1,581 research outputs found
KRas4BG12C/D/PDE6δ Heterodimeric Molecular Complex: A Target Molecular Multicomplex for the Identification and Evaluation of Nontoxic Pharmacological Compounds for the Treatment of Pancreatic Cancer
- Author
- Publication venue
- 'IntechOpen'
- Publication date
- 19/08/2020
- Field of study
The search for new targeted therapies to improve the quality of life of patients with pancreatic cancer has taken about 30 years. Compounds that can inhibit the K-Ras4B oncoprotein signaling pathway have been sought. Taking into account that the interaction of KRas4B with PDE6δ is essential for its transport and subsequent activation in the plasma membrane, our working group identified and evaluated in vitro and in vivo small organic molecules that could act as molecular staples to stabilize the KRas4B/PDE6δ heterodimeric complex. From this group of molecules, 38 compounds with high interaction energies on the structure of the crystallized molecular complex were selected, indicating that they efficiently stabilized the molecular complex. In vitro evaluation of compounds called D14, C22, and C19 showed significant specific effects on the cell viability of pancreatic cancer cells (and not on normal cells), thus inducing death by apoptosis and significantly inhibiting the activation of the pathways, signaling AKT and ERK. In addition to these experimental findings, we were also able to detect that compounds D14 and C22 showed significant tumor growth inhibitory activity in pancreatic cancer cell-induced subcutaneous xenograft models
The Ascomycete Verticillium longisporum Is a Hybrid and a Plant Pathogen with an Expanded Host Range
- Author
- A Collins
- A Johansson
- AA Christen
- AC Hastie
- AC Hastie
- AG Spiers
- Anastasia Litvintseva
- BG Turgeon
- C Eynck
- C Stark
- CD Moon
- CD Moon
- CD Moon
- CE Caten
- CL Schardl
- CM Brasier
- CM Brasier
- CM Brasier
- CM Brasier
- CW Jackson
- D Posada
- DE Soltis
- DJ Barbara
- DL Joly
- DL Swofford
- E Clewes
- E Clewes
- F Ronquist
- G Newcombe
- GF Pegg
- GH Pethybridge
- H Kauserud
- H-F Tsai
- HH Flor
- I Masneuf
- J Beser
- J Heale
- J Sambrook
- JB Heale
- JE Puhalla
- JM Clarkson
- JW Taylor
- JW Taylor
- K O'Donnell
- K O'Donnell
- K Zeise
- K Zeise
- Krishna V. Subbarao
- KV Subbarao
- L Morin
- LA Steventon
- M Babadoost
- M Collado-Romero
- M Collado-Romero
- M Gardes
- M Staats
- MA Hernandez-Bello
- MA Selosse
- MA Typas
- ME Zolan
- MJ Hegarty
- MP Pantou
- MP Pantou
- O Paun
- P Gonthier
- Patrik Inderbitzin
- QM Qin
- R Bhat
- R Ingram
- R. Michael Davis
- Richard M. Bostock
- RL Metzenberg
- S Horiuchi
- T Usami
- T Usami
- TC Osborn
- TJ White
- VK Karapapa
- WA Man in't Veld
- X Lin
- X-F Ma
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
Hybridization plays a central role in plant evolution, but its overall importance in fungi is unknown. New plant pathogens are thought to arise by hybridization between formerly separated fungal species. Evolution of hybrid plant pathogens from non-pathogenic ancestors in the fungal-like protist Phytophthora has been demonstrated, but in fungi, the most important group of plant pathogens, there are few well-characterized examples of hybrids. We focused our attention on the hybrid and plant pathogen Verticillium longisporum, the causal agent of the Verticillium wilt disease in crucifer crops. In order to address questions related to the evolutionary origin of V. longisporum, we used phylogenetic analyses of seven nuclear loci and a dataset of 203 isolates of V. longisporum, V. dahliae and related species. We confirmed that V. longisporum was diploid, and originated three different times, involving four different lineages and three different parental species. All hybrids shared a common parent, species A1, that hybridized respectively with species D1, V. dahliae lineage D2 and V. dahliae lineage D3, to give rise to three different lineages of V. longisporum. Species A1 and species D1 constituted as yet unknown taxa. Verticillium longisporum likely originated recently, as each V. longisporum lineage was genetically homogenous, and comprised species A1 alleles that were identical across lineages
Global, regional, and national comparative risk assessment of 79 behavioural, environmental and occupational, and metabolic risks or clusters of risks, 1990-2015: a systematic analysis for the Global Burden of Disease Study 2015
- Author
- Aasvang GM
- Abajobir AA
- Abate KH
- Abbafati C
- Abbas KM
- Abd-Allah F
- Abdulle AM
- Abera SF
- Abraham B
- Abu-Raddad LJ
- Abyu GY
- Adebiyi AO
- Adedeji IA
- Ademi Z
- Adou AK
- Adsuar JC
- Afshin A
- Agardh EE
- Agarwal A
- Agrawal A
- Ajala ON
- Akinyemiju TF
- Al-Aly Z
- Alam K
- Alam NKM
- Aldhahri SF
- Aldridge RW
- Alemu ZA
- Alexander LT
- Ali R
- Alkerwi A
- Alla F
- Allebeck P
- Alsharif U
- Altirkawi KA
- Alvarez Martin E
- Alves Silveira DG
- Alvis-Guzman N
- Amare AT
- Amberbir A
- Amegah AK
- Amini H
- Ammar W
- Amrock SM
- Andersen HH
- Anderson BO
- Anderson HR
- Antonio CAT
- Anwar P
- Aquino Faraon EJ
- Arnlov J
- Artaman A
- Asayesh H
- Asghar RJ
- Assadi R
- Atique S
- Avokpaho EFGA
- Awasthi A
- Azzopardi P
- Bacha U
- Badawi A
- Baernighausen T
- Bahit MC
- Balakrishnan K
- Bans C
- Barac A
- Barber RM
- Barker-Collo SL
- Barquera S
- Barregard L
- Barrero LH
- Basu S
- Bazargan-Hejazi S
