176 research outputs found
Bases agroecologicas para una agricultura con descanso : TROPANDES - Bolivia
- Publication venue
- Publication date
- 01/01/2006
- Field of study
Comparison of physical fitness between healthy and mild‐to‐moderate asthmatic children with exercise symptoms: A cross‐sectional study
- Author
- Alioglu B
- Azmeh R
- Bao Y
- Bateman ED
- Beasley R
- Carson V
- Cassim R
- Castro‐Piñero J
- De Moraes EZC
- Diette GB
- ElmE Von
- Espeland MA
- Faul F
- Fuhlbrigge AL
- Gulmans VAM
- Herdy AH
- Hussey J
- Illi S
- Jia CE
- Kowalski KC
- Kraemer W
- Lam KM
- Lang JE
- Lu KD
- Maltais F
- Marchese VG
- Marrodán MD
- Mate L
- Measurement O
- Milanese M
- Nair P
- Panagiotou M
- Parsons JP
- Pearce N
- Pellegrino R
- Pérez‐Yarza EG
- Pérez‐Yarza EG
- Quanjer PH
- Reimberg MM
- Ribas L
- Ruiz JR
- Santuz P
- Schindel CS
- Villa F
- Wang H
- Welsh L
- Publication venue
- 'Wiley'
- Publication date
- 06/04/2022
- Field of study
.Objective
Asthma is a chronic disease that may affect physical fitness, although its primary effects on exercise capacity, muscle strength, functionality and lifestyle, in children and adolescents, are still poorly understood. This study aimed to evaluate the differences in cardiorespiratory fitness, muscle strength, lifestyle, lung function, and functionality between asthmatics with exercise symptoms and healthy children. In addition, we have analyzed the association between clinical history and the presence of asthma.
Study Design
Cross-sectional study including 71 patients with a diagnosis of asthma and 71 healthy children and adolescents (7–17 years of age). Anthropometric data, clinical history, disease control, lifestyle (KIDMED and physical activity questionnaires), lung function (spirometry), exercise-induced bronchoconstriction test, aerobic fitness (cardiopulmonary exercise test), muscle strength and functionality (timed up and go; timed up and down stairs) were evaluated.
Results
Seventy-one patients with asthma (mean age 11.5 ± 2.7) and 71 healthy subjects (mean age 10.7 ± 2.5) were included. All asthmatic children had mild to moderate and stable asthma. EIB occurred in 56.3% of asthmatic children. Lung function was significantly (p < .05) lower in the asthmatic group when compared to healthy peers, as well as the cardiorespiratory fitness, muscle strength, lifestyle and functionality. Moreover, asthmatic children were more likely to have atopic dermatitis, allergic reactions, food allergies, and a family history of asthma when compared to healthy children.
Conclusions
Children with mild-to-moderate asthma presenting exercise symptoms show a reduction in cardiorespiratory fitness, muscle strength, lung function, functionality, and lifestyle when compared to healthy peers. The study provides data for pediatricians to support exercise practice aiming to improve prognosis and quality of life in asthmatic children.S
Alveolar hypoxia, alveolar macrophages, and systemic inflammation
- Author
- A Nemmar
- A Tsantes
- AL Souza Jr
- AR Whorton
- AS McCourtie
- B Basnyat
- B Beck-Schimmer
- BA Beidleman
- BJ Morgan
- BZ Simkhovich
- C Koechlin
- C Madjdpour
- CJ Mackins
- D Vuichard
- DJ Ciesla
- DR Steiner
- DR Steiner
- E Abraham
- E Tamagawa
- E Zudaire
- EA Deitch
