139 research outputs found
Prenatal Immune Challenge Is an Environmental Risk Factor for Brain and Behavior Change Relevant to Schizophrenia: Evidence from MRI in a Mouse Model
- Author
- AE Dorr
- AE Rehn
- AJ Bailey
- AM Samuelsson
- AS Brown
- AS Brown
- AS Brown
- AS Brown
- AS Brown
- B Clancy
- B Zhao
- BG Bunney
- BI Turetsky
- C Beltinger
- CD Good
- Charlton Cheung
- DL Braff
- E Chan
- E O'Callaghan
- E Romero
- EA Hazlett
- Ed X. Wu
- Edward S. Hui
- EM Ornitz
- F Limosin
- FV Rijsdijk
- G Torres
- GM McAlonan
- GM McAlonan
- Grainne M. McAlonan
- H Beckmann
- H Beydoun
- HJ Sorensen
- I Ellison-Wright
- I Kostovic
- J Ashburner
- J Borrell
- J Sanjuan
- J Suvisaari
- JH Gilmore
- JL Rausch
- Joram Feldon
- JS Craig
- K Japha
- K Lancaster
- K Ozawa
- K Pierce
- L Shi
- L Shi
- L Zuckerman
- LR Dice
- M Ghaziuddin
- M Hornig
- M Hornig
- MA Hayat
- MJ Hoptman
- ML Bauman
- ML Bauman
- ML Esterberg
- NR Swerdlow
- NR Swerdlow
- OT Wolf
- OT Wolf
- Pak C. Sham
- PC Heinrich
- PC Nopoulos
- PC Sham
- PH Patterson
- Qi Li
- R Galani
- RA Carper
- Ran Wei
- RP Kesner
- S Chess
- S Chess
- S Pietropaolo
- SA Chance
- SA Mednick
- SE Chua
- SE Chua
- SE Chua
- SE Chua
- SE Chua
- SE Smith
- SH Fatemi
- SH Fatemi
- SH Fatemi
- SH Fatemi
- Siew E. Chua
- SJ Sawiak
- SM Smith
- Sookja Chung
- TD Cannon
- Thomas Burne
- TR Traynor
- U Meyer
- U Meyer
- U Meyer
- U Meyer
- U Meyer
- U Meyer
- U Meyer
- Urs Meyer
- V Babulas
- V Cheung
- V Kumari
- W Perry
- WG Staal
- WG Staal
- WI Lipkin
- X Gong
- Y Watanabe
- Y Yamashita
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2009
- Field of study
Objectives: Maternal infection during pregnancy increases risk of severe neuropsychiatric disorders, including schizophrenia and autism, in the offspring. The most consistent brain structural abnormality in patients with schizophrenia is enlarged lateral ventricles. However, it is unknown whether the aetiology of ventriculomegaly in schizophrenia involves prenatal infectious processes. The present experiments tested the hypothesis that there is a causal relationship between prenatal immune challenge and emergence of ventricular abnormalities relevant to schizophrenia in adulthood. Method: We used an established mouse model of maternal immune activation (MIA) by the viral mimic Polyl:C administered in early (day 9) or late (day 17) gestation. Automated voxel-based morphometry mapped cerebrospinal fluid across the whole brain of adult offspring and the results were validated by manual region-of-interest tracing of the lateral ventricles. Parallel behavioral testing determined the existence of schizophrenia-related sensorimotor gating abnormalities. Results: Polyl:C-induced immune activation, in early but not late gestation, caused marked enlargement of lateral ventricles in adulthood, without affecting total white and grey matter volumes. This early exposure disrupted sensorimotor gating, in the form of prepulse inhibition. Identical immune challenge in late gestation resulted in significant expansion of 4th ventricle volume but did not disrupt sensorimotor gating. Conclusions: Our results provide the first experimental evidence that prenatal immune activation is an environmental risk factor for adult ventricular enlargement relevant to schizophrenia. The data indicate immune-associated environmental insults targeting early foetal development may have more extensive neurodevelopmental impact than identical insults in late prenatal life. © 2009 Li et al.published_or_final_versio
Understanding Growth and Malnutrition in Baka Pygmy Children
- Author
- A Froment
- A Indrayan
- A Jelenkovic
- AB Migliano
- AD Blackwell
- AJ Prendergast
- AL Park
- B Winegard
- BJ Akombi
- C Batini
- C Nagahori
- CEG Amorim
- CM Orr
- DM Stasinopoulos
- E Dinerstein
- E Patin
- FV Ramírez Rozzi
- FVR Rozzi
- GH Perry
- GH Perry
- GH Perry
