153 research outputs found
Ripples in a pond: Do social work students need to learn about terrorism?
- Author
- B. Dedeoglu
- B. Kelly
- B. Thomas
- B.E. Bride
- C. Candland
- C. Knight
- C. Skehill
- C. Tosone
- D.L. Strug
- E.B. Foa
- E.K. Proctor
- F. Besthorn
- H. Arendt
- H. Itzhaky
- H. Itzhaky
- H. Itzhaky
- I. Ferguson
- I. Sever
- J. Campbell
- J. Campbell
- J. Parker
- J. Parker
- J. Parker
- J. Sweifach
- J.G. Allen
- K. O’Hagan
- K. Popple
- L. Dominelli
- L.P. Donaldson
- M. Drory
- M. Ekengren
- M. Shamai
- M. Smith
- M. Tsui
- M.L. Spitz
- N. Baum
- N. Linzer
- N. Thompson
- P. Hayes
- P. Williams
- P.E. Hodgkinson
- R. Dekel
- R. Dekel
- R. Kapur
- R. Manktelow
- R. Thompson
- R.M. Scurfield
- R.W. Mackelprang
- S. Atran
- S. Cemlyn
- S. Cemlyn
- S. Guru
- S. Ramon
- S. Vertigans
- Publication venue
- 'Whiting & Birch, Ltd.'
- Publication date
- 01/10/2013
- Field of study
In the face of heightened awareness of terrorism, however it is defined, the challenges for social work are legion. Social work roles may include working with the military to ensure the well-being of service-men and women and their families when bereaved or injured, as well as being prepared to support the public within the emergency context of an overt act of terrorism. This paper reviews some of the literature concerning how social work responds to confl ict and terrorism before reporting a smallscale qualitative study examining the views of social work students, on a qualifying programme in the UK, of terrorism and the need for knowledge and understanding as part of their education
Considerations about ocular neoplasia of dogs and cats
- Author
- Alexandre Pinto Ribeiro
- ANDRADE A.L.
- ATTALI-SOUSSAY K.
- BARRIE K.P.
- BARSOTTI G.
- BERNAYS M.E.
- BOLZAN A.A.
- CASSOTIS N.B.
- CHANDLER H.L.
- COLLINS B.K
- COLLINS B.K.
- DITERS R.W.
- DONALDSON D.
- DONALDSON D.
- DUBIELZIG R.R.
- DUBIELZIG R.R.
- Dunia Yisela Trujillo Piso
- ESSON D.
- GILGER B.C.
- GONZALEZ E.M.
- GRAHN B.H.
- HALLSTROM M.
- HARTLEY C.
- JOHNSON B.W.
- José Luiz Laus
- KALISHMAN J.B.
- KLOSTERMAN E.
- KOMAROMY A.M.
- KREHBIEL J.D.
- LIAPIS I.K.
- LORIMIER L.P.
- Luciano Fernandes da Conceição
- MATTON J.S.
- MILLER P.E.
- MONTIANI-FERREIRA F.
- MULTARI D.
- PATNAIK A.K.
- PEREIRA J.S.
- PIRIE C.G.
- RODRIGUES Jr E.F.
- WILLIS A.M.
- WOOG J.
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
A novel formulation of inhaled sodium cromoglicate (PA101) in idiopathic pulmonary fibrosis and chronic cough: a randomised, double-blind, proof-of-concept, phase 2 trial
- Author
- Abel K.
- Adamali H.
- Adeloye D.
- Adeyemi F.
- Adeyemi O.
- Ahmad S.
- Ahmed R.
- Ainsworth M.
- Al-Sheklly B.
- Alamoudi A.
- Aljaroof M.
- Allan L.
- Allen R.
- Alli A.
- Altmann D.
- Anderson D.
- Andrews M.
- Angyal A.
- Antoniades C.
- Arbane G.
- Armour C.
- Armstrong L.
- Armstrong N.
- Arnold D.
- Arnold H.
- Ashworth A.
- Ashworth M.
- Assefa-Kebede H.
- Atkin P.
- Atkins A.
- Atkins H.
- Aul R.
- Avram C.
- Baggott R.
- Baguley D.
- Baillie J. Kenneth
- Baillie J.K.
- Bain S.
- Bakali M.
- Bakau M.
- Baldry E.
- Baldwin D.
- Ballard C.
- Bambrough J.
- Barker R.E.
- Barratt S.
- Barrett F.
- Basu N.
- Batterham R.
- Baxendale H.
- Bayes H.
- Bayley M.
- Beadsworth M
- Beirne P.
- Bell D.
- Bell R.
- Berry C.
- Betts S.
- Bhui K.
- Bishop L.
- Blaikely J.
- Bloomfield C.
- Bloss A.
- Bolger A.
- Bolton C.E.
- Bonnington J.
- Botkai A.
- Bourne C.
- Bourne M.
- Bradley E.
- Bramham K.
- Brear L.
- Breen G.
- Breeze J.
- Briggs A.
- Bright E.
- Brightling C.E.
- Brightling Christopher E.
- Brill S.
- Brindle K.
- Broad L.
- Broome M.
- Brown A
- Brown A
- Brown J.
- Brown J.
- Brown J.S.
- Brown Jeremy S.
