205 research outputs found
Competition between Magnetic and Structural Transition in CrN
- Author
- A. Filippetti
- A. Mavromaras
- A. Neckel
- B. M. Klein
- B. W. Roberts
- D. A. Papaconstantopoulos
- D. A. Papaconstantopoulos
- D. J. Singh
- D. M. Ceperley
- D. Vanderbilt
- E. G. Moroni
- F. Bernardini
- G. Linker
- H. F. George
- H. Nakagawa
- H. Sawada
- H. Shimizu
- H. Shimizu
- J. Chen
- J. D. Browne
- J. HĂ€glung
- J. P. Perdew
- K. Laasonen
- K. Suzuki
- K. Suzuki
- L. M. Corliss
- L. M. Corliss
- M. Nasr-Eddine
- N. Chetty
- N. Chetty
- P. J. Dismukes
- P. Subramanya Herle
- R. M. Ibberson
- S. G. Louie
- S. P. Lewis
- T. Hinomura
- V. P. Zhukov
- W. E. Pickett
- W. E. Pickett
- W. E. Pickett
- W. L. Roth
- Y. Tsuchiya
- Y. Tsuchiya
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 27/10/1998
- Field of study
CrN is observed to undergo a paramagnetic to antiferromagnetic transition
accompanied by a shear distortion from cubic NaCl-type to orthorhombic
structure. Our first-principle plane wave and ultrasoft pseudopotential
calculations confirm that the distorted antiferromagnetic phase with spin
configuration arranged in double ferromagnetic sheets along [110] is the most
stable. Antiferromagnetic ordering leads to a large depletion of states around
Fermi level, but it does not open a gap. Simultaneous occurence of structural
distortion and antiferromagnetic order is analyzed.Comment: 10 pages, 10 figure
Participation in Church or Religious Groups and its Association with Health: A National Study of Young Canadians
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Cellular therapies for treating pain associated with spinal cord injury
- Author
- A Brown
- A Buss
- A Buss
- A George
- A George
- A George
- A Hoke
- A Mackay-Sim
- A Menssen
- A Scuteri
- AA Kumar
- AJ Bayer
- AJ Obaya
- AP Winnie
- AR Blight
- AR Khalatbary
- AT Hama
- B Bhattacharya
- B Sahin
- B Zorner
- BG Livett
- BS Shah
- C Lima
- C Lima
- C Price
- C Sommer
- CJ Sherr
- CM Morin
- D Siniscalco
- D Turk
- D Usvald
- D Wolfe
- DD French
- DS Tian
- DS Tian
- E Buchser
- E Esposito
- E Sykova
- E Sykova
- EA Carswell
- EM Hagen
- F Feron
- G Moalem
- G Yiu
- GA Moviglia
- GA Moviglia
- GE Pickett
- GW Small
- GZ Ning
- H Blaschko
- H Huang
- H Kondo
- H Nishitani
- H Saberi
- H Wang
- HD Klimm
- HS Chhabra
- HY Yang
- I Cernak
- I Mody
- J Bickels
- J Henle
- J Liu
- J Sagen
- J Sagen
- J Sagen
- J Sagen
- J Sagen
- J Sagen
- J Scholz
- J Silver
- J Vaquero
- J Vaquero
- J Widenfalk
- JC Bes
- JG Boyd
- JG Boyd
- JG Boyd
- JG Hu
- JH Brock
- JL Bodmer
- JM Huard
- JS Bae
- JS Riddell
- JW Fawcett
- JY Lee
- K An
- K Kadoya
- K Le Blanc
- K Miki
- K Stenson
- KC Roet
- KR Byrnes
- Lawrence Leung
- LF Geffner
- LH Sekhon
- LM Ramer
- M Bommer
- M Dinomais
- M Eriksdotter Jonhagen
- M Miyazato
- M Rudelius
- M Schafers
- M Schafers
- M Yamamoto
- MD Kawaja
- MH Tuszynski
- MJ Eaton
- MJ Eaton
- MJ Eaton
- MM Daadi
- MP Jensen
- MS Beattie
- N Beyer Nardi
- N Keyvan-Fouladi
- NA Hillarp
- NA Kishk
- NP Lapointe
- O Dehlin
- O Dehlin
- P Boehm-Sturm
- P Lu
- P Sacerdote
- PE de Almeida
- PJ Cejas
- PJ Siddall
- PJ Siddall
- PJ Siddall
- PJ Siddall
- PM Fischer
- PN Rizek
- R Jancalek
- R Pal
- R Vallejo
- R Wagner
- RJ Franklin
- RJ Franklin
- RJ Swijnenburg
- RM Locksley
- RN Lemon
- S David
- S Di Giovanni
- S Frantz
- S Lu
- S Shamash
- S Yousefzadeh Chabok
- SH Yoon
- SP Cohen
- SP Wilson
- SR Winn
- SS Seyed Jafari
- T Kallur
- T Korhonen
- T Korhonen
- T Korhonen
- T Takami
- TA Ignatowski
- TE Ichim
- TE Ichim
- VI Shubayev
- VI Shubayev
- VM Gomez
- WB Cafferty
- WC Covey
- WC Covey
- X Jiang
- X Li
- X Wu
- XF Zhou
- XH Parsons
- XQ Shu
- Y Lazorthes
- Y Liu
- Y Liu
- Y Liu
- Y Xu
- YJ Gao
- YT Wang
- ZA Sobani
- Publication venue
- BioMed Central
- Publication date
- 01/01/2012
- Field of study
