282 research outputs found
Employer's management of employees affected by cancer
- Author
- A Rijk de
- A. de Boer
- A. Popa
- AG Boer De
- AKY Mak
- AKY Mak
- C Tiedtke
- C Tiedtke
- CJ Main
- D Ehrmann-Feldmann
- D. Yagil
- DE Stewart
- EA Grunfeld
- F. Munir
- FL Tan
- G McKay
- GS Pransky
- H Eguchi
- J Selander
- J Yarker
- K Luker
- KT Ashing-Giwa
- M Stergiou-Kita
- M Stergiou-Kita
- MG Poulsen
- ML Lindbohm
- PD McGrath
- S. Tamminga
- SJ Tamminga
- T Taskila
- VS Blinder
- Z Amir
- Z Amir
- Z Amir
- Z. Amir
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 29/11/2017
- Field of study
Return to work (RTW) following treatment can be problematic for cancer survivors. Although some people affected by cancer are able to continue working, a greater proportion of these survivors end up unemployed, retire early or change jobs than those without a diagnosis of cancer. One of the reasons for not returning to work is the lack of understanding and support from employers and supervisors. Currently, it is not clear what factors are likely to influence the employer’s management of employees recovering from cancer. This article reports the outcome from a review of the published literature on factors related to the current employer management of employed cancer survivors
Time-Dependent Subcellular Distribution and Effects of Carbon Nanotubes in Lungs of Mice
- Author
- A Erdely
- AA Humbles
- AA Kapralov
- AB Murray
- Anne T. Saber
- AT Saber
- AT Saber
- AT Saber
- C Ben-Yehuda
- C Ronzani
- CA Beamer
- CA Poland
- Carole L. Yauk
- Carsten Købler
- D Pantarotto
- DW Porter
- DW Porter
- FT Andón
- H Ali-Boucetta
- H Heath
- H Nagai
- H Persson
- Håkan Wallin
- I Canton
- I Marangon
- I Waern
- J Muller
- J Muller
- J Nio
- J Pauluhn
- JA Bourdon
- JG Teeguarden
- JP Ryman-Rasmussen
- K Aschberger
- K Bhattacharya
- K Donaldson
- K Inoue
- Klaus Qvortrup
- Kristian Mølhave
- KT Al-Jamal
- L Guo
- L Lacerda
- L Ma-Hock
- M Feldmesser
- M Monthioux
- M. Van Zweiten
- MH Ross
- MJ Hoenerhoff
- N Kobayashi
- Nicklas R. Jacobsen
- Q Mu
- R Wierzbicki
- R-F Guo
- RF Hamilton
- S Treumann
- SA Elmore
- Sabina Halappanavar
- Sarah S. Poulsen
- SS Poulsen
- TA Girtsman
- TM Sager
- UC Nygaard
- Ulla Vogel
- Valentin Ceña
- X Wang
- X Wang
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2015
- Field of study
BACKGROUND AND METHODS:Pulmonary deposited carbon nanotubes (CNTs) are cleared very slowly from the lung, but there is limited information on how CNTs interact with the lung tissue over time. To address this, three different multiwalled CNTs were intratracheally instilled into female C57BL/6 mice: one short (850 nm) and tangled, and two longer (4 μm and 5.7 μm) and thicker. We assessed the cellular interaction with these CNTs using transmission electron microscopy (TEM) 1, 3 and 28 days after instillation. RESULTS:TEM analysis revealed that the three CNTs followed the same overall progression pattern over time. Initially, CNTs were taken up either by a diffusion mechanism or via endocytosis. Then CNTs were agglomerated in vesicles in macrophages. Lastly, at 28 days post-exposure, evidence suggesting CNT escape from vesicle enclosures were found. The longer and thicker CNTs more often perturbed and escaped vesicular enclosures in macrophages compared to the smaller CNTs. Bronchoalveolar lavage (BAL) showed that the CNT exposure induced both an eosinophil influx and also eosinophilic crystalline pneumonia. CONCLUSION:Two very different types of multiwalled CNTs had very similar pattern of cellular interactions in lung tissue, with the longer and thicker CNTs resulting in more severe effects in terms of eosinophil influx and incidence of eosinophilic crystalline pneumonia (ECP)
Search for new phenomena in final states with an energetic jet and large missing transverse momentum in pp collisions at √ s = 8 TeV with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abud AA