- Brown M
- Brown M.
- Brown R.
- Brown V.
- Brugha T
- Brunskill N
- Buch M
- Bularga A
- Bullmore E
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- Burns G
- Busby J
- Buttress A
- Byrne S
- Cairns P
- Calder P.C.
- Calvelo E
- Card B
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- Carson G
- Carter P
- Cavanagh J
- Chalder T
- Chalder Trudie
- Chalmers J.D.
- Chalmers James D.
- Chambers R.C.
- Channon K
- Chapman K
- Charalambou A
- Chaudhuri N
- Checkley A
- Chen J
- Chetham L
- Chilvers E.R.
- Chinoy H
- Chong-James K
- Choudhury G
- Choudhury N
- Chowdhury P
- Chowienczyk P
- Christie C
- Clark C
- Clark D
- Clarke J
- Clift P
- Clohisey S
- Coburn Z
- Cole J
- Coleman C
- Connell D
- Connolly B
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- Crisp P
- Cristiano D
- Crooks M.G.
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- Daines L
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- Deakin B
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- Djukanovic R
- Dobson C
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- Docherty A.B.
- Docherty Annemarie B.
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- Dormand N
- Dougherty A
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- Easom Nicholas
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- Elmer A
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- Fairbairn S
- Fairman A
- Fallon K
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- Finch J
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- Glossop J
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- Gupta A
- Gurram S
- Guthrie E
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- Haggar A
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- Halling-Brown M
- Hamil R
- Hanley N.A.
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- Harrington K
- Harris V
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- Harvey A
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- Haworth J
- Hayday A
- Heaney L.G.
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- Hesselden L
- Hillman T
- Hingorani A
- Hiwot T
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- Hoare A
- Hoare M
- Hogarth P
- Holbourn A
- Holdsworth L
- Holgate D
- Holmes K
- Holroyd-Hind B
- Horsley A
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- Hosseini A
- Hotopf M
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- Hurst J.R.
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- Korszun A
- Kotanidis C
- Koychev I
- Kurupati P
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- Landers G
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- Lawrie A
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- Lenagh R
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- Lingford-Hughes A
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- Magee N
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- Majeed N
- Major E
- Malim M
- Mallison G
- Man W
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- Mangion K
- Mansoori P
- Marciniak S
- Mariveles M
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- Matthews L
- Mayet J
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- McAllister-Williams H
- McArdle A
- McArdle P
- McAulay D
- McAuley D.F.
- McAuley H
- McAuley Hamish
- McCafferty K
- McCann G.P.
- McCann Gerry P.
- McCauley H
- McCourt P
- Mcgarvey L
- McGinness J
- McGovern A
- McGuinness H
- McInnes I.B.
- McIvor E
- McIvor K
- McMahon A
- McMahon M.J.
- McMorrow L
- Mcnally T
- McNarry M
- McQueen A
- McShane H
- Megson S
- Meiring J
- Menzies D
- Michael A
- Milligan L
- Mills N
- Mitchell J
- Mohamed A
- Molyneaux P.L.
- Monteiro W
- Morley A
- Morrison L
- Morriss R
- Morrow A
- Moss A
- Moss A.J.
- Moss P
- Mukaetova-Ladinska E
- Munawar U
- Murali E
- Murira J
- Nassa H
- Neill P
- Neubauer S
- Neubauer Stefan
- Newby D
- Newell H
- Newton Cox A
- Nicholson T
- Nicoll D
- Nolan C.M.
- Noonan M.J.
- Novotny P
- Nunag J
- Nyaboko J
- O'Brien L
- Odell N
- Ogg G
- Olaosebikan O
- Oliver C
- Omar Z
- Openshaw P.J.M.
- Openshaw Peter J. M.
- Osbourne R
- Ostermann M
- Overton C
- Oxton J
- Pacpaco E
- Paddick S
- Papineni P
- Paradowski K
- Pareek M
- Parekh D
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- Parkes M
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- Pathmanathan B
- Pavlides M
- Pearl J.E.
- Peckham D
- Pendlebury J
- Peng Y
- Pennington C
- Peralta I
- Perkins E
- Peto T
- Petousi N
- Petrie J
- Pfeffer P
- Pfeffer Paul
- Phipps J
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- Plekhanova T
- Poinasamy K
- Poinasamy Krisnah
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- Porter J.C.
- Portukhay S
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- Rahman Najib M.
- Ralser M
- Raman B
- Raman Betty
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- Sage E.K.
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- Saunders R
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- Selby N
- Semple M.G.
- Semple Malcolm G.
- Sereno M
- Sereno Marco
- Shah A
- Shah Ajay M.
- Shah K
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- Sharpe C
- Sharpe M
- Shaw V
- Sheikh A
- Sheikh Aziz
- Shevket K
- Shikotra A
- Shikotra Aarti
- Short J
- Siddiqui S
- Sigfrid L
- Simons G
- Simpson J
- Singapuri A
- Singapuri Amisha
- Singh C
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- Singh S.J.
- Singh Sally J.
- Skeemer J
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- Smith I
- Smith J
- Smith L
- Soares M
- Southern D
- Spears M
- Spencer L.G.
