241 research outputs found
Elevated Stearoyl-CoA Desaturase in Brains of Patients with Alzheimer's Disease
- Author
- AD Attie
- AJ Lengi
- AM Saunders
- B Hutter-Paier
- Carl W. Cotman
- CE Finch
- Daniele Piomelli
- David H. Cribbs
- E Corpeleijn
- E Warensjo
- E Warensjo
- Elizabeth Head
- F Paillard
- G Astarita
- G Astarita
- G Razay
- Giuseppe Astarita
- H Cao
- H Sampath
- HJ Huttunen
- HW Querfurth
- J Wang
- JM Ntambi
- Kwang-Mook Jung
- L Puglielli
- L Whalley
- L Zhang
- LJ Launer
- M Issandou
- M Prince
- MO Grimm
- Nicholas V. DiPatrizio
- P Cohen
- R Mar-Heyming
- R Sanchez-Mejia
- RG Cutler
- RK McNamara
- RK McNamara
- Sarah K. Martin
- Silvana Gaetani
- SP Kalra
- Vitaly Vasilevko
- W Windig
- WJ Lukiw
- WJ Lukiw
- Y Uto
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
The molecular bases of Alzheimer's disease (AD) remain unclear. We used a lipidomic approach to identify lipid abnormalities in the brains of subjects with AD (N = 37) compared to age-matched controls (N = 17). The analyses revealed statistically detectable elevations in levels of non-esterified monounsaturated fatty acids (MUFAs) and mead acid (20:3n-9) in mid-frontal cortex, temporal cortex and hippocampus of AD patients. Further studies showed that brain mRNAs encoding for isoforms of the rate-limiting enzyme in MUFAs biosynthesis, stearoyl-CoA desaturase (SCD-1, SCD-5a and SCD-5b), were elevated in subjects with AD. The monounsaturated/saturated fatty acid ratio (‘desaturation index’) – displayed a strong negative correlation with measures of cognition: the Mini Mental State Examination test (r = −0.80; P = 0.0001) and the Boston Naming test (r = −0.57; P = 0.0071). Our results reveal a previously unrecognized role for the lipogenic enzyme SCD in AD
Nanohybrids of Silver Particles Immobilized on Silicate Platelet for Infected Wound Healing
- Author
- A Agarwal
- CC Chu
- Chia-Yu Chu
- Chien-Wen Chen
- CLJ Fox
- D Van Den Plas
- EJL Lowbury
- F Robert
- Fu-Chuo Peng
- G Gravante
- H Nagato
- HL Su
- HL Su
- Hsing-Chuan Lee
- J Jain
- J Tian
- JE Feugate
- Jiang-Jen Lin
- Jiun-Chiou Wei
- JJ Lin
- JJ Lin
- Johanna M. Brandner
- M Zanetti
- PR Li
- RK Cribbs
- RR Tice
- S Barrientos
- TT Goodman
- W Liu
- WU Huynh
- X Liu
- XJ Wang
- Ying-Fang Chiu
- Publication venue
- Public Library of Science
- Publication date
- 05/06/2012
- Field of study
Silver nanoparticles supported on nanoscale silicate platelets (AgNP/NSP) possess interesting properties, including a large surface area and high biocide effectiveness. The nanohybrid of AgNP/NSP at a weight ratio 7/93 contains 5-nm Ag particles supported on the surface of platelets with dimensions of approximately 80×80×1 nm3. The nanohybrid expresses a trend of lower cytotoxicity at the concentration of 8.75 ppm Ag and low genotoxicity. Compared with conventional silver ions and the organically dispersed AgNPs, the nanohybrid promotes wound healing. We investigated overall wound healing by using acute burn and excision wound healing models. Tests on both infected wound models of mice were compared among the AgNP/NSP, polymer-dispersed AgNPs, the commercially available Aquacel, and silver sulfadiazine. The AgNP/NSP nanohybrid was superior for wound appearance, but had similar wound healing rates, vascular endothelial growth factor (VEGF)-A levels and transforming growth factor (TGF)-β1 expressions to Aquacel and silver sulfadiazine
Autophagy-mediated turnover of Dynamin-related Protein 1
- Author
- A Sandebring
- AK Lutz
- AM Choi
