The Austrian model for world development: John Komlos' neoklassisches Modell. Eine Kritik

The article opens a debate on a book by John Komlos dealing with the economic history of the eighteenth-century Habsburg Monarchy. Rebel discusses Komlos' approach in regard to his concept of the Industrial Revolution and an ,,anthropometric history", which he classifies as „neoclassical" and „neo- Malthusian". Both Komlos' empirical basis - the height measurement of Austrian recruits - and his approach of establishing a correlation between physical height and national income are seen as problematic. The validity of some of the regional samples as well as the resulting calculations are questioned. Rebel doubts Komlos' thesis of a nutritional crisis of the population as a possible cause of the economic measures by the Austrian government which prepared the way for the lndustrial Revolution. Consequently this concept, which Komlos claimed to be a general model for world development, is rejected by Rebel also as a pattern of industrialization.The article opens a debate on a book by John Komlos dealing with the economic history of the eighteenth-century Habsburg Monarchy. Rebel discusses Komlos' approach in regard to his concept of the Industrial Revolution and an ,,anthropometric history", which he classifies as „neoclassical" and „neo- Malthusian". Both Komlos' empirical basis - the height measurement of Austrian recruits - and his approach of establishing a correlation between physical height and national income are seen as problematic. The validity of some of the regional samples as well as the resulting calculations are questioned. Rebel doubts Komlos' thesis of a nutritional crisis of the population as a possible cause of the economic measures by the Austrian government which prepared the way for the lndustrial Revolution. Consequently this concept, which Komlos claimed to be a general model for world development, is rejected by Rebel also as a pattern of industrialization

Loss of CDKN2A and p14ARF expression occurs frequently in human nonmelanoma skin cancers

The CDKN2A locus on human chromosome 9p21 encodes two proteins named p16INK4a and p14ARF, known to function as tumour suppressors via the retinoblastoma (Rb) or the p53 pathway. The p53 tumour suppressor gene is the most commonly mutated gene in human and mouse cancers. Disruption of the p53 and Rb pathways is a fundamental trend of most human cancer cells. Recent studies have shown that the CDKN2A gene plays an active role in the p53 and Rb tumour suppressor pathways. Genetic abnormalities in CDKN2A have been well documented in human melanoma, but their involvement in nonmelanoma skin cancer (NMSC) is less clear