- Beardsley J
- Beatriz Heredia-Pi I
- Beatriz Lawrynowicz AE
- Bedi N
- Beghi E
- Bell ML
- Bello AK
- Bennett DA
- Bensenor IM
- Berhane A
- Bernabe E
- Betsu BD
- Beyene AS
- Bhala N
- Bhansali A
- Bhatt S
- Bhutta ZA
- Biadgilign S
- Bicer BK
- Bikbov B
- Biryukov S
- Bisanzio D
- Bjertness E
- Blore JD
- Borschmann R
- Boufous S
- Bourne RRA
- Brainin M
- Brauer M
- Brazinova A
- Breitborde NJK
- Brenner H
- Broday DM
- Brugha TS
- Brunekreef B
- Burnett R
- Butt ZA
- Cahill LE
- Caicedo AJP
- Calabria B
- Cardenas R
- Carmen Gomez-Cabrera M
- Carpenter D
- Casey DC
- Castaneda-Oquela CA
- Castillo Rivas J
- Catala-Lopez F
- Cercy K
- Chang J-C
- Charlson FJ
- Chiang PP-C
- Chibalabala M
- Chimed-Ochir O
- Chisumpa VH
- Chitheer AA
- Choi J-YJ
- Christensen H
- Christopher DJ
- Ciobanu LG
- Coates MM
- Cohen AJ
- Colquhoun SM
- Cooper LT
- Cooperrider K
- Cornaby L
- Cortinovis M
- Crump JA
- Cuevas-Nasu L
- Damasceno A
- Dandona L
- Dandona R
- Darby SC
- Dargan PI
- das Neves J
- Davis AC
- Davletov K
- De la Cruz-Gongora V
- De Leo D
- Defo BK
- Degenhardt L
- Del Gobbo LC
- del Pozo-Cruz B
- Dellavalle RP
- Deribew A
- Des Jarlais DC
- Dharmaratne SD
- Dhillon PK
- Diaz-Tome C
- Dicker D
- Dimbuene ZT
- Ding EL
- Dolores Sanchez-Nino M
- Dorsey ER
- Doyle KE
- Driscoll TR
- Duan L
- Dubey M
- Duncan BB
- Elyazar I
- Endries AY
- Ermakov SP
- Erskine HE
- Eshrati B
- Estanislao Castro R
- Esteghamati A
- Estep K
- Factors GBDR
- Fahimi S
- Farid TA
- Faro A
- Farvid MS
- Farzadfar F
- Feigin VL
- Fereshtehnejad S-M
- Fernandes JG
- Ferrari AJ
- Filipa de Castro E
- Fischer F
- Fitchett JRA
- Fleming T
- Foigt N
- Foreman K
- Forouzanfar MH
- Fowkes FGR
- Francisco Hernandez-Llanes N
- Franklin RC
- Frostad JJ
- Fuerst T
- Fullman N
- Futran ND
- Gakidou E
- Garcia-Basteiro AL
- Gebrehiwot TT
- Gebremedhin AT
- Geleijnse JM
- Gessner BD
- Gething PW
- Giref AZ
- Giroud M
- Gishu MD
- Godwin WW
- Goenka S
- Gomez-Dantes H
- Gona P
- Goodridge A
- Gopalani SV
- Gotay CC
- Goto A
- Gouda HN
- Griswold M
- Gugnani HC
- Guillemin F
- Guo Y
- Gupta R
- Gupta R
- Gutierrez RA
- Haagsma JA
- Hafezi-Nejad N
- Haile D
- Hailu GB
- Halasa YA
- Hamadeh RR
- Hamidi S
- Handal AJ
- Hankey GJ
- Hao Y
- Harb HL
- Harikrishnan S
- Hassanvand MS
- Hassen TA
- Havmoeller R
- Heydarpour P
- Hoek HW
- Hoffman HJ
- Horino M
- Horita N
- Hosgood HD
- Hoy DG
- Hsairi M
- Htet AS
- Hu G
- Huang JJ
- Husseini A
- Hutchings SJ
- Huybrechts I
- Iburg KM
- Idrisov BT
- Ileanu BV
- Inoue M
- Jacobs TA
- Jacobsen KH
- Jahanmehr N
- Jakovljevic MB
- Jansen HAFM
- Jassal SK
- Javanbakht M
- Jayatilleke AU
- Jee SH
- Jeemon P
- Jha V
- Jiang Y
- Jibat T
- Jin Y
- Johnson CO
- Jonas JB
- Kabir Z
- Kalkonde Y
- Kamal R
- Kan H
- Karch A
- Karema CK
- Karimkhani C
- Kasaeian A
- Kaul A
- Kawakami N
- Kazi DS
- Keiyoro PN
- Kemp AH
- Kengne AP
- Keren A
- Kesavachandran CN
- Khader YS
- Khan AR
- Khan EA
- Khan G
- Khang Y-H
- Khatibzadeh S
- Khera S
- Khoja TAM
- Khubchandani J
- Kiadaliri AA
- Kieling C
- Kim C-I
- Kim D
- Kimokoti RW
- Kinfu Y
- Kissoon N
- Kivipelto M
- Knibbs LD
- Kokubo Y
- Kopec JA
- Koul PA
- Koyanagi A
- Kravchenko M
- Kromhout H
- Krueger H
- Ku T
- Kuchenbecker RS
- Kuipers EJ
- Kumar GA
- Kwan GF
- Kyu HH
- Lal DK
- Lalloo R
- Lallukka T
- Lan Q
- Landon N
- Larson HJ
- Larsson A
- Latif AA
- Leasher JL
- Leigh J
- Leung J
- Levi M
- Levy TS
- Li X
- Li Y
- Liang J
- Liang X
- Lim SS
- Liu PY
- Liu S
- Lloyd BK
- Logroscino G
- Lopez AD
- Lotufo PA
- Lozano R
- Lunevicius R
- Maclntyre M
- Mahdavi M
- Mahesh PA
- Majdan M
- Majeed A
- Malekzadeh R
- Malta DC
- Manamo WAA
- Mapoma CC
- Marcenes W
- Marczak L
- Maria Haro J
- Martin RV
- Martinez-Raga J
- Masiye F
- Matsushita K
- Matzopoulos R
- Mayosi BM
- McGrath JJ
- McKee M
- Meaney PA
- Medina C
- Mehari A
- Mekonnen AB
- Melaku YA
- Memish ZA
- Mena-Rodriguez F
- Mendoza W
- Mensah GA
- Mensink GBM
- Meretoja A
- Meretoja TJ
- Mesfin YM
- Mhimbira FA
- Miller TR
- Mills EJ
- Mirarefin M
- Misganaw A
- Mock CN
- Mohammadi A
- Mohammed S
- Mokdad AH
- Mola GLD
- Monasta L
- Montanez Hernandez JC
- Montico M
- Moradi-Lakeh M
- Morawska L
- Mori R
- Mozaffarian D
- Mueller UO
- Mullany E
- Mumford JE
- Murray CJL
- Murthy GVS
- Nachega JB
- Naghavi M
- Naheed A
- Nangia V
- Nassiri N
- Neal B
- Newton JN
- Ng M
- Nguyen QL
- Nisar MI
- Norheim OF
- Norman RE
- Norrving B
- Nyakarahuka L
- Obermeyer CM
- Ogbo FA
- Oh I-H
- Oladimeji O
- Olivares PR
- Olsen H
- Olusanya BO
- Olusanya JO
- Opio JN
- Oren E
- Orozco R
- Ortiz A
- Ota E
- Pana A
- Park E-K
- Parry CD
- Parsaeian M
- Patel T
- Patil ST
- Patten SB
- Patton GC
- Pearce N
- Pereira DM
- Perico N
- Pesudovs K
- Pete PMN
- Petzold M
- Phillips MR
- Piel FB
- Pillay JD
- Plass D
- Polinder S
- Pond CD
- Pope CA
- Pope D
- Popova S
- Poulton RG
- Pourmalek F
- Prasad NM
- Qorbani M
- Quintanilla BPA
- Rabiee RHS
- Radfar A
- Rafay A
- Rahimi-Movaghar V
- Rahman M
- Rahman MHU
- Rahman SU
- Rai RK
- Rajsic S
- Raju M
- Ram U
- Rana SM
- Ranganathan K
- Rao P
- Razo Garcia CA
- Refaat AH
- Rehm CD
- Rehm J
- Reinig N
- Reitsma MB
- Remuzzi G
- Resnikoff S
- Ribeiro AL
- Ricardo Campos-Nonato I
- Rivera JA
- Rodriguez A
- Rodriguez-Ramirez S
- Rojas-Rueda D
- Rolm HS
- Roman Y
- Ronfani L
- Roshandel G
- Roth GA
- Rothenbacher D
- Roy A
- Saleh MM
