176 research outputs found
Antibody responses in furunculosis patients vaccinated with autologous formalin-killed Staphylococcus aureus
- Author
- A. van Belkum
- AC Schaffer
- AE Wright
- B Okrasinska-Cholewa
- B. M. Bröker
- C Goerke
- C. de Vogel
- D Harmsen
- E Zaluga
- G Lina
- H. Masiuk
- HF Wertheim
- HP Cu
- HW Boucher
- J Halasa
- J Lewin
- J Ring
- J. Jursa-Kulesza
- J. Kolata
- JA Lindsay
- JD Fraser
- L. Steil
- M Aires-de-Sousa
- M Dryden
- M. Nowosiad
- N Verkaik
- N. J. Verkaik
- NJ Verkaik
- O Nolte
- O Nolte
- RL Goodale
- RM McLoughlin
- RM McLoughlin
- RP Novick
- S Bavari
- S Holtfreter
- S Holtfreter
- S Holtfreter
- S Holtfreter
- S. Giedrys-Kalemba
- S. Holtfreter
- U. Völker
- V Rusch
- W. van Wamel
- Z Novotny
- Publication venue
- Springer-Verlag
- Publication date
- 01/01/2011
- Field of study
Autologous vaccines (short: autovaccines) have been used since the beginning of the 20th century to treat chronic staphylococcal infections, but their mechanisms of action are still obscure. This prospective pilot study involved four patients with furunculosis who were vaccinated with autologous formalin-killed Staphylococcus aureus cells. Vaccines were individually prepared from the infecting S. aureus strain and repeatedly injected subcutaneously in increasing doses over several months. We characterized the virulence gene repertoire and spa genotype of the infecting and colonising S. aureus strains. Serum antibody responses to secreted and surface-bound bacterial antigens were determined by two-dimensional immunoblotting and flow-cytometry based assays (Luminex®). All patients reported clinical improvement. Molecular characterization showed that all strains isolated from one patient over time belonged to the same S. aureus clone. Already before treatment, there was robust antibody binding to a broad range of staphylococcal antigens. Autovaccination moderately boosted the IgG response to extracellular antigens in two patients, while the antibody response of the other two patients was not affected. Similarly, vaccination moderately enhanced the antibody response against some staphylococcal surface proteins, e.g. ClfA, ClfB, SdrD and SdrE. In summary, autovaccination only slightly boosted the pre-existing serum antibody response, predominantly to bacterial surface antigens
Staging Bipolar Disorder.
- Author
- A Duffy
- A Martínez-Arán
- A. R. Rosa
- AB Cunha
- AB Cunha
- AC Andreazza
- AC Andreazza
- AC Swann
- AR Rosa
- AR Rosa
- BN Frey
- CJ Murray
- CU Correll
- E Brietzke
- E Brietzke
- E Vieta
- Eduard Vieta
- F Colom
- F Kapczinski
- F Kapczinski
- F Kapczinski
- F Kapczinski
- G Salvadore
- G Salvadore
- GA Fava
- HP Blumberg
- IK Lyoo
- J Scott
- JC Soares
- JC Walz
- JC Walz
- L Franchini
- LJ Robinson
- LN Yatham
- LV Kessing
- LV Kessing
- M Berk
- M Berk
- M Berk
- M Kauer-Sant’Anna
- M Reinares
- M. Reinares
- N Crumlish
- NM Simon
- P Conus
- PB Moore
- PD McGorry
- PD McGorry
- PD McGorry
- RM Post
- RM Post
- SL McElroy
- SM Strakowski
- T Silverstone
- TA Ketter
- TW Moorhead
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 04/04/2014
- Field of study
The purpose of this study was to analyze the evidence supporting a staging model for bipolar disorder. The authors conducted an extensive Medline and Pubmed search of the published literature using a variety of search terms (staging, bipolar disorder, early intervention) to find relevant articles, which were reviewed in detail. Only recently specific proposals have been made to apply clinical staging to bipolar disorder. The staging model in bipolar disorder suggests a progression from prodromal (at-risk) to more severe and refractory presentations (Stage IV). A staging model implies a longitudinal appraisal of different aspects: clinical variables, such as number of episodes and subsyndromal symptoms, functional and cognitive impairment, comorbidity, biomarkers, and neuroanatomical changes. Staging models are based on the fact that response to treatment is generally better when it is introduced early in the course of the illness. It assumes that earlier stages have better prognosis and require simpler therapeutic regimens. Staging may assist in bipolar disorder treatment planning and prognosis, and emphasize the importance of early intervention. Further research is required in this exciting and novel area
Jet energy measurement with the ATLAS detector in proton-proton collisions at root s=7 TeV
- Author
- Aad G
- Abbott B
- Abdallah J
- Abdelalim AA
- Abdesselam A
- Abdinov O
- Abi B
- Abolins M
- Abramowicz H
- Abreu H
- Acerbi E
