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54 research outputs found

Clinical aspects of Mayer-Rokitansky-Kuester-Hauser syndrome: recommendations for clinical diagnosis and staging

Author: Beckmann Matthias W.
Brucker Sara
Hauser Georges A.
Kellermann Anja
Ludwig Kurt S.
Oppelt Peter
Renner Stefan P.
Strick Reiner
Strissel Pamela L.
Wallwiener Diethelm
Publication venue
Publication date: 02/08/2017
Field of study

BACKGROUND: The Mayer-Rokitansky-Kuester-Hauser (MRKH) syndrome is a malformation of the female genitals (occurring in one in 4000 female live births) as a result of interrupted embryonic development of the Müllerian (paramesonephric) ducts. This retrospective study examined the issue of associated malformations, subtyping, and the frequency distribution of subtypes in MRKH syndrome. METHODS: Fifty-three MRKH patients were investigated using a newly developed standardized questionnaire. Together with the results of clinical and diagnostic examinations, the patients were classified into the three recognized subtypes [typical, atypical and MURCS (Müllerian duct aplasia, renal aplasia, and cervicothoracic somite dysplasia)]. RESULTS: The typical form was diagnosed in 25 patients (47%), the atypical form in 11 patients (21%), and the most marked form—the MURCS type—in 17 patients (32%). Associated malformations were notably frequent among the patients. Malformations of the renal system were the most frequent type of accompanying malformation, with 23 different malformations in 19 patients, followed by 18 different skeletal changes in 15 patients. CONCLUSIONS: In accordance with the literature, this study shows that associated malformations are present in more than a third of cases. Therefore, new basic guidelines for standard diagnostic classification involving patients with suspected MRKH are presente

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Distinct Kinetics of Memory B-Cell and Plasma-Cell Responses in Peripheral Blood Following a Blood-Stage Plasmodium chabaudi Infection in Mice

B cell and plasma cell responses take place in lymphoid organs, but because of the inaccessibility of these organs, analyses of human responses are largely performed using peripheral blood mononuclear cells (PBMC). To determine whether PBMC are a useful source of memory B cells and plasma cells in malaria, and whether they reflect Plasmodium-specific B cell responses in spleen or bone marrow, we have investigated these components of the humoral response in PBMC using a model of Plasmodium chabaudi blood-stage infections in C57BL/6 mice. We detected memory B cells, defined as isotype-switched IgD− IgM− CD19+ B cells, and low numbers of Plasmodium chabaudi Merozoite Surface Protein-1 (MSP1)-specific memory B cells, in PBMC at all time points sampled for up to 90 days following primary or secondary infection. By contrast, we only detected CD138+ plasma cells and MSP1-specific antibody-secreting cells within a narrow time frame following primary (days 10 to 25) or secondary (day 10) infection. CD138+ plasma cells in PBMC at these times expressed CD19, B220 and MHC class II, suggesting that they were not dislodged bone-marrow long-lived plasma cells, but newly differentiated migratory plasmablasts migrating to the bone marrow; thus reflective of an ongoing or developing immune response. Our data indicates that PBMC can be a useful source for malaria-specific memory B cells and plasma cells, but extrapolation of the results to human malaria infections suggests that timing of sampling, particularly for plasma cells, may be critical. Studies should therefore include multiple sampling points, and at times of infection/immunisation when the B-cell phenotypes of interest are likely to be found in peripheral blood

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UCL Discovery

Combinations of single-top-quark production cross-section measurements and vertical bar f(LV)V(tb)vertical bar determinations at root s=7 and 8 TeV with the ATLAS and CMS experiments

