90 research outputs found
Effects of FTY720 on brain neurogenic niches in vitro and after kainic acid-induced injury
- Author
- A Sierra
- A Yazdi
- Alfredo RodrĂguez-AntigĂŒedad
- AM Stessin
- B Hattiangady
- B Hattiangady
- B Hattiangady
- B Tan
- B Waly El
- B Winner
- BL Murphy
- C Matute
- C Zhao
- CA Blanc
- Carlos Matute
- CG Jung
- CT Ekdahl
- E Bieberich
- F Cavaliere
- F Gao
- FH Gage
- FS Hoffmann
- G Anderson
- G Paxinos
- I Kondratiuk
- J Mitchell
- J Zhang
- JE Carpentino
- JE Kralic
- JM Parent
- JM Parent
- Juan Carlos Chara
- JW Choi
- KJ Hopkins
- L Harris
- L Menna Di
- L Nuzzo di
- LA Shapiro
- M Benito-Muñoz
- M LĂ©vesque
- M Zawadzka
- ML Monje
- MS Rao
- NC Hait
- P Efstathopoulos
- Q Wang
- QL Cui
- R Cipriani
- R Deogracias
- R Kuruba
- Raffaela Cipriani
- RJM Franklin
- RP Coelho
- S Jessberger
- T Ravizza
- V Brinkmann
- V Brinkmann
- VE Miron
- VE Miron
- VE Miron
- WD Richardson
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Patient safety and the prevention of skin and mucosal lesions associated with airway invasive devices
- Author
- BrĂŒgemann OM
- CastellÔes TMFW
- Coelho MS
- Deisy Mello de Pinto
- Dornelles C
- Estela dos Santos Schons
- Jerre G
- Josefine Busanello
- Martins I
- Martins RHG
- Minayo MCS
- Mota LAA
- Muñoz VE
- Nascimento CCP
- Nolan JP
- Orlandini GM
- Paiva MCMS
- Peña ELC
- Ricz HMA
- Servin SON
- Shi Z
- Siela D
- Silveira LSS
- Stanzani VL
- Tanios MA
- Valdecir Zavarese da Costa
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
Pathogenic Huntingtin Repeat Expansions in Patients with Frontotemporal Dementia and Amyotrophic Lateral Sclerosis.
- Author
- Aarsland D
- Abramzon Y
- Adams DJ
- Adeleye A
- Aguilar M
- Ahmed S
- Al-Chalabi A
- Al-Sarraj S
- Al-Sarraj S
- Alba C
- Alba C
- Albani D
- Albert MS
- Altschuler S
- Alvarez I
- Amar K
- Ambrose JC
- Archibald N
- Arcila-Londono X
- Arepalli S
- Aronica E
- Arumugam P
- Asress S
- Atassi N
- Attems J
- Auluck P
- Baas F
- Baas F
- Babu S
- Bacikova D
- Bacikova D
- Baloh RH
- Baloh RH
- Bandmann O
- Baple EL
- Barrett MJ
- Beach TG
- Bekris LM
- Bennett DA
- Benussi L
- Berry JD
- Bertolin C
- Besser LM
- Bigio EH
- Binetti G
- Black SE
- Blair IP
- Bleda M
- Boada M
- Boardman-Pretty F
- Boeve BF
- Bohannan RC
- Boissiere JM
- Borroni B
- Boustred CR
- Bowser R
- Bowser R
- Boylan KB
- Brady CB
- Brett F
- Brice A
- Brice A
- Brittain H
- Broach J
- Broach JR
- Broce I
- Brown RH
- Brunetti M
- Bruni A
- Butovsky O
- Calini D
- Calvo A
- Camu W
- Canosa A
- Capps E
- Caraway CA
- Caroppo P
- Castellotti B
- Caulfield MJ
- Cereda C
- Ceroni M
- Chan GC
- Chandran S
- Chia R
- Chiang H-H
- ChiĂČ A
- ChiĂČ A
- Church A
- Clarimon J
- Clarimon J
- Coebergh J
- Colombrita C
- Comi GP
- Cooper-Knock J
- Corcia P
- Corrado L
- Corti S
- Costantini A
- Cox GA
- Craig CEH
- Crary J
- Critchley P
- Dalgard CL
- Danek A
- Danielson EW
- Dardiotis E
- Daugherty LC
- De Belleroche J
- De Burca A
- Del Bo R
- Devereau A
- Dewan R
- Dickson D
- Diehl-Schimd J
- Diekstra FP
- Diez-Fairen M
- Diez-Fairen M
- Ding J
- Dols-Icardo O
- Drepper C
- Drory V
- Drory VE
- Dubnau J
- Duga S
- Dunckley TL
- Duyckaerts C
- DâAlfonso S
- Elgar G
- Esteban-PĂ©rez J
- Faber K
- Faghri F
- Fallini C
- Farren J
- Feldman E
- Ferman T
- Ferrari R
- Ferrucci L
- Finkbeiner S
- Flanagan ME
- Floeter MK
- Floris G
- Foroud TM
- Fortea J
- Foulger RE
- Fowler T
- Fraenkel E
- Fratta P
- Fratta P
- FuriĂł-TarĂ P
- Gagliardi S
- Galimberti D
- Gan-Or Z
- GarcĂa-Redondo A
- Gellera C
- Gentleman S
- Gerhard G
- Ghetti B
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- Gibbs JR
- Gibbs JR
- Gibson SB
- Gkazi AS
- Glass JD
- Goate A
- Goldstein D
- GonzĂĄlez-Aramburu I
- Gotkine M
- Goutman SA
- Graff C
- Graff-Radford NR
- Hackett JM
- Halai D
- Hamblin A
- Hammell MG
- Hardiman O
- Hardy JA
- Harms M
- Harms MB
- Harris B
- Heiman-Patterson T
- Heiman-Patterson TD
- Henderson S
- Hernandez DG
