244 research outputs found
Effective Rheology of Bubbles Moving in a Capillary Tube
- Author
- Abelev B
- Adam J
- Adamova D
- Adare AM
- Aggarwal MM
- Agocs AG
- Agostinelli A
- Ahammed Z
- Ahmad N
- Ahn SU
- Akindinov A
- Alaniz EC
- Aleksandrov D
- Alessandro B
- Alici A
- Alkin A
- Alme J
- Alt T
- Altini V
- Altinpinar S
- Altsybeev I
- Andrei C
- Andronic A
- Anguelov V
- Anielski J
- Anson C
- Anticic T
- Antinori F
- Antonioli P
- Aphecetche L
- Appelshauser H
- Arbor N
- Arcelli S
- Arend A
- Armesto N
- Arnaldi R
- Aronsson T
- Arsene IC
- Arslandok M
- Asryan A
- Augustinus A
- Averbeck R
- Avina EA
- Awes TC
- Aysto J
- Azmi MD
- Bach M
- Badala A
- Baek YW
- Bailhache R
- Baillie OV
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- Balbastre GC
- Baldisseri A
- Baldit A
- Ban J
- Baral RC
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- Barile F
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- Barret V
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- Basile M
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- Baumann C
- Bearden IG
- Beck H
- Belikov I
- Bellini F
- Bellwied R
- Belmont-Moreno E
- Bencedi G
- Beole S
- Berceanu I
- Bercuci A
- Berdnikov Y
- Berenyi D
- Bergmann C
- Berzano D
- Betev L
- Bhasin A
- Bhati AK
- Bianchi L
- Bianchi N
- Bianchin C
- Bielcik J
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- Bjelogrlic S
- Blanco F
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- Blau D
- Blume C
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- Bock N
- Bogdanov A
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- Bogolyubsky M
- Boldizsar L
- Bombara M
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- Borel H
- Borissov A
- Bornec YL
- Bose S
- Bossu F
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- Bottger S
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- Braun-Munzinger P
- Bregant M
- Breitner T
- Browning TA
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- Brun R
- Bruna E
- Bruno GE
- Budnikov D
- Buesching H
- Bufalino S
- Bugaiev K
- Busch O
- Bustamante RTJ
- Buthelezi Z
- Caffarri D
- Cahuantzi MR
- Cai X
- Caines H
- Camerini P
- Carena F
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- Carminati F
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- Chawla I
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- Trzaska WH
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- Zimmermann A
- Zinovjev G
- Zoccarato Y
- Zynovyev M
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 16/03/2012
- Field of study
We calculate the average volumetric flux versus pressure drop of bubbles
moving in a single capillary tube with varying diameter, finding a square-root
relation from mapping the flow equations onto that of a driven overdamped
pendulum. The calculation is based on a derivation of the equation of motion of
a bubble train from considering the capillary forces and the entropy production
associated with the viscous flow. We also calculate the configurational
probability of the positions of the bubbles.Comment: 4 pages, 1 figur
Neonatal cerebrovascular autoregulation.
- Author
- A Tekes
- AC Larson
- AC Larson
- AH Beek van
- AM Watkins
- AN Massaro
- B Smith
- B Wang
- B Wang
- C Chiron
- C Hua
- CA Giller
- CA Lear
- CA Zamora
- Christopher J. Rhee
- CJ Rhee
- CJ Rhee
- Cristine Sortica da Costa
- CS Costa da
- CS Roy
- CW Kuzawa
- D Hori
- D Nusbaum
- DA Osborn
- E Dempsey
- F Tian
- F Wong
- FY Wong
- G Greisen
- GH Hahn
- GJ Bouma
- GV Varsos
- H Bassan
- H Lou
- H O’Leary
- H Skov
- H Versmold
- I Al-Aweel
- J Kaiser
- J Kaiser
- J Lee
- J Nichol
- J Perlman
- J Riera
- J Toro Del
- JA Howlett
- Jennifer K. Lee
- JJ Volpe
- JK Lee
- JK Lee
- JK Lee
- JK Lee
- JK Lee
- JK Lee
- JK Lee
- JK Lynch
- JK Votava-Smith
- JM Saavedra
- JR Kaiser
- JR Kaiser
- JS Soul
- JS Soul
- JS Wyatt
- JW Hamner
- JW Wladimiroff
- Ken M. Brady
- KM Brady
- KM Brady
- KM Brady
- KM Brady
- L Chalak
- L Steiner
- LB Rorke
- LM Foley
- LR Ment
- LR Ment
- M Carrasco
- M Czosnyka
- M Good
- M Jagersberg
- M Johnston
- M Kluckow
- M Kluckow
- M Laughon
- M Pesek
- M Tsuji
- M Zhao
- Marek Czosnyka
- MH Lightburn
- MJH Aries
- MS Vavilala
- O Pryds
- PM Lewis
- R Blaine Easley
- R Chavez-Valdez
- R Panerai
- R Panerai
- R Spinazzola
- R Steinmeier
- Report of a Joint Working Group of the British Association of Perinatal Medicine and the Research Unit of the Royal College of Physicians.