- Acharya BS
- Adams DL
- Addy TN
- Adelman J
- Aderholz M
- Adomeit S
- Adragna P
- Adye T
- Aefsky S
- Aguilar-Saavedra JA
- Aharrouche M
- Ahlen SP
- Ahles F
- Ahmad A
- Ahsan M
- Aielli G
- Akdogana T
- Akesson TPA
- Akimoto G
- Akimov AV
- Akiyama A
- Aktas A
- Alam MA
- Alam MS
- Albert J
- Albrand S
- Aleksa M
- Aleksandrov IN
- Alessandria F
- Alexa C
- Alexander G
- Alexandre G
- Alexopoulos T
- Alhroob M
- Aliev M
- Alimonti G
- Alison J
- Aliyev M
- Allport PP
- Allwood-Spiers SE
- Almenar CC
- Almond J
- Aloisio A
- Alon R
- Alonso A
- Alviggi MG
- Amako K
- Amaral P
- Amelung C
- Ammosov VV
- Amorim A
- Amoros G
- Amram N
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders G
- Anderson KJ
- Andreazza A
- Andrei V
- Andrieux M-L
- Anduaga XS
- Angerami A
- Anghinolfi F
- Anh TV
- Anjos N
- Annovi A
- Antonaki A
- Antonelli M
- Antonov A
- Antos J
- Anulli F
- Aoun S
- Apolle R
- Arabidze G
- Aracena I
- Arai Y
- Aranda CP
- Arce ATH
- Archambault JP
- Arfaoui S
- Arguin J-F
- Arik E
- Arik M
- Armbruster AJ
- Arnaez O
- Arnault C
- Artamonov A
- Artoni G
- Arutinov D
- Asai S
- Asfandiyarov R
- Ask S
- Asman B
- Asner D
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- Aubert B
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- Augsten K
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- Avolio G
- Avramidou R
- Axen D
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- Azuma Y
- Baak MA
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- Backes M
- Backhaus M
- Badescu E
- Bagnaia P
- Bahinipati S
- Bai Y
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- Baker MD
- Baker OK
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- Belenguer MJ
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- Benchouk C
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- Benjamin DP
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- Bensinger JR
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- Boonekamp M
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- Borjanovic I
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- Boscherini D
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- Yilmaz M
- Yoosootiniya R
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- Zarzhitsky P
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- Zhemchugov A
- Zheng S
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- Zitoun R
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- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/03/2013
- Field of study
The jet energy scale and its systematic uncertainty are determined for jets measured with the ATLAS detector at the LHC in proton-proton collision data at a centre-of-mass energy of √s = 7TeV corresponding to an integrated luminosity of 38 pb-1. Jets are reconstructed with the anti-kt algorithm with distance parameters R=0. 4 or R=0. 6. Jet energy and angle corrections are determined from Monte Carlo simulations to calibrate jets with transverse momenta pT≥20 GeV and pseudorapidities {pipe}η{pipe}<4. 5. The jet energy systematic uncertainty is estimated using the single isolated hadron response measured in situ and in test-beams, exploiting the transverse momentum balance between central and forward jets in events with dijet topologies and studying systematic variations in Monte Carlo simulations. The jet energy uncertainty is less than 2. 5 % in the central calorimeter region ({pipe}η{pipe}<0. 8) for jets with 60≤pT<800 GeV, and is maximally 14 % for pT<30 GeV in the most forward region 3. 2≤{pipe}η{pipe}<4. 5. The jet energy is validated for jet transverse momenta up to 1 TeV to the level of a few percent using several in situ techniques by comparing a well-known reference such as the recoiling photon pT, the sum of the transverse momenta of tracks associated to the jet, or a system of low-pT jets recoiling against a high-pT jet. More sophisticated jet calibration schemes are presented based on calorimeter cell energy density weighting or hadronic properties of jets, aiming for an improved jet energy resolution and a reduced flavour dependence of the jet response. The systematic uncertainty of the jet energy determined from a combination of in situ techniques is consistent with the one derived from single hadron response measurements over a wide kinematic range. The nominal corrections and uncertainties are derived for isolated jets in an inclusive sample of high-pT jets. Special cases such as event topologies with close-by jets, or selections of samples with an enhanced content of jets originating from light quarks, heavy quarks or gluons are also discussed and the corresponding uncertainties are determined. © 2013 CERN for the benefit of the ATLAS collaboration
