262 research outputs found
Developing a coding scheme for analysing classroom dialogue across educational contexts
- Author
- Publication venue
- Publication date
- 18/03/2016
- Field of study
The research reported sought to develop a framework for systematically analysing classroom dialogue for application across a range of educational settings. The paper outlines the development and refinement of a coding scheme that attempts to represent and operationalise commonalities amongst some key theorists in the field concerning productive forms of educational dialogue. The team has tested it using video recordings from classroom settings in the UK and Mexico, across age phases, subject areas, and different interactional contexts including whole class, group and paired work. Our Scheme for Educational Dialogue Analysis (SEDA) is situated within a sociocultural paradigm, and draws on Hymes' Ethnography of Communication to highlight the importance of context. We examined how such a tool could be used in practice. We found that concentrating on the ‘communicative act’ to explore dialogue between participants was an appropriate level of granularity, while clustering the 33 resulting codes according to function of the acts helped to highlight dialogic sequences within lessons. We report on the application of the scheme in two different learning contexts and reflect on its fitness for purpose, including perceived limitations. Development of specialised sub-schemes and a version for teachers is underway
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Developing a coding scheme for analysing classroom dialogue across d educational contexts.
- Author
- Publication venue
- Learning, Culture and Social Interaction
- Publication date
- 30/06/2016
- Field of study
The research reported sought to develop a framework for systematically analysing classroom dialogue for application across a range of educational settings. The paper outlines the development and refinement of a coding scheme that attempts to represent and operationalise commonalities amongst some key theorists in the field concerning productive forms of educational dialogue. The team has tested it using video recordings from classroom settings in the UK and Mexico, across age phases, subject areas, and different interactional contexts including whole class, group and paired work. Our Scheme for Educational Dialogue Analysis (SEDA) is situated within a sociocultural paradigm, and draws on Hymes' Ethnography of Communication to highlight the importance of context. We examined how such a tool could be used in practice. We found that concentrating on the ‘communicative act’ to explore dialogue between participants was an appropriate level of granularity, while clustering the 33 resulting codes according to function of the acts helped to highlight dialogic sequences within lessons. We report on the application of the scheme in two different learning contexts and reflect on its fitness for purpose, including perceived limitations. Development of specialised sub-schemes and a version for teachers is underway.This collaborative work was carried out for a project entitled “A Tool for Analysing Dialogic Interactions in Classrooms” (http://tinyurl.com/BAdialogue) funded through the British Academy International Partnership and Mobility Scheme (ref. RG66509), between January 2013 - December 2015. We are most grateful to colleagues on the project teams who made significant