- S Cunningham
- S Helou
- S Mitra
- S Soleymani
- SP Yang
- T Alderliesten
- T Hegyi
- T Hegyi
- T Kusaka
- Topun Austin
- VJ Burton
- VR Eriksen
- VR Eriksen
- WM Armstead
- WM Armstead
- WM Armstead
- WM Armstead
- ZS Katusic
- Publication venue
- Pediatr Res
- Publication date
- 01/11/2018
- Field of study
Cerebrovascular pressure autoregulation is the physiologic mechanism that holds cerebral blood flow (CBF) relatively constant across changes in cerebral perfusion pressure (CPP). Cerebral vasoreactivity refers to the vasoconstriction and vasodilation that occur during fluctuations in arterial blood pressure (ABP) to maintain autoregulation. These are vital protective mechanisms of the brain. Impairments in pressure autoregulation increase the risk of brain injury and persistent neurologic disability. Autoregulation may be impaired during various neonatal disease states including prematurity, hypoxic-ischemic encephalopathy (HIE), intraventricular hemorrhage, congenital cardiac disease, and infants requiring extracorporeal membrane oxygenation (ECMO). Because infants are exquisitely sensitive to changes in cerebral blood flow (CBF), both hypoperfusion and hyperperfusion can cause significant neurologic injury. We will review neonatal pressure autoregulation and autoregulation monitoring techniques with a focus on brain protection. Current clinical therapies have failed to fully prevent permanent brain injuries in neonates. Adjuvant treatments that support and optimize autoregulation may improve neurologic outcomes
RNA interference approaches for treatment of HIV-1 infection
- Author
- A Fire
- A Khvorova
- A Michienzi
- A Ono
- A Prussia
- A Singh
- A Soejitno
- AT Das
- B Berkhout
- BL Levine
- BP Burke
- CA Didigu
- CB Wilen
- D Boden
- D Finzi
- D Grimm
- D Liu
- D Siolas
- D Stone
- D Vlachakis
- DA Maier
- DA Persons
- DB Kohn
- DF Purcell
- DH Kim
- DL DiGiusto
- DS Schwarz
- E Cave
- E Puerta-Fernandez
- E Song
- EB Schleifman
- EE Perez
- EJ Arts
- EM Westerhout
- F Boutimah
- F Buchholz
- F Christ
- F Wong-Staal
- G Hutter
- G Hutter
- GJ McIntyre
- GJ McIntyre
- GR Rettig
- H Ebina
- H Mitsuya
- H Nishitsuji
- H Siomi
- HJ Unwalla
- HP Bogerd
- HS Christensen
- I Sarkar
- IB Hogue
- J Anderson
- J Chung
- J Chung
- J Lunzen van
- J Nejepinska
- J Yuan
- J Zhou
- J Zhou
- J Zhou
- J Zhou
- JC Burnett
- JC Burnett
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- JJ Eekels
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- JJ Rossi
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- JK Wong
- JL Macpherson
- JL Umbach
- JM Jacque
- JN Leonard
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- John J Rossi
- JS Pan
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- K Suzuki
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- MA Kay
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- MA Martinez
- Maggie L Bobbin
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- MJ Buzon
- MJ Li
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- NM Snead
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- P Kumar
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- P Tebas
- PR Gorry
- PS Shah
- PV Maillard
- RJ Scarborough
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- RT Mitsuyasu
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- S Backes
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- S Shimizu
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- S Subramanya
- S Yang
- SA Yukl
- SD Rose
- SG Deeks
- SK Lee
- SK Singh
- SS Kim
- SW Cho
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- T Nagawa
- T Yamamoto
- T Zhang
- TA Karlsen
- TJ Cradick
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- TP Chendrimada
- TS Lau
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- VA Green
- VR Sanghvi
- W Hu
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- WW Xu
- X Zhang
- Y Kashiwakura
- Y Li
- Y Pan
- Y Takeuchi
- YH Ping
- YL Chiu
- YP Liu
- YP Liu
- YP Liu
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
HIV/AIDS is a chronic and debilitating disease that cannot be cured with current antiretroviral drugs. While combinatorial antiretroviral therapy (cART) can potently suppress HIV-1 replication and delay the onset of AIDS, viral mutagenesis often leads to viral escape from multiple drugs. In addition to the pharmacological agents that comprise cART drug cocktails, new biological therapeutics are reaching the clinic. These include gene-based therapies that utilize RNA interference (RNAi) to silence the expression of viral or host mRNA targets that are required for HIV-1 infection and/or replication. RNAi allows sequence-specific design to compensate for viral mutants and natural variants, thereby drastically expanding the number of therapeutic targets beyond the capabilities of cART. Recent advances in clinical and preclinical studies have demonstrated the promise of RNAi therapeutics, reinforcing the concept that RNAi-based agents might offer a safe, effective, and more durable approach for the treatment of HIV/AIDS. Nevertheless, there are challenges that must be overcome in order for RNAi therapeutics to reach their clinical potential. These include the refinement of strategies for delivery and to reduce the risk of mutational escape. In this review, we provide an overview of RNAi-based therapies for HIV-1, examine a variety of combinatorial RNAi strategies, and discuss approaches for ex vivo delivery and in vivo delivery