- I Anderson
- J Lachance
- J Olivero
- JA Ndzo
- JP Jarvis
- KK Ong
- LM Schell
- M de Onis
- M de Onis
- M de Onis
- M Gurven
- M Henneberg
- M Lopez
- M Martin
- MR Robinson
- N Ohenjo
- NCD Risk Factor Collaboration (NCD-RisC)
- NSA Becker
- P Hsieh
- P Pridmore
- P Verdu
- R Walker
- RA Rigby
- RC Bailey
- S Stinson
- S Stinson
- SJ Gray
- SS Urlacher
- T Lohman
- TB Heaton
- The Lancet
- TJ Pemberton
- UNICEF WHO, World Bank
- V Natale
- WHO Multicentre Growth Reference Study Group
- World Health Organization
- Y Diekmann
- Z Foster
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 31/05/2020
- Field of study
We determined stunting, wasting, and obesity frequencies in a total 1092 2-to-12 year old Baka Pygmy children from anthropometric and health data gathered in 34 villages in the Djoum-Mintom region in southeastern Cameroon in four health campaigns in 2010 and 2017–9. We compare these to the WHO Child Growth Standards, Amazonian Tsiname growth references for inter-population comparisons and the study population itself. Population-specific growth charts were constructed using GAMLSS modelling. Our results show that Baka children have one of the highest global rates of stunting relative to the WHO child growth standard with 57.8% for 2-to-12 year olds and 64% and 73% for 2-to-4 year old girls and boys, respectively. Frequencies of wasting, overweight, and low BMI were low at 3.4%, 4.6% and 4.3%, respectively, for 2-to-12 year olds. Underweight was at 25.5%, in the upper range for sub-Saharan Africa. Edemas indicated rare severe malnutrition (0.3%). Uncertainties in age estimation had dramatic effects on the reliability of estimated individual z-scores but distributions of z-scores were robust at a population level. In the context of the recent evidence for genetic adaptation of the Pygmies’ small stature to the tropical forest environment we argue that WHO child standards for weight and BMI are applicable. However, standards for height are clearly not adequate for Pygmy people. To achieve UN Sustainable Development Goals, we recommend that Pygmy specific growth standards are developed for the various, genetically differing Pygmy tribes
Pan-cancer analysis of whole genomes
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- The ICGC/TCGA Pan-Cancer Analysis of Whole Genomes Consortium View Correspondence (jump link)
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- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
Iron Behaving Badly: Inappropriate Iron Chelation as a Major Contributor to the Aetiology of Vascular and Other Progressive Inflammatory and Degenerative Diseases
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- The Long-Term Intervention with Pravastatin in Ischaemic Disease (LIPID) Study Group
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- Publication venue
- Publication date
- 09/08/2008
- Field of study
The production of peroxide and superoxide is an inevitable consequence of
aerobic metabolism, and while these particular "reactive oxygen species" (ROSs)
can exhibit a number of biological effects, they are not of themselves
excessively reactive and thus they are not especially damaging at physiological
concentrations. However, their reactions with poorly liganded iron species can
lead to the catalytic production of the very reactive and dangerous hydroxyl
radical, which is exceptionally damaging, and a major cause of chronic
inflammation. We review the considerable and wide-ranging evidence for the
involvement of this combination of (su)peroxide and poorly liganded iron in a
large number of physiological and indeed pathological processes and
inflammatory disorders, especially those involving the progressive degradation
of cellular and organismal performance. These diseases share a great many
similarities and thus might be considered to have a common cause (i.e.
