240 research outputs found
Effects of cyclooxygenase-1 and -2 gene disruption on Helicobacter pylori-induced gastric inflammation
- Author
- Publication venue
- 'University of Chicago Press'
- Publication date
- 01/01/2006
- Field of study
Background. Cyclooxygenases (COXs) play important roles in inflammation and carcinogenesis. The present study aimed to determine the effects of COX-1 and COX-2 gene disruption on Helicobacter pylori-induced gastric inflammation. Methods. Wild-type (WT), COX-1 and COX-2 heterozygous (COX-1 +/- and COX-2 +/-), and homozygous COX-deficient (COX-1 -/- and COX-2 -/-) mice were inoculated with H. pylori strain TN2 and killed after 24 weeks of infection. Uninfected WT and COX-deficient mice were used as controls. Levels of gastric mucosal inflammation, epithelial cell proliferation and apoptosis, and cytokine expression were determined. Results. COX deficiency facilitated H. pylori-induced gastritis. In the presence of H. pylori infection, apoptosis was increased in both WT and COX-deficient mice, whereas cell proliferation was increased in WT and COX-1-deficient, but not in COX-2-deficient, mice. Tumor necrosis factor (TNF)-α and interleukin-10 mRNA expression was elevated in H. pylori-infected mice, but only TNF-α mRNA expression was further increased by COX deficiency. Prostaglandin E 2 levels were increased in infected WT and COX-2-deficient mice but were at very low levels in infected COX-1-deficient mice. Leukotriene (LT) B 4 and LTC 4 levels were increased to a similar extent in infected WT and COX-deficient mice. Conclusions. COX deficiency enhances H. pylori-induced gastritis, probably via TNF-α expression. COX-2, but not COX-1, deficiency suppresses H. pylori-induced cell proliferation. © 2006 by the Infectious Diseases Society of America. All rights reserved.published_or_final_versio
Recombination dynamics of a human Y-chromosomal palindrome:rapid GC-biased gene conversion, multi-kilobase conversion tracts, and rare inversions
- Author
- A Geraldes
- A Scally
- AJ Jeffreys
- AJ Jeffreys
- B Trombetta
- C Batini
- E Bosch
- E Mancera
- FO Losch
- G Marais
- G Marais
- GA Marais
- Georgina R. Bowden
- H Guillon
- H Skaletsky
- HM Cann
- IL Berg
- J Lange
- J Pecon Slattery
- J Wang
- JA Armour
- JF Hughes
- JF Hughes
- JK Davis
- JM Chen
- JZ Li
- K Holloway
- KM Sullivan
- L Duret
- M Iwase
- M Iwase
- M Mendez-Lago
- Mark A. Jobling
- Mikkel H. Schierup
- N Backström
- N Bouzekri
- Patricia Balaresque
- PE Warburton
- Pille Hallast
- R Assis
- RD Bagnall
- S Repping
- S Rozen
- S Sarbajna
- S Sengupta
- SM Adams
- Stéphane Ballereau
- T Connallon
- TM Karafet
- W Shi
- W Wei
- Y Xue
- ZH Rosser
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2013
- Field of study
The male-specific region of the human Y chromosome (MSY) includes eight large inverted repeats (palindromes) in which arm-to-arm similarity exceeds 99.9%, due to gene conversion activity. Here, we studied one of these palindromes, P6, in order to illuminate the dynamics of the gene conversion process. We genotyped ten paralogous sequence variants (PSVs) within the arms of P6 in 378 Y chromosomes whose evolutionary relationships within the SNP-defined Y phylogeny are known. This allowed the identification of 146 historical gene conversion events involving individual PSVs, occurring at a rate of 2.9-8.4×10(-4) events per generation. A consideration of the nature of nucleotide change and the ancestral state of each PSV showed that the conversion process was significantly biased towards the fixation of G or C nucleotides (GC-biased), and also towards the ancestral state. Determination of haplotypes by long-PCR allowed likely co-conversion of PSVs to be identified, and suggested that conversion tract lengths are large, with a mean of 2068 bp, and a maximum in excess of 9 kb. Despite the frequent formation of recombination intermediates implied by the rapid observed gene conversion activity, resolution via crossover is rare: only three inversions within P6 were detected in the sample. An analysis of chimpanzee and gorilla P6 orthologs showed that the ancestral state bias has existed in all three species, and comparison of human and chimpanzee sequences with the gorilla outgroup confirmed that GC bias of the conversion process has apparently been active in both the human and chimpanzee lineages