Measurement of the inclusive and dijet cross-sections of b-jets in pp collisions at sqrt(s) = 7 TeV with the ATLAS detector
- Author
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- Abbott B
- Abdallah J
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- Abdesselam A
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- Tua A
- Tuggle JM
- Turala M
- Turecek D
- Turlay E
- Tuts PM
- Tykhonov A
- Tylmad M
- Tyndel M
- Typaldos D
- Tyrvainen H
- Tzanakos G
- Uchida K
- Ueda I
- Ueno R
- Ugland M
- Uhlenbrock M
- Uhrmacher M
- Ukegawa F
- Unal G
- Underwood DG
- Undrus A
- Unel G
- Unno Y
- Urbaniec D
- Urkovsky E
- Urquijo P
- Urrejola P
- Usai G
- Uslenghi M
- Vacavant L
- Vacek V
- Vachon B
- Vahsen S
- Valderanis C
- Valenta J
- Valente P
- Valentinetti S
- Valkar S
- Valladolid Gallego E
- Vallecorsa S
- Valls Ferrer JA
- van der Graaf H
- van der Kraaij E
- van der Poel E
- van der Ster D
- Van Eijk B
- van Eldik N
- van Gemmeren P
- van Kesteren Z
- van Vulpen I
- Vandelli W
- Vandoni G
- Vaniachine A
- Vankov P
- Vannucci F
- Vari R
- Varnes EW
- Varouchas D
- Vartapetian A
- Varvell KE
- Vassilakopoulos VI
- Vazeille F
- Vegni G
- Veillet JJ
- Vellidis C
- Veloso F
- Veness R
- Veneziano S
- Ventura A
- Ventura D
- Ventura S
- Venturi M
- Venturi N
- Vercesi V
- Verducci M
- Verkerke W
- Vermeulen JC
- Vertogardov L
- Vest A
- Vetterli MC
- Vichou I
- Vickey T
- Viegas FJTA
- Viehhauser GHA
- Viel S
- Villa M
- Villaplana Perez M
- Vilucchi E
- Vincter MG
- Vinek E
- Vinogradov VB
- Virchaux M
- Viret S
- Virzi J
- Vitale A
- Vitells O
- Vivarelli I
- Vives Vaque F
- Vlachos S
- Vlasak M
- Vlasov N
- Vogel A
- Vokac P
- Volpi M
- Volpini G
- von der Schmitt H
- von Loeben J
- von Radziewski H
- von Toerne E
- Vorobel V
- Vorobiev AP
- Vorwerk V
- Vos M
- Voss R
- Voss TT
- Vossebeld JH
- Vovenko AS
- Vranjes N
- Vrba V
- Vreeswijk M
- Vuillermet R
- Vukotic I
- Wagner P
- Wagner W
- Wahlen H
- Wakabayashi J
- Walbersloh J
- Walch S
- Walder J
- Walker R
- Walkowiak W
- Wall R
- Waller P
- Wang C
- Wang H
- Wang J
- Wang JC
- Wang SM
- Warburton A
- Ward CP
- Warsinsky M
- Watkins PM
- Watson AT
- Watson MF
- Watts G
- Watts S
- Waugh AT
- Waugh BM
- Weber J
- Weber M
- Weber MS
- Weber P
- Weidberg AR
- Weingarten J
- Weiser C
- Wellenstein H
- Wells PS
- Wen M
- Wenaus T
- Wendler S
- Weng Z
- Wengler T
- Wenig S
- Wermes N
- Werner M
- Werner P
- Werth M
- Wessels M
- Whalen K
- Wheeler-Ellis SJ
- Whitaker SP
- White A
- White MJ
- White S
- Whitehead SR
- Whiteson D
- Whittington D
- Wicek F
- Wicke D
- Wickens FJ
- Wiedenmann W
- Wielers M
- Wienemann P
- Wiglesworth C
- Wiik LAM
- Wildauer A
- Wildt MA
- Wilhelm I
- Wilkens HG
- Will JZ
- Williams E
- Williams HH
- Willis W
- Willocq S
- Wilson A
- Wilson JA
- Wilson MG
- Wingerter-Seez I
- Winkelmann S
- Winklmeier F
- Wittgen M
- Wolter MW
- Wolters H
- Wooden G
- Wosiek BK
- Wotschack J
- Woudstra MJ
- Wraight K
- Wright C
- Wrona B
- Wu SL
- Wu X
- Wu Y
- Wulf E
- Wunstorf R
- Wynne BM
- Xaplanteris L
- Xella S
- Xie S
- Xie Y
- Xu C
- Xu D
- Xu G
- Yabsley B
- Yagci KD
- Yamada M
- Yamamoto A
- Yamamoto K
- Yamamoto S
- Yamamura T
- Yamaoka J
- Yamazaki T
- Yamazaki Y
- Yan Z
- Yang H
- Yang S
- Yang UK
- Yang Y
- Yang Y
- Yang Z
- Yanush S
- Yao W-M
- Yao Y
- Yasu Y
- Ye J
- Ye S
- Yildiz HD
- Yilmaz M
- Yoosoofmiya R
- Yorita K
- Yoshida R
- Young C
- Youssef S
- Yu D
- Yu J
- Yu J
- Yuan L
- Yurkewicz A
- Zaets VG
- Zaidan R
- Zaitsev AM
- Zajacova Z
- Zalite YK
- Zanello L
- Zarzhitsky P
- Zaytsev A
- Zdrazil M
- Zeitnitz C
- Zeller M
- Zema PF
- Zemla A
- Zendler C
- Zenin AV
- Zenin O
- Zenis T
- Zenonos Z
- Zenz S
- Zerwas D
- Zhan Z
- Zhang D
- Zhang H
- Zhang J
- Zhang X
- Zhang Z
- Zhao L
- Zhao T
- Zhao Z
- Zhemchugov A
- Zheng S
- Zhong J
- Zhou B
- Zhou N
- Zhou Y
- Zhu CG
- Zhu H
- Zhu Y
- Zhuang X
- Zhuravlov V
- Zieminska D
- Zilka B
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Ziolkowski M
- Zitoun R
- Zivkovic L
- Zmouchko VV
- Zobernig G
- Zoccoli A
- Zolnierowski Y
- Zsenei A
- zur Nedden M
- Zutshi V
- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 31/12/2010
- Field of study
The inclusive and dijet production cross-sections have been measured for jets
containing b-hadrons (b-jets) in proton-proton collisions at a centre-of-mass
energy of sqrt(s) = 7 TeV, using the ATLAS detector at the LHC. The
measurements use data corresponding to an integrated luminosity of 34 pb^-1.