Spinal cord injury leads to immense disability and loss of quality of life in human with no satisfactory clinical cure. Cell-based or cell-related therapies have emerged as promising therapeutic potentials both in regeneration of spinal cord and mitigation of neuropathic pain due to spinal cord injury. This article reviews the various options and their latest developments with an update on their therapeutic potentials and clinical trialing
New Light Source (NLS) project: conceptual design report
- Author
- Aeppli G
- Allemann R
- Almond A
- Angal-Kalinin D
- Ashfold M
- Averbukh V
- Bagshaw C
- Barahona M
- Barker P
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- Baumberger F
- Beard C
- Beeby A
- Bisby R
- Blackburn E
- Bliss N
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- Booth P
- Bowler M
- Bressler C
- Brown W
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- Burge F
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- Chattapadhyay S
- Chergui M
- Christou C
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- Cogdell R
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- Couprie M -E
- Cramer L
- Currell F
- Davis I
- Diakun G
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- Dougan C
- Drescher M
- Dunne AM
- Dunning D
- Durr H
- Emma P
- Evans J
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- Falcone R
- Feldhaus J
- Fell B
- Ferenczi M
- Ferguson-Smith M
- Fernandez-Hernando J
- Fernig D
- Fielding HH
- Flavell W
- Fraser H
- Frasinski L J
- Froud C
- Gardiner J
- Gensch M
- George M
- Gerricke D
- Gleeson A
- Goddard P
- Gould I
- Goulden A
- Gregori G
- Griffiths S
- Hajdu J
- Hamilton B
- Han J -H
- Hanon M
- Hasnain S
- Hatton P
- Hawes C
- Herbert J
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- Heyes D
- Hirst G
- Holbourn M
- Holland D
- Humphries M
- Hunter C
- Irving M
- Ivanov M
- Jackson F
- Jamison S
- Jaroszynski D
- Johnson L
- Jones J
- Kadodwala M
- Kalinin A
- Kapanidis A
- Kaplan A
- Kay J
- Kimel A
- Klug D
- Kuske B
- Lammie D
- Lee RW
- Maia F
- Marangos J
- Marsh M
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- McKenzie J
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- McMahon H
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- McNeil B
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- Michette A
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- Molloy J
- Moss A
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- Nann T
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- O'Neill P
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- Owen H
- Parker T
- Parmigiani F
- Pattalwar S
- Perutz R
- Pickett C
- Pimblott S
- Poletto L
- Poole M
- Powis I
- Priebe G
- Quiney H
- Quinn F
- Radaelli P
- Raff J
- Rehm G
- Reiche S
- Rich P
- Riley D
- Rimmer B
- Robinson I
- Roper M
- Rose S
- Rossbach J
- Rowland J
- Sankar G
- Schlarb H
- Schroeder S
- Scrutton N
- Seddon E
- Segal D
- Shintake T
- Singleton J
- Smith A
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- Sokolowski-Tinten K
- Somekh M
- Springate E
- Stead T
- Stringer M
- Sutcliffe M
- Tallents G
- Taylor K
- Thompson N
- Thompson R
- Tisch J
- Towns-Andrews E
- Townsend D
- Towrie M
- Ueda K
- Underwood J
- van der Laan G
- van Grondelle R
- van Thor J
- Venkitaraman A
- von Haeften K
- Vrakking M
- Walker R
- Walmsley I
- Wann D
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- Wheelhouse A
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- Wishart J
- Wolf A
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- Yaliraki S
- Yates E
- Zeitler A
- Zepf M
- Zholents A
- Publication venue
- Publication date
- 01/01/2010
- Field of study
Impact of primary kidney disease on the effects of empagliflozin in patients with chronic kidney disease: secondary analyses of the EMPA-KIDNEY trial
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- Abat S.
