316 research outputs found
Role of Haptoglobin in Polycystic Ovary Syndrome (PCOS), Obesity and Disorders of Glucose Tolerance in Premenopausal Women
- Author
- A Levy
- A Vermeulen
- AP Levy
- B Bowman
- B Matharoo-Ball
- C Chiellini
- CCJ Kelly
- Cuilin Zhang
- E Codner
- F Alvarez-Blasco
- F Dati
- Francisco Álvarez-Blasco
- G De Pergola
- HF Escobar-Morreale
- HF Escobar-Morreale
- HF Escobar-Morreale
- HF Escobar-Morreale
- Héctor F. Escobar-Morreale
- J Delanghe
- JI Botella-Carretero
- JJ Conn
- JK Zawadzki
- JM Fernandez-Real
- José L. San Millán
- KM Flegal
- M Insenser
- M Langlois
- M Luque-Ramirez
- M Luque-Ramirez
- M Matsuda
- Ma Ángeles Martínez-García
- Manuel Luque-Ramírez
- MR Langlois
- Naiara Parraza
- R Asleh
- R Azziz
- R Azziz
- R Moirand
- RC Christian
- RS Legro
- T Sabuncu
- W Koch
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2009
- Field of study
alleles of the haptoglobin α–chain polymorphism reduce the anti-oxidant properties and increase the pro-inflammatory actions of this acute-phase protein in a gene-dosage fashion. We hypothesized that the haptoglobin polymorphism might contribute to the increased oxidative stress and low-grade chronic inflammation frequently associated with polycystic ovary syndrome, obesity, and abnormalities of glucose tolerance.<0.001), yet no association was found between obesity and haptoglobin genotypes. No differences were observed in haptoglobin levels or genotype frequencies depending on glucose tolerance. Fifty percent of the variation in serum haptoglobin concentrations was explained by the variability in serum C-reactive protein concentrations, BMI, insulin sensitivity and haptoglobin genotypes. alleles suggests that the anti-oxidant and anti-inflammatory properties of haptoglobin may be reduced in these patients
Predictive model of pheochromocytoma based on the imaging features of the adrenal tumours
- Author
- Araujo-Castro M
- Blanco Carrera C
- Calatayud Gutiérrez M
- de Miguel Novoa P
- Escobar-Morreale HF
- Fernández-Ladreda MT
- Garcia Centeno R
- García Gómez Muriel I
- Gracia Gimeno P
- Hanzu F
- Lamas C
- López-García MC
- Marazuela M
- Mora Porta M
- Mínguez Ojeda C
- Parra Ramírez P
- Percovich Hualpa JC
- Robles Lázaro C
- Rojas-Marcos PM
- Sampedro Núñez M
- Valderrábano P
- Valdés Gallego N
- Álvarez Escolà C
- Publication venue
- Publication date
- 17/07/2023
- Field of study
The purpose of our study was to develop a predictive model to rule out pheochromocytoma among adrenal tumours, based on unenhanced computed tomography (CT) and/or magnetic resonance imaging (MRI) features. We performed a retrospective multicentre study of 1131 patients presenting with adrenal lesions including 163 subjects with histological confirmation of pheochromocytoma (PHEO), and 968 patients showing no clinical suspicion of pheochromocytoma in whom plasma and/or urinary metanephrines and/or catecholamines were within reference ranges (non-PHEO). We found that tumour size was significantly larger in PHEO than non-PHEO lesions (44.3 +/- 33.2 versus 20.6 +/- 9.2 mm respectively; P < 0.001). Mean unenhanced CT attenuation was higher in PHEO (52.4 +/- 43.1 versus 4.7 +/- 17.9HU; P < 0.001). High lipid content in CT was more frequent among non-PHEO (83.6% versus 3.8% respectively; P < 