The b-jets are identified using either a lifetime-based method, where secondary
decay vertices of b-hadrons in jets are reconstructed using information from
the tracking detectors, or a muon-based method where the presence of a muon is
used to identify semileptonic decays of b-hadrons inside jets. The inclusive
b-jet cross-section is measured as a function of transverse momentum in the
range 20 < pT < 400 GeV and rapidity in the range |y| < 2.1. The bbbar-dijet
cross-section is measured as a function of the dijet invariant mass in the
range 110 < m_jj < 760 GeV, the azimuthal angle difference between the two jets
and the angular variable chi in two dijet mass regions. The results are
compared with next-to-leading-order QCD predictions. Good agreement is observed
between the measured cross-sections and the predictions obtained using POWHEG +
Pythia. MC@NLO + Herwig shows good agreement with the measured bbbar-dijet
cross-section. However, it does not reproduce the measured inclusive
cross-section well, particularly for central b-jets with large transverse
momenta.Comment: 10 pages plus author list (21 pages total), 8 figures, 1 table, final
version published in European Physical Journal
Biomarkers in T cell therapy clinical trials
- Author
- A Hoos
- A Varela-Rohena
- AM Leen
- AP Rapoport
- AR Hersperger
- AR Hersperger
- B Jena
- BG Till
- BL Levine
- C Berger
- C Berger
- C Yee
- CF Taylor
- CH June
- CH Lamers
- CJ Turtle
- CM Bollard
- CU Louis
- D Gaucher
- D Hollyman
- D Kalamasz
- DB Kohn
- DC Palmer
- DF Stroncek
- DF Stroncek
- DJ Powell Jr
- DJ Stewart
- DL Porter
- DL Porter
- DR Bandura
- EA Walter
- G Makedonas
- G Makedonas
- H Tahara
- HI Scher
- HP Bonarius
- HS Robins
- HT Maecker
- J van Lunzen
- J Zhou
- JC Siebert
- JD Freeman
- JE Cortes
- JJ Melenhorst
- JL Davis
- JW Gratama
- KQ Tran
- KR Schulz
- L Gattinoni
- L Rowen
- LA Johnson
- LH Butterfield
- LH Butterfield
- LH Finke
- M Kalos
- M Raki
- MA Pule
- MA Suni
- MC Jensen
- ME Dudley
- Michael Kalos
- MJ Besser
- MR Betts
- MV Maus
- OI Ornatsky
- PD Greenberg
- PF Robbins
- PF Robbins
- PJ Amrolia
- PK Chattopadhyay
- R Brentjens
- RA Morgan
- RA Morgan
- RA Morgan
- RA Seder
- RJ O'Reilly
- S Ambs
- S Biswas
- S Gnjatic
- S Janetzki
- S Yang
- SC Chow
- SH Beachy
- SP Perfetto
- T Feuchtinger
- TF Gajewski
- U Petrausch
- W Liu
- WT Rogers
- X Wang
- Y Lin
- Y Zhao
- Y Zhao
- Publication venue
- BioMed Central
- Publication date
- 01/01/2011
- Field of study
T cell therapy represents an emerging and promising modality for the treatment of both infectious disease and cancer. Data from recent clinical trials have highlighted the potential for this therapeutic modality to effect potent anti-tumor activity. Biomarkers, operationally defined as biological parameters measured from patients that provide information about treatment impact, play a central role in the development of novel therapeutic agents. In the absence of information about primary clinical endpoints, biomarkers can provide critical insights that allow investigators to guide the clinical development of the candidate product. In the context of cell therapy trials, the definition of biomarkers can be extended to include a description of parameters of the cell product that are important for product bioactivity
The equilibria that allow bacterial persistence in human hosts
- Author
- A Lundgren
- AE Hirsh
- AJ Barnard
- B Gebert
- B Linz
- BD Smith
- BM Murphy
- BR Levin
- C Ghose
- C Kidgell
- CA Mims
- CC McGowan
- CM Krinos
- CR Sinnott
- CU Nwokolo
- D Falush
- D Gammack
- D Kirschner
- D Sud
- D Wodarz
- D Wodarz
- D Young
- DE Kirschner
- Denise Kirschner
- DH Lewis
- DM Monack
- DP O’Brien
- EJ Kuipers
- EJ Munoz-Elias
- EM Bik
- F Espersen
- F Fenner
- F Fiegna
- GF Webb
- HP Wirth
- J Nash
- J Parkhill
- J Parsonnet
- J Raymond
- J Segovia-Juarez
- J Smith
- JD Fontenot
- JD Robbins
- JE Clark-Curtiss
- JE Wigginton
- JF Tomb
- JG Fox
- JM Kang
- JM Smith
- JM Smith
