215 research outputs found
Brucellosis Vaccines: Assessment of Brucella melitensis Lipopolysaccharide Rough Mutants Defective in Core and O-Polysaccharide Synthesis and Export
- Author
- A Cloeckaert
- A Cloeckaert
- A Dricot
- A Sola-Landa
- AJ Winter
- Andrej Weintraub
- B Garin-Bastuji
- B Garin-Bastuji
- B Kneidinger
- BA Freeman
- C Galanos
- C Guzman-Verri
- CA Allen
- CL Baldwin
- Clara-M. Marín
- CM Fernández-Prada
- CM Fernández-Prada
- CR Raetz
- D Cafiso
- D Fretin
- D Monreal
- David González
- DE Heinrichs
- DV Bowser
- E Barquero-Calvo
- E Moreno
- F Godfroid
- F Porte
- FJ Sangari
- G Rajashekara
- GG Alton
- GG Schurig
- GG Schurig
- Göran Widmalm
- I Maudlin
- I Moriyón
- Ignacio López-Goñi
- Ignacio Moriyón
- J Ariza
- J Celli
- J Pei
- J Velasco
- J Zinsstag
- JB Wilson
- JE Ugalde
- Jean-Jacques Letesson
- Jean-Pierre Gorvel
- José-María Blasco
- JR McQuiston
- Kirsten Nielsen
- L Braunschweiler
- M Caroff
- M Iriarte
- M Ito
- M Rosenberg
- Maite Iriarte
- María-Jesús De Miguel
- María-Jesús Grilló
- MB Perry
- MG Rittig
- Michel Zygmunt
- MJ Grilló
- MJ Grilló
- MM Kahl-McDonagh
- MP Jiménez de Bagüés
- N Rojas
- N Vizcaíno
- NP Price
- O Holst
- PG Detilleux
- PG Detilleux
- Pilar Muñoz
- PL Nicoletti
- R Adone
- R Conde-Álvarez
- R Hallez
- R Vemulapalli
- RA Bowden
- Raquel Conde-Álvarez
- RM Crawford
- RM Delrue
- Rose-May Delrue
- S Kohler
- Tara Ali
- V Aragón
- V Haine
- Vilma Arce-Gorvel
- W Braun
- WW Spink
- YA Knirel
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2008
- Field of study
Background: The brucellae are facultative intracellular bacteria that cause brucellosis, one of the major neglected zoonoses. In endemic areas, vaccination is the only effective way to control this disease. Brucella melitensis Rev 1 is a vaccine effective against the brucellosis of sheep and goat caused by B. melitensis, the commonest source of human infection. However, Rev 1 carries a smooth lipopolysaccharide with an O-polysaccharide that elicits antibodies interfering in serodiagnosis, a major problem in eradication campaigns. Because of this, rough Brucella mutants lacking the O-polysaccharide have been proposed as vaccines. Methodology/Principal Findings: To examine the possibilities of rough vaccines, we screened B. melitensis for lipopolysaccharide genes and obtained mutants representing all main rough phenotypes with regard to core oligosaccharide and O-polysaccharide synthesis and export. Using the mouse model, mutants were classified into four attenuation patterns according to their multiplication and persistence in spleens at different doses. In macrophages, mutants belonging to three of these attenuation patterns reached the Brucella characteristic intracellular niche and multiplied intracellularly, suggesting that they could be suitable vaccine candidates. Virulence patterns, intracellular behavior and lipopolysaccharide defects roughly correlated with the degree of protection afforded by the mutants upon intraperitoneal vaccination of mice. However, when vaccination was applied by the subcutaneous route, only two mutants matched the protection obtained with Rev 1 albeit at doses one thousand fold higher than this reference vaccine. These mutants, which were blocked in O-polysaccharide export and accumulated internal O-polysaccharides, stimulated weak anti-smooth lipopolysaccharide antibodies. Conclusions/Significance: The results demonstrate that no rough mutant is equal to Rev 1 in laboratory models and question the notion that rough vaccines are suitable for the control of brucellosis in endemic areas.This work was funded by the European Commission (Research Contract QLK2-CT-2002-00918) and the Ministerio de Ciencia y Tecnología of Spain