- ED Zee
- EF Wouters
- G Hartmann
- G Zallen
- GM VanOtteren
- H Ishii
- J Agorreta
- J Chao
- JG Wood
- JG Wood
- JG Wood
- JM Kisala
- JR Jordan
- JT Mc Donald
- KA Powers
- KA Powers
- LB Moraes
- LB Ware
- LJ Magnotti
- M Frink
- M Lotz
- M Miyazaki
- M Perl
- M Zhao
- MX Wan
- NC Gonzalez
- NC Gonzalez
- NM Punjabi
- NM Punjabi
- P Conti
- P Eickhoff
- P Puneet
- R Dix
- R Schramm
- RB Silver
- RC St John
- RC Wust
- RJ Gonzalez
- S Fernandez-Serrano
- S Jelic
- S Shah
- S Yende
- SF van Eeden
- SK Leeper-Woodford
- SP Kantrow
- T Fujii
- T Orth
- TR Nurkiewicz
- WI de Boer
- Publication venue
- BioMed Central
- Publication date
- 01/01/2009
- Field of study
Diseases featuring abnormally low alveolar PO2 are frequently accompanied by systemic effects. The common presence of an underlying inflammatory component suggests that inflammation may contribute to the pathogenesis of the systemic effects of alveolar hypoxia. While the role of alveolar macrophages in the immune and defense functions of the lung has been long known, recent evidence indicates that activation of alveolar macrophages causes inflammatory disturbances in the systemic microcirculation. The purpose of this review is to describe observations in experimental animals showing that alveolar macrophages initiate a systemic inflammatory response to alveolar hypoxia. Evidence obtained in intact animals and in primary cell cultures indicate that alveolar macrophages activated by hypoxia release a mediator(s) into the circulation. This mediator activates perivascular mast cells and initiates a widespread systemic inflammation. The inflammatory cascade includes activation of the local renin-angiotensin system and results in increased leukocyte-endothelial interactions in post-capillary venules, increased microvascular levels of reactive O2 species; and extravasation of albumin. Given the known extrapulmonary responses elicited by activation of alveolar macrophages, this novel phenomenon could contribute to some of the systemic effects of conditions featuring low alveolar PO2
Antiinflammatory Therapy with Canakinumab for Atherosclerotic Disease
- Author
- A Abbate
- A Abhyankar
- A Adams
- A Agafina
- A Akyea-Djamson
- A Alan
- A Alfieri
- A Alfrey
- A Alvarisqueta
- A Amos
- A Anadiotis
- A Anneveldt
- A Antolick
- A Arthur
- A Aslam
- Abaci F
- Abdullakutty J
- Abhaichand R
- Abib E Jr
- Aboufakher R
- Abrantes J
- Abreu M
- Abulencia K
- Acampora D
- Acampora M
- Accini Mendoza Jl
- Aceto L
- Acevedo B
- Acevedo L
- Acheatel R
- Actis Mv
- Adams M
- Adjic Nc
- Affaki Gs
- Agarwal Dk
- Aggarwal Rk
- Agosta Gf
- Aguilar M
- Aguilar M
- Ahire N
- Ahmad I
- Ahmed Sh
- Ahn Y
- Aish B
- Aiub F
- Aiub J
- Ajax K
- Ajioka M
- Akai Y
- Akatova E
- Akrap B
- Al Joundi T
- Al-Khalili F
- Albisu J
- Alcalde Jm
- Alcide P
- Alfonso T
- Allen J
- Alllison Dc
- Almaliky T
- Amerena J
- Andersen K
- Andor M
- Angelova I
- Angiolillo D
- Anita S
- Anker Sd
- Antalik L
- Antonicelli R
- Aparicio Cv
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- Apostolovic S
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- Avalos V
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- B Becker
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- Zieve F
- Zineldine A.
