657 research outputs found
Oxygen Diffusion Layer on Ti–6Al–4V Alloy: Scratch and Dry Wear Resistance
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/12/2019
- Field of study
Ti–6Al–4V has prominent physical and mechanical properties, which nominate it for various engineering applications. A drawback of the alloy, however, is low scratch and wear resistance, which can be improved by a diffusion treatment. Oxygen diffusion layer (ODL) was produced on Ti–6Al–4V by thermal oxidation at a temperature of 850 °C for 3 and 6 h. Hardness of the samples was measured using a nano-indentation tester. Micro-scratch tests were performed under progressive and constant normal loads of 0–50 N. Dry wear behavior of the samples was studied using a ball-on-disk tribometer. The wear tests were carried out under various normal loads of 7.5, 15 and 30 N for sliding distances of 50, 100, 200 and 300 m using alumina balls as the counterface. The wear and scratch tracks were examined by scanning electron microscope and surface profilometer. The ODL samples showed higher hardness and lower plastic deformation as compared with Ti–6Al–4V. The brittle behavior of the ODL samples caused the formation of cracks and the development of large acoustic signals during scratching under normal loads of 40 and 50 N. The results also showed a higher scratch and wear resistance of the ODL samples compared with Ti–6Al–4V. However, the oxygen diffusion layer formed after 3 h of oxidation showed a sharp increase in the wear rate of the alloy under a normal load of 30 N and a sliding distance of 100 m
Bio-corrosion behaviour of oxygen diffusion layer on Ti-6Al-4V during tribocorrosion
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/11/2017
- Field of study
Oxygen diffusion layers (ODL) on Ti-6Al-4V were produced by thermal oxidation at a temperature of 850 °C for three and six hours. ODL formation resulted in a β-Ti to α-Ti transformation, causing an increase in hardness. Polarization and EIS studies in stagnant and tribocorrosion conditions were performed under normal loads of 3–30 N in a phosphate buffered saline solution. In a stagnant condition, a passive bilayer film was formed on samples, which led to a low icorr. During tribocorrosion, the formation of a protective tribofilm resulted in a higher Ecorr and a lower icorr in ODLs compared with Ti-6Al-4V
Mechanism of tribofilm formation on Ti6Al4V oxygen diffusion layer in a simulated body fluid
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2018
- Field of study
An Oxygen Diffusion Layer (ODL) was generated on the surface of Ti6Al4V alloy by thermal oxidation treatment. In vitro tests for cytotoxicity were performed in the presence of Ti6Al4V and the ODL samples with the culture of G292 Cells and using MTT assay. The results showed a similar cell viability in the presence of the both samples. Wear behavior of Ti6Al4V and the ODL samples was investigated in a phosphate buffered saline solution under a normal load of 30 N at a sliding velocity of 0.1 m/s. The worn surface and subsurface of the samples were studied using SEM, STEM, TEM, XPS, AFM, nano-hardness and surface profilometry. A bio-tribofilm was observed on the worn surface of the ODL. TEM studies showed that the tribofilm had an amorphous structure and contained oxygen and phosphorous as confirmed by XPS and EDS analysis. AFM images also revealed that the tribofilm consisted of compacted fine debris. The formation of the tribofilm on the ODL with higher hardness and strength resulted in a decrease of about 95% in the wear rate compared with Ti6Al4V alloy
Search for new phenomena in final states with an energetic jet and large missing transverse momentum in pp collisions at √ s = 8 TeV with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
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- Acharya BS
- Achkar B