(Proyecto AGL2004-01162/GAN)
Increased Expression of Fatty-Acid and Calcium Metabolism Genes in Failing Human Heart
- Author
- A Galione
- A Galione
- A Garnier
- A Garnier
- A MacGregor
- AF Dulhunty
- AJ Murray
- AJ Murray
- Ana Mosquera-Leal
- AT Turer
- B Desvergne
- B Huang
- B Mettauer
- BN Finck
- D Harzheim
- Diego Rodríguez-Penas
- DJ Crossman
- DS Luciani
- E Roselló-Lletí
- Francisca Lago
- G Karamanlidis
- GP Holloway
- GP Holloway
- HY Fu
- IP Uray
- J Karbowska
- J Pohl
- J Yang
- JD Bayer
- JF Glatz
- JF Glatz
- JG Duncan
- JG Duncan
- JH Rennison
- John Parrington
- Josep Brugada
- José Ramón González-Juanatey
- JS Jaswal
- L Teboul
- LC Heather
- LC Heather
- LH Opie
- LO Go
- M Schupp
- M Sebastiani
- M van Bilsen
- M Yano
- Manuel Calaza
- Manuel Francisco Otero
- Manuel Portolés
- María Álvarez-Barredo
- Miguel Rivera
- MJ Goikoetxea
- MM Pelsers
- MN Sack
- NH Son
- O Sato
- P Razeghi
- P Wu
- Pamela Virginia Lear
- PJ Calcraft
- R Cortés
- R Cortés
- R Cortés
- R Hooper
- Rajesh Gopalrao Katare
- RW Schwenk
- S Fu
- S Sihag
- Sandra Feijóo-Bandín
- T Kato
- T Minamino
- T Nakata
- Teresa Noguera-Moreno
- V Lionetti
- V Sharma
- Vanessa García-Rúa
- WC Stanley
- X Zong
- Publication venue
- Public Library of Science
- Publication date
- 06/06/2012
- Field of study
Heart failure (HF) involves alterations in metabolism, but little is known about cardiomyopathy-(CM)-specific or diabetes-independent alterations in gene expression of proteins involved in fatty-acid (FA) uptake and oxidation or in calcium-(Ca(2+))-handling in the human heart.RT-qPCR was used to quantify mRNA expression and immunoblotting to confirm protein expression in left-ventricular myocardium from patients with HF (n = 36) without diabetes mellitus of ischaemic (ICM, n = 16) or dilated (DCM, n = 20) cardiomyopathy aetiology, and non-diseased donors (CTL, n = 6).Significant increases in mRNA of genes regulating FA uptake (CD36) and intracellular transport (Heart-FA-Binding Protein (HFABP)) were observed in HF patients vs CTL. Significance was maintained in DCM and confirmed at protein level, but not in ICM. mRNA was higher in DCM than ICM for peroxisome-proliferator-activated-receptor-alpha (PPARA), PPAR-gamma coactivator-1-alpha (PGC1A) and CD36, and confirmed at the protein level for PPARA and CD36. Transcript and protein expression of Ca(2+)-handling genes (Two-Pore-Channel 1 (TPCN1), Two-Pore-Channel 2 (TPCN2), and Inositol 1,4,5-triphosphate Receptor type-1 (IP3R1)) increased in HF patients relative to CTL. Increases remained significant for TPCN2 in all groups but for TPCN1 only in DCM. There were correlations between FA metabolism and Ca(2+)-handling genes expression. In ICM there were six correlations, all distinct from those found in CTL. In DCM there were also six (all also different from those found in CTL): three were common to and three distinct from ICM.DCM-specific increases were found in expression of several genes that regulate FA metabolism, which might help in the design of aetiology-specific metabolic therapies in HF. Ca(2+)-handling genes TPCN1 and TPCN2 also showed increased expression in HF, while HF- and CM-specific positive correlations were found among several FA and Ca(2+)-handling genes
Plasma lipid profiles discriminate bacterial from viral infection in febrile children
- Author
- Abdulla Amina F.
- Abelake Eugenia
- Adeel Abdul O.A.
- Aebi C.
- Agapow Paul Michael
- Agbeko R.
- Agyeman P.
- Anderson Suzanne T. B.
- Antonio Martin
- Baer W.
- Barham Heather
- Barral-Arca Ruth
- Barrios Xavier Gabaldó
- Bartlett Maeve
- Baumgart Hinrich
- Baumgartner Markus
- Behrends Uta
- Bellos Evangelos
- Berger C.