- Salomon JA
- Sanabria JR
- Sanchez-Pimienta TG
- Sandar L
- Santomauro DF
- Santos IS
- Sarmiento-Suarez R
- Sartorius B
- Satpathy M
- Savic M
- Sawhney M
- Schmidhuber J
- Schmidt MI
- Schneider IJC
- Schoettker B
- Schutte AE
- Schwebel DC
- Scott JG
- Seedat S
- Sepanlou SG
- Servan-Mori EE
- Shaheen A
- Shahraz S
- Shaikh MA
- Sharma R
- She J
- Sheikhbahaei S
- Shen J
- Sheth KN
- Shi P
- Shibuya K
- Shigematsu M
- Shin M-J
- Shiri R
- Shishani K
- Shiue I
- Shrime MG
- Sigfusdottir ID
- Silva DAS
- Silverberg JI
- Simard EP
- Sindi S
- Singh A
- Singh JA
- Singh PK
- Slepak EL
- Sofia E Sa Farinha C
- Soljak M
- Soneji S
- Sorensen RJD
- Sposato LA
- Sreeramareddy CT
- Stathopoulou V
- Steckling N
- Steel N
- Stein DJ
- Stein MB
- Stockl H
- Stranges S
- Stroumpoulis K
- Sunguya BF
- Sur PJ
- Swaminathan S
- Sykes BL
- Szoeke CEI
- Tabares-Seisdedos R
- Takahashi K
- Talongwa RT
- Tanne D
- Tavakkoli M
- Taye BW
- Taylor HR
- Tedla BA
- Tefera WM
- Tegegne TK
- Tekle DY
- Terkawi AS
- Thakur JS
- Thomas BA
- Thomas ML
- Thomson AJ
- Thorne-Lyman AL
- Thrift AG
- Thurston GD
- Tillmann T
- Tobe-Gai R
- Tobollik M
- Topor-Madry R
- Topouzis F
- Towbin JA
- Tran BX
- Tsilimparis N
- Tura AK
- Tuzcu EM
- Tyrovolas S
- Ukwaja KN
- Undurraga EA
- Uneke CJ
- Uthman OA
- van Donkelaar A
- van Os J
- Varakin YY
- Vasankari T
- Veerman JL
- Venketasubramanian N
- Violante FS
- Vollset SE
- Vos T
- Wagner GR
- Wagner JA
- Waller SG
- Wang H
- Wang J
- Wang L
- Wang Y
- Weichenthal S
- Weiderpass E
- Weintraub RG
- Werdecker A
- Westerman R
- Whiteford HA
- Wijeratne T
- Wiysonge CS
- Wolfe CDA
- Won S
- Woolf AD
- Wubshet M
- Xavier D
- Xu G
- Yadav AK
- Yakob B
- Yalew AZ
- Yano Y
- Yaseri M
- Ye P
- Yip P
- Yonemoto N
- Yoon S-J
- Younis MZ
- Yu C
- Zaidi Z
- Zaki MES
- Zhao Y
- Zhou M
- Zhu J
- Zipkin B
- Zodpey S
- Zuhlke LJ
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2016
- Field of study
SummaryBackground The Global Burden of Diseases, Injuries, and Risk Factors Study 2015 provides an up-to-date synthesis of the evidence for risk factor exposure and the attributable burden of disease. By providing national and subnational assessments spanning the past 25 years, this study can inform debates on the importance of addressing risks in context. Methods We used the comparative risk assessment framework developed for previous iterations of the Global Burden of Disease Study to estimate attributable deaths, disability-adjusted life-years (DALYs), and trends in exposure by age group, sex, year, and geography for 79 behavioural, environmental and occupational, and metabolic risks or clusters of risks from 1990 to 2015. This study included 388 risk-outcome pairs that met World Cancer Research Fund-defined criteria for convincing or probable evidence. We extracted relative risk and exposure estimates from randomised controlled trials, cohorts, pooled cohorts, household surveys, census data, satellite data, and other sources. We used statistical models to pool data, adjust for bias, and incorporate covariates. We developed a metric that allows comparisons of exposure across risk factors—the summary exposure value. Using the counterfactual scenario of theoretical minimum risk level, we estimated the portion of deaths and DALYs that could be attributed to a given risk. We decomposed trends in attributable burden into contributions from population growth, population age structure, risk exposure, and risk-deleted cause-specific DALY rates. We characterised risk exposure in relation to a Socio-demographic Index (SDI). Findings Between 1990 and 2015, global exposure to unsafe sanitation, household air pollution, childhood underweight, childhood stunting, and smoking each decreased by more than 25%. Global exposure for several occupational risks, high body-mass index (BMI), and drug use increased by more than 25% over the same period. All risks jointly evaluated in 2015 accounted for 57·8% (95% CI 56·6–58·8) of global deaths and 41·2% (39·8–42·8) of DALYs. In 2015, the ten largest contributors to global DALYs among Level 3 risks were high systolic blood pressure (211·8 million [192·7 million to 231·1 million] global DALYs), smoking (148·6 million [134·2 million to 163·1 million]), high fasting plasma glucose (143·1 million [125·1 million to 163·5 million]), high BMI (120·1 million [83·8 million to 158·4 million]), childhood undernutrition (113·3 million [103·9 million to 123·4 million]), ambient particulate matter (103·1 million [90·8 million to 115·1 million]), high total cholesterol (88·7 million [74·6 million to 105·7 million]), household air pollution (85·6 million [66·7 million to 106·1 million]), alcohol use (85·0 million [77·2 million to 93·0 million]), and diets high in sodium (83·0 million [49·3 million to 127·5 million]). From 1990 to 2015, attributable DALYs declined for micronutrient deficiencies, childhood undernutrition, unsafe sanitation and water, and household air pollution; reductions in risk-deleted DALY rates rather than reductions in exposure drove these declines. Rising exposure contributed to notable increases in attributable DALYs from high BMI, high fasting plasma glucose, occupational carcinogens, and drug use. Environmental risks and childhood undernutrition declined steadily with SDI; low physical activity, high BMI, and high