112 research outputs found
Postnatal Proteasome Inhibition Induces Neurodegeneration and Cognitive Deficiencies in Adult Mice: A New Model of Neurodevelopment Syndrome
- Author
- A DiAntonio
- A Gruart
- A Hershko
- A Kaffman
- A Matilla-Dueñas
- A Surget
- A Varshavsky
- AE Dityatev
- AL Goldberg
- AL Goldberg
- AL Schwartz
- AN Hegde
- B Bingol
- CA Ross
- CA Ross
- CD Galvan
- CE Stafstrom
- CE Stafstrom
- D Germain
- DA Gill
- DC Rubinsztein
- EB Taylor
- EC Burgard
- ED Levin
- ED Levin
- ER de Kloet
- Francisco Javier Aguilar-Montilla
- GN Patrick
- HC Tai
- HJ Groenewegen
- I Ehrlich
- IA Trounce
- J Dobbing
- J Pines
- JE Malberg
- JF Nouws
- Jiyan Ma
- JJ Yi
- JM Heo
- JN Keller
- K Nakano
- KS Hougaard
- L Bedford
- L Santarelli
- M de los Santos-Arteaga
- M Muratani
- M Zhao
- MT Lin
- MY Sherman
- NF Santos
- NM van Strien
- PG Sullivan
- R Havekes
- R Tyzio
- RK Murphey
- Rocío Romero-Granados
- T Grune
- WE Crusio
- WM Fox
- Y Ben-Ari
- Ángel Manuel Carrión
- Ángela Fontán-Lozano
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
Defects in the ubiquitin-proteasome system have been related to aging and the development of neurodegenerative disease, although the effects of deficient proteasome activity during early postnatal development are poorly understood. Accordingly, we have assessed how proteasome dysfunction during early postnatal development, induced by administering proteasome inhibitors daily during the first 10 days of life, affects the behaviour of adult mice. We found that this regime of exposure to the proteasome inhibitors MG132 or lactacystin did not produce significant behavioural or morphological changes in the first 15 days of life. However, towards the end of the treatment with proteasome inhibitors, there was a loss of mitochondrial markers and activity, and an increase in DNA oxidation. On reaching adulthood, the memory of mice that were injected with proteasome inhibitors postnatally was impaired in hippocampal and amygdala-dependent tasks, and they suffered motor dysfunction and imbalance. These behavioural deficiencies were correlated with neuronal loss in the hippocampus, amygdala and brainstem, and with diminished adult neurogenesis. Accordingly, impairing proteasome activity at early postnatal ages appears to cause morphological and behavioural alterations in adult mice that resemble those associated with certain neurodegenerative diseases and/or syndromes of mental retardation
Comparative genomics of Cluster O mycobacteriophages
- Author
- Adair T
- Anders KR
- Ball S
- Bollivar D
- Bowman CA
- Breitenberger C
- Burnett SH
- Butela K
- Byrnes D
- Carzo S
- Cornely KA
- Cresawn SG
- Cross T
- Daniels RL
- De Los Santos C
- Dedrick RM
- Dunbar D
- Ekanem K
- Findley AM
- Gissendanner CR
- Golebiewska UP
- Hartzog GA
- Hatfull GF
- Hatherill JR
- Hendrix RW
- Hughes LE
- Jacobs-Sera D
- Jalloh CS
- Khambule SL
- King RA
- King-Smith C
- Klyczek K
- Krukonis GP
- Laing C
- Lapin JS
- Lopez AJ
- Mkhwanazi SM
- Molloy SD
- Moran D
- Munsamy V
- Pacey E
- Plymale R
- Pope WH
- Poxleitner M
- Reyna N
- Russell DA
- Schildbach JF
- Stukey J
- Taylor SE
- Ware VC
- Wellmann AL
- Westholm D
- Wodarski D
- Zajko M
- Zikalala TS
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 05/03/2015
- Field of study
Mycobacteriophages - viruses of mycobacterial hosts - are genetically diverse but morphologically are all classified in the Caudovirales with double-stranded DNA and tails. We describe here a group of five closely related mycobacteriophages - Corndog, Catdawg, Dylan, Firecracker, and YungJamal - designated as Cluster O with long flexible tails but with unusual prolate capsids. Proteomic analysis of phage Corndog particles, Catdawg particles, and Corndog-infected cells confirms expression of half of the predicted gene products and indicates a non-canonical mechanism for translation of the Corndog tape measure protein. Bioinformatic analysis identifies 8-9 strongly predicted SigA promoters and all five Cluster O genomes contain more than 30 copies of a 17 bp repeat sequence with dyad symmetry located throughout the genomes. Comparison of the Cluster O phages provides insights into phage genome evolution including the processes of gene flux by horizontal genetic exchange
Performance of the CMS Cathode Strip Chambers with Cosmic Rays
- Author
- Abadjiev K
- Abbaneo D
- Abbiendi G
- Abbrescia M
- Abdullin S
- Abelev B
- Acosta D
- Acosta JG
- Acosta MV
- Actis O
- Adam N
- Adam W