- Zirlik A
- Zucchetti C
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2017
- Field of study
Background: Experimental and clinical data suggest that reducing inflammation without affecting lipid levels may reduce the risk of cardiovascular disease. Yet, the inflammatory hypothesis of atherothrombosis has remained unproved. Methods: We conducted a randomized, double-blind trial of canakinumab, a therapeutic monoclonal antibody targeting interleukin-1β, involving 10,061 patients with previous myocardial infarction and a high-sensitivity C-reactive protein level of 2 mg or more per liter. The trial compared three doses of canakinumab (50 mg, 150 mg, and 300 mg, administered subcutaneously every 3 months) with placebo. The primary efficacy end point was nonfatal myocardial infarction, nonfatal stroke, or cardiovascular death. RESULTS: At 48 months, the median reduction from baseline in the high-sensitivity C-reactive protein level was 26 percentage points greater in the group that received the 50-mg dose of canakinumab, 37 percentage points greater in the 150-mg group, and 41 percentage points greater in the 300-mg group than in the placebo group. Canakinumab did not reduce lipid levels from baseline. At a median follow-up of 3.7 years, the incidence rate for the primary end point was 4.50 events per 100 person-years in the placebo group, 4.11 events per 100 person-years in the 50-mg group, 3.86 events per 100 person-years in the 150-mg group, and 3.90 events per 100 person-years in the 300-mg group. The hazard ratios as compared with placebo were as follows: in the 50-mg group, 0.93 (95% confidence interval [CI], 0.80 to 1.07; P = 0.30); in the 150-mg group, 0.85 (95% CI, 0.74 to 0.98; P = 0.021); and in the 300-mg group, 0.86 (95% CI, 0.75 to 0.99; P = 0.031). The 150-mg dose, but not the other doses, met the prespecified multiplicity-adjusted threshold for statistical significance for the primary end point and the secondary end point that additionally included hospitalization for unstable angina that led to urgent revascularization (hazard ratio vs. placebo, 0.83; 95% CI, 0.73 to 0.95; P = 0.005). Canakinumab was associated with a higher incidence of fatal infection than was placebo. There was no significant difference in all-cause mortality (hazard ratio for all canakinumab doses vs. placebo, 0.94; 95% CI, 0.83 to 1.06; P = 0.31). Conclusions: Antiinflammatory therapy targeting the interleukin-1β innate immunity pathway with canakinumab at a dose of 150 mg every 3 months led to a significantly lower rate of recurrent cardiovascular events than placebo, independent of lipid-level lowering. (Funded by Novartis; CANTOS ClinicalTrials.gov number, NCT01327846.
Timing is everything: the regulation of type III secretion
- Author
- A Blocker
- A Boland
- A Botteaux
- A Botteaux
- A Forsberg
- A Subtil
- A Sukhan
- AJ Blocker
- AJ Olive
- AK Veenendaal
- AL Page
- AU Kresse
- AW Williams
- C Agrain
- C Agrain
- C Parsot
- C Parsot
- CA Mueller
- CA Mueller
- CB O’Connell
- CK Yip
- CM Collazo
- CM Li
- CR Epler
- D Wang
- DM Anderson
- ED Brutinel
- F Ferracci
- F Ferracci
- FD Schubot
- FF Chevance
- FS Cordes
- G Wilharm
- GM Fraser
- GR Cornelis
- GT Lountos
- HU Ferris
- I Sorg
- I Stainier
- J Magdalena
- JA Crawford
- Janet E. Deane
- JB Day
- JE Deane
- JE Deane
- JE Deane
- JE Morello
- JL Hodgkinson
- JL McMurry
- JS Matson
- JW Goss
- K Kutsukake
- K Tamano
- K Tamano
- KE Riordan
- KE Riordan
- KL DeBord
- KS Ramamurthi
- L Journet
- L Sundberg
- L Zhang
- LJ Mota
- LW Cheng
- M Espina
- M Lower
- M Sani
- MH Karavolos
- MJ Pallen
- MN Marenne
- N Moriya
- NA Thomas
- NP West
- P Aldridge
- P Ghosh
- Patrizia Abrusci
- PB Lodato
- PJ Edqvist
- PL Payne
- Q Jin
- R Arnold
- R Menard
- R Samudrala
- R Zarivach
- RC Waters
- S Hakansson
- S Wagner
- SA Lloyd
- SE Wood
- SS Joseph
- Steven Johnson
- Susan M. Lea
- SY He
- T Kubori
- T Kubori
- T Kubori
- T Minamino
- T Minamino
- T Minamino
- TC Marlovits
- TC Marlovits
- TF Moraes
- TG Kimbrough
- U Wiesand
- W Deng
- Y Saijo-Hamano
- Y Wang
- Z Du
- Publication venue
- SP Birkhäuser Verlag Basel
- Publication date