Socio-Economic Instability and the Scaling of Energy Use with Population Size
- Author
- A Goodman
- A Kander
- AJ Stutz
- CT Bauch
- DWE Rees
- EA Rosa
- EA Wrigley
- EA Wrigley
- GF Gause
- H Haberl
- H Samaniego
- J Brown
- J Mokyr
- J Rockstrom
- JC Nekola
- JE Cohen
- JE Cohen
- JG Gauthier
- John P. DeLong
- JP DeLong
- JP DeLong
- JP DeLong
- JP Holdren
- JR Goldstein
- JZ Shik
- K Jaffe
- LA White
- LH Kaack
- LMA Bettencourt
- LMA Bettencourt
- M Berg
- M Giampietro
- M Myrskyla
- M Scheffer
- ME Moses
- MJ Hamilton
- MJ Hamilton
- MJ Hamilton
- MK Shenk
- Mora PM Foerster H von
- Mulder M Borgerhoff
- N Georgescu-Roegen
- NB Schulz
- O Burger
- Oskar Burger
- P Deane
- P Ehrlich
- P Turchin
- P Turchin
- R Lee
- R Pearl
- RE Frisch
- RM Sibly
- RN Adams
- Rodrigo Huerta-Quintanilla
- S Suweis
- T Dietz
- TD Allendorf
- TJ Espenshade
- TR Malthus
- V Smil
- V Smil
- W Lutz
- WP Nel
- WS Thompson
- Y Liu
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/06/2015
- Field of study
The size of the human population is relevant to the development of a sustainable world, yet the forces setting growth or declines in the human population are poorly understood. Generally, population growth rates depend on whether new individuals compete for the same energy (leading to Malthusian or density-dependent growth) or help to generate new energy (leading to exponential and super-exponential growth). It has been hypothesized that exponential and super-exponential growth in humans has resulted from carrying capacity, which is in part determined by energy availability, keeping pace with or exceeding the rate of population growth. We evaluated the relationship between energy use and population size for countries with long records of both and the world as a whole to assess whether energy yields are consistent with the idea of an increasing carrying capacity. We find that on average energy use has indeed kept pace with population size over long time periods. We also show, however, that the energy-population scaling exponent plummets during, and its temporal variability increases preceding, periods of social, political, technological, and environmental change. We suggest that efforts to increase the reliability of future energy yields may be essential for stabilizing both population growth and the global socio-economic system
Observation of associated near-side and away-side long-range correlations in √sNN=5.02 TeV proton-lead collisions with the ATLAS detector
- Author
- Aad G
- Abajyan T
- Abbott B
- Abdallah J
- Abdel Khalek S
- Abdelalim AA
- Abdinov O
- Aben R
- Abi B
- Abolins M
- Abouzeid OS
- Abramowicz H
- Abreu H
- Acharya BS
- Adamczyk L
- Adams DL
- Addy TN
- Adelman J
- Adomeit S
- Adragna P
- Adye T
- Aefsky S
- Aguilar-Saavedra JA
- Agustoni M
- Ahlen SP
- Ahles F
- Ahmad A
- Ahsan M
- Aielli G
- Akesson TP
- Akimoto G
- Akimov AV
- Alam MA
- Albert J
- Albrand S
- Aleksa M
- Aleksandrov IN
- Alessandria F
- Alexa C
- Alexander G
- Alexandre G
- Alexopoulos T
- Alhroob M
- Aliev M
- Alimonti G
- Alison J
- Allbrooke BM
- Allison LJ
- Allport PP
- Allwood-Spiers SE
- Almond J
- Aloisio A
- Alon R
- Alonso A
- Alonso F
- Altheimer A
- Alvarez Gonzalez B
- Alviggi MG
- Amako K
- Amelung C
- Ammosov VV
- Amor Dos Santos SP
- Amorim A
- Amoroso S
- Amram N
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders G
- Anderson KJ
- Andreazza A
- Andrei V
- Anduaga XS
- Angelidakis S
- Anger P
- Angerami A
- Anghinolfi F
- Anisenkov A
- Anjos N
- Annovi A
- Antonaki A
- Antonelli M
- Antonov A
- Antos J
- Anulli F
- Aoki M
- Aperio Bella L
- Apolle R
- Arabidze G
- Aracena I
- Arai Y
- Arce AT
- Arfaoui S
- Arguin JF
- Argyropoulos S
- Arik E
- Arik M
- Armbruster AJ
- Arnaez O
- Arnal V
- Artamonov A
- Artoni G
- Arutinov D
- Asai S
- Ask S
- Asman B
- Asquith L
- Assamagan K
- Astalos R
- Astbury A
- Atkinson M
- ATLAS Collaboration The
- Auerbach B
- Auge E