- AS Tsvetkov
- B Westermann
- C Alexander
- C Delettre
- CR Chang
- D Otsuga
- DH Cho
- E Smirnova
- E Smirnova
- G Banker
- H Chen
- H Wang
- Howard S Fox
- HR Waterham
- J Kim
- JT Cribbs
- K Elgass
- K Itoh
- LC Gomes
- M Gomez-Lazaro
- M Komatsu
- M Manczak
- NC Chan
- PH Reddy
- Phillip R Purnell
- RK Dagda
- S Zuchner
- SC Correia
- TJ Wilson
- UP Shirendeb
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Search for squarks and gluinos in events with isolated leptons, jets and missing transverse momentum at s√=8 TeV with the ATLAS detector
- Author
- A Djouadi
- A Kulesza
- A Kulesza
- A Lazopoulos
- A Neveu
- A Neveu
- A Sherstnev
- A. A. E. Bannoura
- A. A. Elliot
- A. A. Grillo
- A. A. Komar
- A. A. Korol
- A. A. Maier
- A. A. Minaenko
- A. A. Nepomuceno
- A. A. Snesarev
- A. A. Solodkov
- A. A. Talyshev
- A. Aloisio
- A. Alonso
- A. Altheimer
- A. Amorim
- A. Andreazza
- A. Angerami
- A. Annovi
- A. Antonov
- A. Artamonov
- A. Ashkenazi
- A. B. Fenyuk
- A. Baroncelli
- A. Bassalat
- A. Basye
- A. Beddall
- A. Bellerive
- A. Bingul
- A. Bocci
- A. Bogouch
- A. Borisov
- A. Boveia
- A. Brandt
- A. Bruni
- A. Buzatu
- A. C. Bundock
- A. C. Daniells
- A. C. König
- A. C. Martyniuk
- A. C. Schaffer
- A. Calandri
- A. Campoverde
- A. Canepa
- A. Castelli
- A. Catinaccio
- A. Cattai
- A. Cerri
- A. Cervelli
- A. Chafaq
- A. Chegwidden
- A. Cheplakov
- A. Chilingarov
- A. Chitan
- A. Ciocio
- A. Clark
- A. Coccaro
- A. Corso-Radu
- A. Cortes-Gonzalez
- A. D. Hawkins
- A. D. Pilkington
- A. Dafinca
- A. Dattagupta
- A. De Salvo
- A. De Santo
- A. Dell’Acqua
- A. Demilly
- A. Dewhurst
- A. Di Ciaccio
- A. Di Domenico
- A. Di Girolamo
- A. Di Mattia
- A. Di Simone
- A. Dimitrievska
- A. Doria
- A. Dudarev
- A. Durglishvili
- A. E. Baas
- A. E. Barton
- A. E. Kiryunin
- A. E. Loevschall-Jensen
- A. Ereditato
- A. Ezhilov
- A. F. Saavedra
- A. Farbin
- A. Farilla
- A. Favareto
- A. Ferrari
- A. Ferrer
- A. Ferretto Parodi
- A. Filipčič
- A. Firan
- A. Fischer
- A. Floderus
- A. Formica
- A. Forti
- A. G. Bogdanchikov
- A. G. Buckley
- A. G. Goussiou
- A. G. Leister
- A. G. Myagkov
- A. G. Olchevski
- A. Gabrielli
- A. Gabrielli
- A. Gemmell
- A. Gershon
- A. Glazov
- A. Gomes
- A. Gorišek
- A. Grohsjean
- A. Haas
- A. Hamilton
- A. Hasib
- A. Henrichs
- A. Hoecker
- A. Hoummada
- A. Hrynevich
- A. I. Butt
- A. I. Etienvre
- A. I. Mincer
- A. Irles Quiles
- A. J. Armbruster
- A. J. Barr
- A. J. Beddall
- A. J. Bevan
- A. J. Brennan
- A. J. Horton
- A. J. Lankford
- A. J. Lowe
- A. J. Tanasijczuk
- A. J. Turvey
- A. Jinaru
- A. Juste Rozas
- A. K. Ciftci
- A. K. Kopp
- A. K. Morley
- A. Kaczmarska
- A. Kamenshchikov
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- A. Karamaoun
- A. Kastanas
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- A. Khanov
- A. Kharlamov
- A. Khodinov
- A. Khomich
- A. Klimentov
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- A. Strandlie
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- A. T. Doyle
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- B. Abbott
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- B. Dechenaux
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- B. E. Lindquist
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- C. Oropeza Barrera
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- W Beenakker
- W Beenakker
- W Beenakker
- W-M. Yao
- W. A. Cribbs
- W. A. Leight
- W. B. Quayle
- W. Bhimji
- W. Blum
- W. Buttinger
- W. C. Fisher
- W. Cleland
- W. Dabrowski