Author: Aaboud Morad
Aad G.
Abbott B.
Abbott D. C.
Abdinov O.
Abed Abud A.
Abhayasinghe D. K.
Abidi S. H.
AbouZeid O. S.
Abraham N. L.
Abramowicz H.
Abreu H.
Abulaiti Y.
Acharya B. S.
Adachi S.
Adam L.
Adamczyk L.
Adamek L.
Adelman J.
Adersberger M.
Adiguzel A.
Adorni S.
Adye T.
Affolder A. A.
Afik Y.
Agapopoulou C.
Agaras M. N.
Aggarwal A.
Agheorghiesei C.
Aguilar-Saavedra Juan Antonio
Ahmadov F.
Ai X.
Aielli G.
Akatsuka S.
Akilli E.
Akimov A. V.
Al Khoury K.
Alberghi G. L.
Albert J.
Alconada Verzini M. J.
Alderweireldt S.
Aleksa M.
Aleksandrov I. N.
Alexa C.
Alexandre D.
Alexopoulos T.
Alfonsi A.
Alhroob M.
Ali B.
Alimonti G.
Alison J.
Alkire S. P.
Allaire C.
Allbrooke B. M. M.
Allen B. W.
Allport P. P.
Aloisio A.
Alonso A.
Alonso F.
Alpigiani C.
Alshehri A. A.
Alstaty M. I.
Alvarez Estevez M.
Alvarez Gonzalez B.
Alviggi M. G.
Amaral Coutinho Y.
Ambler A.
Ambroz L.
Amelung C.
Amidei D.
Amor Dos Santos S. P.
Amoroso S.
Amrouche C. S.
An F.
Anastopoulos C.
Andari N.
Andeen T.
Anders C. F.
Anders J. K.
Andreazza A.
Andrei V.
Anelli C. R.
Angelidakis S.
Angelozzi I.
Angerami A.
Anisenkov A. V.
Annovi A.
Antel C.
Anthony M. T.
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Aparisi Pozo J. A.
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Smith N.
Smith W. H.
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Álvarez Piqueras D.
Åkesson T. P. A.
Publication venue
Publication date: 01/01/2019
Field of study

This paper presents the combinations of single-top-quark production cross-section measurements by the ATLAS and CMS Collaborations, using data from LHC proton-proton collisions at = 7 and 8 TeV corresponding to integrated luminosities of 1.17 to 5.1 fb(-1) at = 7 TeV and 12.2 to 20.3 fb(-1) at = 8 TeV. These combinations are performed per centre-of-mass energy and for each production mode: t-channel, tW, and s-channel. The combined t-channel cross-sections are 67.5 +/- 5.7 pb and 87.7 +/- 5.8 pb at = 7 and 8 TeV respectively. The combined tW cross-sections are 16.3 +/- 4.1 pb and 23.1 +/- 3.6 pb at = 7 and 8 TeV respectively. For the s-channel cross-section, the combination yields 4.9 +/- 1.4 pb at = 8 TeV. The square of the magnitude of the CKM matrix element V-tb multiplied by a form factor f(LV) is determined for each production mode and centre-of-mass energy, using the ratio of the measured cross-section to its theoretical prediction. It is assumed that the top-quark-related CKM matrix elements obey the relation |V-td|, |V-ts| << |V-tb|. All the |f(LV)V(tb)|(2) determinations, extracted from individual ratios at = 7 and 8 TeV, are combined, resulting in |f(LV)V(tb)| = 1.02 +/- 0.04 (meas.) +/- 0.02 (theo.). All combined measurements are consistent with their corresponding Standard Model predictions.Peer reviewe

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Epigenetic Regulation of a Murine Retrotransposon by a Dual Histone Modification Mark

Author: A Dupressoir
AE Peaston
AF Smit
AL Clayton
Anna Sawicka
AS Wilson
Astrid Hagelkruys
AT Carter
B Irwin
B McClintock
B Panning
BD Strahl
BS Garrison
C Esnault
C Hauser
C Stocking
CD Malone
Christian Seiser
CM Davis
CM Koch
CP Hodgson
CP Walsh
D Branciforte
D Noutsopoulos
D Rotili
DJ Obbard
DT Scholes
E Kalkhoven
Elisabeth Simboeck
ES Lander
ET Prak
G He
G Lagger
Gerda Egger
GJ Todaro
H Ishihara
HP Cam
I Colmegna
IA Maksakova
IA Maksakova
IA Maksakova
J Li
JA Mietz
JH Martens
JJ Fischer
JL Goodier
JL Griffith
JS You
JV Moran
K Ghoshal
K Ikegami
KM Nyswaner
L Eaton
L Li
L Schermelleh
LC Mahadevan
M Aye
M Lusic
M Matzke
M Nilsson
M Puschendorf
MA Glozak
Michel Georges
ML Gonzalgo
N Lane
NL Wiech
NS French
O Heidmann
P Capy
P Cheung
P Marks
Patrick Matthias
PS Espino
R Brunmeir
R Januchowski
Reinhard Brunmeir
RH Waterston
RK Slotkin
S Shankar
S Thomson
S Winter
Sabine Lagger
SK Kurdistani
SP Goff
T Matsui
T Tzavaras
T Wicker
T Yamaguchi
TA Rauch
V Stribinskis
W Seifarth
WJ Costain
WJ Miller
WM Liu
Wolfgang J. Miller
X Song
X Song
XJ Yang
Yu Zhang
Z Wang
Publication venue: Public Library of Science
Publication date: 01/04/2010
Field of study

