33 research outputs found
Genetic polymorphisms in the cyclooxygenase-2 gene, use of nonsteroidal anti-inflammatory drugs, and breast cancer risk
- Author
- A Papafili
- A Ristimaki
- AJ Dannenberg
- AJ Dannenberg
- AJ Dannenberg
- Alfred I Neugut
- AM Brodie
- B Arun
- CL Siezen
- CM Ulrich
- D Campa
- D Campa
- D Hwang
- D Pereg
- DA Dixon
- DA Tregouet
- DG Cox
- DG Cox
- DW Hosmer
- EJ Jacobs
- F Andre
- F Cipollone
- F Liu
- HR Herschman
- IU Ali
- JA Elvin
- Jing Shen
- JM Park
- KJ Rothman
- L Gallicchio
- LA Brosens
- LA Garcia Rodriguez
- LC Sakoda
- M Stephens
- Marilie D Gammon
- Mary Beth Terry
- MB Terry
- MD Gammon
- MD Gammon
- PG Moorman
- PG Moorman
- RC Panguluri
- RE Harris
- Regina M Santella
- RN DuBois
- S Di Marco
- S Kang
- SA Khuder
- SF Marshall
- SJ Cok
- Susan L Teitelbaum
- T Hall-Pogar
- T Kosaka
- U Langsenlehner
- U Vogel
- VM Oliveira
- WP Koh
- X Chen
- X Zhang
- Z Hu
- Publication venue
- BioMed Central
- Publication date
- 01/01/2006
- Field of study
INTRODUCTION: The association between use of nonsteroidal anti-inflammatory drugs (NSAIDs) and breast cancer risk remains unclear. Inconsistencies in previously reported findings may be partly due to differences in expression of cyclooxygenase (COX)-2. We hypothesized that genetic polymorphisms (COX-2 .926, COX-2 .5209, and COX-2 .8473) may reduce overall breast cancer risk or risk for subtypes of breast cancer by modulating the inflammatory response and may interact with aspirin or any NSAID use. METHODS: We conducted a population-based, case-control study in which we genotyped 1,067 breast cancer cases and 1,110 control individuals included in the Long Island Breast Cancer Study Project. RESULTS: No major effects of the three COX-2 variant alleles on breast cancer risk were found. A total of eight distinct haplotypes and 18 diplotypes were observed in the population. Overall, no significant associations between COX-2 haplotypes/diplotypes and breast cancer risk were observed. Among women who used aspirin or any NSAID there was little evidence for an interaction with the at-risk COX-2 genotypes, with one exception. Among women with hormone receptor positive breast cancer, the reduced risk for any NSAID use was only evident among those who had at least one variant C allele of COX-2 .8473 (odds ratio = 0.7, 95% confidence interval = 0.5 to 1.0; P for the interaction = 0.02). There was no corresponding interaction for aspirin use, possibly because of limited power. CONCLUSION: These data provide modest evidence that the C allele of COX-2 .8473 may interact with NSAIDs to reduce risk for hormone receptor positive breast cancer
A review of gene-drug interactions for nonsteroidal anti-inflammatory drug use in preventing colorectal neoplasia.
- Author
- A Ekbom
- A Papafili
- AE Rettie
- AN Kingsnorth
- AO Maree
- AT Chan
- BF McAdam
- BM Psaty
- BT Scott
- C M Ulrich
- CL Siezen
- CM Ulrich
- CM Ulrich
- CM Ulrich
- CM Ulrich
- CW Porter
- DG Cox
- DH Solomon
- E Fritsche
- E M Poole
- E Poole
- EJ Topol
- EL Barry
- EW Gerner
- F Catella-Lawson
- F Scorcioni
- F Wolter
- FE Silverstein
- G Singh
- GE Kuehl
- GM LaMuraglia
- H Takahashi
- HB Koo
- HJ Lin
- IU Ali
- J Bigler
- J Janne
- J Orbe
- J Ostrowski
- J T Cross
- JA Baron
- JA Baron
- JE Goodman
- JO Miners
- JR Vane
- JW Lampe
- L Hansson
- L Turchanowa
- LB Sansbury
- M Ciotti
- M Oshima
- MB Hansen-Petrik
- ME Martinez
- MK Halushka
- MM Bertagnolli
- MM Taketo
- N Arber
- N Babbar
- PC Chulada
- PP Carbone
- RA Gupta
- RA Hubner
- RJ Kulmacz
- RS Bresalier
- RS Sandler
- S Fries
- SD Solomon
- SD Solomon
- SK Peterson
- SP Motsko
- Steering Committee of the Physicians' Health Study Research Group
- T Jess
- WL Smith
- WP Koh