fasting plasma glucose increased with SDI. In 119 countries, metabolic risks, such as high BMI and fasting plasma glucose, contributed the most attributable DALYs in 2015. Regionally, smoking still ranked among the leading five risk factors for attributable DALYs in 109 countries; childhood underweight and unsafe sex remained primary drivers of early death and disability in much of sub-Saharan Africa. Interpretation Declines in some key environmental risks have contributed to declines in critical infectious diseases. Some risks appear to be invariant to SDI. Increasing risks, including high BMI, high fasting plasma glucose, drug use, and some occupational exposures, contribute to rising burden from some conditions, but also provide opportunities for intervention. Some highly preventable risks, such as smoking, remain major causes of attributable DALYs, even as exposure is declining. Public policy makers need to pay attention to the risks that are increasingly major contributors to global burden. Funding Bill & Melinda Gates Foundation
Search for new phenomena in final states with an energetic jet and large missing transverse momentum in pp collisions at √ s = 8 TeV with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abud AA
- Abulaiti Y
- Acharya BS
- Achkar B
- Adachi S
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
- Adersberger M
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmadov F
- Ahmed WS
- Ai X
- Aielli G
- Akatsuka S
- Akesson TPA
- Akilli E
- Akimov A
- Al Khoury K
- Alberghi GL
- Albert J
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Alhroob M
- Ali B
- Aliev M
- Alimonti G
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alvarez Estevez M
- Alviggi MG
- Amaral Coutinho Y
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amor Dos Santos SP
- Amoroso S
- Amrouche CS
- An F
- Anastopoulos C
- Andari N
- Andeen T
- Anders CF
- Anders JK
- Andreazza A
- Andrei V
- Anelli CR
- Angelidakis S
- Angerami A
- Anisenkov A
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Aparisi Pozo JA
- Araque JP
- Araujo Ferraz V
- Araujo Pereira R
- Araya ST
- Arcangeletti C
- Arce ATH
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Arling J-H
- Armbruster AJ
- Armstrong A
- Arnaez O
- Arnold H
- Artoni G
- Artz S
- Asai S
- Asawatavonvanich T
- Asbah N
- Asimakopoulou EM
- Asquith L
- Assahsah J
- Assamagan K
- Astalos R
- Astigarraga MEP
- Atkin RJ
- Atkinson M
- Atlay NB
- Atmani H
- Augsten K
- Avolio G
- Ayoub MK
- Azuelos G
- Bachacou H
- Bachas K
- Backes M
- Backman F
- Bagnaia P
- Bahmani M
- Bahrasemani H
- Bailey AJ
- Bailey VR
- Baines JT
- Bakalis C
- Baker OK
- Bakker PJ
- Balaji S
- Baldin EM
- Balek P
- Balli F
- Balunas WK
- Balz J
- Banas E
- Bandyopadhyay A
- Banerjee S
- Bannoura AAE
- Barak L
- Barbe WM
- Barberio EL
- Barberis D
- Barbero M
- Barbour G
- Barillari T
- Barisits M-S
- Barkeloo J
- Barklow T
- Barnea R
- Barnett BM
- Barnett RM
- Barnovska-Blenessy Z
- Baroncelli A
- Barone G
- Barr AJ
- Barreiro F
- Barsov S
- Bartoldus R
- Bartolini G
- Barton AE
- Bartos P
- Basalaev A
- Basan A
- Bassalat A
- Basso MJ
- Bates RL
- Batlamous S
- Batley JR
- Batool B
- Battaglia M
- Bauce M
- Bauer F
- Bauer KT
- Bawa HS
- Beacham JB
- Beau T
- Beauchemin PH
- Becherer F
- Bechtle P
- Beck HC
- Beck HP
- Becker K
- Becot C
- Beddall A
- Beddall AJ
- Bednyakov VA
- Bedognetti M
- Beermann TA
- Begalli M
- Begel M
- Behera A
- Behr JK
- Beisiegel F
- Bell AS
- Bella G
- Bella LA
- Bellagamba L
- Bellerive A
- Bellos P
- Beloborodov K
- Belotskiy K
- Belyaev NL
- Benchekroun D
- Benekos N
- Benhammou Y
- Benjamin DP
- Benoit M
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Berge D
- Berger N
- Bergmann B
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- Klimentov A
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- Knue A
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- Kodys P
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- Kotwal A
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- Kourkoumelis C
- Kourlitis E
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- Laplace S
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
Results of a search for new phenomena in final states with an energetic jet and large missing transverse momentum are reported. The search uses 20.3 fb−1 of √ s = 8 TeV data collected in 2012 with the ATLAS detector at the LHC. Events are required to have at least one jet with pT > 120 GeV and no leptons. Nine signal regions are considered with increasing missing transverse momentum requirements between Emiss T > 150 GeV and Emiss T > 700 GeV. Good agreement is observed between the number of events in data and Standard Model expectations. The results are translated into exclusion limits on models with either large extra spatial dimensions, pair production of weakly interacting dark matter candidates, or production of very light gravitinos in a gauge-mediated supersymmetric model. In addition, limits on the production of an invisibly decaying Higgs-like boson leading to similar topologies in the final state are presente
Measurement of the cross-section and charge asymmetry of W bosons produced in proton-proton collisions at s=8 TeV with the ATLAS detector
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- Abbott B.
- Abbott D. C.
- Abdinov O.