0.001); and this feature alone had 83.6% sensitivity and 96.2% specificity to rule out pheochromocytoma with an area under the receiver operating characteristics curve (AUC-ROC) of 0.899. The combination of high lipid content and tumour size improved the diagnostic accuracy (AUC-ROC 0.961, sensitivity 88.1% and specificity 92.3%). The probability of having a pheochromocytoma was 0.1% for adrenal lesions smaller than 20 mm showing high lipid content in CT. Ninety percent of non-PHEO presented loss of signal in the out of phase MRI sequence compared to 39.0% of PHEO (P < 0.001), but the specificity of this feature for the diagnosis of non-PHEO lesions low. In conclusion, our study suggests that sparing biochemical screening for pheochromocytoma might be reasonable in patients with adrenal lesions smaller than 20 mm showing high lipid content in the CT scan, if there are no typical signs and symptoms of pheochromocytoma
M2 Macrophages Activate WNT Signaling Pathway in Epithelial Cells: Relevance in Ulcerative Colitis
- Author
- A Mantovani
- A Riaño
- A Sica
- A Tuin
- AC Vos
- Angeles Álvarez
- C Hernández
- Carlos Hernández
- D Ortiz-Masià
- D Ortiz-Masiá
- D Pinto
- Dolores Ortiz-Masiá
- Emiko Mizoguchi
- G Lee
- H Clevers
- HF Farin
- JB Brown
- JC Fleet
- Jesús Cosín-Roger
- JM Mariadason
- Joaquin Hinojosa
- Juan V. Esplugues
- K Oguma
- K Smith
- KJ Maloy
- KR Hughes
- L Boulter
- LG van der Flier
- M Allez
- M van de Wetering
- Maria D. Barrachina
- MF Neurath
- MM Hunter
- N Barker
- P Henderson
- S Asonuma
- S Chen
- S Dahan
- S Liu
- Sara Calatayud
- SL Lin
- SL Pull
- T Nakamura
- TM Yeung
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2013
- Field of study
Macrophages, which exhibit great plasticity, are important components of the inflamed tissue and constitute an essential element of regenerative responses. Epithelial Wnt signalling is involved in mechanisms of proliferation and differentiation and expression of Wnt ligands by macrophages has been reported. We aim to determine whether the macrophage phenotype determines the expression of Wnt ligands, the influence of the macrophage phenotype in epithelial activation of Wnt signalling and the relevance of this pathway in ulcerative colitis. Human monocyte-derived macrophages and U937-derived macrophages were polarized towards M1 or M2 phenotypes and the expression of Wnt1 and Wnt3a was analyzed by qPCR. The effects of macrophages and the role of Wnt1 were analyzed on the expression of β-catenin, Tcf-4, c-Myc and markers of cell differentiation in a co-culture system with Caco-2 cells. Immunohistochemical staining of CD68, CD206, CD86, Wnt1, β-catenin and c-Myc were evaluated in the damaged and non-damaged mucosa of patients with UC. We also determined the mRNA expression of Lgr5 and c-Myc by qPCR and protein levels of β-catenin by western blot. Results show that M2, and no M1, activated the Wnt signaling pathway in co-culture epithelial cells through Wnt1 which impaired enterocyte differentiation. A significant increase in the number of CD206+ macrophages was observed in the damaged mucosa of chronic vs newly diagnosed patients. CD206 immunostaining co-localized with Wnt1 in the mucosa and these cells were associated with activation of canonical Wnt signalling pathway in epithelial cells and diminution of alkaline phosphatase activity. Our results show that M2 macrophages, and not M1, activate Wnt signalling pathways and decrease enterocyte differentiation in co-cultured epithelial cells. In the mucosa of UC patients, M2 macrophages increase with chronicity and are associated with activation of epithelial Wnt signalling and diminution in enterocyte differentiation
