70 research outputs found
Genetic Associations of Type 2 Diabetes with Islet Amyloid Polypeptide Processing and Degrading Pathways in Asian Populations
- Author
- A Ragvin
- AE Kel
- Alice Pik Shan Kong
- Andrew P. Morris
- AV Matveyenko
- BF Voight
- C Lange
- Cheng Hu
- CJ Groves
- CJ Huang
- Claudia Ha Ting Tam
- D Qi
- Daniel Peng-Keat Ng
- DF Steiner
- E Zeggini
- E. Shyong Tai
- ET Jaikaran
- Gang Xu
- H Chen
- H Zhao
- Heung Man Lee
- Hiroto Furuta
- HL Zhao
- Hong Kyu Lee
- I Prokopenko
- J Paulsson
- JC Barrett
- JC Chan
- Jean Woo
- Jianjun Liu
- JK Li
- JK Wolford
- JP Noonan
- Juliana Chung Ngor Chan
- JZ Bai
- K Piwernetz
- KD Jeffrey
- KH Liu
- Kishio Nanjo
- KJ Gaulton
- Kyong Soo Park
- LJ Martin
- M He
- M Stumvoll
- Maggie Chor Yin Ng
- MB Pepys
- MC Ng
- MC Ng
- MI McCarthy
- MK Kim
- ML Stitzel
- MS German
- N Rudovich
- N Utsunomiya
- Nelson Leung Sang Tang
- P Westermark
- Ping Chung Leung
- Q Yang
- R Bhandare
- R DerSimonian
- RG Bennett
- Risa Ozaki
- Ronald Ching Wan Ma
- S Nordman
- SB Harrap
- SH Kwak
- Shiro Maeda
- Tien-Yin Wong
- TS Leak
- V Steinthorsdottir
- Vincent Kwok Lim Lam
- Wei Ping Jia
- Wing-Yee So
- WP Jia
- X Sim
- XL Li
- Xueling Sim
- Y Cai
- Y Furukawa
- Y Wang
- Ying Wang
- YS Cho
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2013
- Field of study
10.1371/journal.pone.0062378PLoS ONE86
Impact of laryngopharyngeal reflux on subjective and objective voice assessments: a prospective study
- Author
- A Printza
- AL Hamdan
- B Naiboglu
- BJ Chun
- BJ Jin
- C Hopkins
- CF Smit
- DA Vincent Jr
- DT Book
- F Ogut
- GY Shaw
- I Garcia
- J Koufman
- J Schoentgen
- JA Koufman
- JA Koufman
- JA Koufman
- JA Koufman
- JA Ross
- JC Selby
- JO Park
- JR Lechien
- K Vashani
- KH Park
- LE Rees
- M Masaany
- M Sereg-Bahar
- M Yana
- MA Corvo
- N Siupsinskiene
- ND Gregory
- O Ozturk
- P Sen
- PC Belafsky
- PC Belafsky
- PO Katz
- R Pribuisiene
- R Vardar
- RC Mamede
- RJ Toohill
- RT Sataloff
- S Vaz Freitas
- Y Maryn
- YS Lee
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Dendritic excitation–inhibition balance shapes cerebellar output during motor behaviour
- Author
- A Arenz
- A Badura
- BY Chow
- D Jaeger
- D Jaeger
- D Jaeger
- DM Armstrong
- DM Armstrong
- DM Armstrong
- E Galliano
- F Bengtsson
- F Santamaria
- G Andersson
- G Stuart
- G Szapiro
- H Jörntell
- H Jörntell
- H Zhou
- I Duguid
- I Llano
- I Ozden
- IM Raman
- J Chaumont
- J Midtgaard
- J Rodrigo
- J Schiemann
- JC Eccles
- JM Bower
- JT Walter
- K Abitbol
- K Powell
- KH Lee
- L Witter
- M Hausser
- M Ito
- M Udo
- M Womack
- MJ Dizon
- NH Barmack
- O Yizhar
- P Chadderton
- P Vincent
- P Wulff
- PJ Mathews
- PM Blazquez
- Q He
- R Llinas
- R Llinas
- RD Howell
- RL Leshan
- S Shinomoto
- SA Edgley
- SA Heiney
- SM Park
- TM Hoogland
- V Yakhnitsa
- W Graf
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
Feedforward excitatory and inhibitory circuits regulate cerebellar output, but how these circuits interact to shape the somatodendritic excitability of Purkinje cells during motor behaviour remains unresolved. Here we perform dendritic and somatic patch-clamp recordings in vivo combined with optogenetic silencing of interneurons to investigate how dendritic excitation and inhibition generates bidirectional (that is, increased or decreased) Purkinje cell output during self-paced locomotion. We find that granule cells generate a sustained depolarization of Purkinje cell dendrites during movement, which is counterbalanced by variable levels of feedforward inhibition from local interneurons. Subtle differences in the dendritic excitation–inhibition balance generate robust, bidirectional changes in simple spike (SSp) output. Disrupting this balance by selectively silencing molecular layer interneurons results in unidirectional firing rate changes, increased SSp regularity and disrupted locomotor behaviour. Our findings provide a mechanistic understanding of how feedforward excitatory and inhibitory circuits shape Purkinje cell output during motor behaviour