contributions and helpful input during development and testing of the scheme and preparation of the manuscript, including Farah Ahmed, Riikka Hofmann, Christine Howe, Ruth Kershner, Fiona Jackson, Karen Littleton, Neil Mercer, Paul Warwick (UK team); Mariana Alarcón, Nube Estrada, Erika Gil, Kissy Guzmán, Flora Hernández, José Hernández, Haydeé Pedraza, Ana Luisa Rubio, Brenda Itzel Sánchez, Ana Laura Trigo, Maricela Velez (Mexico team). We also thank all of the teachers (especially Lloyd and Tania) and students who participated in our previous research from which examples were taken. We appreciate the support of the Economic and Social Research Council, sponsor of most of the UK team’s work in this area over the years
CONTRIBUTIONS TO RICKETTSIOSES RESEARCH IN COLOMBIA (1917-1943), LUIS B. PATIÑO CAMARGO
- Author
- Acosta J
- Arroyave E
- Barrera S
- Carlos A. BOTERO-GARCÍA
- Faccini-Martínez ÁA
- Faccini-Martínez ÁA
- Faccini-Martínez ÁA
- Hidalgo M
- Hidalgo M
- Hidalgo M
- Hidalgo M
- Hidalgo M
- Hidalgo M
- Hidalgo M
- Lombana-Barreneche JM
- Londoño AF
- Marylin HIDALGO
- Merhej V
- Miranda AM
- Miranda J
- Miranda J
- Miranda J
- Pacheco-García OE
- Padmanabha H
- Patino L
- Patiño JF
- Patiño-Camargo L
- Patiño-Camargo L
- Patiño-Camargo L
- Patiño-Camargo L
- Patiño-Camargo L
- Patiño-Camargo L
- Patiño-Niño JA
- Pérez-Osorio CE
- Quintero JC
- Ramírez N
- Ramírez-Hernández A
- Ríos R
- Szabó MP
- Álvaro A. FACCINI-MARTÍNEZ
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
Observation of associated near-side and away-side long-range correlations in √sNN=5.02 TeV proton-lead collisions with the ATLAS detector
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- Aad G
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- Abdallah J
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- Abreu H
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- Alessandria F
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- Sánchez J
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- Thomas JP
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- Thompson PD
- Thompson PD
- Thomsen LA
- Thomson E
- Thomson M
- Thong WM
- Thun RP
- Tian F
- Tibbetts MJ
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- Tikhonov YA
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- Tollefson K
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- Tsarouchas C
- Tseng JC
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- Tsiareshka PV
- Tsionou D
- Tsipolitis G
- Tsiskaridze S
- Tsiskaridze V
- Tskhadadze EG
- Tsukerman II
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- Tsung JW
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- Tudorache A
- Tudorache V
- Tuggle JM
- Turala M
- Turecek D
- Turk Cakir I
- Turra R
- Tuts PM
- Tykhonov A
- Tylmad M
- Tyndel M
- Tzanakos G
- Uchida K
- Ueda I
- Ueno R
- Ughetto M
- Ugland M
- Uhlenbrock M
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- Urbaniec D
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- Valentinetti S
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- Valladolid Gallego E
- Vallecorsa S
- Valls Ferrer JA
- Van Berg R
- Van Der Deijl PC
- van der Geer R
- van der Graaf H
- Van Der Leeuw R
- van der Poel E
- van der Ster D
- van Eldik N
- van Gemmeren P
- Van Nieuwkoop J
- van Vulpen I
- Vanadia M
- Vandelli W
- Vaniachine A
- Vankov P
- Vannucci F
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- Varnes EW