Recommendations for the design of laboratory studies on non-target arthropods for risk assessment of genetically engineered plants
- Author
- A Dutton
- A Raybould
- A Raybould
- A Raybould
- A Raybould
- A Raybould
- A Raybould
- A Rodrigo-Simón
- AC Cohen
- Adinda De Schrijver
- Alan Raybould
- Angharad M. R. Gatehouse
- Annabel Waggoner
- Anthony M. Shelton
- C Grimm
- C Grimm
- C Grimm
- C Ludy
- C Zurbrügg
- CL Wraight
- D Babendreier
- D Stacey
- DE Stanley-Horn
- E Vojtech
- EA Mulligan
- EJM Damme Van
- EPPO
- F Álvarez-Alfageme
- F Álvarez-Alfageme
- G Head
- GP Dively
- H Wandeler
- HA Bell
- HA Bell
- HF Brødsgaard
- J Romeis
- J Romeis
- J Romeis
- J Romeis
- J Romeis
- J Romeis
- J Romeis
- JB Torres
- JD Harwood
- JD Wolt
- JG Lundgren
- JG Lundgren
- JG Lundgren
- JJ Duan
- JJ Duan
- JJ Duan
- JJ Duan
- JJ Duan
- JJ Duan
- JN Perry
- Joseph E. Huesing
- JT Christeller
- Jörg Romeis
- Keri Carstens
- KL Carstens
- L Obrist
- L Thomas
- L Yu
- LA Malone
- LB Obrist
- LB Obrist
- LCH Jesse
- LH Heckmann
- M Chen
- M Chen
- M Garcia-Alonso
- M Meissle
- M Meissle
- MA Marvier
- Marco P. Candolfi
- Morven A. McLean
- N Escher
- N Ferry
- NC Lawo
- NC Lawo
- OECD
- P Aupinel
- P Aupinel
- PAM Hogervorst
- PAM Hogervorst
- PD Jensen
- PF Chapman
- R Konrad
- R Rose
- RA Hill
- RE Shade
- Richard L. Hellmich
- RJ Steidl
- RL Hellmich
- Rod A. Herman
- S Knecht
- SL Brandt
- TH Schuler
- TH Schuler
- TM Caro
- TR Glare
- TS Hoffmeister
- U Heimbach
- U Heimbach
- USEPA
- W Li
- WP Ridley
- WS Abbott
- Y Gao
- Y Gao
- Y Li
- Y Li
- Y Li
- Y Shirai
- YY Bai
- Publication venue
- Springer Netherlands
- Publication date
- 01/01/2010
- Field of study
This paper provides recommendations on experimental design for early-tier laboratory studies used in risk assessments to evaluate potential adverse impacts of arthropod-resistant genetically engineered (GE) plants on non-target arthropods (NTAs). While we rely heavily on the currently used proteins from Bacillus thuringiensis (Bt) in this discussion, the concepts apply to other arthropod-active proteins. A risk may exist if the newly acquired trait of the GE plant has adverse effects on NTAs when they are exposed to the arthropod-active protein. Typically, the risk assessment follows a tiered approach that starts with laboratory studies under worst-case exposure conditions; such studies have a high ability to detect adverse effects on non-target species. Clear guidance on how such data are produced in laboratory studies assists the product developers and risk assessors. The studies should be reproducible and test clearly defined risk hypotheses. These properties contribute to the robustness of, and confidence in, environmental risk assessments for GE plants. Data from NTA studies, collected during the analysis phase of an environmental risk assessment, are critical to the outcome of the assessment and ultimately the decision taken by regulatory authorities on the release of a GE plant. Confidence in the results of early-tier laboratory studies is a precondition for the acceptance of data across regulatory jurisdictions and should encourage agencies to share useful information and thus avoid redundant testing