PALB2, CHEK2 and ATM rare variants and cancer risk: data from COGS
- Author
- Aben KK
- Aittomäki K
- Akushevich L
- Alnæs GG
- Alonso MR
- Ambrosone CB
- Andrulis I
- Anton-Culver H
- Antonenkova NN
- Arndt V
- Ashworth A
- Australian Ovarian Cancer Study Group
- Bacot F
- Bandera EV
- Batra J
- Baynes C
- Bean Y
- Beckmann MW
- Benitez J
- Benlloch S
- Berchuck A
- Bernard L
- Beuselinck B
- Bisogna M
- Bjorge L
- Blomqvist C
- Blot WJ
- Bogdanova NV
- Bojesen S
- Bolla MK
- Bonanni B
- Brand JS
- Brauch H
- Brenner H
- Broeks A
- Brooks-Wilson A
- Brüning T
- Burwinkel B
- Butterbach K
- Butzow R
- Cai H
- Campbell I
- Cannon-Albright L
- Carpenter J
- Catucci I
- Chang-Claude J
- Chanock SJ
- Chen YA
- Chen Z
- Chenevix-Trench G
- Claes KB
- Clarke C
- Clarke CA
- Collée JM
- Cook LS
- Couch F
- Cox A
- Cramer DW
- Cunningham JM
- Cybulski C
- Czene K
- Dansonka-Mieszkowska A
- Dao F
- Darabi H
- Dennis J
- Devilee P
- Dicks E
- Doherty JA
- Donovan JL
- Dos-Santos-Silva I
- Dowty JG
- du Bois A
- Dumont M
- Dunning AM
- Durda K
- Dörk T
- Easton DF
- Eccles DM
- Edwards RP
- Eeles R
- Eilber U
- Ekici AB
- Engelholm SA
- Eriksson M
- Fasching PA
- Fenstermacher D
- Figueroa J
- Flesch-Janys D
- Fletcher O
- Floris G
- Flyger H
- Fogarty ZC
- Foulkes WD
- Fridley BL
- Friel G
- Garcia-Closas M
- Gayther SA
- Gentry-Maharaj A
- Gerty SM
- Giles GG
- Glasspool R
- Glendon G
- Goldgar DE
- González-Neira A
- Goode EL
- Goodman MT
- Grip M
- Gronberg H
- Gronwald J
- Guénel P
- Górski B
- Haeberle L
- Haiman CA
- Hall P
- Hamann U
- Hamdy FC
- Hankinson SE
- Harter P
- Hartikainen JM
- Hartmann A
- Heikkinen T
- Hein A
- Heitz F
- Henderson BE
- Herrero D
- Hildebrandt MA
- Hillemanns PA
- Hogervorst FB
- Hollestelle A
- Hooning MJ
- Hopper JL
- Humphreys K
- Hunter DJ
- Høgdall CK
- Høgdall E
- Investigators K
- Iversen ES
- Jakubowska A
- Jaworska K
- Jensen A
- Johnson N
- Jones M
- Jukkola-Vuorinen A
- Kalli KR
- Kaneva R
- Karlan BY
- Kataja V
- Kauppila S
- Kelemen LE
- Kelley JL
- Kerin MJ
- Khaw KT
- Kibel AS
- Kiemeney LA
- Killeen JL
- Kjaer SK
- Knight JA
- Ko YD
- Kommoss S
- Kosma VM
- Kote-Jarai Z
- Kraft P
- Kristensen V
- Kupryjanczyk J
- Lambrechts D
- Lambretchs S
- Larson MC
- Le Marchand L
- Le ND
- Lester J
- Levine DA
- Li J
- Liang D
- Lin HY
- Lindblom A
- Lindstrom S
- Lissowska J
- Lu KH
- Lubinski J
- Luccarini C
- Lundvall L
- Lurie G
- Maier C
- Mannermaa A
- Margolin S
- Marks JR
- Marme F
- Martens JW
- Massuger LF
- McGuire V
- McLaughlin JR
- McLean C
- McNeish I
- Menegaux F
- Menkiszak J
- Menon U
- Menéndez P
- Michailidou K
- Miller N
- Milne RL
- Modugno F
- Moes-Sosnowska J
- Monteiro AN
- Moysich KB
- Muir K
- Mulligan AM
- Muranen TA
- Narod SA
- Neal D
- Nedergaard L
- Ness RB
- Neuhausen S
- Nevanlinna H
- Nickels S
- Nielsen SF
- Nordestgaard BG
- Odefrey FA
- Odunsi K
- Olama AA
- Olson JE
- Olson SH
- Orlow I
- Orr N
- Pankratz VS
- Park J
- Paul J
- Pearce CL
- Peissel B
- Pejovic T
- Pelttari LM
- Perez JI
- Permuth JB
- Peterlongo P
- Peto J
- Pharoah PD
- Phelan CM
- Pike MC