- Bernhard-Stirnemann S.
- Biebl Ariane
- Binder Alexander
- Birnbacher Robert
- Blanke Jan-Gerd
- Boeddha Navin P.
- Boelke Carsten
- Bojang Kalifa
- Bokhandi Sumit S.
- Breukels Meike A.
- Breuling Kai
- Brinkman Daniëlle M.C.
- Brió Sanagustín Sonia
- Brunner Jürgen
- Buller Maria
- Camuzeaux Stephane
- Carreras Eduardo
- Carrol Enitan
- Carter Stephanie
- Cebey-López Miriam
- Coin Lachlan
- Cole Fatoumata
- Concha-Torre Andres
- Contreras J. Ruiz
- Cooke Elizabeth Fernández
- Crate Lindsay
- Curras-Tuala MJ.
- Dahlem Peter
- Davies Patrick
- de Groot Ronald C.A.
- de Korte Carla C.M.M.
- de Vries Esther
- de Waal Wouter J.
- Deep Akash
- Dekkers Roel
- Dietrich Beate
- Dings-Lammertink Anouk
- Doedens Rienus A.
- Donker AlbertineE.
- Dousma Mieke
- Driessen Gertjan Jan A.
- Eber Ernst
- Eleftherohorinou Hariklia
- Elias Johannes
- Ellacuriagal Antonio Salas
- Emhofer Josef
- Emonts Marieke
- Etschmaier Rosa
- Faber Tina E.
- Farr Sebastian
- Faust Saul
- Fernandez Silveira Laura
- Fernández Reyes
- Fink Colin
- Forte Miguel Sanchez
- Fortune Peter-Marc
- Frederick H.
- Galassini Rachel
- García Natalia
- Gerrits Gerardus Peter Jm M.
- Gerver Jan A.M.
- Giannoni E.
- Giménez-Sánchez Francisco
- Girtler Ylenia
- Gomez-Rial Jose
- Gormley Stuart
- Grela Beiroa Andrea
- Gribbin Andrew
- Grigorow Irina
- Guillén Sara
- Gómez-Carballa Alberto
- Hamilton Shea
- Hardy Katie
- Hasters P.
- Hazelzet Jan
- Heidema Jojanneke
- Heimann Konrad
- Heininger U.
- Herberg Jethro A.
- Hoggart Clive
- Hourmat Bernardo
- Hudnott Phil
- Ibarrondo Cristina Epalza
- Iglesias Pilar Leboráns
- Ihm Ulrike
- Inwald David
- Jackson Heather R.
- Jacobs Monique A.M.
- Jansen Nicolaas Johannes Georgius
- Jaros Zdenek
- Jennings R.
- Johnston J.
- Justicia-Grande Antonio
- Kaforou Myrsini
- Kahlert C.
- Kalhoff Hermann
- Kaulfersch Wilhelm
- Kawczynski Pawel
- Kebbeh Ngange
- Kemen Christoph
- Kenwright R.
- khan Saidy
- Klobassa Daniela S.
- Klocker Nina
- Klucovska Kristine
- Kneyber Martin Christiaan Jacques
- Kohlmaier B.
- Komini Eleni
- Koopman-Keemink Yvonne
- Kramer Lydia
- Kuijpers T. W.
- Köster Bernhard
- Langenhorst Veerle J.
- Leusink José
- Levin Michael
- Lewis Matthew R.
- León-León María Cruz
- Loza Bettina F.
- Lozano María José
- Maconochie Ian
- Martinez-Santos Alba-Elena
- Martinon-Torres Federico
- MartinónTorres Nazareth
- Mashbat Meg
- McClure Myra
- McCorkell Jenni
- McCready Sharon
- Menikou Stephanie
- Merth Istvan T.
- Miedema Carien J.
- Mingorance Álvaro Navarro
- Momodou None
- Moreno David
- Morillo Gutiérrez Beatriz
- Mustafa Sobia
- Nadel Simon
- Navarra Helen
- Navarro Marisa
- Neeleman Chris
- Neth Olaf
- Neubert Antje
- Niederer-Loher A.
- Nijman Ruud Gerard
- Noordzij Jeroen G.
- Nsirim Eniola
- O'Neill Lauren
- Obihara Charlie C.C.