- W. Davey
- W. Edson
- W. Ehrenfeld
- W. F. Mader
- W. Fedorko
- W. H. Bell
- W. H. Hopkins
- W. Iwanski
- W. J. Dearnaley
- W. J. Murray
- W. K. Brooks
- W. Kozanecki
- W. Lampl
- W. Lavrijsen
- W. Liebig
- W. Lukas
- W. M. Thong
- W. Mohr
- W. Okamura
- W. R. Spearman
- W. S. Lockman
- W. Taylor
- W. Trischuk
- W. Van Den Wollenberg
- W. Vandelli
- W. Verkerke
- W. Wagner
- W. Walkowiak
- W. Wiedenmann
- X. Chen
- X. Hu
- X. Ju
- X. Lei
- X. Lou
- X. Ruan
- X. S. Anduaga
- X. Sun
- X. Wang
- X. Wu
- X. Zhang
- X. Zhao
- X. Zhuang
- Y Grossman
- Y. A. Kurochkin
- Y. Abulaiti
- Y. Amaral Coutinho
- Y. Arai
- Y. Azuma
- Y. B. Pan
- Y. Bai
- Y. Benhammou
- Y. Chen
- Y. Cheng
- Y. Coadou
- Y. Davygora
- Y. Enari
- Y. F. Ryabov
- Y. Fang
- Y. Hasegawa
- Y. Heng
- Y. Hernández Jiménez
- Y. Horii
- Y. Huang
- Y. Ikegami
- Y. Ilchenko
- Y. Inamaru
- Y. J. Schnellbach
- Y. Jiang
- Y. Kataoka
- Y. Komori
- Y. Kulchitsky
- Y. Li
- Y. Liu
- Y. Makida
- Y. Minami
- Y. Ming
- Y. Munwes
- Y. Nagai
- Y. Nagasaka
- Y. Nakahama
- Y. Ninomiya
- Y. Okumura
- Y. Oren
- Y. Qin
- Y. Rozen
- Y. S. Gao
- Y. Sakurai
- Y. Sasaki
- Y. Silver
- Y. Smirnov
- Y. Sugaya
- Y. Suzuki
- Y. Takubo
- Y. Tayalati
- Y. Unno
- Y. V. Grishkevich
- Y. Wu
- Y. Yamaguchi
- Y. Yamazaki
- Y. Yang
- Y. Yasu
- Y. Zhao
- Y. Zhu
- Yu. A. Tikhonov
- Z. Barnovska
- Z. D. Greenwood
- Z. Dolezal
- Z. Gecse
- Z. H. Citron
- Z. Hajduk
- Z. Hubacek
- Z. Idrissi
- Z. J. Grout
- Z. Kohout
- Z. L. Ren
- Z. Liang
- Z. M. Karpova
- Z. Marshall
- Z. Rurikova
- Z. V. Krumshteyn
- Z. Vykydal
- Z. Weng
- Z. Yan
- Z. Zhang
- Z. Zhao
- Z. Zinonos
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
The results of a search for supersymmetry in final states containing at least one isolated lepton (electron or muon), jets and large missing transverse momentum with the ATLAS detector at the Large Hadron Collider are reported. The search is based on proton-proton collision data at a centre-of-mass energy s√=8 TeV collected in 2012, corresponding to an integrated luminosity of 20 fb−1. No significant excess above the Standard Model expectation is observed. Limits are set on supersymmetric particle masses for various supersymmetric models. Depending on the model, the search excludes gluino masses up to 1.32 TeV and squark masses up to 840 GeV. Limits are also set on the parameters of a minimal universal extra dimension model, excluding a compactification radius of 1/R c = 950 GeV for a cut-off scale times radius (ΛR c) of approximately 30
Search for pair-produced long-lived neutral particles decaying to jets in the ATLAS hadronic calorimeter in ppcollisions at √s=8TeV
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- Zobernig G
- Zoccoli A
- zur Nedden M
- Zurzolo G
- Zutshi V
- Zwalinski L
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2014
- Field of study
The ATLAS detector at the Large Hadron Collider at CERN is used to search for the decay of a scalar boson to a pair of long-lived particles, neutral under the Standard Model gauge group, in 20.3fb−1of data collected in proton–proton collisions at √s=8TeV. This search is sensitive to long-lived particles that decay to Standard Model particles producing jets at the outer edge of the ATLAS electromagnetic calorimeter or inside the hadronic calorimeter. No significant excess of events is observed. Limits are reported on the product of the scalar boson production cross section times branching ratio into long-lived neutral particles as a function of the proper lifetime of the particles. Limits are reported for boson masses from 100 GeVto 900 GeV, and a long-lived neutral particle mass from 10 GeVto 150 GeV