- Pita G
- Plisiecka-Halasa J
- Poole EM
- Pylkäs K
- Radice P
- Rafiq S
- Ramus SJ
- Reed MW
- Reis-Filho J
- Renner SP
- Rossing MA
- Rothstein JH
- Rudolph A
- Ruebner M
- Runnebaum I
- Rüdiger T
- Salvesen HB
- Sanchez M
- Sawyer EJ
- Schaid DJ
- Schildkraut J
- Schleutker J
- Schmidt MK
- Schumacher F
- Scott RJ
- Seibold P
- Sellers TA
- Seynaeve C
- Shu XO
- Siddiqui N
- Sieh W
- Simard J
- Slager S
- Smeets D
- Song H
- Soucy P
- Southey MC
- Spurdle AB
- Stampfer M
- Stanford JL
- Stegmaier C
- Sucheston-Campbell LE
- Sutphen R
- Swerdlow A
- Tapper WJ
- Tavtigian SV
- Teixeira MR
- Teo ZL
- Terry KL
- Tessier DC
- Teten RT
- Thibodeau S
- Tilanus-Linthorst M
- Tischkowitz M
- Toland AE
- Tollenaar RA
- Tomlinson I
- Torres D
- Travis RC
- Truong T
- Tsai YY
- Tsimiklis H
- Tworoger SS
- Tyrer J
- Ulmer HU
- Vachon C
- van Altena AM
- Van Den Berg D
- Van Nieuwenhuysen E
- van Rensburg EJ
- Vergote I
- Verhoef S
- Vierkant RA
- Vincent D
- Walsh C
- Wang-Gohrke S
- Wauters E
- Weischer M
- Whittemore AS
- Wiklund F
- Wilkens LR
- Winqvist R
- Winship I
- Wu AH
- Wu X
- Yang R
- Yannoukakos D
- Zamora MP
- Zheng W
- Ziogas A
- Álvarez N
- Publication venue
- Publication date
- 01/01/2016
- Field of study
Background: The rarity of mutations in PALB2, CHEK2 and ATM make it difficult to estimate precisely associated cancer risks. Population-based family studies have provided evidence that at least some of these mutations are associated with breast cancer risk as high as those associated with rare BRCA2 mutations. We aimed to estimate the relative risks associated with specific rare variants in PALB2, CHEK2 and ATM via a multicentre case-control study.Methods: We genotyped 10 rare mutations using the custom iCOGS array: PALB2 c.1592delT, c.2816T>G and c.3113G>A, CHEK2c.349A>G, c.538C>T, c.715G>A, c.1036C>T, c.1312G>T, and c.1343T>G and ATM c.7271T>G. We assessed associations with breast cancer risk (42 671 cases and 42 164 controls), as well as prostate (22 301 cases and 22 320 controls) and ovarian (14 542 cases and 23 491 controls) cancer risk, for each variant.Results: For European women, strong evidence of association with breast cancer risk was observed for PALB2 c.1592delT OR 3.44 (95% CI 1.39 to 8.52, p=7.1×10−5), PALB2 c.3113G>A OR 4.21 (95% CI 1.84 to 9.60, p=6.9×10−8) and ATM c.7271T>G OR 11.0 (95% CI 1.42 to 85.7, p=0.0012). We also found evidence of association with breast cancer risk for three variants in CHEK2, c.349A>G OR 2.26 (95% CI 1.29 to 3.95), c.1036C>T OR 5.06 (95% CI 1.09 to 23.5) and c.538C>T OR 1.33 (95% CI 1.05 to 1.67) (p≤0.017). Evidence for prostate cancer risk was observed for CHEK2 c.1343T>G OR 3.03 (95% CI 1.53 to 6.03, p=0.0006) for African men and CHEK2 c.1312G>T OR 2.21 (95% CI 1.06 to 4.63, p=0.030) for European men. No evidence of association with ovarian cancer was found for any of these variants.Conclusions: This report adds to accumulating evidence that at least some variants in these genes are associated with an increased risk of breast cancer that is clinically important.</p
Early phase of plasticity-related gene regulation and SRF dependent transcription in the hippocampus
- Author
- A De Simoni
- A Etkin
- A Fleischmann
- A Gärtner
- A Ivanov
- A Klejman
- A Kucharska
- A Lindecke
- A Ludwig
- A Ludwig
- A Ma'ayan
- A Ma'ayan
- A Mouravlev
- A Shalizi
- A Tabuchi
- AA Oliva
- AJ Oliveira-Dos-Santos
- AM Bode
- AR Vaillant
- AW Johnson
- B Cheng
- B Mioduszewska
- B Xu
- B Xu
- BD Rowland
- BD Rowland
- BE Porter
- BH Gähwiler
- BH Gähwiler
- BP Curran
