67 research outputs found
COPPADIS-2015 (COhort of Patients with PArkinson's DIsease in Spain, 2015), a global--clinical evaluations, serum biomarkers, genetic studies and neuroimaging--prospective, multicenter, non-interventional, long-term study on Parkinson's disease progressio
- Author
- A Brück
- A Cervantes-Arriaga
- A Merola
- A Schrag
- AJ Clark
- AT Beck
- B Dubois
- B Ford
- BC Robinson
- BG Tabachnick
- Biomarkers Definitions Working Group
- C Jenkinson
- C Toth
- CH Adler
- CH Williams-Gray
- CS Burckhardt
- CW Olanow
- D Aarsland
- D Lindqvist
- D Santos García
- D Santos-García
- D Weintraub
- DC Velseboer
- Diego Santos-García
- DJ Brooks
- DJ McGhee
- DV Schaf
- E Cubo
- E Tolosa
- E Ziegler
- ELEP Grupo
- Esther Cubo
- F Agosta
- G Madenci
- GT Stebbins
- H Honig
- H Li
- H Sawada
- J Jankovic
- J Jankovic
- J Pagonabarraga
- Jaime Kulisevsky
- JB Anang
- JD Rogers
- JE Lee
- JG Goldman
- JG Goldman
- JL Cummings
- Jon Infante
- José Matías Arbelo
- JS Paulsen
- JTE Richarson
- K Marek
- K Sathe
- K Seppi
- K Zhu
- K Zhu
- KF Pedersen
- KR Chaudhuri
- KR Chaudhuri
- L Marsh
- L Zhang
- LL Judd
- Lydia Vela
- M Auyeung
- M Balzer-Geldsetzer
- M Dufek
- M Farhoudi
- M Kasten
- M Menéndez-González
- M Novak
- M Reale
- M Rothermundt
- MA Hely
- MA Schwarzschild
- Mari Cruz Rodríguez-Oroz
- Maria José Martí
- MF Folstein
- MM Hoehn
- N Giladi
- N Ibarretxe-Bilbao
- NS Da Rocha
- O Oguh
- OH Gerlach
- P Bugalho
- P Martinez-Martin
- Pablo Martínez-Martín
- Pablo Mir
- PD MED Collaborative Group
- PPM Initiative
- R Katzenschlager
- RS Schwab
- S Fanhn
- S Lesage
- S Vercruysse
- SB Tan
- SE Daniel
- SE Winder-Rhodes
- ST Tsai
- T Nagatsu
- T Stewart
- YH Hiorth
- YY Chen
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
Background: Parkinson?s disease (PD) is a progressive neurodegenerative disorder causing motor and non-motor symptoms that can affect independence, social adjustment and the quality of life (QoL) of both patients and caregivers. Studies designed to find diagnostic and/or progression biomarkers of PD are needed. We describe here the study protocol of COPPADIS-2015 (COhort of Patients with PArkinson?s DIsease in Spain, 2015), an integral PD project based on four aspects/concepts: 1) PD as a global disease (motor and non-motor symptoms); 2) QoL and caregiver issues; 3) Biomarkers; 4) Disease progression.Methods/design: Observational, descriptive, non-interventional, 5-year follow-up, national (Spain), multicenter (45 centers from 15 autonomous communities), evaluation study. Specific goals: (1) detailed study (clinical evaluations, serum biomarkers, genetic studies and neuroimaging) of a population of PD patients from different areas of Spain, (2) comparison with a control group and (3) follow-up for 5 years. COPPADIS-2015 has been specifically designed to assess 17 proposed objectives. Study population: approximately 800 non-dementia PD patients, 600 principal caregivers and 400 control subjects. Study evaluations: (1) baseline includes motor assessment (e.g., Unified Parkinson?s Disease Rating Scale part III), non-motor symptoms (e.g., Non-Motor Symptoms Scale), cognition (e.g., Parkinson?s Disease Cognitive Rating Scale), mood and neuropsychiatric symptoms (e.g., Neuropsychiatric Inventory), disability, QoL (e.g., 39-item Parkinson?s disease Quality of Life Questionnaire Summary-Index) and caregiver status (e.g., Zarit Caregiver Burden Inventory); (2) follow-up includes annual (patients) or biannual (caregivers and controls) evaluations. Serum biomarkers (S-100b protein, TNF-?, IL-1, IL-2, IL-6, vitamin B12, methylmalonic acid, homocysteine, uric acid, C-reactive protein, ferritin, iron) and brain MRI (volumetry, tractography and MTAi [Medial Temporal Atrophy Index]), at baseline and at the end of follow-up, and genetic studies (DNA and RNA) at baseline will be performed in a subgroup of subjects (300 PD patients and 100 control subjects). Study periods: (1) recruitment period, from November, 2015 to February, 2017 (basal assessment); (2) follow-up period, 5 years; (3) closing date of clinical follow-up, May, 2022. Funding: Public/Private. Discussion: COPPADIS-2015 is a challenging initiative. This project will provide important information on the natural history of PD and the value of various biomarkers
Identification of Candidate Parkinson Disease Genes by Integrating Genome-Wide Association Study, Expression, and Epigenetic Data Sets
- Author
- Adarmes-Gómez AD
- Adolfo Mínguez MM
- Aguilar M
- Alcalay RN
- Alvarez I
- Alvarez V
- Arregui ALDM
- Bandres-Ciga S
- Bergareche Yarza JA
- Bernal-Bernal I
- Billingsley K
- Blauwendraat C
- Blazquez M
- Bonilla-Toribio M
- Botiá JA
- Botía J