- 01/01/2009
- Field of study
Type Three Secretion Systems (T3SSs) are essential virulence determinants of many Gram-negative bacteria. The T3SS is an injection device that can transfer bacterial virulence proteins directly into host cells. The apparatus is made up of a basal body that spans both bacterial membranes and an extracellular needle that possesses a channel that is thought to act as a conduit for protein secretion. Contact with a host-cell membrane triggers the insertion of a pore into the target membrane, and effectors are translocated through this pore into the host cell. To assemble a functional T3SS, specific substrates must be targeted to the apparatus in the correct order. Recently, there have been many developments in our structural and functional understanding of the proteins involved in the regulation of secretion. Here we review the current understanding of protein components of the system thought to be involved in switching between different stages of secretion
Estudo terapêutico aberto com sulfato de aminosidine na leishmaniose mucosa causada for Leishmania (viannia) braziliensis
- Author
- Albino V. Magalhães
- Aldina Barrai
- Badaró R
- Berman JD
- Chunge CN
- Correia Filho D
- Edgar M. Carvalho
- Franco MA
- Franke ED
- Franke ED
- Grogl M
- Gustavo A.S. Romero
- Herwaldt BL
- Hélio A. Lessa
- Jones TC Johnson WD
- Krause G
- Magalhães AV Moraes MAP
- Maria G.O. Orge
- Maria VA. Abreu
- Marsden PD
- Marsden PD
- Marsden PD
- Marsden PD
- Neal RA
- Philip D. Marsden
- Reed SG
- Rosa AC
- Sampaio RNR
- Sampaio RNR
- Schillings RT
- Thakur CP
- Vanize O. Macêdo
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
A922 Sequential measurement of 1 hour creatinine clearance (1-CRCL) in critically ill patients at risk of acute kidney injury (AKI)
- Author
- Abbott TE
- Abdallah RI
- Abdel-Aal IR
- Abdelmonem SA
- Abdo WF
- Abellan AN
- Abellán AN
- Abellán AN
- Abellán AN
- Abrams ST
- Ackerley C
- Acuña M
- Adda M
- Adhikari NK
- Adriano G
- Adrie C
- Affatato R
- Affatato R
- Afonso-Rivero D
- Agarossi A
- Agarwal LK
- Ageeva T
- Agrawal R
- Aguayo-DeHoyos E
- Aguilar AL
- Aguilar-Alonso E
- Aguilar-Alonso E
- Aguilar-Alonso E
- Aguirregabyria M
- Ahmadnia E
- Ahn C
- Ahn JY
- Ahn JY
- Ahn MY
- Ahn MY
- Aitken LM
- Akalaev R
- Akbarzadeh A
- Akcan-Arikan A
- Akhlagh SH
- Akhtar N
- AKI Research Group
- Akiduki N
- Akin S
- Akizuki N
- Akkoc T
- Ala-Kokko T
- Alanio A
- Albaiceta GM
- Albaladejo P
- Alberto F
- Albuisson E
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- Aldabo-Pallas T
- Aldana NN
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- Alejandro R
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- Amargianitakis V
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- ARIAM registry of adult cardiac surgery
- ARIAM-ANDALUCIA
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- CAPCRI Study
- Capellier G
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- Castillo-Lorente E
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- Cavalheiro AM
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- Champigneulle B
- Champigneulle B
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- Chang WY
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- Charitidou E
- Charpentier C
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- Charron C
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- Chernyshuk S
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- Chhor V
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- Chiu LC
- Chiumello D
- Chiurazzi C
- Chiurazzi C
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- Chrétien JM
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- Creteur J
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- Crimella F
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- Cuellar AI
- Cui N
- Cunha MM
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- Curiel-Balsera E
- Curinga M
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- Cuthbertson BH
- Cutuli SL
- Câmara M
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- da Silva SL
- da Silva VZ
