3 research outputs found
Iron Behaving Badly: Inappropriate Iron Chelation as a Major Contributor to the Aetiology of Vascular and Other Progressive Inflammatory and Degenerative Diseases
- Author
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- The Long-Term Intervention with Pravastatin in Ischaemic Disease (LIPID) Study Group
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- ZZ Chong
- Publication venue
- Publication date
- 09/08/2008
- Field of study
The production of peroxide and superoxide is an inevitable consequence of
aerobic metabolism, and while these particular "reactive oxygen species" (ROSs)
can exhibit a number of biological effects, they are not of themselves
excessively reactive and thus they are not especially damaging at physiological
concentrations. However, their reactions with poorly liganded iron species can
lead to the catalytic production of the very reactive and dangerous hydroxyl
radical, which is exceptionally damaging, and a major cause of chronic
inflammation. We review the considerable and wide-ranging evidence for the
involvement of this combination of (su)peroxide and poorly liganded iron in a
large number of physiological and indeed pathological processes and
inflammatory disorders, especially those involving the progressive degradation
of cellular and organismal performance. These diseases share a great many
similarities and thus might be considered to have a common cause (i.e.
iron-catalysed free radical and especially hydroxyl radical generation). The
studies reviewed include those focused on a series of cardiovascular, metabolic
and neurological diseases, where iron can be found at the sites of plaques and
lesions, as well as studies showing the significance of iron to aging and
longevity. The effective chelation of iron by natural or synthetic ligands is
thus of major physiological (and potentially therapeutic) importance. As
systems properties, we need to recognise that physiological observables have
multiple molecular causes, and studying them in isolation leads to inconsistent
patterns of apparent causality when it is the simultaneous combination of
multiple factors that is responsible. This explains, for instance, the
decidedly mixed effects of antioxidants that have been observed, etc...Comment: 159 pages, including 9 Figs and 2184 reference
Capital e Poder a serviço da Globalização: os oligopólios da educação superior privada no Brasil
- Author
- ALERIGI JÚNIOR Alberto
- Andreia Gomes da Cruz
- ANTUNES F
- BARREYRO Gladys Beatriz
- BITTAR Mariluce
- BRAGA Ryon
- CARVALHO Cristina Helena Almeida de
- CHAVES Vera Lucia Jacob
- COLADO Eduardo Ibarra
- DELGADO Jaime Ornellas
- DIAS SOBRINHO José
- ERTHAL João Marcelo
- GOIS Ancelmo
- HARTMANN Eva
- KOIKE Beth
- LEHER Roberto
- LEITE Denise
- LEITE Denise
- LIMA Licínio C.
- Maria de Fátima Costa de Paula
- MEIRELLES Jorge Luís Faria
- OLIVEIRA Portela Romualdo
- OSCAR Nayana
- OYAMA Edison Riuitiro
- PAULA Maria de Fatima. Costa de
- PAULA Maria Fátima Costa de
- SANTOS Aline Veiga dos
- SGUISSARDI Valdemar
- SILVA JUNIOR João dos Reis
- SLAUGHTER Sheila
- SORIMA NETO João
- SOUSA Dayanne
- SOUZA Andrea Harada
- TAVARES Pedro Henrique de Souza
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
Guidelines for the use of flow cytometry and cell sorting in immunological studies
- Author
- A. Graham Pockley
- Aalderen
- Abate
- Abdelrahman
- Acosta-Rodriguez
- Agematsu
- Agematsu
- Aghaeepour
- Aghaeepour
- Aghaeepour
- Aghaeepour
- Aghaeepour
- Aichele
- Akkaya
- Akondy
- Alan M. Stall
- Alanio
- Alder
- Alessandro Moretta
- Alexander Scheffold
- Alfadda
- Alfonso Blanco
- Alice Yue
- Alix-Panabières
- Alsayed
- Altman
- Amini
- Amir el
- Amit
- Amy Lovett-Racke
- Ana Cumano
- Anat Shemer
- Anderson
- Anderson
- Anding
- Andrea Cavani
- Andrea Cossarizza
- Andrea Gori
- Andreas Diefenbach
- Andreas Dolf
- Andreas Grützkau
- Andreas Radbruch
- Andrew Filby
- Angelo
- Anis Larbi
- Anita Dreher
- Anja E. Hauser
- Anna Iannone
- Anna Rubartelli
- Anna-Katharina Simon
- Anne Cooke
- Annette Oxenius
- Annika Wiedemann
- Anselmo
- Antonio Cosma
- Appay
- Appay
- Appert-Collin
- Arai
- Ari Waisman
- Armstrong
- Asbury
- Assenmacher
- Attila Tarnok
- Auffray
- Autréaux
- Avey
- Avramidou
- Axel Ronald Schulz
- Baba
- Bacher
- Bacher
- Bacher
- Bacher
- Bacher
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- Baehner
- Baerbel Keller
- Baerlocher
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- Bailey
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- Barber
- Barry Moran
- Bart Everts
- Bartek Rajwa
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- Baumgart
- Baxter
- Beate Ruckert
- Beatriz Jávega
- Becher
- Becker
- Behbehani
- Behbehani
- Belaaouaj
- Bendall
- Bendall
- Benjamini
- Berhanu
- Bernardo
- Bernink
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- Bianco
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- Biasi
- Biasi
- Biburger
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- Bimba F. Hoyer
- Binder
- Birgit Sawitzki
- Biron
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- Björkström
- Bo Huang
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- Bodenmiller
- Boehm
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- Chapman
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- Chow
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- Christian Münz
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- Wölfl
- Xiao
- Xu
- Xuetao Cao
- Yamamoto
- Yamanaka
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- Yanling Liu
- Yeo
- Yi Zhao
- Yilmaz
- Yona
- Young
- Yousuke Takahama
- Yu
- Yu
- Yu
- Yvonne Samstag
- Yáñez
- Zaunders
- Zawada
- Zhang
- Zheng
- ZhiGang Tian
- Zhou
- Zhou
- Zhou
- Zhou
- Zielinski
- Zigmond
- Zimmermann
- Zimmermann
- Zivanovic
- Zuba-Surman
- Zunder
- Zunder
- Publication venue
- 'Wiley'
- Publication date
- 01/01/2017
- Field of study
International audienceThe classical model of hematopoiesis established in the mouse postulates that lymphoid cells originate from a founder population of common lymphoid progenitors. Here, using a modeling approach in humanized mice, we showed that human lymphoid development stemmed from distinct populations of CD127(-) and CD127(+) early lymphoid progenitors (ELPs). Combining molecular analyses with in vitro and in vivo functional assays, we demonstrated that CD127(-) and CD127(+) ELPs emerged independently from lympho-mono-dendritic progenitors, responded differently to Notch1 signals, underwent divergent modes of lineage restriction, and displayed both common and specific differentiation potentials. Whereas CD127(-) ELPs comprised precursors of T cells, marginal zone B cells, and natural killer (NK) and innate lymphoid cells (ILCs), CD127(+) ELPs supported production of all NK cell, ILC, and B cell populations but lacked T potential. On the basis of these results, we propose a "two-family" model of human lymphoid development that differs from the prevailing model of hematopoiesis