Large fractions of eukaryotic genomes contain repetitive sequences of which the vast majority is derived from transposable elements (TEs). In order to inactivate those potentially harmful elements, host organisms silence TEs via methylation of transposon DNA and packaging into chromatin associated with repressive histone marks. The contribution of individual histone modifications in this process is not completely resolved. Therefore, we aimed to define the role of reversible histone acetylation, a modification commonly associated with transcriptional activity, in transcriptional regulation of murine TEs. We surveyed histone acetylation patterns and expression levels of ten different murine TEs in mouse fibroblasts with altered histone acetylation levels, which was achieved via chemical HDAC inhibition with trichostatin A (TSA), or genetic inactivation of the major deacetylase HDAC1. We found that one LTR retrotransposon family encompassing virus-like 30S elements (VL30) showed significant histone H3 hyperacetylation and strong transcriptional activation in response to TSA treatment. Analysis of VL30 transcripts revealed that increased VL30 transcription is due to enhanced expression of a limited number of genomic elements, with one locus being particularly responsive to HDAC inhibition. Importantly, transcriptional induction of VL30 was entirely dependent on the activation of MAP kinase pathways, resulting in serine 10 phosphorylation at histone H3. Stimulation of MAP kinase cascades together with HDAC inhibition led to simultaneous phosphorylation and acetylation (phosphoacetylation) of histone H3 at the VL30 regulatory region. The presence of the phosphoacetylation mark at VL30 LTRs was linked with full transcriptional activation of the mobile element. Our data indicate that the activity of different TEs is controlled by distinct chromatin modifications. We show that activation of a specific mobile element is linked to a dual epigenetic mark and propose a model whereby phosphoacetylation of histone H3 is crucial for full transcriptional activation of VL30 elements

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PubMed Central

Search for new phenomena in high-mass diphoton final states using 37 fb−1 of proton–proton collisions collected at √s = 13 TeV with the ATLAS detector