- Abd Rahman R.
- Abdul Cader R.
- Abdul Hafidz M. I.
- Abdul Wahab M. Z.
- Abdul-Samad T.
- Abdullah N. K.
- Abe M.
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- Acheampong S.
- Achiri P.
- Acosta J. A.
- Adeleke A.
- Adell V.
- Adewuyi-Dalton R.
- Adnan N.
- Africano A.
- Agharazii M.
- Aguilar F.
- Aguilera A.
- Ahmad M.
- Ahmad M. K.
- Ahmad Miswan N.
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- Ahmad Rosdi H.
- Ahmed I.
- Ahmed S.
- Aiello J.
- Aitken A.
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- Aker S.
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- Akinfolarin A.
- Akram S.
- Al-Rabadi L.
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- Alberici F.
- Albert C.
- Aldrich L.
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- Alexander L.
- Alfaress S.
- Alhadj Ali M.
- Ali A.
- Alicic R.
- Aliu A.
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- Almeida J.
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- An Y.
- Andriaccio L.
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- Publication venue
- Publication date
- 01/01/2024
- Field of study
Background: The EMPA KIDNEY trial showed that empagliflozin reduced the risk of the primary composite outcome of kidney disease progression or cardiovascular death in patients with chronic kidney disease mainly through slowing progression. We aimed to assess how effects of empagliflozin might differ by primary kidney disease across its broad population. Methods: EMPA-KIDNEY, a randomised, controlled, phase 3 trial, was conducted at 241 centres in eight countries (Canada, China, Germany, Italy, Japan, Malaysia, the UK, and the USA). Patients were eligible if their estimated glomerular filtration rate (eGFR) was 20 to less than 45 mL/min per 1·73 m2, or 45 to less than 90 mL/min per 1·73 m2 with a urinary albumin-to-creatinine ratio (uACR) of 200 mg/g or higher at screening. They were randomly assigned (1:1) to 10 mg oral empagliflozin once daily or matching placebo. Effects on kidney disease progression (defined as a sustained â„40% eGFR decline from randomisation, end-stage kidney disease, a sustained eGFR below 10 mL/min per 1·73 m2, or death from kidney failure) were assessed using prespecified Cox models, and eGFR slope analyses used shared parameter models. Subgroup comparisons were performed by including relevant interaction terms in models. EMPA-KIDNEY is registered with ClinicalTrials.gov, NCT03594110. Findings: Between May 15, 2019, and April 16, 2021, 6609 participants were randomly assigned and followed up for a median of 2·0 years (IQR 1·5â2·4). Prespecified subgroupings by primary kidney disease included 2057 (31·1%) participants with diabetic kidney disease, 1669 (25·3%) with glomerular disease, 1445 (21·9%) with hypertensive or renovascular disease, and 1438 (21·8%) with other or unknown causes. Kidney disease progression occurred in 384 (11·6%) of 3304 patients in the empagliflozin group and 504 (15·2%) of 3305 patients in the placebo group (hazard ratio 0·71 [95% CI 0·62â0·81]), with no evidence that the relative effect size varied significantly by primary kidney disease (pheterogeneity=0·62). The between-group difference in chronic eGFR slopes (ie, from 2 months to final follow-up) was 1·37 mL/min per 1·73 m2 per year (95% CI 1·16â1·59), representing a 50% (42â58) reduction in the rate of chronic eGFR decline. This relative effect of empagliflozin on chronic eGFR slope was similar in analyses by different primary kidney diseases, including in explorations by type of glomerular disease and diabetes (p values for heterogeneity all >0·1). Interpretation: In a broad range of patients with chronic kidney disease at risk of progression, including a wide range of non-diabetic causes of chronic kidney disease, empagliflozin reduced risk of kidney disease progression. Relative effect sizes were broadly similar irrespective of the cause of primary kidney disease, suggesting that SGLT2 inhibitors should be part of a standard of care to minimise risk of kidney failure in chronic kidney disease. Funding: Boehringer Ingelheim, Eli Lilly, and UK Medical Research Council
