204 research outputs found
The disruption of proteostasis in neurodegenerative diseases
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- Publication venue
- 'American Institute of Mathematical Sciences (AIMS)'
- Publication date
- 01/01/2015
- Field of study
Cells count on surveillance systems to monitor and protect the cellular proteome which, besides being highly heterogeneous, is constantly being challenged by intrinsic and environmental factors. In this context, the proteostasis network (PN) is essential to achieve a stable and functional proteome. Disruption of the PN is associated with aging and can lead to and/or potentiate the occurrence of many neurodegenerative diseases (ND). This not only emphasizes the importance of the PN in health span and aging but also how its modulation can be a potential target for intervention and treatment of human diseases.info:eu-repo/semantics/publishedVersio
Approaches in biotechnological applications of natural polymers
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- Publication venue
- 'American Institute of Mathematical Sciences (AIMS)'
- Publication date
- 01/01/2016
- Field of study
Natural polymers, such as gums and mucilage, are biocompatible, cheap, easily available and non-toxic materials of native origin. These polymers are increasingly preferred over synthetic materials for industrial applications due to their intrinsic properties, as well as they are considered alternative sources of raw materials since they present characteristics of sustainability, biodegradability and biosafety. As definition, gums and mucilages are polysaccharides or complex carbohydrates consisting of one or more monosaccharides or their derivatives linked in bewildering variety of linkages and structures. Natural gums are considered polysaccharides naturally occurring in varieties of plant seeds and exudates, tree or shrub exudates, seaweed extracts, fungi, bacteria, and animal sources. Water-soluble gums, also known as hydrocolloids, are considered exudates and are pathological products; therefore, they do not form a part of cell wall. On the other hand, mucilages are part of cell and physiological products. It is important to highlight that gums represent the largest amounts of polymer materials derived from plants. Gums have enormously large and broad applications in both food and non-food industries, being commonly used as thickening, binding, emulsifying, suspending, stabilizing agents and matrices for drug release in pharmaceutical and cosmetic industries. In the food industry, their gelling properties and the ability to mold edible films and coatings are extensively studied. The use of gums depends on the intrinsic properties that they provide, often at costs below those of synthetic polymers. For upgrading the value of gums, they are being processed into various forms, including the most recent nanomaterials, for various biotechnological applications. Thus, the main natural polymers including galactomannans, cellulose, chitin, agar, carrageenan, alginate, cashew gum, pectin and starch, in addition to the current researches about them are reviewed in this article.. }To the Conselho Nacional de Desenvolvimento Cientfíico e Tecnológico (CNPq) for fellowships (LCBBC and MGCC) and the Coordenação de Aperfeiçoamento de Pessoal de Nvíel Superior (CAPES) (PBSA). This study was supported by the Portuguese Foundation for Science and Technology (FCT) under the scope of the strategic funding of UID/BIO/04469/2013 unit, the Project RECI/BBB-EBI/0179/2012 (FCOMP-01-0124-FEDER-027462) and COMPETE 2020 (POCI-01-0145-FEDER-006684) (JAT)