Author: Aaboud M
Aad G
Abbott B
Abdinov O
Abeloos B
Abidi SH
AbouZeid OS
Abraham NL
Abramowicz H
Abreu H
Abreu R
Abulaiti Y
Acharya BS
Adachi S
Adamczyk L
Adelman J
Adersberger M
Adye T
Affolder AA
Afik Y
Agatonovic-Jovin T
Agheorghiesei C
Aguilar-Saavedra JA
Ahlen SP
Ahmadov F
Aielli G
Akatsuka S
Akerstedt H
Akesson TPA
Akilli E
Akimov AV
Alberghi GL
Albert J
Albicocco R
Alderweireldt SC
Aleksa M
Aleksandrov IN
Alexa C
Alexander G
Alexopoulos T
Alhroob M
Ali B
Aliev M
Alimonti G
Alison J
Alkire SP
Allbrooke BMM
Allen BW
Allport PP
Aloisio A
Alonso A
Alonso F
Alpigiani C
Alshehri AA
Alstaty MI
Alviggi MG
Amadio BT
Amaral Coutinho Y
Amelung C
Amidei D
Amor Dos Santos SP
Amoroso S
Amundsen G
Anastopoulos C
Ancu LS
Andari N
Andeen T
Anders CF
Anders JK
Anderson KJ
Andreazza A
Andrei V
Angelidakis S
Angelozzi I
Angerami A
Anisenkov AV
Anjos N
Annovi A
Antel C
Antonelli M
Antonov A
Antrim DJ
Anulli F
Aoki M
Arabidze G
Arai Y
Aranda CP
Araque JP
Araujo Ferraz V
Araya ST
Arce ATH
Ardell RE
Arduh FA
Arguin J-F
Argyropoulos S
Arik M
Armadans RC
Armbruster AJ
Armitage LJ
Arnaez O
Arnold H
Arratia M
Arslan O
Artamonov A
Artoni G
Artz S
Asai S
Asbah N
Ashkenazi A
Asquith L
Assamagan K
Astalos R
Astigarraga MEP
Atkinson M
Atlay NB
Augsten K
Avolio G
Axen B
Ayoub MK
Azuelos G
Baas AE
Baca MJ
Bachacou H
Bachas K
Backes M
Bagnaia P
Bahmani M
Bahrasemani H
Baines JT
Bajic M
Baker OK
Bakker PJ
Baldin EM
Balek R
Balli F
Balunas WK
Banas E
Bandyopadhyay A
Banerjee S
Bannoura AAE
Barajas CAC
Barak L
Barberio EL
Barberis D
Barbero M
Barillari T
Barisits M-S
Barkeloo JT
Barklow T
Barlow N
Barnes SL
Barnett BM
Barnett RM
Barnovska-Blenessy Z
Baroncelli A
Barone G
Barr AJ
Barranco Navarro L
Barreiro F
Bartoldus R
Barton AE
Bartos R
Basalaev A
Bassalat A
Bates RL
Batista SJ
Batley JR
Battaglia M
Bauce M
Bauer F
Bawa HS
Beacham JB
Beattie MD
Beau T
Beauchemin PH
Bechtle R
Beck HC
Beck HP
Becker K
Becker M
Becot C
Beddall A
Beddall AJ
Bednyakov VA
Bedognetti M
Bee CP
Beermann TA
Begalli M
Begel M
Behr JK
Bell AS
Bella G
Bella LA
Bellagamba L
Bellerive A
Bellomo M
Belotskiy K
Beltramello O
Belyaev NL
Benary O
Benchekroun D
Bender M
Benekos N
Benhammou Y
Benitez J
Benjamin DP
Benoit M
Bensinger JR
Bentvelsen S
Beresford L
Beretta M
Berge D
Berger N
Beringer J
Berlendis S
Berlingen JM
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Bernardi G
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Bertsche D
Besjes GJ
Bessner M
Besson N
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Biedermann D
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Bourdarios C
Boutle SK
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Boyko IR
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Bratzler U
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Bressler S
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Bryngemark L
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Buat Q
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Buckley AG
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Buescher V
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Burgard CD
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Burghgrave B
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Bylund OB
Caforio D
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Calvet D
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Calvet TP
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Cameron D
Camincher C
Campana S
Campanelli M
Camplani A
Campoverde A
Canale V
Cantero J
Cao T
Caprini I
Caprini M
Capua M
Carbone RM
Cardarelli R
Cardillo F
Carli I
Carli T
Carlino G
Carlson BT
Carminati L
Carney RMD
Caron S
Carquin E
Carra S
Carrillo-Montoya GD
Casadei D
Casado MP
Casolino M
Casper DW
Castelijn R
Castillo Gimenez V
Castillo IS
Castillo LRF
Castro NF
Catinaccio A
Catmore JR
Cattai A
Caudron J
Cavaliere V
Cavallaro E
Cavalli D
Cavalli-Sforza M
Cavasinni V
Celebi E
Ceradini F
Cerda Alberich L
Cerqueira AS
Cerri A
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Cerutti F
Cervelli A
Cetin SA
Chafaq A
Chakraborty D
Chan SK
Chan WS
Chan YL
Chang R
Chapman JD
Charlton DG
Chau CC
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Cheatham S
Chegwidden A
Chekanov S
Chekulaev SV
Chelkov GA
Chelstowska MA
Chen C
Chen C
Chen H
Chen J
Chen S
Chen S
Chen X
Chen Y
Cheng HC
Cheng HJ
Cheplakov A
Cheremushkina E
Cheu E
Cheung K
Chevalier L
Chiarella V
Chiarelli G