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- Adelman J
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- Akesson TPA
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- Severini H
- Sevova S
- Sfiligoj T
- Sforza F
- Sfyrla A
- Shabalina E
- Shahinian JD
- Shaikh NW
- Shan LY
- Shank JT
- Shapiro M
- Sharma A
- Sharma AS
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- Shaw K
- Shaw SM
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- Sherman AD
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- Shrif EM
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- Sidebo PE
- Sidiropoulou O
- Sidoti A
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- Silva Oliveira M
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- Silverstein SB
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- Sinev NB
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- Sjolin J
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- Suster CJE
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- Swiatlowski M
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- Swirski T
- Sydorenko A
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- Thomsen LA
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- Thorpe EJ
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- Tikhonov YA
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- Torres RET
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- Tsukerman II
- Tsulaia V
- Tsuno S
- Tsybychev D
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- Tudorache A
- Tudorache V
- Tulbure TT
- Tuna AN
- Turchikhin S
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- Usai G
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- Vacek V
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- Vadla KOH
- Vaidya A
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- Vale MAB
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- Valentinetti S
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- Vandewall ER
- Vaniachine A
- Vannicola D
- Vari R
- Varnes EW
- Varni C
- Varol T
- Varouchas D
- Varvell KE
- Vasile ME
- Vasquez GA
- Vazeille F
- Vazquez Furelos D
- Vazquez CJB
- Vazquez JGP
- Veatch J
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- Veloce LM
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- Vermeulen AT
- Vermeulen JC
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- Vetterli MC
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- Viehhauser GHA
- Vigani L
- Villa M
- Vilucchi E
- Vincter MG
- Virdee GS
- Vishwakarma A
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- von Buddenbrock SE
- Von Toerne E
- Vorobel V
- Vorobev K
- Vos M
- Vossebeld JH
- Vozak M
- Vranjes N
- Vrba V
- Vreeswijk M
- Vuillermet R
- Vukotic I
- Wagner P
- Wagner W
- Wagner-Kuhr J
- Wahdan S
- Wahlberg H
- Walbrecht VM
- Walder J
- Walker R
- Walker SD
- Walkowiak W
- Wallangen V
- Wang AM
- Wang AZ
- Wang C
- Wang F
- Wang H
- Wang J
- Wang P
- Wang Q
- Wang R
- Wang R-J
- Wang SM
- Wang W
- Wang WT
- Wang WX
- Wang Y
- Wang Z
- Wanotayaroj C
- Warburton A
- Ward CP
- Wardrope DR
- Warrack N
- Washbrook A
- Watson AT
- Watson MF
- Watts G
- Waugh BM
- Webb AF
- Webb S
- Weber C
- Weber MS
- Weber SA
- Weber SM
- Weidberg AR
- Weingarten J
- Weirich M
- Weiser C
- Wells PS
- Wenaus T
- Wengler T
- Wenig S
- Wermes N
- Werner MD
- Wessels M
- Weston TD
- Whalen K
- Whallon NL
- Wharton AM
- White A
- White AS
- White MJ
- Whiteson D
- Whitmore BW
- Wiedenmann W
- Wiel C
- Wielers M
- Wieseotte N
- Wiglesworth C
- Wiik-Fuchs LAM
- Wilkens HG
- Wilkins LJ
- Williams HH
- Williams S
- Willis C
- Willocq S
- Wingerter-Seez I
- Winkels E
- Winklmeier F
- Winston OJ
- Winter BT
- Wittgen M
- Wobisch M
- Wolf A
- Wolf TMH
- Wolff R
- Wolker R
- Wollrath J
- Wolter MW
- Wolters H
- Wong VWS
- Woods NL
- Worm SD
- Wosiek BK
- Wozniak KW
- Wraight K
- Wu SL
- Wu X
