645 research outputs found
High "Normal" blood glucose is associated with decreased brain volume and cognitive performance in the 60s: the PATH through Life Study
- Author
- A DeVisser
- A Di Carlo
- A Klein
- A Ott
- AL van Harmelen
- AM Stranahan
- Andrew L. Janke
- B Mezuk
- C Messier
- CR Savage
- D Wechsler
- DC Delis
- EW Gregg
- F Dentali
- FF Johansen
- Friedemann Paul
- GM Halliday
- J Ashburner
- J Ashburner
- JA Luchsinger
- JB Saunders
- JG Sled
- K Esposito
- K van der Borght
- KA Darvall
- Kaarin J. Anstey
- L Bergouignan
- LJ Launer
- M Vanhanen
- Moyra E. Mortby
- N Awad
- N Cherbuin
- N Vasic
- Nicolas Cherbuin
- Perminder S. Sachdev
- RI Scahill
- SE Vermeer
- SH Alzahrani
- SM Haffner
- SM Manschot
- SM Resnick
- T den Heijer
- VR Vaidyula
- WJ Mack
- Y Araki
- Y Yuan
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/09/2013
- Field of study
Context:Type 2 diabetes is associated with cerebral atrophy, cognitive impairment and dementia. We recently showed higher glucose levels in the normal range not to be free of adverse effects and to be associated with greater hippocampal and amygdalar atrophy in older community-dwelling individuals free of diabetes.Objective:This study aimed to determine whether blood glucose levels in the normal range
Search for new phenomena in final states with an energetic jet and large missing transverse momentum in pp collisions at √ s = 8 TeV with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abud AA
- Abulaiti Y
- Acharya BS
- Achkar B
- Adachi S
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
- Adersberger M
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmadov F
- Ahmed WS
- Ai X
- Aielli G
- Akatsuka S
- Akesson TPA
- Akilli E
- Akimov A
- Al Khoury K
- Alberghi GL
- Albert J
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Alhroob M
- Ali B
- Aliev M
- Alimonti G
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alvarez Estevez M
- Alviggi MG
- Amaral Coutinho Y
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amor Dos Santos SP
- Amoroso S
- Amrouche CS
- An F
- Anastopoulos C
- Andari N
- Andeen T
- Anders CF
- Anders JK
- Andreazza A
- Andrei V
- Anelli CR
- Angelidakis S
- Angerami A
- Anisenkov A
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Antrim DJA
- Anulli F
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- Aparisi Pozo JA
- Araque JP
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- Arduh FA
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- Argyropoulos S
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- Svatos M
- Swiatlowski M
- Swift SP
- Swirski T
- Sydorenko A
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- Thompson PD
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- Tikhomirov VO
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- Tobon CAM
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- Tong B
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- Toro RC
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- Torres RET
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- Tovey DR
- Traeet A
- Treado CJ
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- Trigger IM
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- Tseng JC-L
- Tsiareshka P
- Tsirigotis A
- Tsiskaridze V
- Tskhadadze EG
- Tsopoulou M
- Tsukerman II
- Tsulaia V
- Tsuno S
- Tsybychev D
- Tu Y
- Tudorache A
- Tudorache V
- Tulbure TT
- Tuna AN
- Turchikhin S
- Turgeman D
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- Urban J