- WS Samowitz
- Y Cheng
- Y Guo
- Publication venue
- Nature Publishing Group
- Publication date
- 01/08/2008
- Field of study
Nonsteroidal anti-inflammatory drugs (NSAIDs) have been shown to be effective chemopreventive agents for colorectal neoplasia. Polymorphisms in NSAID targets or metabolizing enzymes may affect NSAID efficacy or toxicity. We conducted a literature review to summarize current evidence of gene-drug interactions between NSAID use and polymorphisms in COX1, COX2, ODC, UGT1A6 and CYP2C9 on risk of colorectal neoplasia by searching OVID and PubMed. Of 134 relevant search results, thirteen investigated an interaction. One study reported a significant interaction between NSAID use and the COX1 Pro17Leu polymorphism (P=0.03) whereby the risk reduction associated with NSAID use among homozygous wild-type genotypes was not observed among NSAID users with variant alleles. Recent pharmacodynamic data support the potential for gene-drug interactions for COX1 Pro17Leu. Statistically significant interactions have also been reported for ODC (315G>A), UGT1A6 (Thr181Ala+Arg184Ser or Arg184Ser alone), and CYP2C9 (*2/*3). No statistically significant interactions have been reported for polymorphisms in COX2; however, an interaction with COX2 -765G>C approached significance (P=0.07) in one study. Among seven remaining studies, reported interactions were not statistically significant for COX1, COX2 and ODC gene polymorphisms. Most studies were of limited sample size. Definitions of NSAID use differed substantially between studies. The literature on NSAID-gene interactions to date is limited. Reliable detection of gene-NSAID interactions will require greater sample sizes, consistent definitions of NSAID use and evaluation of clinical trial subjects of chemoprevention studies
Dystonia, facial dysmorphism, intellectual disability and breast cancer associated with a chromosome 13q34 duplication and overexpression of TFDP1: case report
- Author
- A Kupsch
- B Diosdado
- C Cayrol
- C Shen
- D Coverley
- F Hoche
- G Charlesworth
- Garrett L Rampon
- HC Mefford
- HH Li
- HJ Lee
- IA Adzhubei
- IU Isaias
- J Hersheson
- J Hippisley-Cox
- J Xiao
- J Xiao
- JA Rosenfeld
- Jianfeng Xiao
- JK Krauss
- JM Schwarz
- K Yasui
- Kelly D Foote
- L Melchor
- LJ Ozelius
- LL Chen
- M Lomazzi
- M Swift
- M Tagliati
- M Vidailhet
- MA Larkin
- Mariana Moscovich
- Mark S LeDoux
- MC Abba
- Michael S Okun
- MJ Ravitz
- MS LeDoux
- MS Nicoloso
- N Calakos
- OS Cohen
- PC Ng
- Q Cheng
- R Saunders-Pullman
- Ramon L Rodriguez
- RE Burke
- S Girirajan
- S Girirajan
- S Makino
- S Talluri
- Satya R Vemula
- SD Castillo
- SN Silva
- SR Vemula
- T Fuchs
- T Shinomiya
- X Hong
- X Liu
- Y Tian
- YH Koh
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Effect of prophylactic 360° laser treatment for prevention of retinal detachment after phacovitrectomy: (Prophylactic 360° laser treatment for prevention of retinal detachment)
- Author
- A Guillaubey
- AN Stangos
- AS Banker
- B Kirchhof
- Brian C Oveson
- C Territo
- DAH Laidlaw
- E de Juan Jr
- E Mendrinos
- EH Jaccoma
- F Patelli
- GA Williams
- H Ahmadieh
- H Heimann
- HJ Koh
- IP Theocharis
- IU Scott
- J Hwang
- JK Moore
- JT Thompson
- LJ Faia
- M Cowley
- M Rasouli
- PD Brazitikos
- PV Algvere
- R Scartozzi
- RF Escoffery
- RG Michels
- RG Michels
- RN Sjaarda
- RV Azad
- RV Campo
- S Rizzo
- SS Park
- T Avitabile
- Takeshi Iwase
- TJ Wolfensberger
- TS Chang
- V Martinez-Castillo
- Young-Joon Jo
- YR Sharma
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Global burden of 369 diseases and injuries in 204 countries and territories, 1990-2019: a systematic analysis for the Global Burden of Disease Study 2019
- Author
- Abbafati C