- Wu Y
- Wyatt TR
- Wynne BM
- Xella S
- Xi Z
- Xia L
- Xiao X
- Xiotidis I
- Xu D
- Xu H
- Xu L
- Xu T
- Xu W
- Xu Z
- Yabsley B
- Yacoob S
- Yajima K
- Yallup DP
- Yamaguchi N
- Yamaguchi Y
- Yamamoto A
- Yamatani M
- Yamazaki T
- Yamazaki Y
- Yan Z
- Yang HJ
- Yang HT
- Yang S
- Yang T
- Yang X
- Yang Y
- Yao W-M
- Yap YC
- Yasu Y
- Yatsenko E
- Ye H
- Ye J
- Ye S
- Yeletskikh I
- Yexley MR
- Yigitbasi E
- Yildiz HD
- Yorita K
- Yoshihara K
- Young C
- Young CJS
- Yu J
- Yuan R
- Yue X
- Zaazoua M
- Zabinski B
- Zacharis G
- Zaffaroni E
- Zahreddine J
- Zaitsev AM
- Zakareishvili T
- Zakharchuk N
- Zambito S
- Zanzi D
- Zaripovas DR
- Zeissner S
- Zeitnitz C
- Zemaityte G
- Zeng JC
- Zenin O
- Zenis T
- Zerwas D
- Zgubic M
- Zhang B
- Zhang DF
- Zhang G
- Zhang H
- Zhang J
- Zhang L
- Zhang M
- Zhang R
- Zhang S
- Zhang X
- Zhang Y
- Zhang Z
- Zhao P
- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
- Zhou B
- Zhou C
- Zhou M
- Zhou MS
- Zhou N
- Zhou Y
- Zhu C
- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine N
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zivkovic L
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zu Theenhausen HM
- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
Results of a search for new phenomena in final states with an energetic jet and large missing transverse momentum are reported. The search uses 20.3 fb−1 of √ s = 8 TeV data collected in 2012 with the ATLAS detector at the LHC. Events are required to have at least one jet with pT > 120 GeV and no leptons. Nine signal regions are considered with increasing missing transverse momentum requirements between Emiss T > 150 GeV and Emiss T > 700 GeV. Good agreement is observed between the number of events in data and Standard Model expectations. The results are translated into exclusion limits on models with either large extra spatial dimensions, pair production of weakly interacting dark matter candidates, or production of very light gravitinos in a gauge-mediated supersymmetric model. In addition, limits on the production of an invisibly decaying Higgs-like boson leading to similar topologies in the final state are presente
Protocol of the “As du Coeur” study: a randomized controlled trial on physical activity maintenance in cardiovascular patients
- Author
- A Ghasemi
- AJ Rothman
- AL Marshall
- Alain Fuch
- AM Skene
- B Gardner
- B Gardner
- B Verplanken
- BM Pinto
- Christian Pradier
- D Hedeker
- D Tessier
- Fabienne d’Arripe-Longueville
- HM Dalal
- I Arrigo
- I Boutron
- J Adamson
- J Boiché
- J Daly
- J Dorn
- J-AD Chase
- JA Bennett
- Jean-Jacques Domerego
- Jocelyn Gal
- Karim Tifratene
- KM Smith
- L Zheng
- M Barz
- M Coca Perraillon
- M Zelen
- Marion Fournier
- MM Schlüssel
- NM Brière
- P Lally
- P Lally
- Philippe Mossé
- R Campbell
- RJ Beeken
- RW Bohannon
- Rémi Radel
- SJ Hardcastle
- SM Moore
- SM Phillips
- SM Phillips
- ST O’Keeffe
- U Scholz
- W Wood
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Cardiovascular and metabolic influences of fetal smoke exposure
- Author
- A Al Mamun
- A Burdorf
- A Gasparrini
- A Ghasemi
- A Leclerc
- A Oliveira
- A Thompson
- AA Mahabadi
- AC Holloway
- AG Pires de Sousa
- AH Merry
- AM Toschke
- AS Khashan
- B Clarisse
- B Durmus
- B Durmus
- B Durmus
- BM Kim
- BO Verburg
- C Bexelius
- C Heidemann
- C Hublin
- C Power
- C Sabanayagam
- C Thomas
- CC Geerts
- CC Geerts
- CP Barakat-Haddad
- CS Fox
- CV Breton
- DA Lawlor
- DC Greenwood
- DH Heppe
- DM Hafeman
- DO Mook-Kanamori
- DS Lambers
- DS Thelle
- E Corpeleijn
- E Oken
- E Olafsdottir
- E Regidor
- E Somm
- EM Kho
- F Wiesbauer
- G Andreotti
- G Cohen
- G Pichler
- H El Marroun
- H Taal
- Hanneke Bakker
- HM Gardiner
- J Klenk
- J Pekkanen
- JJM Geelhoed
- JM Rogers
- JP Newnham
- KH Greiser
- KV Blake
- L Ay
- L Balk
- L Dubois
- L Falk
- L Ruan
- L Sartiani
- M Dorr
- M Heys
- M Lampl
- M Osler