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- Usai G
- Uysal Z
- Vacek V
- Vachon B
- Vadla KOH
- Vaidya A
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- Vale MAB
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- Valentinetti S
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- Valery L
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- Van Vulpen I
- Vanadia M
- Vandelli W
- Vandenbroucke M
- Vandewall ER
- Vaniachine A
- Vannicola D
- Vari R
- Varnes EW
- Varni C
- Varol T
- Varouchas D
- Varvell KE
- Vasile ME
- Vasquez GA
- Vazeille F
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- Vazquez CJB
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- Viehhauser GHA
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- Vincter MG
- Virdee GS
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- von Buddenbrock SE
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- Vorobel V
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- Vos M
- Vossebeld JH
- Vozak M
- Vranjes N
- Vrba V
- Vreeswijk M
- Vuillermet R
- Vukotic I
- Wagner P
- Wagner W
- Wagner-Kuhr J
- Wahdan S
- Wahlberg H
- Walbrecht VM
- Walder J
- Walker R
- Walker SD
- Walkowiak W
- Wallangen V
- Wang AM
- Wang AZ
- Wang C
- Wang F
- Wang H
- Wang J
- Wang P
- Wang Q
- Wang R
- Wang R-J
- Wang SM
- Wang W
- Wang WT
- Wang WX
- Wang Y
- Wang Z
- Wanotayaroj C
- Warburton A
- Ward CP
- Wardrope DR
- Warrack N
- Washbrook A
- Watson AT
- Watson MF
- Watts G
- Waugh BM
- Webb AF
- Webb S
- Weber C
- Weber MS
- Weber SA
- Weber SM
- Weidberg AR
- Weingarten J
- Weirich M
- Weiser C
- Wells PS
- Wenaus T
- Wengler T
- Wenig S
- Wermes N
- Werner MD
- Wessels M
- Weston TD
- Whalen K
- Whallon NL
- Wharton AM
- White A
- White AS
- White MJ
- Whiteson D
- Whitmore BW
- Wiedenmann W
- Wiel C
- Wielers M
- Wieseotte N
- Wiglesworth C
- Wiik-Fuchs LAM
- Wilkens HG
- Wilkins LJ
- Williams HH
- Williams S
- Willis C
- Willocq S
- Wingerter-Seez I
- Winkels E
- Winklmeier F
- Winston OJ
- Winter BT
- Wittgen M
- Wobisch M
- Wolf A
- Wolf TMH
- Wolff R
- Wolker R
- Wollrath J
- Wolter MW
- Wolters H
- Wong VWS
- Woods NL
- Worm SD
- Wosiek BK
- Wozniak KW
- Wraight K
- Wu SL
- Wu X
- Wu Y
- Wyatt TR
- Wynne BM
- Xella S
- Xi Z
- Xia L
- Xiao X
- Xiotidis I
- Xu D
- Xu H
- Xu L
- Xu T
- Xu W
- Xu Z
- Yabsley B
- Yacoob S
- Yajima K
- Yallup DP
- Yamaguchi N
- Yamaguchi Y
- Yamamoto A
- Yamatani M
- Yamazaki T
- Yamazaki Y
- Yan Z
- Yang HJ
- Yang HT
- Yang S
- Yang T
- Yang X
- Yang Y
- Yao W-M
- Yap YC
- Yasu Y
- Yatsenko E
- Ye H
- Ye J
- Ye S
- Yeletskikh I
- Yexley MR
- Yigitbasi E
- Yildiz HD
- Yorita K
- Yoshihara K
- Young C
- Young CJS
- Yu J
- Yuan R
- Yue X
- Zaazoua M
- Zabinski B
- Zacharis G
- Zaffaroni E
- Zahreddine J
- Zaitsev AM
- Zakareishvili T
- Zakharchuk N
- Zambito S
- Zanzi D
- Zaripovas DR
- Zeissner S
- Zeitnitz C
- Zemaityte G
- Zeng JC
- Zenin O
- Zenis T
- Zerwas D
- Zgubic M
- Zhang B
- Zhang DF
- Zhang G
- Zhang H
- Zhang J
- Zhang L
- Zhang M
- Zhang R
- Zhang S
- Zhang X
- Zhang Y
- Zhang Z
- Zhao P
- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
- Zhou B
- Zhou C
- Zhou M
- Zhou MS
- Zhou N
- Zhou Y
- Zhu C
- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine N
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zivkovic L
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zu Theenhausen HM
- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