- Varol T
- Varouchas D
- Vartapetian A
- Varvell KE
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- Vazeille F
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- Vichou I
- Vickey Boeriu OE
- Vickey T
- Viehhauser GH
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- Villa M
- Villaplana Perez M
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- Vinogradov VB
- Virzi J
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- Vivarelli I
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- von der Schmitt H
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- Vorobel V
- Vorwerk V
- Vos M
- Voss R
- Vossebeld JH
- Vranjes Milosavljevic M
- Vranjes N
- Vrba V
- Vreeswijk M
- Vu Anh T
- Vuillermet R
- Vukotic I
- Vykydal Z
- Wagner P
- Wagner W
- Wahlen H
- Wahrmund S
- Wakabayashi J
- Walch S
- Walder J
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- Waller P
- Walsh B
- Wang C
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- Wang H
- Wang J
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- Wang SM
- Wang T
- Wang X
- Warburton A
- Ward CP
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- Warsinsky M
- Washbrook A
- Wasicki C
- Watanabe I
- Watkins PM
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- Waugh BM
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- Weidberg AR
- Weigell P
- Weingarten J
- Weiser C
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- Wendland D
- Weng Z
- Wengler T
- Wenig S
- Wermes N
- Werner M
- Werner P
- Werth M
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- Wetter J
- Weydert C
- Whalen K
- White A
- White MJ
- White S
- Whitehead SR
- Whiteson D
- Whittington D
- Wicke D
- Wickens FJ
- Wiedenmann W
- Wielers M
- Wienemann P
- Wiglesworth C
- Wiik-Fuchs LA
- Wijeratne PA
- Wildauer A
- Wildt MA
- Wilhelm I
- Wilkens HG
- Will JZ
- Williams E
- Williams HH
- Williams S
- Willis W
- Willocq S
- Wilson A
- Wilson JA
- Wilson MG
- Wingerter-Seez I
- Winkelmann S
- Winklmeier F
- Wittgen M
- Wittig T
- Wittkowski J
- Wollstadt SJ
- Wolter MW
- Wolters H
- Wong WC
- Wooden G
- Wosiek BK
- Wotschack J
- Woudstra MJ
- Wozniak KW
- Wraight K
- Wright M
- Wrona B
- Wu SL
- Wu X
- Wu Y
- Wulf E
- Wynne BM
- Xella S
- Xiao M
- Xie S
- Xu C
- Xu D
- Xu L
- Yabsley B
- Yacoob S
- Yamada M
- Yamaguchi H
- Yamamoto A
- Yamamoto K
- Yamamoto S
- Yamamura T
- Yamanaka T
- Yamauchi K
- Yamazaki T
- Yamazaki Y
- Yan Z
- Yang H
- Yang H
- Yang UK
- Yang Y
- Yang Z
- Yanush S
- Yao L
- Yasu Y
- Yatsenko E
- Ye J
- Ye S
- Yen AL
- Yilmaz M
- Yoosoofmiya R
- Yorita K
- Yoshida R
- Yoshihara K
- Young C
- Young CJ
- Youssef S
- Yu D
- Yu DR
- Yu J
- Yu J
- Yuan L
- Yurkewicz A
- Zabinski B
- Zaidan R
- Zaitsev AM
- Zambito S
- Zanello L
- Zanzi D
- Zaytsev A
- Zeitnitz C
- Zeman M
- Zemla A
- Zenin O
- Zeniš T
- Zerwas D
- Zevi Della Porta G
- Zhang D
- Zhang H
- Zhang J
- Zhang L
- Zhang X
- Zhang Z
- Zhao L
- Zhao Z
- Zhemchugov A
- Zhong J
- Zhou B
- Zhou N
- Zhou Y
- Zhu CG
- Zhu H
- Zhu J
- Zhu Y
- Zhuang X
- Zhuravlov V
- Zibell A
- Zieminska D
- Zimin NI
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zitoun R
- Zivković L
- Zmouchko VV
- Zobernig G
- Zoccoli A
- Zur Nedden M
- Zutshi V
- Zwalinski L
- Publication venue
- American Physical Society
- Publication date
- 01/01/2012
- Field of study