- Augsten K
- Aurousseau M
- Avolio G
- Axen D
- Azuelos G
- Azuma Y
- Baak MA
- Baccaglioni G
- Bacci C
- Bach AM
- Bachacou H
- Bachas K
- Backes M
- Backhaus M
- Backus Mayes J
- Badescu E
- Bagnaia P
- Bai Y
- Bailey DC
- Bain T
- Baines JT
- Baker OK
- Baker S
- Balek P
- Balli F
- Banas E
- Banerjee P
- Banerjee S
- Banfi D
- Bangert A
- Bansal V
- Bansil HS
- Barak L
- Baranov SP
- Barber T
- Barberio EL
- Barberis D
- Barbero M
- Bardin DY
- Barillari T
- Barisonzi M
- Barklow T
- Barlow N
- Barnett BM
- Barnett RM
- Baroncelli A
- Barone G
- Barr AJ
- Barreiro Guimarães da Costa J
- Barreiro F
- Bartoldus R
- Barton AE
- Bartsch V
- Basye A
- Bates RL
- Batkova L
- Batley JR
- Battaglia A
- Battistin M
- Bauer F
- Bawa HS
- Beale S
- Beau T
- Beauchemin PH
- Beccherle R
- Bechtle P
- Beck HP
- Becker K
- Becker S
- Beckingham M
- Becks KH
- Beddall A
- Beddall AJ
- Bedikian S
- Bednyakov VA
- Bee CP
- Beemster LJ
- Beermann TA
- Begel M
- Behar Harpaz S
- Belanger-Champagne C
- Bell PJ
- Bell WH
- Bella G
- Bellagamba L
- Bellomo M
- Belloni A
- Beloborodova O
- Belotskiy K
- Beltramello O
- Benary O
- Benchekroun D
- Bendtz K
- Benekos N
- Benhammou Y
- Benhar Noccioli E
- Benitez Garcia JA
- Benjamin DP
- Benoit M
- Bensinger JR
- Benslama K
- Bentvelsen S
- Berge D
- Bergeaas Kuutmann E
- Berger N
- Berghaus F
- Berglund E
- Beringer J
- Bernat P
- Bernhard R
- Bernius C
- Bernlochner FU
- Berry T
- Bertella C
- Bertin A
- Bertolucci F
- Besana MI
- Besjes GJ
- Besson N
- Bethke S
- Bhimji W
- Bianchi RM
- Bianchini L
- Bianco M
- Biebel O
- Bieniek SP
- Bierwagen K
- Biesiada J
- Biglietti M
- Bilokon H
- Bindi M
- Binet S
- Bingul A
- Bini C
- Biscarat C
- Bittner B
- Black CW
- Black JE
- Black KM
- Blair RE
- Blanchard JB
- Blazek T
- Bloch I
- Blocker C
- Blocki J
- Blum W
- Blumenschein U
- Bobbink GJ
- Bobrovnikov VS
- Bocchetta SS
- Bocci A
- Boddy CR
- Boehler M
- Boek J
- Boek TT
- Boelaert N
- Bogaerts JA
- Bogdanchikov A
- Bogouch A
- Bohm C
- Bohm J
- Boisvert V
- Bold T
- Boldea V
- Bolnet NM
- Bomben M
- Bona M
- Boonekamp M
- Bordoni S
- Borer C
- Borisov A
- Borissov G
- Borjanovic I
- Borri M
- Borroni S
- Bortfeldt J
- Bortolotto V
- Bos K
- Boscherini D
- Bosman M
- Boterenbrood H
- Bouchami J
- Boudreau J
- Bouhova-Thacker EV
- Boumediene D
- Bourdarios C
- Bousson N
- Boutouil S
- Boveia A
- Boyd J
- Boyko IR
- Bozovic-Jelisavcic I
- Bracinik J
- Branchini P
- Brandt A
- Brandt G
- Brandt O
- Bratzler U
- Brau B
- Brau JE
- Braun HM
- Brazzale SF
- Brelier B
- Bremer J
- Brendlinger K
- Brenner R
- Bressler S
- Bristow TM
- Britton D
- Brochu FM
- Brock I
- Brock R
- Broggi F
- Bromberg C
- Bronner J
- Brooijmans G
- Brooks T
- Brooks WK
- Brown G
- Bruckman de Renstrom PA
- Bruncko D
- Bruneliere R
- Brunet S
- Bruni A
- Bruni G
- Bruschi M
- Bryngemark L
- Buanes T
- Buat Q
- Bucci F
- Buchanan J
- Buchholz P
- Buckingham RM
- Buckley AG
- Buda SI
- Budagov IA
- Budick B
- Bugge L
- Bulekov O
- Bundock AC
- Bunse M
- Buran T
- Burckhart H
- Burdin S
- Burgess T
- Burke S
- Busato E
- Bussey P
- Buszello CP
- Butler B
- Butler JM
- Buttar CM
- Butterworth JM
- Buttinger W
- Byszewski M
- Büscher V
- Cabrera Urbán S
- Caforio D
- Cakir O
- Calafiura P
- Calderini G
- Calfayan P
- Calkins R
- Caloba LP
- Caloi R
- Calvet D
- Calvet S
- Camacho Toro R
- Camarri P
- Cameron D
- Caminada LM
- Caminal Armadans R
- Campana S
- Campanelli M
- Canale V
- Canelli F
- Canepa A
- Cantero J
- Cantrill R
- Cao T
- Capeans Garrido MD
- Caprini I
- Caprini M
- Capriotti D
- Capua M
- Caputo R
- Cardarelli R
- Carli T
- Carlino G
- Carminati L
- Caron S
- Carquin E
- Carrillo-Montoya GD
- Carter AA
- Carter JR
- Carvalho J
- Casadei D
- Casado MP
- Cascella M
- Caso C
- Castaneda-Miranda E
- Castillo Gimenez V
- Castro NF
- Cataldi G
- Catastini P
- Catinaccio A
- Catmore JR
- Cattai A
- Cattani G
- Caughron S
- Cavaliere V
- Cavalleri P
- Cavalli D
- Cavalli-Sforza M
- Cavasinni V
- Ceradini F
- Cerqueira AS
- Cerri A
- Cerrito L
- Cerutti F
- Cetin SA
- Chafaq A
- Chakraborty D
- Chalupkova I
- Chan K
- Chang P
- Chapleau B
- Chapman JD
- Chapman JW