Chromatin remodelling factor SMARCD2 regulates transcriptional networks controlling differentiation of neutrophil granulocytes
- Author
- A Alajem
- A Khanna-Gupta
- A Komiyama
- A Rau
- AD Friedman
- Alejandro A Schäffer
- AR Quinlan
- Asbjørg Stray-Pedersen
- BG Wilson
- BR Cairns
- C Hall
- C Liongue
- Christian Mertes
- Christoph Klein
- Christoph Ziegenhain
- CT Griffin
- D Croft
- D Kotlarz
- D Álvarez-Errico
- Daniel Petersheim
- Eckhard Wolf
- Ehsan Bahrami
- EO Glocker
- F Ellett
- FJ Sedlazeck
- FM Cernilogar
- G Bohn
- Graham J Lieschke
- H Cedar
- H Hu
- H Li
- H Li
- H Lickert
- Hans-Peter Horny
- Heinrich Schmidt
- HF Lin
- HT Huang
- IL de la Serna
- J Breton-Gorius
- J Harrow
- J Krosl
- J Krumsiek
- J Lekstrom-Himes
- J Shi
- JA Dahl
- JA Gagnon
- JA Lekstrom-Himes
- Jacek Puchałka
- JD Buenrostro
- JI Gallin
- Julien Gagneur
- LJ Zhu
- M Adli
- M Buscarlet
- M Koike
- M Koulnis
- M Lawrence
- M Lek
- M Milacic
- M Tanaka
- Maik Dahlhoff
- Marlon R Schneider
- Maximilian Witzel
- ME Newman
- Meino Rohlfs
- MI Love
- Miguel R Abboud
- MY Tolstorukov
- N Iriyama
- ND Meeker
- P Krawitz
- P Shannon
- PB Rahl
- Peter D Arkwright
- Peter M Krawitz
- R Yamanaka
- RF Wynn
- S Picelli
- SA Renshaw
- SH Orkin
- T Gkikopoulos
- T Hamada
- T Wada
- Tomas Racek
- V Jojic
- V Madan
- VA Cruickshank
- Vahid Pazhakh
- VR Ramirez-Carrozzi
- W Verbeek
- Wolfgang Enard
- Y Liao
- Y Liu
- Yanxin Fan
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/05/2017
- Field of study
Traditional Mapuche ecological knowledge in Patagonia, Argentina: fishes and other living beings inhabiting continental waters, as a reflection of processes of change
- Author
- A Begossi
- A Begossi
- A Begossi
- A Begossi
- A Begossi
- A D’Orbigny
- A Escobar
- A Hajduk
- A Ladio
- A Llagostera
- A Moyano
- A Viedma
- AE Valle Del
- AH Ladio
- AH Soldano
- AJ Bath
- AL Silva Da
- AM Miquelarena
- Ana Ladio
- B Berlin
- B Hernández
- B Vilá
- BA Fernández
- C Briones
- C Brown
- C Eyssartier
- C Folke
- C Villagrán
- CA Fernández
- CR El
- D Feeny
- D Santos Fita
- D Wegrzyn
- E Camac
- E Crivelli
- E Hunn
- E Ostrom
- E Ostrom
- EJ Cano-Contreras
- EM Costa-Neto
- EW Moesbach
- F Berkes
- F Berkes
- F Berkes
- F Berkes
- F Berkes
- F Berkes
- F Berkes
- F Tola
- F Trentini
- FO Zuloaga
- FR Barbarán
- G Galafassi
- G Álvarez
- GC Musters
- GF Scarpa
- GJ Martínez
- GJ Osemeobo
- H Zapater
- HF Magalhães
- I Davidson Hunt
- J Aigo
- J Aigo
- J Bengoa
- J Colding
- J Colding
- J Gómez Otero
- J Hernandez
- J Nantes
- J Valbo-Jørgensen
- JA Cordero
- JB Callicott
- JGW Marques
- JM Nolan
- JM Silveira
- JP Barriga
- JP Barriga
- JS Mourão
- JS Mourão
- Juana Aigo
- L Buria
- L Kropff
- L Prates
- LR Parodi
- M Calvo
- M Espósito
- M Gadgil
- M Gusinde
- M Höft
- M Lozada
- M Lucherini
- M Mermoz
- M Olivera
- M Pascual
- M Vargas-Clavijo
- MA Pascual
- MC Medrano
- ME Grebe
- MF Pinto
- MS Reis
- P Arenas
- P Arenas
- P Coña
- P Descola
- P Navarro Floria
- PH Vigliano
- PJ Fargey