Two-particle correlations in relative azimuthal angle (Δϕ) and pseudorapidity (Δη) are measured in √sNN=5.02 TeV p+Pb collisions using the ATLAS detector at the LHC. The measurements are performed using approximately 1 μb-1 of data as a function of transverse momentum (pT) and the transverse energy (ΣETPb) summed over 3.1<η<4.9 in the direction of the Pb beam. The correlation function, constructed from charged particles, exhibits a long-range (2<|Δη|<5) “near-side” (Δϕ∼0) correlation that grows rapidly with increasing ΣETPb. A long-range “away-side” (Δϕ∼π) correlation, obtained by subtracting the expected contributions from recoiling dijets and other sources estimated using events with small ΣETPb, is found to match the near-side correlation in magnitude, shape (in Δη and Δϕ) and ΣETPb dependence. The resultant Δϕ correlation is approximately symmetric about π/2, and is consistent with a dominant cos2Δϕ modulation for all ΣETPb ranges and particle pT
Metabolic flux analysis and the NAD(P)H/NAD(P) + ratios in chemostat cultures of Azotobacter vinelandii
- Author
- A Díaz-Barrera
- A Díaz-Barrera
- A Díaz-Barrera
- A Díaz-Barrera
- A Kaplan
- A Richard
- AIC Obanye
- AM Sanchez
- Andres García
- AP Zeng
- C Flores
- C Peña
- C Peña
- Carlos Peña
- CF Snook
- CM Henriksen
- Daniel Segura
- E Galindo
- E Lozano
- E Post
- FL Felipe De
- G Miller
- H Bergmeyer
- I Rocha
- J Fu
- J Oelze
- JC Choi
- JN Lee
- Joan Albiol
- K Gómez-Pazarín
- K Inomura
- K Linkerhägner
- KJ Livak
- MT Castillo
- MT Castillo
- OH Lowry
- P Tatnell
- Pau Ferrer
- PF Stanbury
- PJ Senior
- R Noguez
- S Centeno-Leija
- S Heux
- SJ Berrios-Rivera
- SJ Berrios-Rivera
- SJ Lim
- SK Spaans
- T Anderlei
- T Bao
- T Barry
- Tania Castillo
- U Maier
- W Sabra
- W Sabra
- YV Bertsova
- YV Bertsova
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2018
- Field of study
Azotobacter vinelandii is a bacterium that produces alginate and polyhydroxybutyrate (P3HB); however, the role of NAD(P)H/NAD(P) + ratios on the metabolic fluxes through biosynthesis pathways of these biopolymers remains unknown. The aim of this study was to evaluate the NAD(P)H/NAD(P) + ratios and the metabolic fluxes involved in alginate and P3HB biosynthesis, under oxygen-limiting and non-limiting oxygen conditions. The results reveal that changes in the oxygen availability have an important effect on the metabolic fluxes and intracellular NADPH/NADP + ratio, showing that at the lowest OTR (2.4 mmol L −1 h −1), the flux through the tricarboxylic acid (TCA) cycle decreased 27.6-fold, but the flux through the P3HB biosynthesis increased 6.6-fold in contrast to the cultures without oxygen limitation (OTR = 14.6 mmol L −1 h −1). This was consistent with the increase in the level of transcription of phbB and the P3HB biosynthesis. In addition, under conditions without oxygen limitation, there was an increase in the carbon uptake rate (twofold), as well as in the flux through the pentose phosphate (PP) pathway (4.8-fold), compared to the condition of 2.4 mmol L −1 h −1. At the highest OTR condition, a decrease in the NADPH/NADP + ratio of threefold was observed, probably as a response to the high respiration rate induced by the respiratory protection of the nitrogenase under diazotrophic conditions, correlating with a high expression of the uncoupled respiratory chain genes (ndhII and cydA) and induction of the expression of the genes encoding the nitrogenase complex (nifH). We have demonstrated that changes in oxygen availability affect the internal redox state of the cell and carbon metabolic fluxes. This also has a strong impact on the TCA cycle and PP pathway as well as on alginate and P3HB biosynthetic fluxes