- Ortner Daniel
- Oñate Vergara Eider
- Pacheco Pablo Obando
- Padilla Ma del Carmen Martínez
- Pajkrt Dasja
- Palacios Alba
- Pardo-Seco Jacobo
- Patel Rekha
- Patel Sanjay Valabh
- Pathan Nazima
- Paulus Stéphane
- Pescollderungg Lydia
- Peters Mark
- Pfurtscheller Klaus
- Philipsen H.L.A.
- Pinnock Elli
- Pischedda Sara
- Pollard AJ.
- Poortman Geriska H.
- Posfay-Barbe K.M.
- Potgieter Steph T.
- Potjewijd Joke
- Power Sue
- Pérez Belén Mosquera
- Rahman Rahmeen
- Ramaiah Raghu
- Redondo Collazo Lorenzo
- Reiter Karl
- Relly C.
- Reyes Susana Beatriz
- Ridout Jenna
- Ristic Goran
- Rivero Calle Irene
- Rodríguez-Tenreiro Carmen
- Rojo Pablo
- Rosias Philippe P.R.
- Royad C.
- Rödl Siegfried
- Sagmeister Manfred
- Salas Antonio
- Sancho Shimizu Vanessa
- Sanders Elisabeth A. M.
- Sands Caroline
- Sarr Isatou
- Schlapbach Luregn
- Schweintzger Nina
- Secka Fatou
- Sellner Andrea
- Serén Fernández Sonia
- Sey Gibbi
- Silva María Del Sol Porto
- Sonnleitner Astrid
- Sperl Matthias
- Sprong Tom
- Standing Fran
- Stelzl Wolfgang
- Stocker M.
- Sánchez José María
- Tato Luis Manuel Prieto
- ten Tussher Gavin W.
- Thakker Clare
- Thio Bernard J.
- Thomas Gilleh
- Thurston Sarah
- Till Holger Karl H.
- Tramper-Stranders Gerdien A.
- Trobisch Andreas
- Tuffrey Catherine
- Universitat Autònoma de Barcelona
- van de Beek Diederik
- van der Ende Arie
- Van der Flier Michiel
- van der Meer H.
- van Deuren M.
- van Furth Marceline
- van Kuppevelt A.J.M.
- van Overbeek - van Gils A. Lidy T.
- van Wermeskerken A.M.
- Veen J. Homan-van der
- Vega Ana Karina
- Verwijs W.A.
- Vicente Luisa García
- Vierzig Anne
- Vilanova-Trillo Lucía
- Vivanco Ana
- Vogel Ulrich
- Wang Xinzhu
- Weingarten Christina
- Welke Stefanie
- White Deborah K.
- White Sonia
- Willis Louise
- Wimmer Andreas
- Wintergerst Uwe
- Wolfs T.F.W.
- Wright Victoria
- Wüller Daniel
- Young Zoe
- Zaunschirm Andrew
- Zenz Werner
- Ziuraite Ieva
- Álvarez Cristina Álvarez
- Álvez González Fernando
- Žukovskaja Veslava
- Publication venue
- Publication date
- 01/01/2019
- Field of study
Fever is the most common reason that children present to Emergency Departments. Clinical signs and symptoms suggestive of bacterial infection are often non-specific, and there is no definitive test for the accurate diagnosis of infection. The 'omics' approaches to identifying biomarkers from the host-response to bacterial infection are promising. In this study, lipidomic analysis was carried out with plasma samples obtained from febrile children with confirmed bacterial infection (n = 20) and confirmed viral infection (n = 20). We show for the first time that bacterial and viral infection produces distinct profile in the host lipidome. Some species of glycerophosphoinositol, sphingomyelin, lysophosphatidylcholine and cholesterol sulfate were higher in the confirmed virus infected group, while some species of fatty acids, glycerophosphocholine, glycerophosphoserine, lactosylceramide and bilirubin were lower in the confirmed virus infected group when compared with confirmed bacterial infected group. A combination of three lipids achieved an area under the receiver operating characteristic (ROC) curve of 0.911 (95% CI 0.81 to 0.98). This pilot study demonstrates the potential of metabolic biomarkers to assist clinicians in distinguishing bacterial from viral infection in febrile children, to facilitate effective clinical management and to the limit inappropriate use of antibiotics
Present and future evolution of advanced breast cancer therapy
- Author
- A Citri
- A Frassoldati
- A Kolman
- A Moreno-Aspitia
- A Tutt
- AL Harris
- AL Thomas
- AU Buzdar
- BB Friday
- BI Rini
- BT Hennessy
- C Bunnell
- C Erlichman
- C Giordano
- C Twelves
- CC Portera
- CE Geyer
- CG Kleer
- CJ Torrance
- CL Vogel
- D Cunningham
- D Hanahan
- D Miles
- DB Agus
- DB Agus
- DB Mendel
- DD Von Hoff