- PZ Mora
- R Guber
- R Lista
- R Rozzi
- R Rozzi
- R Rozzi
- RA Ringuelet
- RC Menni
- RE Johannes
- RE Johannes
- RK Sinha
- RM Casamiquela
- RRN Alves
- S Molares
- S Molares
- S Montecino
- S Ortubay
- SJ Tambiah
- SR Carpenter
- T Marini
- TC Silva
- TD Dillehay
- TM Herrmann
- UP Alburquerque
- V Castro
- V Castro
- V Cussac
- V Reyes-García
- VE Cussac
- VM Toledo
- VS Lema
- Y Kuramochi
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Combined measurement of differential and total cross sections in the H → γγ and the H → ZZ* → 4ℓ decay channels at s=13 TeV with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abhayasinghe DK
- Abidi SH
- Abouzeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Adachi S
- Adamczyk L
- Adelman J
- Adersberger M
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmadov F
- Aielli G
- Akatsuka S
- Akilli E
- Akimov AV
- Alberghi GL
- Albert J
- Albicocco P
- Alconada Verzini MJ
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alhroob M
- Ali B
- Alimonti G
- Alison J
- Alkire SP
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alstaty MI
- Alvarez Gonzalez B
- Alviggi MG
- Amadio BT
- Amaral Coutinho Y
- Ambroz L
- Amelung C
- Amidei D
- Amor Dos Santos SP
- Amoroso S
- Amrouche CS
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders JK
- Anderson KJ
- Andreazza A
- Andrei V
- Anelli CR
- Angelidakis S
- Angelozzi I
- Angerami A
- Anisenkov AV
- Annovi A
- Antel C
- Anthony MT
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Aperio Bella L
- Arabidze G
- Arai Y
- Araque JP
- Araujo Ferraz V
- Araujo Pereira R
- Arce ATH
- Ardell RE
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Armbruster AJ
- Armitage LJ
- Armstrong A
- Arnaez O
- Arnold H
- Arratia M
- Arslan O
- Artamonov A
- Artoni G
- Artz S
- Asai S
- Asbah N
- Ashkenazi A
- Asimakopoulou EM
- Asquith L
- Assamagan K
- Astalos R
- Atkin RJ
- Atkinson M
- Atlas Collaboration
- Atlay NB
- Augsten K
- Avolio G
- Avramidou R
- Axen B
- Ayoub MK
- Azuelos G
- Baas AE
- Baca MJ
- Bachacou H
- Bachas K
- Backes M
- Bagnaia P
- Bahmani M
- Bahrasemani H
- Bailey AJ
- Baines JT
- Bajic M
- Bakalis C
- Baker OK
- Bakker PJ
- Bakshi Gupta D
- Baldin EM
- Balek P
- Balli F
- Balunas WK
- Balz J
- Banas E
- Bandyopadhyay A
- Banerjee S
- Bannoura AAE
- Barak L
- Barbe WM
- Barberio EL
- Barberis D
- Barbero M
- Barillari T
- Barisits M-S
- Barkeloo J
- Barklow T
- Barlow N
- Barnea R
- Barnes SL
- Barnett BM
- Barnett RM
- Barnovska-Blenessy Z
- Baroncelli A
- Barone G
- Barr AJ
- Barranco Navarro L
- Barreiro Guimarães Da Costa J
- Barreiro F
- Bartoldus R
- Barton AE
- Bartos P
- Basalaev A
- Bassalat A
- Bates RL
- Batista SJ
- Batlamous S
- Batley JR
- Battaglia M
- Bauce M
- Bauer F
- Bauer KT
- Bawa HS
- Beacham JB
- Beattie MD
- Beau T
- Beauchemin PH
- Bechtle P
- Beck HC
- Beck HP
- Becker K
- Becker M
- Becot C
- Beddall A
- Beddall AJ
- Bednyakov VA
- Bedognetti M
- Bee CP
- Beermann TA
- Begalli M
- Begel M
- Behera A
- Behr JK
- Bell AS
- Bella G
- Bellagamba L
- Bellerive A
- Bellomo M
- Bellos P
- Belotskiy K
- Belyaev NL
- Benary O
- Benchekroun D
- Bender M
- Benekos N
- Benhammou Y
- Benhar Noccioli E
- Benitez J
- Benjamin DP
- Benoit M
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Berge D
- Bergeaas Kuutmann E