Management of hypersensivity pneumonitis
- Author
- A Ampere
- A Churg
- A Cui
- A Kruit
- A Pardo
- A Thornburg
- A Zarbock
- AD Joshi
- AM Patel
- AR Tonelli
- CA Dinarello
- CA Saltoun
- CI Silva
- CM Fenoglio
- CR Cordeiro
- D Koschel
- D Koschel
- D Reghellin
- DE León
- DJ Fong
- DM Caillaud
- DS Koschel
- E Israel-Assayag
- E Israel-Assayag
- E Kupeli
- E Lerch
- E Uzaslan
- F Buchvald
- F Casilli
- F Morell
- F Morell
- G Gudmundsson
- G Reboux
- G Reboux
- G Rhodes
- G Roshnee
- GY Chew
- H Gbaguidi-Haore
- H Otera
- HA Abdelsamed
- HJ Park
- I Tillie-Leblond
- J Kuramochi
- J Kuramochi
- J Schäfer
- J Sennekamp
- JE Losa García
- JI Kokkarinen
- JJ McDevitt
- JL Myers
- JN Fink
- JN Fink
- K Mitaka
- K Unoura
- K Usui
- L Barrera
- L Curtis
- L Howard
- L Jiménez-Alvarez
- LP Hariri
- M Drent
- M Girard
- M Girard
- M Heron
- M Miyajima
- M Sterclova
- MJ Rodrigo
- ML Metersky
- MR Blanchet
- N Inase
- N Inase
- N Inase
- N Inase
- N Ishikawa
- N Martin
- PJ Bouic
- PJ Hansen
- PL Simonian
- PL Simonian
- PL Simonian
- PM Roelofs
- Q Ye
- Q Ye
- R Erkinjuntti-Pekkanen
- R Toribio
- RM Strieter
- S Alexandru
- S Nakae
- S Roussel
- S Roussel
- SJ Hwang
- SL Walsh
- SP Reynolds
- SZ Ben-Sasson
- T Akashi
- T Arikawa
- T Jinta
- T Okamoto
- T Shirai
- T Takao
- T Tateishi
- TJ Craig
- U Bhan
- U Bhan
- U Costabel
- U Greinert
- VL Capelozzi
- Y Cormier
- Y Cormier
- Y Lacasse
- Y Miyazaki
- Y Ohtani
- Y Ohtani
- Y Park
- Y Yoshizawa
- Y Yoshizawa
- Z Tong
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Association of genomic domains in BRCA1 and BRCA2 with prostate cancer risk and aggressiveness
- Author
- Adlard J
- Agata S
- Agnarsson BA
- Ahmed M
- Aittomäki K
- Alducci E
- Andrulis IL
- Antoniou AC
- Arason A
- Arnold N
- Artioli G
- Arver B
- Auber B
- Azzollini J
- Balmaña J
- Barkardottir RB
- Barnes DR
- Barroso A
- Barrowdale D
- Belotti M
- Benitez J
- Bertelsen B
- Blok MJ
- Bodrogi I
- Bonadona V
- Bonanni B
- Bondavalli D
- Boonen SE
- Borde J
- Borg A
- Bradbury AR
- Brady A
- Brewer C
- Brunet J
- Buecher B
- Busch EL
- Buys SS
- Cabezas-Camarero S
- Caldés T
- Caliebe A
- Caligo MA
- Calvello M
- Campbell IG
- Carnevali I
- Carrasco E
- Chan TL
- Chenevix-Trench G
- Chu ATW
- Chung WK
- Claes KBM
- Collaborators E
- Collaborators GS
- Cook J
- Cortesi L
- Couch FJ
- Cronin A
- D'Amico AV
- Daly MB
- Damante G
- Darder E
- Davidson R
- de la Hoya M
- Dennis J
- Ding YC
- Ditsch N
- Domchek SM
- Donaldson A
- Dworniczak B
- Díez O
- Easton DF
- Eccles DM
- Eeles RA
- Ehrencrona H
- Ejlertsen B
- Engel C
- Evans DG
- Faivre L
- Faust U
- Feliubadaló L
- Foretova L
- Fostira F
- Fountzilas G
- Freedman ML
- Friebel TM
- Frost D
- García-Barberán V
- Garre P
- Gauthier-Villars M
- Gehrig A
- Gerdes A-M
- Gesta P
- Giannini G
- Glendon G
- Godwin AK
- Goldgar DE
- Gomes DM
- Greene MH
- Gutierrez-Barrera AM
- Géczi L
- Hahnen E
- Hamann U
- Hauke J
- Herold N
- Hogervorst FBL
- Honisch E
- Hopper JL
- Hulick PJ
- Investigators H
- Investigators K
- Izatt L
- Jager A
- James P
- Janavicius R
- Jensen TD
- Jensen UB
- Johannsson OT
- John EM
- Joseph V
- Kang E
- Kast K
- Kiiski JI
- Kim S-W
- Kim Z
- Ko K-P
- Konstantopoulou I
- Kramer G
- Krogh L
- Kruse TA
- Kwong A
- Larsen M
- Lasset C
- Lautrup C
- Lazaro C
- Lee J
- Lee JW
- Lee MH
- Lemke J
- Leslie G
- Lesueur F
