27 research outputs found
Endothelial nitric oxide synthase gene polymorphism (Glu298Asp) and development of pre-eclampsia: a case-control study and a meta-analysis
- Author
- A Persu
- AD Hingorani
- AD Hingorani
- AM Lachmeijer
- Aroon D Hingorani
- AT Papageorghiou
- CB Tempfer
- Christina KH Yu
- CK Yu
- DJ Williams
- G Guo
- G Kobashi
- G Pridjian
- GC Smith
- H Salonen Ros
- H Watanabe
- HM Colhoun
- IP Kosmas
- JA Lade
- JP Casas
- JP Casas
- Juan P Casas
- JW Choi
- K Ohta
- Kypros H Nicolaides
- L Morgan
- M Egger
- M Tesauro
- MA Brown
- Makrina D Savvidou
- Manpreet K Sahemey
- MD Savvidou
- MD Savvidou
- MJ Khoury
- NC Serrano
- R DerSimonian
- R Hillermann
- R Landau
- S Krotz
- SC Robson
- SP Seligman
- T Hakli
- T Yoshimura
- T Yoshimura
- TA Fairchild
- WR Shadish
- Publication venue
- BioMed Central
- Publication date
- 01/01/2006
- Field of study
BACKGROUND: Pre-eclampsia is thought to have an important genetic component. Recently, pre-eclampsia has been associated in some studies with carriage of a common eNOS gene Glu298Asp polymorphism, a variant that leads to the replacement of glutamic acid by aspartic acid at codon 298. METHOD: Healthy women with singleton pregnancies were recruited from 7 district general hospitals in London, UK. Women at high risk of pre-eclampsia were screened by uterine artery Doppler velocimetry at 22–24 weeks of gestation and maternal blood was obtained to genotype the eNOS Glu298Asp polymorphism. Odds ratios (OR) and 95%CI, using logistic regression methods, were obtained to evaluate the association between the Glu298Asp polymorphism and pre-eclampsia. A meta-analysis was then undertaken of all published studies up to November 2005 examining the association of eNOS Glu298Asp genotype and pre-eclampsia. RESULTS: 89 women with pre-eclampsia and 349 controls were included in the new study. The Glu298Asp polymorphism in a recessive model was not significantly associated with pre-eclampsia (adjusted-OR: 0.83 [95%CI: 0.30–2.25]; p = 0.7). In the meta-analysis, under a recessive genetic model (1129 cases & 2384 controls) women homozygous for the Asp298 allele were not at significantly increased risk of pre-eclampsia (OR: 1.28 [95%CI: 0.76–2.16]; p = 0.34). A dominant model (1334 cases & 2894 controls) was associated with no increase of risk of pre-eclampsia for women carriers of the Asp298 allele (OR: 1.12 [95%CI: 0.84–1.49]; p = 0.42). CONCLUSION: From the data currently available, the eNOS Glu298Asp polymorphism is not associated with a significant increased risk of pre-eclampsia. However, published studies have been underpowered, much larger studies are needed to confirm or refute a realistic genotypic risk of disease, but which might contribute to many cases of pre-eclampsia in the population
Extended-Spectrum-Beta-Lactamases, AmpC Beta-Lactamases and Plasmid Mediated Quinolone Resistance in Klebsiella spp. from Companion Animals in Italy
- Author
- A Briales
- A Carattoli
- A Carattoli
- A Coelho
- A García-Fernández
- A García-Fernández
- Alessia Franco
- AM Hujer
- Antonio Battisti
- AT Vo
- Aurora García-Fernández
- Axel Cloeckaert
- B Périchon
- C Fontana
- C Fontana
- C Mammina
- C Mammina
- CH Park
- Christina Aaby Svendsen
- D Boccia
- D Jonas
- DE Roberts
- DL Paterson
- E Miró
- E Voulgari
- Fabiola Feltrin
- FJ Pérez-Pérez
- G Arlet
- Gessica Cordaro
- J Ma
- J Yang
- JB Nielsen
- KH Roh
- L Diancourt
- L Hidalgo
- L Poirel
- L Villa
- LM Cavaco
- LM Cavaco
- M Haenni
- M Lavollay
- M Österblad
- P Roy Chowdhury
- R Durmaz
- R Migliavacca
- R Podschun
- Raniero Lorenzetti
- Rene S. Hendriksen
- S Lavilla
- SA Jemima
- Serena Lorenzetti
- T Frank
- T Ode
- V Cattoir