- Botía JA
- Boungiorno MT
- Bras J
- Brice A
- Brockmann K
- Buiza-Rueda D
- Carrillo F
- Carrión-Claro M
- Cerdan D
- Chelban V
- Clarimón J
- Compta Y
- Cookson MR
- Corvol J-C
- Craig DW
- Càmara A
- D'sa K
- Danjou F
- Diez-Fairen M
- Dols-Icardo O
- Duarte J
- Duran R
- Errazquin FP
- Escamilla-Sevilla F
- Escott-Price V
- Ezquerra M
- Faghri F
- Feliz C
- Fernàndez M
- Fernàndez-Santiago R
- Ferrari R
- Finkbeiner S
- Foltynie T
- Forabosco P
- Gan-Or Z
- Garcia C
- García-Ruiz P
- Gasser T
- Gibbs JR
- Gomez Heredia MJ
- Gonzalez-Aramburu I
- González MM
- Guelfi MS
- Guelfi S
- Guelfi S
- Guerreiro R
- Gómez-Garre P
- Hardy J
- Hardy J
- Hassin-Baer S
- Hernandez DG
- Heutink P
- Hoenicka J
- Holmans P
- Houlden H
- Hubbard L
- Infante J
- Javier Barrero F
- Jimenez-Escrig A
- Kaiyrzhanov R
- Kia DA
- Kia DA
- Kinghorn KJ
- Koks S
- Krohn L
- Kulisevsky J
- Labrador-Espinosa MA
- Leonard HL
- Lesage S
- Lewis P
- Lewis PA
- Lopez-Sendon JL
- Lovering R
- Lubbe S
- Lungu C
- Macias D
- Manzoni C
- Manzoni C
- Marinus J
- Marti MJ
- Martinez M
- Martínez-Castrillo JC
- Marín J
- Mencacci NE
- Middlehurst B
- Mir P
- Mok KY
- Morris HR
- Muñoz E
- Mèndez-del-Barrio C
- Nalls MA
- Nicolas A
- Noyce AJ
- Pagola AG
- Pagonabarraga J
- Pastor P
- Perinán-Tocino T
- PhD JQ
- Pihlstrom L
- Plun-Favreau H
- R'Bibo L
- Ramasamy A
- Reed X
- Reynolds RH
- Rezola EM
- Rizig M
- Rizzu P
- Robak L
- Rodriguez AS
- Rouleau GA
- Ruiz-Martínez J
- Ruz C
- Ryten M
- Ryten M
- Scholz SW
- Schreglmann S
- Schulte C
- Sharma M
- Shulman JM
- Sierra M
- Simón-Sánchez J
- Singleton AB
- Smith C
- Suarez-Sanmartin E
- Taba P
- Tabernero C
- Tan M
- Tartari JP
- Tejera-Parrado C
- Toft M
- Tolosa E
- Torres IM
- Trabzuni D
- Trabzuni D
- Valldeoriola F
- van Hilten JJ
- Van Keuren-Jensen K
- Vargas-González L
- Vela L
- Vives F
- Weale M
- Williams N
- Williams N
- Wood NW
- Wood NW
- Zhang D
- Zimprich A
- Publication venue
- 'American Medical Association (AMA)'
- Publication date
- 01/02/2021
- Field of study
Importance Substantial genome-wide association study (GWAS) work in Parkinson disease (PD) has led to the discovery of an increasing number of loci shown reliably to be associated with increased risk of disease. Improved understanding of the underlying genes and mechanisms at these loci will be key to understanding the pathogenesis of PD. / Objective To investigate what genes and genomic processes underlie the risk of sporadic PD. / Design and Setting This genetic association study used the bioinformatic tools Coloc and transcriptome-wide association study (TWAS) to integrate PD case-control GWAS data published in 2017 with expression data (from Braineac, the Genotype-Tissue Expression [GTEx], and CommonMind) and methylation data (derived from UK Parkinson brain samples) to uncover putative gene expression and splicing mechanisms associated with PD GWAS signals. Candidate genes were further characterized using cell-type specificity, weighted gene coexpression networks, and weighted protein-protein interaction networks. / Main Outcomes and Measures It was hypothesized a priori that some genes underlying PD loci would alter PD risk through changes to expression, splicing, or methylation. Candidate genes are presented whose change in expression, splicing, or methylation are associated with risk of PD as well as the functional pathways and cell types in which these genes have an important role. / Results Gene-level analysis of expression revealed 5 genes (WDR6 [OMIM 606031], CD38 [OMIM 107270], GPNMB [OMIM 604368], RAB29 [OMIM 603949], and TMEM163 [OMIM 618978]) that replicated using both Coloc and TWAS analyses in both the GTEx and Braineac expression data sets. A further 6 genes (ZRANB3 [OMIM 615655], PCGF3 [OMIM 617543], NEK1 [OMIM 604588], NUPL2 [NCBI 11097], GALC [OMIM 606890], and CTSB [OMIM 116810]) showed evidence of disease-associated splicing effects. Cell-type specificity analysis revealed that gene expression was overall more prevalent in glial cell types compared with neurons. The weighted gene coexpression performed on the GTEx data set showed that NUPL2 is a key gene in 3 modules implicated in catabolic processes associated with protein ubiquitination and in the ubiquitin-dependent protein catabolic process in the nucleus accumbens, caudate, and putamen. TMEM163 and ZRANB3 were both important in modules in the frontal cortex and caudate, respectively, indicating regulation of signaling and cell communication. Protein interactor analysis and simulations using random networks demonstrated that the candidate genes interact significantly more with known mendelian PD and parkinsonism proteins than would be expected by chance. / Conclusions and Relevance Together, these results suggest that several candidate genes and pathways are associated with the findings observed in PD GWAS studies