- Liljegren A
- Lindblom A
- Llovet P
- Lopez-Fernández A
- Lopez-Perolio I
- Lorca V
- Loud JT
- Ma ESK
- Mai PL
- Manoukian S
- Mari V
- Martin L
- Matricardi L
- McGuffog L
- Mebirouk N
- Medici V
- Meijers-Heijboer HEJ
- Meindl A
- Mensenkamp AR
- Miller C
- Montagna M
- Mooij TM
- Moserle L
- Mouret-Fourme E
- Mulligan AM
- Nathanson KL
- Navratilova M
- Neuhausen SL
- Nevanlinna H
- Niederacher D
- Nielsen FCC
- Nielsen HR
- Nikitina-Zake L
- Offit K
- Olah E
- Olopade OI
- Ong K-R
- Osorio A
- Ott C-E
- Ottini L
- Palli D
- Park SK
- Parsons MT
- Patel VL
- Pedersen IS
- Peissel B
- Peixoto A
- Peterlongo P
- Petersen AH
- Pomerantz MM
- Porteous ME
- Pujana MA
- Puppa LD
- Pérez-Segura P
- Radice P
- Ramser J
- Rantala J
- Rashid MU
- Rebbeck TR
- Rhiem K
- Rizzolo P
- Robson ME
- Rookus MA
- Rossing CM
- Ruddy KJ
- Santos C
- Saule C
- Scarpitta R
- Schmutzler RK
- Schuster H
- Senter L
- Seynaeve CM
- Shah PD
- Sharma P
- Shin VY
- Silvestri V
- Simard J
- Singer CF
- Skytte A-B
- Snape K
- Solano AR
- Soucy P
- Southey MC
- Spurdle AB
- Steele L
- Steinemann D
- Stoppa-Lyonnet D
- Stradella A
- Sunde L
- Sutter C
- Tan YY
- Teixeira MR
- Teo SH
- Thomassen M
- Tibiletti MG
- Tischkowitz M
- Tognazzo S
- Toland AE
- Tommasi S
- Torres D
- Toss A
- Trainer AH
- Tung N
- van Asperen CJ
- van der Baan FH
- van der Kolk LE
- van der Luijt RB
- van Hest LP
- Varesco L
- Varon-Mateeva R
- Viel A
- Vierstrate J
- Villa R
- von Wachenfeldt A
- Wagner P
- Wang-Gohrke S
- Wappenschmidt B
- Weitzel JN
- Wieme G
- Yadav S
- Yannoukakos D
- Yoon S-Y
- Zanzottera C
- Zorn KK
- Publication venue
- Publication date
- 01/01/2019
- Field of study
Pathogenic sequence variants (PSV) in BRCA1 or BRCA2 (BRCA1/2) are associated with increased risk and severity of prostate cancer. Weevaluated whether PSVs inBRCA1/2 were associated with risk of overall prostate cancer or high grade (Gleason 8+) prostate cancer using an international sample of 65 BRCA1 and 171 BRCA2 male PSV carriers with prostate cancer, and 3,388 BRCA1 and 2,880 BRCA2 male PSV carriers without prostate cancer. PSVs in the 30 region of BRCA2 (c.7914+) were significantly associated with elevated risk of prostate cancer compared with reference bin c.1001c.7913 [HR = 1.78; 95% confidence interval (CI), 1.25-2.52; P = 0.001], as well as elevated risk of Gleason 8+ prostate cancer (HR = 3.11; 95% CI, 1.63-5.95; P = 0.001). c.756-c.1000 was also associated with elevated prostate cancer risk (HR = 2.83; 95% CI, 1.71-4.68; P = 0.00004) and elevated risk of Gleason 8+prostate cancer (HR = 4.95; 95% CI, 2.12-11.54; P = 0.0002). No genotype-phenotype associations were detected for PSVs in BRCA1. These results demonstrate that specific BRCA2 PSVs may be associated with elevated risk of developing aggressive prostate cancer. Significance: Aggressive prostate cancer risk in BRCA2 mutation carriers may vary according to the specific BRCA2 mutation inherited by the at-risk individual.Peer reviewe
Pan-cancer analysis of whole genomes
- Author
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- Jamieson
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- The ICGC/TCGA Pan-Cancer Analysis of Whole Genomes Consortium View Correspondence (jump link)
- Thiessen
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- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
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- Abdel-Aziz AK
- Abdelfatah S
- Abdellatif M