- BY Lam
- C Chen
- C Katche
- C Marie-Claire
- C Niu
- C Sala
- C Sala
- C Tárrega
- C Yu
- CE Nunes-Xavier
- Claudio Altafini
- CR Bramham
- CR Bramham
- CR Raymond
- CR Raymond
- CR Weston
- CS Park
- D Canzoniere
- D Han
- D Junghans
- D Passeri
- D Zheng
- daW Huang
- DJ Lockhart
- DK Ma
- DL Moore
- DM Owens
- DP Seeburg
- DS Roberts
- E Benito
- E Kadar
- E Messaoudi
- E Portales-Casamar
- E Yilmaz-Rastoder
- ED Martín
- EE Irvine
- EM Rial Verde
- F Jeanneteau
- FD Broccard
- G Borlikova
- G Chen
- G Lynch
- G Pinato
- G Pinato
- G Turrigiano
- G Turrigiano
- Giovanni Iacono
- GJ Augustine
- GT Williams
- H Bading
- H Cheval
- H Scharfman
- H Yang
- H Yang
- HD MacGillavry
- HD MacGillavry
- I Uchida
- IE Holopainen
- IM Reti
- J Han
- J Jaworski
- J Lee
- J Zhao
- JD Shepherd
- JF Guzowski
- JH Do
- JH Kim
- JH Kotaleski
- JJ Ventura
- JS Francis
- JW Tullai
- K Inokuchi
- K Kalita
- K Kawaai
- K Stolle
- K Yamagata
- KA Slanina
- KC Chung
- KH Kim
- KH Wang
- KJ Lee
- L Li
- LA Garrett-Sinha
- LF Chang
- M Barnat
- M Crispino
- M Decressac
- M Domercq
- M Dragunow
- M Mandl
- M Racaniello
- M Ramsden
- M Schubert
- M VanElzakker
- M Yoo
- Malka Cohen-Armon
- MC Chang
- MG Liu
- MH Mohajerani
- MJ Chen
- MP Kelly
- MP Kelly
- MR Daliri
- MV Olesen
- MW Jones
- N Kamei
- N Kojima
- N Plath
- N Ramanan
- N Sang
- N Volakakis
- NC O'Sullivan
- NR Holm
- O Heidenreich
- OO Sokolova
- P Gass
- P Uvarov
- PA Brooks
- PL Greer
- R O'Brien
- R Otal
- R Poirier
- RA Irizarry
- RJ O'Brien
- RM Hutchison
- S Bhattacharya
- S Bisler
- S Chowdhury
- S Davis
- S Davis
- S Farioli-Vecchioli
- S Farioli-Vecchioli
- S Ha
- S Lähteinen
- S Papadia
- S Pegoraro
- S Peña de Ortiz
- S Reibel
- S Ribeiro
- S Rozen
- S Ryser
- S Ryser
- S Yasuda
- S Zhu
- SA Pintchovski
- SC Danzer
- SD Bamforth
- SD Kuipers
- SJ Zhang
- SJ Zhang
- SJ Zhang
- SW Flavell
- T Celikel
- T Hökfelt
- T Lemberger
- T Pönniö
- T Seredenina
- T Zhang
- TF Dijkmans
- V Kumar
- V Pencea
- V Volmat
- Vincent Torre
- W Sun
- WC Abraham
- WC Sin
- WF Odenwald
- WI Colón-Cesario
- Y Inoue
- Y Kim
- Y Watanabe
- Y Zhu
- YR Gonzalez
- Z Penke
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2013
- Field of study
Hippocampal organotypic cultures are a highly reliable in vitro model for studying neuroplasticity: in this paper, we analyze the early phase of the transcriptional response induced by a 20 \ub5M gabazine treatment (GabT), a GABA-Ar antagonist, by using Affymetrix oligonucleotide microarray, RT-PCR based time-course and chromatin-immuno-precipitation. The transcriptome profiling revealed that the pool of genes up-regulated by GabT, besides being strongly related to the regulation of growth and synaptic transmission, is also endowed with neuro-protective and pro-survival properties. By using RT-PCR, we quantified a time-course of the transient expression for 33 of the highest up-regulated genes, with an average sampling rate of 10 minutes and covering the time interval [10 3690] minutes. The cluster analysis of the time-course disclosed the existence of three different dynamical patterns, one of which proved, in a statistical analysis based on results from previous works, to be significantly related with SRF-dependent regulation (p-value<0.05). The chromatin immunoprecipitation (chip) assay confirmed the rich presence of working CArG boxes in the genes belonging to the latter dynamical pattern and therefore validated the statistical analysis. Furthermore, an in silico analysis of the promoters revealed the presence of additional conserved CArG boxes upstream of the genes Nr4a1 and Rgs2. The chip assay confirmed a significant SRF signal in the Nr4a1 CArG box but not in the Rgs2 CArG box