- Abbas KM
- Abbasi M
- Abbasi-Kangevari M
- Abbasifard M
- Abbastabar H
- Abd El Razek HM
- Abd El Razek MM
- Abd-Allah F
- Abdelalim A
- Abdollahi M
- Abdollahpour I
- Abdulkader RS
- Abdullah KH
- Abedi A
- Abedi P
- Abegaz KH
- Abolhassani H
- Abosetugn AE
- Aboyans V
- Abrams EM
- Abreu LG
- Abrigo MRM
- Abu Haimed AK
- Abu-Gharbieh E
- Abu-Raddad LJ
- Abualhasan A
- Abushouk AI
- Acebedo A
- Ackerman IN
- Adabi M
- Adair T
- Adamu AA
- Adebayo OM
- Adedeji IA
- Adekanmbi V
- Adelson JD
- Adeoye AM
- Adetokunboh OO
- Adham D
- Advani SM
- Afarideh M
- Afshari M
- Afshin A
- Agardh EE
- Agarwal G
- Agasthi P
- Agesa KM
- Aghaali M
- Aghamir SMK
- Agrawal A
- Ahmad T
- Ahmadi A
- Ahmadi K
- Ahmadi M
- Ahmadieh H
- Ahmadpour E
- Ahmed MB
- Aji B
- Akalu TY
- Akinyemi RO
- Akinyemiju T
- Akombi B
- Akunna CJ
- Al-Aly Z
- Al-Mekhlafi HM
- Al-Raddadi RM
- Alahdab F
- Alam K
- Alam N
- Alam S
- Alam T
- Alanezi FM
- Alanzi TM
- Albertson SB
- Alcalde-Rabanal JE
- Alema NM
- Alemu BW
- Alemu YM
- Alhabib KF
- Alhassan RK
- Ali M
- Ali S
- Alicandro G
- Alijanzadeh M
- Alinia C
- Alipour V
- Alizade H
- Aljunid SM
- Alla F
- Allebeck P
- Almadi MAH
- Almasi A
- Almasi-Hashiani A
- Almasri NA
- Almulhim AM
- Alonso J
- Altirkawi KA
- Alumran AK
- Alvis-Guzman N
- Alvis-Zakzuk NJ
- Amare AT
- Amare B
- Amini S
- Amini-Rarani M
- Aminorroaya A
- Amiri F
- Amit AML
- Amugsi DA
- Amul GGH
- Anbesu EW
- Ancuceanu R
- Anderlini D
- Anderson JA
- Andrei CL
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- Androudi S
- Angus C
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- Ansari I
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- Norrving B
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- Olagunju AT
- Olusanya BO
- Olusanya JO
- Oluwasanu MM
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- Soofi M
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- Sudaryanto A
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- Tadakamadla SK
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- Truelsen TC
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- Umeokonkwo CD
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- Upadhyay E
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- Venketasubramanian N
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- Wickramasinghe ND
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- Wijeratne T
- Wilner LB
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- Wiysonge CS
- Wojtyniak B
- Woldu G
- Wolfe CDA
- Wondmeneh TG
- Wondmieneh AB
- Wool EE
- Wozniak SS
- Wu A-M
- Wu C
- Wu J
- Wunrow HY
- Xie Y
- Xu G
- Xu R
- Yadgir S
- Yamada T
- Yamagishi K
- Yaminfirooz M
- Yano Y
- Yaya S
- Yazdi-Feyzabadi V
- Yearwood JA
- Yeheyis TY
- Yeshitila YG
- Yilgwan CS
- Yilma MT
- Yip P
- Yonemoto N
- Yoon S-J
- York HW
- Younis MZ
- Younker TP
- Yousefi B
- Yousefi Z
- Yousefifard M
- Yousefinezhadi T
- Yousuf AY
- Yu C
- Yu Y
- Yuan C-W
- Yuce D
- Yusefzadeh H
- Zadey S
- Zahabi SS
- Zaidi SS
- Zaki L
- Zaki MES
- Zakzuk J
- Zamani M
- Zamanian M
- Zandian H
- Zangeneh A
- Zarafshan H
- Zastrozhin MS
- Zewdie KA
- Zhang J
- Zhang Y
- Zhang Z-J
- Zhao JT
- Zhao X-JG
- Zhao Y
- Zheleva B
- Zheng P
- Zhou M
- Zhu C
- Ziapour A
- Zimsen SRM
- Zlavog BS
- Zodpey S
- Publication venue
- 'Elsevier BV'
- Publication date
- 17/10/2020
- Field of study
Five insights from the Global Burden of Disease Study 2019
- Author
- Abbafati C
- Abbas KM
- Abbasi M
- Abbasi-Kangevari M
- Abbasifard M
- Abbastabar H
- Abd El Razek HM
- Abd El Razek MM
- Abd-Allah F
- Abdelalim A
- Abdollahi M
- Abdollahpour I
- Abdulkader RS
- Abdullah KH
- Abedi A
- Abedi P
- Abegaz KH
- Abolhassani H
- Abosetugn AE
- Aboyans V