- Abulaiti Y
- Acharya BS
- Achkar B
- Adachi S
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
- Adersberger M
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmadov F
- Ahmed WS
- Ai X
- Aielli G
- Akatsuka S
- Akesson TPA
- Akilli E
- Akimov A
- Al Khoury K
- Alberghi GL
- Albert J
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Alhroob M
- Ali B
- Aliev M
- Alimonti G
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alvarez Estevez M
- Alviggi MG
- Amaral Coutinho Y
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amor Dos Santos SP
- Amoroso S
- Amrouche CS
- An F
- Anastopoulos C
- Andari N
- Andeen T
- Anders CF
- Anders JK
- Andreazza A
- Andrei V
- Anelli CR
- Angelidakis S
- Angerami A
- Anisenkov A
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Aparisi Pozo JA
- Araque JP
- Araujo Ferraz V
- Araujo Pereira R
- Araya ST
- Arcangeletti C
- Arce ATH
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Arling J-H
- Armbruster AJ
- Armstrong A
- Arnaez O
- Arnold H
- Artoni G
- Artz S
- Asai S
- Asawatavonvanich T
- Asbah N
- Asimakopoulou EM
- Asquith L
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- Astalos R
- Astigarraga MEP
- Atkin RJ
- Atkinson M
- Atlay NB
- Atmani H
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- Ayoub MK
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- Bachacou H
- Bachas K
- Backes M
- Backman F
- Bagnaia P
- Bahmani M
- Bahrasemani H
- Bailey AJ
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- Scholer PG
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- Schroeder TV
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- Sydorenko A
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- Tskhadadze EG
- Tsopoulou M
- Tsukerman II
- Tsulaia V
- Tsuno S
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- Tu Y
- Tudorache A
- Tudorache V
- Tulbure TT
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- Turchikhin S
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- Urban J
- Urquijo P
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- Vacek V
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- Vadla KOH
- Vaidya A
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- Vale MAB
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- Valentinetti S
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- Van Vulpen I
- Vanadia M
- Vandelli W
- Vandenbroucke M
- Vandewall ER
- Vaniachine A
- Vannicola D
- Vari R
- Varnes EW
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- Varol T
- Varouchas D
- Varvell KE
- Vasile ME
- Vasquez GA
- Vazeille F
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- Vazquez CJB
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- Vermeulen JC
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- von Buddenbrock SE
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- Vossebeld JH
- Vozak M
- Vranjes N
- Vrba V
- Vreeswijk M
- Vuillermet R
- Vukotic I
- Wagner P
- Wagner W
- Wagner-Kuhr J
- Wahdan S
- Wahlberg H
- Walbrecht VM
- Walder J
- Walker R
- Walker SD
- Walkowiak W
- Wallangen V
- Wang AM
- Wang AZ
- Wang C
- Wang F
- Wang H
- Wang J
- Wang P
- Wang Q
- Wang R
- Wang R-J
- Wang SM
- Wang W
- Wang WT
- Wang WX
- Wang Y
- Wang Z
- Wanotayaroj C
- Warburton A
- Ward CP
- Wardrope DR
- Warrack N
- Washbrook A
- Watson AT
- Watson MF
- Watts G
- Waugh BM
- Webb AF
- Webb S
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- Weber MS
- Weber SA
- Weber SM
- Weidberg AR
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- Wengler T
- Wenig S
- Wermes N
- Werner MD
- Wessels M
- Weston TD
- Whalen K
- Whallon NL
- Wharton AM
- White A
- White AS
- White MJ
- Whiteson D
- Whitmore BW
- Wiedenmann W
- Wiel C
- Wielers M
- Wieseotte N
- Wiglesworth C
- Wiik-Fuchs LAM
- Wilkens HG
- Wilkins LJ
- Williams HH
- Williams S