- Daga-Ruiz D
- Dalhuisen A
- Dalorzo M
- Damas P
- Dambrosio M
- Damås JK
- Das Neves A
- David H
- Davis C
- Davis P
- Day A
- de Abreu MG
- De Backer A
- De Brito-Ashurst I
- De Cassai A
- De Freitas FF
- De Gasperi A
- de Hoyos EA
- De la Calle-Pedrosa N
- de la Fuente MV
- De La Fuente-Martos C
- De La Fuente-Martos C
- De La Rica AS
- De la Torre MA
- De Maglie M
- De Maglie M
- de Molina FJ
- De Morton N
- de Oca Sandoval MA
- De Pascale G
- De Rosa S
- De Sanctis F
- De Silva AP
- De Sousa I
- de Vera AP
- de Wilde W
- De Zani D
- Deblier I
- DeBrunnar R
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- Del Olmo R
- Del Rosario CG
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- Dimopoulou IK
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- ENVIN-HELICS Study Group
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- Ferrón FR
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- FINNRESUSCI Study Group
- Fischer G
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- Follin A
- Follin A
- Fong KY
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- Fontana V
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- GETGAG Working Group
- GETGAG/SEMICYUC
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- Iijima H
- Ilan R
- Ilyina V
- Imanaka H
- Inaloo S
- Ince C
- Iotti GA
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- Iotti GA
- IPREA Study Group
- Irazabal JM
- Irazabal JM
- Ishimatsu S
- Itenov TS
- Iwamoto M
- Izawa M
- Izdes S
- Jaber S
- Jacky A
- Jacobs FM
- Jacques T
- Jaffal K
- Jamali S
- Jang BH
- Janotka M
- Janotka M
- Janotka M
- Jansen D
- Jansen N
- Japan Septic Disseminated Intravascular Coagulation (JSEPTIC DIC) study group
- Jardim JJ
- Jardim M
- Jareño A
- Javadpour S
- Jayasinghe S
- Jean-Baptiste S
- Jean-Baptiste S
- Jensen AE
- Jensen JU
- Jeon YD
- Jeon YD
- Jeong IY
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- Jian Z
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- Jibaja M
- Jimenez-Herrera MF
- Jiménez GJ
- Jo YH
- Joachim J
- Jochmans S
- John G
- Jonas M
- Jonas M
- Jonas M
- Jones C
- Joshi R
- Joshi V
- Jovaisa T
- JSEPTIC (Japanese Society of Education for Physicians and Trainees in Intensive Care) Clinical Trial Group
- Juan WC
- Juanas CA
- Judickas S
- Juez A
- Juliarena A
- Jun IG
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- Jung K
- Jung KW
- Junior JM
- Kaczirek K
- Kaese S
- Kalani N
- Kalenka A
- Kalfon P
- Kamali E
- Kaminsky GE
- Kaneko M
- Kang M
- Kang M
- Kang PL
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- Kang Y
- Kao KC
- Kappler F
- Kara A
- Karachi F
- Karanjia N
- Karbing DS
- Karsten E
- Kasdan H
- Kashiya S
- Kashyap R
- Katabami K
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- Katira BH
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- Kawamura N
- Kawano S
- Kay FU
- Kayaaslan B
- Kayambu G
- Kazutoshi F
- Ke MW
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- Keep J
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- Keller E
- Kellner F
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- Kerhuel L
- Kerr C
- Kezyte G
- Khadjibaev A
- Khaliq W
- Khine H
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- King HS
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- Kirakli C
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- Kirca H
- Kirman M
- Kirwan CJ
- Kittnar O
- Klapsing P
- Klaus DA
- Kleinpell R
- Kluge S
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- Ko JI
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- Kobayashi A
- Koco E
- Kodate A
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- Kokkini S
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- Komuro T
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- Kongpolprom N
- Kooner G
- Kostalas M
- Kosuk ME
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- Kou PS
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- Kovacikova L
- Kox M
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- Kristof J
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- Ku NS
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- Kubik M