Standard perioperative management in gastrointestinal surgery
- Author
- A Brauer
- A Brauer
- A Butterly
- A Carignan
- A Donati
- A Gottschalk
- A Qaseem
- A Schweizer
- A Singh
- A Stanley
- A Windsor
- AA Ghaferi
- AB Haynes
- AD Ryding
- AI Duncan
- American Society of Anesthesiologists Task Force on Perioperative Management of Patients with Cardiac Rhythm Management Devices
- B Kabon
- B. Michael Ghadimi
- BC Tsui
- BD Kavanagh
- BG Hammill
- BH Cuthbertson
- C Jambor
- CE Edmiston
- CE Pineda
- CE Schneemilch
- CL Grines
- D Lindstrom
- D Poldermans
- D Ryan
- DC Classen
- Deutsche Gesellschaft für Anästhesiologie und Intensivmedizin DGfIM Deutsche Gesellschaft für Chirurgie
- DO Warner
- DR Flum
- DW Bratzler
- E Bouza
- E Futier
- E Futier
- E Tacconelli
- EN Vries de
- ER Fernandez-Perez
- F Bozzetti
- F Bozzetti
- F Bretagnol
- F Schortgen
- F Schortgen
- FA Manian
- FM Brunkhorst
- FM Williams
- G Berghe van den
- G Imberger
- G Marx
- G Youssef
- GA Hans
- GH Lyman
- GL Ackland
- GL Snyder
- GP Copeland
- GR Park
- GW Smetana
- GW Smetana
- H Kehlet
- H Kehlet
- H Kehlet
- H Kehlet
- H Kehlet
- H Kehlet
- H Kehlet
- H Petrowsky
- HD Young
- HH Feringa
- HH Stone
- I Uckay
- J Benes
- J Gibbs
- J Klein
- JB Froehlich
- JC Santos Jr
- JE Aguilar-Nascimento
- JL Vincent
- JW Blom
- K Holte
- KA Brennan
- KE Fleischmann
- KH Burkitt
- L Magnusson
- LA Fleisher
- M Bundgaard-Nielsen
- M Diana
- M Duggan
- M Licker
- M Scatizzi
- M Tokunaga
- M Werner-Wasik
- MA Rozner
- Marian Grade
- ME Cohen
- Michael Quintel
- MJ Pena-Soria
- MK Diener
- ML Cheatham
- MW Buchler
- MW Cook
- N Lees
- O Ahlers
- O Zmora
- P Burke
- P Forget
- P Michelet
- PF White
- PF White
- PM Markus
- PY Wuethrich
- R Coleman
- R Finkelstein
- R Latham
- R Nelson
- RJ Fotiadis
- RJ Moraca
- RL Koretz
- RP Merkow
- RP Miettinen
- S Benington
- S Finfer
- S Ganai
- S Jhanji
- S Kurosawa
- S Noble
- SE Capes
- SF Jencks
- SF Khuri
- SH Dougherty
- SH Dougherty
- SJ Lewis
- SK Sahai
- SL Cohn
- T Thomsen
- TD Crabtree
- The Acute Respiratory Distress Syndrome Network
- TJ Gan
- V Squadrone
- VA Lawrence
- WG Cheadle
- WG Stevenson
- WH Geerts
- WJ Mauermann
- WM Popescu
- Y Akutsu
- Y Haga
- Y Sakr
- Z Yang
- Publication venue
- Springer-Verlag
- Publication date
- 01/01/2011
- Field of study
Multi-messenger observations of a binary neutron star merger
- Author
- Aab A
- Aartsen MG
- Abbott BP
- Abbott R
- Abbott TD
- Abbott TMC
- Abdalla H
- Abe F
- Abeysekara AU
- Abramo LR
- Abramowski A
- Abreu P
- Acernese F
- Acero F
- Ackermann M
- Ackley K
- Ackley K
- Adams C
- Adams J
- Adams SM
- Adams T
- Addesso P
- Adhikari RX
- Adya VB
- Affeldt C
- Afrough M
- Agarwal B
- Agathos M
- Agatsuma K
- Aggarwal N
- Aglietta M
- Agliozzo C
- Agudo I
- Aguiar OD
- Aguilar JA
- Aharonian F
- Ahlers M
- Ahrens M
- Aiello L
- Ain A
- Ajith P
- Akras S
- Al Samarai I
- Albert A
- Albert A
- Albuquerque IFM
- Albury JM
- Alcaniz JS
- Alexander KD
- Alfaro R
- Alighieri S Di Serego
- Allam S
- Allekotte I
- Allen B
- Allen G
- Allison J
- Allison JR
- Allocca A
- Almela A
- Altin PA
- Altmann D
- Alvarez C
- Alvarez JD
- Alvarez M Dovale
- Alvarez-Muiz J
- Amati L
- Amato A
- An T
- Ananyeva A
- Anastasi GA
- Anchordoqui L
- Andeen K
- Anderson JP
- Anderson SB
- Anderson T
- Anderson WG
- Andrada B
- Andre M
- Andreoni I
- Andreoni I
- Andringa S
- Angelova SV
- Anghinolfi M
- Angner EO