Identification of sixteen novel candidate genes for late onset Parkinson’s disease
- Author
- Acampora D
- Adarmes-Gómez AD
- Aguilar M
- Aitkulova A
- Akhmetzhanov V
- Alcalay RN
- Alvarez I
- Alvarez V
- Bandres-Ciga S
- Barrero FJ
- Bernal-Bernal I
- Billingsley K
- Blauwendraat C
- Blazquez M
- Bonilla-Toribio M
- Botía JA
- Boungiorno MT
- Bras J
- Brice A
- Brockmann K
- Bubb V
- Buiza-Rueda D
- Carrillo F
- Carrión-Claro M
- Cerdan D
- Chelban V
- Ciullo M
- Clarimón J
- Clarke C
- Compta Y
- Cookson MR
- Corvol J-C
- Craig DW
- Danjou F
- de Munain Arregui AL
- Di Giovannantonio LG
- Diez-Fairen M
- Dols-Icardo O
- Duarte J
- Duran R
- D’Esposito M
- Errazquin FP
- Escamilla-Sevilla F
- Escott-Price V
- Esposito T
- Ezquerra M
- Faghri F
- Feliz C
- Fernández M
- Fernández-Santiago R
- Finkbeiner S
- Foltynie T
- Gambardella S
- Gan-Or Z
- Garcia C
- García-Ruiz P
- Gasser T
- Gialluisi A
- Gianfrancesco F
- Gibbs JR
- Gonzalez-Aramburu I
- González MM
- Guelfi S
- Guerreiro R
- Gómez-Garre P
- Hardy J
- Hassin-Baer S
- Heredia MJG
- Hernandez DG
- Heutink P
- Hoenicka J
- Holmans P
- Houlden H
- Iacoviello L
- Infante J
- Iwaki H
- Jesús S
- Jimenez-Escrig A
- Kaishybayeva G
- Kaiyrzhanov R
- Kaiyrzhanov R
- Karimova A
- Kia DA
- Kinghorn KJ
- Koks S
- Krohn L
- Krohn L
- Kulisevsky J
- Labrador-Espinosa MA
- Leonard HL
- Lesage S
- Lewis P
- Lombardi A
- Lopez-Sendon JL
- Lovering R
- Lubbe S
- Lungu C
- Macias D
- Majamaa K
- Manzoni C
- Marinus J
- Marti MJ
- Martinez M
- Martínez-Castrillo JC
- Marín J
- Mata M
- Mencacci NE
- Middlehurst B
- Mir P
- Modugno N
- Mok KY
- Morris HR
- Morrison KE
- Muñoz E
- Méndez-del-Barrio C
- Mínguez A
- Nalls MA
- Narendra D
- Noyce AJ
- Nutile T
- Ojo OO
- Okubadejo NU
- Pagola AG
- Pagonabarraga J
- Pastor P
- Periñán-Tocino T
- Pietracupa S
- Pihlstrom L
- Plun-Favreau H
- Quinn J
- Reccia MG
- Reed X
- Rezola EM
- Rizig M
- Rizzu P
- Robak L
- Rodriguez AS
- Rouleau GA
- Ruggiero D
- Ruiz-Martínez J
- Ruz C
- Ryten M
- R’Bibo L
- Sadykova D
- Scala S
- Scholz SW
- Schreglmann S
- Schulte C
- Sharma M
- Shashkin C
- Shulman JM
- Sierra M
- Siitonen A
- Simeone A
- Simón-Sánchez J
- Singleton AB
- Suarez-Sanmartin E
- Taba P
- Tabernero C
- Tan M
- Tartari JP
- Tejera-Parrado C
- Toft M
- Tolosa E
- Torres IM
- Trabzuni D
- Valldeoriola F
- van Hilten JJ
- Van Keuren-Jensen K
- Vargas-González L
- Vela L
- Vives F
- Williams N
- Wood NW
- Yarza JAB
- Zharkynbekova N
- Zharmukhanov Z
- Zholdybayeva E
- Zimprich A
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 21/06/2021
- Field of study
Background
Parkinson’s disease (PD) is a neurodegenerative movement disorder affecting 1–5% of the general population for which neither effective cure nor early diagnostic tools are available that could tackle the pathology in the early phase. Here we report a multi-stage procedure to identify candidate genes likely involved in the etiopathogenesis of PD.
Methods
The study includes a discovery stage based on the analysis of whole exome data from 26 dominant late onset PD families, a validation analysis performed on 1542 independent PD patients and 706 controls from different cohorts and the assessment of polygenic variants load in the Italian cohort (394 unrelated patients and 203 controls).
Results
Family-based approach identified 28 disrupting variants in 26 candidate genes for PD including PARK2, PINK1, DJ-1(PARK7), LRRK2, HTRA2, FBXO7, EIF4G1, DNAJC6, DNAJC13, SNCAIP, AIMP2, CHMP1A, GIPC1, HMOX2, HSPA8, IMMT, KIF21B, KIF24, MAN2C1, RHOT2, SLC25A39, SPTBN1, TMEM175, TOMM22, TVP23A and ZSCAN21. Sixteen of them have not been associated to PD before, were expressed in mesencephalon and were involved in pathways potentially deregulated in PD. Mutation analysis in independent cohorts disclosed a significant excess of highly deleterious variants in cases (p = 0.0001), supporting their role in PD.
Moreover, we demonstrated that the co-inheritance of multiple rare variants (≥ 2) in the 26 genes may predict PD occurrence in about 20% of patients, both familial and sporadic cases, with high specificity (> 93%; p = 4.4 × 10− 5). Moreover, our data highlight the fact that the genetic landmarks of late onset PD does not systematically differ between sporadic and familial forms, especially in the case of small nuclear families and underline the importance of rare variants in the genetics of sporadic PD.
Furthermore, patients carrying multiple rare variants showed higher risk of manifesting dyskinesia induced by levodopa treatment.