Results of a search for new phenomena in final states with an energetic jet and large missing transverse momentum are reported. The search uses 20.3 fb−1 of √ s = 8 TeV data collected in 2012 with the ATLAS detector at the LHC. Events are required to have at least one jet with pT > 120 GeV and no leptons. Nine signal regions are considered with increasing missing transverse momentum requirements between Emiss T > 150 GeV and Emiss T > 700 GeV. Good agreement is observed between the number of events in data and Standard Model expectations. The results are translated into exclusion limits on models with either large extra spatial dimensions, pair production of weakly interacting dark matter candidates, or production of very light gravitinos in a gauge-mediated supersymmetric model. In addition, limits on the production of an invisibly decaying Higgs-like boson leading to similar topologies in the final state are presente
HIV-Specific Antibodies Capable of ADCC Are Common in Breastmilk and Are Associated with Reduced Risk of Transmission in Women with High Viral Loads
- Author
- A Coutsoudis
- A Piantadosi
- AJ Hessell
- AS Goldman
- B Bottiger
- BL Lohman
- C Duprat
- C Verhofstede
- CA Blish
- CA Blish
- CF Barbas 3rd
- CM Rousseau
- CM Rousseau
- D Ott
- DD Panteleeff
- DH Barouch
- DN Forthal
- DN Forthal
- DN Forthal
- DN Forthal
- DN Sather
- DT Dunn
- Dylan Peterson
- E Tuaillon
- EM Fenyo
- EM Long
- EM Obimbo
- ES Russell
- F Barin
- FX Lü
- G John-Stewart
- G Kafulafula
- G Scarlatti
- G Scarlatti
- GC John
- GG Fouda
- H Zhang
- HJ Hosea Blewett
- HL Yilmaz
- I Thior
- J Homsy
- J Kagaayi
- J Neilson
- JB Lynch
- Jennifer Mabuka
- JH Humphrey
- JL Mellquist
- JR Fiore
- Julie Overbaugh
- K Battle-Miller
- K Semrau
- Katherine Odem-Davis
- L Bélec
- L Kuhn
- L Kuhn
- L Kuhn
- L Kuhn
- LL Baum
- M Poss
- M Raux
- M Sagar
- MA Naarding
- MJ Pineda
- MT Asensi
- N Ahmad
- ND Banks
- P Becquart
- P Nowak
- P Xiao
- R Ahmad
- R Bosire
- R Dickover
- R Garofalo
- R Nduati
- RE Dickover
- RL Hengel
- RM Lawrence
- RN Husson
- Ronald C. Desrosiers
- Ruth Nduati
- S Sabbaj
- SC Kliks
- SL Lamers
- SM Wolinsky
- T Doherty
- T Hirbod
- T Horvath
- T Samleerat
- T Taha
- TL Creek
- VR Gomez-Roman
- VR Gomez-Roman
- WH Oddy
- X Wu
- X Yang
- Y Sun
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2012
- Field of study
There are limited data describing the functional characteristics of HIV-1 specific antibodies in breast milk (BM) and their role in breastfeeding transmission. The ability of BM antibodies to bind HIV-1 envelope, neutralize heterologous and autologous viruses and direct antibody-dependent cell cytotoxicity (ADCC) were analyzed in BM and plasma obtained soon after delivery from 10 non-transmitting and 9 transmitting women with high systemic viral loads and plasma neutralizing antibodies (NAbs). Because subtype A is the dominant subtype in this cohort, a subtype A envelope variant that was sensitive to plasma NAbs was used to assess the different antibody activities. We found that NAbs against the subtype A heterologous virus and/or the woman's autologous viruses were rare in IgG and IgA purified from breast milk supernatant (BMS) – only 4 of 19 women had any detectable NAb activity against either virus. Detected NAbs were of low potency (median IC50 value of 10 versus 647 for the corresponding plasma) and were not associated with infant infection (p = 0.58). The low NAb activity in BMS versus plasma was reflected in binding antibody levels: HIV-1 envelope specific IgG titers were 2.2 log10 lower (compared to 0.59 log10 lower for IgA) in BMS versus plasma. In contrast, antibodies