Search for new phenomena in final states with an energetic jet and large missing transverse momentum in pp collisions at √ s = 8 TeV with the ATLAS detector
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- Tskhadadze EG
- Tsopoulou M
- Tsukerman II
- Tsulaia V
- Tsuno S
- Tsybychev D
- Tu Y
- Tudorache A
- Tudorache V
- Tulbure TT
- Tuna AN
- Turchikhin S
- Turgeman D
- Turner RJ
- Turra RT
- Tuts PM
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- Tzovara E
- Ucchielli G
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- Vaidya A
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- Valentinetti S
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- Van Vulpen I
- Vanadia M
- Vandelli W
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- Vandewall ER
- Vaniachine A
- Vannicola D
- Vari R
- Varnes EW
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- Varouchas D
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- Vasile ME
- Vasquez GA
- Vazeille F
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- Vazquez CJB
- Vazquez JGP
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- Virdee GS
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- Von Toerne E
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- Vossebeld JH
- Vozak M
- Vranjes N
- Vrba V
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- Vukotic I
- Wagner P
- Wagner W
- Wagner-Kuhr J
- Wahdan S
- Wahlberg H
- Walbrecht VM
- Walder J
- Walker R
- Walker SD
- Walkowiak W
- Wallangen V
- Wang AM
- Wang AZ
- Wang C
- Wang F
- Wang H
- Wang J
- Wang P
- Wang Q
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- Wang R-J
- Wang SM
- Wang W
- Wang WT
- Wang WX
- Wang Y
- Wang Z
- Wanotayaroj C
- Warburton A
- Ward CP
- Wardrope DR
- Warrack N
- Washbrook A
- Watson AT
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- Waugh BM
- Webb AF
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- Weidberg AR
- Weingarten J
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- Weston TD
- Whalen K
- Whallon NL
- Wharton AM
- White A
- White AS
- White MJ
- Whiteson D
- Whitmore BW
- Wiedenmann W
- Wiel C
- Wielers M
- Wieseotte N
- Wiglesworth C
- Wiik-Fuchs LAM
- Wilkens HG
- Wilkins LJ
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- Williams S
- Willis C
- Willocq S
- Wingerter-Seez I
- Winkels E
- Winklmeier F
- Winston OJ
- Winter BT
- Wittgen M
- Wobisch M
- Wolf A
- Wolf TMH
- Wolff R
- Wolker R
- Wollrath J
- Wolter MW
- Wolters H
- Wong VWS
- Woods NL
- Worm SD
- Wosiek BK
- Wozniak KW
- Wraight K
- Wu SL
- Wu X
- Wu Y
- Wyatt TR
- Wynne BM
- Xella S
- Xi Z
- Xia L
- Xiao X
- Xiotidis I
- Xu D
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- Yabsley B
- Yacoob S
- Yajima K
- Yallup DP
- Yamaguchi N
- Yamaguchi Y
- Yamamoto A
- Yamatani M
- Yamazaki T
- Yamazaki Y
- Yan Z
- Yang HJ
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- Yao W-M
- Yap YC
- Yasu Y
- Yatsenko E
- Ye H
- Ye J
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- Yeletskikh I
- Yexley MR
- Yigitbasi E
- Yildiz HD
- Yorita K
- Yoshihara K
- Young C
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- Yu J
- Yuan R
- Yue X
- Zaazoua M
- Zabinski B
- Zacharis G
- Zaffaroni E
- Zahreddine J
- Zaitsev AM
- Zakareishvili T
- Zakharchuk N
- Zambito S
- Zanzi D
- Zaripovas DR
- Zeissner S
- Zeitnitz C
- Zemaityte G
- Zeng JC
- Zenin O
- Zenis T
- Zerwas D
- Zgubic M
- Zhang B
- Zhang DF
- Zhang G
- Zhang H
- Zhang J
- Zhang L
- Zhang M
- Zhang R
- Zhang S
- Zhang X
- Zhang Y
- Zhang Z
- Zhao P
- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
- Zhou B
- Zhou C
- Zhou M
- Zhou MS
- Zhou N
- Zhou Y
- Zhu C
- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine N