Search for direct pair production of the top squark in all-hadronic final states in proton-proton collisions at s√=8 TeV with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abdallah J
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- Charfeddine D
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- Youssef S
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- Zaidan R
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- Zambito S
- Zanello L
- Zanzi D
- Zeitnitz C
- Zeman M
- Zemla A
- Zengel K
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- Zhemchugov A
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- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann C
- Zimmermann R
- Zimmermann S
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- Zoccoli A
- zur Nedden M
- Zurzolo G
- Zutshi V
- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2014
- Field of study
The results of a search for direct pair production of the scalar partner to the top quark using an integrated luminosity of 20.1fb−1 of proton–proton collision data at √s = 8 TeV recorded with the ATLAS detector at the LHC are reported. The top squark is assumed to decay via t˜→tχ˜01 or t˜→ bχ˜±1 →bW(∗)χ˜01 , where χ˜01 (χ˜±1 ) denotes the lightest neutralino (chargino) in supersymmetric models. The search targets a fully-hadronic final state in events with four or more jets and large missing transverse momentum. No significant excess over the Standard Model background prediction is observed, and exclusion limits are reported in terms of the top squark and neutralino masses and as a function of the branching fraction of t˜ → tχ˜01 . For a branching fraction of 100%, top squark masses in the range 270–645 GeV are excluded for χ˜01 masses below 30 GeV. For a branching fraction of 50% to either t˜ → tχ˜01 or t˜ → bχ˜±1 , and assuming the χ˜±1 mass to be twice the χ˜01 mass, top squark masses in the range 250–550 GeV are excluded for χ˜01 masses below 60 GeV
Mitochondrial Alterations in PINK1 Deficient Cells Are Influenced by Calcineurin-Dependent Dephosphorylation of Dynamin-Related Protein 1
- Author
- A Beilina
- A Petit
- A Weihofen
- A Wood-Kaczmar
- AC Poole
- Alexandra Beilina
- Angel Cedazo-Mínguez
- Anna Sandebring
- C Zhou
- CA Gautier
- CR Chang
- D Narendra
- D Stojanovski
- David W. Miller
- DC Chan
- DF Babcock
- E Greggio
- EA Reits
- EM Valente
- F Darios
- G Benard
- G Szabadkai
- G Twig
- G Twig
- GM Cereghetti
- H Deng
- H Deng
- H Mortiboys
- H Plun-Favreau
- HH Hoepken
- HM McBride
- Homira Behbahani
- Ibardo Zambrano
- IE Clark
- J Herrington
- J Park
- J Park
- JC Greene
- JJ Palacino
- JL Biedler
- JT Cribbs
- JW Pridgeon
- K Ishii
- K Nowikovsky
- Kelly Jean Thomas
- L Plecita-Hlavata
- M Karbowski
- M Karbowski
- M Priault
- Marcel van der Brug
- Mark R. Cookson
- ME Gegg
- ME Haque
- Megan M. Cleland
- MJ Baptista
- MJ Barsoum
- MMT O'Hare
- MR Cookson
- N Exner
- N Taguchi
- OM de Brito
- R Ichishita
- R Marongiu
- R Rahman-Roblick
- R Zunino
- RD Mills
- Richard F. Cowburn
- Rili Ahmad
- RK Dagda
- RM Canet-Aviles
- S Gandhi
- S Przedborski
- SA Detmer
- T Kitada
- W Liu
- WJ Rettig
- XJ Han
- XJ Han
- Y Yang
- Y Yang
- Yue Feng
- Publication venue
- Public Library of Science
- Publication date
- 01/05/2009
- Field of study