Conclusions
Besides confirming the extreme genetic heterogeneity of PD, these data provide novel insights into the genetic of the disease and may be relevant for its prediction, diagnosis and treatment
Global Impact of the COVID-19 Pandemic on Cerebral Venous Thrombosis and Mortality
- Author
- Abdalkader M
- Acampa M
- Adami A
- Adebayo TT
- Ahmad S
- Al Hashmi AM
- Alexiev F
- Araújo AP
- Arsovska AA
- Asif KS
- Aykac O
- Ayo-Martin O
- Baek JH
- Bajrami A
- Banerjee S
- Barrientos Guerra JD
- Bello AH
- Below K
- Bernava G
- Bessa F
- Biller J
- Blasco J
- Bolognese M
- Bonnet T
- Boskamp S
- Brady M
- Brola W
- Carrera E
- Castro P
- Charidimou A
- Chebl A
- Chen HS
- Chen M
- Chen Y
- Chieh CY
- Churojana A
- Ciopleias B
- Colasurdo M
- Conforto AB
- Cora EA
- Cruz-Culebras A
- Czap A
- Czlonkowska A
- Dao VP
- de Havenon AH
- de la Cruz Fernández B
- de Oliveira Neves L
- De Raedt S
- de Sousa DA
- Demeestere J
- Dharmadhikari S
- Dimitriade A
- Dippel D
- Dirren E
- Dorobek M
- Drenck N
- Duong HQ
- Dušek P
- Dy JSH
- Dębiec A
- Enzinger C
- Escartín López J
- Eskey CJ
- Falup-Pecurariu C
- Farhoudi M
- Farooqui M
- Fernández Ferro JC
- Ferreira P
- Feske SK
- Fibrich P
- Field TS
- Fonseca L
- Fujita K
- Fukuda H
- García Sánchez MJ
- Gattringer T
- Ghoreishi A
- Gongora-Rivera F
- Goyal N
- Grimmett KB
- Guillán Rodríguez M
- Guzik AK
- Hamid MA
- Haring J
- Hashemilar M
- Haussen DC
- Hernandez-Fernandez F
- Hidalgo RCT
- Hlaváčová H
- Hovingh M
- Hrabanovska E
- Huded V
- Hussain SI
- Imamura H
- Infante-Valenzuela A
- Inoa V
- Jancar N
- Jillela D
- John S
- Jurák L
- Kadlčíková J
- Kalpachki R
- Kan PT
- Karapanayiotides T
- Kargiotis O
- Karlinski MA
- Karpowicz I
- Kelly ME
- Khatri R
- Khoury NN
- Kiley NL
- Klein P
- Klečka L
- Kolikonda MK
- Kosuke M
- Koutroulou I
- Kovář M
- Krastev G
- Krishnan K
- Kristoffersen ES
- Kwan J
- Labuz-Roszak BM
- Lameirinhas da Silva I
- Lara S
- Lasek-Bal A
- Le HV
- Lee JS
- Lee SB
- Lemmens R
- Lenthall R
- Lereis VP
- Lesaine E
- Li G
- Ligot N
- Linfante I
- Lo H
- Longoni M
- Loochtan AI
- Lopez CD
- Lycan S
- Ma A
- Maali L
- Machi P
- Male SS
- Mansour OY
- Martinez-Marino M
- Marto JP
- Martíns RT
- Masoud HE
- Matsumaru Y
- Mbroh J
- Michel P
- Mikulik R
- Min J
- Mohamed GA
- Mohammaden M
- Mohammadzadeh N
- Moniche F
- Montaner J
- Mora Cuervo DL
- Nagel S
- Nagendra S
- Nahab F
- Nalleballe K
- Neumann J
- Nguyen TN
- Nogueira RG
- Nunes AP
- Ohara N
- Omran SS
- Orgeta-Gutierrez S
- Ornello R
- Palaiodimou L
- Paloušková H
- Peeling L
- Perez-Sanchez S
- Pham TN
- Pikula A
- Pinho E Melo T
- Poli S
- Prajapati C
- Punter M
- Qureshi MM
- Radaideh Y
- Rahal F
- Rahman A
- Ramakrishnan P
- Rashid U
- Rayo-Taranto B
- Rebello LC
- Reiner P
- Reiser M
- Renieri L
- Rocha M
- Rodrigues M
- Rodriguez-Kadota L
- Rodríguez Fernández C
- Rodríguez-Vázquez A
- Rohan V
- Rojas-Soto DM
- Roje Bedekovic M
- Romoli M
- Rosenkranz M
- Rouanet F
- Ruland S
- Sabben C
- Sabetay SI
- Sacco S
- Sadeghi Hokmabadi E
- Sakaguchi M
- Sakai K
- Sakai N
- Sakelarova T
- Salmaggi A
- Sangalli D
- Sani AF
- Santiago IB
- Sathya A
- Scozzari D.
- Sedova P
- Senadim S
- Seo KD
- Sheth SA
- Shindo S
- Siegler JE
- Sienkiewicz-Jarosz H
- Silva ML
- Simpkins AN
- Skoda O
- Sobh K
- Sobolewski P
- Sohn SI
- Sonoda K
- Starosciak AK
- Staszewski J
- Strambo D
- Strbian D
- Sugiura Y
- Sundseth A
- Sunmonu TA
- Suryadevara NC
- SVIN COVID-19 Global COVID Stroke Registry
- Sylaja PN
- Takenobu Y
- Tarlov NE
- Taylor RA
- Tenora D
- Thomalla G
- Ton MD
- Toyoda K
- Tran AD
- Tsivgoulis G
- Uwatoko T
- van Dam-Nolen DHK
- Vanacker P
- Vandervorst F
- Vences MÁ
- Venketasubramanian N
- Voetsch B
- Wahab KW
- Wan Yahya WNN
- Wan Zaidi WA
- Wiącek M
- Wu TY
- Yaghi S
- Yalung PM
- Yamagami H
- Yamamoto N
- Yamamoto R
- Yang S
- Yazawa Y
- Yoshimoto T
- Zaidat OO
- Zhang L
- Zhang L
- Zielinska-Turek J
- Zini A
- Özdemir AÖ
- Čabal M
- Šimůnek L
- Škorňa M
- Šrámek M
- Publication venue
- Publication date
- 01/01/2022
- Field of study
Background and purpose: Recent studies suggested an increased incidence of cerebral venous thrombosis (CVT) during the coronavirus disease 2019 (COVID-19) pandemic. We evaluated the volume of CVT hospitalization and in-hospital mortality during the 1st year of the COVID-19 pandemic compared to the preceding year.