- Abdoli A
- Abel S
- Abeliovich H
- Abildgaard MH
- Abudu YP
- Acevedo-Arozena A
- Adamopoulos IE
- Adeli K
- Adolph TE
- Adornetto A
- Aflaki E
- Agam G
- Agarwal A
- Aggarwal BB
- Agnello M
- Agostinis P
- Agrewala JN
- Agrotis A
- Aguilar PV
- Ahmad ST
- Ahmed ZM
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- Aits S
- Aizawa S
- Akkoc Y
- Akoumianaki T
- Akpinar HA
- Al-Abd AM
- Al-Akra L
- Al-Gharaibeh A
- Alaoui-Jamali MA
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- Alessandri C
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- Alizadeh J
- Almacellas E
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- Alonso GD
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- Álvarez ÉMC
- Ávalos Y
- Önal G
- Üstün S
- Čolić M
- Đokić J
- Žerovnik E
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field
Performance of the ATLAS muon trigger in pp collisions at [Formula: see text] TeV
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- Abdallah J
- Abdel Khalek S
- Abdinov O
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- Wildauer A
- Wildt MA
- Wilkens HG
- Will JZ
- Williams HH
- Williams S
- Willis C
- Willocq S
- Wilson A
- Wilson JA
- Wingerter-Seez I
- Winklmeier F
- Winter BT
- Wittgen M
- Wittig T
- Wittkowski J
- Wollstadt SJ
- Wolter MW
- Wolters H
- Wosiek BK
- Wotschack J
- Woudstra MJ
- Wozniak KW
- Wright M
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- Wu SL
- Wu X
- Wu Y
- Wulf E
- Wyatt TR
- Wynne BM
- Xella S
- Xiao M
- Xu D
- Xu L
- Yabsley B
- Yacoob S
- Yakabe R
- Yamada M
- Yamaguchi H
- Yamaguchi Y
- Yamamoto A
- Yamamoto K
- Yamamoto S
- Yamamura T
- Yamanaka T
- Yamauchi K
- Yamazaki Y
- Yan Z
- Yang H
- Yang H
- Yang UK
- Yang Y
- Yanush S
- Yao L
- Yao WM
- Yasu Y
- Yatsenko E
- Yau Wong KH
- Ye J
- Ye S
- Yeletskikh I
- Yen AL
- Yildirim E
- Yilmaz M
- Yoosoofmiya R
- Yorita K
- Yoshida R
- Yoshihara K
- Young C
- Young CJ
- Youssef S
- Yu DR
- Yu J
- Yu J
- Yu JM
- Yuan L
- Yurkewicz A
- Yusuff I
- Zabinski B
- Zaidan R
- Zaitsev AM
- Zaman A
- Zambito S
- Zanello L
- Zanzi D
- Zeitnitz C
- Zeman M
- Zemla A
- Zengel K
- Zenin O
- Zerwas D
- Zevi Della Porta G
- Zhang D
- Zhang F
- Zhang H
- Zhang J
- Zhang L
- Zhang X
- Zhang Z
- Zhao Z
- Zhemchugov A
- Zhong J
- Zhou B
- Zhou L
- Zhou N
- Zhu CG
- Zhu H
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann C
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zobernig G
- Zoccoli A
- Zur Nedden M
- Zurzolo G
- Zutshi V
- Zwalinski L
- Åkesson TP
- Åsman B
- Ženiš T
- Publication venue
- Publication date
- 01/03/2015
- Field of study
The performance of the ATLAS muon trigger system is evaluated with proton-proton collision data collected in 2012 at the Large Hadron Collider at a centre-of-mass energy of 8 TeV. It is primarily evaluated using events containing a pair of muons from the decay of [Formula: see text] bosons. The efficiency of the single-muon trigger is measured for muons with transverse momentum [Formula: see text] GeV, with a statistical uncertainty of less than 0.01 % and a systematic uncertainty of 0.6 %. The [Formula: see text] range for efficiency determination is extended by using muons from decays of [Formula: see text] mesons, [Formula: see text] bosons, and top quarks. The muon trigger shows highly uniform and stable performance. The performance is compared to the prediction of a detailed simulation
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