capable of ADCC were common and could be detected in the BMS from all 19 women. BMS envelope-specific IgG titers were associated with both detection of IgG NAbs (p = 0.0001)and BMS ADCC activity (p = 0.014). Importantly, BMS ADCC capacity was inversely associated with infant infection risk (p = 0.039). Our findings indicate that BMS has low levels of envelope specific IgG and IgA with limited neutralizing activity. However, this small study of women with high plasma viral loads suggests that breastmilk ADCC activity is a correlate of transmission that may impact infant infection risk
Nurses' experiences, expectations, and preferences for mind-body practices to reduce stress
- Author
- A Alacacioglu
- A Chiesa
- A Chiesa
- AC Barron
- AE Beddoe
- BS McEwen
- CS Mackenzie
- D Koszycki
- D McElligott
- Deborah Krueger
- DW Smith
- EC de Carvalho
- F Zeidan
- G Elkins
- G Pagnoni
- GS Birdee
- J Cohen-Katz
- J Cohen-Katz
- J Cohen-Katz
- Jane A McCool
- JE Emery
- JR Halbesleben
- K Kemper
- K McCabe Ruff
- K Richards
- Kathi Kemper
- KJ Kemper
- KM Hayes
- L Keegan
- L Poghosyan
- LE Carlson
- LM Bossi
- M Braithwaite
- M King
- M Ramirez
- M Verdon
- MA Hays
- Mary Jane Ott
- MC Poncet
- MF Tracy
- MF Tracy
- MI Fitch
- MJ Kreitzer
- MS Krasner
- NE Schoenberger
- P Goffaux
- Paula Gardiner
- Preference Collaborative Review G
- R Chattha
- R Lindquist
- R Tang
- RB Wall
- RE Graham
- RE Taylor-Piliae
- S Cutshall
- S Rosenzweig
- S Telles
- Sally Bulla
- SBS Khalsa
- SC Danhauer
- T Mitchell
- VA Barnes
- VR Aggarwal
- WR Davies
- Publication venue
- BioMed Central
- Publication date
- 01/01/2011
- Field of study
BACKGROUND: Most research on the impact of mind-body training does not ask about participants\u27 baseline experience, expectations, or preferences for training. To better plan participant-centered mind-body intervention trials for nurses to reduce occupational stress, such descriptive information would be valuable.
METHODS: We conducted an anonymous email survey between April and June, 2010 of North American nurses interested in mind-body training to reduce stress. The e-survey included: demographic characteristics, health conditions and stress levels; experiences with mind-body practices; expected health benefits; training preferences; and willingness to participate in future randomized controlled trials.
RESULTS: Of the 342 respondents, 96% were women and 92% were Caucasian. Most (73%) reported one or more health conditions, notably anxiety (49%); back pain (41%); GI problems such as irritable bowel syndrome (34%); or depression (33%). Their median occupational stress level was 4 (0 = none; 5 = extreme stress). Nearly all (99%) reported already using one or more mind-body practices to reduce stress: intercessory prayer (86%), breath-focused meditation (49%), healing or therapeutic touch (39%), yoga/tai chi/qi gong (34%), or mindfulness-based meditation (18%). The greatest expected benefits were for greater spiritual well-being (56%); serenity, calm, or inner peace (54%); better mood (51%); more compassion (50%); or better sleep (42%). Most (65%) wanted additional training; convenience (74% essential or very important), was more important than the program\u27s reputation (49%) or scientific evidence about effectiveness (32%) in program selection. Most (65%) were willing to participate in a randomized trial of mind-body training; among these, most were willing to collect salivary cortisol (60%), or serum biomarkers (53%) to assess the impact of training.