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- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
This paper presents measurements of the W+→μ+ν and W−→μ−ν cross-sections and the associated charge asymmetry as a
function of the absolute pseudorapidity of the decay muon. The data were
collected in proton--proton collisions at a centre-of-mass energy of 8 TeV with
the ATLAS experiment at the LHC and correspond to a total integrated luminosity
of 20.2~\mbox{fb^{-1}}. The precision of the cross-section measurements
varies between 0.8% to 1.5% as a function of the pseudorapidity, excluding the
1.9% uncertainty on the integrated luminosity. The charge asymmetry is measured
with an uncertainty between 0.002 and 0.003. The results are compared with
predictions based on next-to-next-to-leading-order calculations with various
parton distribution functions and have the sensitivity to discriminate between
them.Comment: 38 pages in total, author list starting page 22, 5 figures, 4 tables,
submitted to EPJC. All figures including auxiliary figures are available at
https://atlas.web.cern.ch/Atlas/GROUPS/PHYSICS/PAPERS/STDM-2017-13
Defective Membrane Remodeling in Neuromuscular Diseases: Insights from Animal Models
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- A Bolino
- A Bolino
- A Bolino
- A Bolis
- A Buj-Bello
- A Buj-Bello
- A Echaniz-Laguna
- A Labrousse
- A Raimondi
- A Razzaq
- A Toussaint
- AC Durieux
- AC Durieux
- AH Beggs
- AJ Muller
- Anne Toussaint
- AS Nicot
- AS Nicot
- Belinda S. Cowling
- BJ Peter
- BS Cowling
- DJ Hernandez-Deviez
- DJ Owen
- E Johnson
- E Lee
- EE Patterson
- FL Robinson
- FL Robinson
- G Di Paolo
- G Di Paolo
- GJ Praefcke
- GK van Wijngaarden
- Gregory A. Cox
- H Azzedine
- H Dang
- H Fares
- HH Nandurkar
- I Ribeiro
- J Bohm
- J Laporte
- J Laporte
- J Laporte
- J Laporte
- J Schessl
- J Senderek
- J Shen
- J Zou
- JA Kenniston
- Jean Muller
- JJ Dowling
- Jocelyn Laporte
- K Hnia
- K Hnia
- K Tersar
- L Al-Qusairi
- L Klinge
- L Wang
- LJ Neukomm
- M Bitoun
- M Bitoun
- M Bitoun
- M Ramaswami
- M Silhankova
- M Velichkova
- M Vicinanza
- MA De Matteis
- MA McNiven
- MJ Begley
- MJ Wishart
- MM Reilly
- N Kusumi
- P Berger
- P De Camilli
- PS Estes
- R Wechsler-Reya
- RJ Wechsler-Reya
- S Bonneick
- S Pant
- S Zuchner
- SA Kim
- SA Robb
- SG Clark
- SM Ferguson
- SM Ferguson
- SM Jones
- T Kosaka
- T Tsuruhara
- U Suter
- W Zou
- X Cui
- X Yu
- Y Mochizuki
- Y Nakayama
- Y Xue
- Publication venue
- Public Library of Science
- Publication date
- 05/04/2012
- Field of study
Proteins involved in membrane remodeling play an essential role in a plethora of cell functions including endocytosis and intracellular transport. Defects in several of them lead to human diseases. Myotubularins, amphiphysins, and dynamins are all proteins implicated in membrane trafficking and/or remodeling. Mutations in myotubularin, amphiphysin 2 (BIN1), and dynamin 2 lead to different forms of centronuclear myopathy, while mutations in myotubularin-related proteins cause Charcot-Marie-Tooth neuropathies. In addition to centronuclear myopathy, dynamin 2 is also mutated in a dominant form of Charcot-Marie-Tooth neuropathy. While several proteins from these different families are implicated in similar diseases, mutations in close homologues or in the same protein in the case of dynamin 2 lead to diseases affecting different tissues. This suggests (1) a common molecular pathway underlying these different neuromuscular diseases, and (2) tissue-specific regulation of these proteins. This review discusses the pathophysiology of the related neuromuscular diseases on the basis of animal models developed for proteins of the myotubularin, amphiphysin, and dynamin families. A better understanding of the common mechanisms between these neuromuscular disorders will lead to more specific health care and therapeutic approaches
Search for chargino-neutralino production with mass splittings near the electroweak scale in three-lepton final states in √s=13 TeV pp collisions with the ATLAS detector
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- Wingerter-Seez I
- Winkels E.
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- Wyatt T. R.
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- Xella S.
- Xi Z.
- Xia L.
- Xiao X.
- Xiotidis I
- Xu D.
- Xu H.
- Xu L.
- Xu T.
- Xu W.
- Xu Z.
- Yabsley B.
- Yacoob S.
- Yajima K.
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- Yamaguchi D.
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- Yamamoto A.
- Yamatani M.
- Yamazaki T.
- Yamazaki Y.
- Yan Z.
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- Yao W-M
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- Yexley M. R.
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- Yildiz H. Duran
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- Zaazoua M.
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- Zaffaroni E.
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- Zambito S.
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- Zeissner S.
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- Zeng J. C.
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- Zhang D. F.
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- Zhang J.
- Zhang L.
- Zhang M.
- Zhang R.
- Zhang X.
- Zhang Y.
- Zhang Z.
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- Zhao Y.
- Zhao Z.
- Zhemchugov A.
- Zheng Z.
- Zhong D.
- Zhou B.
- Zhou C.
- Zhou M.
- Zhou M. S.
- Zhou N.
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- Zhu C. G.
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- Zhu J.
- Zhu Y.
- Zhuang X.
- Zhukov K.
- Zhulanov V
- Zieminska D.
- Zimine N.
- Zimmermann S.
- Zinonos Z.
- Ziolkowski M.
- Zivkovic L.
- Zobernig G.
- Zoccoli A.
- Zoch K.
- Zorbas T. G.
- Zou R.
- Zu Theenhausen H. Meyer
- Zwalinski L.