- HernĂĄndez I
- Hickman RA
- Hjermind LE
- Hodges AK
- Holman JE
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- Hu H-C
- Hu MTM
- Hubbard TJP
- Hupalo D
- Hupalo DN
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- IllĂĄn-Gala I
- Infante J
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- Jackson R
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- Kaufmann H
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- Klein G
- Kobylecki C
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- Kowal N
- Kowall NW
- KrĂŒger R
- Kukull W
- Kuzma A
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- Laaksovirta H
- Lage C
- Lahnstein L
- Landers JE
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- Rothstein JD
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- Troncoso JC
- Troncoso JC
- Tucci A
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- Turner MR
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- Van Damme P
- Van Deerlin V
- Van den Berg LH
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- Vonsattel JP
- Waldo ML
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- Wilkerson MD
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- Woltjer R
- Wood SM
- Wszolek ZK
- Wu L
- Wu Z
- Xiromerisiou G
- Zaitlen N
- Zarowiecki M
- Zecca C
- Zhang B
- Zhang X
- Zinman L
- Ăijerstedt L
- Publication venue
- Neuron
- Publication date
- 01/01/2020
- Field of study
We examined the role of repeat expansions in the pathogenesis of frontotemporal dementia (FTD) and amyotrophic lateral sclerosis (ALS) by analyzing whole-genome sequence data from 2,442 FTD/ALS patients, 2,599 Lewy body dementia (LBD) patients, and 3,158 neurologically healthy subjects. Pathogenic expansions (range, 40-64 CAG repeats) in the huntingtin (HTT) gene were found in three (0.12%) patients diagnosed with pure FTD/ALS syndromes but were not present in the LBD or healthy cohorts. We replicated our findings in an independent collection of 3,674 FTD/ALS patients. Postmortem evaluations of two patients revealed the classical TDP-43 pathology of FTD/ALS, as well as huntingtin-positive, ubiquitin-positive aggregates in the frontal cortex. The neostriatal atrophy that pathologically defines Huntington's disease was absent in both cases. Our findings reveal an etiological relationship between HTT repeat expansions and FTD/ALS syndromes and indicate that genetic screening of FTD/ALS patients for HTT repeat expansions should be considered
Antiinflammatory Therapy with Canakinumab for Atherosclerotic Disease
- Author
- A Abbate
- A Abhyankar
- A Adams
- A Agafina
- A Akyea-Djamson
- A Alan
- A Alfieri
- A Alfrey
- A Alvarisqueta
- A Amos
- A Anadiotis
- A Anneveldt
- A Antolick
- A Arthur
- A Aslam
- Abaci F
- Abdullakutty J
- Abhaichand R
- Abib E Jr
- Aboufakher R
- Abrantes J
- Abreu M
- Abulencia K
- Acampora D
- Acampora M
- Accini Mendoza Jl
- Aceto L
- Acevedo B
- Acevedo L
- Acheatel R
- Actis Mv
- Adams M
- Adjic Nc
- Affaki Gs
- Agarwal Dk
- Aggarwal Rk
- Agosta Gf
- Aguilar M
- Aguilar M
- Ahire N
- Ahmad I
- Ahmed Sh
- Ahn Y
- Aish B
- Aiub F
- Aiub J
- Ajax K
- Ajioka M
- Akai Y
- Akatova E
- Akrap B
- Al Joundi T
- Al-Khalili F
- Albisu J
- Alcalde Jm
- Alcide P
- Alfonso T
- Allen J
- Alllison Dc
- Almaliky T
- Amerena J
- Andersen K
- Andor M
- Angelova I
- Angiolillo D
- Anita S
- Anker Sd
- Antalik L
- Antonicelli R
- Aparicio Cv
- Apel T
- Apostolovic S
- Ara M
- Aragorn L
- Arango Jl
- Araujo Fonseca M
- Ardissino D
- Arenas Leon Jl
- Arevalo S
- Argiolas G
- Arif I
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- Ashcom T
- Ashford M
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- Asim Oktay Ao
- Assarsson E
- Ata B
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- Avalos V
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- Azizad M
- Azize Gm
- B Becker
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- Zielinski M
- Zieve F
- Zineldine A.