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zivkovic L
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zu Theenhausen HM
- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
Results of a search for new phenomena in final states with an energetic jet and large missing transverse momentum are reported. The search uses 20.3 fb−1 of √ s = 8 TeV data collected in 2012 with the ATLAS detector at the LHC. Events are required to have at least one jet with pT > 120 GeV and no leptons. Nine signal regions are considered with increasing missing transverse momentum requirements between Emiss T > 150 GeV and Emiss T > 700 GeV. Good agreement is observed between the number of events in data and Standard Model expectations. The results are translated into exclusion limits on models with either large extra spatial dimensions, pair production of weakly interacting dark matter candidates, or production of very light gravitinos in a gauge-mediated supersymmetric model. In addition, limits on the production of an invisibly decaying Higgs-like boson leading to similar topologies in the final state are presente
Chromosome 9p21 SNPs associated with multiple disease phenotypes correlate with ANRIL expression
- Author
- A Gimelbrant
- A Gschwendtner
- A Helgadottir
- A Helgadottir
- A Lal
- AL Dixon
- AM Goldstein
- B Rhead
- BE Stranger
- Bernard Keavney
- Bongani M. Mayosi
- CA McKenzie
- CJ Hussussian
- D Farre
- D Melzer
- DT Bishop
- DW Parsons
- E Hara
- E Pasmant
- E Zeggini
- E Zeggini
- EE Schadt
- ET Cirulli
- Greg Gibson
- HM Broadbent
- J Chen
- J Chen
- J Gil
- J Healy
- J Krishnamurthy
- J Vandesompele
- J-M Li
- John Burn
- K Duesing
- K Silander
- K Zhang
- KD Robertson
- KE Driver
- L Folkersen
- L Yan
- LJ Scott
- M Andreassi
- M Berwick
- M Matarin
- M Moritani
- M Wrensch
- Mauro Santibanez Koref
- MC Campbell
- MD Teare
- Michael S. Cunnington
- MQ Yang
- MY Kang
- NJ Samani
- O Jarinova
- O Straume
- R Kumar
- R McPherson
- S Campino
- S Gonzalez
- S Griffiths-Jones
- S Rozen
- S Sakano
- S Shete
- S Züchner
- SA Gayther
- T Debniak
- T Debniak
- T Dębniak
- T Minamino
- T Pastinen
- T Pastinen
- TA Manolio
- TR Mercer
- V Plagnol
- W Wang
- W Yu
- WY Kim
- X Messeguer
- X Wang
- X Zhang
- Y Liu
- Y Liu
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2010
- Field of study
Author Summary Genetic variants on chromosome 9p21 have been associated with several important diseases including coronary artery disease, diabetes, and multiple cancers. Most of the risk variants in this region do not alter any protein sequence and are therefore likely to act by influencing the expression of nearby genes. We investigated whether chromosome 9p21 variants are correlated with expression of the three nearest genes ( CDKN2A , CDKN2B , and ANRIL ) which might mediate the association with disease. Using two different techniques to study effects on expression in blood from two separate populations of healthy volunteers, we show that variants associated with disease are all correlated with ANRIL expression, but associations with the other two genes are weaker and less consistent. Multiple genetic variants are independently associated with expression of all three genes. Although total expression levels of CDKN2A , CDKN2B , and ANRIL are positively correlated, individual genetic variants influence ANRIL and CDKN2B expression in opposite directions, suggesting a possible role of ANRIL in CDKN2B regulation. Our study suggests that modulation of ANRIL expression mediates susceptibility to several important human diseases
A Directed Molecular Evolution Approach to Improved Immunogenicity of the HIV-1 Envelope Glycoprotein