- Adams MR
- Adams T
- Adiguzel A
- Adler V
- Adolphi R
- Adzic P
- Afaq MA
- Agostino L
- Agram JL
- Aguilar-Benitez M
- Ahmad M
- Ahmad WH
- Ahmed I
- Ahmed W
- Ahuja S
- Aisa D
- Aisa S
- Akchurin N
- Akgun B
- Akgun U
- Akimenko S
- Akin IV
- Alagoz E
- Alampi G
- Albajar C
- Albayrak EA
- Alberdi J
- Albergo S
- Albert E
- Albrow M
- Alexander J
- Alidra M
- Aliev T
- Allfrey P
- Almeida N
- Altenhofer G
- Altsybeev I
- Alver B
- Alverson G
- Alves GA
- Amaglobeli N
- Amapane N
- Ambroglini F
- Amsler C
- Anagnostou G
- Ananthan B
- Anastassov A
- Andelin D
- Anderson M
- Andrea J
- Andreev V
- Andreev Y
- Anghel IM
- Anguelov T
- Anh T. Vu
- Anisimov A
- Antillon E
- Antipov P
- Antonelli L
- Anttila E
- Antunes JR
- Antunovic Z
- Apanasevich L
- Apollinari G
- Apresyan A
- Arce P
- Arcidiacono R
- Arenton MW
- Arfaei H
- Argiro S
- Arisaka K
- Arneodo M
- Arnold B
- Arora S
- Artamonov A
- Asaadi J
- Asghar MI
- Ashby S
- Askew A
- Atac M
- Atramentov O
- Auffray E
- Aurisano A
- Autermann C
- Avery P
- Avetisyan A
- Avila C
- Awan MIM
- Ayan AS
- Ayhan A
- Azhgirey I
- Aziz T
- Azzi P
- Azzurri P
- Baarmand MM
- Babb J
- Babucci E
- Baccaro S
- Bacchetta N
- Bacchi W
- Bachtis M
- Baden D
- Badgett W
- Baechler J
- Baer H
- Baesso P
- Baffioni S
- Bagby L
- Bagliesi G
- Bahk SY
- Bailleux D
- Baillon P
- Bainbridge R
- Bakhshiansohi H
- Bakirci MN
- Bakken JA
- Balazs M
- Baldin B
- Ball G
- Ball H
- Ballin J
- Bally SL
- Ban Y
- Bandurin D
- Banerjee S
- Banerjee S
- Banicz K
- Bansal S
- Banzuzi K
- Barashko V
- Barbagli G
- Barberis E
- Barbone L
- Barcala JM
- Barcellan L
- Bard R
- Bargassa P
- Baringer P
- Barnes VE
- Barnett BA
- Barney D
- Barone L
- Bartalini P
- Bartoloni A
- Bartz E
- Basegmez S
- Battilana C
- Baty C
- Baud A
- Bauer G
- Bauer J
- Bauerdick LAT
- Baur U
- Bawa HS
- Bazterra VE
- Bean A
- Beauceron S
- Beaudette F
- Beaumont W
- Bechtel F
- Bedjidian M
- Bedoya CF
- Beetz CP
- Behrens U
- Bejar JC
- Belforte S
- Beliy N
- Bell KW
- Bellan P
- Bellan R
- Bellato M
- Bellinger JN
- Belotelov I
- Benaglia A
- Bencze G
- Bendavid J
- Bender W
- Benedetti D
- Benelli G
- Benettoni M
- Beni N
- Benucci L
- Benussi L
- Benvenuti AC
- Berengueres JOL
- Beretvas A
- Bergauer H
- Bergauer T
- Beri SB
- Bernardini J
- Bernet C
- Berntzon L
- Berretta L
- Berry D
- Berry E
- Berryhill J
- Bertani M
- Bertl W
- Bertoldi M
- Berzano U
- Besancon M
- Besson A
- Betchart B
- Betev B
- Betts RR
- Beuselinck R
- Bhat PC
- Bhatnagar V
- Bhattacharya S
- Bhattacharya S
- Bhatti A
- Biallass P
- Bianchini L
- Bianco S
- Biasini M
- Biasotto M
- Biery K
- Biino C
- Bilei GM
- Bilki B
- Bilmis S
- Binkley M
- Bisello D
- Bitioukov S
- Blaha J
- Blekman F
- Bloch D
- Bloch I
- Bloch P
- Bloom K
- Bluj M
- Blum P
- Blumenfeld B
- Blyweert S
- Boccali T
- Bocci A
- Bockelman B
- Bodek A
- Bodin D
- Boeriu O
- Boldini M
- Boldizsar L
- Bolla G
- Bolognesi S
- Bolton T
- Bona M
- Bonacorsi D
- Bonato A
- Bondar N
- Bontenackels M
- Boos E
- Borcherding F
- Borgia MA
- Bornheim A
- Borras K
- Borrello L
- Borsato E
- Bortoletto D
- Bos J
- Bose M
- Bose S
- Bose T
- Bosi F
- Bostock F
- Botta C
- Boudoul G
- Bouhali O
- Bourgeois N
- Bourilkov D
- Bourrel T
- Boutemeur M
- Boutle S
- Braibant-Giacomelli S
- Branca A
- Branson JG
- Brauer R
- Braunschweig W
- Breedon R
- Brett AM
- Breuker H
- Brew C
- Bricola S
- Briggs R
- Brigljevic V
- Broccolo G
- Brom JM
- Brooke JJ
- Brown RM
- Brun H
- Bruno G
- Buchmuller O
- Budd H
- Buege V
- Buehler M
- Bunin P
- Bunkowski K
- Bunn J
- Buontempo S
- Burkett K
- Burtovoy V
- Busson P
- Busza W
- Butler JN
- Butler PH
- Butt J
- Butz E
- Bylsma B
- Caballero IG
- Cabrillo IJ
- Cafaro VD
- Cai J
- Caiazza SS
- Cakir A
- Cakir I. Turk
- Calderon A
- Cali IA
- Callner J
- Calloni M
- Calvo E
- Calzolari F
- Camanzi B
- Caminada L
- Campagnari C
- Campbell A
- Campderros JD
- Campi D
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- Cankocak K
- Cano E
- Capiluppi P
- Caponeri B
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- Carlsmith D
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- Cartiglia N
- Carvalho W
- Case M
- Cassel D
- Castaldi R
- Castellani L
- Castello R
- Castro A
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- Castro MA
- Cattai A
- Caudron J
- Cavallari F
- Cavallo FR
- Cavallo N
- Cavanaugh R
- Ceballos GG
- Cebra D
- Cepeda M
- Cerati GB
- Cerci S
- Cerizza G
- Cerminara G
- Ceron C
- Cerrada M
- Chabert EC
- Chan M