- JV Neumann
- K Dietz
- K Putsep
- K Yokoyama
- L Margulis
- M Bledzka-Sarek
- M Glickman
- M Nowak
- M Nowak
- Martin J. Blaser
- MG Dominguez-Bello
- MJ Blaser
- MJ Blaser
- MJ Blaser
- MJ Blaser
- NJ Saunders
- P Bellwood
- P Roumagnac
- PG Falk
- PL Lin
- R Law
- RA Aras
- RA Aras
- RA Aras
- RB Hornick
- RD Fleischmann
- RE Hope-Simpson
- RM Anderson
- RM Anderson
- RM Peek
- Ruslan Medzhitov
- S Bonhoeffer
- S Gagneux
- S Ganguli
- S Marino
- S Marino
- S Odenbreit
- S Sakaguchi
- S-Y Kim
- SM Blower
- SP Faucher
- ST Cole
- SV Capuano
- T Rosebury
- V Lazarevic
- WR Schwan
- Y Belkaid
- Y Chen
- Y Zheng
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 18/10/2007
- Field of study
We propose that microbes that have developed persistent relationships with human hosts have evolved cross-signalling mechanisms that permit homeostasis that conforms to Nash equilibria and, more specifically, to evolutionarily stable strategies. This implies that a group of highly diverse organisms has evolved within the changing contexts of variation in effective human population size and lifespan, shaping the equilibria achieved, and creating relationships resembling climax communities. We propose that such ecosystems contain nested communities in which equilibrium at one level contributes to homeostasis at another. The model can aid prediction of equilibrium states in the context of further change: widespread immunodeficiency, changing population densities, or extinctions.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/62883/1/nature06198.pd
Lung epithelium as a sentinel and effector system in pneumonia – molecular mechanisms of pathogen recognition and signal transduction
- Author
- A Apisarnthanarak
- A Combes
- A Dunne
- A Kadioglu
- A Muir
- A Poltorak
- A Schoenemeyer
- A Takaoka
- A Uehara
- AB Molofsky
- AI Chin
- AJ Ratner
- AK Perry
- AM Ferrara
- AM Hajjar
- AM LeVine
- AN Honko
- AN Theofilopoulos
- AO Aliprantis
- AR Koczulla
- B Barnes
- B Beutler
- B Opitz
- B Opitz
- B Opitz
- B Opitz
- B Schmeck
- B Schmeck
- B Schmeck
- BB Moore
- BD Rudd
- BD Rudd
- BD Strahl
- BD Uhal
- BG Williams
- BN Lambrecht
- C Delclaux
- C McDonald
- CA Hewson
- CA Sinaniotis
- CAJ Janeway
- CFJ Thomas
- CH Mody
- CJ Hertz
- CM Greene
- CM Greene
- CP Costa
- CP Dillon
- CU Da Costa
- D Droemann
- D Prawitt
- DA Groneberg
- DJ Groskreutz
- DJ Philpott
- DR Cundell
- DS Zamboni
- DW Shimabukuro
- E Bouza
- E Cario
- E Crouch
- E Diez
- E Meylan
- EK Wright
- F Hayashi
- F Heil
- F Martinon
- F Martinon
- FG Hayden
- FS Sutterwala
- FY Liew
- G Bonizzi
- G Ferwerda
- G Segal
- G Soong
- G Tal
- G Zhang
- H Chen
- H Fehrenbach
- H Hemmi
- H Kato
- H Lemjabbar
- H Sano
- H Slevogt
- H Tada
- HP Hauber
- HP Jia
- HU Jahn
- HW Chu
- IC Michelow
- J Aarbiou
- J Andrejeva
- J Chastre
- J Harder
- J Pizarro-Cerda
- J Viala
- J Wang
- JEJ Crowe
- JM Stark
- JP Janssens
- JP Ting
- JPIII Lynch
- JT Wang
- JV Williams
- JW Hollingsworth
- JY Fagon
- K Burns
- K De Smet
- K Hoebe
- K Honda
- K Honda
- K Ito
- K Kobayashi
- K Kuba
- K Muegge
- K Takeda
- KA Fitzgerald
- KA Fitzgerald
- KA Fitzgerald
- KC Meyer
- KF Chung
- KL Williams
- KS Kobayashi
- KS Li
- L Alexopoulou
- L Armstrong
- L Edwards
- L Guillot
- L Guillot
- L Mueller-Anneling
- LA Mandell
- LG Xu
- LH Travassos
- LJ Mota
- LP Nicod
- M Chamaillard
- M Chamaillard
- M Hu
- M Hu
- M Krull
- M Nakamura
- M Pichavant
- M Ritter
- M Ulanova
- M Yamamoto
- M Yamamoto
- M Yoneyama
- M Yoshida
- MA Matthay
- ME Poynter
- MG Ison
- MG Netea
- MH Kollef
- MI Gomez
- MJ Alcorn
- MJ Griffiths
- MJ Shaw
- ML Yu
- MM Monick
- MN Tsolia
- MT Abreu
- MT Klockmann
- MW Pound
- N Barnich
- N Chaudhuri
- N Inohara
- N Inohara
- N Kambe
- N Noulin
- NC Arbour
- NP Cianciotto
- NW Schroder
- O Avni
- O Takeuchi
- P Cooper
- P Cossart
- P Hysi
- P Rosenstiel
- PD N'Guessan
- PJ Barnes
- R Adamo
- R Bals
- R Chaby
- R Claus
- R Malley
- R Medzhitov
- RA Fouchier
- RA Fouchier
- RA Fowler
- RB Seth