- Speranza F
- Stadon L
- Stanel S
- Steiner M
- Stensel D
- Stern M
- Stewart I
- Stockley J
- Stone R
- Storrie A
- Storton K
- Stringer E
- Subbe C
- Sudlow C
- Suleiman Z
- Summers C
- Summersgill C
- Sutherland D
- Sykes D.L.
- Sykes R
- Talbot N
- Tan A.L.
- Taylor A
- Taylor C
- Te A
- Tedd H
- Tee C.J.
- Tench H
- Terry S
- Thackray-Nocera S
- Thaivalappil F
- Thickett D
- Thomas A.K.
- Thomas D
- Thomas D.C.
- Thomas David
- Thompson A.A.R.
- Thompson T
- Thornton T
- Thwaites R.S.
- Tobin M
- Toingson G.F.
- Tong C
- Toshner M
- Toshner Mark
- Touyz R
- Tripp K.A.
- Tunnicliffe E
- Turner E
- Turtle L
- Turton H
- Ugwuoke R
- Upthegrove R
- Valabhji J
- Vellore K
- Wade E
- Wain L.V.
- Wain Louise V.
- Wajero L.O.
- Walder S
- Walker S
- Wall E
- Wallis T
- Walmsley S
- Walsh J.A.
- Walsh S
- Watson E
- Watson J
- Watson L
- Watson L
- Welch C
- Welch H
- Welsh B
- Wessely S
- West S
- Wheeler H
- Whitehead V
- Whitney J
- Whittaker S
- Whittam B
- Wild J
- Wilkins M
- Wilkinson D
- Williams B
- Williams J
- Williams N
- Williams-Howard S.A.
- Willicombe M
- Willis G
- Wilson D
- Wilson I
- Window N
- Witham M
- Wolf-Roberts R
- Woodhead F
- Woods J
- Wootton D
- Worsley J
- Wraith D
- Wright C
- Wright L
- Wright S
- Xie C
- Yasmin S
- Yates T
- Yip K.P.
- Young A
- Young B
- Young S
- Yousuf A
- Yousuf A.J.
- Zawia A
- Zhao B
- Zongo O
- Publication venue
- Elsevier
- Publication date
- 01/03/2013
- Field of study
Background Cough can be a debilitating symptom of idiopathic pulmonary fibrosis (IPF) and is difficult to treat. PA101 is a novel formulation of sodium cromoglicate delivered via a high-efficiency eFlow nebuliser that achieves significantly higher drug deposition in the lung compared with the existing formulations. We aimed to test the efficacy and safety of inhaled PA101 in patients with IPF and chronic cough and, to explore the antitussive mechanism of PA101, patients with chronic idiopathic cough (CIC) were also studied. Methods This pilot, proof-of-concept study consisted of a randomised, double-blind, placebo-controlled trial in patients with IPF and chronic cough and a parallel study of similar design in patients with CIC. Participants with IPF and chronic cough recruited from seven centres in the UK and the Netherlands were randomly assigned (1:1, using a computer-generated randomisation schedule) by site staff to receive PA101 (40 mg) or matching placebo three times a day via oral inhalation for 2 weeks, followed by a 2 week washout, and then crossed over to the other arm. Study participants, investigators, study staff, and the sponsor were masked to group assignment until all participants had completed the study. The primary efficacy endpoint was change from baseline in objective daytime cough frequency (from 24 h acoustic recording, Leicester Cough Monitor). The primary efficacy analysis included all participants who received at least one dose of study drug and had at least one post-baseline efficacy measurement. Safety analysis included all those who took at least one dose of study drug. In the second cohort, participants with CIC were randomly assigned in a study across four centres with similar design and endpoints. The study was registered with ClinicalTrials.gov (NCT02412020) and the EU Clinical Trials Register (EudraCT Number 2014-004025-40) and both cohorts are closed to new participants. Findings Between Feb 13, 2015, and Feb 2, 2016, 24 participants with IPF were randomly assigned to treatment groups. 28 participants with CIC were enrolled during the same period and 27 received study treatment. In patients with IPF, PA101 reduced daytime cough frequency by 31·1% at day 14 compared with placebo; daytime cough frequency decreased from a mean 55 (SD 55) coughs per h at baseline to 39 (29) coughs per h at day 14 following treatment with PA101, versus 51 (37) coughs per h at baseline to 52 (40) cough per h following placebo treatment (ratio of least-squares [LS] means 0·67, 95% CI 0·48–0·94, p=0·0241). By contrast, no treatment benefit for PA101 was observed in the CIC cohort; mean reduction of daytime cough frequency at day 14 for PA101 adjusted for placebo was 6·2% (ratio of LS means 1·27, 0·78–2·06, p=0·31). PA101 was well tolerated in both cohorts. The incidence of adverse events was similar between PA101 and placebo treatments, most adverse events were mild in severity, and no severe adverse events or serious adverse events were reported. Interpretation This study suggests that the mechanism of cough in IPF might be disease specific. Inhaled PA101 could be a treatment option for chronic cough in patients with IPF and warrants further investigation
Introdução ao teorema de Nash e às Branas-mundo
- Author
- Abdalla E.
- Alcaniz J.S.
- Arkani-Hamed N.
- Boyer C.B.
- Burstin C.
- Bustamante M.
- Campbell J.E.