- Chandra A
- Chang P
- Chang S
- Chang YH
- Chanon N
- Chao Y
- Charaf O
- Charlot C
- Chatelain JP
- Chatrchyan S
- Chatterjee A
- Chauhan S
- Chauvey M
- Checchia P
- Checcucci B
- Chekhovsky V
- Chen EA
- Chen GM
- Chen HS
- Chen J
- Chen KF
- Chen M
- Chen WT
- Chen Z
- Cheng TL
- Chertok M
- Chetluru V
- Cheung HWK
- Chien CY
- Chierici R
- Chiochia V
- Chiorboli M
- Chipaux R
- Chiumarulo F
- Chlebana F
- Choi M
- Choi S
- Choi Y
- Chou JP
- Choudhary BC
- Choudhury RK
- Christian G
- Christiansen T
- Chtchipounov L
- Chuang SH
- Chung J
- Chung K
- Chung YS
- Churin I
- Chwalek T
- Cihangir S
- Cimmino A
- Cirino G
- Cittolin S
- Ciulli V
- Civinini C
- Claes DR
- Clare R
- Clarida W
- Clemente A
- Clemente F
- Clerbaux B
- Cline D
- Cockerill DJA
- Codispoti G
- Colafranceschi S
- Colaleo A
- Cole JE
- Colino N
- Colling D
- Colonna D
- Conetti S
- Contardo D
- Conte E
- Conti E
- Conway J
- Cooper SI
- Cordonnier AM
- Cortabitarte RV
- Cossutti F
- Costa M
- Costa S
- Coughlan JA
- Cousins R
- Covarelli R
- Cox B
- Cox PT
- Crawford M
- Creanza D
- Cremaldi LM
- Cripps N
- Crotty I
- Cuevas J
- Cuffiani M
- Cumalat JP
- Cuplov V
- Cure B
- Cuscela G
- Cushman P
- Cussans D
- Cutts D
- Cwiok M
- Czellar S
- D'Alessandro R
- D'Alfonso M
- D'Angelo P
- D'Antone I
- D'Enterria D
- D'Hondt J
- Dabrowski R
- Dafinei I
- Dagenhart W
- Dahmes B
- Dal Corso F
- Dallavalle GM
- Dambach S
- Damgov J
- Damiao DD
- Dammann D
- Daniel M
- Danielson T
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- Daskalakis G
- Dasu S
- Dattola D
- Daubie E
- David A
- Davids M
- Davies G
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- de Barbaro P
- De Benedetti A
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- de Cassagnac RG
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- De Gruttola M
- De Guio F
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- De Mattia M
- de Monchenault GH
- De Palma M
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- De Souza SF
- de Troconiz JF
- De Visscher S
- De Weirdt S
- De Wolf EA
- Debbins P
- Debreczeni G
- Deiters K
- Dejardin M
- Del Alamo MB
- del Arbol PMR
- Del Re D
- Delachenal V
- Delaere C
- Deliomeroglu M
- Dell'Orso R
- Della Negra M
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- Dellacasa G
- Delmeire E
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- Demarteau M
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- Denegri D
- Denisov A
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- Dermenev A
- Dero V
- Derylo G
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- Dharmaratna WGD
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- Di Giovanni GP
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- Diemoz M
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- Dimitrov A
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- Dinardo ME
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- Dittmar M
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- Djordjevic M
- Do Amaral SMS
- Dobrzynski L
- Dobur D
- Dolen J
- Dolgopolov A
- Dominguez A
- Dominik W
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- Draeger J
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- Dragoiu C
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- Drozdetskiy A
- Druzhkin D
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- Dumanoglu I
- Dumitrache F
- Dupasquier T
- Dupont T
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- Durkin LS
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- Dutta D
- Dutta S
- Dvornikov O
- Dykstra D
- Dyulendarova M
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- Eartly DP
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- Ecklund KM
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- Fasanella D
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- Ferrando A
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- Hatakeyama K
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- Heering AH
- Hegner B
- Heier S
- Heikkinen A
- Heinrich M
- Heister A
- Hektor A
- Held H
- Heltsley B
- Hermanns T
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- Hernath S
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- Heyninck J
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- Iglesias LL
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- Wingham M
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- Wu S
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- Wulf E.