Pan-cancer analysis of whole genomes
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- X. k Mbabaali
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- Xiang
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- xo Abeshouse
- xp Benz
- xp Edwards
- xr Ghori
- xs Gundem
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- xv Luxton
- xw McDermott
- xy Visakorpi
- xz Whitaker
- Y. -E. ot
- Y. -J. k
- Y. -J. xn
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- ya Al-Ahmadie
- Yachida
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- Yamaguchi T. N. k
- Yamamoto
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- z ah
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- Zapatka
- zd Gershenwald
- Zenklusen
- Zeps
- zf Ghossein
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang
- Zhang H. b
- Zhao
- Zheng
- Zhou
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- Zhu
- Zhu
- zi zj
- zm Jayaseelan
- Zou
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- zt Kim
- zw Lewis
- Ă. A. th
- Ă. sk
- Publication venue
- Publication date
- 11/12/2019
- Field of study
Cancer is driven by genetic change, and the advent of massively parallel sequencing has enabled systematic documentation of this variation at the whole-genome scale(1-3). Here we report the integrative analysis of 2,658 whole-cancer genomes and their matching normal tissues across 38 tumour types from the Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium of the International Cancer Genome Consortium (ICGC) and The Cancer Genome Atlas (TCGA). We describe the generation of the PCAWG resource, facilitated by international data sharing using compute clouds. On average, cancer genomes contained 4-5 driver mutations when combining coding and non-coding genomic elements; however, in around 5% of cases no drivers were identified, suggesting that cancer driver discovery is not yet complete. Chromothripsis, in which many clustered structural variants arise in a single catastrophic event, is frequently an early event in tumour evolution; in acral melanoma, for example, these events precede most somatic point mutations and affect several cancer-associated genes simultaneously. Cancers with abnormal telomere maintenance often originate from tissues with low replicative activity and show several mechanisms of preventing telomere attrition to critical levels. Common and rare germline variants affect patterns of somatic mutation, including point mutations, structural variants and somatic retrotransposition. A collection of papers from the PCAWG Consortium describes non-coding mutations that drive cancer beyond those in the TERT promoter(4); identifies new signatures of mutational processes that cause base substitutions, small insertions and deletions and structural variation(5,6); analyses timings and patterns of tumour evolution(7); describes the diverse transcriptional consequences of somatic mutation on splicing, expression levels, fusion genes and promoter activity(8,9); and evaluates a range of more-specialized features of cancer genomes(8,10-18).Peer reviewe
The impact of surgical delay on resectability of colorectal cancer: An international prospective cohort study
- Author
- Abadia M
- Abassy J
- Abate E
- Abbott Tom EF