CONCLUSIONS: Most nurses interested in mind-body training already engage in such practices. They have greater expectations about spiritual and emotional than physical benefits, but are willing to participate in studies and to collect biomarker data. Recruitment may depend more on convenience than a program\u27s scientific basis or reputation. Knowledge of participants\u27 baseline experiences, expectations, and preferences helps inform future training and research on mind-body approaches to reduce stress
Metabolic Regulation in Progression to Autoimmune Diabetes
- Author
- A Giongo
- A Kupila
- A Subramanian
- A Tripathi
- A Zhernakova
- Abhishek Tripathi
- AK Jain
- Andrey Ermolov
- AV Chervonsky
- B Freiesleben De Blasio
- B Keymeulen
- B Singh
- BM Bolstad
- CC Patterson
- CJ Fox
- DL Eizirik
- DL Eizirik
- E Hypponen
- EAM Gale
- Eriika Savontaus
- Erno Lindfors
- F Ortis
- G Matarese
- G Matarese
- GK Smyth
- GL Szot
- GM Lord
- H Nygren
- H Sokol
- I Truyen
- Ismo Mattila
- J Maukonen
- J Maukonen
- J Nikkilä
- J Ostman
- J Seufert
- Janne Nikkilä
- JCJ Floyd
- JD Powell
- JD Storey
- JD Storey
- Jennifer L. Reed
- Johanna Maukonen
- Jorma Ilonen
- K Atarashi
- L Ozcan
- L Wen
- L Yetukuri
- Laura Vähätalo
- Laxman Yetukuri
- M Ashburner
- M Kanehisa
- M Katajamaa
- M Katajamaa
- M Oresic
- M Oresic
- M Ott
- M Ozata
- M Rapoport
- MA Atkinson
- Maria Saarela
- Marko Sysi-Aho
- Matej Orešič
- Mikael Knip
- MJ Dunning
- MS Anderson
- MS Ronkainen
- N Salomonis
- Olli Simell
- P Achenbach
- P Newsholme
- P Xu
- Peddinti V. Gopalacharyulu
- R Gentleman
- R Laaksonen
- RJ Chaparro
- RM Satokari
- S Castillo
- S Fourlanos
- S Mrena
- S Sakaguchi
- S Sharma
- Samuel Kaski
- SD Allison
- Suvi T. Ruohonen
- SYS Kimm
- T Calzascia
- T Kadowaki
- Taina Härkönen
- Tuulikki Seppänen-Laakso
- U Nevo
- V De Rosa
- V Harjutsalo
- VR Velagapudi
- W Welthagen
- WA Hagopian
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
Recent evidence from serum metabolomics indicates that specific metabolic disturbances precede β-cell autoimmunity in humans and can be used to identify those children who subsequently progress to type 1 diabetes. The mechanisms behind these disturbances are unknown. Here we show the specificity of the pre-autoimmune metabolic changes, as indicated by their conservation in a murine model of type 1 diabetes. We performed a study in non-obese prediabetic (NOD) mice which recapitulated the design of the human study and derived the metabolic states from longitudinal lipidomics data. We show that female NOD mice who later progress to autoimmune diabetes exhibit the same lipidomic pattern as prediabetic children. These metabolic changes are accompanied by enhanced glucose-stimulated insulin secretion, normoglycemia, upregulation of insulinotropic amino acids in islets, elevated plasma leptin and adiponectin, and diminished gut microbial diversity of the Clostridium leptum group. Together, the findings indicate that autoimmune diabetes is preceded by a state of increased metabolic demands on the islets resulting in elevated insulin secretion and suggest alternative metabolic related pathways as therapeutic targets to prevent diabetes
Gut microbiota and diabetes: from pathogenesis to therapeutic perspective
- Author
- A Bjorneklett
- A Cerutti
- A Cerutti
- A Green
- A Velayudham
- AC Vreugdenhil
- AL Goodman
- BE Gustafsson
- C King
- C Knauf
- C Levy-Marchal
- C Manichanh
- C Maziere
- C Mering von
- C Palmer
- C Postic
- CC Patterson
- CF Favier
- Chantal Chabo
- CJ Wiedermann
- CM Edwards
- CP Day
- D Knights
- D Thabut
- DD Black
- DW Han
- E Esposito
- E Firatli
- ED Sonnenburg
- EF Murphy
- EG Zoetendal
- EG Zoetendal
- F Backhed
- F Backhed
- F Backhed
- F D’Aiuto
- F Guarner
- G Dalmasso
- G Danaei
- G Eisenbarth
- G Musso
- GC Baker
- GR Gibson
- GS Hotamisligil
- H Buchwald
- H Löe
- H Zhang
- HW Harris
- HY Zhang
- I Pappo
- J Amar
- J Amar