- Willis C
- Willocq S
- Wingerter-Seez I
- Winkels E
- Winklmeier F
- Winston OJ
- Winter BT
- Wittgen M
- Wobisch M
- Wolf A
- Wolf TMH
- Wolff R
- Wolker R
- Wollrath J
- Wolter MW
- Wolters H
- Wong VWS
- Woods NL
- Worm SD
- Wosiek BK
- Wozniak KW
- Wraight K
- Wu SL
- Wu X
- Wu Y
- Wyatt TR
- Wynne BM
- Xella S
- Xi Z
- Xia L
- Xiao X
- Xiotidis I
- Xu D
- Xu H
- Xu L
- Xu T
- Xu W
- Xu Z
- Yabsley B
- Yacoob S
- Yajima K
- Yallup DP
- Yamaguchi N
- Yamaguchi Y
- Yamamoto A
- Yamatani M
- Yamazaki T
- Yamazaki Y
- Yan Z
- Yang HJ
- Yang HT
- Yang S
- Yang T
- Yang X
- Yang Y
- Yao W-M
- Yap YC
- Yasu Y
- Yatsenko E
- Ye H
- Ye J
- Ye S
- Yeletskikh I
- Yexley MR
- Yigitbasi E
- Yildiz HD
- Yorita K
- Yoshihara K
- Young C
- Young CJS
- Yu J
- Yuan R
- Yue X
- Zaazoua M
- Zabinski B
- Zacharis G
- Zaffaroni E
- Zahreddine J
- Zaitsev AM
- Zakareishvili T
- Zakharchuk N
- Zambito S
- Zanzi D
- Zaripovas DR
- Zeissner S
- Zeitnitz C
- Zemaityte G
- Zeng JC
- Zenin O
- Zenis T
- Zerwas D
- Zgubic M
- Zhang B
- Zhang DF
- Zhang G
- Zhang H
- Zhang J
- Zhang L
- Zhang M
- Zhang R
- Zhang S
- Zhang X
- Zhang Y
- Zhang Z
- Zhao P
- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
- Zhou B
- Zhou C
- Zhou M
- Zhou MS
- Zhou N
- Zhou Y
- Zhu C
- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine N
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zivkovic L
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zu Theenhausen HM
- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
Results of a search for new phenomena in final states with an energetic jet and large missing transverse momentum are reported. The search uses 20.3 fb−1 of √ s = 8 TeV data collected in 2012 with the ATLAS detector at the LHC. Events are required to have at least one jet with pT > 120 GeV and no leptons. Nine signal regions are considered with increasing missing transverse momentum requirements between Emiss T > 150 GeV and Emiss T > 700 GeV. Good agreement is observed between the number of events in data and Standard Model expectations. The results are translated into exclusion limits on models with either large extra spatial dimensions, pair production of weakly interacting dark matter candidates, or production of very light gravitinos in a gauge-mediated supersymmetric model. In addition, limits on the production of an invisibly decaying Higgs-like boson leading to similar topologies in the final state are presente
Improving the effectiveness of sickness benefit case management through a public-private partnership? A difference-in-difference analysis in eighteen Danish municipalities
- Author
- A Oakley
- AC Cameron
- AE Young
- AE Young
- B Aust
- Birgit Aust
- BK Graversen
- BT Gardner
- C Caroll
- CDG Stoltenberg
- E MacEachen
- E Yelin
- Eurostat
- F Schaafsma
- H Hlobil
- J Høgelund
- J Høgelund
- Jan Høgelund
- JD Angrist
- K Johansen
- K Johansen
- K Nielsen
- KT Palmer
- Malene Rode Larsen
- MB Nielsen
- MF Andersen
- MHT Martin
- MHT Martin
- MHT Martin
- OECD
- OM Poulsen
- P Craig
- P Loisel
- P Loisel
- PE Greenberg
- R Bille
- R-L Franche
- RL Franche
- S Schandelmaier
- T Lund
- The Danish Health and Medicines Authority Danish Centre for Health Technology Assessment
- V Borg
- W Shaw
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Activation of mGlu3 Receptors Stimulates the Production of GDNF in Striatal Neurons
- Author
- A Cozzi
- A Eslamboli
- A Schober
- A Schober
- A. Lehmann
- AE Lang
- B Greco
- B Pakkenberg
- Barbara Riozzi
- C Corti
- CA Nosrat
- Carla L. Busceti
- Corrado Corti
- D Sharon
- DD Schoepp
- DM Gash
- DM Lovinger
- DM Lovinger
- Domenico Bucci
- ER Matarredona
- F Nicoletti
- Ferdinando Nicoletti
- G Battaglia
- G Battaglia
- G Battaglia
- G Battaglia
- G Battaglia
- Gemma Molinaro
- Giuseppe Battaglia
- Giuseppina Mudò
- Hitoshi Okazawa
- HJG Gundersen
- HJG Gundersen