iron-catalysed free radical and especially hydroxyl radical generation). The
studies reviewed include those focused on a series of cardiovascular, metabolic
and neurological diseases, where iron can be found at the sites of plaques and
lesions, as well as studies showing the significance of iron to aging and
longevity. The effective chelation of iron by natural or synthetic ligands is
thus of major physiological (and potentially therapeutic) importance. As
systems properties, we need to recognise that physiological observables have
multiple molecular causes, and studying them in isolation leads to inconsistent
patterns of apparent causality when it is the simultaneous combination of
multiple factors that is responsible. This explains, for instance, the
decidedly mixed effects of antioxidants that have been observed, etc...Comment: 159 pages, including 9 Figs and 2184 reference
Search for physics beyond the standard model in events with two leptons of same sign, missing transverse momentum, and jets in proton-proton collisions at root s=13TeV
- Author
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- Abbaneo D
- Abbiendi G
- Abbrescia M
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- Abdulsalam A
- Ackert A
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- Publication venue
- Publication date
- 01/01/2017
- Field of study
Peer reviewe
A Deep Neural Network for Simultaneous Estimation of b Jet Energy and Resolution
- Author
- Aarrestad TK
- Abbaneo D
- Abbiendi G
- Abbrescia M
- Abdalla H
- Abdullah WATW
- Abdullin S
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- Publication venue
- Publication date
- 01/01/2020
- Field of study
We describe a method to obtain point and dispersion estimates for the energies of jets arising from b quarks produced in proton-proton collisions at an energy of s = 13 TeV at the CERN LHC. The algorithm is trained on a large sample of simulated b jets and validated on data recorded by the CMS detector in 2017 corresponding to an integrated luminosity of 41 fb - 1 . A multivariate regression algorithm based on a deep feed-forward neural network employs jet composition and shape information, and the properties of reconstructed secondary vertices associated with the jet. The results of the algorithm are used to improve the sensitivity of analyses that make use of b jets in the final state, such as the observation of Higgs boson decay to b b ¯
Assimilation of preexisting Pleistocene intrusions at Long Valley by periodic magma recharge accelerates rhyolite generation: rethinking the remelting model
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- A Goolaerts
- A Heumann
- A Heumann
- AD Burnham
- AF Glazner
- AF Glazner
- AIS Kemp
- AJ Pietruszka
- AK Schmitt
- AN Halliday
- AN Halliday
- AT Anderson
- Axel K. Schmitt
- BLA Charlier
- BW Evans
- C Annen
- C Weiland
- CD Miller
- CJ Hawkesworth
- CJN Wilson
- CK Wilson
- CM Johnson
- CR Bacon
- DA Wark
- DA Wark
- DJ DePaolo
- DJ DePaolo
- DJ DePaolo
- Dominique Weis
- Donald J. DePaolo
- DS Coleman
- EA Mankinen
- EB Watson
- EB Watson
- FJ Spera
- FV Perry
- G Zandt
- GA Mahood
- GAR Gualda
- GAR Gualda
- GR Davies
- GR Davies
- H Chiba
- IN Bindeman
- J Dufek
- J Michel
- J Roberge
- JA Crisp
- JA Vazquez
- JA Wolff
- JB Paces
- JB Thomas
- JEP Matzel
- JF Wotzlaw
- JG Bryce
- JI Simon
- JI Simon
- JI Simon
- JI Simon
- JL Crowley
- JM Lees
- JM Metz
- JM Metz
- JN Christensen