- Valentina Donati
- VL Yu
- WC Ko
- Y Pfeifer
- Y Qi
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2014
- Field of study
We report the genetic characterization of 15 Klebsiella pneumoniae (KP) and 4 isolates of K. oxytoca (KO) from clinical cases in dogs and cats and showing extended-spectrum cephalosporin (ESC) resistance. Extended spectrum beta-lactamase (ESBL) and AmpC genes, plasmid-mediated quinolone resistance (PMQR) and co-resistances were investigated. Among KP isolates, ST101 clone was predominant (8/15, 53%), followed by ST15 (4/15, 27%). ST11 and ST340, belonging to Clonal Complex (CC)11, were detected in 2012 (3/15, 20%). MLST on KP isolates corresponded well with PFGE results, with 11 different PFGE patterns observed, including two clusters of two (ST340) and four (ST101) indistinguishable isolates, respectively. All isolates harbored at least one ESBL or AmpC gene, all carried on transferable plasmids (IncR, IncFII, IncI1, IncN), and 16/19 were positive for PMQR genes (qnr family or aac(6')-Ib-cr). The most frequent ESBL was CTX-M-15 (11/19, 58%), detected in all KP ST101, in one KP ST15 and in both KP ST340. blaCTX-M-15 was carried on IncR plasmids in all but one KP isolate. All KP ST15 isolates harbored different ESC resistance genes and different plasmids, and presented the non-transferable blaSHV-28 gene, in association with blaCTX-M-15, blaCTX-M-1 (on IncR, or on IncN), blaSHV-2a (on IncR) or blaCMY-2 genes (on IncI1). KO isolates were positive for blaCTX-M-9 gene (on IncHI2), or for the blaSHV-12 and blaDHA-1 genes (on IncL/M). They were all positive for qnr genes, and one also for the aac(6')-Ib-cr gene. All Klebsiella isolates showed multiresistance towards aminoglycosides, sulfonamides, tetracyclines, trimethoprim and amphenicols, mediated by strA/B, aadA2, aadB, ant (2")-Ia, aac(6')-Ib, sul, tet, dfr and cat genes in various combinations. The emergence in pets of multidrug-resistant Klebsiella with ESBL, AmpC and PMQR determinants, poses further and serious challenges in companion animal therapy and raise concerns for possible bi-directional transmission between pets and humans, especially at household level
Secretion of Genome-Free Hepatitis B Virus – Single Strand Blocking Model for Virion Morphogenesis of Para-retrovirus
- Author
- A Alberti
- A Gallina
- A Machida
- AJ Thompson
- AM Roseman
- C Seeger
- C Seeger
- Christina Adams
- CM Hilditch
- D Perlman
- DA Fallows
- David Nguyen
- DH Nguyen
- DH Nguyen
- DH Perlman
- EF Pettersen
- F Birnbaum
- G Sommer
- H Delius
- H Guo
- H Guo
- Hyunwook Lee
- J Hu
- J Hu
- J Hu
- J Hu
- J Summers
- J Summers
- Jianming Hu
- Jing-hsiung James Ou
- JL Gerin
- JM Lien
- JZ Porterfield
- KH Heermann
- Laura Mentzer
- M Deguchi
- M Kann
- M Nassal
- M Newman
- M Newman
- M Yu
- MA Petit
- O Rosmorduc
- P Wingfield
- PK Chua
- PM Kaplan
- RE Lanford
- RE Lanford
- RH Miller
- RJ Lenhoff
- Robert Ashley
- S Asabe
- S Le Pogam
- S Liu
- S Terre
- SF Chang
- SH Basagoudanavar
- SK Ladner
- Susan Hafenstein
- T Gerelsaikhan
- T Hatton
- T Kimura
- T Volz
- T Watanabe
- TT Yuan
- TT Yuan
- W Gao
- W Schormann
- X Yan
- Xiaojun Ning
- Y Sakamoto
- Y Wei
- YL Chiu
- Z Zhang
- Publication venue
- Public Library of Science
- Publication date
- 01/09/2011
- Field of study
