Counting Carbon: The Politics of Carbon Footprints and Climate Governance from the Individual to the Global

This article considers carbon footprints as a form of climate governance. Drawing on science studies to consider the contingent nature of calculative devices and governmentality studies to examine the intrinsic relationship between how problems are framed and remedied, this article advances two arguments. First, it argues that efforts to define and deploy carbon footprints contributed to a conceptual shift in emissions accounting, from a narrower metric focused on emissions from fossil fuel and electricity use—Carbon Footprint 1.0—to a more expansive metric that includes emissions embodied in consumption and trade—Carbon Footprint 2.0. Second, this article argues that these approaches to carbon footprints at the individual level have intersected with broader discussions about allocating emissions responsibilities and examining mitigation strategies at the national and international levels, offering alternative grounds for assigning responsibility for climate-change mitigation and expanding the range of policy options available for addressing emissions

A Rooftop Revolution? A Multidisciplinary Analysis of State-Level Residential Solar Programs in New Jersey and Massachusetts

The success of alternative energy policies is usually measured in terms of energy capacity. By this metric, state-level policies to promote solar installations in New Jersey and Massachusetts have been a success. To fully evaluate these policies, however, it is necessary to consider how these policy programs are structured and funded, who participates in these programs, and the complete life-cycle consequences of \u27clean\u27 energy technologies. This paper focuses specifically on residential solar installations, which represent more than half of the total U.S. rooftop solar capacity potential. It takes a multidisciplinary approach that draws on policy analysis, spatial and demographic analyses, and life-cycle assessment. The analyses reveal three key conclusions: First, state-level policies have shifted from subsidies for solar installations to incentive-based support based on system performance, which have reduced the payback period for residential solar to less than ten years and have contributed to the growth of third-party leasing companies. Second, communities with low median income and/or a high percentage of non-white residents generally remain at lower than expected levels of participation. Third, while residential solar installations significantly offset greenhouse gas emissions and compounds that harm human respiratory health after 18 months, switching to photovoltaic panels generates a net increase in the production of ecotoxic chemicals. Drawing on these observations we recommend policy changes to encourage broader geographic and demographic participation, to recognize the importance of solar leasing companies and landlords, and to promote the use of solar panels with lower environmental impacts across the lifecycle

Writing in Britain and Ireland, c. 400 to c. 800

No abstract available

Playing Position is Associated with Injury Incidence Rate in Male Academy Soccer Players

Context: It is unclear whether playing position influences injury in male academy soccer players (ASP). Objective: To determine if playing position is associated with injury in ASP. Design: Descriptive Epidemiology Study. Setting: English, Spanish, Uruguayan and Brazilian soccer academies. Participants: 369 ASP from Under 14 (U14) to U23 age groups, classified as ‘post-peak height velocity' using maturity offset, and grouped as goalkeepers (GK), lateral defenders (LD), central defenders (CD), lateral midfielders (LM), central midfielders (CM) and forwards (FWD). Additional analysis compared central (CENT) with lateral/forward (LAT/FWD) positions. Main Outcome Measures: Injuries were recorded prospectively over one season. Injury prevalence proportion (IPP), days missed and injury incidence rate (IIR, injuries per 1000 training/match hours, n=116) were analysed according to playing position. Results: No association with playing position was observed for any injury type/location regarding IPP (P≥0.089) or days missed (P≥0.235). The IIR was higher in CD than LD for general (9.30 vs. 4.18 injuries/1000h, P=0.009), soft-tissue (5.14 vs. 1.95 injuries/1000h, P=0.026) and ligament/tendon injuries (2.69 vs. 0.56 injuries/1000h, P=0.040). Regarding CENT vs. LAT/FWD, there were no associations with IPP (P≥0.051) or days missed (P≥0.083), but general IIR was greater in CENT than LAT/FWD (8.67 vs. 6.12 injuries/1000h, P=0.047). Conclusions: ASP playing position was not associated with IPP or days missed but the higher general, soft-tissue and ligament/tendon IIR in CD suggests this position warrants specific attention regarding injury prevention strategies. These novel findings highlight the importance of including training/match exposure when investigating the influence of playing position on injury in ASP

Lupus-related single nucleotide polymorphisms and risk of diffuse large B-cell lymphoma

Objective: Determinants of the increased risk of diffuse large B-cell lymphoma (DLBCL) in SLE are unclear. Using data from a recent lymphoma genome-wide association study (GWAS), we assessed whether certain lupus-related single nucleotide polymorphisms (SNPs) were also associated with DLBCL. Methods: GWAS data on European Caucasians from the International Lymphoma Epidemiology Consortium (InterLymph) provided a total of 3857 DLBCL cases and 7666 general-population controls. Data were pooled in a random-effects meta-analysis. Results: Among the 28 SLE-related SNPs investigated, the two most convincingly associated with risk of DLBCL included the CD40 SLE risk allele rs4810485 on chromosome 20q13 (OR per risk allele=1.09, 95% CI 1.02 to 1.16, p=0.0134), and the HLA SLE risk allele rs1270942 on chromosome 6p21.33 (OR per risk allele=1.17, 95% CI 1.01 to 1.36, p=0.0362). Of additional possible interest were rs2205960 and rs12537284. The rs2205960 SNP, related to a cytokine of the tumour necrosis factor superfamily TNFSF4, was associated with an OR per risk allele of 1.07, 95% CI 1.00 to 1.16, p=0.0549. The OR for the rs12537284 (chromosome 7q32, IRF5 gene) risk allele was 1.08, 95% CI 0.99 to 1.18, p=0.0765. Conclusions: These data suggest several plausible genetic links between DLBCL and SLE