- DJ Riese 2nd
- DJ Slamon
- DM Bollag
- DW End
- DW Rusnak
- E Di Gennaro
- E Raymond
- E Thomas
- EA Perez
- EH Romond
- ER Plummer
- ER Wood
- ES Thomas
- F Ciardiello
- FJ Esteva
- FY Lee
- GE Konecny
- Group EBCTC
- GW Sledge
- H Roche
- HA Burris 3rd
- HE Jones
- HJ Burstein
- HJ Burstein
- HJ Burstein
- HR Tsou
- HS Rugo
- J Baselga
- J Baselga
- J Baselga
- J Cortes
- J Drevs
- J Drevs
- J Folkman
- J Nemunaitis
- J O'Shaughnessy
- J O'Shaughnessy
- J O'Shaughnessy
- J Pierga
- J Zha
- J Zujewski
- JA Low
- JA Sparano
- JH Nettles
- JM Wood
- K Gelmon
- K Kamath
- K Margolin
- K Miller
- K Yu
- KD Miller
- KD Miller
- KJ Kim
- KK Wong
- KL Carraway 3rd
- KL van Golen
- KM Dameron
- L Adnane
- L Chow
- L Gianni
- LA Carey
- LC Cantley
- LW Jones
- M Atkins
- M Beeram
- M Bucan
- M Cristofanilli
- M Guix
- M Wartmann
- M Zambetti
- MA Cobleigh
- MA Cobleigh
- MA Fabian
- MC Franklin
- MJ Piccart-Gebhart
- MN Dickler
- MS Gordon
- MX Sliwkowski
- N Denduluri
- N Ferrara
- NI Goldstein
- P Kiewe
- P Kiewe
- PA Ganz
- PC Fong
- QS Chu
- R Fossati
- R Nahta
- R Swaby
- RI Nicholson
- Ricardo H Álvarez
- RJ Kowalski
- RM O'Regan
- S Chan
- S Faivre
- S Johnston
- S Jones
- S Modi
- SE Lipshultz
- SN Holden
- SR Johnston
- SR Wedge
- T Li
- TS Lewis
- V Guarneri
- V Schreiber
- VA Pollack
- W Xia
- WE Holmes
- Y Mi
- Y Yarden
- Publication venue
- BioMed Central
- Publication date
- Field of study
Although the introduction of novel therapies and drug combinations has improved the prognosis of metastatic breast cancer, the disease remains incurable. Increased knowledge of the biology and the molecular alterations in breast cancer has facilitated the design of targeted therapies. These agents include receptor and nonreceptor tyrosine kinase inhibitors (epidermal growth factor receptor family), intracellular signaling pathways (phosphatidylinositol-3-kinase, AKT, mammalian target of rapamycin) angiogenesis inhibitors and agents that interfere with DNA repair (poly(ADP-ribose) polymerase inhibitors). In the present review, we present the most promising studies of these new targeted therapies and novel combinations of targeted therapies with cytotoxic agents
The management of acute venous thromboembolism in clinical practice. Results from the European PREFER in VTE Registry
- Author
- Abbadie F
- Achkar A
- Ackroyd A
- Adamczyk A
- Adarraga Cansino Md
- Addala A
- Adnet F
- Agarwal G
- Ageno W
- Agnelli G
- Agnelli G
- Al Ahmad W
- Al-Refaie F
- Alexandra Jf
- Alikhan R
- Allameddine A
- Altarriba Es
- Amado Fernández C
- Amitrano M
- Amore M
- Angelini E
- Antignani Pl
- Aquilanti S
- Aranda Blazquez R
- Arden C
- Arellano Rodrigo E
- Armengou Arxe A
- Arone A
- Asaro G
- Attanzio Mt
- Attinasi V
- Austin A
- Axthelm C
- Axthelm P
- Bakhai A
- Balbarini A
- Ballaz Quincoces A
- Banfi E
- Banyai M
- Barcellona D
- Barillari G
- Bartolini C
- Barton T
- Bartone M
- Bauersachs R
- Bauersachs R
- Beato Pérez Jl
- Bedate Díaz P
- Belhassane A
- Bellanuova I
- Bellisi M
- Bellizzi A
- Benaroya B
- Bergmann K
- Berremili T
- Beschorner U
- Bianchi M
- Bianchini G
- Binias Kh
- Birr V
- Blanco-Molina Ma
- Boari B
- Body R
- Bonnin C
- Boral M
- Bortoluzzi C
- Bosler F
- Bramlage P
- Bratsch H
- Brauer G
- Bresin Durand Mg
- Brisot D
- Brodmann M
- Brousse C
- Brucoli C
- Burghard S
- Calandra G
- Calvo Mijares N
- Camporese G
- Canafoglia L
- Cancellieri E
- Cannavacciuolo F
- Caprioli C
- Carlizza A
- Carnovali M
- Carrasco Expósito M
- Carrasco Mas A
- Carugati A
- Casarsa C
- Castellote Mc
- Castrodeza Sanz R
- Catalini R
- Cavazza S
- Cayman C
- Cazaubon M
- Cañas Alcántara I
- Cenci C
- Ceresa F
- Cereto Castro F
- Chacko J
- Champion O
- Chandra D
- Chanut M
- Charters F
- Chevalet P
- Church A
- Ciampa A
- Ciarambino T
- Cillari E
- Cisneros de la Fuente E
- Clemente M
- Clements J
- Clifford P
- Cohen At
