51 research outputs found
A922 Sequential measurement of 1 hour creatinine clearance (1-CRCL) in critically ill patients at risk of acute kidney injury (AKI)
- Author
- Abbott TE
- Abdallah RI
- Abdel-Aal IR
- Abdelmonem SA
- Abdo WF
- Abellan AN
- Abellán AN
- Abellán AN
- Abellán AN
- Abrams ST
- Ackerley C
- Acuña M
- Adda M
- Adhikari NK
- Adriano G
- Adrie C
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- Affatato R
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- Aguirregabyria M
- Ahmadnia E
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- Ahn MY
- Aitken LM
- Akalaev R
- Akbarzadeh A
- Akcan-Arikan A
- Akhlagh SH
- Akhtar N
- AKI Research Group
- Akiduki N
- Akin S
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- Knight LD
- Ko JI
- Ko SB
- Kobayashi A
- Koco E
- Kodate A
- Kodate A
- Kodate A
- Kokkini S
- Kolnikova I
- Komuro T
- Kondili E
- Kongpolprom N
- Kooner G
- Kostalas M
- Kosuk ME
- Kotsopoulos A
- Kou PS
- Kouatchet A
- Kovacikova L
- Kox M
- Kraegpoeth A
- Krenn CG
- Krishna B
- Kristof J
- Kruger A
- Kruger A
- Krüger A
- Ku NS
- Ku NS
- Kubik M
- Kubota K
- Kuebler WM
- Kulaga EV
- Kulkarni AP
- Kumari BK
- Kumbamu A
- Kunze-Szikszay N
- Kurmukov IA
- Kurth T
- Kurtz P
- Kurtz P
- Kwon HM
- Kwon HM
- Kwon WY
- Kwon WY
- Kwon WY
- Kye YC
- Kyle UG
- Kyle UG
- Labaune MA
- Labra C
- Lacerda P
- Laerkner E
- Lafaurie M
- Lafuente E
- Lagonidis D
- Lagos R
- Lahmer T
- Lai CC
- Lai CH
- Lake K
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- Lapteva K
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- Latini R
- Latini R
- Latini R
- Latorre J
- Lattuada M
- Lattuada M
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- Laurent A
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- Le Brocque R
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- Lebiedz P
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- Lee EY
- Lee JH
- Lee S
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- Lee SJ
- Lee WY
- Lee YJ
- Lee YJ
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- Legrand M
- Legrand M
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- Leitao AL
- Leite RT
- Leonard H
- Leone M
- Leone M
- Lerma FA
- Lesmes SP
- Lesmes SP
- Lesmes SP
- Lesmes SP
- Lesmes SP
- Lesmes SP
- Letizia T
- Levrat Q
- Levy B
- León JL
- León JP
- León JP
- León JP
- Lheureux O
- Lheureux O
- Lheureux O
- Lheureux O
- Li R
- Li SH
- Liao X
- Lichtenstein D
- Lim TH
- Limuti R
- Lin KC
- Lin KC
- Lin KC
- Lin KC
- Lin KL
- Lin PH
- Lin SC
- Lin SC
- Lin SC
- Lin SC
- Lin WC
- Lin WC
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- Lischinsky A
- Lissonde F
- Lissonde F
- Litchfield K
- Liu CP
- Liu CP
- Liu CP
- Liu CP
- Liu D
- Llaurado-Serra M
- Llorente B
- Llorente MÁ
- Lobato MS
- Lobo-Civico A
- Loizou C
- Lombardo MD
- Londoño JG
- Longani N
- Longhi L
- Lopes JA
- Lopez GD
- Lopez R
- Lopez-Caler C
- Lopez-Gude MJ
- Lorini L
- Lortat-Jacob B
- Lortat-Jacob B
- Lotay R
- Loudet CI
- Louf-Durier A
- Louf-Durier A
- Louis B
- Lourido CM
- Lozano JA
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- Lucendo AP
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- Lucinio NM
- Luini M
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- Lukaszewicz AC
- Luna RR
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- Lusk J
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- Lázaro AS
- López AR
- López L
- López MA
- Macfarlane B
- MacKay A
- MacKay A
- Mackey G
- Mackey G
- MacPhee I
- Macrì M
- Madueño VP
- Maekawa K
- Maekawa K
- Maekawa K
- Maekawa K
- Maertens B
- Maggiorini M
- Maghsoudi B
- Magnanimi E
- Magnoni S
- Magret M
- Mainardi JL
- Maistry N
- Maitra S
- Makiko S
- Malagurski B
- Malek F
- Malo LR
- Maly M
- Malyshev A
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- Mancebo J
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- Mancino A
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- Marcos-Zambrano LJ
- Mariani M
- Mariman A
- Marin-Mateos H
- Marine-Vidal J
- Mariotte E
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- Marmanidou K
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- Martinez-Reyes L
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- Martínez F
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- Martínez-Carmona JF
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- Melo N
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- Mendonca AV
- Mendsaikhan N
- Menendez R
- Mengelle C
- Mercat A
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- Mergulhão PJ
- Merino-Vega D
- Messenger D
- Mezidi M
- Mezidi M
- Michel L
- Midwinter MJ
- Mignot T
- Miguelena PR
- Mihara Y
- Millar M
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- Mimoz O
- Min A
- Min HJ
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- Miranda F
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- Mistry M
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- Mitchell M
- Mitchell S
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- Mizugaki A
- Mizugaki A
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- Olaechea P
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- Philips BJ
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- Radu V
- Rai S
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- Rodríguez FG
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- Romero I
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- Spahn DR
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- Spijkstra JJ
- Spronk PE
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- Sprung CL
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- Stamm J