Methods: We conducted a cross-sectional retrospective study of 171 stroke centers from 49 countries. We recorded COVID-19 admission volumes, CVT hospitalization, and CVT in-hospital mortality from January 1, 2019, to May 31, 2021. CVT diagnoses were identified by International Classification of Disease-10 (ICD-10) codes or stroke databases. We additionally sought to compare the same metrics in the first 5 months of 2021 compared to the corresponding months in 2019 and 2020 (ClinicalTrials.gov Identifier: NCT04934020).
Results: There were 2,313 CVT admissions across the 1-year pre-pandemic (2019) and pandemic year (2020); no differences in CVT volume or CVT mortality were observed. During the first 5 months of 2021, there was an increase in CVT volumes compared to 2019 (27.5%; 95% confidence interval [CI], 24.2 to 32.0; P<0.0001) and 2020 (41.4%; 95% CI, 37.0 to 46.0; P<0.0001). A COVID-19 diagnosis was present in 7.6% (132/1,738) of CVT hospitalizations. CVT was present in 0.04% (103/292,080) of COVID-19 hospitalizations. During the first pandemic year, CVT mortality was higher in patients who were COVID positive compared to COVID negative patients (8/53 [15.0%] vs. 41/910 [4.5%], P=0.004). There was an increase in CVT mortality during the first 5 months of pandemic years 2020 and 2021 compared to the first 5 months of the pre-pandemic year 2019 (2019 vs. 2020: 2.26% vs. 4.74%, P=0.05; 2019 vs. 2021: 2.26% vs. 4.99%, P=0.03). In the first 5 months of 2021, there were 26 cases of vaccine-induced immune thrombotic thrombocytopenia (VITT), resulting in six deaths.
Conclusions: During the 1st year of the COVID-19 pandemic, CVT hospitalization volume and CVT in-hospital mortality did not change compared to the prior year. COVID-19 diagnosis was associated with higher CVT in-hospital mortality. During the first 5 months of 2021, there was an increase in CVT hospitalization volume and increase in CVT-related mortality, partially attributable to VITT
Combined measurement of differential and total cross sections in the H → γγ and the H → ZZ* → 4ℓ decay channels at s=13 TeV with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abhayasinghe DK
- Abidi SH
- Abouzeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Adachi S
- Adamczyk L
- Adelman J
- Adersberger M
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmadov F
- Aielli G
- Akatsuka S
- Akilli E
- Akimov AV
- Alberghi GL
- Albert J
- Albicocco P
- Alconada Verzini MJ
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alhroob M
- Ali B
- Alimonti G
- Alison J
- Alkire SP
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alstaty MI
- Alvarez Gonzalez B
- Alviggi MG
- Amadio BT
- Amaral Coutinho Y
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- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- Elsevier
- Publication date
- 01/01/2018
- Field of study
A combined measurement of differential and inclusive total cross sections of Higgs boson production is performed using 36.1 fb−1 of 13 TeV proton–proton collision data produced by the LHC and recorded by the ATLAS detector in 2015 and 2016. Cross sections are obtained from measured H→γγ and H→ZZ*(→4ℓ event yields, which are combined taking into account detector efficiencies, resolution, acceptances and branching fractions. The total Higgs boson production cross section is measured to be 57.0−5.9 +6.0 (stat.) −3.3 +4.0 (syst.) pb, in agreement with the Standard Model prediction. Differential cross-section measurements are presented for the Higgs boson transverse momentum distribution, Higgs boson rapidity, number of jets produced together with the Higgs boson, and the transverse momentum of the leading jet. The results from the two decay channels are found to be compatible, and their combination agrees with the Standard Model predictions
Measurement of the t¯tZ and t¯tW cross sections in proton-proton collisions at √s=13 TeV with the ATLAS detector
- Author
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- Aad G
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- Abdinov O
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- Zeng Q
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- Zhao P
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- Zhemchugov A
- Zheng Z
- Zhong D
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- Zhu CG
- Zhu H
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- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zur Nedden M
- Zwalinski L
- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2019
- Field of study
