943 research outputs found
Entrapment neuropathy results in different microRNA expression patterns from denervation injury in rats
- Author
- A Git
- AH Friedman
- AH Williams
- AH Williams
- B John
- CC Chen
- CH Hsieh
- Cheng-Shyuan Rau
- Chia-Jung Lin
- Chia-Jung Wu
- Ching-Hua Hsieh
- D Baek
- E van Rooij
- F Meng
- G Schratt
- H Wang
- H Wang
- HW Hwang
- I Eisenberg
- JA Chan
- JG Zhang
- JJ McCarthy
- Jonathan Chris Jeng
- K Jeyaseelan
- K Pham
- K Yuasa
- LP Lim
- M Jovanovic
- M Selbach
- M Tatsuguchi
- McCarthy J John
- MJ Potthoff
- P Sethupathy
- PK Rao
- Po-Chou Liliang
- RS Pillai
- S Dong
- SE Mackinnon
- SE Mackinnon
- Seng-Feng Jeng
- SF Jeng
- SP Maggi
- SP Maggi
- T Mozaffar
- TE Callis
- Tsu-Hsiang Lu
- TW Nilsen
- V Ambros
- WP Kloosterman
- Y Cheng
- Y Zhao
- Yi-Chun Chen
- Publication venue
- BioMed Central
- Publication date
- 01/01/2010
- Field of study
<p>Abstract</p> <p>Background</p> <p>To compare the microRNA (miRNA) expression profiles in neurons and innervated muscles after sciatic nerve entrapment using a non-constrictive silastic tube, subsequent surgical decompression, and denervation injury.</p> <p>Methods</p> <p>The experimental L4-L6 spinal segments, dorsal root ganglia (DRGs), and soleus muscles from each experimental group (sham control, denervation, entrapment, and decompression) were analyzed using an Agilent rat miRNA array to detect dysregulated miRNAs. In addition, muscle-specific miRNAs (miR-1, -133a, and -206) and selectively upregulated miRNAs were subsequently quantified using real-time reverse transcription-polymerase chain reaction (real-time RT-PCR).</p> <p>Results</p> <p>In the soleus muscles, 37 of the 47 miRNAs (13.4% of the 350 unique miRNAs tested) that were significantly downregulated after 6 months of entrapment neuropathy were also among the 40 miRNAs (11.4% of the 350 unique miRNAs tested) that were downregulated after 3 months of decompression. No miRNA was upregulated in both groups. In contrast, only 3 miRNAs were upregulated and 3 miRNAs were downregulated in the denervated muscle after 6 months. In the DRGs, 6 miRNAs in the entrapment group (miR-9, miR-320, miR-324-3p, miR-672, miR-466b, and miR-144) and 3 miRNAs in the decompression group (miR-9, miR-320, and miR-324-3p) were significantly downregulated. No miRNA was upregulated in both groups. We detected 1 downregulated miRNA (miR-144) and 1 upregulated miRNA (miR-21) after sciatic nerve denervation. We were able to separate the muscle or DRG samples into denervation or entrapment neuropathy by performing unsupervised hierarchal clustering analysis. Regarding the muscle-specific miRNAs, real-time RT-PCR analysis revealed an ~50% decrease in miR-1 and miR-133a expression levels at 3 and 6 months after entrapment, whereas miR-1 and miR-133a levels were unchanged and were decreased after decompression at 1 and 3 months. In contrast, there were no statistical differences in the expression of miR-206 during nerve entrapment and after decompression. The expression of muscle-specific miRNAs in entrapment neuropathy is different from our previous observations in sciatic nerve denervation injury.</p> <p>Conclusions</p> <p>This study revealed the different involvement of miRNAs in neurons and innervated muscles after entrapment neuropathy and denervation injury, and implied that epigenetic regulation is different in these two conditions.</p
A Naturally Occurring Mutation in ropB Suppresses SpeB Expression and Reduces M1T1 Group A Streptococcal Systemic Virulence
- Author
- A Jeng
- Adam J. Ratner
- AH Tart
- Andrew Hollands
- AT Pulliainen
- AV Dmitriev
- AV Dmitriev
- B Demers
- CD Ashbaugh
- CD Ashbaugh
- EL Kaplan
- JD McArthur
- JM Musser
- JN Cole
- JR Carapetis
- JT Buchanan
- M Collin
- MA Chaussee
- Malak Kotb
- Mark J. Walker