- Abrams EM
- Abreu LG
- Abrigo MRM
- Abu Haimed AK
- Abu-Gharbieh E
- Abu-Raddad LJ
- Abualhasan A
- Abushouk AI
- Acebedo A
- Ackerman IN
- Adabi M
- Adair T
- Adamu AA
- Adebayo OM
- Adedeji IA
- Adekanmbi V
- Adelson JD
- Adeoye AM
- Adetokunboh OO
- Adham D
- Advani SM
- Afarideh M
- Afshari M
- Afshin A
- Agardh EE
- Agarwal G
- Agasthi P
- Agesa KM
- Aghaali M
- Aghamir SMK
- Agrawal A
- Ahmad T
- Ahmadi A
- Ahmadi K
- Ahmadi M
- Ahmadieh H
- Ahmadpour E
- Ahmed MB
- Aji B
- Akalu TY
- Akinyemi RO
- Akinyemiju T
- Akombi B
- Akunna CJ
- Al-Aly Z
- Al-Mekhlafi HM
- Al-Raddadi RM
- Alahdab F
- Alam K
- Alam N
- Alam S
- Alam T
- Alanezi FM
- Alanzi TM
- Albertson SB
- Alcalde-Rabanal JE
- Alema NM
- Alemu BW
- Alemu YM
- Alhabib KF
- Alhassan RK
- Ali M
- Ali S
- Alicandro G
- Alijanzadeh M
- Alinia C
- Alipour V
- Alizade H
- Aljunid SM
- Alla F
- Allebeck P
- Almadi MAH
- Almasi A
- Almasi-Hashiani A
- Almasri NA
- Almulhim AM
- Alonso J
- Altirkawi KA
- Alumran AK
- Alvis-Guzman N
- Alvis-Zakzuk NJ
- Amare AT
- Amare B
- Amini S
- Amini-Rarani M
- Aminorroaya A
- Amiri F
- Amit AML
- Amugsi DA
- Amul GGH
- Anbesu EW
- Ancuceanu R
- Anderlini D
- Anderson JA
- Andrei CL
- Andrei T
- Androudi S
- Angus C
- Anjomshoa M
- Ansari F
- Ansari I
- Ansari-Moghaddam A
- Antonazzo IC
- Antonio CAT
- Antony CM
- Antriyandarti E
- Anvari D
- Anwer R
- Appiah SCY
- Arab-Zozani M
- Arabloo J
- Aravkin AY
- Arba AAK
- Aremu O
- Ariani F
- Aripov T
- Armoon B
- Arnlov J
- Arowosegbe OO
- Aryal KK
- Arzani A
- Asaad M
- Asadi-Aliabadi M
- Asadi-Pooya AA
- Asgari S
- Asghari B
- Ashbaugh C
- Assmus M
- Atafar Z
- Athari SS
- Atnafu DD
- Atout MMW
- Atre SR
- Atteraya MS
- Ausloos F
- Ausloos M
- Avila-Burgos L
- Avokpaho EFGA
- Ayano G
- Ayanore MA
- Aynalem GL
- Aynalem YA
- Ayza MA
- Azari S
- Azarian G
- Azene ZN
- Azhar G
- Azzopardi PS
- Babaee E
- Badawi A
- Badiye AD
- Bagherzadeh M
- Bagli E
- Bahrami MA
- Baig AA
- Bairwa M
- Bakhshaei MH
- Bakhtiari A
- Bakkannavar SM
- Balachandran A
- Balakrishnan S
- Balalla S
- Balassyano S
- Baldasseroni A
- Bali AO
- Ball K
- Ballew SH
- Balzi D
- Banach M
- Bandpei MAM
- Banerjee SK
- Banik PC
- Bannick MS
- Bante AB
- Bante SA
- Baraki AG
- Barboza MA
- Barker-Collo SL
- Barnighausen TW
- Barrero LH
- Barthelemy CM
- Barua L
- Barzegar A
- Basaleem H
- Bassat Q
- Basu S
- Baune BT
- Bayati M
- Baye BA
- Bazmandegan G
- Becker JS
- Bedi N
- Beghi E
- Behzadifar M
- Bejot Y
- Bekuma TTT
- Bell ML
- Bello AK
- Ben L
- Bender RG
- Bennett DA
- Bennitt FB
- Bensenor IM
- Benziger CP
- Berhe K
- Berman AE
- Bernabe E
- Bernstein RS
- Bertolacci GJ
- Bhagavathula AS
- Bhageerathy R
- Bhala N
- Bhandari D
- Bhardwaj P
- Bhat AG
- Bhattacharyya K
- Bhattarai S
- Bhutta ZA
- Bibi S
- Bicer BK
- Biehl MH
- Bijani A
- Bikbov B
- Bilano V
- Bin Sayeed MS
- Bin Zaman S
- Biondi A
- Birihane BM
- Bisanzio D
- Bisignano C
- Biswas RK
- Bitew H
- Bjorge T
- Bockarie MJ
- Bohlouli S
- Bohluli M
- Bojia HA
- Bolla SR
- Boloor A
- Boon-Dooley AS
- Borges G
- Borzi AM
- Borzouei S
- Bose D
- Bosetti C
- Boston S
- Boufous S
- Bourne R
- Brady OJ
- Braithwaite D
- Brauer M
- Brayne C
- Breitborde NJK
- Breitner S
- Brenner H
- Breusov AV
- Briant PS
- Briggs AM
- Briko AN
- Briko NI
- Britton GB
- Brugha T
- Bryazka D
- Buchbinder R
- Bumgarner BR
- Burkart K
- Burnett RT
- Busse R
- Butt ZA
- Caetano dos Santos FL
- Cahill LE
- Cahuana-Hurtado L
- Cai T
- Callender CSKH
- Camera LA
- Campos-Nonato IR
- Cao J
- Car J
- Car LT
- Cardenas R
- Carreras G
- Carrero JJ
- Carvalho F
- Castaldelli-Maia JM
- Castaneda-Orjuela CA
- Castelpietra G
- Castle CD
- Castro E
- Castro F
- Catala-Lopez F
- Causey K
- Cederroth CR
- Cercy KM
- Cerin E
- Chalek J