- Author
- A Crameri
- A Morner
- A Trkola
- AD Brideau-Andersen
- AJ Hessell
- AK Pilgrim
- AL Kurtzman
- B Dey
- BD Walker
- BF Haynes
- C Grundner
- CC Huang
- CF Barbas 3rd
- Christos J. Petropoulos
- Clive M. Gray
- CN Scanlan
- D Apt
- DD Richman
- Dennis R. Burton
- DL Kothe
- DL Kothe
- DN Sather
- EA Weaver
- Ellaine B. Mariano
- EO Saphire
- ET Crooks
- G Zanetti
- GB Karlsson Hedestam
- Helen Chen
- HX Liao
- IK Srivastava
- J Haas
- J Hammonds
- J Liu
- J Minshull
- JE Ness
- JM Bacher
- JM Binley
- JM Binley
- JM Binley
- John A. Ballantyne
- JR Mascola
- K Bojang
- L Stamatatos
- LA Castle
- LC Dupuy
- Li Xu
- LM Walker
- M Pancera
- Michael B. Zwick
- Michael Chambers
- NA Doria-Rose
- NR Derby
- P Carotenuto
- P Mooij
- P Roben
- P Zhu
- Paul W. H. I. Parren
- PD Kwong
- PD Kwong
- PF Zhang
- R Hofmann-Lehmann
- R Kunert
- R Pantophlet
- R Pantophlet
- R Pantophlet
- R Shibata
- R Wyatt
- Rebecca I. Boenig
- Robert G. Whalen
- S Beddows
- S Kumar
- S Lazetic
- S Rerks-Ngarm
- S Selvarajah
- S Wang
- S Wang
- SD Frost
- SD Frost
- Sean X. Du
- SR Leong
- Susan Tang
- SW Barnett
- SX Du
- SX Du
- T Zhou
- Terri Wrin
- TT Wu
- TW Baba
- Wenge Zhang
- WP Stemmer
- X Wei
- X Wu
- X Wu
- X Xiao
- X Yang
- X Yang
- X Yang
- Y Li
- Y Li
- Y Nishimura
- YK Kang
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
A prophylactic vaccine is needed to slow the spread of HIV-1 infection. Optimization of the wild-type envelope glycoproteins to create immunogens that can elicit effective neutralizing antibodies is a high priority. Starting with ten genes encoding subtype B HIV-1 gp120 envelope glycoproteins and using in vitro homologous DNA recombination, we created chimeric gp120 variants that were screened for their ability to bind neutralizing monoclonal antibodies. Hundreds of variants were identified with novel antigenic phenotypes that exhibit considerable sequence diversity. Immunization of rabbits with these gp120 variants demonstrated that the majority can induce neutralizing antibodies to HIV-1. One novel variant, called ST-008, induced significantly improved neutralizing antibody responses when assayed against a large panel of primary HIV-1 isolates. Further study of various deletion constructs of ST-008 showed that the enhanced immunogenicity results from a combination of effective DNA priming, an enhanced V3-based response, and an improved response to the constant backbone sequences
Erratum to: 36th International Symposium on Intensive Care and Emergency Medicine
- Author
- A Boing
- A Gaspari
- A Kübler
- A Marudi
- A Matheson
- A Tridente
- A. A. Amelia
- A. Abbas
- A. Adams
- A. Agnoli
- A. Al Khulaifi
- A. Al Khulaifi
- A. Aldawood
- A. AlHussain
- A. Ali
- A. Almenyar
- A. Alrashid
- A. Alsheikhly
- A. Alves
- A. Ankuli
- A. Arbouti
- A. Armaganidis
- A. Artigas
- A. Asimakos
- A. B. Belli
- A. B. Cayci Sivri
- A. Bandyopadhyay
- A. Basu
- A. Bedova
- A. Belli
- A. Ben Souissi
- A. Ben Souissi
- A. Ben Souissi
- A. Ben Souissi
- A. Bond
- A. Borgman
- A. Borowska
- A. Boutin
- A. Boutin
- A. Boyce
- A. Braschi
- A. Budnyuk
- A. C. Rodrigues
- A. Cariou
- A. Casazza
- A. Chakraborty
- A. Cirodde
- A. Crizaldo Toledo
- A. Cudia
- A. Cuoghi
- A. D. Dubrawski
- A. Dardashti
- A. Dean
- A. Dean
- A. Delfosse
- A. Dhaliwal
- A. Dhaliwal
- A. Dobru
- A. Docherty
- A. Doronzio
- A. Douvdevani
- A. Dukoff-Gordon
- A. Durward
- A. E Van den Berg
- A. Eden
- A. Eissa
- A. Enguix-Armada
- A. Escalante
- A. Escalante
- A. Ezzamouri
- A. F. Francke
- A. F. Turgeon
- A. F. Turgeon
- A. F. Van Rootselaar
- A. F. Van Rootselaar
- A. Fayed
- A. Fernandez
- A. Fischer