- Charlton DG
- Chavda V
- Chavez Barajas CA
- Cheatham S
- Chekanov S
- Chekulaev SV
- Chelkov GA
- Chelstowska MA
- Chen C
- Chen H
- Chen S
- Chen X
- Chen Y
- Cheng Y
- Cheplakov A
- Cherkaoui El Moursli R
- Chernyatin V
- Cheu E
- Cheung SL
- Chevalier L
- Chiefari G
- Chikovani L
- Childers JT
- Chilingarov A
- Chiodini G
- Chisholm AS
- Chislett RT
- Chitan A
- Chizhov MV
- Choudalakis G
- Chouridou S
- Chow BK
- Christidi IA
- Christov A
- Chromek-Burckhart D
- Chu ML
- Chudoba J
- Ciapetti G
- Ciftci AK
- Ciftci R
- Cinca D
- Cindro V
- Ciocio A
- Cirilli M
- Cirkovic P
- Citron ZH
- Citterio M
- Ciubancan M
- Clark A
- Clark PJ
- Clarke RN
- Cleland W
- Clemens JC
- Clement B
- Clement C
- Coadou Y
- Cobal M
- Coccaro A
- Cochran J
- Coffey L
- Cogan JG
- Coggeshall J
- Colas J
- Cole B
- Cole S
- Colijn AP
- Collins NJ
- Collins-Tooth C
- Collot J
- Colombo T
- Colon G
- Compostella G
- Conde Muiño P
- Coniavitis E
- Conidi MC
- Consonni SM
- Consorti V
- Constantinescu S
- Conta C
- Conti G
- Conventi F
- Cooke M
- Cooper BD
- Cooper-Sarkar AM
- Copic K
- Cornelissen T
- Corradi M
- Corriveau F
- Cortes-Gonzalez A
- Cortiana G
- Costa G
- Costa MJ
- Costanzo D
- Cottin G
- Courneyea L
- Cowan G
- Cox BE
- Cranmer K
- Crescioli F
- Cristinziani M
- Crosetti G
- Crépé-Renaudin S
- Cuciuc CM
- Cuenca Almenar C
- Cuhadar Donszelmann T
- Cummings J
- Curatolo M
- Curtis CJ
- Cuthbert C
- Cwetanski P
- Czirr H
- Czodrowski P
- Czyczula Z
- Côté D
- D'Auria S
- D'Onofrio M
- D'Orazio A
- Da Cunha Sargedas De Sousa MJ
- Da Via C
- Dabrowski W
- Dafinca A
- Dai T
- Dallaire F
- Dallapiccola C
- Dam M
- Damiani DS
- Danielsson HO
- Dao V
- Darbo G
- Darlea GL
- Dassoulas JA
- Davey W
- Davidek T
- Davidson N
- Davidson R
- Davies E
- Davies M
- Davignon O
- Davison AR
- Davygora Y
- Dawe E
- Dawson I
- Daya-Ishmukhametova RK
- de Asmundis R
- De Castro S
- De Cecco S
- de Graat J
- De Groot N
- de Jong P
- De La Taille C
- De la Torre H
- De Lorenzi F
- De Nooij L
- De Pedis D
- De Salvo A
- De Sanctis U
- De Santo A
- De Vivie De Regie JB
- De Zorzi G
- De K
- Dearnaley WJ
- Debbe R
- Debenedetti C
- Dechenaux B
- Dedovich DV
- Degenhardt J
- Del Peso J
- Del Prete T
- Delemontex T
- Deliyergiyev M
- Dell'acqua A
- Dell'asta L
- Della Pietra M
- Della Volpe D
- Delmastro M
- Delsart PA
- Deluca C
- Demers S
- Demichev M
- Demirkoz B
- Denisov SP
- Derendarz D
- Derkaoui JE
- Derue F
- Dervan P
- Desch K
- Deviveiros PO
- Dewhurst A
- Dewilde B
- Dhaliwal S
- Dhullipudi R
- Di Ciaccio A
- Di Ciaccio L
- Di Donato C
- Di Girolamo A
- Di Girolamo B
- Di Luise S
- Di Mattia A
- Di Micco B
- Di Nardo R
- Di Simone A
- Di Sipio R
- Diaz MA
- Diehl EB
- Dietrich J
- Dietzsch TA
- Diglio S
- Dindar Yagci K
- Dingfelder J
- Dinut F
- Dionisi C
- Dita P
- Dita S
- Dittus F
- Djama F
- Djobava T
- do Vale MA
- Do Valle Wemans A
- Doan TK
- Dobbs M
- Dobos D
- Dobson E
- Dodd J
- Doglioni C
- Doherty T
- Dohmae T
- Doi Y
- Dolejsi J
- Dolezal Z
- Dolgoshein BA
- Donadelli M
- Donini J
- Dopke J
- Doria A
- Dos Anjos A
- Dotti A
- Dova MT
- Doyle AT
- Dressnandt N
- Dris M
- Dubbert J
- Dube S
- Dubreuil E
- Duchovni E
- Duckeck G
- Duda D
- Dudarev A
- Dudziak F
- Duerdoth IP
- Duflot L
- Dufour MA
- Duguid L
- Dunford M
- Duran Yildiz H
- Duxfield R
- Dwuznik M
- Dührssen M
- Düren M
- Ebenstein WL
- Ebke J
- Eckweiler S
- Edson W
- Edwards CA
- Edwards NC
- Ehrenfeld W
- Eifert T
- Eigen G
- Einsweiler K
- Eisenhandler E
- Ekelof T
- El Kacimi M
- Ellert M
- Elles S
- Ellinghaus F
- Ellis K
- Ellis N
- Elmsheuser J
- Elsing M
- Emeliyanov D
- Engelmann R
- Engl A
- Epp B
- Erdmann J
- Ereditato A
- Eriksson D
- Ernst J
- Ernst M
- Ernwein J
- Errede D
- Errede S
- Ertel E
- Escalier M
- Esch H
- Escobar C
- Espinal Curull X
- Esposito B
- Etienne F
- Etienvre AI
- Etzion E
- Evangelakou D
- Evans H
- Fabbri L
- Fabre C
- Facini GJ
- Fakhrutdinov RM
- Falciano S
- Fang Y
- Fanti M
- Farbin A
- Farilla A
- Farley J
- Farooque T
- Farrell S
- Farrington SM
- Farthouat P
- Fassi F
- Fassnacht P
- Fassouliotis D
- Fatholahzadeh B
- Favareto A
- Fayard L
- Federic P
- Fedin OL
- Fedorko W