- RE Dixon
- RIII Paine
- RM Strieter
- RM Strieter
- RT Sadikot
- RT Sadikot
- RT Sadikot
- S Akira
- S Basu
- S Epelman
- S Hippenstiel
- S Maeda
- S Mariathasan
- S Matikainen
- S Saccani
- S Sharma
- S Spaan
- S Traub
- S Uematsu
- S van Wetering
- S van Wetering
- S Weidinger
- SB Greenberg
- SB Neff
- SE Girardin
- SE Girardin
- SE Girardin
- SJ Skerrett
- SL Johnston
- SM Travis
- SN Sarkar
- SR Lane
- SR Vavricka
- SS Diebold
- T Decker
- T Hisamatsu
- T Homma
- T Horng
- T Jenuwein
- T Joseph
- T Kawai
- T Kawai
- T Kawai
- T Kawai
- T Ren
- T Saito
- T Watanabe
- TD Kanneganti
- TJ Marrie
- TM File
- TR Hawn
- U Buwitt-Beckmann
- V Bitko
- V Gant
- WC Tan
- WR Bishai
- X Wang
- Y Gon
- Y Imai
- Y Tesfaigzi
- Y Tsutsumi-Ishii
- YS Lopez-Boado
- Z Zhang
- Publication venue
- BioMed Central
- Publication date
- 01/01/2006
- Field of study
Pneumonia, a common disease caused by a great diversity of infectious agents is responsible for enormous morbidity and mortality worldwide. The bronchial and lung epithelium comprises a large surface between host and environment and is attacked as a primary target during lung infection. Besides acting as a mechanical barrier, recent evidence suggests that the lung epithelium functions as an important sentinel system against pathogens. Equipped with transmembranous and cytosolic pathogen-sensing pattern recognition receptors the epithelium detects invading pathogens. A complex signalling results in epithelial cell activation, which essentially participates in initiation and orchestration of the subsequent innate and adaptive immune response. In this review we summarize recent progress in research focussing on molecular mechanisms of pathogen detection, host cell signal transduction, and subsequent activation of lung epithelial cells by pathogens and their virulence factors and point to open questions. The analysis of lung epithelial function in the host response in pneumonia may pave the way to the development of innovative highly needed therapeutics in pneumonia in addition to antibiotics
Measurement of the cross-section for b-jets produced in association with a Z boson at root s=7 TeV with the ATLAS detector ATLAS Collaboration
- Author
- Aad G
- Abbott B
- Abdallah J
- Abdelalim AA
- Abdesselam A
- Abdinov O
- Abi B
- Abolins M
- Abramowicz H
- Abreu H
- Acerbi E
- Acharya BS
- Adams DL
- Addy TN
- Adelman J
- Aderholz M
- Adomeit S
- Adragna P
- Adye T
- Aefsky S
- Aguilar-Saavedra JA
- Aharrouche M
- Ahlen SP
- Ahles F
- Ahmad A
- Ahsan M
- Aielli G
- Akdogan T
- Akesson TPA
- Akimoto G
- Akimov AV
- Akiyama A
- Alam MA
- Alam MS
- Albert J
- Albrand S
- Aleksa M
- Aleksandrov IN
- Alessandria F
- Alexa C
- Alexander G
- Alexandre G
- Alexopoulos T
- Alhroob M
- Aliev M
- Alimonti G
- Alison J
- Aliyev M
- Allport PP
- Allwood-Spiers SE
- Almenar CC
- Almond J
- Aloisio A
- Alon R
- Alonso A
- Alviggi MG
- Amako K
- Amaral P
- Amelung C
- Ammosov VV
- Amorim A
- Amoros G
- Amram N
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders G
- Anderson KJ
- Andreazza A
- Andrei V
- Andrieux M-L
- Anduaga XS
- Angerami A
- Anghinolfi F
- Anh TV
- Anjos N
- Annovi A
- Antonaki A
- Antonelli M
- Antonov A
- Antos J
- Anulli F
- Aoun S
- Apolle R
- Arabidze G
- Aracena I
- Arai Y
- Arce ATH
- Archambault JP
- Arfaoui S
- Arguin J-F
- Arik E
- Arik M
- Armbruster AJ
- Arnaez O
- Arnault C
- Artamonov A
- Artoni G
- Arutinov D
- Asai S
- Asfandiyarov R
- Ask S
- Asman B
- Asquith L
- Assamagan K
- Astbury A
- Astvatsatourov A
- Atoian G
- Aubert B
- Auge E
- Augsten K
- Aurousseau M
- Austin N
- Avolio G
- Avramidou R
- Axen D
- Ay C
- Azuelos G
- Azuma Y
- Baak MA
- Baccaglioni G
- Bacci C
- Bach AM
- Bachacou H
- Bachas K
- Bachy G
- Backes M
- Backhaus M
- Badescu E
- Bagnaia P
- Bahinipati S
- Bai Y
- Bailey DC
- Bain T
- Baines JT
- Baker MD
- Baker OK
- Baker S
- Banas E
- Banerjee P
- Banerjee S
- Banfi D
- Bangert A
- Bansal V
- Bansil HS
- Barajas CAC
- Barak L