- M Touvier
- M Wideroe
- MJ Brion
- MJ Brion
- MS Kramer
- MS Pearce
- MS Setia
- N Dragano
- N Fallah
- N Prabhu
- O Mayer Jr
- P Rzehak
- PD Gluckman
- PH Whincup
- QM Nguyen
- R Bakker
- R Guerrero-Preston
- R Huxley
- R Huxley
- R Morley
- R Quansah
- RP Stolk
- S Aarts
- S Alatupa
- S Holzhauer
- S Walter
- S Ziaei
- SJ Roza
- SJ Roza
- SL White
- SP Ng
- T Chimonas
- T Vikan
- TA Slotkin
- TC Turin
- TJ VanderWeele
- TN Jacobsen
- U Nur
- U Stromberg
- VA Houten van
- VE Murphy
- Vincent W. V. Jaddoe
- VW Jaddoe
- VW Jaddoe
- VW Jaddoe
- VWV Jaddoe
- VWV Jaddoe
- Publication venue
- Springer Netherlands
- Publication date
- 01/01/2011
- Field of study
Many epidemiological studies showed associations of low birth weight with cardiovascular disease, type 2 diabetes and obesity. The associations seem to be consistent and stronger among subjects with a postnatal catch up growth. It has been suggested that developmental changes in response to adverse fetal exposures might lead to changes in the fetal anatomy and physiology. These adaptations may be beneficial for short term, but may lead to common diseases in adulthood. Maternal smoking during pregnancy is one of the most important adverse fetal exposures in Western countries, and is known to be associated with a 150–200 g lower birth weight. An accumulating body of evidence suggests that maternal smoking during pregnancy might be involved in pathways leading to both low birth weight and common diseases, including cardiovascular disease, type 2 diabetes and obesity, in adulthood. In this review, we discuss epidemiological studies focused on the associations of maternal smoking with fetal growth and development and cardiovascular and metabolic disease in later life. We also discuss potential biological mechanisms, and challenges for future epidemiological studies
Genome-wide association and Mendelian randomisation analysis provide insights into the pathogenesis of heart failure
- Author
- Almgren P
- Andersson C
- Aragam KG
- Asselbergs FW
- Backman JD
- Biggs ML
- Bloom HL
- Brandimarto J
- Brown MR
- Buckbinder L
- Cappola TP
- Carey DJ
- Chaffin MD
- Chasman DI
- Chen X
- Chen X
- Chung J
- Chutkow W
- Cook JP
- Dehghan A
- Delgado GE
- Denaxas S
- Doney AS
- Dudley SC
- Dunn ME
- Dörr M
- Ellinor PT
- Engström G
- Esko T
- Fatemifar G
- Felix SB
- Finan C
- Ford I
- Ghanbari M
- Ghasemi S
- Giedraitis V
- Giulianini F
- Gottdiener JS
- Gross S
- Gutmann R
- Guðbjartsson DF
- Haggerty CM
- Hedman ÅK
- Helgadottir A
- Hemingway H
- Henry A
- Hingorani AD
- Holm H
- Holmes MV
- Hyde CL
- Ingelsson E
- Jukema JW
- Kavousi M
- Khaw K-T
- Kleber ME
- Koekemoer A
- Kuchenbaecker K
- Køber L
- Lang CC
- Langenberg C
- Lin H
- Lind L
- Lindgren CM
- London B
- Lotta LA
- Lovering RC
- Luan J
- Lubitz SA
- Lumbers RT
- Magnusson P
- Mahajan A
- Malarstig A
- Margulies KB
- McMurray JJV
- Melander O
- Mordi IR
- Morgan T
- Morley MP
- Morris AD
- Morris AP
- Morrison AC
- März W
- Nagle MW
- Nelson CP
- Newton-Cheh C
- Niessner A
- Niiranen T
- O'Donoghue ML
- Owens AT
- Palmer CNA
- Parry HM
- Perola M
- Portilla-Fernandez E
- Psaty BM
- Regeneron Genetics Center
- Rice KM
- Ridker PM
- Romaine SPR
- Roselli C
- Rotter JI
- Salo P
- Salomaa V
- Samani NJ
- Sattar N
- Shah S
- Shalaby AA
- Smelser DT
- Smith JG
- Smith NL
- Stefansson K
- Stender S
- Stott DJ
- Sveinbjörnsson G
- Svensson P
- Swerdlow DI
- Tammesoo M-L
- Taylor KD
- Teder-Laving M
- Teumer A
- Thorgeirsson G
- Thorsteinsdottir U
- Torp-Pedersen C
- Trompet S
- Tyl B
- Uitterlinden AG
- van der Harst P
- van Setten J
- Vasan RS
- Veluchamy A
- Verweij N
- Visscher PM
- Voors AA
- Völker U
- Wang X
- Wareham NJ
- Waterworth D
- Weeke PE
- Weiss R
- Wiggins KL
- Wilk JB
- Xing H
- Yang J
- Yerges-Armstrong LM
- Yu B
- Zannad F
- Zhao JH
- Ärnlöv J
- Publication venue
- Publication date
- 09/01/2020
- Field of study