As a para-retrovirus, hepatitis B virus (HBV) is an enveloped virus with a double-stranded (DS) DNA genome that is replicated by reverse transcription of an RNA intermediate, the pregenomic RNA or pgRNA. HBV assembly begins with the formation of an “immature” nucleocapsid (NC) incorporating pgRNA, which is converted via reverse transcription within the maturing NC to the DS DNA genome. Only the mature, DS DNA-containing NCs are enveloped and secreted as virions whereas immature NCs containing RNA or single-stranded (SS) DNA are not enveloped. The current model for selective virion morphogenesis postulates that accumulation of DS DNA within the NC induces a “maturation signal” that, in turn, triggers its envelopment and secretion. However, we have found, by careful quantification of viral DNA and NCs in HBV virions secreted in vitro and in vivo, that the vast majority of HBV virions (over 90%) contained no DNA at all, indicating that NCs with no genome were enveloped and secreted as empty virions (i.e., enveloped NCs with no DNA). Furthermore, viral mutants bearing mutations precluding any DNA synthesis secreted exclusively empty virions. Thus, viral DNA synthesis is not required for HBV virion morphogenesis. On the other hand, NCs containing RNA or SS DNA were excluded from virion formation. The secretion of DS DNA-containing as well as empty virions on one hand, and the lack of secretion of virions containing single-stranded (SS) DNA or RNA on the other, prompted us to propose an alternative, “Single Strand Blocking” model to explain selective HBV morphogenesis whereby SS nucleic acid within the NC negatively regulates NC envelopment, which is relieved upon second strand DNA synthesis
Minimal information for studies of extracellular vesicles 2018 (MISEV2018):a position statement of the International Society for Extracellular Vesicles and update of the MISEV2014 guidelines
- Author
- Afd Pharmaceutics
- Afd Pharmacology
- Aikawa Elena
- Alcaraz Maria Jose
- Anderson Johnathon D
- Andriantsitohaina Ramaroson
- Antoniou Anna
- Arab Tanina
- Archer Fabienne
- Atkin-Smith Georgia K
- Ayre D Craig
- Bach Jean-Marie
- Bachurski Daniel
- Baharvand Hossein
- Balaj Leonora
- Baldacchino Shawn
- Bauer Natalie N
- Baxter Amy A
- Bebawy Mary
- Beckham Carla
- Bedina Zavec Apolonija
- Benmoussa Abderrahim
- Berardi Anna C
- Bergese Paolo
- Bielska Ewa
- Blenkiron Cherie
- Bobis-Wozowicz Sylwia
- Boilard Eric
- Boireau Wilfrid
- Bongiovanni Antonella
- Borràs Francesc E
- Bosch Steffi
- Boulanger Chantal M
- Breakefield Xandra
- Breglio Andrew M
- Brennan Meadhbh Á
- Brigstock David R
- Brisson Alain
- Broekman Marike Ld
- Bromberg Jacqueline F
- Bryl-Górecka Paulina
- Buch Shilpa
- Buck Amy H
- Burger Dylan
- Busatto Sara
- Buschmann Dominik
- Bussolati Benedetta
- Buzás Edit I
- Byrd James Bryan
- Camussi Giovanni
- Carter David Rf
- Caruso Sarah
- Celbiologie
- Chamley Lawrence W
- Chang Yu-Ting
- Chen Chihchen
- Chen Shuai
- Cheng Lesley
- Chin Andrew R
- Clayton Aled
- Clerici Stefano P
- Cocks Alex
- Cocucci Emanuele
- Coffey Robert J
- Cordeiro-da-Silva Anabela
- Couch Yvonne
- Coumans Frank Aw
- Coyle Beth
- Crescitelli Rossella
- Criado Miria Ferreira
- D'Souza-Schorey Crislyn
- Das Saumya
- Datta Chaudhuri Amrita
- dB&C I&I
- de Candia Paola
- De Santana Eliezer F
- De Wever Olivier
- Del Portillo Hernando A
- Demaret Tanguy
- Deville Sarah
- Devitt Andrew
- Dhondt Bert
- Di Vizio Dolores
- Dieterich Lothar C
- Dolo Vincenza
- Dominguez Rubio Ana Paula
- Dominici Massimo
- Dourado Mauricio R
- Driedonks Tom Ap
- Duarte Filipe V
- Duncan Heather M
- Eichenberger Ramon M
- Ekström Karin
- El Andaloussi Samir
- Elie-Caille Celine
- Erdbrügger Uta
- Falcón-Pérez Juan M
- Fatima Farah
- Fish Jason E
- Flores-Bellver Miguel
- Frelet-Barrand Annie
- Fricke Fabia
- Fuhrmann Gregor
- Försönits András
- Gabrielsson Susanne
- Gardiner Chris
- Gaudin Raphael
- Gho Yong Song
- Giebel Bernd
- Gilbert Caroline
- Gimona Mario
- Giusti Ilaria
- Goberdhan Deborah Ci
- Gorski Sharon M
- Greening David W
- Gross Julia Christina