Epigenetic activities of flavonoids in the prevention and treatment of cancer
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Pan-cancer analysis of whole genomes
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- The ICGC/TCGA Pan-Cancer Analysis of Whole Genomes Consortium View Correspondence (jump link)
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- Ó. A. th
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- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
Measurement and interpretation of same-sign W boson pair production in association with two jets in pp collisions at s = 13 TeV with the ATLAS detector
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- Abidi SH
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- Abusleme Hoffman AC
- Acharya BS
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- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 01/04/2024
- Field of study
This paper presents the measurement of fducial and diferential cross sections for both the inclusive and electroweak production of a same-sign W-boson pair in association with two jets (W±W±jj) using 139 fb−1 of proton-proton collision data recorded at a centre-of-mass energy of √s = 13 TeV by the ATLAS detector at the Large Hadron Collider. The analysis is performed by selecting two same-charge leptons, electron or muon, and at least two jets with large invariant mass and a large rapidity diference. The measured fducial cross sections for electroweak and inclusive W±W±jj production are 2.92 ± 0.22 (stat.) ± 0.19 (syst.)fb and 3.38±0.22 (stat.)±0.19 (syst.)fb, respectively, in agreement with Standard Model predictions. The measurements are used to constrain anomalous quartic gauge couplings by extracting 95% confdence level intervals on dimension-8 operators. A search for doubly charged Higgs bosons H±± that are produced in vector-boson fusion processes and decay into a same-sign W boson pair is performed. The largest deviation from the Standard Model occurs for an H±± mass near 450 GeV, with a global signifcance of 2.5 standard deviations
Effect of ozone oxidative preconditioning in preventing early radiation-induced lung injury in rats
- Author
- Agrawal A
- Ajamieh HH
- Arsalane K
- B. Guven
- B.H. Bakkal
- Barber E
- Bette M
- Bhalla DK
- Bocci VA
- Chen H
- Coleman CN
- Connor AJ
- Eliakim R
- Epperly MW
- F.A. Gultekin
- Gauter-Fleckenstein B
- Gracy RW
- Gultekin FA
- Guven A
- Haagsman HP
- Hernandez F
- Kesik V
- Laskin DL
- Leon OS
- Lowry OH
- M. Can
- Mettler Jr FA
- Mustafa MG
- Peralta C
- Rabbani ZN
- Re L
- Riepe MW
- Riley PA
- Roberts RA
- Rodriguez ZZ
- Rubin P
- S. Bektas
- Shimbo T
- Silva RA
- Souza YM
- Sun Y
- Sunil VR
- U.O. Turkcu
- Vujaskovic Z
- Yamanel L
- Zamora ZB
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
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- Abtew YD
- Accrombessi MMK
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- Temsah M-H
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- Thiyagarajan A
- Ticoalu JHV
- Tigabu BM
- Tiyuri A
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- Tovani-Palone MR
- Tusa BS
- Ullah I
- Umer AA
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- Valdez PR
- Vart P
- Varthya SB
- Vaziri S
- Verma M
- Veroux M
- Vervoort D
- Vilchis-Tella VM
- Wagaye B
- Weldegebreal F
- Wickramasinghe ND
- Woldemariam M
- Wonde TE
- Wubetie GA
- Xu X
- Yari K
- Yazdanpanah F
- Yehualashet SS
- Yigit A
- Yigit V
- Yisihak E
- Yon DK
- Yonemoto N
- Young MF
- Yu C
- Yunusa I
- Zahir M
- Zaki L
- Zaman BA
- Zamora N
- Zare I
- Zareshahrabadi Z
- Zenebe GA
- Zhang Z-J
- Zheng P
- Zoeckler LZ
- Zoladl M
- Publication venue
- Elsevier
- Publication date
- 29/08/2023
- Field of study
Background
Anaemia is a major health problem worldwide. Global estimates of anaemia burden are crucial for developing appropriate interventions to meet current international targets for disease mitigation. We describe the prevalence, years lived with disability, and trends of anaemia and its underlying causes in 204 countries and territories.