Heart failure (HF) is a leading cause of morbidity and mortality worldwide. A small proportion of HF cases are attributable to monogenic cardiomyopathies and existing genome-wide association studies (GWAS) have yielded only limited insights, leaving the observed heritability of HF largely unexplained. We report results from a GWAS meta-analysis of HF comprising 47,309 cases and 930,014 controls. Twelve independent variants at 11 genomic loci are associated with HF, all of which demonstrate one or more associations with coronary artery disease (CAD), atrial fibrillation, or reduced left ventricular function, suggesting shared genetic aetiology. Functional analysis of non-CAD-associated loci implicate genes involved in cardiac development (MYOZ1, SYNPO2L), protein homoeostasis (BAG3), and cellular senescence (CDKN1A). Mendelian randomisation analysis supports causal roles for several HF risk factors, and demonstrates CAD-independent effects for atrial fibrillation, body mass index, and hypertension. These findings extend our knowledge of the pathways underlying HF and may inform new therapeutic strategies
Fetal and infant origins of asthma
- Author
- A Beyerlein
- A Castagnaro
- A Castello
- A Engeland
- A Ghasemi
- A Gugten van der
- A Lebon
- A Lowe
- AD Ngo
- AD Stein
- AJ Lowe
- AJ Oosterhout van
- AL Lux
- AL Wright
- B Durmus
- B Durmus
- B Morseth
- BD Gessner
- BH Strand
- BT Heijmans
- C Crump
- C Minelli
- C Ober
- CA Camargo Jr
- CA Edwards
- CE Lopuhaa
- CL Grainge
- CM Fisk
- CV Breton
- CV Breton
- D Canoy
- D Caudri
- D Gimeno
- D Rothenbacher
- DA Lawlor
- DA Meyers
- DA Stern
- DA Stern
- DB Hancock
- DC Greenwood
- DH Heppe
- DJ Barker
- DJ Barker
- DO Mook-Kanamori
- DS Newburg
- E Baraldi
- E Bouzigon
- E Horak
- E Lannero
- E Mutius von
- E Noguchi
- E Schnabel
- E Sellier
- EN Schultz
- EW Tobi
- FD Gilliland
- FD Martinez
- G Davey Smith
- G Devereux
- G Haland
- G Liu
- G Nagel
- GI Rours
- GI Rours
- GS Pedersen
- H Bakker
- H Bisgaard
- H Bisgaard
- H Slachtova
- HK Reddel
- HM Inskip
- I Diouf
- IK Sundar
- IP Hall
- J Fawke
- J Murdzoska
- J Riedler
- JA Labout
- JH Flory
- JJ Geelhoed
- JJ Jaakkola
- JT McBride
- K Abe
- K Tamakoshi
- K Waller
- KC Pike
- L Ay
- L Elliott
- Liesbeth Duijts
- LL Magnusson
- LM Schachter
- M Erkkola
- M Hallman
- M Heys
- M Huisman
- M Masoli
- M Osler
- M Reuser
- M Siltanen
- MA Ferreira
- MF Moffatt
- MI Asher
- MI Both
- MJ Brion
- MJ Ege
- MJ Whitrow
- ML Tang
- MR Sears
- MR Sears
- N Prabhu
- NJ Friedman
- P Gudmundsson
- P Lagiou
- PD Danziger
- PD Gluckman
- PD Sly
- PM Sleiman
- PW Tennant
- R Gaillard
- R Granell
- R Kumar
- RJ Hancox
- RM Freathy
- RP Steegers-Theunissen
- RS Aysola
- RT Stein
- RW Beasley
- S Alatupa
- S Broughton
- S Cnattingius
- S Illi
- S Panasevich
- S Scholtens
- S Scholtens
- S Timmermans
- S Turner
- SE Haberg
- SE Haberg
- SJ Hepworth
- SJ Kotecha
- SL Friedlander
- SO Shaheen
- SO Shaheen
- SO Shaheen
- SO Shaheen
- SO Shaheen
- SO Shaheen
- SO Shaheen
- SP Peters
- SW Turner
- T Behrens
- TM Ball
- U Hoppu
- VW Jaddoe
- W Shi
- Y Borne
- Y Miyake
- Y Miyake
- Publication venue
- Springer Netherlands
- Publication date
- 01/01/2012
- Field of study
Previous studies have suggested that asthma, like other common diseases, has at least part of its origin early in life. Low birth weight has been shown to be associated with increased risks of asthma, chronic obstructive airway disease, and impaired lung function in adults, and increased risks of respiratory symptoms in early childhood. The developmental plasticity hypothesis suggests that the associations between low birth weight and diseases in later life are explained by adaptation mechanisms in fetal life and infancy in response to various adverse exposures. Various pathways leading from adverse fetal and infant exposures to growth adaptations and respiratory health