- Connault J
- Constans J
- Conti F
- Cordeanu M
- Cosmi B
- Couturaud F
- Cox D
- Crouch M
- Crowther M
- D'Alessandro A
- D'Angelo A
- D'Antoni S
- Davies E
- Davies M
- de Ancos Aracil C
- De Bartolomeo G
- De Campora P
- de Daborenea González Md
- De Dedker L
- De Franciscis S
- De Gaudenzi E
- de la Fuente Aguado J
- De La Fuente T
- de las Revillas Fa
- De Santis F
- De Tommaso I
- Decoulx E
- del Carmen Fernández-Capitán M
- del Toro Cervera J
- Delgado Bregel Jl
- Delpin S
- Demann C
- Demmig A
- Derrien B
- Di Francesco L
- Di Maggio R
- Di Micco P
- Di Michele D
- Di Minno G
- Di Nisio M
- Di Salvo M
- Diamand Jm
- Diard A
- Dimitri S
- Donadio F
- Douadi Y
- Drebes A
- Dres P
- Dupas S
- Durant C
- Díez Fernández F
- D’Avino M
- Eberhad Rieker E
- Eberlein U
- Edhery S
- Eischer L
- Elias Hernández T
- Emter E
- Enea I
- Enger F
- Eschenburg J
- Eschenburg Ju
- Ewad H
- Fabbian F
- Falanga A
- Falvo N
- Farcas Taralunga C
- Felis S
- Fernández Iglesias A
- Ferrari E
- Ferri Bataler R
- Fiore V
- Fontanella A
- Forkmann L
- Franco E
- Frank J
- Frank U
- Franklin S
- Frausini G
- Freischmidt H
- Fritsche I
- Fronk Em
- Fuorlo M
- Gaillard C
- Gallerani M
- Gamberini S
- Garcia Ma
- García García Jm
- García Loizaga A
- García-Bragado Dalmau F
- Garrigues D
- Gassauer M
- George J
- Gerofke H
- Ghirarduzzi A
- Ghizzi G
- Gianniello F
- Gibbs C
- Gillet Jl
- Giordana P
- Girke F
- Gitt Ak
- Giudice G
- Goebels Mc
- Goh T
- González Porras Jr
- González Lg
- Grange C
- Grare F
- Grenot Mc
- Grün-Himmelmann K
- Guarnera G
- Guercini F
- Guggenbichler S
- Guil García M
- Gupta S
- Gutierrez Ja
- Hadj Henni A
- Hartmann K
- Heilberger P
- Heinsius A
- Held M
- Herman G
- Hernando López E
- Herold J
- Hertrich F
- Heuser S
- Hidden-Henic V
- Hirschl M
- Holmes J
- Holtea D
- Hommel H
- Hottin D
- Härtel D
- Hütte G
- Imbert B
- Imberti D
- Iosub Di
- Iozzi E
- Irvine N
- Jackson-Voyzey E
- Jacquinandi V
- Jakob D
- Jara Palomares L
- Jaras Mj
- Jeanneret C
- Jiménez Castro D
- Jiménez-Rodríguez Madridejos R
- Jones N
- Joya Seijo D
- Jungandreas K
- Jurus C
- Jörg Gr
- Kalka C
- Kallat A
- Karcher J
- Karl-Wollweber S
- Keilhau Da
- Kerr P
- Kesteven P
- Kittel K
- Knittel M
- Knolinski T
- Kopplin U
- Kroschel U
- Kröger K
- Kubicek–Hofmann C
- Köhler C
- Körner I
- La Forgia S
- La Regina M
- Lacoste A
- Lacut K
- Laeis P
- Landolfi R
- Laroche Jp
- Lecumberri R
- Lench T
- Leschke M
- Lester W
- Lewis M
- Lluís Padierna M
- Lodigiani C
- Lorenzo Hernández A
- Lowe G
- Lutz A
- López Castellanos Gt
- López Jiménez L
- López Gt
- López-Sáez Jb
- Lübbert G
- Madridano Cobo O
- Mainez Saiz C
- Mancuso G
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- Zanatta N
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- Zingaretti O
- Zorzi F
- zur Nieden T
- Publication venue
- 'Georg Thieme Verlag KG'
- Publication date
- 01/01/2017
- Field of study
Venous thromboembolism (VTE) is a significant cause of morbidity and mortality in Europe. Data from real-world registries are necessary, as clinical trials do not represent the full spectrum of VTE patients seen in clinical practice. We aimed to document the epidemiology, management and outcomes of VTE using data from a large, observational database. PREFER in VTE was an international, non-interventional disease registry conducted between January 2013 and July 2015 in primary and secondary care across seven European countries. Consecutive patients with acute VTE were documented and followed up over 12 months. PREFER in VTE included 3,455 patients with a mean age of 60.8 ± 17.0 years. Overall, 53.0 % were male. The majority of patients were assessed in the hospital setting as inpatients or outpatients (78.5 %). The diagnosis was deep-vein thrombosis (DVT) in 59.5 % and pulmonary embolism (PE) in 40.5 %. The most common comorbidities were the various