PTEN-induced novel kinase 1 (PINK1) mutations are associated with autosomal recessive parkinsonism. Previous studies have shown that PINK1 influences both mitochondrial function and morphology although it is not clearly established which of these are primary events and which are secondary. Here, we describe a novel mechanism linking mitochondrial dysfunction and alterations in mitochondrial morphology related to PINK1. Cell lines were generated by stably transducing human dopaminergic M17 cells with lentiviral constructs that increased or knocked down PINK1. As in previous studies, PINK1 deficient cells have lower mitochondrial membrane potential and are more sensitive to the toxic effects of mitochondrial complex I inhibitors. We also show that wild-type PINK1, but not recessive mutant or kinase dead versions, protects against rotenone-induced mitochondrial fragmentation whereas PINK1 deficient cells show lower mitochondrial connectivity. Expression of dynamin-related protein 1 (Drp1) exaggerates PINK1 deficiency phenotypes and Drp1 RNAi rescues them. We also show that Drp1 is dephosphorylated in PINK1 deficient cells due to activation of the calcium-dependent phosphatase calcineurin. Accordingly, the calcineurin inhibitor FK506 blocks both Drp1 dephosphorylation and loss of mitochondrial integrity in PINK1 deficient cells but does not fully rescue mitochondrial membrane potential. We propose that alterations in mitochondrial connectivity in this system are secondary to functional effects on mitochondrial membrane potential
Redox regulation of mitochondrial fission, protein misfolding, synaptic damage, and neuronal cell death: potential implications for Alzheimer’s and Parkinson’s diseases
- Author
- A Nikolaev
- AB Knott
- AC Poole
- AK Lutz
- AM Isaacs
- AV Panov
- B Westermann
- BR Hu
- C Delettre
- C Haass
- C Hetz
- C Iadecola
- C Wang
- CR Chang
- D Trotti
- D Yao
- DG Breckenridge
- DH Cho
- DS Bredt
- DS Bredt
- DT Hess
- E Bonfoco
- E Bossy-Wetzel
- E Bossy-Wetzel
- E Bossy-Wetzel
- E Clementi
- EM Schuman
- ER Kandel
- ES Wong
- G Liot
- G Melino
- G Wang
- GE Hardingham
- GM Shankar
- GT Liberatore
- H Chen
- H Li
- H Shimura
- H Wang
- H Yuan
- HH Low
- HM Abu-Soud
- HR Waterham
- HS Chen
- HS Ko
- I Sliskovic
- J Emerit
- J Garthwaite
- J Haendeler
- JA White
- JB Mannick
- JC Greene
- JD Atkin
- JS Beckman
- JS Stamler
- JS Stamler
- JS Stamler
- JS Stamler
- JS Stamler
- JS Stamler
- JT Cribbs
- JW Olney
- JW Olney
- K Hirai
- K Kijima
- KJ Barnham
- KJ Conn
- KK Chung
- KK Chung
- KL Lim
- KN Houk
- KR Pitts
- L Nowak
- L Tenneti
- M Ankarcrona
- M Gu
- M Karbowski
- M Lafon-Cazal
- M Matos
- MF Beal
- MJ Barsoum
- MJ LaVoie
- ML Mayer
- MM Lyles
- MR Cookson
- MR Hara
- MT Lin
- MW Cleeter
- N Exner
- N Kang-Decker
- N Nakamura
- N Taguchi
- P Hantraye
- PE Chabrier
- PH Reddy
- PJ Muchowski
- PP Zhu
- Q Wang
- R Medeiros
- R Ramachandran
- R Yonashiro
- RA Quintanilla
- RJ Youle
- RK Dagda
- RL Frederick
- RV Rao
- S Dimmeler
- S Li
- S Papadia
- S Papadia
- S Przedborski
- S Tanaka
- S Wasiak
- S Zuchner
- SA Lipton
- SA Lipton
- SA Lipton
- SA Lipton
- SJ Baloyannis
- SL Budd
- SR Jaffrey
- Stuart A. Lipton
- SZ Lei
- T Kitada
- T Uehara
- TJ O’Dell
- Tomohiro Nakamura
- VL Dawson
- VS Laar Van
- WD Parker Jr
- WK Kim
- WS Liang
- X Wang
- X Wang
- X Wang
- YB Choi
- Z Gu
- Z Huang
- Z Li
- Publication venue
- Springer US
- Publication date
- 01/01/2010
- Field of study