- Abdelaal Samir A
- Abdeldayem H
- Abdelkabir M
- Abdelsamed A
- Abdulwahed E
- Abiyere H
- Abou Chaar MK
- Abukhalaf SA
- Abukhalaf Sadi
- Adamina Michel
- Ademuyiwa Adesoji
- Ademuyiwa Adesoji O
- Adeyeye A
- Adisa Adewale
- Afzal MF
- Agarwal Arnav
- Aggarwal M
- Agnes A
- Agostini C
- Agrawal A
- Agrawal N
- Agresta F
- Aguiar Junior S
- Ahmed A
- Ahmed K
- Ahmed Nafees R
- Ahmed Z
- Ahn JH
- Aime A
- Akhtar N
- Akkulak Murat
- Akrivou D
- Al Abdallah M
- Al Riyami S
- Al Sayed M
- Al-Masri M
- Al-Najjar H
- Al-Slaibi I
- Alaa S
- Alakaloko Felix
- Alameer Ehab
- Alamri Alex
- Alawneh F
- Albertsmeier M
- Albertsmeier Markus
- Alderson Derek
- Aldokali N
- Alemanno G
- Alfieri S
- Algallai M
- Ali AA
- Alkadiki Ghadah Z
- Almaadany Faraj S
- Almeida AC
- Almeida-Reis R
- Almezghwi H
- Almond Max
- Alrahawy M
- Alser Osaid
- Alshaar Muhammad
- Alshareea E
- Alshareef K
- Alshryda Sattar
- Alsoufi A
- Altamirano Riquoir C
- Altomare DF
- Altomi I
- Alurralde C
- Alvarez MR
- Alvi A
- Alwarfly S
- AlZAEDE S
- Alzeerelhouseini IAH
- Amaral MJ
- Amira G
- Amorim E
- Amrani L
- Anania G
- Anastasi A
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- Publication venue
- 'Royal College of Obstetricians & Gynaecologists (RCOG)'
- Publication date
- 01/06/2022
- Field of study
AIM: The SARS-CoV-2 pandemic has provided a unique opportunity to explore the impact of surgical delays on cancer resectability. This study aimed to compare resectability for colorectal cancer patients undergoing delayed versus non-delayed surgery. METHODS: This was an international prospective cohort study of consecutive colorectal cancer patients with a decision for curative surgery (January-April 2020). Surgical delay was defined as an operation taking place more than 4âweeks after treatment decision, in a patient who did not receive neoadjuvant therapy. A subgroup analysis explored the effects of delay in elective patients only. The impact of longer delays was explored in a sensitivity analysis. The primary outcome was complete resection, defined as curative resection with an R0 margin. RESULTS: Overall, 5453 patients from 304 hospitals in 47 countries were included, of whom 6.6% (358/5453) did not receive their planned operation. Of the 4304 operated patients without neoadjuvant therapy, 40.5% (1744/4304) were delayed beyond 4âweeks. Delayed patients were more likely to be older, men, more comorbid, have higher body mass index and have rectal cancer and early stage disease. Delayed patients had higher unadjusted rates of complete resection (93.7% vs. 91.9%, PÂ =Â 0.032) and lower rates of emergency surgery (4.5% vs. 22.5%, Pâ<â0.001). After adjustment, delay was not associated with a lower rate of complete resection (OR 1.18, 95% CI 0.90-1.55, PÂ =Â 0.224), which was consistent in elective patients only (OR 0.94, 95% CI 0.69-1.27, PÂ =Â 0.672). Longer delays were not associated with poorer outcomes. CONCLUSION: One in 15 colorectal cancer patients did not receive their planned operation during the first wave of COVID-19. Surgical delay did not appear to compromise resectability, raising the hypothesis that any reduction in long-term survival attributable to delays is likely to be due to micro-metastatic disease
Outcomes from elective colorectal cancer surgery during the SARS-CoV-2 pandemic
- Author
- 14 | COVIDSurg COllabOratIVe Tzovaras G. (General University Hospital of Larissa)
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- COVIDSurg COllabOratIVe | 15 Violante T. (IRCCS Azienda Ospedaliero â Universitaria di Bologna
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- ĂÄĂŒcĂŒ H.
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- Äoza I.