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- Stanić R
- Starczewska M
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- Stiphout C
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- Stocchetti N
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- Stretti F
- Struijs A
- Stubbs C
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- Stümpfle R
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- Su PL
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- Suchomel P
- Suh GJ
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- Suh JW
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- Sun WZ
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- Suzuki T
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- Sánchez B
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- Sánchez EC
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- Tabatabaei HR
- Tabei SH
- Taccone F
- Taccone FS
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- Taccone FS
- Taccone FS
- Taccone FS
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- Taggu A
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- Takaki S
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- Takeda M
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- Takei T
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- Tamura T
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- Tanaka S
- Tanaka S
- Tane N
- Tang KB
- Tang LK
- Tapia A
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- Tapponnier R
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- Temprano-Gómez I
- Tena SA
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- Terzi N
- Terzi N
- Terzi N
- Terzi N
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- Thabut G
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- Thompson E
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- Tirkkonen J
- Tirumala S
- Tjepkema-Cloostermans MC
- Tobar M
- Tofiño AL
- Toh CH
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- Tomas E
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- Tonetti T
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- Yılmaz G
- Yilmazer F
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- Yoshida T
- Yoshihiro S
- Yoshitake E
- You KM
- Youn AM
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- Yu KM
- Yumoto T
- Yuzawa J
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- Zabini D
- Zahorec M
- Zaien I
- Zakynthinos S
- Zanello M
- Zani D
- Zarantonello F
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- Zeroual N
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- Zhang J
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- Zijlstra F
- Zimmerman J
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- Zogheib E
- Zogheib E
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- Zýková I
- Álvarez JT
- Álvarez-Lerma F
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
Meeting abstrac
Pan-cancer analysis of whole genomes
- Author
- A. -L. rv
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- The ICGC/TCGA Pan-Cancer Analysis of Whole Genomes Consortium View Correspondence (jump link)
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- zt Kim
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- Ó. A. th
- Ø. sk
- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
Wage Rate: Is this Return to Education or Return to Physical Capability? Evidence from Rural India
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Using Nonlinear Tools to Evaluate Movement of Fragile Objects
- Author
- Publication venue
- 'Human Kinetics'
- Publication date
- Field of study
Cell-Based Therapy for Neural Disorders — Anticipating Challenges
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Exome sequencing identifies frequent inactivating mutations in BAP1, ARID1A and PBRM1 in intrahepatic cholangiocarcinomas.
- Author
- Alexandrescu St
- Anders Ra
- Argani P
- Capelli P
- de Braud F
- Dima S
- Fassan Matteo
- Gores Gj
- Guglielmi A
- Guthrie Vb
- Hruban Rh
- Jarnagin W
- Jiao Y
- Karchin R
- Kinzler Kw
- Klimstra D
- Lawlor Rt
- Lucas Dj
- Mafficini A
- Maitra A
- Niknafs N
- Offerhaus Gj
- Papadopoulos N
- Pawlik Tm
- Popescu I
- Roa Jc
- Roberts Lr
- Ruzzenente A
- Scarpa A
- Selaru Fm
- Simbolo M
- Streppel Mm
- Tortora G
- Velculescu Ve
- Vogelstein B
- Wood Ld
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2013
- Field of study
Exome sequencing identifies frequent inactivating mutations in BAP1, ARID1A and PBRM1 in intrahepatic cholangiocarcinomas
- Author
- Alexandrescu ST
- Anders RA
- Argani P
- Capelli P
- de Braud F
- Dima S
- Fassan M
- Gores GJ
- Guglielmi A
- Guthrie VB
- Hruban RH
- Jarnagin W
- Jiao Y
- Karchin R
- Kinzler KW
- Klimstra D
- Lawlor RT
- Lucas DJ
- Mafficini A
- Maitra A
- Niknafs N
- Offerhaus GJ
- Papadopoulos N
- Pawlik TM
- Popescu I
- Roa JC
- Roberts LR
- Ruzzenente A
- Scarpa A
- Selaru FM
- Simbolo M
- Streppel MM
- Tortora G
- Velculescu VE
- Vogelstein B
- Wood LD
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2013
- Field of study
Through exomic sequencing of 32 intrahepatic cholangiocarcinomas, we discovered frequent inactivating mutations in multiple chromatin-remodeling genes (including BAP1, ARID1A and PBRM1), and mutation in one of these genes occurred in almost half of the carcinomas sequenced. We also identified frequent mutations at previously reported hotspots in the IDH1 and IDH2 genes encoding metabolic enzymes in intrahepatic cholangiocarcinomas. In contrast, TP53 was the most frequently altered gene in a series of nine gallbladder carcinomas. These discoveries highlight the key role of dysregulated chromatin remodeling in intrahepatic cholangiocarcinomas.