- J McCarthy
- J Medina
- J Qin
- J Slots
- J Tap
- Jacques Amar
- JC Pickup
- JG Caporaso
- JH Fritz
- JM Neefs
- JM Wells
- JP Furet
- K Hettne
- KM Pantalone
- L Bry
- L Darré
- L Dethlefsen
- L Dethlefsen
- L Miller
- L Wen
- LG Ooi
- LV Hooper
- LV Hooper
- M Boirivant
- M Bukoff
- M Feuerer
- M Kalliomaki
- M Kanehisa
- M Membrez
- M Oresic
- M Poggi
- M Roberfroid
- M Schenk
- M Serino
- M Soell
- M Spencer
- M Vijay-Kumar
- M Westerterp
- Matteo Serino
- MB Roberfroid
- MC Collado
- ME Dumas
- ME Johansson
- MF McInerney
- MJ Claesson
- MJ Song
- MT Asquith
- MW Sadelain
- N Doan
- N Larsen
- N Osman
- O Koren
- P Gerard
- P Pozzilli
- P Turnbaugh
- PC Rensen
- PD Cani
- PD Cani
- PD Cani
- PD Cani
- PG Holt
- PG Holt
- PJ Turnbaugh
- PJ Turnbaugh
- PJ Turnbaugh
- Q Wang
- R Andraws
- R Assan
- R Caesar
- R Calvani
- R Ley
- R Menghini
- R Valladares
- RD Heijtz
- RE Ley
- RE Ley
- RE Ley
- RE Ley
- RE Ley
- RG Burcelin
- RI Amann
- RI Mackie
- Rémy Burcelin
- S Akira
- S Brugman
- S Caspar-Bauguil
- S Ding
- S Epstein
- S Fukuda
- S Ghoshal
- S Grossi
- S Hapfelmeier
- S Kondo
- S Menard
- S Ott
- S Rakoff-Nahoum
- S Shoelson
- S Winer
- SA Kocaman
- SK Mazmanian
- SM Ruchat
- SP Weisberg
- T Pischon
- T Saito
- TA Kufer
- V Gaboriau-Routhiau
- Vincent Blasco-Baque
- VO Rotimi
- VR Velagapudi
- WHO
- Y Fukushima
- Y Nishizawa
- YH Paik
- ZL Kumwenda
- Publication venue
- Springer Milan
- Publication date
- 01/01/2011
- Field of study
More than several hundreds of millions of people will be diabetic and obese over the next decades in front of which the actual therapeutic approaches aim at treating the consequences rather than causes of the impaired metabolism. This strategy is not efficient and new paradigms should be found. The wide analysis of the genome cannot predict or explain more than 10–20% of the disease, whereas changes in feeding and social behavior have certainly a major impact. However, the molecular mechanisms linking environmental factors and genetic susceptibility were so far not envisioned until the recent discovery of a hidden source of genomic diversity, i.e., the metagenome. More than 3 million genes from several hundreds of species constitute our intestinal microbiome. First key experiments have demonstrated that this biome can by itself transfer metabolic disease. The mechanisms are unknown but could be involved in the modulation of energy harvesting capacity by the host as well as the low-grade inflammation and the corresponding immune response on adipose tissue plasticity, hepatic steatosis, insulin resistance and even the secondary cardiovascular events. Secreted bacterial factors reach the circulating blood, and even full bacteria from intestinal microbiota can reach tissues where inflammation is triggered. The last 5 years have demonstrated that intestinal microbiota, at its molecular level, is a causal factor early in the development of the diseases. Nonetheless, much more need to be uncovered in order to identify first, new predictive biomarkers so that preventive strategies based on pre- and probiotics, and second, new therapeutic strategies against the cause rather than the consequence of hyperglycemia and body weight gain
Search for Dark Matter and Supersymmetry with a Compressed Mass Spectrum in the Vector Boson Fusion Topology in Proton-Proton Collisions at root s=8 TeV
- Author
- Aarrestad TK
- Abbaneo D
- Abbiendi G
- Abbrescia M
- Abdullin S
- Abdulsalam A
- Abu Zeid S
- Ackert A
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- Publication venue
- Publication date
- 01/01/2016
- Field of study
Peer reviewe
Pan-cancer analysis of whole genomes
- Author
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- ya Al-Ahmadie
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- Yamaguchi T. N. k
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- Zhang
- Zhang
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- Zhao