types of cardiovascular disease (excluding hypertension; 45.5 %), hypertension (42.3 %) and dyslipidaemia (21.1 %). Following the index VTE, a large proportion of patients received initial therapy with heparin (73.2 %), almost half received a vitamin K antagonist (48.7 %) and nearly a quarter received a DOAC (24.5 %). Almost a quarter of all presentations were for recurrent VTE, with >80 % of previous episodes having occurred more than 12 months prior to baseline. In conclusion, PREFER in VTE has provided contemporary insights into VTE patients and their real-world management, including their baseline characteristics, risk factors, disease history, symptoms and signs, initial therapy and outcomes
Search for supersymmetry in events with four or more leptons in √s =13 TeV pp collisions with ATLAS
- Author
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- Zhukov K
- Zibell A
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- Zimine NI
- Zimmermann C
- Zimmermann S
- Zinonos Z
- Zinser M
- Ziolkowski M
- Zobernig G
- Zoccoli A
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- zur Nedden M
- Zwalinski L
- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2018
- Field of study
Results from a search for supersymmetry in events with four or more charged leptons (electrons, muons and taus) are presented. The analysis uses a data sample corresponding to 36.1 fb −1 of proton-proton collisions delivered by the Large Hadron Collider at s √ =13 TeV and recorded by the ATLAS detector. Four-lepton signal regions with up to two hadronically decaying taus are designed to target a range of supersymmetric scenarios that can be either enriched in or depleted of events involving the production and decay of a Z boson. Data yields are consistent with Standard Model expectations and results are used to set upper limits on the event yields from processes beyond the Standard Model. Exclusion limits are set at the 95% confidence level in simplified models of General Gauge Mediated supersymmetry, where higgsino masses are excluded up to 295 GeV. In R -parity-violating simplified models with decays of the lightest supersymmetric particle to charged leptons, lower limits of 1.46 TeV, 1.06 TeV, and 2.25 TeV are placed on wino, slepton and gluino masses, respectively
Combined measurement of differential and total cross sections in the H → γγ and the H → ZZ* → 4ℓ decay channels at s=13 TeV with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abhayasinghe DK
- Abidi SH
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- Zhao Z
- Zhemchugov A
- Zhou B
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- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinonos Z
- Zinser M
- Ziolkowski M
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zur Nedden M
- Zwalinski L
- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- Elsevier
- Publication date
- 01/01/2018
- Field of study
A combined measurement of differential and inclusive total cross sections of Higgs boson production is performed using 36.1 fb−1 of 13 TeV proton–proton collision data produced by the LHC and recorded by the ATLAS detector in 2015 and 2016. Cross sections are obtained from measured H→γγ and H→ZZ*(→4ℓ event yields, which are combined taking into account detector efficiencies, resolution, acceptances and branching fractions. The total Higgs boson production cross section is measured to be 57.0−5.9 +6.0 (stat.) −3.3 +4.0 (syst.) pb, in agreement with the Standard Model prediction. Differential cross-section measurements are presented for the Higgs boson transverse momentum distribution, Higgs boson rapidity, number of jets produced together with the Higgs boson, and the transverse momentum of the leading jet. The results from the two decay channels are found to be compatible, and their combination agrees with the Standard Model predictions