- J González-Maeso
- J Konieczny
- J Schiefer
- JA Aguirre
- JA Monn
- JH Kordower
- JH Kordower
- JL Venero
- JP Conn
- JP Pin
- JT Slevin
- K Rose
- KBJ Franklin
- KT Poulsen
- L Dawson
- LF Lin
- M D'Onofrio
- M Osikowicz
- M Trupp
- M Yokoi
- Marianna Storto
- Mauro Corsi
- MF Olive
- MJ Fell
- ML Woolley
- MM Bespalov
- MS Airaksinen
- N Belluardo
- Natale Belluardo
- NP Visanji
- O Weinreb
- Paola Spinsanti
- R Ciccarelli
- R Schmidt-Kastner
- SA Mandel
- SS Gill
- ST Patil
- TM Mayhew
- V Bruno
- V Bruno
- V Bruno
- Valentina Di Liberto
- Valeria Bruno
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2009
- Field of study
Metabotropic glutamate (mGlu) receptors have been considered potential targets
for the therapy of experimental parkinsonism. One hypothetical advantage
associated with the use of mGlu receptor ligands is the lack of the adverse
effects typically induced by ionotropic glutamate receptor antagonists, such as
sedation, ataxia, and severe learning impairment. Low doses of the mGlu2/3
metabotropic glutamate receptor agonist, LY379268 (0.25–3 mg/kg, i.p.)
increased glial cell line-derived neurotrophic factor (GDNF) mRNA and protein
levels in the mouse brain, as assessed by in situ
hybridization, real-time PCR, immunoblotting, and immunohistochemistry. This
increase was prominent in the striatum, but was also observed in the cerebral
cortex. GDNF mRNA levels peaked at 3 h and declined afterwards, whereas GDNF
protein levels progressively increased from 24 to 72 h following LY379268
injection. The action of LY379268 was abrogated by the mGlu2/3 receptor
antagonist, LY341495 (1 mg/kg, i.p.), and was lost in mGlu3 receptor knockout
mice, but not in mGlu2 receptor knockout mice. In pure cultures of striatal
neurons, the increase in GDNF induced by LY379268 required the activation of the
mitogen-activated protein kinase and phosphatidylinositol-3-kinase pathways, as
shown by the use of specific inhibitors of the two pathways. Both in
vivo and in vitro studies led to the conclusion
that neurons were the only source of GDNF in response to mGlu3 receptor
activation. Remarkably, acute or repeated injections of LY379268 at doses that
enhanced striatal GDNF levels (0.25 or 3 mg/kg, i.p.) were highly protective
against nigro-striatal damage induced by
1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine in mice, as assessed by
stereological counting of tyrosine hydroxylase-positive neurons in the pars
compacta of the substantia nigra. We speculate that selective mGlu3 receptor
agonists or enhancers are potential candidates as neuroprotective agents in
Parkinson's disease, and their use might circumvent the limitations
associated with the administration of exogenous GDNF
Diabetes, periodontitis, and the subgingival microbiota
- Author
- Aldridge JP
- Axelsson P
- Bissada NF
- Campus G
- Cianciola LJ
- Crasta K
- Cullinan MP
- D'Aiuto F
- Ebersole JL
- Edward J. Ohlrich
- Emrich LJ
- Engebretson S
- Ficara AJ
- Ford PJ
- Forner L
- Frantzis TG
- Gemmell E
- Griffiths GS
- Grossi SG
- Hamlet SM
- Hart TC
- Havemose-Poulsen A
- Hintao J
- Janket SJ
- Johnson LL
- Jonathan W. Leichter
- Jones JA
- Kardesler L
- Kent MJ
- Khader YS
- Khaw KT
- Kiran M
- Kolenbrander PE
- Lalla E
- Lalla E
- Lamster IB
- Lindhe J
- Listgarten MA
- Loe H
- Loe H
- Loesche WJ
- Loos BG
- Mandell RL
- Manouchehr-Pour M
- Manouchehr-Pour M
- Marsh PD
- Mary P. Cullinan
- Mashimo PA
- Mattila KJ
- Nelson RG
- Nesse W
- Noack B
- Offenbacher S
- Ojima M
- Papapanou P
- Preshaw PM
- Ramamurthy NS
- Ramamurthy NS
- Reynolds JJ
- Rodrigues DC
- Rosling B
- Rylander H
- Salvi GE
- Salvi GE
- Sastrowijoto SH
- Sastrowijoto SH
- Sbordone L
- Sbordone L
- Schnider SL
- Seppala B
- Seppala B
- Seymour GJ
- Seymour GJ
- Simpson TC
- Skaleric U
- Slade GD
- Socransky SS
- Socransky SS
- Taylor GW
- Taylor GW
- Teeuw WJ
- Tervonen T