- Chandan JS
- Chang AR
- Chang AY
- Chang J-C
- Chang K-L
- Charan J
- Charlson FJ
- Chattu VK
- Chaturvedi S
- Cherbuin N
- Chimed-Ochir O
- Chin KL
- Chirinos-Caceres JL
- Cho DY
- Choi J-YJ
- Christensen H
- Chu D-T
- Chung MT
- Chung S-C
- Cicuttini FM
- Ciobanu LG
- Cirillo M
- Cislaghi B
- Classen TKD
- Cohen AJ
- Collins EL
- Comfort H
- Compton K
- Conti S
- Cooper OR
- Corso B
- Cortesi PA
- Costa VM
- Cousin E
- Cowden RG
- Cowie BC
- Cromwell EA
- Croneberger AJ
- Cross DH
- Cross M
- Crowe CS
- Cruz JA
- Cummins S
- Cunningham M
- D'Amico E
- da Silva DD
- Dahlawi SMA
- Dai H
- Dai H
- Damasceno AAM
- Damiani G
- Dandona L
- Dandona R
- Daneshpajouhnejad P
- Dangel WJ
- Danielsson A-K
- Dargan PI
- Darshan BB
- Darwesh AM
- Daryani A
- Das Gupta R
- Das Neves J
- Das JK
- Dash AP
- Davey G
- Davila-Cervantes CA
- Davis AC
- Davitoiu DV
- Davletov K
- De Leo D
- De Neve J-W
- Dean FE
- DeCleene NK
- Deen A
- Defo BK
- Degenhardt L
- DeLang M
- Dellavalle RP
- Demeke FM
- Demoz GT
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- Zangeneh A
- Zarafshan H
- Zastrozhin MS
- Zewdie KA
- Zhang J
- Zhang Y
- Zhang Z-J
- Zhao JT
- Zhao X-JG
- Zhao Y
- Zheleva B
- Zheng P
- Zhou M
- Zhu C
- Ziapour A
- Zimsen SRM
- Zlavog BS
- Zodpey S
- Publication venue
- Publication date
- 01/01/2020
- Field of study
The Global Burden of Diseases, Injuries, and Risk Factors Study (GBD) 2019 provides a rules-based synthesis of the available evidence on levels and trends in health outcomes, a diverse set of risk factors, and health system responses. GBD 2019 covered 204 countries and territories, as well as first administrative level disaggregations for 22 countries, from 1990 to 2019. Because GBD is highly standardised and comprehensive, spanning both fatal and non-fatal outcomes, and uses a mutually exclusive and collectively exhaustive list of hierarchical disease and injury causes, the study provides a powerful basis for detailed and broad insights on global health trends and emerging challenges. GBD 2019 incorporates data from 281 586 sources and provides more than 3.5 billion estimates of health outcome and health system measures of interest for global, national, and subnational policy dialogue. All GBD estimates are publicly available and adhere to the Guidelines on Accurate and Transparent Health Estimate Reporting. From this vast amount of information, five key insights that are important for health, social, and economic development strategies have been distilled. These insights are subject to the many limitations outlined in each of the component GBD capstone papers.Peer reviewe
Pan-cancer analysis of whole genomes
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- Weinberger
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- Weischenfeldt J. g
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- zd Gershenwald
- Zenklusen
- Zeps
- zf Ghossein
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang H. b
- Zhao
- Zheng
- Zhou
- Zhou
- Zhu
- Zhu
- zi zj
- zm Jayaseelan
- Zou
- Zou
- zt Kim
- zw Lewis
- Ă. A. th
- Ă. sk
- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
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- Abdelfatah S
- Abdellatif M
- Abdoli A
- Abel S
- Abeliovich H
- Abildgaard MH
- Abudu YP
- Acevedo-Arozena A
- Adamopoulos IE
- Adeli K
- Adolph TE
- Adornetto A
- Aflaki E
- Agam G
- Agarwal A
- Aggarwal BB
- Agnello M
- Agostinis P
- Agrewala JN
- Agrotis A
- Aguilar PV
- Ahmad ST
- Ahmed ZM
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- Aits S
- Aizawa S
- Akkoc Y
- Akoumianaki T
- Akpinar HA
- Al-Abd AM
- Al-Akra L
- Al-Gharaibeh A
- Alaoui-Jamali MA
- Alberti S
- Alcocer-GĂłmez E
- Alessandri C