- JN Christensen
- Justin I. Simon
- JW Davis
- KF Kuiper
- KW Min
- LL Claiborne
- M Wiedenbeck
- Masturyono
- MJ Tappa
- MR Reid
- MR Reid
- MR Reid
- MR Reid
- MR Reid
- MS Ghiorso
- O Bachmann
- O Bachmann
- P Boehnke
- P Bogaard van den
- P Wallace
- Paul R. Renne
- PJ Wallace
- PR Renne
- PR Renne
- PR Renne
- PW Lipman
- R Mundil
- RA Bailey
- RD Mills
- RL Hervig
- RL Smith
- Roland Mundil
- RSJ Sparks
- S Nomade
- S Sherburn
- SA Sañudo-Wilhelmy
- SM White
- TA Rivera
- TE Krogh
- TH Druitt
- U Schärer
- W Hildreth
- W Hildreth
- W Hildreth
- W Hildreth
- W Pretorius
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Evidence for the 125 GeV Higgs boson decaying to a pair of tau leptons
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- Abbiendi G
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- Abdullin S
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- Virto AL
- Vischia P
- Vishnevskiy D
- Vitulo P
- Vlasov E
- Vlimant JR
- Vogel H
- Volkov A
- Volobouev I
- Volpe R
- Vorobiev I
- Vorobyev A
- Vorobyev A
- Voutilainen M
- Vuosalo C
- Vutova M
- Wagner SR
- Wagner-Kuhr J
- Wakefield S
- Walker M
- Wallny R
- Walsh R
- Walsh S
- Waltenberger W
- Wang D
- Wang F
- Wang J
- Wang M
- Wang Q
- Wang X
- Wang Z
- Wardle N
- Wasserbaech S
- Wayand S
- Wayne M
- Weber H
- Weber HA
- Weber M
- Weber M
- Weiler T
- Weinberg M
- Wendland L
- Weng Y
- Werner JS
- West C
- Wetzel J
- Whitbeck A
- Whitmore J
- Wickramage N
- Wilbur S
- Wilken R
- Wilkinson R
- Williams T
- Willmott C
- Wimpenny S
- Winer BL
- Winn D
- Winstrom L
- Wissing C
- Wittich P
- Wittmer B
- Wohri HK
- Wolf M
- Wolf R
- Wolfe H
- Wolszczak W
- Womersley WJ
- Won S
- Wood D
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- Wood JS
- Woodard A
- Woods N
- Worm SD
- Wright D
- Wrochna G
- Wu W
- Wulsin HW
- Wulz CE
- Wurthwein F
- Wyslouch B
- Xiao H
- Xie S
- Xie W
- Xu L
- Yagil A
- Yalvac M
- Yang F
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- Yang Y
- Yang ZC
- Yazgan E
- Yelton J
- Yetkin T
- Yi K
- Yilmaz Y
- Yohay R
- Yoo HD
- Yoo J
- Yoon AS
- York A
- Yu I
- Yu SS
- Yucel M
- Yumiceva F
- Yun JC
- Yzquierdo APC
- Zabel J
- Zabi A
- Zablocki J
- Zaganidis N
- Zakaria M
- Zalewski P
- Zanetti A
- Zanetti M
- Zarubin A
- Zeinali M
- Zeise M
- Zenz SC
- Zeuner WD
- Zeyrek M
- Zhang J
- Zhang L
- Zheng Y
- Zhokin A
- Zhu RY
- Zhukov V
- Zhukova V
- Zielinski M
- Zito G
- Zotto P
- Zou W
- Zsigmond AJ
- Zucchetta A
- Zumerle G
- Zuranski A
- Publication venue
- Publication date
- 26/11/2015
- Field of study
A search for a standard model Higgs boson decaying into a pair of tau leptons is performed using events recorded by the CMS experiment at the LHC in 2011 and 2012. The dataset corresponds to an integrated luminosity of 4.9 fb(-1) at a centre-of-mass energy of 7 TeV and 19.7 fb(-1) at 8 TeV. Each tau lepton decays hadronically or leptonically to an electron or a muon, leading to six different final states for the tau-lepton pair, all considered in this analysis. An excess of events is observed over the expected background contributions, with a local significance larger than 3 standard deviations for m (H) values between 115 and 130 GeV. The best fit of the observed H -> tau tau signal cross section times branching fraction for m(H) = 125 GeV is 0.78 +/- 0.27 times the standard model expectation. These observations constitute evidence for the 125 GeV Higgs boson decaying to a pair of tau leptons