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 28/05/2019
- Field of study
A search for supersymmetry through the pair production of electroweakinos with mass splittings near the electroweak scale and decaying via on-shell W and Z bosons is presented for a three-lepton final state. The analyzed proton-proton collision data taken at a center-of-mass energy of √s=13 TeV were collected between 2015 and 2018 by the ATLAS experiment at the Large Hadron Collider, corresponding to an integrated luminosity of 139 fb−1. A search, emulating the recursive jigsaw reconstruction technique with easily reproducible laboratory-frame variables, is performed. The two excesses observed in the 2015–2016 data recursive jigsaw analysis in the low-mass three-lepton phase space are reproduced. Results with the full data set are in agreement with the Standard Model expectations. They are interpreted to set exclusion limits at the 95% confidence level on simplified models of chargino-neutralino pair production for masses up to 345 GeV
Global Impact of the COVID-19 Pandemic on Cerebral Venous Thrombosis and Mortality
- Author
- Abdalkader M
- Acampa M
- Adami A
- Adebayo TT
- Ahmad S
- Al Hashmi AM
- Alexiev F
- Araújo AP
- Arsovska AA
- Asif KS
- Aykac O
- Ayo-Martin O
- Baek JH
- Bajrami A
- Banerjee S
- Barrientos Guerra JD
- Bello AH
- Below K
- Bernava G
- Bessa F
- Biller J
- Blasco J
- Bolognese M
- Bonnet T
- Boskamp S
- Brady M
- Brola W
- Carrera E
- Castro P
- Charidimou A
- Chebl A
- Chen HS
- Chen M
- Chen Y
- Chieh CY
- Churojana A
- Ciopleias B
- Colasurdo M
- Conforto AB
- Cora EA
- Cruz-Culebras A
- Czap A
- Czlonkowska A
- Dao VP
- de Havenon AH
- de la Cruz Fernández B
- de Oliveira Neves L
- De Raedt S
- de Sousa DA
- Demeestere J
- Dharmadhikari S
- Dimitriade A
- Dippel D
- Dirren E
- Dorobek M
- Drenck N
- Duong HQ
- Dušek P
- Dy JSH
- Dębiec A
- Enzinger C
- Escartín López J
- Eskey CJ
- Falup-Pecurariu C
- Farhoudi M
- Farooqui M
- Fernández Ferro JC
- Ferreira P
- Feske SK
- Fibrich P
- Field TS
- Fonseca L
- Fujita K
- Fukuda H
- García Sánchez MJ
- Gattringer T
- Ghoreishi A
- Gongora-Rivera F
- Goyal N
- Grimmett KB
- Guillán Rodríguez M
- Guzik AK
- Hamid MA
- Haring J
- Hashemilar M
- Haussen DC
- Hernandez-Fernandez F
- Hidalgo RCT
- Hlaváčová H
- Hovingh M
- Hrabanovska E
- Huded V
- Hussain SI
- Imamura H
- Infante-Valenzuela A
- Inoa V
- Jancar N
- Jillela D
- John S
- Jurák L
- Kadlčíková J
- Kalpachki R
- Kan PT
- Karapanayiotides T
- Kargiotis O
- Karlinski MA
- Karpowicz I
- Kelly ME
- Khatri R
- Khoury NN
- Kiley NL
- Klein P
- Klečka L
- Kolikonda MK
- Kosuke M
- Koutroulou I
- Kovář M
- Krastev G
- Krishnan K
- Kristoffersen ES
- Kwan J
- Labuz-Roszak BM
- Lameirinhas da Silva I
- Lara S
- Lasek-Bal A
- Le HV
- Lee JS
- Lee SB
- Lemmens R
- Lenthall R
- Lereis VP
- Lesaine E
- Li G
- Ligot N
- Linfante I
- Lo H
- Longoni M
- Loochtan AI
- Lopez CD
- Lycan S
- Ma A
- Maali L
- Machi P
- Male SS
- Mansour OY
- Martinez-Marino M
- Marto JP
- Martíns RT
- Masoud HE
- Matsumaru Y
- Mbroh J
- Michel P
- Mikulik R
- Min J
- Mohamed GA
- Mohammaden M
- Mohammadzadeh N
- Moniche F
- Montaner J
- Mora Cuervo DL
- Nagel S
- Nagendra S
- Nahab F
- Nalleballe K
- Neumann J
- Nguyen TN
- Nogueira RG
- Nunes AP
- Ohara N
- Omran SS
- Orgeta-Gutierrez S
- Ornello R
- Palaiodimou L
- Paloušková H
- Peeling L
- Perez-Sanchez S
- Pham TN
- Pikula A
- Pinho E Melo T
- Poli S
- Prajapati C
- Punter M
- Qureshi MM
- Radaideh Y
- Rahal F
- Rahman A
- Ramakrishnan P
- Rashid U
- Rayo-Taranto B
- Rebello LC
- Reiner P
- Reiser M
- Renieri L
- Rocha M
- Rodrigues M
- Rodriguez-Kadota L
- Rodríguez Fernández C
- Rodríguez-Vázquez A
- Rohan V
- Rojas-Soto DM
- Roje Bedekovic M
- Romoli M
- Rosenkranz M
- Rouanet F
- Ruland S
- Sabben C
- Sabetay SI
- Sacco S
- Sadeghi Hokmabadi E
- Sakaguchi M
- Sakai K
- Sakai N
- Sakelarova T
- Salmaggi A
- Sangalli D
- Sani AF
- Santiago IB
- Sathya A
- Scozzari D.
- Sedova P
- Senadim S
- Seo KD
- Sheth SA
- Shindo S
- Siegler JE
- Sienkiewicz-Jarosz H
- Silva ML
- Simpkins AN
- Skoda O
- Sobh K
- Sobolewski P
- Sohn SI
- Sonoda K
- Starosciak AK
- Staszewski J
- Strambo D
- Strbian D
- Sugiura Y
- Sundseth A
- Sunmonu TA
- Suryadevara NC
- SVIN COVID-19 Global COVID Stroke Registry
- Sylaja PN
- Takenobu Y
- Tarlov NE
- Taylor RA
- Tenora D
- Thomalla G
- Ton MD
- Toyoda K
- Tran AD
- Tsivgoulis G
- Uwatoko T
- van Dam-Nolen DHK
- Vanacker P
- Vandervorst F
- Vences MÁ
- Venketasubramanian N
- Voetsch B
- Wahab KW
- Wan Yahya WNN
- Wan Zaidi WA
- Wiącek M
- Wu TY
- Yaghi S
- Yalung PM
- Yamagami H
- Yamamoto N
- Yamamoto R
- Yang S
- Yazawa Y
- Yoshimoto T
- Zaidat OO
- Zhang L
- Zhang L
- Zielinska-Turek J
- Zini A
- Özdemir AÖ
- Čabal M
- Šimůnek L
- Škorňa M
- Šrámek M
- Publication venue
- Publication date
- 01/01/2022
- Field of study
Background and purpose: Recent studies suggested an increased incidence of cerebral venous thrombosis (CVT) during the coronavirus disease 2019 (COVID-19) pandemic. We evaluated the volume of CVT hospitalization and in-hospital mortality during the 1st year of the COVID-19 pandemic compared to the preceding year.
Methods: We conducted a cross-sectional retrospective study of 171 stroke centers from 49 countries. We recorded COVID-19 admission volumes, CVT hospitalization, and CVT in-hospital mortality from January 1, 2019, to May 31, 2021. CVT diagnoses were identified by International Classification of Disease-10 (ICD-10) codes or stroke databases. We additionally sought to compare the same metrics in the first 5 months of 2021 compared to the corresponding months in 2019 and 2020 (ClinicalTrials.gov Identifier: NCT04934020).