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- Zhu B
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- Zhu G-Q
- Zhu H
- Zhu H
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- Zhu W-G
- Zhu Y
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- Zhuang H
- Zhuang X
- Zientara-Rytter K
- Zimmermann CM
- Ziviani E
- Zoladek T
- Zong W-X
- Zorov DB
- Zorzano A
- Zou W
- Zou Z
- Zou Z
- Zuryn S
- Zwerschke W
- Ălvarez ĂMC
- Ăvalos Y
- Ănal G
- ĂstĂŒn S
- ÄoliÄ M
- ÄokiÄ J
- Ćœerovnik E
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field
Global, regional, and national incidence of six major immune-mediated inflammatory diseases: findings from the global burden of disease study 2019
- Author
- Abate MD
- Abbasi-Kangevari M
- Abbasi-Kangevari Z
- Abbasian M
- Abd-Allah F
- Abdelmasseh M
- Abdollahifar M-A
- Abdou Elmeligy OA
- Abdulah DM
- Abebe EC
- Abedi A
- Abedi V
- Abidi H
- Aboagye RG
- Abolhassani H
- Abuabara K
- Abyadeh M
- Addo IY
- Adeniji KN
- Adepoju AV
- Adesina MA
- Afarideh M
- Aghamiri S
- Agodi A
- Agrawal A
- Aguilera Arriagada CE
- Ahmad A
- Ahmad D
- Ahmad S
- Ahmad S
- Ahmadi A
- Ahmed A
- Ahmed A
- Aithala JP
- Ajadi AA
- Ajami M
- Akbarzadeh-Khiavi M
- Al Mamun Sohag A
- Al-Worafi YM
- Alahdab F
- AlBataineh MT
- Alemi S
- Ali L
- Alif SM
- Almazan JU
- Almustanyir S
- Alqahtani JS
- Alqasmi I
- Alvis-Guzman N
- Alvis-Zakzuk NJ
- Aly H
- Amani R
- Amu H
- Amusa GA
- Andrei CL
- Ansar A
- Ansariniya H
- Anyasodor AE
- Arabloo J
- Arefnezhad R
- Arulappan J
- Asghari-Jafarabadi M
- Ashraf T
- Atata JA
- Athari SS
- Atlaw D
- Aujayeb A
- Awan AT
- Ayatollahi H
- Azadnajafabad S
- Azzam AY
- Badawi A
- Badiye AD
- Bagherieh S
- Baig AA
- Bantie BB
- Barchitta M
- Bardhan M
- Barker-Collo SL
- Barone-Adesi F
- Batra K
- Bayileyegn NS
- Behnoush AH
- Belete Gemeda BN
- Belgaumi UI
- Bemanalizadeh M
- Bensenor IM
- Beyene KA
- Bhagavathula AS
- Bhardwaj P
- Bhaskar S
- Bhat AN
- Bich Le TT
- Bitaraf S
- Bitra VR
- Boloor A
- Bora K
- Botelho JS
- Buchbinder R
- Buendia Rodriguez JA
- Calina D
- Carabeo Medina JR
- Carvalho AF
- Chattu VK
- Chichagi F
- Choi S
- Chou T-C
- Chu D-T
- Coberly K
- Costa VM
- Couto RAS
- Cruz-Martins N
- CĂĄmera LA
- Dadras O
- Dai X
- Damiani G
- Dascalu AM
- Dashti M
- Debela SA
- Dellavalle RP
- Demetriades AK
- Demlash AA
- Deng X
- Desai HD
- Desai R
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- Dharmaratne SD
- Diaz D
- Dibas M
- Dinis-Oliveira RJ
- Diress M
- Do TC
- Doan DK
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- Dodangeh M
- Dongarwar D
- Dube J
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- Ed-Dra A
- Edinur HA
- Eissazade N
- Ekholuenetale M
- Ekundayo TC
- Elemam NM
- Elhadi M
- Elmehrath AO
- Emamverdi M
- Emeto TI
- Esayas HL
- Eshetu HB
- Etaee F
- Fagbamigbe AF
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- Fakhradiyev IR
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- Fetensa G
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- Ganesan B
- Ghamari S-H
- Ghasemi M
- Gholamalizadeh M
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- Gillum RF
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- Gupta B
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- Hadi NR
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- Hien Nguyen HT
- Hiraike Y
- Holla R
- Hosseini M-S
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- Hwang B-F
- Ibitoye SE
- Ilic IM
- Ilic MD
- Iranmehr A
- Iravanpour F
- Ismail NE
- Iwagami M
- Iwu CCD
- Jacob L
- Jafarinia M
- Jafarzadeh A
- Jahankhani K
- Jahrami H
- Jakovljevic M
- Jamshidi E
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- Janodia MD
- Jayapal SK
- Jayaram S
- Jeganathan J