- MD Svensson
- MJ Marouni
- MJ Walker
- MJ Walker
- MN Neely
- MN Neely
- MS Chaussee
- MS Chaussee
- MS Chaussee
- MS Rezcallah
- MW Cunningham
- P Nyberg
- P Sumby
- P Sumby
- PP Cleary
- R Raeder
- Ramy K. Aziz
- RG Kansal
- RG Kansal
- Rita Kansal
- RK Aziz
- RK Aziz
- S Beckert
- S Chatellier
- S Lukomski
- SU Kazmi
- U Ringdahl
- Victor Nizet
- W Hynes
- WR Lyon
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2008
- Field of study
Epidemiological studies of group A streptococcus (GAS) have noted an inverse relationship between SpeB expression and invasive disease. However, the role of SpeB in the course of infection is still unclear. In this study we utilize a SpeB-negative M1T1 clinical isolate, 5628, with a naturally occurring mutation in the gene encoding the regulator RopB, to elucidate the role of RopB and SpeB in systemic virulence. Allelic exchange mutagenesis was used to replace the mutated ropB allele in 5628 with the intact allele from the well characterized isolate 5448. The inverse allelic exchange was also performed to replace the intact ropB in 5448 with the mutated allele from 5628. An intact ropB was found to be essential for SpeB expression. While the ropB mutation was shown to have no effect on hemolysis of RBC's, extracellular DNase activity or survival in the presence of neutrophils, strains with the mutated ropB allele were less virulent in murine systemic models of infection. An isogenic SpeB knockout strain containing an intact RopB showed similarly reduced virulence. Microarray analysis found genes of the SpeB operon to be the primary target of RopB regulation. These data show that an intact RopB and efficient SpeB production are necessary for systemic infection with GAS
MicroRNA profiling in ischemic injury of the gracilis muscle in rats
- Author
- A Dharap
- A Wicki
- AM Krichevsky
- BP Lewis
- BP Lewis
- C Yin
- CH Hsieh
- Cheng-Shyuan Rau
- Chia-Jung Lin
- Chia-Jung Wu
- Ching-Hua Hsieh
- D Baek
- DP Bartel
- E van Rooij
- F Meng
- G Meister
- H Wang
- HW Hwang
- I Eisenberg
- IR Fletcher
- J Brennecke
- J Tsang
- JA Chan
- JM Ng
- Jonathan Chris Jeng
- JS Guimbellot
- JS Guimbellot
- K Jeyaseelan
- KA Solonen
- KJ Kapinya
- M Jovanovic
- M Tatsuguchi
- M Yousef
- ME Crosby
- MJ Potthoff
- MO Leonard
- MO Leonard
- N Rajewsky
- NJ Martinez
- PK Shireman
- Po-Chou Liliang
- RS Pillai
- S Dong
- S Patterson
- S Sasaki
- Seng-Feng Jeng
- SF Jeng
- SP Lyden
- ST Carmichael
- TE Callis
- Tsu-Hsiang Lu
- TW Nilsen
- V Ambros
- V Jayaswal
- V Schacht
- WP Kloosterman
- XP Ren
- Y Cheng
- Y Zhao
- Yi-Chun Chen
- Publication venue
- BioMed Central
- Publication date
- 01/01/2010
- Field of study
<p>Abstract</p> <p>Background</p> <p>To profile the expression of microRNAs (miRNAs) and their potential target genes in the gracilis muscles following ischemic injury in rats by monitoring miRNA and mRNA expression on a genome-wide basis.</p> <p>Methods</p> <p>Following 4 h of ischemia and subsequent reperfusion for 4 h of the gracilis muscles, the specimens were analyzed with an Agilent rat miRNA array to detect the expressed miRNAs in the experimental muscles compared to those from the sham-operated controls. Their expressions were subsequently quantified by real-time reverse transcription polymerase chain reaction (real-time RT-PCR) to determine their expression pattern after different durations of ischemia and reperfusion. In addition, the expression of the mRNA in the muscle specimens after 4 h of ischemia and reperfusion for 1, 3, 7, and 14 d were detected with the Agilent Whole Rat Genome 4 × 44 k oligo microarray. A combined approach using a computational prediction algorithm that included miRanda, PicTar, TargetScanS, MirTarget2, RNAhybrid, and the whole genome microarray experiment was performed by monitoring the mRNA:miRNA association to identify potential target genes.