- Gualerzi Alice
- Gupta Gopal N
- Gustafson Dakota
- Gámez-Valero Ana
- Gärtner Kathrin
- Görgens André
- Handberg Aase
- Haraszti Reka A
- Harrison Paul
- Hegyesi Hargita
- Hendrix An
- Hill Andrew F
- Hochberg Fred H
- Hoffmann Karl F
- Holder Beth
- Holthofer Harry
- Hosseinkhani Baharak
- Hu Guoku
- Huang Yiyao
- Huber Veronica
- Hunt Stuart
- Ibrahim Ahmed Gamal-Eldin
- Ikezu Tsuneya
- Inal Jameel M
- Isin Mustafa
- Ivanova Alena
- Jackson Hannah K
- Jacobsen Soren
- Jay Steven M
- Jayachandran Muthuvel
- Jenster Guido
- Jiang Lanzhou
- Johnson Suzanne M
- Jones Jennifer C
- Jong Ambrose
- Jovanovic-Talisman Tijana
- Jung Stephanie
- Kalluri Raghu
- Kano Shin-Ichi
- Kaur Sukhbir
- Kawamura Yumi
- Keller Evan T
- Khamari Delaram
- Khomyakova Elena
- Khvorova Anastasia
- Kierulf Peter
- Kim Kwang Pyo
- Kislinger Thomas
- Klingeborn Mikael
- Klinke David J
- Kornek Miroslaw
- Kosanović Maja M
- Kovács Árpád Ferenc
- Krasemann Susanne
- Krause Mirja
- Krämer-Albers Eva-Maria
- Kurochkin Igor V
- Kusuma Gina D
- Kuypers Sören
- Laitinen Saara
- Langevin Scott M
- Languino Lucia R
- Lannigan Joanne
- Laurent Louise C
- Lavieu Gregory
- Le Lay Soazig
- Lee Myung-Shin
- Lee Yi Xin Fiona
- Lemos Debora S
- Lenassi Metka
- Leszczynska Aleksandra
- Li Isaac Ts
- Liao Ke
- Libregts Sten F
- Ligeti Erzsebet
- Lim Rebecca
- Lim Sai Kiang
- Linnemannstöns Karen
- Linē Aija
- Llorente Alicia
- Lombard Catherine A
- Lorenowicz Magdalena J
- Lovett Jason
- Lowry Michelle C
- Loyer Xavier
- LS Celbiologie-Algemeen
- Lu Quan
- Lukomska Barbara
- Lunavat Taral R
- Lázaro-Ibáñez Elisa
- Lässer Cecilia
- Lörincz Ákos M
- Lötvall Jan
- Maas Sybren Ln
- Malhi Harmeet
- Marcilla Antonio
- Mariani Jacopo
- Mariscal Javier
- Martens-Uzunova Elena S
- Martin-Jaular Lorena
- Martinez M Carmen
- Martins Vilma Regina
- Mathieu Mathilde
- Mathivanan Suresh
- Maugeri Marco
- McGinnis Lynda K
- McVey Mark J
- Meckes David G
- Meehan Katie L
- Mertens Inge
- Minciacchi Valentina R
- Morales-Kastresana Aizea
- Morhayim Jess
- Mullier François
- Muraca Maurizio
- Musante Luca
- Mussack Veronika
- Muth Dillon C
- Myburgh Kathryn H
- Möller Andreas
- Møller Jørgensen Malene
- Najrana Tanbir
- Nawaz Muhammad
- Nazarenko Irina
- Nejsum Peter
- Neri Christian
- Neri Tommaso
- Nieuwland Rienk
- Nimrichter Leonardo
- Nolan John P
- Nolte-'t Hoen Esther NM
- Noren Hooten Nicole
- O'Driscoll Lorraine
- O'Grady Tina
- O'Loghlen Ana
- Ochiya Takahiro
- Olivier Martin
- Ortiz Alberto
- Ortiz Luis A
- Osteikoetxea Xabier
- Ostrowski Matias
- Park Jaesung
- Pegtel D Michiel
- Peinado Hector
- Perut Francesca
- Pfaffl Michael W
- Phinney Donald G
- Pieters Bartijn Ch
- Pink Ryan C
- Pisetsky David S
- Pogge von Strandmann Elke
- Polakovicova Iva
- Poon Ivan Kh
- Powell Bonita H
- Prada Ilaria
- Pulliam Lynn
- Quesenberry Peter
- Radeghieri Annalisa
- Raffai Robert L
- Raimondo Stefania
- Rak Janusz
- Ramirez Marcel I
- Raposo Graça
- Rayyan Morsi S
- Regev-Rudzki Neta
- Ricklefs Franz L
- Robbins Paul D
- Roberts David D
- Rodrigues Silvia C
- Rohde Eva
- Rome Sophie
- Rouschop Kasper Ma
- Rughetti Aurelia
- Russell Ashley E
- Sahoo Susmita
- Salas-Huenuleo Edison
- Saugstad Julie A
- Saul Meike J
- Saá Paula
- Schiffelers Raymond M
- Schneider Raphael
- Schøyen Tine Hiorth
- Scott Aaron
- Shahaj Eriomina
- Sharma Shivani
- Shatnyeva Olga
- Shekari Faezeh
- Shelke Ganesh Vilas
- Shetty Ashok K
- Shiba Kiyotaka
- Siljander Pia R-M
- Silva Andreia M
- Skowronek Agata
- Snyder Orman L
- Soares Rodrigo Pedro
- Soekmadji Carolina
- Sotillo Javier
- Stahl Philip D
- Stoorvogel Willem
- Stott Shannon L
- Strasser Erwin F
- Sub Biomol.Mass Spect. and Proteomics