Measurement of the t¯tZ and t¯tW cross sections in proton-proton collisions at √s=13 TeV with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abbott DC
- Abdinov O
- Abeloos B
- Abhayasinghe DK
- Abidi SH
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- Zhemchugov A
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- Zimmermann S
- Zinonos Z
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- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
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- Zur Nedden M
- Zwalinski L
- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2019
- Field of study
A measurement of the associated production of a top-quark pair (t¯t) with a vector boson (W, Z) in proton-proton collisions at a center-of-mass energy of 13 TeV is presented, using 36.1 fb−1 of integrated luminosity collected by the ATLAS detector at the Large Hadron Collider. Events are selected in channels with two same- or opposite-sign leptons (electrons or muons), three leptons or four leptons, and each channel is further divided into multiple regions to maximize the sensitivity of the measurement. The t¯tZ and t¯tW production cross sections are simultaneously measured using a combined fit to all regions. The best-fit values of the production cross sections are σt¯tZ=0.95±0.08stat±0.10syst pb and σt¯tW=0.87±0.13stat±0.14syst pb in agreement with the Standard Model predictions. The measurement of the t¯tZ cross section is used to set constraints on effective field theory operators which modify the t¯tZ vertex
Recommended from our members
Search for Dark Matter Produced in Association with a Higgs Boson Decaying to bb[over ¯] Using 36 fb^{-1} of pp Collisions at sqrt[s]=13 TeV with the ATLAS Detector.
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abidi SH
- AbouZeid OS
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- Acharya BS
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- Zur Nedden M
- Zwalinski L
- Álvarez Piqueras D
- Åkesson TPA
- Ženiš T
- Živković L
- Publication venue
- Phys Rev Lett
- Publication date
- 03/11/2017
- Field of study
Several extensions of the standard model predict associated production of dark-matter particles with a Higgs boson. Such processes are searched for in final states with missing transverse momentum and a Higgs boson decaying to a bb[over ¯] pair with the ATLAS detector using 36.1 fb^{-1} of pp collisions at a center-of-mass energy of 13 TeV at the LHC. The observed data are in agreement with the standard model predictions and limits are placed on the associated production of dark-matter particles and a Higgs boson
Measurement of the inclusive isolated-photon cross section at √s = 13 TeV using fb⁻¹ of ATLAS data
- Author
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- Abbott B
- Abbott DC
- Abdinov O
- Abed Abud A
- Abeling K
- Abhayasinghe DK
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/10/2019
- Field of study
The differential cross section for isolated-photon production in pp collisions is measured at a centre-of-mass energy of 13 TeV with the ATLAS detector at the LHC using an integrated luminosity of 36.1 fb−1. The differential cross section is presented as a function of the photon transverse energy in different regions of photon pseudorapidity. The differential cross section as a function of the absolute value of the photon pseudorapidity is also presented in different regions of photon transverse energy. Next-to-leading-order QCD calculations from Jetphox and Sherpa as well as next-to-next-to-leading-order QCD calculations from Nnlojet are compared with the measurement, using several parameterisations of the proton parton distribution functions. The predictions provide a good description of the data within the experimental and theoretical uncertainties
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