- Berger N
- Bergsten LJ
- Beringer J
- Berlendis S
- Bernard NR
- Bernardi G
- Bernius C
- Bernlochner FU
- Berry T
- Berta P
- Bertella C
- Bertoli G
- Bertram IA
- Besjes GJ
- Bessidskaia Bylund O
- Bessner M
- Besson N
- Bethani A
- Bethke S
- Betti A
- Bevan AJ
- Beyer J
- Bianchi RM
- Biebel O
- Biedermann D
- Bielski R
- Bierwagen K
- Biesuz NV
- Biglietti M
- Billoud TRV
- Bindi M
- Bingul A
- Bini C
- Biondi S
- Bisanz T
- Biswal JP
- Bittrich C
- Bjergaard DM
- Black JE
- Black KM
- Blair RE
- Blazek T
- Bloch I
- Blocker C
- Blue A
- Blumenschein U
- Blunier D
- Bobbink GJ
- Bobrovnikov VS
- Bocchetta SS
- Bocci A
- Boerner D
- Bogavac D
- Bogdanchikov AG
- Bohm C
- Boisvert V
- Bokan P
- Bold T
- Boldyrev AS
- Bolz AE
- Bomben M
- Bona M
- Bonilla JS
- Boonekamp M
- Borisov A
- Borissov G
- Bortfeldt J
- Bortoletto D
- Bortolotto V
- Boscherini D
- Bosman M
- Bossio Sola JD
- Bouaouda K
- Boudreau J
- Bouhova-Thacker EV
- Boumediene D
- Bourdarios C
- Boutle SK
- Boveia A
- Boyd J
- Boyko IR
- Bozson AJ
- Bracinik J
- Brahimi N
- Brandt A
- Brandt G
- Brandt O
- Braren F
- Bratzler U
- Brau B
- Brau JE
- Breaden Madden WD
- Brendlinger K
- Brennan AJ
- Brenner L
- Brenner R
- Bressler S
- Brickwedde B
- Briglin DL
- Britton D
- Britzger D
- Brock I
- Brock R
- Brooijmans G
- Brooks T
- Brooks WK
- Brost E
- Broughton JH
- Bruckman De Renstrom PA
- Bruncko D
- Bruni A
- Bruni G
- Bruni LS
- Bruno S
- Brunt BH
- Bruschi M
- Bruscino N
- Bryant P
- Bryngemark L
- Buanes T
- Buat Q
- Buchholz P
- Buckley AG
- Budagov IA
- Buehrer F
- Bugge MK
- Bulekov O
- Bullock D
- Burch TJ
- Burdin S
- Burgard CD
- Burger AM
- Burghgrave B
- Burka K
- Burke S
- Burmeister I
- Burr JTP
- Buschmann E
- Bussey P
- Butler JM
- Buttar CM
- Butterworth JM
- Butti P
- Buttinger W
- Buzatu A
- Buzykaev AR
- Büscher D
- Büscher V
- Cabras G
- Cabrera Urbán S
- Caforio D
- Cai H
- Cairo VMM
- Cakir O
- Calace N
- Calafiura P
- Calandri A
- Calderini G
- Calfayan P
- Callea G
- Caloba LP
- Calvente Lopez S
- Calvet D
- Calvet S
- Calvet TP
- Calvetti M
- Camacho Toro R
- Camarda S
- Camarri P
- Cameron D
- Caminal Armadans R
- Camincher C
- Campana S
- Campanelli M
- Camplani A
- Campoverde A
- Canale V
- Cano Bret M
- Cantero J
- Cao T
- Cao Y
- Capeans Garrido MDM
- Caprini I
- Caprini M
- Capua M
- Carbone RM
- Cardarelli R
- Cardillo FC
- Carli I
- Carli T
- Carlino G
- Carlson BT
- Carminati L
- Carney RMD
- Caron S
- Carquin E
- Carrillo-Montoya GD
- Carrá S
- Casadei D
- Casado MP
- Casha AF
- Casolino M
- Casper DW
- Castelijn R
- Castillo Gimenez V
- Castillo FL
- Castro NF
- Catinaccio A
- Catmore JR
- Cattai A
- Caudron J
- Cavaliere V
- Cavallaro E
- Cavalli D
- Cavalli-Sforza M
- Cavasinni V
- Celebi E
- Ceradini F
- Cerda Alberich L
- Cerqueira AS
- Cerri A
- Cerrito L
- Cerutti F
- Cervelli A
- Cetin SA
- Chafaq A
- Chakraborty D
- Chan SK
- Chan WS
- Chan YL
- Chang P
- Chapman JD
- Charlton DG
- Chau CC
- Chavez Barajas CA
- Che S
- Chegwidden A
- Chekanov S
- Chekulaev SV
- Chelkov GA
- Chelstowska MA
- Chen C
- Chen CH
- Chen H
- Chen J
- Chen J
- Chen S
- Chen SJ
- Chen X
- Chen Y
- Chen Y-H
- Cheng HC
- Cheng HJ
- Cheplakov A
- Cheremushkina E
- Cherkaoui El Moursli R
- Cheu E
- Cheung K
- Chevalier L
- Chiarella V
- Chiarelli G