- Sub General Pharmaceutics
- Swift Simon
- Sánchez Catherine
- Sódar Barbara W
- Tahara Hidetoshi
- Tewari Muneesh
- Théry Clotilde
- Timms Kate
- Tiwari Swasti
- Tixeira Rochelle
- Tkach Mercedes
- Toh Wei Seong
- Tomasini Richard
- Torrecilhas Ana Claudia
- Tosar Juan Pablo
- Toxavidis Vasilis
- Urbanelli Lorena
- Vader Pieter
- van Balkom Bas Wm
- van der Grein Susanne G
- Van Deun Jan
- van Herwijnen Martijn Jc
- Van Keuren-Jensen Kendall
- van Niel Guillaume
- van Royen Martin E
- van Wijnen Andre J
- Vasconcelos M Helena
- Vechetti Ivan J
- Veit Tiago D
- Vella Laura J
- Velot Émilie
- Verweij Frederik J
- Vestad Beate
- Visnovitz Tamás
- Viñas Jose L
- Vukman Krisztina V
- Wahlgren Jessica
- Watson Dionysios C
- Wauben Marca Hm
- Weaver Alissa
- Webber Jason P
- Weber Viktoria
- Wehman Ann M
- Weiss Daniel J
- Welsh Joshua A
- Wendt Sebastian
- Wheelock Asa M
- Wiener Zoltán
- Witte Leonie
- Witwer Kenneth W
- Wolfram Joy
- Xagorari Angeliki
- Xander Patricia
- Xu Jing
- Yan Xiaomei
- Yin Hang
- Yuana Yuana
- Yáñez-Mó María
- Zappulli Valentina
- Zarubova Jana
- Zhang Jian-Ye
- Zhao Zezhou
- Zheng Lei
- Zheutlin Alexander R
- Zickler Antje M
- Zimmermann Pascale
- Zivkovic Angela M
- Zocco Davide
- Zuba-Surma Ewa K
- Østergaard Ole
- Žėkas Vytautas
- Publication venue
- Publication date
- 01/01/2018
- Field of study
The last decade has seen a sharp increase in the number of scientific publications describing physiological and pathological functions of extracellular vesicles (EVs), a collective term covering various subtypes of cell-released, membranous structures, called exosomes, microvesicles, microparticles, ectosomes, oncosomes, apoptotic bodies, and many other names. However, specific issues arise when working with these entities, whose size and amount often make them difficult to obtain as relatively pure preparations, and to characterize properly. The International Society for Extracellular Vesicles (ISEV) proposed Minimal Information for Studies of Extracellular Vesicles (“MISEV”) guidelines for the field in 2014. We now update these “MISEV2014” guidelines based on evolution of the collective knowledge in the last four years. An important point to consider is that ascribing a specific function to EVs in general, or to subtypes of EVs, requires reporting of specific information beyond mere description of function in a crude, potentially contaminated, and heterogeneous preparation. For example, claims that exosomes are endowed with exquisite and specific activities remain difficult to support experimentally, given our still limited knowledge of their specific molecular machineries of biogenesis and release, as compared with other biophysically similar EVs. The MISEV2018 guidelines include tables and outlines of suggested protocols and steps to follow to document specific EV-associated functional activities. Finally, a checklist is provided with summaries of key points
A Genome-Wide Association Study of Diabetic Kidney Disease in Subjects With Type 2 Diabetes
- Author
- Abdalla Moustafa
- Adams F
- Adelman D
- Agardh E
- Ahlqvist E
- Ahlqvist Emma
- Ahluwalia Tarunveer S
- Ahola AJ
- Ahonen R
- Ahonen-Bishopp A
- Aiello L
- Aimolahti A
- Airas MA
- Aizawa K
- Ajanki T
- Akbar T
- Al Maghrabi N
- Albertini M
- Almgren P
- Almgren Peter
- Anderson S
- Ang Su Fen
- Apelqvist J
- Aranko S
- Arends V
- Asplund B
- Babu M
- Bain SC
- Ball C
- Bantle J
- Barbarini N
- Barnie A
- Bassi L
- Belch J
- Bellazzi R
- Bellenger N
- Bengtsson E
- Benigni A
- Bergenstal R
- Berglund L
- Bernal ML
- Betsholtz C
- Bhan A
- Björckbacka H
- Björkholm A
- Blodi B
- Blom-Nilsson U
- Bokvist K
- Boldrini B
- Boni E
- Borell M