A measurement of the associated production of a top-quark pair (t¯t) with a vector boson (W, Z) in proton-proton collisions at a center-of-mass energy of 13 TeV is presented, using 36.1 fb−1 of integrated luminosity collected by the ATLAS detector at the Large Hadron Collider. Events are selected in channels with two same- or opposite-sign leptons (electrons or muons), three leptons or four leptons, and each channel is further divided into multiple regions to maximize the sensitivity of the measurement. The t¯tZ and t¯tW production cross sections are simultaneously measured using a combined fit to all regions. The best-fit values of the production cross sections are σt¯tZ=0.95±0.08stat±0.10syst pb and σt¯tW=0.87±0.13stat±0.14syst pb in agreement with the Standard Model predictions. The measurement of the t¯tZ cross section is used to set constraints on effective field theory operators which modify the t¯tZ vertex
Search for excited electrons singly produced in proton–proton collisions at \sqrt{s} = 13 TeV with the ALAS experiment at the LHC
- Author
- Aaboud M
- Aad G
- Abbott B
- Abbott DC
- Abdinov O
- Abeloos B
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Adachi S
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
- Adersberger M
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmadov F
- Aielli G
- Akatsuka S
- Akilli E
- Akimov AV
- Alberghi GL
- Alberich LC
- Albert J
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexandre D
- Alexopoulos T
- Alhroob M
- Ali B
- Alimonti G
- Alison J
- Alkire SP
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
- Alonso A
- Alonso F
- Alpigiani C
- Alshehri AA
- Alstaty MI
- Alviggi MG
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amoroso S
- Amrouche CS
- An F
- Anastopoulos C
- Andari N
- Andeen T
- Anders CF
- Anders JK
- Andreazza A
- Andrei V
- Anelli CR
- Angelidakis S
- Angelozzi I
- Angerami A
- Anisenkov AV
- Annovi A
- Antel C
- Anthony MT
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Arabidze G
- Aranda CP
- Araque JP
- Araya ST
- Arce ATH
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Arling J-H
- Armadans RC
- Armbruster AJ
- Armitage LJ
- Armstrong A
- Arnaez O
- Arnold H
- Artamonov A
- Artoni G
- Artz S
- Asai S
- Asbah N
- Asimakopoulou EM
- Asquith L
- Assamagan K
- Astalos R
- Astigarraga MEP
- Atkin RJ
- Atkinson M
- Atlay NB
- Augsten K
- Avolio G
- Avramidou R
- Ayoub MK
- Azoulay AM
- Azuelos G
- Baas AE
- Baca MJ
- Bachacou H
- Bachas K
- Backes M
- Backman F
- Bagnaia P
- Bahilo JJL
- Bahmani M
- Bahrasemani H
- Bailey AJ
- Bailey VR
- Baines JT
- Bajic M
- Bakalis C
- Baker OK
- Bakker PJ
- Balaji S
- Baldin EM
- Balek P
- Balli F
- Balunas WK
- Balz J
- Banas E
- Bandyopadhyay A
- Banerjee S
- Bannoura AAE
- Barajas CAC
- Barak L
- Barbe WM
- Barberio EL
- Barberis D
- Barbero M
- Barillari T
- Barisits M-S
- Barkeloo J
- Barklow T
- Barnea R
- Barnes SL
- Barnett BM
- Barnett RM
- Barnovska-Blenessy Z
- Baroncelli A
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- Zhemchugov A
- Zheng Z
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- Zobernig G
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- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/09/2019
- Field of study
A search for excited electrons produced in pp collisions at s√ = 13 TeV via a contact interaction qq¯→ee∗ is presented. The search uses 36.1 fb −1 of data collected in 2015 and 2016 by the ATLAS experiment at the Large Hadron Collider. Decays of the excited electron into an electron and a pair of quarks ( eqq¯ ) are targeted in final states with two electrons and two hadronic jets, and decays via a gauge interaction into a neutrino and a W boson ( νW ) are probed in final states with an electron, missing transverse momentum, and a large-radius jet consistent with a hadronically decaying W boson. No significant excess is observed over the expected backgrounds. Upper limits are calculated for the pp→ee∗→eeqq¯ and pp→ee∗→eνW production cross sections as a function of the excited electron mass me∗ at 95% confidence level. The limits are translated into lower bounds on the compositeness scale parameter Λ of the model as a function of me∗ . For me∗<0.5 TeV , the lower bound for Λ is 11 TeV . In the special case of me∗=Λ , the values of me∗<4.8 TeV are excluded. The presented limits on Λ are more stringent than those obtained in previous searches
Measurement of the inclusive isolated-photon cross section at √s = 13 TeV using fb⁻¹ of ATLAS data
- Author
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- Zhemchugov A
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- Åkesson TPA
- Šforza F
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/10/2019
- Field of study
The differential cross section for isolated-photon production in pp collisions is measured at a centre-of-mass energy of 13 TeV with the ATLAS detector at the LHC using an integrated luminosity of 36.1 fb−1. The differential cross section is presented as a function of the photon transverse energy in different regions of photon pseudorapidity. The differential cross section as a function of the absolute value of the photon pseudorapidity is also presented in different regions of photon transverse energy. Next-to-leading-order QCD calculations from Jetphox and Sherpa as well as next-to-next-to-leading-order QCD calculations from Nnlojet are compared with the measurement, using several parameterisations of the proton parton distribution functions. The predictions provide a good description of the data within the experimental and theoretical uncertainties