- Carmo M.P.
- Cartan E.
- Donaldson S.K.
- Dunnington W.
- Eisenhart L.P.
- Friedman A.
- Fronsdal C.
- Greene R.
- Gupta S.N.
- Heydary-Fard M.
- Holdom B.
- Holdom B.
- Israel W.
- Jalalzadeh S.
- Janet M.
- Joseph D.W.
- Kaluza T.
- Kant I.
- Lopes J.L.
- Maia M.D.
- Maia M.D.
- Maia M.D.
- Maia M.D.
- Maia M.D.
- Monte E.M.
- Nash J.
- Nash J.
- Nash J.
- Ne'eman Y.
- O'Raifeartaigh L.
- Odon P.I.
- Penrose R.
- Poincaré H.
- Randall L.
- Regge T.
- Riemann B.
- Rosen J.
- Schlaefli L.
- Spradlin M.
- Taubes C.H.
- Weinberg S.
- Weyl H.
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
Dexamethasone intravitreal implant in previously treated patients with diabetic macular edema : Subgroup analysis of the MEAD study
- Author
- A. Antoszyk
- A. Barak
- A. Cruess
- A. Gupta
- A. Gupta
- A. Loewenstein
- A. Lotery
- A. Paranhos
- A. Pollack
- A.B. Majji
- A.F. Sanz
- A.J. Augustin
- A.M. Casella
- A.M..V. Gomes
- B. Foster
- B. Kalvodova
- B. Katz
- B.D. Kuppermann
- B.G. Goldstein
- B.H. Doft
- C. Creuzot-Garcher
- C.C. Awh
- C.R. Baumal
- D. Chechik
- D. Eliott
- D. Glaser
- D. Han
- D. Kecik
- D. Lu
- D. Sandner
- D. Tognetto
- D. Weinberger
- D.J. Perez-Ortiz
- D.M. Brown
- D.S. Boyer
- E. Midena
- E. Wong
- E. Wylegala
- E.H. Souied
- F. Ali
- F. Viola
- F.G. Holz
- F.J. Rodriguez-Alvira
- G. Menon
- G. Ravalico
- G. Sartani
- G. Soubrane
- G. Staurenghi
- G.L. Wing
- G.M. Fox
- H. Cui
- H. Newland
- H.G. Yu
- H.M. Engel
- H.S. Uy
- H.V. De Moraes
- I. Fiser
- I. Klemperer
- I. Loose
- I. Vicha
- I.E. Zimmer-Galler
- J. Acton
- J. Corwin
- J. Ernest
- J. Figueira
- J. Kellaway
- J. Lehr
- J. Miller
- J. Moisseiev
- J. Nemeth
- J. Nichols
- J. Orellana
- J. Peace
- J. Rehak
- J. Romanet
- J. Studnicka
- J. Yan
- J.A. Martinez
- J.L. Ferreira
- J.M. Ruiz-Moreno
- J.R. Ferencz
- J.R. Gonder
- K. Carnevale
- K. Ramaswamy
- K. Rezaei
- K. Sall
- K.U. Bartz-Schmidt
- L. Gopal
- L. Halperin
- L. Morse
- L. Rossetti
- L. Visser
- L.H. Young
- L.J. Ulanski
- L.P. Chong
- M. Bhende
- M. Daniell
- M. Donaldson
- M. Gillies
- M. Leys
- M. Michels
- M. Singer
- M. Suarez-Figueroa
- M. Varano
- M. Veith
- M.B. Parodi
- M.P. De Avila
- N. Orzalesi
- P. Gous
- P. Kaiser
- P. Lanzetta
- P. Massin
- P. Mccartney
- P. Mitchell
- P. Soucek
- P.L. Tsai
- P.M. Falcone
- P.R. Pavan
- R. Belfort
- R. Dreyer
- R. Lattanzio
- R. Navarro
- R. Newsom
- R. Park
- R. Ratnakaram
- R.B. Rosen
- R.G. Devenyi
- R.K. Maturi
- R.S. Katz
- S. Abujamra
- S. Chen
- S. Fekrat
- S. Park
- S. Rizzo
- S. Rose
- S. Sadda
- S. Schmickler
- S. Sheu
- S. Sivaprasad
- S.D. Schwartz
- S.M. Hariprasad
- S.M. Whitcup
- S.P. Garg
- T. Carmichael
- T. Chang
- T.F. Essman
- T.Y. Wong
- U. Menchini
- V. Chong
- W. Lee
- W.R. Freeman
- W.Y. Takahashi
- W.Z. Bridges
- X. Li
- Y. Hashad
- Y.H. Yoon
- Y.R. Sharma
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 30/10/2015
- Field of study
Background: Dexamethasone intravitreal implant 0.7 mg (DEX 0.7) was approved for treatment of diabetic macular edema (DME) after demonstration of its efficacy and safety in the MEAD registration trials. We performed subgroup analysis of MEAD study results to evaluate the efficacy and safety of DEX 0.7 treatment in patients with previously treated DME. Methods: Three-year, randomized, sham-controlled phase 3 study in patients with DME, best-corrected visual acuity (BCVA) of 34.68 Early Treatment Diabetic Retinopathy Study letters (20/200.20/50 Snellen equivalent), and central retinal thickness (CRT) 65300 \u3bcm measured by time-domain optical coherence tomography. Patients were randomized to 1 of 2 doses of DEX (0.7 mg or 0.35 mg), or to sham procedure, with retreatment no more than every 6 months. The primary endpoint was 6515-letter gain in BCVA at study end. Average change in BCVA and CRT from baseline during the study (area-under-the-curve approach) and adverse events were also evaluated. The present subgroup analysis evaluated outcomes in patients randomized to DEX 0.7 (marketed dose) or sham based on prior treatment for DME at study entry. Results: Baseline characteristics of previously treated DEX 0.7 (n = 247) and sham (n=261) patients