- Fehling-Kaschek M
- Feligioni L
- Feng C
- Feng EJ
- Fenyuk AB
- Ferencei J
- Fernando W
- Ferrag S
- Ferrando J
- Ferrara V
- Ferrari A
- Ferrari P
- Ferrari R
- Ferreira de Lima DE
- Ferrer A
- Ferrere D
- Ferretti C
- Ferretto Parodi A
- Fiascaris M
- Fiedler F
- Filipčič A
- Filthaut F
- Fincke-Keeler M
- Fiolhais MC
- Fiorini L
- Firan A
- Fischer J
- Fisher MJ
- Fitzgerald EA
- Flechl M
- Fleck I
- Fleischmann P
- Fleischmann S
- Fletcher G
- Fletcher GT
- Flick T
- Floderus A
- Flores Castillo LR
- Florez Bustos AC
- Flowerdew MJ
- Fonseca Martin T
- Formica A
- Forti A
- Fortin D
- Fournier D
- Fowler AJ
- Fox H
- Francavilla P
- Franchini M
- Franchino S
- Francis D
- Frank T
- Franklin M
- Franz S
- Fraternali M
- Fratina S
- French ST
- Friedrich C
- Friedrich F
- Froidevaux D
- Frost JA
- Fukunaga C
- Fullana Torregrosa E
- Fulsom BG
- Fuster J
- Gabaldon C
- Gabizon O
- Gadatsch S
- Gadfort T
- Gadomski S
- Gagliardi G
- Gagnon P
- Galea C
- Galhardo B
- Gallas EJ
- Gallo V
- Gallop BJ
- Gallus P
- Gan KK
- Gandrajula RP
- Gao YS
- Gaponenko A
- Garay Walls FM
- Garberson F
- Garcia-Sciveres M
- García Navarro JE
- García C
- Gardner RW
- Garelli N
- Garonne V
- Gatti C
- Gaudio G
- Gaur B
- Gauthier L
- Gauzzi P
- Gavrilenko IL
- Gay C
- Gaycken G
- Gazis EN
- Ge P
- Gecse Z
- Gee CN
- Geerts DA
- Geich-Gimbel CH
- Gellerstedt K
- Gemme C
- Gemmell A
- Genest MH
- Gentile S
- George M
- George S
- Gerbaudo D
- Gerlach P
- Gershon A
- Geweniger C
- Ghazlane H
- Ghodbane N
- Giacobbe B
- Giagu S
- Giangiobbe V
- Gianotti F
- Gibbard B
- Gibson A
- Gibson SM
- Gilchriese M
- Gillam TP
- Gillberg D
- Gillman AR
- Gingrich DM
- Ginzburg J
- Giokaris N
- Giordani MP
- Giordano R
- Giorgi FM
- Giovannini P
- Giraud PF
- Giugni D
- Giunta M
- Gjelsten BK
- Gladilin LK
- Glasman C
- Glatzer J
- Glazov A
- Glonti GL
- Goddard JR
- Godfrey J
- Godlewski J
- Goebel M
- Goeringer C
- Goldfarb S
- Golling T
- Golubkov D
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- Zaidan R
- Zaitsev AM
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- Zanzi D
- Zaytsev A
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- Zemla A
- Zenin O
- Zeniš T
- Zerwas D
- Zevi Della Porta G
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- Zhao Z
- Zhemchugov A
- Zhong J
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- Zhuravlov V
- Zibell A
- Zieminska D
- Zimin NI
- Zimmermann R
- Zimmermann S
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- Zinonos Z
- Ziolkowski M
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- Zmouchko VV
- Zobernig G
- Zoccoli A
- Zur Nedden M
- Zutshi V
- Zwalinski L
- Publication venue
- American Physical Society
- Publication date
- 01/01/2012
- Field of study
Two-particle correlations in relative azimuthal angle (Δϕ) and pseudorapidity (Δη) are measured in √sNN=5.02 TeV p+Pb collisions using the ATLAS detector at the LHC. The measurements are performed using approximately 1 μb-1 of data as a function of transverse momentum (pT) and the transverse energy (ΣETPb) summed over 3.1<η<4.9 in the direction of the Pb beam. The correlation function, constructed from charged particles, exhibits a long-range (2<|Δη|<5) “near-side” (Δϕ∼0) correlation that grows rapidly with increasing ΣETPb. A long-range “away-side” (Δϕ∼π) correlation, obtained by subtracting the expected contributions from recoiling dijets and other sources estimated using events with small ΣETPb, is found to match the near-side correlation in magnitude, shape (in Δη and Δϕ) and ΣETPb dependence. The resultant Δϕ correlation is approximately symmetric about π/2, and is consistent with a dominant cos2Δϕ modulation for all ΣETPb ranges and particle pT
Search for the neutral Higgs bosons of the minimal supersymmetric standard model in pp collisions at root s=7 TeV with the ATLAS detector
- Author
- Aad G
- Abajyan T
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- Abdallah J
- Abdelalim AA
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- Yamauchi K
- Yamazaki T
- Yamazaki Y
- Yan Z
- Yang H
- Yang H
- Yang UK
- Yang Y
- Yang Z
- Yanush S
- Yao L
- Yasu Y
- Yatsenko E