- Kubota K
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- Kunze-Szikszay N
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- Kurtz P
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- Kwon HM
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- Labaune MA
- Labra C
- Lacerda P
- Laerkner E
- Lafaurie M
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- Lagonidis D
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- Leone M
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- Lissonde F
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- Litchfield K
- Liu CP
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- Liu D
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- Stubbs C
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- Student Research Committee - Shiraz University of Medical Sciences
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- Yılmaz G
- Yilmazer F
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- Yokoyama M
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- Yonis H
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- Yoshida T
- Yoshida T
- Yoshihiro S
- Yoshitake E
- You KM
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- Zani D
- Zarantonello F
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- Zepeda EM
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- Zýková I
- Álvarez JT
- Álvarez-Lerma F
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
Meeting abstrac
A Deep Neural Network for Simultaneous Estimation of b Jet Energy and Resolution
- Author
- Aarrestad TK
- Abbaneo D
- Abbiendi G
- Abbrescia M
- Abdalla H
- Abdullah WATW
- Abdullin S
- Abercrombie D
- Acharya H
- Acosta D
- Acosta JG
- Adam W
- Adams MR
- Adams T
- Adzic P
- Adán DP
- Afanasiev S
- Agapitos A
- Agarwal G
- Aggleton R
- Agram J-L
- Aguilar-Benitez M
- Ahmad A
- Ahmad M
- Ahmad M
- Ahmad WH
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- Akbiyik M
- Akchurin N
- Akgun B
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- Aleksandrov A
- Alexander J
- Alhusseini M
- Alimena J
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- Alvarez JDR
- Alverson G
- Alves FL
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- Alyari M
- Amapane N
- Ambrogi F
- Amendola C
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- Amsler C
- Anagnostou G
- Anampa KH
- Anderson D
- Andrea J
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- Anuar AAB
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- Auzinger G
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- Bachiller I
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- Bhawandeep U
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- Bialkowska H
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- Bitioukov S
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- Butler PH
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- Bylinkin A
- Bylsma B
- Byszuk A
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- Chatterjee S
- Chauhan S
- Chauhan S
- Chawla R
- Checchia P
- Chen G
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- Chen HS
- Chen KF
- Chen M
- Chen PH
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- Chen Y
- Chenarani S
- Cheng T
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- Cherepanov V
- Chernyavskaya N
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- Cheung HWK
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- Chu J
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- Cifuentes JAB
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- Cockerill DJA
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- Crescenzo AD
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- Cucciati G
- Cuevas J
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- Cumalat JP
- Cummings G
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- D'Alessandro R
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- d'Enterria D
- D'Hondt J
- Da Costa EM
- Da Rold A
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- Damiani DD
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- Darwish MR