- Wulz CE
- Wurthwein F
- Wynne B. M.
- Wyslouch B
- Xaplanteris L.
- Xella S.
- Xie S
- Xie S.
- Xie Z
- Xu D.
- Xu N.
- Xue Z
- Yagil A
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- Yan M
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- Yang ZC
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- Yilmaz Y
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- Yun JC
- Yuste C
- Zabi A
- Zabolotny W
- Zachariadou A
- Zalewski P
- Zampieri A
- Zanetti M
- Zang SL
- Zarubin A
- Zatzerklyany A
- Zeidler C
- Zeinali M
- Zeise M
- Zelepoukine S
- Zeuner WD
- Zeyrek M
- Zhang J
- Zhang L
- Zhang Y
- Zhang Z
- Zheng Y
- Zhiltsov V
- Zhokin A
- Zhu B
- Zhu K
- Zhu RY
- Zhukov V
- Zhukova V
- Ziebarth EB
- Zielinski M
- Zilizi G
- Zinonos Z
- Zito G
- Zoeller MH
- Zotto P
- Zub S
- Zumerle G
- Zuranski A
- Zuyeuski R
- Zych P
- Publication venue
- 'IOP Publishing'
- Publication date
- 01/01/2009
- Field of study
The Cathode Strip Chambers (CSCs) constitute the primary muon tracking device
in the CMS endcaps. Their performance has been evaluated using data taken
during a cosmic ray run in fall 2008. Measured noise levels are low, with the
number of noisy channels well below 1%. Coordinate resolution was measured for
all types of chambers, and fall in the range 47 microns to 243 microns. The
efficiencies for local charged track triggers, for hit and for segments
reconstruction were measured, and are above 99%. The timing resolution per
layer is approximately 5 ns
The A-Current Modulates Learning via NMDA Receptors Containing the NR2B Subunit
- Author
- A Fontán-Lozano
- A Fontán-Lozano
- A Fontán-Lozano
- A Frick
- A Gruart
- A Lockridge
- A Ruiz-Gomez
- AJ Norris
- AM Carrion
- C Bellone
- C Lilliehook
- CG Thomas
- Colin Combs
- D Campos
- D Johnston
- D Johnston
- DA Hoffman
- David González-Forero
- E Klann
- ER Kandel
- F Ledo
- F Ledo
- G Daoudal
- G Kohr
- GM Ramakers
- HY Cheng
- Irene Suárez-Pereira
- J Kim
- J Kim
- J Kim
- JC Alexander
- JF Storm
- JH Goldberg
- K Takimoto
- KJ Rhodes
- LJ Wu
- LR Squire
- M de los Santos-Arteaga
- M Maletic-Savatic
- M Menegola
- M Rivas
- M Sheng
- MA Sutton
- MC Inda
- NE Schoppa
- R Romero-Granados
- R Shibata
- RA Al-Hallaq
- RC Malenka
- RJ Kelleher 3rd
- S Cash
- S Watanabe
- SC Jung
- SC Jung
- TV Bliss
- WF An
- X Chen
- Y Dudai
- Y Zhang
- Z Lei
- Ángel Manuel Carrión
- Ángela Fontán-Lozano
- Publication venue
- Public Library of Science
- Publication date
- 26/09/2011
- Field of study
Synaptic plasticity involves short- and long-term events, although the molecular mechanisms that underlie these processes are not fully understood. The transient A-type K+ current (IA) controls the excitability of the dendrites from CA1 pyramidal neurons by regulating the back-propagation of action potentials and shaping synaptic input. Here, we have studied how decreases in IA affect cognitive processes and synaptic plasticity. Using wild-type mice treated with 4-AP, an IA inhibitor, and mice lacking the DREAM protein, a transcriptional repressor and modulator of the IA, we demonstrate that impairment of IA decreases the stimulation threshold for learning and the induction of early-LTP. Hippocampal electrical recordings in both models revealed alterations in basal electrical oscillatory properties toward low-theta frequencies. In addition, we demonstrated that the facilitated learning induced by decreased IA requires the activation of NMDA receptors containing the NR2B subunit. Together, these findings point to a balance between the IA and the activity of NR2B-containing NMDA receptors in the regulation of learning
Global dataset of soil organic carbon in tidal marshes.