- A. Fogagnolo
- A. Fuller
- A. Fuller
- A. G. Murchison
- A. G. Proudfoot
- A. Garcia
- A. Garcia-Alcantara
- A. Garcia-De la Torre
- A. Garland
- A. Garland
- A. Gaspari
- A. Ghuysen
- A. Giannini
- A. Gobatto
- A. Goloubev
- A. Grubb
- A. Gruber
- A. Gultekin
- A. H. Zwinderman
- A. Hagiwara
- A. Hana
- A. Hana
- A. Harts
- A. Hejazi
- A. Hernández-Lozano
- A. Hernández-Lozano
- A. Hovingh
- A. Huber
- A. Itani
- A. J. Groeneveld
- A. J. Hoogendijk
- A. K. Kink
- A. K. Kuzovlev
- A. K. Tashima
- A. Kapuagasi
- A. Kar
- A. Kaskovagheorgescu
- A. Kaushal
- A. Khaghani
- A. Kindawi
- A. Kleinsasser
- A. Kodaira
- A. Kutz
- A. Kuzovlev
- A. Körner
- A. L Luciani
- A. L. Lange
- A. Lepape
- A. Lesmes
- A. Lima
- A. Lima
- A. Linder
- A. Loza
- A. López Lago
- A. M. Magnuson
- A. M. Tuip-de Boer
- A. Maaoui
- A. Machado
- A. Mahrous
- A. Malamas
- A. Marinho
- A. Marinho
- A. Marinho
- A. Marinho
- A. Marinho
- A. Marinho
- A. Marinho
- A. Martins
- A. Marudi
- A. Marudi
- A. Matroud
- A. Mebazaa
- A. Mignon
- A. Min
- A. Molokhia
- A. Molokhia
- A. Molokhia
- A. Molokhia
- A. Molokhia
- A. Morelli
- A. Morris
- A. Motos
- A. N. Ahmad
- A. Nadeem
- A. Nau
- A. Neill
- A. Nyman
- A. Núñez
- A. Oglinda
- A. Omar
- A. Omar
- A. Omar
- A. Omar
- A. Orlando
- A. Osipov
- A. Ozcan
- A. O’Loughlin
- A. O’Sullivan
- A. O’Sullivan
- A. O’Sullivan
- A. P. Pagano
- A. P. Torelly
- A. Pagano
- A. Patarchi
- A. Pautova
- A. Pavli
- A. Pesenti
- A. Prekates
- A. Psirides
- A. Puerto-Morlan
- A. Puxty
- A. Pérez-Calatayud
- A. R. Robles Caballero
- A. Radford
- A. Raithatha
- A. Raithatha
- A. Raithatha
- A. Reiter
- A. Rhodes
- A. Rhodes
- A. Rhodes
- A. Riahi
- A. Riahi
- A. Riahi
- A. Riahi
- A. Riviere
- A. Riviere
- A. Riviere
- A. Robles
- A. Rogers
- A. Rousseau
- A. Roze des Ordons
- A. Ruijter
- A. Rutten
- A. S. Alsaawi
- A. S. Stieglitz
- A. Salgueiro
- A. Salgueiro
- A. Salgueiro
- A. Sarmento
- A. Sarmento
- A. Savitsky
- A. Scheirer
- A. Schiavi
- A. Seldon
- A. Sell
- A. Sen
- A. Sergeev
- A. Shabanov
- A. Shafie
- A. Shafquat
- A. Shiraishi
- A. Silva-Pinto
- A. Silva-Pinto
- A. Spassov
- A. Stella
- A. Steuber
- A. Stieglitz
- A. Stilwell
- A. Sánchez-López
- A. Sánchez-López
- A. Taha
- A. Taha
- A. Taskin
- A. Temple
- A. Tinmouth
- A. Tinmouth
- A. Torres
- A. Torres
- A. Torres
- A. Touborg Lassen
- A. Tridente
- A. Tridente
- A. Tridente
- A. Tridente
- A. Trifi
- A. Trifi
- A. Tsimogianni
- A. Turgeon
- A. Turgeon
- A. Vakalos
- A. Vakalos
- A. Vakalos
- A. Vakalos
- A. Van Assche
- A. Van Assche
- A. Varma
- A. Vassi
- A. Vercueil
- A. Vieillard-Baron
- A. Violaki
- A. Violaki
- A. Vuylsteke
- A. W. Waldmann
- A. Wald
- A. Waldmann
- A. Waldmann
- A. Webber
- A. Wheeler
- A. Yalcin
- A. Zepeda-Mendoza
- A. Zerman
- AF Donneau
- AK Güler
- AT Tesnieres
- B Adamik
- B Avard
- B. Al Raiy
- B. AlGhamdi
- B. Alhamadi
- B. Alotaibi
- B. Avard
- B. Avard
- B. Balicco
- B. Basarýk Aydogan
- B. Besen
- B. Bilgili
- B. Bilgili
- B. Borgatta
- B. Bouhemad
- B. C. Civantos
- B. Carr
- B. Civantos
- B. De Cagny
- B. De Cagny
- B. De Cagny
- B. De Cagny
- B. De Laat
- B. Delwarde
- B. Eilers
- B. François
- B. François
- B. Fyntanidou
- B. Fyntanidou
- B. Fyntanidou
- B. Goncalves
- B. Guidet
- B. J. Hunt
- B. Jeribi
- B. Jones
- B. K. Lopansri
- B. Khwannimit
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- Z. Aidoni
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- Z. Uzundurukan
- Z. Ülger
- Ó. Martínez González
- Ö. Özdedeoglu
- Ü. Gaygýsýz
- Ý. Kara
- Publication venue
- Springer Nature
- Publication date
- 01/01/2016
- Field of study
[This corrects the article DOI: 10.1186/s13054-016-1208-6.]