Chiodini G
Chisholm AS
Chitan A
Chiu YH
Chizhov MV
Choi K
Chomont AR
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Chow YS
Christodoulou V
Chu MC
Chudoba J
Chuinard AJ
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Chytka L
Ciftci AK
Cinca D
Cindro V
Cioara IA
Ciocio A
Cirotto F
Citron ZH
Citterio M
Ciubancan M
Clark A
Clark BL
Clark MR
Clark PJ
Clarke RN
Clement C
Coadou Y
Cobal M
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Collaboration ATLAS
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Cooper-Sarkar AM
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Cornell SD
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Costa MJ
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Cox BE
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Crawley SJ
Creager RA
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Cummings J
Curatolo M
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Czekierda S
Czodrowski R
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D'Auria S
D'eramo L
D'Onofrio M
da Costa JBG
Da Costa JGPF
Da Cunha Sargedas De Sousa MJ
Da Via C
Dabrowski W
Dado T
Dai T
Dale O
Dallaire F
Dallapiccola C
Dam M
Damazio DO
Dandoy JR
Daneri MF
Dang NP
Daniells AC
Dann NS
Danninger M
Dao V
Darbo G
Darmora S
Dassoulas J
Dattagupta A
Daubney T
Davey W
David C
Davidek T
Davis DR
Davison R
Dawe E
Dawson I
de Andrade Filho LM
de Asmundis R
De Benedetti A
De Bruin PHS
De Castro S
De Cecco S
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de Jong R
de la Hoz SG
De la Torre H
de Lima DEF
De Lorenzi F
De Maria A
De Pedis D
De Regie JBDV
de Renstrom PAB
De Salvo A
De Sanctis U
De Santo A
De K
Debbe R
Debenedetti C
Dedovich DV
Dehghanian N
Deigaard I
Del Gaudio M
Del Peso J
Delgado AT
Delgove D
Deliot F
Delitzsch CM
Dell'Acqua A
Dell'Asta L
Dell'Orso M
Della Pietra M
Della Volpe D
Delmastro M
Delporte C
Delsart PA
DeMarco DA
Demers S
Demichev M
Demilly A
Denisov SP
Denysiuk D
Derendarz D
Derkaoui JE
Derue F
Dervan R
Desch K
Deterre C
Dette K
Devesa MR
Deviveiros PO
Dewhurst A
Dhaliwal S
Di Bello FA
Di Ciaccio A
Di Ciaccio L
Di Clemente WK
Di Donato C
Di Girolamo A
Di Girolamo B
Di Micco B
Di Nardo R
Di Petrillo KF
Di Simone A
Di Sipio R
Di Valentino D
Diaconu C
Diamond M
Dias FA
Diaz MA
Diehl EB
Dietrich J
Dimitrievska A
Dingfelder J
Dita P
Dita S
Dittus F
Djama F
Djobava T
Djuvsland JI
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Dobos D
Dobre M
Dodsworth D
Doglioni C
Dolejsi J
Dolezal Z
Donadelli M
Donati S
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Donszelmann TC
Dopke J
Doria A
Dova MT
Doyle AT
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Dris M
Du Y
Duarte-Campderros J
Dubreuil A
Duchovni E
Duckeck G
Ducourthial A
Ducu OA
Duda D
Dudarev A
Dudder AC
Dueren M
Duffield EM
Duflot L
Duhrssen M
Dulsen C
Dumancic M
Dumitriu AE
Duncan AK
Dunford M
Duperrin A
Durglishvili A
Duschinger D
Dutta B
Duvnjak D
Dyndal M
Dziedzic BS
Eckardt C
Ecker KM
Edgar RC
Eifert T
Eigen G
Einsweiler K
Ekelof T
El Kacimi M
El Kosseifi R
El Moursli RC
Ellajosyula V
Ellert M
Elles S
Ellinghaus F
Elliot AA
Ellis N
Elmsheuser J
Elsing M
Emeliyanov D
Enari Y
Endner OC
Ennis JS
Epland MB
Erdmann J
Ereditato A
Ernst M
Errede S
Escalier M
Escobar C
Esposito B
Etienvre AI
Etzion E
Evans H
Ezhilov A
Ezzi M
Fabbri F
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Fabiani V
Facini G
Fakhrutdinov RM
Falciano S
Falla RJ
Faltova J
Fang Y
Fanti M
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Farilla A
Farina C
Farina EM
Farooque T
Farrell S
Farrington SM
Farthouat R
Fassi F
Fassnacht R
Fassouliotis D
Favareto A
Fawcett WJ
Fayard L
Fedin OL
Fedorko W
Feigl S
Feligioni L
Feng C
Feng EJ
Fenton MJ
Fenyuk AB
Feremenga L
Fernandez Martinez R
Fernandez Perez S
Ferrando J
Ferrari A
Ferrari R
Ferrari R
Ferrer A
Ferrer JAV
Ferrere D
Ferretti C
Fiedler F
Filipcic A
Filipuzzi M
Filthaut F
Fincke-Keeler M
Finelli KD
Fiolhais MCN
Fiorini L
Fischer A
Fischer C
Fischer J
Fisher WC
Flasche N
Fleck I
Fleischmann R
Fletcher RRM
Flick T
Flierl BM
Flowerdew MJ
Forcolin GT
Formica A
Forster FA
Forti A
Foster AG
Fournier D
Fox H
Fracchia S
Francavilla R
Franchini M
Franchino S
Francis D
Franconi L
Franklin M
Frate M
Fraternali M
Freeborn D
Fressard-Batraneanu SM
Freund B
Froidevaux D
Frost JA
Fukunaga C
Furelos DV
Fusayasu T
Fuster J
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Gabrielli A
Gabrielli A