- Tervonen T
- Tervonen T
- Thorstensson H
- Thorstensson H
- Wild S
- Willershausen-Zonnchen B
- Yamazaki K
- Yamazaki K
- Publication venue
- CoAction Publishing
- Publication date
- 01/01/2010
- Field of study
Both type 1 and type 2 diabetes have been associated with increased severity of periodontal disease for many years. More recently, the impact of periodontal disease on glycaemic control has been investigated. The role of the oral microbiota in this two-way relationship is at this stage unknown. Further studies, of a longitudinal nature and investigating a wider array of bacterial species, are required in order to conclusively determine if there is a difference in the oral microbiota of diabetics and non-diabetics and whether this difference accounts, on the one hand, for the increased severity of periodontal disease and on the other for the poorer glycaemic control seen in diabetics
Epigenetic regulation of caloric restriction in aging
- Author
- A Brunet
- A Melk
- A Petronis
- A Razin
- A Richardson
- A Vageuro
- A Vaquero
- A Vaquero
- AB Crujeiras
- AJ de Ruijter
- AL Clayton
- AP Bracken
- AY So
- B Agarwal
- B Merry
- BD Mitchell
- BD Strahl
- BK Kennedy
- BS Hass
- C Cruzen
- CB Yoo
- CK Taylor
- CM McCay
- D Sun
- DA Alcorta
- DA Sinclair
- E Langley
- E Li
- E Li
- FI Milagro
- G Egger
- GS Roth
- H Vaziri
- H Wong
- HL Cheng
- HY Cohen
- HY Cohen
- IB Leibiger
- J Campión
- J Gil
- J Jeong
- J Ketonen
- J Knapowski
- J Koubova
- J Krishnamurthy
- J Luo
- J Mill
- JA Baur
- JC Mathers
- JG Wood
- JJ Jacobs
- JL Barger
- JM Dhahbi
- JO Holloszy
- JP Issa
- JP Issa
- JP Issa
- JT Kadonaga
- K Luger
- KR Patel
- KT Howitz
- L Bordone
- L Bouchard
- L Chouliaras
- L Guarente
- L Subramanian
- LA Wakeling
- LC Sambucetti
- M Forster
- M Hernandez-Valencia
- M Meyerson
- M Okano
- M Okano
- M Okano
- MC Haigis
- MC Motta
- MF Chan
- MG Goll
- Michael Daniel
- MM Schilling
- MW McBurney
- NA Oates
- P Fischer-Posovszky
- P Oberdoerffer
- P Poulsen
- P Wu
- PA Callinan
- R Sohal
- R Weindruch
- R Weindruch
- RB Vega
- RJ Colman
- RP Singhal
- RS Ahima
- SD Gore
- SH Cross
- SJ Lin
- SK Kia
- SM Meeran
- SM Meeran
- T Chen
- T Kanaya
- T Kouzarides
- T Kouzarides
- T Waki
- TD Pugh
- TD Shanafelt
- TM Bass
- TM Cooper
- TM Larsen
- Trygve O Tollefsbol
- TY Kim
- U Muñoz-Najar
- V Jayagopal
- VL Wilson
- VM Richon
- VM Richon
- W Fischle
- X Ma
- Y Kanfi
- Y Li
- Y Li
- Y Li
- Y Li
- Y Li
- Y Li
- YH Kao
- Yuanyuan Li
- ZD Sharp
- Publication venue
- BioMed Central
- Publication date
- 01/01/2011
- Field of study
The molecular mechanisms of aging are the subject of much research and have facilitated potential interventions to delay aging and aging-related degenerative diseases in humans. The aging process is frequently affected by environmental factors, and caloric restriction is by far the most effective and established environmental manipulation for extending lifespan in various animal models. However, the precise mechanisms by which caloric restriction affects lifespan are still not clear. Epigenetic mechanisms have recently been recognized as major contributors to nutrition-related longevity and aging control. Two primary epigenetic codes, DNA methylation and histone modification, are believed to dynamically influence chromatin structure, resulting in expression changes of relevant genes. In this review, we assess the current advances in epigenetic regulation in response to caloric restriction and how this affects cellular senescence, aging and potential extension of a healthy lifespan in humans. Enhanced understanding of the important role of epigenetics in the control of the aging process through caloric restriction may lead to clinical advances in the prevention and therapy of human aging-associated diseases