Results: There were 2,313 CVT admissions across the 1-year pre-pandemic (2019) and pandemic year (2020); no differences in CVT volume or CVT mortality were observed. During the first 5 months of 2021, there was an increase in CVT volumes compared to 2019 (27.5%; 95% confidence interval [CI], 24.2 to 32.0; P<0.0001) and 2020 (41.4%; 95% CI, 37.0 to 46.0; P<0.0001). A COVID-19 diagnosis was present in 7.6% (132/1,738) of CVT hospitalizations. CVT was present in 0.04% (103/292,080) of COVID-19 hospitalizations. During the first pandemic year, CVT mortality was higher in patients who were COVID positive compared to COVID negative patients (8/53 [15.0%] vs. 41/910 [4.5%], P=0.004). There was an increase in CVT mortality during the first 5 months of pandemic years 2020 and 2021 compared to the first 5 months of the pre-pandemic year 2019 (2019 vs. 2020: 2.26% vs. 4.74%, P=0.05; 2019 vs. 2021: 2.26% vs. 4.99%, P=0.03). In the first 5 months of 2021, there were 26 cases of vaccine-induced immune thrombotic thrombocytopenia (VITT), resulting in six deaths.
Conclusions: During the 1st year of the COVID-19 pandemic, CVT hospitalization volume and CVT in-hospital mortality did not change compared to the prior year. COVID-19 diagnosis was associated with higher CVT in-hospital mortality. During the first 5 months of 2021, there was an increase in CVT hospitalization volume and increase in CVT-related mortality, partially attributable to VITT
Human matrix metalloproteinases: An ubiquitarian class of enzymes involved in several pathological processes
- Author
- Adachi
- Adachi
- Agarwal
- Agarwal
- Agrawal
- Ahmad
- Ahmed
- Ahn
- Ahokas
- Ahokas
- Aihara
- Airola
- Akhtar
- Ala-aho
- Ala-Aho
- Alam
- Albrecht
- Amberger
- Annabi
- Anthony
- Apte
- Araujo
- Ardi
- Aresu
- Arlt
- Arthur
- Asahina
- Atkinson
- Atkinson
- Atkinson
- Aureli
- Avolio
- Ayoub
- Azar
- Babusyte
- Baciu
- Badiga
- Bafetti
- Baines
- Balbin
- Balbin
- Balbin
- Banda
- Bar-Or
- Baramova
- Barascuk
- Barksby
- Barmina
- Barnes
- Barret
- Barrowman
- Basset
- Basset
- Bassi
- Bau
- Bayes-Genis
- Beck
- Becker
- Belaaouaj
- Belaaouaj
- Beliveau
- Beliën
- Belkin
- Bendardaf
- Bendrik
- Benesova
- Benesova
- Bergers
- Bergmann
- Bernal
- Bernal
- Bernstein
- Berry
- Betsuyaku
- Bhaskar
- Bhaskaran
- Billinghurst
- Bilousova
- Binder
- Binker
- Bisson
- Bister
- Bister
- Biswas
- Björklund
- Bjørn
- Bland
- Blavier
- Blobel
- Blumenthal
- Bode
- Bode
- Boldt
- Bolignano
- Bolon
- Bonacci
- Bord
- Borges
- Borkakoti
- Borrell-Pages
- Boulay
- Bourboulia
- Bourguignon
- Bozzi
- Bradbury
- Bralet
- Brauer
- Brew
- Briknarova
- Brinckerhoff
- Brinckerhoff
- Brinckerhoff
- Broide
- Brooks
- Brown
- Buhler
- Buhmeida
- Bujak
- Burke
- Burrage
- Butoi Dragomir
- Bylander
- Byun
- Caceres
- Cai
- Campbell
- Cao
- Cao
- Capell
- Capell
- Carl-McGrath
- Carrel
- Carrell
- Carty
- Cataldo
- Cataldo
- Cauwe
- Cauwe
- Cauwe
- Cazorla
- Cha
- Chabottaux
- Chabry
- Chaffer
- Chambers
- Chan
- Chandler
- Chang
- Chang
- Chang
- Chantrain
- Chapman
- Charbonneau
- Chaudhary
- Checler
- Chen
- Chen
- Chen
- Chen
- Chen
- Chen
- Chen
- Cheng
- Chetty
- Chetty
- Chiao
- Chiara Ciaccio
- Chiba
- Chin
- Cho
- Cho
- Choi
- Choke
- Chung
- Chung
- Churg
- Churg
- Churg
- Churg
- Clements
- Clements
- Cleutjens
- Colige
- Collier
- Colotti
- Comabella
- Conant
- Conover
- Conover
- Consuegra-Sanchez
- Consuegra-Sanchez
- Copin
- Cosgrove
- Coulson-Thomas
- Coussens
- Coussens
- Coussens
- Culpitt
- Curci
- Cyr
- Daniele
- Danielsen
- Daull
- Davidson
- Davidson
- Davies
- Davis
- De Coignac
- Deb
- Decock
- Deguchi
- Dehan
- Del Zoppo
- Delassus
- Demedts
- Denecke
- Deng
- Denny
- Deryugina
- Deryugina
- Deryugina
- Deshmukh
- Devarajan
- Di Bella
- Diaz-Sanchez
- Diego Sbardella
- Doerner
- Doi
- Domagala-Kulawik
- Dong
- Dong
- Du
- Du
- Dubois
- Duffy
- Dufour
- Dumin
- Dunn
- Durham
- D’Armiento
- D’Ercole
- Eckman
- Eckman
- Edwards
- Egeblad
- Ehrenfeld
- Eliason
- Elkington
- Elkington
- Elkins
- Ellenrieder
- Elnemr
- Emlen
- Emoto
- Endo
- England
- English
- English
- Erler
- Esser
- Ezhilarasan
- Fabriek
- Fager
- Fahrenholz
- Fainardi
- Fariñas
- Fasciglione
- Fenton
- Fernandes
- Fernandes
- Fernandez-Catalan
- Ferrara
- Ferreirinha
- Ferro
- Fields
- Finder
- Fingleton
- Fingleton
- Finlay
- Fiore
- Fiotti
- Firestein
- Flex
- Folgueras
- Folgueras
- Fortin
- Fosang
- Fowlkes
- Frederick
- Freedland
- Freemont
- Freije
- Fujikawa
- Fujita
- Furlan
- Furuse
- Gaetje
- Gakh
- Galis
- Galis
- Galis
- Gall
- Galvez-Gastelum
- Ganji
- Garcia
- Garcia-Montojo
- Garcia-Prieto
- Garg
- Garton
- Garvican
- Gaultier
- Gauthier
- Ge
- Genden
- Gerstein
- Ghaderian
- Giannelli
- Giannobile
- Gill
- Gill
- Gilles
- Gilles
- Gilles
- Gingras
- Gioia
- Gioia
- Gioia
- Gioia
- Giovanni Francesco Fasciglione
- Giricz
- Giricz
- Glass-Marmor
- Goldbach-Mansky
- Gondi
- Gottlieb
- Gough
- Gounni-Abdelilah
- Grazia Raffaella Tundo
- Gronski
- Gruber
- Grzmil
- Gu
- Guadamillas
- Gueders
- Guo
- Gutierrez-Fernandez
- Ha
- Haas
- Habelhah
- Hafezi-Moghadam
- Hahn
- Hahn
- Hamann
- Hanemaaijer
- Hansson
- Hansson
- Hansson
- Hao
- Haq
- Harrell
- Harrison
- Harty
- Hashimoto
- Hashimoto
- Hasty
- Hattori
- Hattori
- Hauser
- Hautamaki
- Hawinkels
- Hawkins