Methods
We estimated population-level distributions of haemoglobin concentration by age and sex for each location from 1990 to 2021. We then calculated anaemia burden by severity and associated years lived with disability (YLDs). With data on prevalence of the causes of anaemia and associated cause-specific shifts in haemoglobin concentrations, we modelled the proportion of anaemia attributed to 37 underlying causes for all locations, years, and demographics in the Global Burden of Disease Study 2021.
Findings
In 2021, the global prevalence of anaemia across all ages was 24·3% (95% uncertainty interval [UI] 23·9–24·7), corresponding to 1·92 billion (1·89–1·95) prevalent cases, compared with a prevalence of 28·2% (27·8–28·5) and 1·50 billion (1·48–1·52) prevalent cases in 1990. Large variations were observed in anaemia burden by age, sex, and geography, with children younger than 5 years, women, and countries in sub-Saharan Africa and south Asia being particularly affected. Anaemia caused 52·0 million (35·1–75·1) YLDs in 2021, and the YLD rate due to anaemia declined with increasing Socio-demographic Index. The most common causes of anaemia YLDs in 2021 were dietary iron deficiency (cause-specific anaemia YLD rate per 100 000 population: 422·4 [95% UI 286·1–612·9]), haemoglobinopathies and haemolytic anaemias (89·0 [58·2–123·7]), and other neglected tropical diseases (36·3 [24·4–52·8]), collectively accounting for 84·7% (84·1–85·2) of anaemia YLDs.
Interpretation
Anaemia remains a substantial global health challenge, with persistent disparities according to age, sex, and geography. Estimates of cause-specific anaemia burden can be used to design locally relevant health interventions aimed at improving anaemia management and prevention.
Funding
Bill & Melinda Gates Foundation
Global, regional, and national incidence of six major immune-mediated inflammatory diseases: findings from the global burden of disease study 2019
- Author
- Abate MD
- Abbasi-Kangevari M
- Abbasi-Kangevari Z
- Abbasian M
- Abd-Allah F
- Abdelmasseh M
- Abdollahifar M-A
- Abdou Elmeligy OA
- Abdulah DM
- Abebe EC
- Abedi A
- Abedi V
- Abidi H
- Aboagye RG
- Abolhassani H
- Abuabara K
- Abyadeh M
- Addo IY
- Adeniji KN
- Adepoju AV
- Adesina MA
- Afarideh M
- Aghamiri S
- Agodi A
- Agrawal A
- Aguilera Arriagada CE
- Ahmad A
- Ahmad D
- Ahmad S
- Ahmad S
- Ahmadi A
- Ahmed A
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- Aithala JP
- Ajadi AA
- Ajami M
- Akbarzadeh-Khiavi M
- Al Mamun Sohag A
- Al-Worafi YM
- Alahdab F
- AlBataineh MT
- Alemi S
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- Alif SM
- Almazan JU
- Almustanyir S
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- Barker-Collo SL
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- Nnyanzi LA
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- Nzoputam CI
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- Okonji OC
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- Tan K-K
- Tat NY
- Tavasol A
- Temsah M-H
- Thangaraju P
- Tharwat S
- Thu Le TT
- Tibebu NS
- Tillawi T
- Tiruye TY
- Tiyuri A
- Tovani-Palone MR
- Tripathi M
- Tsegay GM
- Tualeka AR
- Ty SS
- Ubah CS
- Ullah S
- Ullah S
- Umair M
- Umakanthan S
- Upadhyay E
- Ur Rahman MH
- Vahabi SM
- Vaishali K
- Vaithinathan AG
- Valizadeh R
- Varthya SB
- Vasankari TJ
- Venketasubramanian N
- Vera Ticoalu JH
- Verras G-I
- Villafañe JH
- Vlassov V
- Vo DC
- Wahbi Atout MM
- Waheed Y
- Waris A
- Welegebrial BG
- Westerman R
- Wickramasinghe DP
- Wickramasinghe ND
- Willekens B
- Woldegeorgis BZ
- Woldemariam M
- Wu D
- Xiao H
- Xing Y
- Yada DY
- Yahya G
- Yang L
- Yazdanpanah F
- Yon DK
- Yonemoto N
- You Y
- Zahir M
- Zaidi SS
- Zangiabadian M
- Zare I
- Zeineddine MA
- Zemedikun DT
- Zeru NG
- Zhang C
- Zhao H
- Zhong C
- Zielińska M
- Zoladl M
- Zumla A
- Publication venue
- Elsevier BV
- Publication date
- 04/09/2023
- Field of study