outcomes have been studied, including fetal and early infant growth patterns, maternal smoking and diet, children’s diet, respiratory tract infections and acetaminophen use, and genetic susceptibility. Still, the specific adverse exposures in fetal and early postnatal life leading to respiratory disease in adult life are not yet fully understood. Current studies suggest that both environmental and genetic factors in various periods of life, and their epigenetic mechanisms may underlie the complex associations of low birth weight with respiratory disease in later life. New well-designed epidemiological studies are needed to identify the specific underlying mechanisms. This review is focused on specific adverse fetal and infant growth patterns and exposures, genetic susceptibility, possible respiratory adaptations and perspectives for new studies
Measurement of the W±Z boson pair-production cross section in pp collisions at √s=13TeV with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdallah J
- Abdinov O
- Abeloos B
- Aben R
- AbouZeid OS
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- Yamanaka T
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- Yao W-M
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- Yeletskikh I
- Yen AL
- Yildirim E
- Yildiz HD
- Yorita K
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- Yoshihara K
- Young C
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- Youssef S
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- Yusuff I
- Zabinski B
- Zaidan R
- Zaitsev AM
- Zakharchuk N
- Zalieckas J
- Zaman A
- Zambito S
- Zanello L
- Zanzi D
- Zeitnitz C
- Zeman M
- Zemla A
- Zeng JC
- Zeng Q
- Zengel K
- Zenin O
- Zenis T
- Zerwas D
- Zhang D
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- Zhang G
- Zhang H
- Zhang J
- Zhang L
- Zhang R
- Zhang R
- Zhang X
- Zhang Z
- Zhao X
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zhong J
- Zhou B
- Zhou C
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- Zhou L
- Zhou M
- Zhou N
- Zhu CG
- Zhu H
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- Zhu Y
- Zhuang X
- Zhukov K
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann C
- Zimmermann S
- Zinonos Z
- Zinser M
- Ziolkowski M
- Zivkovic L
- Zobernig G
- Zoccoli A
- Zu Theenhausen HM
- zur Nedden M
- Zwalinski L
- Publication venue
- Netherlands
- Publication date
- 01/01/2016
- Field of study
published_or_final_versio
Combined measurement of differential and total cross sections in the H → γγ and the H → ZZ* → 4ℓ decay channels at s=13 TeV with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abhayasinghe DK
- Abidi SH
- Abouzeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Adachi S
- Adamczyk L
- Adelman J
- Adersberger M
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
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- Aguilar-Saavedra JA
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- Aielli G
- Akatsuka S
- Akilli E
- Akimov AV
- Alberghi GL
- Albert J
- Albicocco P
- Alconada Verzini MJ
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alhroob M
- Ali B
- Alimonti G
- Alison J
- Alkire SP
- Allaire C
- Allbrooke BMM
- Allen BW
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- Alvarez Gonzalez B
- Alviggi MG
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- Amidei D
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- Anisenkov AV
- Annovi A
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- Anthony MT
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Aperio Bella L
- Arabidze G
- Arai Y
- Araque JP
- Araujo Ferraz V
- Araujo Pereira R
- Arce ATH
- Ardell RE
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Armbruster AJ
- Armitage LJ
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- Arnaez O
- Arnold H
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- Artamonov A