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- Kamal VK
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- Kazemian S
- Kebede SA
- Keikavoosi-Arani L
- Keykhaei M
- Khader YS
- Khajuria H
- Khamesipour F
- Khan Altaf IU
- Khan EA
- Khan IA
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- Khan MA
- Khan MAB
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- Khatatbeh H
- Khatatbeh MM
- Khateri S
- Khayat Kashani HR
- Kim MS
- Kisa A
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- Koh HY
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- Korzh O
- Kotnis AL
- Koul PA
- Koyanagi A
- Krishan K
- Kuddus M
- Kulkarni VV
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- Kundu S
- Kurmi OP
- La Vecchia C
- Lahariya C
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- Lee YH
- Lenzi J
- Levi M
- Li W
- Ligade VS
- Lim SS
- Liu G
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- Llanaj E
- Lo C-H
- Lukenze Tamuzi JJL
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- Machado VS
- Maghazachi AA
- Mahesh PA
- Mahmoud MA
- Mai TA
- Majeed A
- Makram OM
- Malhotra K
- Malik AA
- Malik I
- Mallhi TH
- Malta DC
- Mansournia MA
- Mantovani LG
- Martorell M
- Masoudi S
- Masoumi SZ
- Mathangasinghe Y
- Mathews E
- Mathioudakis AG
- Maugeri A
- Mayeli M
- Meles GG
- Mendes JJ
- Menezes RG
- Mestrovic T
- Michalek IM
- Micheletti Gomide Nogueira de SĂĄ AC
- Mihretie ET
- Mirfakhraie R
- Mirrakhimov EM
- Misganaw A
- Mohamadkhani A
- Mohamed Redwan EM
- Mohamed NS
- Mohammadi F
- Mohammadi S
- Mohammed S
- Mohammed S
- Mohan S
- Mohseni A
- Mokdad AH
- Momtazmanesh S
- Monasta L
- Moni MA
- Moniruzzaman M
- Moradi Y
- Morovatdar N
- Mostafavi E
- Mousavi P
- Mukoro GD
- Mulita A
- Mulu GB
- Murillo-Zamora E
- Musaigwa F
- Mustafa G
- Muthu S
- Nainu F
- Nangia V
- Natto ZS
- Navaraj P
- Nayak BP
- Nazri-Panjaki A
- Negash H
- Nematollahi MH
- Nguyen DH
- Nguyen HQ
- Nguyen PT
- Nguyen VT
- Nhat Minh LH
- Niazi RK
- Nikolouzakis TK
- Nnyanzi LA
- Noreen M
- Nzoputam CI
- Nzoputam OJ
- Oancea B
- Oh I-H
- Okati-Aliabad H
- Okonji OC
- Okwute PG
- Olagunju AT
- Olatubi MI
- Oliaee RT
- Olufadewa II
- Ordak M
- Otstavnov N
- Owolabi MO
- Padubidri JR
- Pak A
- Pakzad R
- Palladino R
- Pana A
- Pantazopoulos I
- Papadopoulou P
- Pardhan S
- Parthasarathi A
- Pashaei A
- Patel J
- Pathan AR
- Patil S
- Paudel U
- Pawar S
- Pedersini P
- Pensato U
- Pereira DM
- Pereira J
- Pereira MO
- Pereira RB
- Peres MFP
- Perianayagam A
- Perna S
- Petcu I-R
- Pezeshki PS
- Pham HT
- Philip AK
- Piradov MA
- Podder I
- Podder V
- Poddighe D
- Qattea I
- Rad EM
- Radfar A
- Raee P
- Rafiei A
- Raggi A
- Rahim F
- Rahimi M
- Rahimi-Movaghar V
- Rahimifard M
- Rahman Dewan SM
- Rahman M
- Rahman MA
- Rahman MO
- Rahmani AM
- Rahmani M
- Rahmani S
- Rahmanian V
- Ramasubramani P
- Rancic N
- Rao IR
- Rashedi S
- Rashid AM
- Ravikumar N
- Rawaf S
- Rezaei N
- Rezaei N
- Rezaei N
- Rezaeian M
- Ribeiro D
- Rodrigues M
- Roever L
- Romero-RodrĂguez E
- Saad AMA
- Saddik B
- Sadeghian S
- Sady Prates EJ
- Saeed Al-Gheethi AA
- Saeed U
- Safary A
- Safdarian M
- Safi SZ
- Saghazadeh A
- Sagoe D
- Sahebkar A
- Sahoo H
- Sahraian MA
- Sajid MR
- Sakhamuri S
- Sakilah Adnani QE
- Sakshaug JW
- Saleh MA
- Salehi L
- Salehi S
- Samadzadeh S
- Samargandy S
- Samieefar N
- Samy AM
- Sanadgol N
- Sanaye PM
- Sanjeev RK
- Sawhney M
- Saya GK
- Schuermans A
- Senthilkumaran S
- Sepanlou SG
- Sethi Y
- Shafie M
- Shah H
- Shahid I
- Shahid S
- Shaikh MA
- Sharfaei S
- Sharif-Askari FS
- Sharif-Askari NS
- Sharma M
- Shayan M
- Shehata HS
- Sheikh A
- Shetty JK
- Shin JI
- Shirkoohi R
- Shitaye NA
- Shivakumar KM
- Shivarov V
- Shobeiri P
- Siabani S
- Sibhat MM
- Siddig EE
- Simpson CR