- Ye J
- Ye S
- Yen AL
- Yilmaz M
- Yoosoofmiya R
- Yorita K
- Yoshida R
- Yoshihara K
- Young C
- Young CJ
- Youssef S
- Yu D
- Yu DR
- Yu J
- Yu J
- Yuan L
- Yurkewicz A
- Zabinski B
- Zaidan R
- Zaitsev AM
- Zanello L
- Zanzi D
- Zaytsev A
- Zeitnitz C
- Zeman M
- Zemla A
- Zenin O
- Zenis T
- Zerwas D
- Zhang D
- Zhang H
- Zhang J
- Zhang X
- Zhang Z
- Zhao L
- Zhao Z
- Zhemchugov A
- Zhong J
- Zhou B
- Zhou N
- Zhou Y
- Zhu CG
- Zhu H
- Zhu J
- Zhu Y
- Zhuang X
- Zhuravlov V
- Zibell A
- Zieminska D
- Zimin NI
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zitoun R
- Zivkovic L
- Zmouchko VV
- Zobernig G
- Zoccoli A
- zur Nedden M
- Zutshi V
- Zwalinski L
- Publication venue
- Publication date
- 01/02/2013
- Field of study
A search for neutral Higgs bosons of the Minimal Supersymmetric Standard Model (MSSM) is reported. The analysis is based on a sample of proton-proton collisions at a centre-of-mass energy of 7TeV recorded with the ATLAS detector at the Large Hadron Collider. The data were recorded in 2011 and correspond to an integrated luminosity of 4.7 fb-1 to 4.8 fb-1. Higgs boson decays into oppositely-charged muon or τ lepton pairs are considered for final states requiring either the presence or absence of b-jets. No statistically significant excess over the expected background is observed and exclusion limits at the 95% confidence level are derived. The exclusion limits are for the production cross-section of a generic neutral Higgs boson, φ, as a function of the Higgs boson mass and for h/A/H production in the MSSM as a function of the parameters mA and tan β in the mhmax scenario for mA in the range of 90GeV to 500 GeV. Copyright CERN
Search for R-parity-violating supersymmetry in events with four or more leptons in sqrt(s) =7 TeV pp collisions with the ATLAS detector
- Author
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- Abajyan T
- Abbott B
- Abdallah J
- Khalek SA
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- Yilmaz M
- Yoosoofmiya R
- Yorita K
- Yoshida R
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- Zeitnitz C
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- Zerwas D
- della Porta GZ
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- Zhemchugov A
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- Zibell A
- Zieminska D
- Zimin NI
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Ziolkowski M
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- Zivkovic L
- Zmouchko VV
- Zobernig G
- Zoccoli A
- zur Nedden M
- Zutshi V
- Zwalinski L
- Collaboration ATLAS
- Publication venue
- Springer Verlag
- Publication date
- 01/01/2008
- Field of study
A search for new phenomena in final states with four or more leptons (electrons or muons) is presented. The analysis is based on 4.7 fb−1 of s=7TeV proton-proton collisions delivered by the Large Hadron Collider and recorded with the ATLAS detector. Observations are consistent with Standard Model expectations in two signal regions: one that requires moderate values of missing transverse momentum and another that requires large effective mass. The results are interpreted in a simplified model of R-parity-violating supersymmetry in which a 95% CL exclusion region is set for charged wino masses up to 540 GeV. In an R-parity-violating MSUGRA/CMSSM model, values of m 1/2 up to 820 GeV are excluded for 10 < tan β < 40
Search for high-mass resonances decaying to dilepton final states in pp collisions at s√=7 TeV with the ATLAS detector
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- Abdelalim AA
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- Zhemchugov A
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- Zimmermann R
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- Zinonos Z
- Ziolkowski M
- Zitoun R
- Zivkovic L
- Zmouchko VV
- Zobernig G
- Zoccoli A
- zur Nedden M
- Zutshi V
- Zwalinski L
- Publication venue
- Springer
- Publication date
- 23/11/2012
- Field of study