- Boulton AJM
- Bourne P
- Boustany C
- Braffett B
- Brandslund Ivan
- Brennan EP
- Brosnan J
- Brosnan Mary Julia
- Bucalo M
- Burström A
- Byskata U
- Campbell C
- Canady J
- Casanova F
- Caulder S
- Chan JCN
- Chan KKH
- Chan TF
- Chan Juliana C N
- Chassin L
- Cheung GPY
- Christensen Cramer
- Cilio C
- Cimino L
- Cinthio M
- Clara Tan Si Hua
- Cleary P
- Clerin V
- Cobelli C
- Colhoun H
- Colhoun Helen M
- Colombo M
- Colson S
- Cowie C
- Crowther D
- Dagogo-Jack S
- Dahlström E
- Dallai A
- Dalton N
- Danis R
- Deanfield J
- Demaine AG
- Deshmukh H
- Deshmukh Harshal
- Di Camillo B
- Dijkstra M
- Donadelli R
- Dorajoo Rajkumar
- Dove F
- Dreja K
- Driscoll M
- Duffy Seamus
- Dunger D
- Dunger David
- Dunér P
- Ehlgen A
- Eliasson A
- Ellingford A
- Engelbertsen D
- Erola RM
- Ervasti S
- Fadista J
- Fagerholm E
- Fagerudd J
- Fahlstrom R
- Fan X
- Farran B
- Fauman E
- Fauman Eric
- Feodoroff M
- Ferguson M
- Ferrannini E
- Finotello F
- Flaherty N
- Florez JC
- Florez Jose C
- Formentini I
- Foroogh F
- Forsblom C
- Forsblom C
- Forsblom Carol
- Forsen M
- Forslund T
- Frayling T
- Gan LM
- Gassenhuber J
- Gatcomb P
- Gates P
- Genové G
- Gerl M
- Gertow K
- Gieger C
- Gigante B
- Godson C
- Golden E
- Goldstein D
- Gomez M
- Goncalves I
- Gooding K
- Gordin D
- Gordin D
- Grallert H
- Granlund H
- Grarup Niels
- Gregory NS
- Groop L
- Groop PH
- Groop PH
- Groop Leif C
- Groop Per-Henrik
- Groves C
- Gubitosi-Klug R
- Guidi F
- Guiducci C
- Gunyou K
- Gurzeler E
- Gyorgy Beata
- Haaga J
- Hadjadj Samy
- Hainsworth D
- Halonen A
- Hammarstedt A
- Hamsten A
- Hansen Torben
- Harjutsalo V
- Harjutsalo V
- Harjutsalo Valma
- Harth J
- Hattersley A
- Haukkala J
- Havulinna A
- He B
- Hedblad B
- Heikkilä O
- Heikkilä O
- Heinonen S
- Henderson G
- Hensley S
- Hentunen P
- Herman W
- Herrala T
- Hess S
- Hietala K
- Hietala K
- Hiraki
- Hirschhorn JN
- Hirschhorn Joel N
- Hitt S
- Horsford J
- Houston G
- Huang Y
- Huber J
- Hubschle T
- Hultgårdh A
- Huovinen E
- Huttunen E
- Huusko J
- Hyde C
- Hämäläinen A
- Häring H
- Igo Robert P
- Ikäheimo R
- Ilkka V
- Imamura Minako
- Isakova T
- Ivaska-Suomela M
- Jablonka B
- Jatkola E
- Jauhiainen A
- Jiang G
- Jiang Guozhi
- Johansson ME
- Johnson M
- Johnsonbaugh S
- Jokelainen KS
- Jones JK
- Jonsson AC
- Juliusdottir T
- Juretschke HP
- Jönsson-Rylander AC
- Kalliomäki FT
- Kananen K
- Kanninen T
- Kaprio SA
- Karhapää P
- Kari PH
- Karjalainen M
- Karpe F
- Kauppinen-Mäkelin R
- Keasler L
- Kekäläinen P
- Kemppainen P
- Kennbäck C
- Khan F
- Kholová I
- Kinnunen P
- Klemetti S
- Knuuti J
- Koistinen A
- Koivula S
- Korolainen M
- Koskelainen J
- Koskiaho P
- Koskinen E
- Kozakova M
- Kravic J
- Kristiansson K
- Krolewski Andrzej S
- Kruger D
- Kubo Michiaki
- Kuusela M
- Kuusisto A
- Kyllönen L
- Kytö J
- Kytö J
- Kärkkäinen J
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- Publication venue
- Publication date
- 01/01/2018
- Field of study
dentification of sequence variants robustly associated with predisposition to diabetic kidney disease (DKD) has the potential to provide insights into the pathophysiological mechanisms responsible. We conducted a genome-wide association study (GWAS) of DKD in type 2 diabetes (T2D) using eight complementary dichotomous and quantitative DKD phenotypes: the principal dichotomous analysis involved 5,717 T2D subjects, 3,345 with DKD. Promising association signals were evaluated in up to 26,827 subjects with T2D (12,710 with DKD). A combined T1D+T2D GWAS was performed using complementary data available for subjects with T1D, which, with replication samples, involved up to 40,340 subjects with diabetes (18,582 with DKD). Analysis of