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- Artz S
- Asai S
- Asbah N
- Ashkenazi A
- Asimakopoulou EM
- Asquith L
- Assamagan K
- Astalos R
- Atkin RJ
- Atkinson M
- Atlas Collaboration
- Atlay NB
- Augsten K
- Avolio G
- Avramidou R
- Axen B
- Ayoub MK
- Azuelos G
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- Bachacou H
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- Bahmani M
- Bahrasemani H
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- Baines JT
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- Barreiro Guimarães Da Costa J
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- Brock I
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- Capeans Garrido MDM
- Caprini I
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- Cardillo FC
- Carli I
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- Carlino G
- Carlson BT
- Carminati L
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- Carrillo-Montoya GD
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- Casado MP
- Casha AF
- Casolino M
- Casper DW
- Castelijn R
- Castillo Gimenez V
- Castillo FL
- Castro NF
- Catinaccio A
- Catmore JR
- Cattai A
- Caudron J
- Cavaliere V
- Cavallaro E
- Cavalli D
- Cavalli-Sforza M
- Cavasinni V
- Celebi E
- Ceradini F
- Cerda Alberich L
- Cerqueira AS
- Cerri A
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- Cervelli A
- Cetin SA
- Chafaq A
- Chakraborty D
- Chan SK
- Chan WS
- Chan YL
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- Chapman JD
- Charlton DG
- Chau CC
- Chavez Barajas CA
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- Chekulaev SV
- Chelkov GA
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- De Groot N
- De Jong P
- De La Torre H
- De Lorenzi F
- De Maria A
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- De Salvo A
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- De Santo A
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- Debenedetti C
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- Delmastro M
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- Delsart PA
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- Denysiuk D
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- Zemaityte G
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- Zeng Q
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- Zgubič M
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- Zhao P
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- Zhao Z
- Zhemchugov A
- Zhou B
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- Zhou N
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- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinonos Z
- Zinser M
- Ziolkowski M
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zur Nedden M
- Zwalinski L
- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- Elsevier
- Publication date
- 01/01/2018
- Field of study
A combined measurement of differential and inclusive total cross sections of Higgs boson production is performed using 36.1 fb−1 of 13 TeV proton–proton collision data produced by the LHC and recorded by the ATLAS detector in 2015 and 2016. Cross sections are obtained from measured H→γγ and H→ZZ*(→4ℓ event yields, which are combined taking into account detector efficiencies, resolution, acceptances and branching fractions. The total Higgs boson production cross section is measured to be 57.0−5.9 +6.0 (stat.) −3.3 +4.0 (syst.) pb, in agreement with the Standard Model prediction. Differential cross-section measurements are presented for the Higgs boson transverse momentum distribution, Higgs boson rapidity, number of jets produced together with the Higgs boson, and the transverse momentum of the leading jet. The results from the two decay channels are found to be compatible, and their combination agrees with the Standard Model predictions
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