- Sinaei E
- Singh H
- Singh I
- Singh JA
- Singh P
- Singh S
- Siraj MS
- Solanki R
- Solikhah S
- Solomon Y
- Soltani-Zangbar MS
- Sun J
- Swamy SN
- Szeto MD
- Tabarés-Seisdedos R
- Tabatabaei SM
- Tabish M
- Tahbaz SV
- Taheri E
- Tahvildari A
- Talaat IM
- Tam L-S
- Tan K-K
- Tat NY
- Tavasol A
- Temsah M-H
- Thangaraju P
- Tharwat S
- Thu Le TT
- Tibebu NS
- Tillawi T
- Tiruye TY
- Tiyuri A
- Tovani-Palone MR
- Tripathi M
- Tsegay GM
- Tualeka AR
- Ty SS
- Ubah CS
- Ullah S
- Ullah S
- Umair M
- Umakanthan S
- Upadhyay E
- Ur Rahman MH
- Vahabi SM
- Vaishali K
- Vaithinathan AG
- Valizadeh R
- Varthya SB
- Vasankari TJ
- Venketasubramanian N
- Vera Ticoalu JH
- Verras G-I
- Villafañe JH
- Vlassov V
- Vo DC
- Wahbi Atout MM
- Waheed Y
- Waris A
- Welegebrial BG
- Westerman R
- Wickramasinghe DP
- Wickramasinghe ND
- Willekens B
- Woldegeorgis BZ
- Woldemariam M
- Wu D
- Xiao H
- Xing Y
- Yada DY
- Yahya G
- Yang L
- Yazdanpanah F
- Yon DK
- Yonemoto N
- You Y
- Zahir M
- Zaidi SS
- Zangiabadian M
- Zare I
- Zeineddine MA
- Zemedikun DT
- Zeru NG
- Zhang C
- Zhao H
- Zhong C
- ZieliĆska M
- Zoladl M
- Zumla A
- Publication venue
- Elsevier BV
- Publication date
- 04/09/2023
- Field of study
Background The causes for immune-mediated inflammatory diseases (IMIDs) are diverse and the incidence trends of IMIDs from specific causes are rarely studied. The study aims to investigate the pattern and trend of IMIDs from 1990 to 2019. Methods We collected detailed information on six major causes of IMIDs, including asthma, inflammatory bowel disease, multiple sclerosis, rheumatoid arthritis, psoriasis, and atopic dermatitis, between 1990 and 2019, derived from the Global Burden of Disease study in 2019. The average annual percent change (AAPC) in number of incidents and age standardized incidence rate (ASR) on IMIDs, by sex, age, region, and causes, were calculated to quantify the temporal trends. Findings In 2019, rheumatoid arthritis, atopic dermatitis, asthma, multiple sclerosis, psoriasis, inflammatory bowel disease accounted 1.59%, 36.17%, 54.71%, 0.09%, 6.84%, 0.60% of overall new IMIDs cases, respectively. The ASR of IMIDs showed substantial regional and global variation with the highest in High SDI region, High-income North America, and United States of America. Throughout human lifespan, the age distribution of incident cases from six IMIDs was quite different. Globally, incident cases of IMIDs increased with an AAPC of 0.68 and the ASR decreased with an AAPC of â0.34 from 1990 to 2019. The incident cases increased across six IMIDs, the ASR of rheumatoid arthritis increased (0.21, 95% CI 0.18, 0.25), while the ASR of asthma (AAPC = â0.41), inflammatory bowel disease (AAPC = â0.72), multiple sclerosis (AAPC = â0.26), psoriasis (AAPC = â0.77), and atopic dermatitis (AAPC = â0.15) decreased. The ASR of overall and six individual IMID increased with SDI at regional and global level. Countries with higher ASR in 1990 experienced a more rapid decrease in ASR. Interpretation The incidence patterns of IMIDs varied considerably across the world. Innovative prevention and integrative management strategy are urgently needed to mitigate the increasing ASR of rheumatoid arthritis and upsurging new cases of other five IMIDs, respectively. Funding The Global Burden of Disease Study is funded by the Bill and Melinda Gates Foundation. The project funded by Scientific Research Fund of Sichuan Academy of Medical Sciences & Sichuan Provincial People's Hospital (2022QN38)
Research of Influence Preliminary Water Compression to the Processes of Methane-Containing Gas Mixtures Hydrate Formation
- Author
- Publication venue
- 'IOP Publishing'
- Publication date
- Field of study