- Author
- Adame MF
- Adams JB
- Antisari LV
- Austin WEN
- Beasy K
- Boscutti F
- Bouma TJ
- Bulmer RH
- Burden A
- Burke SA
- Böttcher ME
- Camacho S
- Chaudhary DR
- Chmura GL
- Copertino M
- Cott GM
- Craft C
- Day J
- de Los Santos CB
- Denis L
- Ding W
- Ellison JC
- Ewers Lewis CJ
- Giani L
- Gispert M
- Gontharet S
- González-Alcaraz MN
- González-Pérez JA
- Gorham C
- Graversen AEL
- Grey A
- Guerra R
- He Q
- Holmquist JR
- Jones AR
- Juanes JA
- Kelleher BP
- Kohfeld KE
- Krause-Jensen D
- Lafratta A
- Landis E
- Lavery PS
- Laws EA
- Leiva-Dueñas C
- Loh PS
- Lovelock CE
- Lundquist CJ
- Macreadie PI
- Maxwell TL
- Mazarrasa I
- Megonigal JP
- Neto JM
- Nogueira J
- Osland MJ
- Pagès JF
- Perera N
- Pfeiffer E-M
- Pollmann T
- Raw JL
- Recio M
- Rovai AS
- Ruiz-Fernández AC
- Russell SK
- Rybczyk JM
- Sammul M
- Sanders C
- Santos R
- Serrano O
- Siewert M
- Smart L
- Smeaton C
- Song Z
- Spalding M
- Trasar-Cepeda C
- Twilley RR
- Van de Broek M
- Vitti S
- Voltz B
- Wails CN
- Ward M
- Ward RD
- Wolfe J
- Worthington TA
- Yang R
- Zubrzycki S
- Álvarez-Rogel J
- Publication venue
- Nature Research
- Publication date
- 11/11/2023
- Field of study
Tidal marshes store large amounts of organic carbon in their soils. Field data quantifying soil organic carbon (SOC) stocks provide an important resource for researchers, natural resource managers, and policy-makers working towards the protection, restoration, and valuation of these ecosystems. We collated a global dataset of tidal marsh soil organic carbon (MarSOC) from 99 studies that includes location, soil depth, site name, dry bulk density, SOC, and/or soil organic matter (SOM). The MarSOC dataset includes 17,454 data points from 2,329 unique locations, and 29 countries. We generated a general transfer function for the conversion of SOM to SOC. Using this data we estimated a median (± median absolute deviation) value of 79.2 ± 38.1 Mg SOC ha-1 in the top 30 cm and 231 ± 134 Mg SOC ha-1 in the top 1 m of tidal marsh soils globally. This data can serve as a basis for future work, and may contribute to incorporation of tidal marsh ecosystems into climate change mitigation and adaptation strategies and policies
Aptamers for pharmaceuticals and their application in environmental analytics
- Author
- A Arola
- A Sassolas
- A Shoji
- A Vallee-Belisle
- A Wochner
- A Wochner
- A Wochner
- AA Rowe
- AC Connor
- AD Ellington
- AD Ellington
- AE Radi
- AL Nuijs van
- AM Evans
- B Kasprzyk-Hordern
- B Sun
- BR Baker
- Bundesministeriums für Gesundheit und Umweltbundesamt
- C Berens
- C Ma
- C Mannironi
- C Niemeyer
- C Tuerk
- CB Joeng
- CE Tucker
- CK O’Sullivan
- DH Burke
- DP Morse
- DP Wernette
- DS Alberts
- E Gonzalez-Fernandez
- E Katz
- E Zuccato
- FW Scheller
- G Zagotto
- GD Huy
- GJ Martin
- GL Nielsen
- H Han
- H Noppe
- H Park
- H Schapiro
- H Schürer
- H Schürer
- H Xiao
- HF Noller
- HJ Kim
- I Willner
- J Chen
- J Rogers
- J Schwaiger
- JA Chapman
- JA Cowan
- JC Jennings
- JE Vandenengel
- JF Lee
- JG Bruno
- JH Niazi
- JH Niazi
- JL Oaks
- JS Swensen
- JW Liu
- JW Liu
- JY Cho
- K Hamasaki
- K Kern
- KJ Cash
- KM Song
- KP Williams
- L Gold
- L Jiang
- L Jiang
- L Kiesel
- LC Jiang
- M Cargouet
- M Cleuvers
- M Djordjevic
- M Famulok
- M Huerta-Fontela
- M Kim
- M Kwon
- M Leone
- M Wagner
- MA Kohanski
- MAA Siddiqui
- MC Hammond-Kosack
- MG Wallis
- MG Wallis
- MN Stojanovic
- MN Stojanovic
- MN Stojanovic
- MN Win
- MR Ladisch
- MR Servos
- N de-los-Santos-Alvarez
- N Pastor-Navarro
- N Piganeau
- NA Tritos
- OA Sadik
- P Calik
- P Cekan
- P Chen
- PA Doris
- PD Hansen
- PJ Barnes
- R Condray
- R Freeman
- R Savica
- R Schroeder
- R Stoltenburg
- R Stoltenburg
- R Stoltenburg
- R Walsh
- R White
- RAC Roos
- RD Jenison
- RF Little
- S Jensen
- S Klussmann
- S Leva
- S Shultz
- S Stampfl
- S Sun
- S Tombelli
- SCB Gopinath
- SE Jorgensen
- SJ Klug
- SK Teo
- SM Lato
- SM Lato
- ST Wallace
- T Hermann
- T Mairal
- T Simonsson
- TA Ternes
- V Tereshko
- W Yoshida
- W Yoshida
- XL Zuo
- Y Du
- Y Du
- Y Du
- Y Hashimoto
- Y Jin
- Y Li
- Y Tor
- Y Wang
- Y Wang
- Y Xiao
- Y Xiao
- YJ Kim
- YS Huh
- YS Kim
- YS Kim
- YS Kim
- YS Kim
- Publication venue
- Springer Vienna
- Publication date
- 01/01/2011
- Field of study