- Chiodini G
- Chisholm AS
- Chitan A
- Chiu I
- Chiu YH
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- Zimmermann S
- Zinonos Z
- Zinser M
- Ziolkowski M
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zur Nedden M
- Zwalinski L
- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- Elsevier
- Publication date
- 01/01/2018
- Field of study
A combined measurement of differential and inclusive total cross sections of Higgs boson production is performed using 36.1 fb−1 of 13 TeV proton–proton collision data produced by the LHC and recorded by the ATLAS detector in 2015 and 2016. Cross sections are obtained from measured H→γγ and H→ZZ*(→4ℓ event yields, which are combined taking into account detector efficiencies, resolution, acceptances and branching fractions. The total Higgs boson production cross section is measured to be 57.0−5.9 +6.0 (stat.) −3.3 +4.0 (syst.) pb, in agreement with the Standard Model prediction. Differential cross-section measurements are presented for the Higgs boson transverse momentum distribution, Higgs boson rapidity, number of jets produced together with the Higgs boson, and the transverse momentum of the leading jet. The results from the two decay channels are found to be compatible, and their combination agrees with the Standard Model predictions
Search for supersymmetry in events with four or more leptons in √s =13 TeV pp collisions with ATLAS
- Author
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- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abidi SH
- AbouZeid OS
- Abraham NL
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- Zinser M
- Ziolkowski M
- Zobernig G
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- zur Nedden M
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- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2018
- Field of study
Results from a search for supersymmetry in events with four or more charged leptons (electrons, muons and taus) are presented. The analysis uses a data sample corresponding to 36.1 fb −1 of proton-proton collisions delivered by the Large Hadron Collider at s √ =13 TeV and recorded by the ATLAS detector. Four-lepton signal regions with up to two hadronically decaying taus are designed to target a range of supersymmetric scenarios that can be either enriched in or depleted of events involving the production and decay of a Z boson. Data yields are consistent with Standard Model expectations and results are used to set upper limits on the event yields from processes beyond the Standard Model. Exclusion limits are set at the 95% confidence level in simplified models of General Gauge Mediated supersymmetry, where higgsino masses are excluded up to 295 GeV. In R -parity-violating simplified models with decays of the lightest supersymmetric particle to charged leptons, lower limits of 1.46 TeV, 1.06 TeV, and 2.25 TeV are placed on wino, slepton and gluino masses, respectively
Measurement of the t¯tZ and t¯tW cross sections in proton-proton collisions at √s=13 TeV with the ATLAS detector
- Author
- Aaboud M
- Aad G
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- Abbott DC
- Abdinov O
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- Zeng Q
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- Zhao P