- Angus CR
- Annis J
- Ansseau I
- Antier S
- Anton G
- Antonelli LA
- Antonelli LA
- Anupama GC
- Aoki K
- Aoki W
- Appert S
- Aptekar RL
- Arai K
- Arakawa M
- Aramo C
- Araya MC
- Arcavi I
- Arceo R
- Ardid M
- Areeda JS
- Aresu G
- Argan A
- Argelles C
- Arnaud N
- Array LWA Long Wavelength
- Array MWA Murchison Widefield
- Arrieta M
- Arsene N
- Arteaga-Velzquez JC
- Artola R
- Arun KG
- Asakura Y
- Asaoka Y
- Ascenzi S
- Ashall C
- Ashley MCB
- Ashton G
- Asorey H
- Assis P
- Ast M
- Aston SM
- Astone P
- Atallah DV
- ATLAS
- Atwood WB
- Aubert J-J
- Aubert P
- Aublin J
- Auffenberg J
- Aufmuth P
- Aulbert C
- AultONeal K
- Austin C
- Avgitas T
- Avila G
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- Awad A Kuotb
- Axani S
- Baar S
- Babak S
- Bachetti M
- Backes M
- Bacon P
- Bader MKM
- Badescu AM
- Bae S
- Bagherpour H
- Bai X
- Bailes M
- Baker PT
- Balaceanu A
- Balanutsa PV
- Balasubramanian A
- Balbinot E
- Baldaccini F
- Baldini L
- Ballardin G
- Ballmer SW
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- Bannister KW
- Barayoga JC
- Barbarino C
- Barbato F
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- Barsuglia M
- Barta D
- Barthelmy SD
- Bartlett J
- Bartos I
- Barway S
- Barway S
- Barwick SW
- Basa S
- Bassiri R
- Basti A
- Bastieri D
- Batch JC
- Bauer FE
- Bauer FE
- Baum V
- Bawaj M
- Bay R
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- Bazzan M
- Bazzano A
- Bchele M
- Beardmore AP
- Beardsley A
- Beatty JJ
- Becerril A
- Becherini Y
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- Bernardini MG
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- Bertin E
- Bertin V
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- Borner M
- Borodai N
- Bos F
- Boschi V
- Bose D
- Bose S
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- Botner O
- Bottacini E
- Bottcher M
- Botti AM
- Botticella MT
- Bouffanais Y
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- Bourbeau J
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- Bozzi A
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- Cano Z
- Cao H
- Cao J
- Cao XL
- Capaccioli M
- Capano CD
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- Caramete L
- Caraveo P
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- Carbognani F
- Carboni G
- Cardenas B Vargas
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- Chatterjee D
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- Coughlin M
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- Coutu S
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- Coward DM
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- Cowart MJ
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- Creusot A
- Cripe J
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- Drouhin D
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- El Bojaddaini I
- El Khayati N
- El Moursli R Cherkaoui
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- Elipe G Torralba
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- Fenu F
- Fermi GBM
- Fernandes MV
- Fernandez D
- Fernandez E
- Fernandez G Rodriguez
- Fernandez-Galiana A
- Feroci M
- Ferrante I
- Ferrari A
- Ferreira EC
- Ferrigno C
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- Fiori I
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- Frail DA
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- Fulda P
- Funk S
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- Furusawa H
- Fuschino F
- Fusco LA
- Fusco P
- Fuster A
- Fyffe M
- Fynbo JPU
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- Gadre BU