- Publication venue
- 'Wiley'
- Publication date
- 01/01/2020
- Field of study
This study aimed to describe the change in surgical practice and the impact of SARS-CoV-2 on mortality after surgical resection of colorectal cancer during the initial phases of the SARS-CoV-2 pandemic
Taming the tiger by the tail: modulation of DNA damage responses by telomeres
- Author
- Addinall SG
- Ahmed S
- Artandi SE
- Askree SH
- Aubert G
- Aylon Y
- Baldo V
- Barlow JH
- Baumann P
- Bertuch AA
- Bi X
- Bianchi A
- Blackburn EH
- Boerckel J
- Booth C
- Boulton SJ
- Bucholc M
- Capper R
- Carballo JA
- Caspi RR
- Celli GB
- Celli GB
- Cenci G
- Cesare AJ
- Chai W
- Chan SW
- Chang M
- Churikov D
- Ciapponi L
- Cooper JP
- Cui T
- d&rsquo
- D&rsquo
- David Lydall
- de Lange T
- Denchi EL
- Deng Y
- Deriano L
- Dimitrova N
- DuBois ML
- Dubrana K
- Enomoto S
- Fellerhoff B
- Ferguson TA
- Ferreira MG
- Fisher TS
- Foster SS
- Francia S
- Frank-Vaillant M
- Gao H
- Garvik B
- Gatbonton T
- George JA
- Grandin N
- Gravel S
- Greenall A
- Greider CW
- Griffith JD
- Hartwell LH
- Hector RE
- Hirano Y
- Hockemeyer D
- Hockemeyer D
- Hockemeyer D
- IJpma A
- Jeyapalan JC
- Jia X
- Kanaar R
- Kim HS
- Kim JS
- Laroche T
- Larrivee M
- Larrivee M
- Lazzaro F
- Lee JY
- Lee SE
- Lei M
- Li B
- Lingner J
- Lisby M
- Longhese MP
- Longhese MP
- Louis EJ
- Lundblad V
- Lydall D
- Lydall D
- Majka J
- Mantiero D
- Maringele L
- Maringele L
- Maringele L
- Maringele L
- Maser RS
- McEachern MJ
- Melnikova L
- Michelson RJ
- Miller KM
- Mimitou EP
- Morin I
- Muller HJ
- Murnane JP
- Myung K
- Naito T
- Nakada D
- Nautiyal S
- Nugent CI
- Nugent CI
- Oikemus SR
- Pardo B
- Petreaca RC
- Pickett HA
- Polakova S
- Polotnianka RM
- Proctor CJ
- Raices M
- Ranganathan V
- Raynard S
- Riha K
- Ritchie KB
- Ritchie KB
- Rog O
- Rog O
- Rong YS
- Sabourin M
- Sabourin M
- Sandell LL
- Schaetzlein S
- Shachar R
- Shore D
- Stellwagen AE
- Stewart SA
- Streilein JW
- Symington LS
- Takai H
- Tarsounas M
- Teixeira MT
- Teo SH
- Titen SW
- Tomita K
- Tomita K
- Tourand Y
- Usui T
- van Leeuwen F
- van Steensel B
- Verdun RE
- Verdun RE
- Verdun RE
- Vodenicharov MD
- Wang F
- Weinert TA
- Weinert TA
- Wotton D
- Wu D
- Wu L
- Wu Y
- Zhu Z
- Zubko MK
- Zubko MK
- Publication venue
- Nature Publishing Group
- Publication date
- Field of study
Telomeres are by definition stable and inert chromosome ends, whereas internal chromosome breaks are potent stimulators of the DNA damage response (DDR). Telomeres do not, as might be expected, exclude DDR proteins from chromosome ends but instead engage with many DDR proteins. However, the most powerful DDRs, those that might induce chromosome fusion or cell-cycle arrest, are inhibited at telomeres. In budding yeast, many DDR proteins that accumulate most rapidly at double strand breaks (DSBs), have important functions in physiological telomere maintenance, whereas DDR proteins that arrive later tend to have less important functions. Considerable diversity in telomere structure has evolved in different organisms and, perhaps reflecting this diversity, different DDR proteins seem to have distinct roles in telomere physiology in different organisms. Drawing principally on studies in simple model organisms such as budding yeast, in which many fundamental aspects of the DDR and telomere biology have been established; current views on how telomeres harness aspects of DDR pathways to maintain telomere stability and permit cell-cycle division are discussed
- âŠ