Normal mitochondrial dynamics consist of fission and fusion events giving rise to new mitochondria, a process termed mitochondrial biogenesis. However, several neurodegenerative disorders manifest aberrant mitochondrial dynamics, resulting in morphological abnormalities often associated with deficits in mitochondrial mobility and cell bioenergetics. Rarely, dysfunctional mitochondrial occur in a familial pattern due to genetic mutations, but much more commonly patients manifest sporadic forms of mitochondrial disability presumably related to a complex set of interactions of multiple genes (or their products) with environmental factors (G × E). Recent studies have shown that generation of excessive nitric oxide (NO), in part due to generation of oligomers of amyloid-β (Aβ) protein or overactivity of the NMDA-subtype of glutamate receptor, can augment mitochondrial fission, leading to frank fragmentation of the mitochondria. S-Nitrosylation, a covalent redox reaction of NO with specific protein thiol groups, represents one mechanism contributing to NO-induced mitochondrial fragmentation, bioenergetic failure, synaptic damage, and eventually neuronal apoptosis. Here, we summarize our evidence in Alzheimer’s disease (AD) patients and animal models showing that NO contributes to mitochondrial fragmentation via S-nitrosylation of dynamin-related protein 1 (Drp1), a protein involved in mitochondrial fission. These findings may provide a new target for drug development in AD. Additionally, we review emerging evidence that redox reactions triggered by excessive levels of NO can contribute to protein misfolding, the hallmark of a number of neurodegenerative disorders, including AD and Parkinson’s disease. For example, S-nitrosylation of parkin disrupts its E3 ubiquitin ligase activity, and thereby affects Lewy body formation and neuronal cell death
Experimental burns: Comparison between silver sulfadiazine and photobiomodulation
- Author
- Andrade AG
- Christiane Pavani
- Coelho RCP
- Cribbs RK
- Cristiane Miranda França
- Daniela de Fátima Teixeira da Silva
- Fabio Lopes
- Gabriela Russo Soeiro Campos
- Gragnani A
- Guelton Hirano Guedes
- Henriques ACG
- Kumar V
- Mariana Teixeira Gomes
- Natália Piccolo
- Núñez SC
- Peck MD
- Piccolo M
- Prazeres SJ
- Prestes M
- Rafael do Nascimento de Lima
- Rahmanian-Schwarz A
- Reinke JM
- Renata A. Belotto
- Ribeiro MS
- Rossi LA
- Silva DFT
- Silva DFT
- Tavares CS
- Taylor EW
- Vidal BC
- Wassermann D
- Wright EH
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
Search for new phenomena in final states with an energetic jet and large missing transverse momentum in pp collisions at root s = 8 TeV with the ATLAS detector (vol 75, 299, 2015)
- Author
- Aad G
- Abbott B
- Abdallah J
- Abdinov O
- Aben R
- Abi B
- Abolins M
- AbouZeid OS
- Abramowicz H
- Abreu H
- Abreu R
- Abulaiti Y
- Acharya BS
- Adam ER
- Adamczyk L
- Adams DL
- Adelman J
- Adomeit S
- Adye T
- Agatonovic-Jovin T
- Aguilar-Saavedra JA
- Agustoni M
- Ahlen SP
- Ahmadov F
- Aielli G
- Akerstedt H
- Akesson TPA
- Akimoto G
- Akimov AV
- Alberghi GL
- Albert J
- Albrand S
- Alconada Verzini MJ
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexander G
- Alexandre G
- Alexopoulos T
- Alhroob M
- Alimonti G
- Alio L
- Alison J
- Allbrooke BMM
- Allison LJ
- Allport PP
- Aloisio A
- Alonso A
- Alonso F
- Alpigiani C
- Altheimer A
- Alviggi MG
- Amako K
- Amaral Coutinho Y
- Amelung C
- Amidei D
- Amor Dos Santos SP
- Amorim A
- Amoroso S
- Amram N
- Amundsen G
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders G
- Anderson KJ
- Andreazza A
- Andrei V
- Anduaga XS
- Angelidakis S