- Gaebel SM
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- Gaior R
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- Gal-Yam A
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- Ghia PL
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- Ghosh Abhirup
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- Goldstein A
- Goldstein DA
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- Golup G
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- Publication venue
- 'American Astronomical Society'
- Publication date
- 01/01/2017
- Field of study
On 2017 August 17 a binary neutron star coalescence candidate (later designated GW170817) with merger time 12:41:04 UTC was observed through gravitational waves by the Advanced LIGO and Advanced Virgo detectors. The Fermi Gamma-ray Burst Monitor independently detected a gamma-ray burst (GRB 170817A) with a time delay of ~1.7 s with respect to the merger time. From the gravitational-wave signal, the source was initially localized to a sky region of 31 deg2 at a luminosity distance of 40+8-8 Mpc and with component masses consistent with neutron stars. The component masses were later measured to be in the range 0.86 to 2.26 Mo. An extensive observing campaign was launched across the electromagnetic spectrum leading to the discovery of a bright optical transient (SSS17a, now with the IAU identification of AT 2017gfo) in NGC 4993 (at ~40 Mpc) less than 11 hours after the merger by the One- Meter, Two Hemisphere (1M2H) team using the 1 m Swope Telescope. The optical transient was independently detected by multiple teams within an hour. Subsequent observations targeted the object and its environment. Early ultraviolet observations revealed a blue transient that faded within 48 hours. Optical and infrared observations showed a redward evolution over ~10 days. Following early non-detections, X-ray and radio emission were discovered at the transient’s position ~9 and ~16 days, respectively, after the merger. Both the X-ray and radio emission likely arise from a physical process that is distinct from the one that generates the UV/optical/near-infrared emission. No ultra-high-energy gamma-rays and no neutrino candidates consistent with the source were found in follow-up searches. These observations support the hypothesis that GW170817 was produced by the merger of two neutron stars in NGC4993 followed by a short gamma-ray burst (GRB 170817A) and a kilonova/macronova powered by the radioactive decay of r-process nuclei synthesized in the ejecta
Pan-cancer analysis of whole genomes
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- Y. yc
- ya Al-Ahmadie
- Yachida
- Yamaguchi
- Yamaguchi T. N. k
- Yamamoto
- Yamaue
- Yang
- Yang
- Yang
- Yang
- Yang
- Yao
- Yaspo
- Yates
- Yau
- yb Ally
- yd Cherniack
- yd Lai
- yd Schadendorf
- ye Auman
- ye Umbricht
- Yellapantula
- Yellapantula
- yh Bathe
- yj Baylin
- Yoon
- Yu
- Yu
- Yu
- Yuan
- Yugawa
- z ah
- Z. ej
- Z. ep
- Z. fr
- Z. hv
- Z. hy
- Z. jq
- Z. kt
- Z. oe
- Zaikova
- Zapatka
- zd Gershenwald
- Zenklusen
- Zeps
- zf Ghossein
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang H. b
- Zhao
- Zheng
- Zhou
- Zhou
- Zhu
- Zhu
- zi zj
- zm Jayaseelan
- Zou
- Zou
- zt Kim
- zw Lewis
- Ó. A. th
- Ø. sk
- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
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- Abdel-Aziz AK
- Abdelfatah S
- Abdellatif M
- Abdoli A
- Abel S
- Abeliovich H
- Abildgaard MH
- Abudu YP
- Acevedo-Arozena A
- Adamopoulos IE
- Adeli K
- Adolph TE