Randomized clinical trials of dental bleaching – Compliance with the CONSORT Statement: a systematic review
- Author
- Acosta Gómez AP
- Akarslan ZZ
- Al Shethri S
- Al-Zarea BK
- Alessandra REIS
- Alessandro Dourado LOGUERCIO
- Alexandra Mara de PAULA
- Almeida LCA
- Alomari Q
- Alonso de la Peña V
- Alonso de la Peña V
- Altman DG
- Auschill T
- Auschill TM
- Barbui C
- Barlow A
- Barnes DM
- Basting RT
- Bearn DR
- Berga-Caballero A
- Bernardon JK
- Bernardon JK
- Bernardon JK
- Bianca Medeiros MARAN
- Bizhang M
- Bizhang M
- Bonafé E
- Borenstein M
- Bortolatto JF
- Braun A
- Browning WD
- Browning WD
- Bruhn AM
- Burns PB
- Cairo F
- Callan RS
- Cardoso PC
- Carvalho BCF
- Cerqueira RR
- Chan AW
- Cibirka RM
- Cioffi I
- Collins LZ
- Corbella S
- Correa AC
- Costa J
- Costa JB
- Cronin M
- Dawson PF
- De Angelis C
- Delgado E
- Deliperi S
- Donly K
- Donly KJ
- Donly KJ
- Donly KJ
- Donly KJ
- Egger M
- Farhat PBA
- Farrell S
- Fernández E
- Fernández E
- Ferrari M
- Flint HE
- Freitas PM
- Gallagher A
- Gallo JR
- Garcia-Godoy F
- Gerlach R
- Gerlach RW
- Gerlach RW
- Gerlach RW
- Gerlach RW
- Gerlach RW
- Gerlach RW
- Gerlach RW
- Geus JL
- Geus JL
- Geus JL
- Geus JL
- Giachetti L
- Giniger M
- Giniger M
- Gomes RS
- Goodson JM
- Grobler SR
- Guerrero JC
- Gurgan S
- Guyatt GH
- Guênes GMT
- Hanks CT
- Hannig C
- Henry RK
- Higgins JP
- Higgins JP
- Hopewell S
- Houle TT
- Hyland BW
- Ishikawa-Nagai S
- Jadad E
- Javaheri DS
- Jorge PERDIGÃO
- Jüni P
- Karpinia K
- Karpinia KA
- Kihn P
- Kihn P
- Kina JF
- Kloukos D
- Knösel M
- Knösel M
- Kose C
- Kose C
- Kossatz S
- Kossatz S
- Kozlovsky A
- Krause F
- Kugel G
- Kugel G
- Kugel G
- Kugel G
- Kugel G
- Lempesi E
- Leonard Jr RH
- Leonard Jr RH
- Leonard Jr RH
- Lewgoy HR
- Li Y
- Lo EC
- Lo Giudice R
- Loguercio AD
- Loyola-Rodriguez JP
- Luo W
- Machado LS
- Machado LS
- Maghaireh GA
- Marson FC
- Martins GC
- Martín J
- Matis BA
- Matis BA
- Matis BA
- Matis BA
- Matis BA
- Matis BA
- Matis BA
- Matis BA
- Medeiros MCS
- Mehta D
- Meireles SS
- Meireles SS
- Meireles SS
- Meireles SS
- Meireles SS
- Mena-Serrano AP
- Mena-Serrano AP
- Miller MB
- Moghadam FV
- Mohan N
- Moher D
- Moher D
- Mokhlis GR
- Moncada G
- Mondelli RF
- Montenegro R
- Montenegro-Arana A
- Morgan S
- Myers ML
- Nathoo S
- Nathoo S
- Navarra CO
- Nutter BJ
- Oginni AO
- Ontiveros JC
- Palé M
- Pandis N
- Pandis N
- Papageorgiou SN
- Papathanasiou A
- Papathanasiou A
- Paula EA
- Paula EA
- Paula EA
- Perry R
- Pocock SJ
- Polydorou O
- Posso Moreno SL
- Rajasekharan S
- Reis A
- Reis A
- Reis A
- Reis A
- Rennie D
- Rezende M
- Rezende M
- Rezende M
- Rezende M
- Rezende M
- Rezende M
- Roderjan DA
- Rosenstiel SF
- Santana MAP
- Sarkis-Onofre R
- Schulz KF
- Schulz KF
- Schulz KF
- Schulz KF
- Shahidi H
- Shamseer L
- Shanbhag R
- Sielski C
- Silva FM
- Simon JF
- Sjögren P
- Soares CJ
- Soares DG
- Soares DG
- Strobl A
- Sundfeld RH
- Swift EJ
- Swift Jr EJ
- Swift Jr EJ
- Swift Jr EJ
- T Tavares M
- Tay LY
- Tay LY
- Taíse Alessandra HANZEN
- Tsubura S
- Tsubura S
- Turkun M
- Turner L
- Vano M
- Ward M
- Wetter NU
- Xu X
- Yudhira R
- Zantner C
- Zekonis R
- Zhao K
- Ziebolz D
- Ziemba SL
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
Recommended from our members
SYSTHESIS OF VOLCANISM STUDIES FOR THE YUCCA MOUNTAIN SITE CHARACTERIZATION PROJECT
- Publication venue
- United States. Department of Energy. Yucca Mountain Project Office.