were similar. In the previously treated subgroup, mean number of treatments over 3 years was 4.1 for DEX 0.7 and 3.2 for sham, 21.5 % of DEX 0.7 patients versus 11.1 % of sham had 6515-letter BCVA gain from baseline at study end (P = 0.002), mean average BCVA change from baseline was +3.2 letters with DEX 0.7 versus +1.5 letters with sham (P = 0.024), and mean average CRT change from baseline was -126.1 \u3bcm with DEX 0.7 versus -39.0 \u3bcm with sham(P < 0.001). Cataract-related adverse events were reported in 70.3 % of baseline phakic patients in the previously treated DEX 0.7 subgroup; vision gains were restored following cataract surgery. Conclusions: DEX 0.7 significantly improved visual and anatomic outcomes in patients with DME previously treated with laser, intravitreal anti-vascular endothelial growth factor, intravitreal triamcinolone acetonide, or a combination of these therapies. The safety profile of DEX 0.7 in previously treated patients was similar to its safety profile in the total study population
Heterozygous Variants in KMT2E Cause a Spectrum of Neurodevelopmental Disorders and Epilepsy.
- Author
- Abou Jamra Rami
- Accogli Andrea
- Ahmed Munaza
- Aitken Stuart
- Akawi Nadia
- Alvi Mohsan,
- Ambridge Kirsty
- Amburgey Kimberly
- Anderlid Britt Marie
- Anjum Uruj
- Archer Hayley
- Armstrong Ruth
- Awada Jana
- Azzarello-Burri Silvia
- Balasubramanian Meena
- Banka Siddharth
- Baralle Diana
- Barnicoat Angela
- Barrett Daniel
- Barrett Jeffrey
- Basinger Alice
- Batstone Paul
- Baty David
- Baxter Samantha
- Bayzetinova Tanya
- Bennett Chris
- Berg Jonathan
- Bernhard Birgitta
- Bevan A. Paul
- Bianchini Claudia,
- Bird Lynne
- Bitner-Glindzicz Maria
- Blair Edward
- Blyth Moira
- Bohanna David
- Bourdon Louise
- Bourn David
- Bradley Lisa
- Brady Angela,
- Brent Simon
- Brewer Carole
- Brunstrom Kate
- Buchert Rebecca
- Bunyan David
- Burn John
- Canham Natalie
- Carré Wilfrid
- Castle Bruce
- Ceulemans Sophia
- Chandler Kate
- Charles Perrine
- Chatzimichali Elena
- Cilliers Deirdre
- Clarke Angus
- Clasper Susan
- Clayton Stephen
- Clayton-Smith Jill
- Clowes Virginia
- Coates Andrea
- Cole Trevor
- Colgiu Irina
- Collins Amanda
- Collinson Morag
- Connell Fiona
- Cooper Nicola
- Cox Helen
- Cresswell Lara
- Cross Gareth
- Crow Yanick
- Culliton Lisa
- Currò Aurora
- Dabir Tabib
- Davidson Rosemarie
- Davies Sally
- de Vries Dylan
- Dean John
- Demurger Florence
- Deshpande Charu
- Devlin Gemma
- Dixit Abhijit
- Dobbie Angus
- Donaldson Alan
- Donnai Dian
- Donnelly Carina
- Donnelly Deirdre
- Douglas Angela
- Douzgou Sofia
- Dowling James,
- Duban-Bedu Bénédicte
- Dubourg Christèle
- Duncan Alexis
- D’Alessandro Mariella
- Eason Jacqueline
- Eiset Saga Elise
- Ellard Sian
- Ellis Ian
- Elmslie Frances
- Escobar Luis
- Evans Karenza
- Everest Sarah
- Faundes Víctor
- Fendick Tina
- Ferrarini Alessandra
- Firth Helen
- Fisher Richard
- Fitzgerald Tomas
- Fitzpatrick David
- Flinter Frances
- Foulds Nicola
- Fry Andrew
- Fryer Alan
- Gardiner Carol
- Gaunt Lorraine
- Ghali Neeti
- Gibbons Richard
- Gill Harinder
- Goodship Judith
- Goudie David
- Gray Emma
- Green Andrew
- Greene Philip
- Greenhalgh Lynn
- Gribble Susan
- Haack Tobias,
- Harrison Lucy
- Harrison Rachel
- Harrison Victoria
- Hashim Mona
- Hawkins Rose
- He Liu
- Heide Solveig
- Helbig Ingo
- Helbig Katherine,
- Hellens Stephen
- Henderson Alex
- Heredia Raul
- Hewitt Sarah
- Hildyard Lucy
- Hobson Emma
- Holden Simon
- Holder Muriel
- Holder Susan
- Hollingsworth Georgina
- Homfray Tessa
- Humphreys Mervyn
- Hurles Matthew,
- Hurst Jane
- Hutton Ben
- Héron Delphine
- Ingram Stuart
- Irving Melita
- Isidor Bertrand
- Islam Lily
- Jackson Andrew
- Jarvis Joanna
- Jenkins Lucy
- Johnson Diana
- Jonasson Amy
- Jones Elizabeth
- Jones Philip,
- Jones Wendy
- Joset Pascal
- Josifova Dragana
- Joss Shelagh
- Kaemba Beckie
- Kaplanis Joanna
- Kazembe Sandra
- Kelsell Rosemary
- Keren Boris
- Kerr Bronwyn
- King Daniel
- Kingston Helen
- Kini Usha
- Kinning Esther
- Kirby Gail
- Kirk Claire
- Kivuva Emma
- Kok Fernando
- Kraus Alison
- Krishnappa Netravathi
- Kroes Hester
- Kumar Dhavendra
- Kumar V.
