Permitido o excluido: la arqueología científica y el mantenimiento de los mitos de legitimación

‘Scientific’ archaeology advocated by mainstream American archaeologists since the 1960s has tended to narrow ‘science’ to what Kuhn termed normal science, that is, research constrained by a ruling paradigm. This paradigm is based on the Myth of Columbus legitimating European invasions, conquests, and dispossessions of American nations by asserting that until October 1492, the Americas were a wilderness inhabited by savages. European international law held that Christians had the right to invade non-Christian nations and convert them by force if necessary. American schools teach that American Indians lacked the arts of civilization, and this early socialization persists in archaeologists’ models of pre-contact American nations. The paper looks at the “Core system” of the discipline, a recent interest in ‘historicizing’ the pre-contact American past, and the issue of transoceanic contacts before Columbus.La arqueología ‘científica’ defendida por los principales arqueólogos americanos desde los años sesenta ha tendido a limitar su definición de ciencia a lo que Kuhn denomina ‘ciencia normal’, es decir, a la investigación realizada bajo el paradigma dominante. Dicho paradigma está basado en ‘el mito de Colón’, un mito que legitima las invasiones europeas y el expolio de las naciones americanas asegurando que, hasta 1492, América era un territorio inhóspito habitado por salvajes. Dicho mito tiene su origen en la legislación europea internacional que establecía que los cristianos tenían el derecho de invadir las naciones no cristianas y convertirlas por la fuerza. Las escuelas americanas han contribuido a perpetuar el ‘mito de Colón’ al insistir en que los ‘indios’ americanos carecían de civilización. Dicha mito ha persistido también en los modelos desarrollados por los arqueólogos a propósito del periodo de pre-contacto con las naciones americanas. Este artículo examina las convicciones fundamentales de la disciplina, el reciente interés por historizar la fase del ‘pre-contacto’ y la cuestión de los contactos transoceánicos antes de Colón

Arsenic and Old Pelts: An Update on Deadly Pesticides in Museum Collections

The use of toxic chemicals within museums is an issue only recently addressed by anthropologists and scholars in related fields. A case of arsenic poisoning in an anthropologist during the 1960s is reviewed for what it may tell us about a mysterious ailment that afflicted Clark Wissler 50 years earlier. While no conclusive diagnosis can be made, Wissler’s case reminds us that we have come a long way in protecting against one of the lesser known dangers confronting anthropologists. All museums use pesticides and preservatives of some form, though the health impact of these agents is not always known. This necessary evil in the preservation of ethnographic collections can thus pose a health risk to people who work with or come in contact with treated objects. Here we open one cold case file, in which we believe a prominent American anthropologist may have directly suffered from the effects of poisons commonly used in the early nineteenth-century. Our own experience and recent inquiries provide one possible answer to the cause of the illness suffered by Clark Wissler

Measurement of χ c1 and χ c2 production with s√ = 7 TeV pp collisions at ATLAS

The prompt and non-prompt production cross-sections for the χ c1 and χ c2 charmonium states are measured in pp collisions at s√ = 7 TeV with the ATLAS detector at the LHC using 4.5 fb−1 of integrated luminosity. The χ c states are reconstructed through the radiative decay χ c → J/ψγ (with J/ψ → μ + μ −) where photons are reconstructed from γ → e + e − conversions. The production rate of the χ c2 state relative to the χ c1 state is measured for prompt and non-prompt χ c as a function of J/ψ transverse momentum. The prompt χ c cross-sections are combined with existing measurements of prompt J/ψ production to derive the fraction of prompt J/ψ produced in feed-down from χ c decays. The fractions of χ c1 and χ c2 produced in b-hadron decays are also measured

Case Reports1. A Late Presentation of Loeys-Dietz Syndrome: Beware of TGFβ Receptor Mutations in Benign Joint Hypermobility

Background: Thoracic aortic aneurysms (TAA) and dissections are not uncommon causes of sudden death in young adults. Loeys-Dietz syndrome (LDS) is a rare, recently described, autosomal dominant, connective tissue disease characterized by aggressive arterial aneurysms, resulting from mutations in the transforming growth factor beta (TGFβ) receptor genes TGFBR1 and TGFBR2. Mean age at death is 26.1 years, most often due to aortic dissection. We report an unusually late presentation of LDS, diagnosed following elective surgery in a female with a long history of joint hypermobility. Methods: A 51-year-old Caucasian lady complained of chest pain and headache following a dural leak from spinal anaesthesia for an elective ankle arthroscopy. CT scan and echocardiography demonstrated a dilated aortic root and significant aortic regurgitation. MRA demonstrated aortic tortuosity, an infrarenal aortic aneurysm and aneurysms in the left renal and right internal mammary arteries. She underwent aortic root repair and aortic valve replacement. She had a background of long-standing joint pains secondary to hypermobility, easy bruising, unusual fracture susceptibility and mild bronchiectasis. She had one healthy child age 32, after which she suffered a uterine prolapse. Examination revealed mild Marfanoid features. Uvula, skin and ophthalmological examination was normal. Results: Fibrillin-1 testing for Marfan syndrome (MFS) was negative. Detection of a c.1270G > C (p.Gly424Arg) TGFBR2 mutation confirmed the diagnosis of LDS. Losartan was started for vascular protection. Conclusions: LDS is a severe inherited vasculopathy that usually presents in childhood. It is characterized by aortic root dilatation and ascending aneurysms. There is a higher risk of aortic dissection compared with MFS. Clinical features overlap with MFS and Ehlers Danlos syndrome Type IV, but differentiating dysmorphogenic features include ocular hypertelorism, bifid uvula and cleft palate. Echocardiography and MRA or CT scanning from head to pelvis is recommended to establish the extent of vascular involvement. Management involves early surgical intervention, including early valve-sparing aortic root replacement, genetic counselling and close monitoring in pregnancy. Despite being caused by loss of function mutations in either TGFβ receptor, paradoxical activation of TGFβ signalling is seen, suggesting that TGFβ antagonism may confer disease modifying effects similar to those observed in MFS. TGFβ antagonism can be achieved with angiotensin antagonists, such as Losartan, which is able to delay aortic aneurysm development in preclinical models and in patients with MFS. Our case emphasizes the importance of timely recognition of vasculopathy syndromes in patients with hypermobility and the need for early surgical intervention. It also highlights their heterogeneity and the potential for late presentation. Disclosures: The authors have declared no conflicts of interes

Measurement of the inclusive jet cross-section in proton-proton collisions at √s=7 TeV using 4.5 fb−1 of data with the ATLAS detector

The inclusive jet cross-section is measured in proton-proton collisions at a centre-of-mass energy of 7 TeV using a data set corresponding to an integrated luminosity of 4.5 fb−1 collected with the ATLAS detector at the Large Hadron Collider in 2011. Jets are identified using the anti-kt algorithm with radius parameter values of 0.4 and 0.6. The double-differential cross-sections are presented as a function of the jet transverse momentum and the jet rapidity, covering jet transverse momenta from 100 GeV to 2 TeV. Next-to-leading-order QCD calculations corrected for non-perturbative effects and electroweak effects, as well as Monte Carlo simulations with next-to-leading-order matrix elements interfaced to parton showering, are compared to the measured cross-sections. A quantitative comparison of the measured cross-sections to the QCD calculations using several sets of parton distribution functions is performed