The ATLAS detector at the Large Hadron Collider is used to search for high-mass resonances decaying to an electron-positron pair or a muon-antimuon pair. The search is sensitive to heavy neutral Z′ gauge bosons, Randall-Sundrum gravitons, Z * bosons, techni-mesons, Kaluza-Klein Z/γ bosons, and bosons predicted by Torsion models. Results are presented based on an analysis of pp collisions at a center-of-mass energy of 7 TeV corresponding to an integrated luminosity of 4.9 fb−1 in the e + e − channel and 5.0 fb−1 in the μ + μ −channel. A Z ′ boson with Standard Model-like couplings is excluded at 95 % confidence level for masses below 2.22 TeV. A Randall-Sundrum graviton with coupling k/MPl=0.1 is excluded at 95 % confidence level for masses below 2.16 TeV. Limits on the other models are also presented, including Technicolor and Minimal Z′ Models
A three-country comparison of psychotropic medication prevalence in youth
- Author
- A Clavenna
- A Knellwolf
- AM Epstein
- B Dawson
- B Vitiello
- C Frances
- C Moreno
- CB Forrest
- CF Frances
- CL Chiang
- DA Zarin
- Daniel J Safer
- E Schirm
- F Rasmussen
- G Tripp
- Gerd Glaeske
- GR Byck
- H Tobi
- IC Wong
- J Mancini
- James F Gardner
- JG Hugtenburg
- JG Simeon
- JJ Criado Alvarez
- JM Fegert
- JM Swanson
- JM Zito
- JM Zito
- JM Zito
- JM Zito
- Joerg M Fegert
- Julie M Zito
- JZ Ayanian
- K McPherson
- Katrin Janhsen
- KH Sheehan
- Lolkje TW de Jong-van den Berg
- M Andretta
- M Holtmann
- M Percudani
- ML Murray
- N Middleton
- NC Patel
- P Starr
- PH Thomsen
- RM Scheffler
- Satish C Valluri
- SO Schmidt-Troschke
- Publication venue
- BioMed Central
- Publication date
- 01/01/2008
- Field of study
<p>Abstract</p> <p>Background</p> <p>The study aims to compare cross-national prevalence of psychotropic medication use in youth.</p> <p>Methods</p> <p>A population-based analysis of psychotropic medication use based on administrative claims data for the year 2000 was undertaken for insured enrollees from 3 countries in relation to age group (0–4, 5–9, 10–14, and 15–19), gender, drug subclass pattern and concomitant use. The data include insured youth aged 0–19 in the year 2000 from the Netherlands (n = 110,944), Germany (n = 356,520) and the United States (n = 127,157).</p> <p>Results</p> <p>The annual prevalence of any psychotropic medication in youth was significantly greater in the US (6.7%) than in the Netherlands (2.9%) and in Germany (2.0%). Antidepressant and stimulant prevalence were 3 or more times greater in the US than in the Netherlands and Germany, while antipsychotic prevalence was 1.5–2.2 times greater. The atypical antipsychotic subclass represented only 5% of antipsychotic use in Germany, but 48% in the Netherlands and 66% in the US. The less commonly used drugs e.g. alpha agonists, lithium and antiparkinsonian agents generally followed the ranking of US>Dutch>German youth with very rare (less than 0.05%) use in Dutch and German youth. Though rarely used, anxiolytics were twice as common in Dutch as in US and German youth. Prescription hypnotics were half as common as anxiolytics in Dutch and US youth and were very uncommon in German youth. Concomitant drug use applied to 19.2% of US youth which was more than double the Dutch use and three times that of German youth.</p> <p>Conclusion</p> <p>Prominent differences in psychotropic medication treatment patterns exist between youth in the US and Western Europe and within Western Europe. Differences in policies regarding direct to consumer drug advertising, government regulatory restrictions, reimbursement policies, diagnostic classification systems, and cultural beliefs regarding the role of medication for emotional and behavioral treatment are likely to account for these differences.</p
Identification of Sequence Variants in Genetic Disease-Causing Genes Using Targeted Next-Generation Sequencing
- Author
- A Bonnefond
- A Efstratiadis
- A Gnirke
- B Aissani
- B Arezi
- B Buehler
- CJ Bell
- G Jimenez-Sanchez
- Guanghui Yang
- H Li
- Hui Jiang
- Illumina
- J Li
- J Wang
- Jian Huang
- Jinming Wang
- JS Berg
- JZ Levin
- L Mamanova
- L Wan
- M Choi
- Ming Qi
- ML Metzker
- MM Deguti
- Ning Qu
- P Sham
- P Shen
- Qian Zhu
- R Li
- R Tewhey
- RA Gibbs
- Ruan Zhen
- S Liao
- SB Ng
- SB Ng
- SE Calvo
- TC Cheng
- Tengfei Liu
- V Bansal
- V Bansal