A second generation human haplotype map of over 3.1 million SNPs
- Author
- Abecasis GR
- Adebamowo CA
- Adewole IF
- Ajayi I
- Altshuler D
- Altshuler D
- Altshuler D
- An D
- Aniagwu T
- Auton A
- Ballinger DG
- Barrett J
- Barry R
- Bellemare G
- Belmont JW
- Belmont JW
- Bentley DR
- Bird CP
- Birren BW
- Blumenstiel B
- Bottolo L
- Boudreau A
- Brooks LD
- Burton J
- Cai D
- Camargo A
- Cardin N
- Cardon LR
- Carter NP
- Chagnon F
- Chakravarti A
- Chee MS
- Chen PE
- Chen Z
- Chretien YR
- Chu X
- Clarke G
- Clayton EW
- Clee CM
- Collins FS
- Cox DR
- Cutler DJ
- Daly MJ
- Daly MJ
- de Bakker PI
- Defelice M
- Delgado M
- Deloukas P
- Dermitzakis ET
- Dixon M
- Donnelly P
- Evans DM
- Eyheramendy S
- Faggart M
- Fan JB
- Feolo M
- Ferretti V
- Foster MW
- Frazer KA
- Freeman C
- Fu H
- Fukushima Y
- Fulton LL
- Gabriel SB
- Gabriel SB
- Galver LM
- Gao Y
- Gibbs RA
- Gibbs RA
- Godbout M
- Goyette M
- Griffiths M
- Guan W
- Gunderson K
- Gupta S
- Guyer MS
- Gwilliam R
- Han H
- Hardenbol P
- He Y
- Hinds DA
- Holden AL
- Hu H
- Hu W
- Huang W
- Hudson TJ
- Hunt S
- Jamieson R
- Jin L
- Jones MC
- Kang L
- Kashuk CS
- Kato K
- Kawaguchi T
- Kennedy K
- Kent A
- Kitamoto T
- Knoppers BM
- Koboldt DC
- Krishnan L
- Kwok PY
- L'Archevêque P
- Leal SM
- Leboeuf M
- Leppert MF
- Li C
- Li Q
- Li Y
- Lin S
- Lin W
- Liu S
- Liu Y
- Macer DR
- Mak W
- Maller J
- Marchini J
- Marshall PA
- Matsuda I
- McCarroll S
- McEwen JE
- McLay K
- McVean G
- Miller RD
- Montpetit A
- Moore J
- Morizono T
- Morris AP
- Morrison J
- Mullikin JC
- Munro HM
- Murray SS
- Muzny D
- Myers S
- Nagashima A
- Nakamura Y
- Nazareth L
- Nguyen H
- Niikawa N
- Nkwodimmah C
- Ohnishi Y
- Oliphant AR
- Olivier JF
- Onofrio RC
- Onofrio RC
- Pan H
- Parkin M
- Pasternak S
- Patterson N
- Pawlikowska L
- Pe'er I
- Peiffer A
- Peterson JL
- Phillips MS
- Plumb RW
- Powell D
- Price A
- Purcell S
- Qin ZS
- Qiu R
- Richter DJ
- Richter DJ
- Rogers J
- Ross MT
- Rotimi CN
- Roumy S
- Roy J
- Royal CD
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- Tam PK
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- Tang X
- Tello-Ruiz MK
- Thomas DJ
- Thorisson GA
- Tsui LC
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- Tsunoda T
- Tsunoda T
- Varilly P
- Verner A
- Wallenburg JC
- Wang H
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- Wang Y
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- Watkin J
- Waye MM
- Weinstock GM
- Weir BS
- Wheeler DA
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- Whittaker P
- Willey DL
- Willis TD
- Wilson RK
- Winchester E
- Wong JT
- Xiao M
- Xiong X
- Xu L
- Xue H
- Yakub I
- Yang H
- Yao Z
- Yu F
- Yu J
- Zacharia LF
- Zeng C
- Zeng C
- Zhang B
- Zhang H
- Zhang Q
- Zhao H
- Zhao H
- Zhao H
- Zhou J
- Ziaugra L
- Ziaugra L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2007
- Field of study
We describe the Phase II HapMap, which characterizes over 3.1 million human single nucleotide polymorphisms (SNPs) genotyped in 270 individuals from four geographically diverse populations and includes 25-35% of common SNP variation in the populations surveyed. The map is estimated to capture untyped common variation with an average maximum r(2) of between 0.9 and 0.96 depending on population. We demonstrate that the current generation of commercial genome-wide genotyping products captures common Phase II SNPs with an average maximum r(2) of up to 0.8 in African and up to 0.95 in non-African populations, and that potential gains in power in association studies can be obtained through imputation. These data also reveal novel aspects of the structure of linkage disequilibrium. We show that 10-30% of pairs of individuals within a population share at least one region of extended genetic identity arising from recent ancestry and that up to 1% of all common variants are untaggable, primarily because they lie within recombination hotspots. We show that recombination rates vary systematically around genes and between genes of different function. Finally, we demonstrate increased differentiation at non-synonymous, compared to synonymous, SNPs, resulting from systematic differences in the strength or efficacy of natural selection between populations.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/62863/1/nature06258.pd
