Two-photon widths of the χ c0,2 states and helicity analysis for χ c2→γγ

Based on a data sample of 106×106 ψ ′ events collected with the BESIII detector, the decays ψ ′→γχ c0,2, χ c0,2→γγ are studied to determine the two-photon widths of the χ c0,2 states. The two-photon decay branching fractions are determined to be B(χ c0→γγ)=(2. 24±0.19±0.12±0.08)×10 -4 and B(χ c2→γγ)=(3.21±0.18±0. 17±0.13)×10 -4. From these, the two-photon widths are determined to be Γ γγ(χ c0)=(2. 33±0.20±0.13±0.17)keV, Γ γγ(χ c2)=(0.63±0.04±0. 04±0.04)keV, and R=Γ γγ(χ c2)/ Γ γγ(χ c0)=0.271±0. 029±0.013±0.027, where the uncertainties are statistical, systematic, and those from the PDG B(ψ ′→γχ c0,2) and Γ(χ c0,2) errors, respectively. The ratio of the two-photon widths for helicity λ=0 and helicity λ=2 components in the decay χ c2→γγ is measured for the first time to be f 0/2=Γγγλ= 0(χ c2)/Γγγλ=2(χ c2)=0. 00±0.02±0.02. © 2012 American Physical Society.published_or_final_versio

Search for a light exotic particle in J/psi radiative decays

Using a data sample containing 1.06x10^8 psi' events collected with the BESIII detector at the BEPCII electron-positron collider, we search for a light exotic particle X in the process psi' -> pi^+ pi^- J/psi, J/psi -> gamma X, X -> mu^+ mu^-. This light particle X could be a Higgs-like boson A^0, a spin-1 U boson, or a pseudoscalar sgoldstino particle. In this analysis, we find no evidence for any mu^+mu^- mass peak between the mass threshold and 3.0 GeV/c^2. We set 90%-confidence-level upper limits on the product-branching fractions for J/psi -> gamma A^0, A^0 -> mu^+ mu^- which range from 4x10^{-7} to 2.1x10^{-5}, depending on the mass of A^0, for M(A^0)<3.0 GeV/c^2. Only one event is seen in the mass region below 255 MeV/c^2 and this has a mu^+mu^- mass of 213.3 MeV/c^2 and the product branching fraction upper limit 5x10^{-7}.Comment: 7 pages, 3 figures, submitted to Physical Review

Determination of the number of J/ψ events with J/ψ → inclusive decays

postprin

Pan-cancer analysis of whole genomes

Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe

Study of the decay D0→K¯0π−e+νe

We report a study of the decay D0→K̄0π-e+νe based on a sample of 2.93 fb-1 e+e- annihilation data collected at the center-of-mass energy of 3.773 GeV with the BESIII detector at the BEPCII collider. The total branching fraction is determined to be B(D0→K̄0π-e+νe)=(1.434±0.029(stat.)±0.032(syst.))%, which is the most precise to date. According to a detailed analysis of the involved dynamics, we find this decay is dominated with the K∗(892)- contribution and present an improved measurement of its branching fraction to be B(D0→K∗(892)-e+νe)=(2.033±0.046(stat.)±0.047(syst.))%. We further access their hadronic form-factor ratios for the first time as rV=V(0)/A1(0)=1.46±0.07(stat.)±0.02(syst.) and r2=A2(0)/A1(0)=0.67±0.06(stat.)±0.01(syst.). In addition, we observe a significant K̄0π- S-wave component accounting for (5.51±0.97(stat.)±0.62(syst.))% of the total decay rate

Study of the decay D0→K¯0π−e+νe

We report a study of the decay D0→K̄0π-e+νe based on a sample of 2.93 fb-1 e+e- annihilation data collected at the center-of-mass energy of 3.773 GeV with the BESIII detector at the BEPCII collider. The total branching fraction is determined to be B(D0→K̄0π-e+νe)=(1.434±0.029(stat.)±0.032(syst.))%, which is the most precise to date. According to a detailed analysis of the involved dynamics, we find this decay is dominated with the K∗(892)- contribution and present an improved measurement of its branching fraction to be B(D0→K∗(892)-e+νe)=(2.033±0.046(stat.)±0.047(syst.))%. We further access their hadronic form-factor ratios for the first time as rV=V(0)/A1(0)=1.46±0.07(stat.)±0.02(syst.) and r2=A2(0)/A1(0)=0.67±0.06(stat.)±0.01(syst.). In addition, we observe a significant K̄0π- S-wave component accounting for (5.51±0.97(stat.)±0.62(syst.))% of the total decay rate