- Lachlan Katherine
- Lam Wayne
- Lampe Anne
- Langman Caroline
- Lavillaureix Alinoë
- Lees Melissa
- Lim Derek
- Longman Cheryl
- Lowther Gordon
- Lu Xin
- Luria Victor
- Lynch Sally
- Maas Saskia
- Maegawa Gustavo H.B.
- Magee Alex
- Maher Eddy
- Male Alison
- Mansour Sahar
- Marcelis Carlo L.M.
- Mark Paul
- Marks Karen
- Martin Katherine,
- Mason Laura
- Masruha Marcelo
- Maye Una
- McCann Emma
- McConnell Vivienne
- McEntagart Meriel
- McGowan Ruth
- McKay Kirsten
- McKee Shane
- McLaughlin Heather
- McMullan Dominic
- McNerlan Susan
- McRae Jeremy
- McWalter Kirsty
- McWilliam Catherine
- Mehta Sarju
- Melchinger Esther
- Mercimek-Andrews Saadet
- Metcalfe Kay
- Middleton Anna
- Miedzybrodzka Zosia
- Miles Emma
- Mohammed Shehla
- Montgomery Tara
- Moore David
- Morgan Sian,
- Morton Jenny
- Mugalaasi Hood
- Murday Victoria
- Murphy Helen
- Naik Swati
- Nava Caroline
- Nellåker Chris
- Nemeth Andrea
- Nevitt Louise
- Newbury-Ecob Ruth
- Norman Andrew
- Ogilvie Caroline
- Ong Kai-Ren
- O’Donnell-Luria Anne
- O’Shea Rosie
- Pais Lynn
- Park Soo-Mi
- Parker Michael
- Patel Chirag
- Paterson Joan
- Payne Stewart
- Pendziwiat Manuela
- Perrett Daniel
- Person Richard,
- Phipps Julie
- Pilz Daniela,
- Pollard Martin
- Pottinger Caroline
- Poulton Joanna
- Pratt Norman
- Prescott Katrina
- Price Sue
- Pridham Abigail
- Prigmore Elena
- Procter Annie
- Purnell Hellen
- Quarrell Oliver
- Ragge Nicola
- Rahbari Raheleh
- Rajan Diana
- Ramelli Gian Paolo
- Ramos Luiza L.P.
- Randall Josh
- Rankin Julia
- Rauch Anita
- Raymond Lucy
- Reavey Caitlin
- Renieri Alessandra
- Rice Debbie
- Rieß Angelika
- Robert Leema
- Roberts Eileen
- Roberts Gillian
- Roberts Jonathan
- Roberts Paul
- Rodan Lance,
- Ross Alison
- Rosser Elisabeth
- Saggar Anand
- Samant Shalaka
- Sampson Julian
- Sanchez-Valle Amarilis
- Sandford Richard
- Sarkar Ajoy
- Sattar Shifteh
- Saunders Carol
- Schwarz Niklas
- Schweiger Susann
- Scott Richard
- Scurr Ingrid
- Selby Ann
- Seller Anneke
- Sequeira Cheryl
- Shannon Nora
- Sharif Saba
- Shaw-Smith Charles
- Shearing Emma
- Shears Debbie
- Sheridan Eamonn
- Sifrim Alejandro
- Simonic Ingrid
- Singh Tarjinder
- Singzon Roldan
- Skitt Zara
- Smith Audrey
- Smith Kath
- Smithson Sarah
- Smol Thomas
- Sneddon Linda
- Splitt Miranda
- Squires Miranda
- Srour Myriam
- Steindl Katharina
- Stewart Fiona
- Stewart Helen
- Straub Volker
- Suri Mohnish
- Sutton Vivienne
- Sveden Abigail
- Swaminathan Ganesh Jawahar
- Sweeney Elizabeth
- Syrbe Steffen
- Tatton-Brown Kate
- Taylor Cat
- Taylor Jenny
- Taylor Rohan
- Tein Mark
- Telegrafi Aida
- Temple I. Karen
- Thiffault Isabelle
- Thomson Jenny
- Tischkowitz Marc
- Tivey Adrian
- Tomkins Susan
- Torokwa Audrey
- Trauner Doris
- Treacy Becky
- Turner Claire
- Turnpenny Peter
- Tysoe Carolyn
- van der Linden Helio
- van Koningsbruggen Silvana
- Vandersteen Anthony,
- Varghese Vinod
- Vasudevan Pradeep
- Vijayarangakannan Parthiban
- Villard Laurent
- Vogel Ida
- Vogt Julie
- Wakeling Emma
- Wallwark Sarah
- Waters Jonathon
- Weber Astrid
- Weber Yvonne
- Wellesley Diana
- Wentzensen Ingrid
- Whiteford Margo
- Widaa Sara
- Widjaja Elysa
- Wilcox Sarah
- Wilkinson Emily
- Williams Denise
- Williams Nicola
- Wilson Louise
- Wood Jordan
- Woods Geoff
- Wragg Christopher
- Wright Caroline
- Wright Michael
- Yates Laura
- Yau Michael
- Zak Jaroslav
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2019
- Field of study
We delineate a KMT2E-related neurodevelopmental disorder on the basis of 38 individuals in 36 families. This study includes 31 distinct heterozygous variants in KMT2E (28 ascertained from Matchmaker Exchange and three previously reported), and four individuals with chromosome 7q22.2-22.23 microdeletions encompassing KMT2E (one previously reported). Almost all variants occurred de novo, and most were truncating. Most affected individuals with protein-truncating variants presented with mild intellectual disability. One-quarter of individuals met criteria for autism. Additional common features include macrocephaly, hypotonia, functional gastrointestinal abnormalities, and a subtle facial gestalt. Epilepsy was present in about one-fifth of individuals with truncating variants and was responsive to treatment with anti-epileptic medications in almost all. More than 70% of the individuals were male, and expressivity was variable by sex; epilepsy was more common in females and autism more common in males. The