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- Zhemchugov A
- Zheng Z
- Zhong D
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- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zur Nedden M
- Zwalinski L
- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2019
- Field of study
A measurement of the associated production of a top-quark pair (t¯t) with a vector boson (W, Z) in proton-proton collisions at a center-of-mass energy of 13 TeV is presented, using 36.1 fb−1 of integrated luminosity collected by the ATLAS detector at the Large Hadron Collider. Events are selected in channels with two same- or opposite-sign leptons (electrons or muons), three leptons or four leptons, and each channel is further divided into multiple regions to maximize the sensitivity of the measurement. The t¯tZ and t¯tW production cross sections are simultaneously measured using a combined fit to all regions. The best-fit values of the production cross sections are σt¯tZ=0.95±0.08stat±0.10syst pb and σt¯tW=0.87±0.13stat±0.14syst pb in agreement with the Standard Model predictions. The measurement of the t¯tZ cross section is used to set constraints on effective field theory operators which modify the t¯tZ vertex
Search for strongly interacting massive particles generating trackless jets in proton-proton collisions at s = 13 TeV
- Author
- Aarrestad TK
- Aarup Petersen H
- Abbaneo D
- Abbiendi G
- Abbrescia M
- Abdullin S
- Abercrombie D
- Acharya H
- Acosta D
- Acosta JG
- Adam W
- Adams E
- Adams MR
- Adams T
- Adloff C
- Adzic P
- Agapitos A
- Agarwal G
- Aggleton R
- Agram J-L
- Aguilar-Benitez M
- Ahmad A
- Ahmad M
- Ahmed A
- Ahuja S
- Aime C
- Akchurin N
- Akgun B
- Akpinar A
- Albajar C
- Albergo S
- Albert A
- Albrow M
- Alcaraz Maestre J
- Aldaya Martin M
- Aldá Júnior WL
- Aleksandrov A
- Alexander J
- Alhusseini M
- Alimena J
- Alison J
- Allen B
- Almond J
- Alvarez Gonzalez B
- Alverson G
- Alves GA
- Aly R
- Alyari M
- Amapane N
- Amendola C
- Amin N
- Amram O
- Amsler C
- Anagnostou G
- Andrea J
- Andreev V
- Andreev Y
- Andrews MB
- Androsov K
- Antchev G
- Antunovic Z
- Apanasevich L
- Apollinari G
- Appelt E
- Apresyan A
- Apyan A
- Araujo M
- Arcaro D
- Arcidiacono R
- Arenton MW
- Argiro S
- Arneodo M
- Aruta C
- Asavapibhop B
- Asawatangtrakuldee C
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- Özçelik Ö
- Široký J
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2022
- Field of study
A search for dark matter in the form of strongly interacting massive particles (SIMPs) using the CMS detector at the LHC is presented. The SIMPs would be produced in pairs that manifest themselves as pairs of jets without tracks. The energy fraction of jets carried by charged particles is used as a key discriminator to suppress efficiently the large multijet background, and the remaining background is estimated directly from data. The search is performed using proton-proton collision data corresponding to an integrated luminosity of 16.1 fb - 1 , collected with the CMS detector in 2016. No significant excess of events is observed above the expected background. For the simplified dark matter model under consideration, SIMPs with masses up to 100 GeV are excluded and further sensitivity is explored towards higher masses
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