Aptamers are single-stranded DNA or RNA oligonucleotides, which are able to bind with high affinity and specificity to their target. This property is used for a multitude of applications, for instance as molecular recognition elements in biosensors and other assays. Biosensor application of aptamers offers the possibility for fast and easy detection of environmental relevant substances. Pharmaceutical residues, deriving from human or animal medical treatment, are found in surface, ground, and drinking water. At least the whole range of frequently administered drugs can be detected in noticeable concentrations. Biosensors and assays based on aptamers as specific recognition elements are very convenient for this application because aptamer development is possible for toxic targets. Commonly used biological receptors for biosensors like enzymes or antibodies are mostly unavailable for the detection of pharmaceuticals. This review describes the research activities of aptamer and sensor developments for pharmaceutical detection, with focus on environmental applications
Multi-messenger observations of a binary neutron star merger
- Author
- Aab A
- Aartsen MG
- Abbott BP
- Abbott R
- Abbott TD
- Abbott TMC
- Abdalla H
- Abe F
- Abeysekara AU
- Abramo LR
- Abramowski A
- Abreu P
- Acernese F
- Acero F
- Ackermann M
- Ackley K
- Ackley K
- Adams C
- Adams J
- Adams SM
- Adams T
- Addesso P
- Adhikari RX
- Adya VB
- Affeldt C
- Afrough M
- Agarwal B
- Agathos M
- Agatsuma K
- Aggarwal N
- Aglietta M
- Agliozzo C
- Agudo I
- Aguiar OD
- Aguilar JA
- Aharonian F
- Ahlers M
- Ahrens M
- Aiello L
- Ain A
- Ajith P
- Akras S
- Al Samarai I
- Albert A
- Albert A
- Albuquerque IFM
- Albury JM
- Alcaniz JS
- Alexander KD
- Alfaro R
- Alighieri S Di Serego
- Allam S
- Allekotte I
- Allen B
- Allen G
- Allison J
- Allison JR
- Allocca A
- Almela A
- Altin PA
- Altmann D
- Alvarez C
- Alvarez JD
- Alvarez M Dovale
- Alvarez-Muiz J
- Amati L
- Amato A
- An T
- Ananyeva A
- Anastasi GA
- Anchordoqui L
- Andeen K
- Anderson JP
- Anderson SB
- Anderson T
- Anderson WG
- Andrada B
- Andre M
- Andreoni I
- Andreoni I
- Andringa S
- Angelova SV
- Anghinolfi M
- Angner EO
- Angus CR
- Annis J
- Ansseau I
- Antier S
- Anton G
- Antonelli LA
- Antonelli LA
- Anupama GC
- Aoki K
- Aoki W
- Appert S
- Aptekar RL
- Arai K
- Arakawa M
- Aramo C
- Araya MC
- Arcavi I
- Arceo R
- Ardid M
- Areeda JS
- Aresu G
- Argan A
- Argelles C
- Arnaud N
- Array LWA Long Wavelength
- Array MWA Murchison Widefield
- Arrieta M
- Arsene N
- Arteaga-Velzquez JC
- Artola R
- Arun KG
- Asakura Y
- Asaoka Y
- Ascenzi S
- Ashall C
- Ashley MCB
- Ashton G
- Asorey H
- Assis P
- Ast M
- Aston SM
- Astone P
- Atallah DV
- ATLAS
- Atwood WB
- Aubert J-J
- Aubert P
- Aublin J
- Auffenberg J
- Aufmuth P
- Aulbert C
- AultONeal K
- Austin C
- Avgitas T
- Avila G
- Avila-Alvarez A
- Awad A Kuotb
- Axani S
- Baar S
- Babak S
- Bachetti M
- Backes M
- Bacon P
- Bader MKM
- Badescu AM
- Bae S
- Bagherpour H
- Bai X
- Bailes M
- Baker PT
- Balaceanu A
- Balanutsa PV
- Balasubramanian A
- Balbinot E
- Baldaccini F
- Baldini L
- Ballardin G
- Ballmer SW
- Bally J
- Balzer A
- Banagiri S
- Banerji M
- Bannister KW
- Barayoga JC
- Barbarino C
- Barbato F
- Barber AS
- Barbiellini G
- Barbiellini G
- Barclay SE
- Baret B
- Barish BC
- Barker D
- Barkett K
- Barnard M
- Barnes J
- Barone F
- Barr B
- Barrios-Marti J
- Barron JP
- Barsotti L
- Barsuglia M
- Barta D
- Barthelmy SD
- Bartlett J
- Bartos I
- Barway S
- Barway S
- Barwick SW
- Basa S
- Bassiri R
- Basti A
- Bastieri D
- Batch JC
- Bauer FE
- Bauer FE
- Baum V
- Bawaj M
- Bay R
- Bayley JC
- Bazzan M
- Bazzano A
- Bchele M
- Beardmore AP
- Beardsley A
- Beatty JJ
- Becerril A
- Becherini Y
- Bechtol K
- Becker KH
- Becsy B
- Beer C
- Bejger M
- Belahcene I
- Belhorma B
- Bell AS
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- Botticella MT
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- Funk S
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- Furusawa H
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- Publication venue
- 'American Astronomical Society'
- Publication date
- 01/01/2017
- Field of study
On 2017 August 17 a binary neutron star coalescence candidate (later designated GW170817) with merger time 12:41:04 UTC was observed through gravitational waves by the Advanced LIGO and Advanced Virgo detectors. The Fermi Gamma-ray Burst Monitor independently detected a gamma-ray burst (GRB 170817A) with a time delay of ~1.7 s with respect to the merger time. From the gravitational-wave signal, the source was initially localized to a sky region of 31 deg2 at a luminosity distance of 40+8-8 Mpc and with component masses consistent with neutron stars. The component masses were later measured to be in the range 0.86 to 2.26 Mo. An extensive observing campaign was launched across the electromagnetic spectrum leading to the discovery of a