- Hayashita-Kinoh
- Hegedüs
- Heiskanen
- Heljasvaara
- Henderson
- Henderson
- Henderson
- Henney
- Heo
- Heo
- Herman
- Hernandez-Guillamon
- Hernandez-Guillamon
- Herrmann
- Hersh
- Heymans
- Heymans
- Hiden
- Hieta
- Hiller
- Himelstein
- Hiraoka
- Hiratsuka
- Hirose
- Hirose
- Hofmann
- Hohlbaum
- Hojilla
- Holman
- Holmbeck
- Hopkins
- Horton
- Hoshino
- Hotary
- Hotary
- Houghton
- Howard
- Hu
- Hu
- Huang
- Hulkkonen
- Hunninghake
- Huo
- Hwang
- Hägglund
- Ihling
- Ihling
- Ii
- Iida
- Iimuro
- Ikari
- Illman
- Illman
- Illman
- Imai
- Imai
- Imai
- Impola
- Impola
- Inoue
- Iredale
- Ito
- Itoh
- Itoh
- Itoh
- Itoh
- Itoh
- Iwata
- Janssens
- Jaworski
- Jefferis
- Jeffries
- Jeng
- Jensen
- Jia
- Jian
- Jin
- Jo
- Joergensen
- Johansson
- Johansson
- Johansson
- Johnson
- Johnson
- Johnson
- Johnson
- Johnston
- Jones
- Jones
- Jonsson-Rylander
- Jormsjo
- Jost
- Juncker-Jensen
- Jung
- Jung
- Jury
- Jüngel
- Kadono
- Kadonosono
- Kajita
- Kajita
- Kaminska
- Kanayama
- Kaneko
- Kanemitsu
- Kang
- Kang
- Kang
- Kaplan
- Kaplan
- Karlberg
- Karroum
- Kato
- Katzenstein
- Kay
- Kenny
- Kenny
- Kerkela
- Kerkela
- Kessenbrock
- Kessler
- Kest
- Kevorkian
- Khokha
- Kiili
- Kim
- Kim
- Kim
- Kim
- Kim
- Kim
- Kirkegaard
- Kiryu-Seo
- Kiryu-Seo
- Kitagawa
- Klatt
- Klawitter
- Klein
- Knox
- Knäuper
- Knäuper
- Knäuper
- Kobayashi
- Kobayashi
- Kodali
- Koehne
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- Xia
- Xiong
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- Xu
- Xu
- Xu
- Yamada
- Yamamoto
- Yamamoto
- Yamamoto
- Yamanaka
- Yan
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- Ye
- Ye
- Yen
- Yoon
- Yoshihara
- Yoshizaki
- Yoshizaki
- Young
- Young
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- Yu
- Zeng
- Zhang
- Zhang
- Zhang
- Zhao
- Zhao
- Zhao
- Zhao
- Zheng
- Zhong
- Zhou
- Zhou
- Zhu
- Zijlstra
- Zitta
- Zucker
- Zuo
- Publication venue
- Publication date
- 01/04/2012
- Field of study
Human matrix metalloproteinases (MMPs) belong to the M10 family of the MA clan of endopeptidases. They are ubiquitarian enzymes, structurally characterized by an active site where a Zn(2+) atom, coordinated by three histidines, plays the catalytic role, assisted by a glutamic acid as a general base. Various MMPs display different domain composition, which is very important for macromolecular substrates recognition. Substrate specificity is very different among MMPs, being often associated to their cellular compartmentalization and/or cellular type where they are expressed. An extensive review of the different MMPs structural and functional features is integrated with their pathological role in several types of diseases, spanning from cancer to cardiovascular diseases and to neurodegeneration. It emerges a very complex and crucial role played by these enzymes in many physiological and pathological processes
Global patient outcomes after elective surgery: prospective cohort study in 27 low-, middle- and high-income countries.
- Author
- Ab Majid MA
- Ab Rahman AS
- Ab Rahman R
- Abayasinghe C
- Abbott T
- Abdullah NH
- Abdur-Rahman L
- Abeloos J
- Abernethy C
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- Aimoniotou-Georgiou E
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- Zoulamoglou M
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- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- 01/01/2016
- Field of study
BACKGROUND: As global initiatives increase patient access to surgical treatments, there remains a need to understand the adverse effects of surgery and define appropriate levels of perioperative care. METHODS: We designed a prospective international 7-day cohort study of outcomes following elective adult inpatient surgery in 27 countries. The primary outcome was in-hospital complications. Secondary outcomes were death following a complication (failure to rescue) and death in hospital. Process measures were admission to critical care immediately after surgery or to treat a complication and duration of hospital stay. A single definition of critical care was used for all countries. RESULTS: A total of 474 hospitals in 19 high-, 7 middle- and 1 low-income country were included in the primary analysis. Data included 44 814 patients with a median hospital stay of 4 (range 2-7) days. A total of 7508 patients (16.8%) developed one or more postoperative complication and 207 died (0.5%). The overall mortality among patients who developed complications was 2.8%. Mortality following complications ranged from 2.4% for pulmonary embolism to 43.9% for cardiac arrest. A total of 4360 (9.7%) patients were admitted to a critical care unit as routine immediately after surgery, of whom 2198 (50.4%) developed a complication, with 105 (2.4%) deaths. A total of 1233 patients (16.4%) were admitted to a critical care unit to treat complications, with 119 (9.7%) deaths. Despite lower baseline risk, outcomes were similar in low- and middle-income compared with high-income countries. CONCLUSIONS: Poor patient outcomes are common after inpatient surgery. Global initiatives to increase access to surgical treatments should also address the need for safe perioperative care. STUDY REGISTRATION: ISRCTN5181700
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