Pan-cancer analysis of whole genomes
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- The ICGC/TCGA Pan-Cancer Analysis of Whole Genomes Consortium View Correspondence (jump link)
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- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
Pooled analysis of WHO Surgical Safety Checklist use and mortality after emergency laparotomy
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- Aamer A
- Aaniana K
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- Abaliksta T
- Abantanga F
- Abantanga F
- Abbas A
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- Abbasy J
- Abbo O
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- Abbott T
- Abbouch M
- Abd El Galeel A
- Abd El Hameed OS
- Abd El Hamid N
- Abd El Salam Y
- Abd El Slam M
- Abd El-Salam F
- Abd Elrasoul Y
- Abd-Elmawla M
- Abd-Elrasoul Y
- Abdallah E
- Abdallah E
- Abdel-Aty A
- Abdel-Hameed N
- Abdel-Kader SM
- Abdel-Wahab NYE
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- Abdelazeam AR
- Abdelazim G
- Abdelaziz MA
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- Abdelfatah A
- AbdelFattah I
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- Abdelghany S
- Abdelhady S
- Abdelhamed R
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- Kosai NR
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- Mohamed M
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- Zulkifli A
- Zumbuhl L
- Publication venue
- Publication date
- 15/10/2018
- Field of study
Background The World Health Organization (WHO) Surgical Safety Checklist has fostered safe practice for 10 years, yet its place in emergency surgery has not been assessed on a global scale. The aim of this study was to evaluate reported checklist use in emergency settings and examine the relationship with perioperative mortality in patients who had emergency laparotomy. Methods In two multinational cohort studies, adults undergoing emergency laparotomy were compared with those having elective gastrointestinal surgery. Relationships between reported checklist use and mortality were determined using multivariable logistic regression and bootstrapped simulation. Results Of 12 296 patients included from 76 countries, 4843 underwent emergency laparotomy. After adjusting for patient and disease factors, checklist use before emergency laparotomy was more common in countries with a high Human Development Index (HDI) (2455 of 2741, 89.6 per cent) compared with that in countries with a middle (753 of 1242, 60.6 per cent; odds ratio (OR) 0.17, 95 per cent c.i. 0.14 to 0.21, P <0001) or low (363 of 860, 422 per cent; OR 008, 007 to 010, P <0.001) HDI. Checklist use was less common in elective surgery than for emergency laparotomy in high-HDI countries (risk difference -94 (95 per cent c.i. -11.9 to -6.9) per cent; P <0001), but the relationship was reversed in low-HDI countries (+121 (+7.0 to +173) per cent; P <0001). In multivariable models, checklist use was associated with a lower 30-day perioperative mortality (OR 0.60, 0.50 to 073; P <0.001). The greatest absolute benefit was seen for emergency surgery in low- and middle-HDI countries. Conclusion Checklist use in emergency laparotomy was associated with a significantly lower perioperative mortality rate. Checklist use in low-HDI countries was half that in high-HDI countries.Peer reviewe