Background The causes for immune-mediated inflammatory diseases (IMIDs) are diverse and the incidence trends of IMIDs from specific causes are rarely studied. The study aims to investigate the pattern and trend of IMIDs from 1990 to 2019. Methods We collected detailed information on six major causes of IMIDs, including asthma, inflammatory bowel disease, multiple sclerosis, rheumatoid arthritis, psoriasis, and atopic dermatitis, between 1990 and 2019, derived from the Global Burden of Disease study in 2019. The average annual percent change (AAPC) in number of incidents and age standardized incidence rate (ASR) on IMIDs, by sex, age, region, and causes, were calculated to quantify the temporal trends. Findings In 2019, rheumatoid arthritis, atopic dermatitis, asthma, multiple sclerosis, psoriasis, inflammatory bowel disease accounted 1.59%, 36.17%, 54.71%, 0.09%, 6.84%, 0.60% of overall new IMIDs cases, respectively. The ASR of IMIDs showed substantial regional and global variation with the highest in High SDI region, High-income North America, and United States of America. Throughout human lifespan, the age distribution of incident cases from six IMIDs was quite different. Globally, incident cases of IMIDs increased with an AAPC of 0.68 and the ASR decreased with an AAPC of −0.34 from 1990 to 2019. The incident cases increased across six IMIDs, the ASR of rheumatoid arthritis increased (0.21, 95% CI 0.18, 0.25), while the ASR of asthma (AAPC = −0.41), inflammatory bowel disease (AAPC = −0.72), multiple sclerosis (AAPC = −0.26), psoriasis (AAPC = −0.77), and atopic dermatitis (AAPC = −0.15) decreased. The ASR of overall and six individual IMID increased with SDI at regional and global level. Countries with higher ASR in 1990 experienced a more rapid decrease in ASR. Interpretation The incidence patterns of IMIDs varied considerably across the world. Innovative prevention and integrative management strategy are urgently needed to mitigate the increasing ASR of rheumatoid arthritis and upsurging new cases of other five IMIDs, respectively. Funding The Global Burden of Disease Study is funded by the Bill and Melinda Gates Foundation. The project funded by Scientific Research Fund of Sichuan Academy of Medical Sciences & Sichuan Provincial People's Hospital (2022QN38)
Search for dark photons in rare Z boson decays with the ATLAS detector
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- Aad G
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Abusleme Hoffman AC
- Acharya BS
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- Addison MJ
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- Zang J
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- Zeitnitz C
- Zeng H
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- Zenger DT
- Zenin O
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- Zhang Z
- Zhao H
- Zhao P
- Zhao T
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- Zhemchugov A
- Zheng K
- Zheng X
- Zheng Z
- Zhong D
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- Zhou Y
- Zhu CG
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- Zhu Y
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- Zhuang X
- Zhukov K
- Zhulanov V
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- American Physical Society
- Publication date
- 01/01/2023
- Field of study
A search for events with a dark photon produced in association with a dark Higgs boson via rare decays of the standard model
Z
boson is presented, using
139
fb
−
1
of
√
s
=
13
TeV
proton-proton collision data recorded by the ATLAS detector at the Large Hadron Collider. The dark boson decays into a pair of dark photons, and at least two of the three dark photons must each decay into a pair of electrons or muons, resulting in at least two same-flavor opposite-charge lepton pairs in the final state. The data are found to be consistent with the background prediction, and upper limits are set on the dark photon’s coupling to the dark Higgs boson times the kinetic mixing between the standard model photon and the dark photon,
α
D
ϵ
2
, in the dark photon mass range of [5, 40] GeV except for the
Υ
mass window [8.8, 11.1] GeV. This search explores new parameter space not previously excluded by other experiments
- …