- Xiangchun Ju
- Xiaoming Wei
- Xiaoyan Li
- Xin Yi
- XM Xu
- Yanfang Guang
- Yang Chen
- Yi Yang
- YL Shih
- Yuxing Chu
- Zhangzhang Lan
- Zhanjiang Liu
- Publication venue
- Public Library of Science
- Publication date
- 21/12/2011
- Field of study
Identification of gene variants plays an important role in research on and diagnosis of genetic diseases. A combination of enrichment of targeted genes and next-generation sequencing (targeted DNA-HiSeq) results in both high efficiency and low cost for targeted sequencing of genes of interest.To identify mutations associated with genetic diseases, we designed an array-based gene chip to capture all of the exons of 193 genes involved in 103 genetic diseases. To evaluate this technology, we selected 7 samples from seven patients with six different genetic diseases resulting from six disease-causing genes and 100 samples from normal human adults as controls. The data obtained showed that on average, 99.14% of 3,382 exons with more than 30-fold coverage were successfully detected using Targeted DNA-HiSeq technology, and we found six known variants in four disease-causing genes and two novel mutations in two other disease-causing genes (the STS gene for XLI and the FBN1 gene for MFS) as well as one exon deletion mutation in the DMD gene. These results were confirmed in their entirety using either the Sanger sequencing method or real-time PCR.Targeted DNA-HiSeq combines next-generation sequencing with the capture of sequences from a relevant subset of high-interest genes. This method was tested by capturing sequences from a DNA library through hybridization to oligonucleotide probes specific for genetic disorder-related genes and was found to show high selectivity, improve the detection of mutations, enabling the discovery of novel variants, and provide additional indel data. Thus, targeted DNA-HiSeq can be used to analyze the gene variant profiles of monogenic diseases with high sensitivity, fidelity, throughput and speed
Protein interaction network of alternatively spliced isoforms from brain links genetic risk factors for autism
- Author
- A Bayes
- AC Need
- AC Need
- B Ewing
- B Zhang
- BJ O'Roak
- BM Neale
- C Zweier
- CR Marshall
- D Levy
- D Malhotra
- D Pinto
- DR Mowat
- E Ben-David
- ET Wang
- FD Hannes
- FF Hamdan
- G Temple
- H Goehler
- H Hibino
- H Li
- H Stefansson
- H Yu
- HC Mefford
- HJ Noh
- HR Christofk
- I Iossifov
- I Voineagu
- J Lim
- J Sebat
- J Sebat
- JC Darnell
- JJ Michaelson
- JM Berg
- JW Smoller
- JZ Wang
- K Salehi-Ashtiani
- K Venkatesan
- K Wang
- LA Weiss
- LM Camargo
- M Bucan
- M Dreze
- ME Cusick
- ME Talkowski
- N Eswar
- NM Williams
- P Braun
- P Lamesch
- Q Pan
- QC Zhang
- R Anney
- R Ullmann
- RB Roth
- S Eyckerman
- S Horvath
- S Raychaudhuri
- S Steinberg
- SH Lee
- SJ Sanders
- SJ Sanders
- SR Gilman
- TD Wu
- X Gai
- X Huang
- X Kuang
- X Yang
- Y Sakai
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2014
- Field of study
Increased risk for autism spectrum disorders (ASD) is attributed to hundreds of genetic loci. The convergence of ASD variants have been investigated using various approaches, including protein interactions extracted from the published literature. However, these datasets are frequently incomplete, carry biases and are limited to interactions of a single splicing isoform, which may not be expressed in the disease-relevant tissue. Here we introduce a new interactome mapping approach by experimentally identifying interactions between brain-expressed alternatively spliced variants of ASD risk factors. The Autism Spliceform Interaction Network reveals that almost half of the detected interactions and about 30% of the newly identified interacting partners represent contribution from splicing variants, emphasizing the importance of isoform networks. Isoform interactions greatly contribute to establishing direct physical connections between proteins from the de novo autism CNVs. Our findings demonstrate the critical role of spliceform networks for translating genetic knowledge into a better understanding of human diseases
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