four individuals with microdeletions encompassing KMT2E generally presented similarly to those with truncating variants, but the degree of developmental delay was greater. The group of four individuals with missense variants in KMT2E presented with the most severe developmental delays. Epilepsy was present in all individuals with missense variants, often manifesting as treatment-resistant infantile epileptic encephalopathy. Microcephaly was also common in this group. Haploinsufficiency versus gain-of-function or dominant-negative effects specific to these missense variants in KMT2E might explain this divergence in phenotype, but requires independent validation. Disruptive variants in KMT2E are an under-recognized cause of neurodevelopmental abnormalities
Post-acute COVID-19 neuropsychiatric symptoms are not associated with ongoing nervous system injury
- Author
- Abel K.
- Adeloye D.
- Adeyemi O.
- Adrego R.
- Aguilar Jimenez L.A.
- Ahmad S.
- Ahmed R.
- Ahwireng N.
- Ainsworth M.
- Al-Sheklly B.
- Alamoudi A.
- Ali M.
- Aljaroof M.
- All A.M.
- Allan L.
- Allen R.J.
- Allerton L.
- Allsop L.
- Almeida P.
- Altmann D.
- Amoils S.
- Anderson D.
- Anifowose S.
- Antoniades C.
- Arbane G.
- Arias A.
- Armour C.
- Armour C.
- Armstrong L.
- Armstrong N.
- Armstrong N.
- Arnold H.
- Ashish A.
- Ashworth A.
- Ashworth M.
- Aslani S.
- Assefa-Kebede H.
- Atkin C.
- Atkin P.
- Atkins H.
- Aul R.
- Aul R.
- Aung H.
- Austin L.
- Avram C.
- Ayoub A.
- Babores M.
- Baggott R.
- Bagshaw J.
- Baguley D.
- Bailey L.
- Baillie J.K.
- Baillie J.K.
- Baillie J.K.
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- Baillie J.K.
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- Bakali M.
- Bakau M.
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- Ballard C.
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- Bang B.
- Barker R.E.
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- Basu N.
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- Baxendale H.
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- Beadsworth M.
- Beckett P.
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- Publication venue
- Oxford University Press
- Publication date
- 27/12/2023
- Field of study
A proportion of patients infected with severe acute respiratory syndrome coronavirus 2 experience a range of neuropsychiatric symptoms months after infection, including cognitive deficits, depression and anxiety. The mechanisms underpinning such symptoms remain elusive. Recent research has demonstrated that nervous system injury can occur during COVID-19. Whether ongoing neural injury in the months after COVID-19 accounts for the ongoing or emergent neuropsychiatric symptoms is unclear. Within a large prospective cohort study of adult survivors who were hospitalized for severe acute respiratory syndrome coronavirus 2 infection, we analysed plasma markers of nervous system injury and astrocytic activation, measured 6 months post-infection: neurofilament light, glial fibrillary acidic protein and total tau protein. We assessed whether these markers were associated with the severity of the acute COVID-19 illness and with post-acute neuropsychiatric symptoms (as measured by the Patient Health Questionnaire for depression, the General Anxiety Disorder assessment for anxiety, the Montreal Cognitive Assessment for objective cognitive deficit and the cognitive items of the Patient Symptom Questionnaire for subjective cognitive deficit) at 6 months and 1 year post-hospital discharge from COVID-19. No robust associations were found between markers of nervous system injury and severity of acute COVID-19 (except for an association of small effect size between duration of admission and neurofilament light) nor with post-acute neuropsychiatric symptoms. These results suggest that ongoing neuropsychiatric symptoms are not due to ongoing neural injury
Accelarated immune ageing is associated with COVID-19 disease severity
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- Publication venue
- BMC
- Publication date
- 11/01/2024
- Field of study
Background
The striking increase in COVID-19 severity in older adults provides a clear example of immunesenescence, the age-related remodelling of the immune system. To better characterise the association between convalescent immunesenescence and acute disease severity, we determined the immune phenotype of COVID-19 survivors and non-infected controls.