- Zheng
- Zhou
- Zhou
- Zhu
- Zhu
- zi zj
- zm Jayaseelan
- Zou
- Zou
- zt Kim
- zw Lewis
- Ó. A. th
- Ø. sk
- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
Iron Behaving Badly: Inappropriate Iron Chelation as a Major Contributor to the Aetiology of Vascular and Other Progressive Inflammatory and Degenerative Diseases
- Author
- A Abbott
- A Abou-Raya
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- Y Almog
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- Y Wu
- Y Zhang
- Y Zhang
- Y Zhang
- Y Zhao
- Y Zheng
- YE Henrotin
- YF Chu
- YH Lee
- YH Suh
- YH Wei
- YJ Surh
- YJ Surh
- YJ Surh
- YK Wu
- YM Kim
- YS Keum
- YS Sohn
- YX Ma
- YZ Fang
- Z Cai
- Z Cai
- Z Cheng
- Z Pei
- Z Serdar
- Z Tong
- Z Tong
- Z Yu
- Z Zhang
- ZD Liu
- ZD Liu
- ZE Karanjawala
- ZE Suntres
- ZI Cabantchik
- ZZ Chong
- ZZ Chong
- Publication venue
- Publication date
- 09/08/2008
- Field of study
The production of peroxide and superoxide is an inevitable consequence of
aerobic metabolism, and while these particular "reactive oxygen species" (ROSs)
can exhibit a number of biological effects, they are not of themselves
excessively reactive and thus they are not especially damaging at physiological
concentrations. However, their reactions with poorly liganded iron species can
lead to the catalytic production of the very reactive and dangerous hydroxyl
radical, which is exceptionally damaging, and a major cause of chronic
inflammation. We review the considerable and wide-ranging evidence for the
involvement of this combination of (su)peroxide and poorly liganded iron in a
large number of physiological and indeed pathological processes and
inflammatory disorders, especially those involving the progressive degradation
of cellular and organismal performance. These diseases share a great many
similarities and thus might be considered to have a common cause (i.e.
iron-catalysed free radical and especially hydroxyl radical generation). The
studies reviewed include those focused on a series of cardiovascular, metabolic
and neurological diseases, where iron can be found at the sites of plaques and
lesions, as well as studies showing the significance of iron to aging and
longevity. The effective chelation of iron by natural or synthetic ligands is
thus of major physiological (and potentially therapeutic) importance. As
systems properties, we need to recognise that physiological observables have
multiple molecular causes, and studying them in isolation leads to inconsistent
patterns of apparent causality when it is the simultaneous combination of
multiple factors that is responsible. This explains, for instance, the
decidedly mixed effects of antioxidants that have been observed, etc...Comment: 159 pages, including 9 Figs and 2184 reference
Search for heavy neutral Higgs bosons produced in association with b-quarks and decaying into b-quarks at root s=13 TeV with the ATLAS detector
- Author
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- Zaripovas DR
- Zeiner S
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- Zhemchugov A
- Zheng Z
- Zhong D
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- Zorbas TG
- Zou R
- Zu Theenhausen HM
- Zwalinski L
- Publication venue
- AMER PHYSICAL SOC
- Publication date
- 13/08/2020
- Field of study
A search for heavy neutral Higgs bosons produced in association with one or two
b
-quarks and decaying to
b
-quark pairs is presented using
27.8
fb
−
1
of
√
s
=
13
TeV
proton-proton collision data recorded by the ATLAS detector at the Large Hadron Collider during 2015 and 2016. No evidence of a signal is found. Upper limits on the heavy neutral Higgs boson production cross section times its branching ratio to
b
¯
b
are set, ranging from 4.0 to 0.6 pb at 95% confidence level over a Higgs boson mass range of 450 to 1400 GeV. Results are interpreted within the two-Higgs-doublet model and the minimal supersymmetric Standard Model
- …