- Zirlik A
- Zucchetti C
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2017
- Field of study
Background: Experimental and clinical data suggest that reducing inflammation without affecting lipid levels may reduce the risk of cardiovascular disease. Yet, the inflammatory hypothesis of atherothrombosis has remained unproved. Methods: We conducted a randomized, double-blind trial of canakinumab, a therapeutic monoclonal antibody targeting interleukin-1ÎČ, involving 10,061 patients with previous myocardial infarction and a high-sensitivity C-reactive protein level of 2 mg or more per liter. The trial compared three doses of canakinumab (50 mg, 150 mg, and 300 mg, administered subcutaneously every 3 months) with placebo. The primary efficacy end point was nonfatal myocardial infarction, nonfatal stroke, or cardiovascular death. RESULTS: At 48 months, the median reduction from baseline in the high-sensitivity C-reactive protein level was 26 percentage points greater in the group that received the 50-mg dose of canakinumab, 37 percentage points greater in the 150-mg group, and 41 percentage points greater in the 300-mg group than in the placebo group. Canakinumab did not reduce lipid levels from baseline. At a median follow-up of 3.7 years, the incidence rate for the primary end point was 4.50 events per 100 person-years in the placebo group, 4.11 events per 100 person-years in the 50-mg group, 3.86 events per 100 person-years in the 150-mg group, and 3.90 events per 100 person-years in the 300-mg group. The hazard ratios as compared with placebo were as follows: in the 50-mg group, 0.93 (95% confidence interval [CI], 0.80 to 1.07; P = 0.30); in the 150-mg group, 0.85 (95% CI, 0.74 to 0.98; P = 0.021); and in the 300-mg group, 0.86 (95% CI, 0.75 to 0.99; P = 0.031). The 150-mg dose, but not the other doses, met the prespecified multiplicity-adjusted threshold for statistical significance for the primary end point and the secondary end point that additionally included hospitalization for unstable angina that led to urgent revascularization (hazard ratio vs. placebo, 0.83; 95% CI, 0.73 to 0.95; P = 0.005). Canakinumab was associated with a higher incidence of fatal infection than was placebo. There was no significant difference in all-cause mortality (hazard ratio for all canakinumab doses vs. placebo, 0.94; 95% CI, 0.83 to 1.06; P = 0.31). Conclusions: Antiinflammatory therapy targeting the interleukin-1ÎČ innate immunity pathway with canakinumab at a dose of 150 mg every 3 months led to a significantly lower rate of recurrent cardiovascular events than placebo, independent of lipid-level lowering. (Funded by Novartis; CANTOS ClinicalTrials.gov number, NCT01327846.
Pan-cancer analysis of whole genomes
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- The ICGC/TCGA Pan-Cancer Analysis of Whole Genomes Consortium View Correspondence (jump link)
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- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018.