bright optical transient (SSS17a, now with the IAU identification of AT 2017gfo) in NGC 4993 (at ~40 Mpc) less than 11 hours after the merger by the One- Meter, Two Hemisphere (1M2H) team using the 1 m Swope Telescope. The optical transient was independently detected by multiple teams within an hour. Subsequent observations targeted the object and its environment. Early ultraviolet observations revealed a blue transient that faded within 48 hours. Optical and infrared observations showed a redward evolution over ~10 days. Following early non-detections, X-ray and radio emission were discovered at the transient’s position ~9 and ~16 days, respectively, after the merger. Both the X-ray and radio emission likely arise from a physical process that is distinct from the one that generates the UV/optical/near-infrared emission. No ultra-high-energy gamma-rays and no neutrino candidates consistent with the source were found in follow-up searches. These observations support the hypothesis that GW170817 was produced by the merger of two neutron stars in NGC4993 followed by a short gamma-ray burst (GRB 170817A) and a kilonova/macronova powered by the radioactive decay of r-process nuclei synthesized in the ejecta
Canagliflozin and Renal Outcomes in Type 2 Diabetes and Nephropathy
- Author
- Abdallah J
- Abdul Ghani R
- Abdul Kadir K
- Abdullah R
- Abejuela Z
- Abouglila K
- Abraham O
- Abrahamsen I
- Abraira Munoz Ld
- Abramowitz M
- Abusnana S
- Accini Mendoza Jl
- Acosta I
- Agafyina A
- Agarwal R
- Agarwal Rajiv
- Aggarwal N
- Agra J
- Ahmed F
- Ahuad Guerrero Ra
- Aiello J
- Ainsworth P
- Aizenberg D
- Akiyama H
- Akright L
- Akyea-Djamson A
- Al-Karadsheh A
- Alamartine E
- Alappan R
- Albisu Jp
- Ali N
- Ali Z
- Alicic R
- Allison Dc
- Alvarez Garcia P
- Alvarisqueta A
- Alves da Costa Fa
- Amerena J
- Amigo A
- Amodeo C
- Andreeva V
- Andreotti Turatti La
- Angelescu Lm
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- Anghel V
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- Aoki H
- Aquitania G
- Arauz-Pacheco C
- Arcos E
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- Zanoli L
- Zaoui P
- Zateyshchikova A
- Zateyshshikov D
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- Zemek S
- Zhang H
- Zhang Hong
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- Zheng H
- Zinman B
- Zinman Bernard
- Zlova T
- Zsom M
- Zub L
- Zykova T
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2019
- Field of study
BACKGROUND Type 2 diabetes mellitus is the leading cause of kidney failure worldwide, but few effective long-term treatments are available. In cardiovascular trials of inhibitors of sodium–glucose cotransporter 2 (SGLT2), exploratory results have suggested that such drugs may improve renal outcomes in patients with type 2 diabetes. METHODS In this double-blind, randomized trial, we assigned patients with type 2 diabetes and albuminuric chronic kidney disease to receive canagliflozin, an oral SGLT2 inhibitor, at a dose of 100 mg daily or placebo. All the patients had an estimated glomerular filtration rate (GFR) of 30 to 300 to 5000) and were treated with renin–angiotensin system blockade. The primary outcome was a composite of end-stage kidney disease (dialysis, transplantation, or a sustained estimated GFR of <15 ml per minute per 1.73 m 2), a doubling of the serum creatinine level, or death from renal or cardiovascular causes. Prespecified secondary outcomes were tested hierarchically. RESULTS The trial was stopped early after a planned interim analysis on the recommendation of the data and safety monitoring committee. At that time, 4401 patients had undergone randomization, with a median follow-up of 2.62 years. The relative risk of the primary outcome was 30% lower in the canagliflozin group than in the placebo group, with event rates of 43.2 and 61.2 per 1000 patient-years, respectively (hazard ratio, 0.70; 95% confidence interval [CI], 0.59 to 0.82; P=0.00001). The relative risk of the renal-specific composite of end-stage kidney disease, a doubling of the creatinine level, or death from renal causes was lower by 34% (hazard ratio, 0.66; 95% CI, 0.53 to 0.81; P<0.001), and the relative risk of end-stage kidney disease was lower by 32% (hazard ratio, 0.68; 95% CI, 0.54 to 0.86; P=0.002). The canagliflozin group also had a lower risk of cardiovascular death, myocardial infarction, or stroke (hazard ratio, 0.80; 95% CI, 0.67 to 0.95; P=0.01) and hospitalization for heart failure (hazard ratio, 0.61; 95% CI, 0.47 to 0.80; P<0.001). There were no significant differences in rates of amputation or fracture. CONCLUSIONS In patients with type 2 diabetes and kidney disease, the risk of kidney failure and cardiovascular events was lower in the canagliflozin group than in the placebo group at a median follow-up of 2.62 years