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- Zuolo D
- Özçelik Ö
- Publication venue
- Publication date
- 01/01/2020
- Field of study
We describe a method to obtain point and dispersion estimates for the energies of jets arising from b quarks produced in proton-proton collisions at an energy of s = 13 TeV at the CERN LHC. The algorithm is trained on a large sample of simulated b jets and validated on data recorded by the CMS detector in 2017 corresponding to an integrated luminosity of 41 fb - 1 . A multivariate regression algorithm based on a deep feed-forward neural network employs jet composition and shape information, and the properties of reconstructed secondary vertices associated with the jet. The results of the algorithm are used to improve the sensitivity of analyses that make use of b jets in the final state, such as the observation of Higgs boson decay to b b ¯
Search for dark matter produced in association with a Higgs boson decaying to a pair of bottom quarks in proton-proton collisions at root s=13TeV
- Author
- Aarrestad TK
- Abbaneo D
- Abbiendi G
- Abbrescia M
- Abdullah WATW
- Abdullin S
- Abercrombie D
- Acharya H
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- Acosta JG
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- Wang B
- Wang D
- Wang F
- Wang H
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- Wang L
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- Wang S
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- Wang X
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- Zorbilmez C
- Zotto P
- Zou D
- Zucchetta A
- Zumerle G
- Zuo X
- Zuolo D
- Zygala L
- Álvarez CR
- Özçelik Ö
- Publication venue
- Publication date
- 01/01/2018
- Field of study
A search for dark matter produced in association with a Higgs boson decaying to a pair of bottom quarks is performed in proton-proton collisions at a center-of-mass energy of 13 TeV collected with the CMS detector at the LHC. The analyzed data sample corresponds to an integrated luminosity of 35.9 fb(-1). The signal is characterized by a large missing transverse momentum recoiling against a bottom quark-antiquark system that has a large Lorentz boost. The number of events observed in the data is consistent with the standard model background prediction. Results are interpreted in terms of limits both on parameters of the type-2 two-Higgs doublet model extended by an additional light pseudoscalar boson a (2HDM+a) and on parameters of a baryonic Z simplified model. The 2HDM+a model is tested experimentally for the first time. For the baryonic Z model, the presented results constitute the most stringent constraints to date.Peer reviewe
Early life child micronutrient status, maternal reasoning, and a nurturing household environment have persistent influences on child cognitive development at age 5 years: Results from MAL-ED
- Author
- A Catharine Ross
- A M Shamsir Ahmed
- Aboud
- Ajila T George
- Alberto M Soares
- Aldo A M Lima
- Alessandra Di Moura
- Alexandre Havt
- Algarin
- Ali Turab
- Amidou Samie
- Angel Mendez Acosta
- Angel Orbe Vasquez
- Angelina Maphula
- Angelina Maphula
- Anita K M Zaidi
- Anup Ramachandran
- Anuradha Bose
- Anuradha Bose
- Archana Mohale
- Asad Ali
- Aubrey Bauck
- Bacharach
- Baitun Nahar
- Barbara Schaefer
- Beena Koshy
- Beena Koshy
- Benjamin J J McCormick
- Benjamin J J McCormick
- Bentley
- Beusenberg
- Biesalski
- Binob Shrestha
- Black
- Black
- Bradley
- Bruna L L Maciel
- Buliga Mujaga Swema
- Caldwell
- Carl J Mason
- Caroline Amour
- Caulfield
- Cesar Banda Chavez
- Christel Hoest
- Cloupas Mahopo
- Cláudia B Abreu
- Crystal L Patil
- Dennis R Lang
- Didar Alam
- Dinesh Hariraju
- Dinesh Mondal
- Dixner Rengifo Trigoso
- Dror
- Eliwaza Bayyo
- Elizabeth T Rogawski
- Emanuel Nyathi
- Engle
- Eric Houpt
- Erling Svensen
- Erling Svensen
- Eshel
- Estomih R Mduma
- Fahmida Tofail
- Francisco S Mota
- Gagandeep Kang
- Gaurvika Nayyar
- Georgiadis
- Georgiadis
- Gwenyth Lee
- Hilda Costa
- Horta
- Houpt
- Ila F Lima
- Imran Ahmed
- Iqbal Hossain
- J Daniel Carreon
- Jacobs
- James Platts-Mills
- Jayaprakash Muliyil
- Jean Gratz