Comment in
Genetics: genetics of biliary tract cancer. [Nat Rev Gastroenterol Hepatol. 2013
Old age pension and intergenerational living arrangements: a regression discontinuity design
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Multicomponent Analysis of the Pancreatic Adenocarcinoma Progression Model Using a Pancreatic Intraepithelial Neoplasia Tissue Microarray
- Author
- Abraham SC
- Abraham SC
- Abraham SC
- Anton RC
- Apple SK
- Argani P
- Argani P
- Barton CM
- Biankin AV
- Bouras T
- de Bolos C
- DiGiuseppe JA
- Fan D
- Fukushima N
- Gansauge S
- Gasco M
- Goggins M
- Greenlee RT
- Hahn SA
- Hassan R
- Hermeking H
- Hollingsworth MA
- Hruban RH
- Hruban RH
- Iacobuzio-Donahue C
- Iacobuzio-Donahue CA
- Kononen J
- Kornmann M
- Linder S
- Luttges J
- Maitra A
- Maitra A
- Makinen K
- Meerzaman D
- Ostergaard M
- Pandey P
- Raffeld M
- Redston MS
- Ruggeri B
- Ryu B
- Ryu B
- Scarpa A
- Schraml P
- Schutte M
- Torhorst J
- van Heek NT
- Wilentz RE
- Wilentz RE
- Wilentz RE
- Wilentz RE
- Yamano M
- Yonezawa S
- Zhang SY
- Publication venue
- 'Ovid Technologies (Wolters Kluwer Health)'
- Publication date
- Field of study
Plasma concentrations of neuroactive steroids before and after electroconvulsive therapy in major depression
- Author
- A Strohle
- Augusto Pasini
- C Corpechot
- C Schule
- CA Altar
- CM Florkowski
- Cornelius Schüle
- Daniela Eser
- DP Uzunov
- E Romeo
- E Romeo
- Elena Romeo
- F Padberg
- Flavia di Michele
- G d'Elia
- G d'Elia
- HA Sackeim
- HA Sackeim
- HW Folkerts
- J Prudic
- J Prudic
- JC Wu
- JN Crawley
- L Grunhaus
- LD Griffin
- M Hamilton
- MA Smith
- MS George
- Peter Zwanzger
- PG Janicak
- R Abrams
- R Abrams
- R Maitra
- R Rupprecht
- R Rupprecht
- Rainer Rupprecht
- RJ Prince
- RT Khisti
- SM Paul
- Thomas C Baghai
- V Uzunova
- Publication venue
- Nature Publishing Group
- Publication date
- 01/01/2005
- Field of study
There is evidence that both cerebrospinal fluid (CSF) and plasma concentrations of 3 alpha-reduced neuroactive steroids are decreased in major depressive disorder. Successful antidepressant pharmacotherapy, for example, with selective serotonin reuptake inhibitors (SSRIs), over several weeks is accompanied by an increase in CSF and plasma concentrations of these neuroactive steroids. However, no such increase has been observed during nonpharmacological treatments such as partial sleep deprivation or repetitive transcranial magnetic stimulation. In order to investigate whether concentration changes in neuroactive steroids are an important component of clinically effective antidepressant treatment, we examined plasma concentrations of the neuroactive steroids 3 alpha,5 alpha-tetrahydroprogesterone, 3 alpha, 5 beta-tetrahydroprogesterone, 3 beta, 5 alpha-tetrahydroprogesterone, and their precursors progesterone, 5 alpha-dihydroprogesterone, and 5 beta-dihydroprogesterone in 31 pharmacotherapy- resistant depressed in-patients before and after unilateral electroconvulsive therapy (ECT) as a monotherapy over 4 weeks. Samples were quantified for neuroactive steroids by means of a highly sensitive and specific combined gas chromatography/mass spectrometry analysis. In all, 51.6% of the patients were treatment responders. There was no influence of ECT on the plasma concentrations of any neuroactive steroid studied. Moreover, neuroactive steroid levels did not differ between treatment responders and nonresponders. Our study shows that changes in neuroactive steroid plasma levels are not a mandatory factor for successful antidepressant treatment by ECT. Thus, the previously observed changes in plasma concentrations of neuroactive steroids following treatment with antidepressants such as SSRIs more likely reflect distinct pharmacological properties of these compounds rather than clinical improvement
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