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- Author
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- Zachariadou A
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- Publication venue
- INSTITUTE OF PHYSICS PUBLISHING
- Publication date
- 01/01/2009
- Field of study
Peer reviewe
Antiinflammatory Therapy with Canakinumab for Atherosclerotic Disease
- Author
- A Abbate
- A Abhyankar
- A Adams
- A Agafina
- A Akyea-Djamson
- A Alan
- A Alfieri
- A Alfrey
- A Alvarisqueta
- A Amos
- A Anadiotis
- A Anneveldt
- A Antolick
- A Arthur
- A Aslam
- Abaci F
- Abdullakutty J
- Abhaichand R
- Abib E Jr
- Aboufakher R
- Abrantes J
- Abreu M
- Abulencia K
- Acampora D
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- Aceto L
- Acevedo B
- Acevedo L
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- Aggarwal Rk
- Agosta Gf
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- Aguilar M
- Ahire N
- Ahmad I
- Ahmed Sh
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- Aiub J
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- Akatova E
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- Al-Khalili F
- Albisu J
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- Amerena J
- Andersen K
- Andor M
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- Angiolillo D
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- Ayala M
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- D Denham
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- Da Silva O Jr
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- Zineldine A.
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- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2017
- Field of study
Background: Experimental and clinical data suggest that reducing inflammation without affecting lipid levels may reduce the risk of cardiovascular disease. Yet, the inflammatory hypothesis of atherothrombosis has remained unproved. Methods: We conducted a randomized, double-blind trial of canakinumab, a therapeutic monoclonal antibody targeting interleukin-1β, involving 10,061 patients with previous myocardial infarction and a high-sensitivity C-reactive protein level of 2 mg or more per liter. The trial compared three doses of canakinumab (50 mg, 150 mg, and 300 mg, administered subcutaneously every 3 months) with placebo. The primary efficacy end point was nonfatal myocardial infarction, nonfatal stroke, or cardiovascular death. RESULTS: At 48 months, the median reduction from baseline in the high-sensitivity C-reactive protein level was 26 percentage points greater in the group that received the 50-mg dose of canakinumab, 37 percentage points greater in the 150-mg group, and 41 percentage points greater in the 300-mg group than in the placebo group. Canakinumab did not reduce lipid levels from baseline. At a median follow-up of 3.7 years, the incidence rate for the primary end point was 4.50 events per 100 person-years in the placebo group, 4.11 events per 100 person-years in the 50-mg group, 3.86 events per 100 person-years in the 150-mg group, and 3.90 events per 100 person-years in the 300-mg group. The hazard ratios as compared with placebo were as follows: in the 50-mg group, 0.93 (95% confidence interval [CI], 0.80 to 1.07; P = 0.30); in the 150-mg group, 0.85 (95% CI, 0.74 to 0.98; P = 0.021); and in the 300-mg group, 0.86 (95% CI, 0.75 to 0.99; P = 0.031). The 150-mg dose, but not the other doses, met the prespecified multiplicity-adjusted threshold for statistical significance for the primary end point and the secondary end point that additionally included hospitalization for unstable angina that led to urgent revascularization (hazard ratio vs. placebo, 0.83; 95% CI, 0.73 to 0.95; P = 0.005). Canakinumab was associated with a higher incidence of fatal infection than was placebo. There was no significant difference in all-cause mortality (hazard ratio for all canakinumab doses vs. placebo, 0.94; 95% CI, 0.83 to 1.06; P = 0.31). Conclusions: Antiinflammatory therapy targeting the interleukin-1β innate immunity pathway with canakinumab at a dose of 150 mg every 3 months led to a significantly lower rate of recurrent cardiovascular events than placebo, independent of lipid-level lowering. (Funded by Novartis; CANTOS ClinicalTrials.gov number, NCT01327846.
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