- Angelozzi I
- Anger P
- Angerami A
- Anghinolfi F
- Anisenkov AV
- Anjos N
- Annovi A
- Antonelli M
- Antonov A
- Antos J
- Anulli F
- Aoki M
- Arabidze G
- Arai Y
- Araque JP
- Arce ATH
- Arduh FA
- Arguin J-F
- Argyropoulos S
- Arik M
- Armbruster AJ
- Arnaez O
- Arnal V
- Arnold H
- Arratia M
- Arslan O
- Artamonov A
- Artoni G
- Asai S
- Asbah N
- Ashkenazi A
- Asman B
- Asquith L
- Assamagan K
- Astalos R
- Atkinson M
- Atlay NB
- Auerbach B
- Augsten K
- Aurousseau M
- Avolio G
- Axen B
- Ayoub MK
- Azuelos G
- Baak MA
- Baas AE
- Bacci C
- Bachacou H
- Bachas K
- Backes M
- Backhaus M
- Bagiacchi P
- Bagnaia P
- Bai Y
- Bain T
- Baines JT
- Baker OK
- Balek P
- Balestri T
- Balli F
- Banas E
- Banerjee S
- Bannoura AAE
- Bansil HS
- Barajas CAC
- Barak L
- Baranov SP
- Barberio EL
- Barberis D
- Barbero M
- Barillari T
- Barisonzi M
- Barklow T
- Barlow N
- Barnes SL
- Barnett BM
- Barnett RM
- Barnovska Z
- Baroncelli A
- Barone G
- Barr AJ
- Barreiro F
- Barrera CO
- Bartoldus R
- Barton AE
- Bartos P
- Bassalat A
- Basye A
- Bates RL
- Batista SJ
- Batley JR
- Battaglia M
- Bauce M
- Bauer F
- Bawa HS
- Beacham JB
- Beattie MD
- Beau T
- Beauchemin PH
- Beccherle R
- Bechtle P
- Beck HP
- Becker K
- Becker S
- Beckingham M
- Becot C
- Beddall A
- Beddall AJ
- Bednyakov VA
- Bee CP
- Beemster LJ
- Beermann TA
- Begel M
- Behr K
- Belanger-Champagne C
- Bell PJ
- Bell WH
- Bella G
- Bella LA
- Bellagamba L
- Bellerive A
- Bellomo M
- Belotskiy K
- Beltramello O
- Benary O
- Benchekroun D
- Bender M
- Bendtz K
- Benekos N
- Benhammou Y
- Benjamin DP
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Berge D
- Berger N
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- Bernius C
- Bernlochner FU
- Berry T
- Berta P
- Bertella C
- Bertoli G
- Bertolucci F
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- Besjes GJ
- Bessner M
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- Bevan AJ
- Bhimji W
- Bianchi RM
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- Biebel O
- Bieniek SP
- Biglietti M
- Bilokon H
- Bindi M
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- Bingul A
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- Black JE
- Black KM
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- Blair RE
- Blanchard J-B
- Blanco JE
- Blazek T
- Bloch I
- Blocker C
- Blum W
- Blumenschein U
- Bobbink GJ
- Bobrovnikov VS
- Bocchetta SS
- Bocci A
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- Boddy CR
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- Boeriu OEV
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- Bogdanchikov AG
- Bohm C
- Boisvert V
- Bold T
- Boldea V
- Boldyrev AS
- Bomben M
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- Boonekamp M
- Borisov A
- Borissov G
- Borroni S
- Bortfeldt J
- Bortolotto V
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- Boscherini D
- Bosman M
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- Boudreau J
- Bouffard J
- Bouhova-Thacker EV
- Boumediene D
- Bourdarios C
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- Boutouil S
- Boveia A
- Boyd J
- Boyko IR
- Bozic I
- Bracinik J
- Brandt A
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- Bristow TM
- Britton D
- Brochu FM
- Brock I
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- Brooijmans G
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- Brosamer J
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- Bruncko D
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- Bryngemark L