Gach GP
Gadatsch S
Gadomski S
Gagliardi G
Gagnon LG
Galea C
Galhardo B
Gallas EJ
Gallop BJ
Gallus P
Galster G
Gan KK
Ganguly S
Gao Y
Gao YS
Garay Walls FM
Garcia Navarro JE
Garcia BRM
Garcia C
Garcia RFN
Garcia-Sciveres M
Gardner RW
Garelli N
Garonne V
Garrido MDMC
Gasnikova K
Gatti C
Gaudiello A
Gaudio G
Gavrilenko IL
Gay C
Gaycken G
Gazis EN
Gee CNP
Geisen J
Geisen M
Geisler MP
Gellerstedt K
Gemme C
Genest MH
Geng C
Gentile S
Gentsos C
Georges S
Gerbaudo D
Gessner G
Ghasemi S
Ghneimat M
Giacobbe B
Giagu S
Giangiacomi N
Giannelli MF
Giannetti P
Gibson SM
Gignac M
Gilchriese M
Gillberg D
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Gingrich DM
Giordani MP
Giorgi FM
Giraud PF
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Giugliarelli G
Giugni D
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Giulini M
Gjelsten BK
Gkaitatzis S
Gkialas I
Gkougkousis EL
Gkountoumis P
Gladilin LK
Glasman C
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Glaysher PCF
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Goblirsch-Kolb M
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Golling T
Golubkov D
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Goncalves Gama R
Gonella G
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Gongadze A
Gonzalez BA
Gonzalez-Sevilla S
Goossens L
Gorbounov RA
Gordon HA
Gorelov I
Gorini B
Gorini E
Gorisek A
Goshaw AT
Gostkin MI
Gottardo CA
Goudet CR
Goujdami D
Goussiou AG
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Gozani E
Grabowska-Bold I
Gradin POJ
Gramling J
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Greenwood ZD
Grefe C
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Gregor IM
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Grivaz J-F
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Grosse-Knetter J
Grossi GC
Grout ZJ
Grummer A
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Guindon S
Gul U
Gumpert C
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Gupta R
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Gurbuz S
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Gutelman BJ
Gutierrez P
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Guzik MP
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Hallewell GD
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Hamnett PG
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Hansen MC
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Hawkes CM
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Knue A
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Komar AA
Kondo T
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Konig AC
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Korolkova EV
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Kostyukhin VV
Kotwal A
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Kourkoumeli-Charalampidi A
Kourkoumelis C
Kourlitis E
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Kowalski TZ
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Kurochkin YA
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Kuutmann EB
Kuwertz ES
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Lavrijsen W
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Loebinger FK
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Lundberg O
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Mjornmark JU
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zur Nedden M
Zwalinski L
Publication venue: Elsevier
Publication date: 01/01/2017
Field of study

Searches for new phenomena in high-mass diphoton final states with the ATLAS experiment at the LHC are presented. The analysis is based on pp collision data corresponding to an integrated luminosity of 36.7 fb−1 at a centre-of-mass energy √s = 13 TeV recorded in 2015 and 2016. Searches are performed for resonances with spin 0, as predicted by theories with an extended Higgs sector, and for resonances with spin 2, using a warped extra-dimension model as a benchmark model, as well as for non-resonant signals, assuming a large extra-dimension scenario. No significant deviation from the Standard Model is observed. Upper limits are placed on the production cross section times branching ratio to two photons as a function of the resonance mass. In addition, lower limits are set on the ultraviolet cutoff scale in the large extra-dimensions model

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