Cancer Biomarker Discovery: The Entropic Hallmark
- Author
- A Alcaraz
- A Arabzadeh
- A Bagri
- A Balbin
- A Bantis
- A Ben-Naim
- A Bolander
- A Buhmeida
- A Burykin
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- T Murakami
- T Oyama
- T Schlomm
- T Schlomm
- T Sheridan
- T Shimizu
- T Sobolik-Delmaire
- T Sun
- T Takahashi
- T Takeda
- T Thornburg
- T Watanabe
- T Yu
- TB Lewis
- TD Prickett
- TH Ecke
- TJ McDonnell
- TJ Park
- TL Schmit
- TL Schmit
- TO Nielsen
- TR Mercer
- U Brinck
- U Graeven
- U Gruneberg
- UV Wesley
- V Ambros
- V Barak
- V Fradet
- V Molinie
- V Molinie
- V Molinie
- V Molinie
- V Molinie
- V Molinie
- V Pekovic
- V Rajaram
- V Samanna
- V Wessagowit
- V Winnepenninckx
- VD Acevedo
- VK Vanguri
- VL Verstraeten
- W Chen
- W Cosme-Blanco
- W Gade
- W Ritchie
- W Spirkl
- W Tettelbach
- W Wang
- W Weichert
- W Weichert
- W Weichert
- WB Isaacs
- WD Dunsmuir
- WE Naugler
- William C. S. Cho
- WK Kaufmann
- WK Lee
- X Chen
- X Cheng
- X Dong
- X Dong
- X Huang
- X Li
- X Liu
- X Liu
- X Meng
- X Ouyang
- X Tang
- X Wu
- X Zhang
- XJ Yang
- XQ Wang
- Y Bu
- Y Cai
- Y Deng
- Y Ito
- Y Katagiri
- Y Kozuka
- Y Lai
- Y Liu
- Y Lu
- Y Lu
- Y Ohyama
- Y Shav-Tal
- Y Sugiyama
- Y Sun
- Y Wang
- Y Wang
- Y Wu
- Y Yamamoto
- Y Yang
- Y Zhou
- YB Lim
- YC Chao
- YJ Ko
- YL Liu
- YN Liu
- YT Sasaki
- YT Sasaki
- YU Katagiri
- Z Jiang
- Z Jiang
- Z Jiang
- Z Jiang
- Z Jiang
- Z Rudzki
- ZA Dotan
- ZR Yurkovetsky
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2010
- Field of study
Background: It is a commonly accepted belief that cancer cells modify their transcriptional state during the progression of the disease. We propose that the progression of cancer cells towards malignant phenotypes can be efficiently tracked using high-throughput technologies that follow the gradual changes observed in the gene expression profiles by employing Shannon's mathematical theory of communication. Methods based on Information Theory can then quantify the divergence of cancer cells' transcriptional profiles from those of normally appearing cells of the originating tissues. The relevance of the proposed methods can be evaluated using microarray datasets available in the public domain but the method is in principle applicable to other high-throughput methods. Methodology/Principal Findings: Using melanoma and prostate cancer datasets we illustrate how it is possible to employ Shannon Entropy and the Jensen-Shannon divergence to trace the transcriptional changes progression of the disease. We establish how the variations of these two measures correlate with established biomarkers of cancer progression. The Information Theory measures allow us to identify novel biomarkers for both progressive and relatively more sudden transcriptional changes leading to malignant phenotypes. At the same time, the methodology was able to validate a large number of genes and processes that seem to be implicated in the progression of melanoma and prostate cancer. Conclusions/Significance: We thus present a quantitative guiding rule, a new unifying hallmark of cancer: the cancer cell's transcriptome changes lead to measurable observed transitions of Normalized Shannon Entropy values (as measured by high-throughput technologies). At the same time, tumor cells increment their divergence from the normal tissue profile increasing their disorder via creation of states that we might not directly measure. This unifying hallmark allows, via the the Jensen-Shannon divergence, to identify the arrow of time of the processes from the gene expression profiles, and helps to map the phenotypical and molecular hallmarks of specific cancer subtypes. The deep mathematical basis of the approach allows us to suggest that this principle is, hopefully, of general applicability for other diseases
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