Results
We performed detailed immune phenotyping of peripheral blood mononuclear cells isolated from 103 COVID-19 survivors 3–5 months post recovery who were classified as having had severe (n = 56; age 53.12 ± 11.30 years), moderate (n = 32; age 52.28 ± 11.43 years) or mild (n = 15; age 49.67 ± 7.30 years) disease and compared with age and sex-matched healthy adults (n = 59; age 50.49 ± 10.68 years). We assessed a broad range of immune cell phenotypes to generate a composite score, IMM-AGE, to determine the degree of immune senescence. We found increased immunesenescence features in severe COVID-19 survivors compared to controls including: a reduced frequency and number of naïve CD4 and CD8 T cells (p < 0.0001); increased frequency of EMRA CD4 (p < 0.003) and CD8 T cells (p < 0.001); a higher frequency (p < 0.0001) and absolute numbers (p < 0.001) of CD28−ve CD57+ve senescent CD4 and CD8 T cells; higher frequency (p < 0.003) and absolute numbers (p < 0.02) of PD-1 expressing exhausted CD8 T cells; a two-fold increase in Th17 polarisation (p < 0.0001); higher frequency of memory B cells (p < 0.001) and increased frequency (p < 0.0001) and numbers (p < 0.001) of CD57+ve senescent NK cells. As a result, the IMM-AGE score was significantly higher in severe COVID-19 survivors than in controls (p < 0.001). Few differences were seen for those with moderate disease and none for mild disease. Regression analysis revealed the only pre-existing variable influencing the IMM-AGE score was South Asian ethnicity (
= 0.174, p = 0.043), with a major influence being disease severity (
= 0.188, p = 0.01).
Conclusions
Our analyses reveal a state of enhanced immune ageing in survivors of severe COVID-19 and suggest this could be related to SARS-Cov-2 infection. Our data support the rationale for trials of anti-immune ageing interventions for improving clinical outcomes in these patients with severe disease
Constraints on spin-0 dark matter mediators and invisible Higgs decays using ATLAS 13 TeV pp collision data with two top quarks and missing transverse momentum in the final state
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- Zhemchugov A.
- Zheng Z.
- Zhong D.
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- Zhu C.
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- Zhuang X.
- Zhukov K.
- Zhulanov V.
- Zimine N.I.
- Zinsser J.
- Ziolkowski M.
- Zoccoli A.
- Zoch K.
- Zorbas T.G.
- Zormpa O.
- Zou W.
- Zwalinski L.
- Åkesson T.P.A.
- Öncel ÖO.
- Ženiš T.
- Živković L.
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2023
- Field of study
This paper presents a statistical combination of searches targeting final states with two top quarks and invisible particles, characterised by the presence of zero, one or two leptons, at least one jet originating from a b-quark and missing transverse momentum. The analyses are searches for phenomena beyond the Standard Model consistent with the direct production of dark matter in pp collisions at the LHC, using 139 fb−1 of data collected with the ATLAS detector at a centre-of-mass energy of 13 TeV. The results are interpreted in terms of simplified dark matter models with a spin-0 scalar or pseudoscalar mediator particle. In addition, the results are interpreted in terms of upper limits on the Higgs boson invisible branching ratio, where the Higgs boson is produced according to the Standard Model in association with a pair of top quarks. For scalar (pseudoscalar) dark matter models, with all couplings set to unity, the statistical combination extends the mass range excluded by the best of the individual channels by 50 (25) GeV, excluding mediator masses up to 370 GeV. In addition, the statistical combination improves the expected coupling exclusion reach by 14% (24%), assuming a scalar (pseudoscalar) mediator mass of 10 GeV. An upper limit on the Higgs boson invisible branching ratio of 0.38 (0.30+0.13−0.09) is observed (expected) at 95% confidence level
Study of Z → llγ decays at √s = 8 TeV with the ATLAS detector
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- Abbott B.
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- Åkesson T.P.A.
- Öncel ÖO.
- Ženiš T.
- Živković L.
- Publication venue
- Springer Science and Business Media LLC
- Publication date
- 26/02/2024
- Field of study
This paper presents a study of Z → llγ decays
with the ATLAS detector at the Large Hadron Collider. The
analysis uses a proton–proton data sample corresponding to
an integrated luminosity of 20.2 fb−1 collected at a centre-ofmass energy √s = 8 TeV. Integrated fiducial cross-sections
together with normalised differential fiducial cross-sections,
sensitive to the kinematics of final-state QED radiation, are
obtained. The results are found to be in agreement with stateof-the-art predictions for final-state QED radiation. First
measurements of Z → llγ γ decays are also reported
- …