- Jessica C Seidman
- Jessica C Seidman
- Jhanelle Graham
- John Pascal
- Jones
- Josiane Quetz
- José Quirino Filho
- Julian Torres Flores
- Karen H Tountas
- Karthikeyan Ramanujam
- Kerry Schulze
- Kosek
- Kosek
- Ladaporn Bodhidatta
- Ladislaus Blacy
- Ladislaus Yarrot
- Laura E Caulfield
- Laura E Caulfield
- Laura E Murray-Kolb
- Laura E Murray-Kolb
- Laura L Pendergast
- Laura Pendergast
- Leah Barrett
- Liu
- Liu
- Lozoff
- Lu
- M Steffi Jennifer
- MAL-ED Network Investigators
- MAL-ED Network Investigators
- Manjeswori Ulak
- Margaret N Kosek
- Maribel Paredes Olotegui
- Mark A Miller
- McCormick
- Mery Siguas Salas
- Michael Gottlieb
- Milena Moraes
- Mohan Venkata Raghava
- Monica McGrath
- Muneera Rasheed
- Muneera Rasheed
- Munirul Islam
- Murray-Kolb
- Mustafa Mahfuz
- Namaste
- Nkrumah
- Noélia L Lima
- Pablo Peñataro Yori
- Pascal Bessong
- Pedro H Q S Medeiros
- Pendergast
- Petry
- Pinkerton
- Platts-Mills
- Prado
- Prakash Sunder Shrestha
- Priyadarshani Karunakaran
- Psaki
- Pérez-Escamilla
- Rahul J Thomas
- Rakhi Ramadas
- Ram Krishna Chandyo
- Ramya Ambikapathi
- Rashidul Haque
- Raven
- Rebecca Blank
- Rebecca Dillingham
- Rebecca Scharf
- Rebecca Scharf
- Reeba Roshan
- Reeba Roshan
- Regisiana Mvungi
- Reinaldo B Oriá
- Reinaldo Oria
- Richard
- Richard
- Richard L Guerrant
- Rita Shrestha
- Rita Shrestha
- Robert E Black
- Robin P Lazarus
- Ronfani
- Rosa M S Mota
- Rosa Rios de Burga
- Rosemary Nshama
- Ruan
- Sajid Soofi
- Samuel P Scott
- Sanjaya Kumar Shrestha
- Santos
- Sayma Haque
- Shahida Qureshi
- Shanmuga Sundaram E
- Shiny Kaki
- Silvia Rengifo Pinedo
- Sophy Raju
- Srujan L Sharma
- Stacey Knobler
- Stephanie A Richard
- Stephanie A Richard
- Stephanie Psaki
- Sudhir Babji
- Sushil John
- Suzanne Simons
- Tahmeed Ahmed
- Tor Strand
- Tucker
- van Buuren
- Victora
- Vivek Charu
- Vivian Wang
- Viyada Doan
- Walker
- Weschler
- WHO
- WHO
- Willett
- William A Petri
- William Checkley
- William K Pan
- Zamora
- Zeba Rasmussen
- Zeba Rasmussen
- Zulfiqar A Bhutta
- Álvaro M Leite
- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- 01/01/2019
- Field of study
Background: Child cognitive development is influenced by early-life insults and protective factors. To what extent these factors have a long-term legacy on child development and hence fulfillment of cognitive potential is unknown. Objective: The aim of this study was to examine the relation between early-life factors (birth to 2 y) and cognitive development at 5 y. Methods: Observational follow-up visits were made of children at 5 y, previously enrolled in the community-based MAL-ED longitudinal cohort. The burden of enteropathogens, prevalence of illness, complementary diet intake, micronutrient status, and household and maternal factors from birth to 2 y were extensively measured and their relation with the Wechsler Preschool Primary Scales of Intelligence at 5 y was examined through use of linear regression. Results: Cognitive T-scores from 813 of 1198 (68%) children were examined and 5 variables had significant associations in multivariable models: mean child plasma transferrin receptor concentration (β: −1.81, 95% CI: −2.75, −0.86), number of years of maternal education (β: 0.27, 95% CI: 0.08, 0.45), maternal cognitive reasoning score (β: 0.09, 95% CI: 0.03, 0.15), household assets score (β: 0.64, 95% CI: 0.24, 1.04), and HOME child cleanliness factor (β: 0.60, 95% CI: 0.05, 1.15). In multivariable models, the mean rate of enteropathogen detections, burden of illness, and complementary food intakes between birth and 2 y were not significantly related to 5-y cognition. Conclusions: A nurturing home context in terms of a healthy/clean environment and household wealth, provision of adequate micronutrients, maternal education, and cognitive reasoning have a strong and persistent influence on child cognitive development. Efforts addressing aspects of poverty around micronutrient status, nurturing caregiving, and enabling home environments are likely to have lasting positive impacts on child cognitive development.publishedVersio
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