specific DKD phenotypes identified a novel signal near GABRR1 (rs9942471, P = 4.5 x 10(-8)) associated with microalbuminuria in European T2D case subjects. However, no replication of this signal was observed in Asian subjects with T2D or in the equivalent T1D analysis. There was only limited support, in this substantially enlarged analysis, for association at previously reported DKD signals, except for those at UMOD and PRKAG2, both associated with estimated glomerular filtration rate. We conclude that, despite challenges in addressing phenotypic heterogeneity, access to increased sample sizes will continue to provide more robust inference regarding risk variant discovery for DKD.Peer reviewe
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- Publication venue
- EUROPEAN PHYSICAL JOURNAL C
- Publication date
- 08/03/2022
- Field of study
The design and performance of the inner detector trigger for the high level
trigger of the ATLAS experiment at the Large Hadron Collider during the 2016-18
data taking period is discussed. In 2016, 2017, and 2018 the ATLAS detector
recorded 35.6 fb−1, 46.9 fb−1, and 60.6 fb−1 respectively of
proton-proton collision data at a centre-of-mass energy of 13 TeV. In order to
deal with the very high interaction multiplicities per bunch crossing expected
with the 13 TeV collisions the inner detector trigger was redesigned during the
long shutdown of the Large Hadron Collider from 2013 until 2015. An overview of
these developments is provided and the performance of the tracking in the
trigger for the muon, electron, tau and b-jet signatures is discussed. The
high performance of the inner detector trigger with these extreme interaction
multiplicities demonstrates how the inner detector tracking continues to lie at
the heart of the trigger performance and is essential in enabling the ATLAS
physics programme
The ATLAS inner detector trigger performance in pp collisions at 13 TeV during LHC Run 2
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
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- Acharya BS
- Achkar B
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- Adamek L
- Adelman J
- Adiguzel A
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- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
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- Agullo P Martinez
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- Alberghi GL
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- Yallup David
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- Zagazeta LF Gutierrez
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- Zeitnitz C
- Zemaityte G
- Zeng JC
- Zenin O
- Zenis T
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- Zerradi S
- Zerwas D
- Zgubic M
- Zhang B
- Zhang DF
- Zhang G
- Zhang J
- Zhang K
- Zhang L
- Zhang L
- Zhang M
- Zhang R
- Zhang S
- Zhang X
- Zhang X
- Zhang Y
- Zhang Z
- Zhao P
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
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- Zhou H
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- Zhu C
- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zivkovic L
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zou W
- Zwalinski L
- Publication venue
- EUROPEAN PHYSICAL JOURNAL C
- Publication date
- 01/01/2022
- Field of study
The design and performance of the inner detector trigger for the high level
trigger of the ATLAS experiment at the Large Hadron Collider during the 2016-18
data taking period is discussed. In 2016, 2017, and 2018 the ATLAS detector
recorded 35.6 fb−1, 46.9 fb−1, and 60.6 fb−1 respectively of
proton-proton collision data at a centre-of-mass energy of 13 TeV. In order to