- Baranov SP
- Barashkou A
- Barber T
- Barberio EL
- Barberis D
- Barbero M
- Bardin DY
- Barillari T
- Barisonzi M
- Barklow T
- Barlow N
- Barnett BM
- Barnett RM
- Baroncellia A
- Barone G
- Barr AJ
- Barreiro F
- Barrera CO
- Barrillon P
- Bartoldus R
- Barton AE
- Bartsch D
- Bartsch V
- Bates RL
- Batkovaa L
- Batley JR
- Battaglia A
- Battistin M
- Battistonia G
- Bauer F
- Bawa HS
- Beare B
- Beau T
- Beauchemin PH
- Beccherlea R
- Bechtle R
- Beck HP
- Beckingham M
- Becks KH
- Beddallc A
- Beddallc AJ
- Bedikian S
- Bednyakov VA
- Bee CP
- Begel M
- Behera PK
- Beimforde M
- Belanger-Champagne C
- Belenguer MJ
- Bell PJ
- Bell WH
- Bella G
- Bella LA
- Bellagambaa L
- Bellina F
- Bellomo M
- Belloni A
- Beloborodova O
- Belotskiy K
- Beltramello O
- Ben Ami S
- Benary O
- Benchekrouna D
- Benchouk C
- Bendel M
- Benekos N
- Benhammou Y
- Benjamin DP
- Benoit M
- Bensinger JR
- Benslama K
- Bentvelsen S
- Berge D
- Berger N
- Berghaus F
- Berglund E
- Beringer J
- Bernardet K
- Bernat P
- Bernhard R
- Bernius C
- Berry T
- Bertina A
- Bertinelli F
- Bertoluccia F
- Besanaa MI
- Besson N
- Bethke S
- Bhimji W
- Bianchi RM
- Biancoa M
- Biebel O
- Bieniek SR
- Bierwagen K
- Biesiada J
- Bigliettia M
- Bilokon H
- Bindia M
- Binet S
- Bingulc A
- Binia C
- Biscarat C
- Bitenc U
- Black KM
- Blair RE
- Blanchard J-B
- Blanchot G
- Blazeka T
- Blocker C
- Blocki J
- Blondel A
- Blum W
- Blumenschein U
- Bobbink GJ
- Bobrovnikov VB
- Bocchetta SS
- Bocci A
- Boddy CR
- Boehler M
- Boek J
- Boelaert N
- Boeriu OEV
- Boeser S
- Bogaerts JA
- Bogdanchikov A
- Bogouch A
- Bohma C
- Boisvert V
- Bold T
- Boldeaa V
- Bolnet NM
- Bona M
- Bondarenko VG
- Bondioli M
- Boonekamp M
- Boorman G
- Booth CN
- Bordoni S
- Borer C
- Borisov A
- Borissov G
- Borjanovica I
- Borronia S
- Bos K
- Boscherinia D
- Bosman M
- Boterenbrood H
- Botterill D
- Bouchami J
- Boudreau J
- Bouhova-Thacker EV
- Bourdarios C
- Bousson N
- Boveia A
- Boyd J
- Boyko IR
- Bozhko NI
- Bozovic-Jelisavcicb I
- Bracinik J
- Braem A
- Branchinia P
- Brandenburg GW
- Brandt A
- Brandt G
- Brandt O
- Bratzler U
- Brau B
- Brau JE
- Braun HM
- Brelier B
- Bremer J
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- Zwalinski L
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2011
- Field of study
A measurement is presented of the inclusive cross-section for b-jet production in association with a Z boson in pp collisions at a centre-of-mass energy of root s = 7 TeV. The analysis uses the data sample collected by the ATLAS experiment in 2010, corresponding to an integrated luminosity of approximately 36 pb(-1). The event selection requires a Z boson decaying into high P-T electrons or muons, and at least one b-jet, identified by its displaced vertex, with transverse momentum p(T) > 25 GeV and rapidity vertical bar y vertical bar < 2.1. After subtraction of background processes, the yield is extracted from the vertex mass distribution of the candidate b-jets. The ratio of this cross-section to the inclusive Z cross-section (the average number of b-jets per Z event) is also measured. Both results are found to be in good agreement with perturbative QCD predictions at next-to-leading order
Demographic, clinical and antibody characteristics of patients with digital ulcers in systemic sclerosis: data from the DUO Registry
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- Adoue D
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- Camps MT
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- Castellvi I
- Castellvi I
- Cazalets-lacoste C
- Chakravarty K
- Charlanne H
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- Chiffot H
- Choong LM
- Chromik S
- Cipriani P
- Coelho P
- Cohen JD
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- Couraud A
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- Cuomo G
- D'Cruz D
- Dadban A
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- de Bois M
- De la Puente C
- de la Torre RG
- de Long AJ