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- Y Fuchs
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- Y Ito
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- Y Li
- Y Li
- Y Liu
- Y Liu
- Y Luo
- Y Ma
- Y Ma
- Y Morizane
- Y Ou
- Y Pang
- Y Rong
- Y Shlyakhtina
- Y Subburaj
- Y Suzuki
- Y Tsujimoto
- Y Wang
- Y Wang
- Y Wang
- Y Wang
- Y Xie
- Y Xie
- Y Xie
- Y Zaltsman
- Y Zermati
- Y Zhang
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- Y Zhu
- Y Zhu
- YB Chen
- YC Hou
- YH Liao
- YI Nakajima
- Ying Wang
- YN Gong
- Yoshihide Tsujimoto
- YS Cho
- Yufang Shi
- Z Cai
- Z Cai
- Z Wang
- Z Wang
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- Z You
- Z Zhang
- Z Zhong
- Zahra Zakeri
- ZG Liu
- ZN Oltvai
- ZT Schafer
- ZX Chen
- ZX Chen
- Publication venue
- Cell Death Differ
- Publication date
- 01/01/2018
- Field of study
Over the past decade, the Nomenclature Committee on Cell Death (NCCD) has formulated guidelines for the definition and interpretation of cell death from morphological, biochemical, and functional perspectives. Since the field continues to expand and novel mechanisms that orchestrate multiple cell death pathways are unveiled, we propose an updated classification of cell death subroutines focusing on mechanistic and essential (as opposed to correlative and dispensable) aspects of the process. As we provide molecularly oriented definitions of terms including intrinsic apoptosis, extrinsic apoptosis, mitochondrial permeability transition (MPT)-driven necrosis, necroptosis, ferroptosis, pyroptosis, parthanatos, entotic cell death, NETotic cell death, lysosome-dependent cell death, autophagy-dependent cell death, immunogenic cell death, cellular senescence, and mitotic catastrophe, we discuss the utility of neologisms that refer to highly specialized instances of these processes. The mission of the NCCD is to provide a widely accepted nomenclature on cell death in support of the continued development of the field
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
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- Zimmermann CM
- Ziviani E
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- Zorzano A
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- Zwerschke W
- Ălvarez ĂMC
- Ăvalos Y
- Ănal G
- ĂstĂŒn S
- ÄoliÄ M
- ÄokiÄ J
- Ćœerovnik E
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field
Genetic signatures of adaptation revealed from transcriptome sequencing of Arctic and red foxes
- Author
- A Conesa
- A Dobin
- A Drummond
- A Löytynoja
- A McKenna
- A Mercure
- A Muñoz-Mérida
- A Roberts
- A Sboner
- A Schneider
- AD Foote
- AM Audet
- AR Saltiel
- AV Kukekova
- Axel Janke
- B Langmead
- B Yngvadottir
- BM Hallström
- C Camacho
- C Hidai
- CE Valdiosera
- DC Koboldt
- DL Stern
- E Geffen
- EC Swindell
- F Delsuc
- F Hailer
- FA Perini
- G Hewitt
- G Jobb
- G Jordan
- H Li
- H Li
- H-X Cui
- HO Clark
- I Seim
- I Shagina
- I Stirling
- J Abella
- J Crouse
- J Sambrook
- J Zhang
- K Katoh
- K Lindblad-Toh
- K Penta
- L Dalén
- L Taher
- M Benton
- M Clement
- M Enari
- M Evert
- M Gouy
- M Kullberg
- M Lechner
- Maria A. Nilsson
- MG Grabherr
- MJ Statham
- MM Mulvihill
- N Gruenheit
- N Zambrano
- P Chomczynski
- P Danecek
- P Librado
- P Prestrud
- P Prestrud
- P Rice
- PG Engström
- Q-Y Zhao
- R Kofler
- S Chanprasert
- S LariviĂšre
- S Liu
- S Rozen
- T Lassmann
- T SchĂŒrpf
- TA Kupriyanova
- TI Shaw
- VE Kutschera
- Verena E. Kutschera
- Vikas Kumar
- W Li
- WC Wozencraft
- WH Gharib
- WJ Kent
- X Liu
- X Wang
- Y Mitsuuchi
- Y Zheng
- YJ Crow
- Z Yang
- Z Yang
- Z Yang
- Z Yang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Skeletal muscle wasting and renewal: a pivotal role of myokine IL-6