deal with the very high interaction multiplicities per bunch crossing expected
with the 13 TeV collisions the inner detector trigger was redesigned during the
long shutdown of the Large Hadron Collider from 2013 until 2015. An overview of
these developments is provided and the performance of the tracking in the
trigger for the muon, electron, tau and b-jet signatures is discussed. The
high performance of the inner detector trigger with these extreme interaction
multiplicities demonstrates how the inner detector tracking continues to lie at
the heart of the trigger performance and is essential in enabling the ATLAS
physics programme
Recommended from our members
Observation of electroweak production of two jets in association with an isolated photon and missing transverse momentum, and search for a Higgs boson decaying into invisible particles at 13 TeV with the ATLAS detector
- Author
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- Abbott B
- Abbott DC
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- Abhayasinghe DK
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- Zhemchugov A
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- Zhulanov V
- Zieminska D
- Zimine NI
- Zimmermann S
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- Zorbas TG
- Zormpa O
- Zou W
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- Publication venue
- EUROPEAN PHYSICAL JOURNAL C
- Publication date
- 03/02/2022
- Field of study
This paper presents the measurement of the electroweak production of two jets
in association with a Zγ pair with the Z boson decaying into two
neutrinos. It also presents the search for invisible or partially invisible
decays of a Higgs boson with a mass of 125 GeV produced through vector-boson
fusion with a photon in the final state. These results use data from LHC
proton-proton collisions at s = 13 TeV collected with the ATLAS
detector corresponding to an integrated luminosity of 139 fb−1. The event
signature, shared by all benchmark processes considered for measurements and
searches, is characterized by a significant amount of unbalanced transverse
momentum and a photon in the final state, in addition to a pair of forward
jets. For electroweak production of Zγ in association with two jets, the
background-only hypothesis is rejected with an observed (expected) significance
of 5.2 (5.1) standard deviations. The measured fiducial cross-section for this
process is 1.31±0.29 fb. Observed (expected) upper limit of 0.37 (0.34) at
95% confidence level is set on the branching ratio of a 125 GeV Higgs boson to
invisible particles, assuming the Standard Model production cross-section. The
signature is also interpreted in the context of decays of a Higgs boson to a
photon and a dark photon. An observed (expected) 95% CL upper limit on the
branching ratio for this decay is set at 0.018 (0.017), assuming the 125 GeV
Standard Model Higgs boson production cross-section
Second-Trimester Uterine Artery Doppler in the Prediction of Stillbirths
- Author
- Akolekar R
- Alfirevic Z
- Argyro Syngelaki
- Berghella V
- Brosens I
- Brunella Muto
- Bujold E
- Celik E
- Christina K.H. Yu
- De Wolf F
- Dommisse J
- Flenady V
- Fonseca EB
- Frøen JF
- Harrington K
- Hassan SS
- Karagiannis G
- Khong TY
- Khong TY
- Kypros H. Nicolaides
- Leona C.Y. Poon
- Meekins JW
- Moldenhauer JS
- Nicola Volpe
- Nicolaides KH
- Nicolaides KH
- Papageorghiou AT
- Pijnenborg R
- Pijnenborg R
- Roberge S
- Robinson HP
- Romero R
- Sebire NJ
- Singh T
- Smith GC
- Smith GC
- Stephansson O
- Syngelaki A
- Tikkanen M
- Willinger M
- Yu CK
- Publication venue
- 'S. Karger AG'
- Publication date
- 01/01/2013
- Field of study