- Publication date
- 23/09/1997
- Field of study
This report synthesizes the results of volcanism studies conducted by scientists at the Los Alamos National Laboratory and collaborating institutions on behalf of the Department of Energy's Yucca Mountain Project. Chapter 1 introduces the volcanism issue for the Yucca Mountain site and provides the reader with an overview of the organization, content, and significant conclusions of this report. The hazard of future basaltic volcanism is the primary topic of concern including both events that intersect a potential repository and events that occur near or within the waste isolation system of a repository. Future volcanic events cannot be predicted with certainty but instead are estimated using formal methods of probabilistic volcanic hazard assessment (PVHA). Chapter 2 describes the volcanic history of the Yucca Mountain region (YMR) and emphasizes the Pliocene and Quaternary volcanic record, the interval of primary concern for volcanic risk assessment. The distribution, eruptive history, and geochronology of Plio-Quaternary basalt centers are described by individual center emphasizing the younger postcaldera basalt (<5 Ma). The Lathrop Wells volcanic center is described in detail because it is the youngest basalt center in the YMR. The age of the Lathrop Wells center is now confidently determined to be about 75 thousand years old. Chapter 3 describes the tectonic setting of the YMR and presents and assesses the significance of multiple alternative tectonic models. The distribution of Pliocene and Quaternary basaltic volcanic centers is evaluated with respect to tectonic models for detachment, caldera, regional and local rifting, and the Walker Lane structural zone. Geophysical data are described for the YMR and are used as an aid to understand the distribution of past basaltic volcanic centers and possible future magmatic processes. Chapter 4 discusses the petrologic and geochemical features of basaltic volcanism in the YMR, the southern Great Basin and the Basin and Range province. Geochemical and isotopic data are presented for post-Miocene basalts of the Yucca Mountain region. Alternative petrogenetic models are assessed for the formation of the Lathrop Wells volcanic center. Based on geochemical data, basaltic ash in fault trenches near Yucca Mountain is shown to have originated from the Lathrop Wells center. Chapter 5 synthesizes eruptive and subsurface effects of basaltic volcanism on a potential repository and summarizes current concepts of the segregation, ascent, and eruption of basalt magma. Chapter 6 synthesizes current knowledge of the probability of disruption of a potential repository at Yucca Mountain. In 1996, an Expert Elicitation panel was convened by DOE that independently conducted PVHA for the Yucca Mountain site. Chapter 6 does not attempt to revise this PVHA; instead, it further examines the sensitivity of variables in PVHA. The approaches and results of PVHA by the expert judgment panel are evaluated and incorporated throughout this chapter. The disruption ratio (E2) is completely re-evaluated using simulation modeling that describes volcanic events based on the geometry of basaltic feeder dikes. New estimates of probability bounds are developed. These comparisons show that it is physically implausible for the probability of magmatic disruption of the Yucca Mountain site to be greater than 10{sup -7} events per year. Bounding probability estimates are used to assess possible implications of not drilling aeromagnetic anomalies in the Arnargosa Valley and Crater Flat. The results of simulation modeling are used to assess the sensitivity of the disruption probability for the location of northeast boundaries of volcanic zones near the Yucca Mountain site. A new section on modeling of radiological releases associated with surface and subsurface magmatic activity has been added to chapter 6. The modeling results are consistent with past total system performance assessments that show future volcanic and magmatic events are not significant components of repository performance and volcanism is not a priority issue for performance assessment studies
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