Combined measurement of differential and total cross sections in the H → γγ and the H → ZZ* → 4ℓ decay channels at s=13 TeV with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abhayasinghe DK
- Abidi SH
- Abouzeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Adachi S
- Adamczyk L
- Adelman J
- Adersberger M
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmadov F
- Aielli G
- Akatsuka S
- Akilli E
- Akimov AV
- Alberghi GL
- Albert J
- Albicocco P
- Alconada Verzini MJ
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alhroob M
- Ali B
- Alimonti G
- Alison J
- Alkire SP
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alstaty MI
- Alvarez Gonzalez B
- Alviggi MG
- Amadio BT
- Amaral Coutinho Y
- Ambroz L
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- Angelozzi I
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- Zwalinski L
- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- Elsevier
- Publication date
- 01/01/2018
- Field of study
A combined measurement of differential and inclusive total cross sections of Higgs boson production is performed using 36.1 fb−1 of 13 TeV proton–proton collision data produced by the LHC and recorded by the ATLAS detector in 2015 and 2016. Cross sections are obtained from measured H→γγ and H→ZZ*(→4ℓ event yields, which are combined taking into account detector efficiencies, resolution, acceptances and branching fractions. The total Higgs boson production cross section is measured to be 57.0−5.9 +6.0 (stat.) −3.3 +4.0 (syst.) pb, in agreement with the Standard Model prediction. Differential cross-section measurements are presented for the Higgs boson transverse momentum distribution, Higgs boson rapidity, number of jets produced together with the Higgs boson, and the transverse momentum of the leading jet. The results from the two decay channels are found to be compatible, and their combination agrees with the Standard Model predictions
Search for High-Mass Resonances Decaying to τν in pp Collisions at √s=13 TeV with the ATLAS Detector
- Author
- Aaboud M
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- Yildiz H Duran
- Yorita K
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- Yuen SPY
- Yusuff I
- Zabinski B
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- Zalieckas J
- Zambito S
- Zanzi D
- Zeitnitz C
- Zemaityte G
- Zeng JC
- Zeng Q
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- Zerwas D
- Zgubic M
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- Zhao X
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zhou B
- Zhou C
- Zhou L
- Zhou M
- Zhou M
- Zhou N
- Zhou Y
- Zhu CG
- Zhu H
- Zhu H
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinonos Z
- Zinser M
- Ziolkowski M
- Zivkovic L
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zu Theenhausen H Meyer
- zur Nedden M
- Zwalinski L
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2018
- Field of study
A search for high-mass resonances decaying to τν using proton-proton collisions at √s=13 TeV produced by the Large Hadron Collider is presented. Only τ-lepton decays with hadrons in the final state are considered. The data were recorded with the ATLAS detector and correspond to an integrated luminosity of 36.1 fb−1. No statistically significant excess above the standard model expectation is observed; model-independent upper limits are set on the visible τν production cross section. Heavy W′ bosons with masses less than 3.7 TeV in the sequential standard model and masses less than 2.2–3.8 TeV depending on the coupling in the nonuniversal G(221) model are excluded at the 95% credibility level
Measurement of differential cross sections and W + /W − cross-section ratios for W boson production in association with jets at √s =8 TeV with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abreu R
- Abulaiti Y
- Acharya BS
- Adachi S
- Adamczyk L
- Adelman J
- Adersberger M
- Adye T
- Affolder AA
- Afik Y
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahlen SP