- Buanes T
- Buat Q
- Bucci F
- Buchholz P
- Buckley AG
- Buda SI
- Budagov IA
- Buehrer F
- Buescher D
- Buescher V
- Bugge L
- Bugge MK
- Bulekov O
- Burckhart H
- Burdin S
- Burghgrave B
- Burke S
- Burmeister I
- Busato E
- Bussey P
- Buszello CP
- Butler JM
- Butt AI
- Buttar CM
- Butterworth JM
- Butti P
- Buttinger W
- Buzatu A
- Bylund OB
- Cabrera Urban S
- Caforio D
- Cakira O
- Calafiura P
- Calandri A
- Calderini G
- Calfayan P
- Caloba LP
- Calvet D
- Calvet S
- Camarda S
- Cameron D
- Caminada LM
- Caminal Armadans R
- Campana S
- Campanelli M
- Campoverde A
- Canale V
- Canepa A
- Cantero J
- Cantrill R
- Cao T
- Caprini I
- Caprini M
- Capua M
- Caputo R
- Cardarelli R
- Carli T
- Carlino G
- Carminati L
- Caron S
- Carquin E
- Carrillo-Montoya GD
- Carter JR
- Carvalho J
- Casadei D
- Casado MP
- Casolino M
- Castaneda-Miranda E
- Castanheira MTD
- Castelli A
- Castillo Gimenez V
- Castillo IS
- Castillo LRF
- Castro NF
- Catastini P
- Catinaccio A
- Catmore JR
- Cattai A
- Cattani G
- Caudron J
- Cavaliere V
- Cavalli D
- Cavalli-Sforza M
- Cavasinni V
- Ceradini F
- Cerio BC
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- Cerqueira AS
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- Cerv M
- Cervelli A
- Cetin SA
- Chafaq A
- Chakraborty D
- Chalupkova I
- Chang P
- Chapleau B
- Chapman JD
- Charfeddine D
- Charlton DG
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- Chekanov S
- Chekulaev SV
- Chelkov GA
- Chelstowska MA
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- Cheng HC
- Cheng Y
- Cheplakov A
- Cheremushkina E
- Chernyatin V
- Cheu E
- Chevalier L
- Chiarella V
- Childers JT
- Chilingarov A
- Chiodini G
- Chisholm AS
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- Chromek-Burckhart D
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- Cribbs WA
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- Croft V
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- Cummings J
- Curatolo M
- Cuthbert C
- Czirr H
- Czodrowski P
- D'Auria S
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- da Costa JBG
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- Dabrowski W
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- Dallaire F
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- De Bruin PHS
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- de Lima DEF
- De Lorenzi F
- De Mendizabal JB
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- Finelli KD
- Fiolhais MCN
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
Results of a search for new phenomena in final states with an energetic jet and large missing transverse momentum are reported. The search uses 20.3 fb−1 of √s=8 TeV data collected in 2012 with the ATLAS detector at the LHC. Events are required to have at least one jet with pT>120 GeV and no leptons. Nine signal regions are considered with increasing missing transverse momentum requirements between EmissT>150 GeV and EmissT>700 GeV. Good agreement is observed between the number of events in data and Standard Model expectations. The results are translated into exclusion limits on models with either large extra spatial dimensions, pair production of weakly interacting dark matter candidates, or production of very light gravitinos in a gauge-mediated supersymmetric model. In addition, limits on the production of an invisibly decaying Higgs-like boson leading to similar topologies in the final state are presented
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