- Author
- A Bonetto
- A Chalaris
- A Ciechanover
- A Cuenda
- A Inui
- A Laine
- A Oliff
- A Steensberg
- AA Narsale
- AC McPherron
- AL Goldberg
- AP Russell
- AS Brack
- B Egan
- B Gayraud-Morel
- B Sangiuliano
- BH Tan
- BH Tan
- BK Pedersen
- BK Pedersen
- BK Pedersen
- BK Pedersen
- BK Pedersen
- C Mammucari
- C Pop
- C Weigert
- CA Hunter
- CM Ardies
- CM Pickart
- D Attaix
- D Bechet
- D Elia
- D Graf
- D Zhang
- DD Cornelison
- DJ Glass
- DJ Glass
- DM Jablons
- DV Do
- E Lach-Trifilieff
- F Haddad
- F Martinon
- F Relaix
- FB Benatti
- G Hall van
- G Skiniotis
- G Straface
- GJ Inman
- H Rubin
- H Yin
- HD Kollias
- HM Blau
- J Du
- J Fujita
- J Mullberg
- J Scheller
- J Schellera
- J Takeuchi
- J Wolf
- J Zhao
- JA Hawley
- JD Bernet
- JE Belizario
- JE Belizario
- JE BelizĂĄrio
- JG Cannon
- JM Argiles
- JM Argiles
- JM Argiles
- JM Argiles
- JM Sacheck
- K Ando
- K Fearon
- K Matsushita
- KA Baltgalvis
- KA Baltgalvis
- KAS Silva
- KC Fearon
- KC Fearon
- KF Kusano
- KG Toth
- KJ Boonen
- KJ Kallen
- KJ Savage
- KK Takeda
- KS Kwak
- L Duprez
- LA Megeney
- LM Baehr
- M Cantini
- M Muscaritoli
- M Pal
- M Pelosi
- M Sandri
- M Sandri
- M Sandri
- M Sandri
- M Seelaender
- M Shibata
- MA Febbraio
- MA Luz
- MD Gomes
- ME Carlson
- MH Parker
- MH Zaki
- MJ Tisdale
- MJ Tisdale
- MT Tierney
- N Fasnacht
- N Motorashi
- NA Riobo
- NC Chang
- NE Brooks
- O Halevy
- P Bonaldo
- P Brien
- P Muñoz-Cånoves
- P Seale
- P Seale
- PC Heinrich
- PS Zammit
- R Bassel-Duby
- R Holmer
- RJ Bryson-Richardson
- S Cohen
- S DaCosta Byfield
- S Lokireddy
- S Schiaffino
- S Shaw
- S Zvonic
- S-J Lee
- SC Bodine
- SC Kandarian
- SC Koeberle
- SD Gopinath
- SH Lecker
- SH Lecker
- SJ Lee
- SM Judge
- T Centner
- T Hirano
- T Lahiri
- T Tsujinaka
- T Tsujinaka
- VE Baracos
- VE Baracos
- W Zhou
- WJ Evans
- X Shi
- X Zhou
- XH Wang
- Y Fong
- Z Yablonka-Reuveni
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
O risco das aminas biogĂȘnicas nos alimentos
- Author
- Abriouel H
- Almeida C
- An H
- AncĂn-Azpilicueta C
- BardĂłcz S
- Björnsdóttir-Butler K
- Bodmer S
- Bover-Cid S
- Bover-Cid S
- Bover-Cid S
- Bruna Amatto Duarte Pires
- Caccioppoli I
- Casella IG
- Coton E
- Coton E
- Cotter PD
- Curiel JA
- De las Rivas B
- De las Rivas B
- Del Prete V
- DelbĂšs-Pausa C
- Duflos G
- Eerola S
- Erkan N
- Fadhlaoui-Zida K
- FernĂĄndez M
- FernĂĄndez M
- FernĂĄndez M
- FernĂĄndez M
- FernĂĄndez-GarcĂa EJT
- Ferreira IM
- Galgano F
- Galgano F
- Garai G
- GarcĂa-Ruiz A
- GlĂłria MBA
- Gomaa MN
- Gonzaga VE
- Greif G
- HalĂĄsz A
- Hernandez-Jover T
- Hwang CC
- Innocente N
- Izquierdo-Pulido M
- Kalac P
- Kanki M
- Kanki M
- KaroviĂšova J
- Kim MK
- Kim SH
- Ladero V
- Ladero VM
- Lakshmanan R
- Landete JM
- Landete JM
- Landete JM
- Laranjeira R
- Larpin-Laborde S
- Lavizzari T
- LeitĂŁo MFF
- Lonvaud-Funel A
- Lucas P
- Lucas PM
- Maijala RL
- Marcobal A
- Marcobal A
- Marcobal A
- Marcobal A
- Marcobal A
- MartĂnez-Villaluenga C
- Matilla AJ
- Mohan CO
- Moreno-Arribas V
- Moret S
- Muñoz-Atienza E
- Myrna Barbosa Gomes
- Naila A
- Nannelli F
- Novella-RodrĂguez S
- Parente E
- Polo L
- Rezaei M
- Roig-Sagués AX
- Russo P
- Sarkadi LS
- Schneider I
- Shalaby AR
- Shalaby AR
- Silla Santos MH
- Silva CMG
- Singh VP
- Slomkowska A
- Smit AY
- Smit AY
- Sohrabvandi S
- Spano G
- Stratton JE
- Straub BW
- Suely Aparecida Pimenta Fracalanzza
- Sumner SS
- Suzzi G
- Tapingkae W
- Ten Brink B
- Til HP
- Triki M
- Tsai YH
- Vale SR
- Valsamaki K
- Victor Augustus Marin
- Wennberg M
- Yongmei L
- Ăzogul F
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
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