- de Ramon E
- de Vicuna RG
- De Vita S
- Deboves O
- Decaux O
- Deininger F
- del Carmen Ortega de la O M
- del Carmen Torres Martin M
- del la Pena Lefebvre PG
- del Mar Ripoll Macias M
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- del Rio AH
- Delgado EG
- den Hengst C
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- Dohse A
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- DUO Registry investigators. Collaborators: Stetter M
- Durant C
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- Enderlein M
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- Skyes H
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- Vacca A
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- van Hagen M
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- van der Veen MJ
- van Gameren I
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- van Paassen H
- van Woerkom JM
- van Zeben D
- Varcasia G
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- Vazquez JL
- Vengemyr K
- Vercoutere W
- Verner D
- Victoria Egurbide M
- Vidal E
- Villaverde V
- Vincent R
- Vingren T
- Visser H
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- Wernitzsch H
- Westedt ML
- Westra R
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- Wilhelm HU
- Willems H
- Willfort-Ehringer A
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- Wlarvens M
- Woijtasik G
- Wollersdorfer E
- Worm M
- Wragg E
- Wuerzburg I
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- Zabel M
- Zemanova I
- Zeni S
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- Ziai M
- Zijlstra TR
- Zimba S
- Zuily S
- Zultak M
- Zuper R
- Zwenger S
- Publication venue
- BMJ Group
- Publication date
- 01/01/2012
- Field of study
OBJECTIVES: The Digital Ulcers Outcome (DUO) Registry was designed to describe the clinical and antibody characteristics, disease course and outcomes of patients with digital ulcers associated with systemic sclerosis (SSc).
METHODS: The DUO Registry is a European, prospective, multicentre, observational, registry of SSc patients with ongoing digital ulcer disease, irrespective of treatment regimen. Data collected included demographics, SSc duration, SSc subset, internal organ manifestations, autoantibodies, previous and ongoing interventions and complications related to digital ulcers.
RESULTS: Up to 19 November 2010 a total of 2439 patients had enrolled into the registry. Most were classified as either limited cutaneous SSc (lcSSc; 52.2%) or diffuse cutaneous SSc (dcSSc; 36.9%). Digital ulcers developed earlier in patients with dcSSc compared with lcSSc. Almost all patients (95.7%) tested positive for antinuclear antibodies, 45.2% for anti-scleroderma-70 and 43.6% for anticentromere antibodies (ACA). The first digital ulcer in the anti-scleroderma-70-positive patient cohort occurred approximately 5 years earlier than the ACA-positive patient group.
CONCLUSIONS: This study provides data from a large cohort of SSc patients with a history of digital ulcers. The early occurrence and high frequency of digital ulcer complications are especially seen in patients with dcSSc and/or anti-scleroderma-70 antibodies
Pan-cancer analysis of whole genomes
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- aab Marks
- aac Marra
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- aaz Schein
- ab ac
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- abd Sipahimalani
- abk Winterhoff
- abn Wong
- abp Baez-Ortega
- abp Kazanov
- abp Kong
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- abpEmail Author
- Aburatani
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- Zaikova
- Zapatka
- zd Gershenwald
- Zenklusen
- Zeps
- zf Ghossein
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang H. b
- Zhao
- Zheng
- Zhou
- Zhou
- Zhu
- Zhu
- zi zj
- zm Jayaseelan
- Zou
- Zou
- zt Kim
- zw Lewis
- Ó. A. th
- Ø. sk
- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
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