Observation of electroweak production of two jets and a Z-boson pair
- Author
- Aad G
- Abbott B
- Abbott DC
- Abed Abud A
- Abeling K
- Abhayasinghe DK
- Abidi SH
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- Wanotayaroj C
- Warburton A
- Ward CP
- Wardrope DR
- Warrack N
- Washbrook A
- Watson AT
- Watson MF
- Watts G
- Waugh BM
- Webb AF
- Weber C
- Weber MS
- Weber SA
- Weber SM
- Weidberg AR
- Weingarten J
- Weirich M
- Weiser C
- Wells PS
- Wenaus T
- Wengler T
- Wenig S
- Wermes N
- Werner MD
- Wessels M
- Weston TD
- Whalen K
- Whallon NL
- Wharton AM
- White A
- White AS
- White MJ
- Whiteson D
- Whitmore BW
- Wiedenmann W
- Wiel C
- Wielers M
- Wieseotte N
- Wiglesworth C
- Wiik-Fuchs LAM
- Wilkens HG
- Wilkins LJ
- Williams HH
- Williams S
- Willis C
- Willocq S
- Wingerter-Seez I
- Winkels E
- Winklmeier F
- Winston OJ
- Winter BT
- Wittgen M
- Wobisch M
- Wolf A
- Wolf TMH
- Wolff R
- Wollrath J
- Wolter MW
- Wolters H
- Wong VWS
- Woods NL
- Worm SD
- Wosiek BK
- Woźniak KW
- Wraight K
- Wu SL
- Wu X
- Wu Y
- Wyatt TR
- Wynne BM
- Wölker R
- Xella S
- Xi Z
- Xiao X
- Xiotidis I
- Xu D
- Xu H
- Xu L
- Xu T
- Xu W
- Xu Z
- Xu Z
- Yabsley B
- Yacoob S
- Yajima K
- Yallup DP
- Yamaguchi N
- Yamaguchi Y
- Yamamoto A
- Yamatani M
- Yamazaki T
- Yamazaki Y
- Yan J
- Yan Z
- Yang HJ
- Yang HT
- Yang S
- Yang T
- Yang X
- Yang Y
- Yao W-M
- Yap YC
- Yasu Y
- Yatsenko E
- Ye H
- Ye J
- Ye S
- Yeletskikh I
- Yexley MR
- Yigitbasi E
- Yildiz H Duran
- Yorita K
- Yoshihara K
- Young C
- Young CJS
- Yu J
- Yuan R
- Yue X
- Zaazoua M
- Zabinski B
- Zacharis G
- Zaffaroni E
- Zaitsev AM
- Zakareishvili T
- Zakharchuk N
- Zambito S
- Zanzi D
- Zaripovas DR
- Zeitnitz C
- Zeißner SV
- Zemaityte G
- Zeng JC
- Zenin O
- Zerwas D
- Zgubič M
- Zhang B
- Zhang DF
- Zhang G
- Zhang H
- Zhang J
- Zhang L
- Zhang L
- Zhang M
- Zhang R
- Zhang S
- Zhang X
- Zhang X
- Zhang Y
- Zhang Z
- Zhang Z
- Zhao P
- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
- Zhou B
- Zhou C
- Zhou M
- Zhou MS
- Zhou N
- Zhou Y
- Zhu C
- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zwalinski L
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 09/02/2023
- Field of study
Electroweak symmetry breaking explains the origin of the masses of elementary particles through their interactions with the Higgs field. Besides the measurements of the Higgs boson properties, the study of the scattering of massive vector bosons with spin 1 allows the nature of electroweak symmetry breaking to be probed. Among all processes related to vector-boson scattering, the electroweak production of two jets and a Z-boson pair is a rare and important one. Here we report the observation of this process from proton–proton collision data corresponding to an integrated luminosity of 139 fb−1 recorded at a centre-of-mass energy of 13 TeV with the ATLAS detector at the Large Hadron Collider. We consider two different final states originating from the decays of the Z-boson pair: one containing four charged leptons and another containing two charged leptons and two neutrinos. The hypothesis of no electroweak production is rejected with a statistical significance of 5.7σ, and the measured cross-section for electroweak production is consistent with the Standard Model prediction. In addition, we report cross-sections for inclusive production of a Z-boson pair and two jets for the two final states
Global Impact of the COVID-19 Pandemic on Cerebral Venous Thrombosis and Mortality.
- Author
- Abdalkader M
- Acampa M
- Adami A
- Adebayo TT
- Ahmad S
- Al Hashmi AM
- Alexiev F
- Araújo AP
- Arsovska AA
- Asif KS
- Aykac O
- Ayo-Martin O
- Baek J-H
- Bajrami A
- Banerjee S
- Barrientos Guerra JD
- Bello AH
- Below K
- Bernava G
- Bessa F
- Biller J
- Blasco J
- Bolognese M
- Bonnet T
- Boskamp S
- Brady M
- Brola W
- Carrera E
- Castro P
- Charidimou A
- Chebl A
- Chen H-S
- Chen M
- Chen Y
- Chieh CY
- Churojana A
- Ciopleias B
- Colasurdo M
- Conforto AB
- Cora EA
- Cruz-Culebras A
- Czap A
- Czlonkowska A
- Dao V-P
- de Havenon AH
- de la Cruz Fernández B
- de Oliveira Neves L
- De Raedt S
- de Sousa DA
- Demeestere J
- Dharmadhikari S
- Dimitriade A
- Dippel D
- Dirren E
- Dorobek M
- Drenck N
- Duong HQ
- Dušek P
- Dy JSH
- Dębiec A
- Enzinger C
- Escartín López J
- Eskey CJ
- Falup-Pecurariu C
- Farhoudi M
- Farooqui M
- Fernández Ferro JC
- Ferreira P
- Feske SK
- Fibrich P
- Field TS
- Fonseca L
- Fujita K
- Fukuda H
- García Sánchez MJ
- Gattringer T
- Ghoreishi A
- Gongora-Rivera F
- Goyal N
- Grimmett KB
- Guillán Rodríguez M
- Guzik AK
- Hamid MA
- Haring J
- Hashemilar M
- Haussen DC
- Hernandez-Fernandez F
- Hidalgo RCT