- Ahmadov F
- Aielli G
- Akatsuka S
- Akerstedt H
- Akesson TPA
- Akilli E
- Akimov AV
- Alberghi GL
- Alberich L Cerda
- Albert J
- Albicocco P
- Alderweireldt SC
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexander G
- Alexopoulos T
- Alhroob M
- Ali B
- Aliev M
- Alimonti G
- Alison J
- Alkire SP
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alstaty MI
- Alviggi MG
- Amadio BT
- Amelung C
- Amidei D
- Amoroso S
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders JK
- Anderson KJ
- Andreazza A
- Andrei V
- Angelidakis S
- Angelozzi I
- Angerami A
- Anisenkov AV
- Anjos N
- Annovi A
- Antel C
- Antonelli M
- Antonov A
- Antrim DJ
- Anulli F
- Aoki M
- Arabidze G
- Arai Y
- Araque JP
- Arce ATH
- Ardell RE
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Arik M
- Armadans R Caminal
- Armbruster AJ
- Armitage LJ
- Arnaez O
- Arnold H
- Arratia M
- Arslan O
- Artamonov A
- Artoni G
- Artz S
- Asai S
- Asbah N
- Ashkenazi A
- Asquith L
- Assamagan K
- Astalos R
- Astigarraga ME Pozo
- Atkinson M
- Atlay NB
- Augsten K
- Avolio G
- Axen B
- Ayoub MK
- Azuelos G
- Baas AE
- Baca MJ
- Bachacou H
- Bachas K
- Backes M
- Bagnaia P
- Bahmani M
- Bahrasemani H
- Baines JT
- Bajic M
- Baker OK
- Bakker PJ
- Baldin EM
- Balek P
- Balli F
- Balunas WK
- Banas E
- Bandyopadhyay A
- Banerjee Sw
- Bannoura AAE
- Barajas CA Chavez
- Barak L
- Barberio EL
- Barberis D
- Barbero M
- Barillari T
- Barisits M-S
- Barkeloo JT
- Barklow T
- Barlow N
- Barnes SL
- Barnett BM
- Barnett RM
- Barnovska-Blenessy Z
- Baroncelli A
- Barone G
- Barr AJ
- Barreiro F
- Bartoldus R
- Barton AE
- Bartos P
- Basalaev A
- Bassalat A
- Bates RL
- Batista SJ
- Batley JR
- Battaglia M
- Bauce M
- Bauer F
- Bauer KT
- Bawa HS
- Beacham JB
- Beattie MD
- Beau T
- Beauchemin PH
- Bechtle P
- Beck HC
- Beck HP
- Becker K
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- Becot C
- Beddall A
- Beddall AJ
- Bednyakov VA
- Bedognetti M
- Bee CP
- Beermann TA
- Begalli M
- Begel M
- Behr JK
- Bell AS
- Bella G
- Bella L Aperio
- Bellagamba L
- Bellerive A
- Bellomo M
- Belotskiy K
- Beltramello O
- Belyaev NL
- Benary O
- Benchekroun D
- Bender M
- Benekos N
- Benhammou Y
- Benitez J
- Benjamin DP
- Benoit M
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Berge D
- Berger N
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- Zhemchugov A
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- Zibell A
- Zieminska D
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- Zimmermann C
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- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2018
- Field of study
This paper presents a measurement of the W boson production cross section and the W + /W − cross-section ratio, both in association with jets, in proton--proton collisions at s √ =8 TeV with the ATLAS experiment at the Large Hadron Collider. The measurement is performed in final states containing one electron and missing transverse momentum using data corresponding to an integrated luminosity of 20.2 fb −1 . Differential cross sections for events with one or two jets are presented for a range of observables, including jet transverse momenta and rapidities, the scalar sum of transverse momenta of the visible particles and the missing transverse momentum in the event, and the transverse momentum of the W boson. For a subset of the observables, the differential cross sections of positively and negatively charged W bosons are measured separately. In the cross-section ratio of W + /W − the dominant systematic uncertainties cancel out, improving the measurement precision by up to a factor of nine. The observables and ratios selected for this paper provide valuable input for the up quark, down quark, and gluon parton distribution functions of the proto
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