- Adornetto A
- Aflaki E
- Agam G
- Agarwal A
- Aggarwal BB
- Agnello M
- Agostinis P
- Agrewala JN
- Agrotis A
- Aguilar PV
- Ahmad ST
- Ahmed ZM
- Ahumada-Castro U
- Aits S
- Aizawa S
- Akkoc Y
- Akoumianaki T
- Akpinar HA
- Al-Abd AM
- Al-Akra L
- Al-Gharaibeh A
- Alaoui-Jamali MA
- Alberti S
- Alcocer-Gómez E
- Alessandri C
- Ali M
- Alim Al-Bari MA
- Aliwaini S
- Alizadeh J
- Almacellas E
- Almasan A
- Alonso A
- Alonso GD
- Altan-Bonnet N
- Altieri DC
- Alves da Costa C
- Alves S
- Alzaharna MM
- Amadio M
- Amantini C
- Amaral C
- Ambrosio S
- Amer AO
- Ammanathan V
- An Z
- Andersen SU
- Andrabi SA
- Andrade-Silva M
- Andres AM
- Angelini S
- Ann D
- Anozie UC
- Ansari MY
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- Arikkath J
- Arimoto H
- Ariosa AR
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- Artero R
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- Zientara-Rytter K
- Zimmermann CM
- Ziviani E
- Zoladek T
- Zong W-X
- Zorov DB
- Zorzano A
- Zou W
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- Zou Z
- Zuryn S
- Zwerschke W
- Álvarez ÉMC
- Ávalos Y
- Önal G
- Üstün S
- Čolić M
- Đokić J
- Žerovnik E
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field
Iron Behaving Badly: Inappropriate Iron Chelation as a Major Contributor to the Aetiology of Vascular and Other Progressive Inflammatory and Degenerative Diseases
- Author
- A Abbott
- A Abou-Raya
- A Aljandali
- A Ambesi-Impiombato
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- A Brzezinski
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- Publication venue
- Publication date
- 09/08/2008
- Field of study
The production of peroxide and superoxide is an inevitable consequence of
aerobic metabolism, and while these particular "reactive oxygen species" (ROSs)
can exhibit a number of biological effects, they are not of themselves
excessively reactive and thus they are not especially damaging at physiological
concentrations. However, their reactions with poorly liganded iron species can
lead to the catalytic production of the very reactive and dangerous hydroxyl
radical, which is exceptionally damaging, and a major cause of chronic
inflammation. We review the considerable and wide-ranging evidence for the
involvement of this combination of (su)peroxide and poorly liganded iron in a
large number of physiological and indeed pathological processes and
inflammatory disorders, especially those involving the progressive degradation
of cellular and organismal performance. These diseases share a great many
similarities and thus might be considered to have a common cause (i.e.
iron-catalysed free radical and especially hydroxyl radical generation). The
studies reviewed include those focused on a series of cardiovascular, metabolic
and neurological diseases, where iron can be found at the sites of plaques and
lesions, as well as studies showing the significance of iron to aging and
longevity. The effective chelation of iron by natural or synthetic ligands is
thus of major physiological (and potentially therapeutic) importance. As
systems properties, we need to recognise that physiological observables have
multiple molecular causes, and studying them in isolation leads to inconsistent
patterns of apparent causality when it is the simultaneous combination of
multiple factors that is responsible. This explains, for instance, the
decidedly mixed effects of antioxidants that have been observed, etc...Comment: 159 pages, including 9 Figs and 2184 reference
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