- Hlaváčová H
- Hovingh M
- Hrabanovska E
- Huded V
- Hussain SI
- Imamura H
- Infante-Valenzuela A
- Inoa V
- Jancar N
- Jillela D
- John S
- Jurák L
- Kadlčíková J
- Kalpachki R
- Kan PT
- Karapanayiotides T
- Kargiotis O
- Karlinski MA
- Karpowicz I
- Kelly ME
- Khatri R
- Khoury NN
- Kiley NL
- Klein P
- Klečka L
- Kolikonda MK
- Kosuke M
- Koutroulou I
- Kovář M
- Krastev G
- Krishnan K
- Kristoffersen ES
- Kwan J
- Labuz-Roszak BM
- Lameirinhas da Silva I
- Lara S
- Lasek-Bal A
- Le HV
- Lee JS
- Lee SB
- Lemmens R
- Lenthall R
- Lereis VP
- Lesaine E
- Li G
- Ligot N
- Linfante I
- Lo H
- Longoni M
- Loochtan AI
- Lopez CD
- Lycan S
- Ma A
- Maali L
- Machi P
- Male SS
- Mansour OY
- Martinez-Marino M
- Marto JP
- Martíns RT
- Masoud HE
- Matsumaru Y
- Mbroh J
- Michel P
- Mikulik R
- Min J
- Mohamed GA
- Mohammaden M
- Mohammadzadeh N
- Moniche F
- Montaner J
- Mora Cuervo DL
- Nagel S
- Nagendra S
- Nahab F
- Nalleballe K
- Neumann J
- Nguyen TN
- Nogueira RG
- Nunes AP
- Ohara N
- Omran SS
- Orgeta-Gutierrez S
- Ornello R
- Palaiodimou L
- Paloušková H
- Peeling L
- Perez-Sanchez S
- Pham TN
- Pikula A
- Pinho E Melo T
- Poli S
- Prajapati C
- Punter M
- Qureshi MM
- Radaideh Y
- Rahal F
- Rahman A
- Ramakrishnan P
- Rashid U
- Rayo-Taranto B
- Rebello LC
- Reiner P
- Reiser M
- Renieri L
- Rocha M
- Rodrigues M
- Rodriguez-Kadota L
- Rodríguez Fernández C
- Rodríguez-Vázquez A
- Rohan V
- Rojas-Soto DM
- Roje Bedekovic M
- Romoli M
- Rosenkranz M
- Rouanet F
- Ruland S
- Sabben C
- Sabetay SI
- Sacco S
- Sadeghi Hokmabadi E
- Sakaguchi M
- Sakai K
- Sakai N
- Sakelarova T
- Salmaggi A
- Sangalli D
- Sani AF
- Santiago IB
- Sathya A
- Scozzari D
- Sedova P
- Senadim S
- Seo K-D
- Sheth SA
- Shindo S
- Siegler JE
- Sienkiewicz-Jarosz H
- Silva ML
- Simpkins AN
- Skoda O
- Sobh K
- Sobolewski P
- Sohn S-I
- Sonoda K
- Starosciak AK
- Staszewski J
- Strambo D
- Strbian D
- Sugiura Y
- Sundseth A
- Sunmonu TA
- Suryadevara NC
- SVIN COVID-19 Global COVID Stroke Registry
- Sylaja PN
- Takenobu Y
- Tarlov NE
- Taylor RA
- Tenora D
- Thomalla G
- Ton MD
- Toyoda K
- Tran AD
- Tsivgoulis G
- Uwatoko T
- van Dam-Nolen DHK
- Vanacker P
- Vandervorst F
- Vences MÁ
- Venketasubramanian N
- Voetsch B
- Wahab KW
- Wan Yahya WNN
- Wan Zaidi WA
- Wiącek M
- Wu TY
- Yaghi S
- Yalung PM
- Yamagami H
- Yamamoto N
- Yamamoto R
- Yang S
- Yazawa Y
- Yoshimoto T
- Zaidat OO
- Zhang L
- Zhang L
- Zielinska-Turek J
- Zini A
- Özdemir AÖ
- Čabal M
- Šimůnek L
- Škorňa M
- Šrámek M
- Publication venue
- 'Korean Stroke Society'
- Publication date
- 01/05/2022
- Field of study
BACKGROUND AND PURPOSE: Recent studies suggested an increased incidence of cerebral venous thrombosis (CVT) during the coronavirus disease 2019 (COVID-19) pandemic. We evaluated the volume of CVT hospitalization and in-hospital mortality during the 1st year of the COVID-19 pandemic compared to the preceding year. METHODS: We conducted a cross-sectional retrospective study of 171 stroke centers from 49 countries. We recorded COVID-19 admission volumes, CVT hospitalization, and CVT in-hospital mortality from January 1, 2019, to May 31, 2021. CVT diagnoses were identified by International Classification of Disease-10 (ICD-10) codes or stroke databases. We additionally sought to compare the same metrics in the first 5 months of 2021 compared to the corresponding months in 2019 and 2020 (ClinicalTrials.gov Identifier: NCT04934020). RESULTS: There were 2,313 CVT admissions across the 1-year pre-pandemic (2019) and pandemic year (2020); no differences in CVT volume or CVT mortality were observed. During the first 5 months of 2021, there was an increase in CVT volumes compared to 2019 (27.5%; 95% confidence interval [CI], 24.2 to 32.0; P<0.0001) and 2020 (41.4%; 95% CI, 37.0 to 46.0; P<0.0001). A COVID-19 diagnosis was present in 7.6% (132/1,738) of CVT hospitalizations. CVT was present in 0.04% (103/292,080) of COVID-19 hospitalizations. During the first pandemic year, CVT mortality was higher in patients who were COVID positive compared to COVID negative patients (8/53 [15.0%] vs. 41/910 [4.5%], P=0.004). There was an increase in CVT mortality during the first 5 months of pandemic years 2020 and 2021 compared to the first 5 months of the pre-pandemic year 2019 (2019 vs. 2020: 2.26% vs. 4.74%, P=0.05; 2019 vs. 2021: 2.26% vs. 4.99%, P=0.03). In the first 5 months of 2021, there were 26 cases of vaccine-induced immune thrombotic thrombocytopenia (VITT), resulting in six deaths. CONCLUSIONS: During the 1st year of the COVID-19 pandemic, CVT hospitalization volume and CVT in-hospital mortality did not change compared to the prior year. COVID-19 diagnosis was associated with higher CVT in-hospital mortality. During the first 5 months of 2021, there was an increase in CVT hospitalization volume and increase in CVT-related mortality, partially attributable to VITT
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