</p> <p>Results</p> <p>Three miRNAs (miR-21, miR-200c, and miR-205) of 350 tested rat miRNAs were found to have an increased expression in the miRNA array. Real-time RT-PCR demonstrated that, with 2-fold increase after 4 h of ischemia, a maximum 24-fold increase at 7 d, and a 7.5-fold increase at 14 d after reperfusion, only the miR-21, but not the miR-200c or miR-205 was upregulated throughout the experimental time. In monitoring the target genes of miR-21 in the expression array at 1, 3, 7, 14 d after reperfusion, with persistent expression throughout the experiment, we detected the same 4 persistently downregulated target genes (<it>Nqo1</it>, <it>Pdpn</it>, <it>CXCL3</it>, and <it>Rad23b</it>) with the prediction algorithms miRanda and RNAhybrid, but no target gene was revealed with PicTar, TargetScanS, and MirTarget2.</p> <p>Conclusions</p> <p>This study revealed 3 upregulated miRNAs in the gracilis muscle following ischemic injury and identified 4 potential target genes of miR-21 by examining miRNAs and mRNAs expression patterns in a time-course fashion using a combined approach with prediction algorithms and a whole genome expression array experiment.</p
Bacteremic community-acquired pneumonia due to Klebsiella pneumoniae: Clinical and microbiological characteristics in Taiwan, 2001-2008
- Author
- Publication venue
- BioMed Central
- Publication date
- 01/01/2010
- Field of study
<p>Abstract</p> <p>Background</p> <p><it>Klebsiella pneumoniae </it>is the major cause of community-acquired pyogenic infections in Taiwan. This retrospective study evaluated the clinical and microbiological characteristics of bacteremic community-acquired pneumonia due to <it>K. pneumoniae </it>in Taiwanese adults.</p> <p>Methods</p> <p>The clinical characteristics of bacteremic community-acquired pneumonia (CAP) in adults due to <it>K. pneumoniae </it>were compared to those of adults with bacteremic CAP due to <it>Streptococcus pneumoniae </it>at a tertiary medical center in Taiwan from 2001-2008. Risk factors for mortality of bacteremic CAP due to <it>K. pneumoniae </it>were analyzed. All clinical isolates of <it>K. pneumoniae </it>were examined for capsular serotypes, hypermucoviscosity phenotype, aerobactin and <it>rmpA </it>gene.</p> <p>Results</p> <p><it>K. pneumoniae </it>was the dominant cause of bacteremic CAP and was associated with a more fulminant course and a worse prognosis than bacteremic CAP due to <it>Streptococcus pneumoniae</it>. Initial presentation with septic shock and respiratory failure were independent risk factors for both early and total mortality. Serotype K1 and K2 comprised around half of all isolates. There were no significant differences in the clinical characteristics of patients with bacteremic CAP due to K1/K2 and non-K1/K2 isolates. Hypermucoviscosity phenotype as well as the aerobactin and <it>rmpA </it>genes were highly prevalent in the <it>K. pneumoniae </it>isolates.</p> <p>Conclusions</p> <p><it>K. pneumoniae </it>continued to be the dominant cause of bacteremic CAP in Taiwanese adults during 2001-2008. Initial presentation with septic shock and respiratory failure were independent risk factors for both early and total mortality from <it>K. pneumoniae </it>bacteremic CAP. Serotypes K1/K2 comprised around half of all isolates, but did not predispose patients to a poor clinical outcome. Physicians should be aware of the poor prognosis of any patient with bacteremic <it>K. pneumoniae </it>CAP and monitor these patients more closely.</p
DNase Sda1 Allows Invasive M1T1 Group A Streptococcus to Prevent TLR9-Dependent Recognition
- Author
- A Dalpke
- A Jeng
- A Norrby-Teglund
- AA Navarini
- AJ Talati
- AK Yi
- AM Krieg
- Annelies S. Zinkernagel
- AS Zinkernagel
- AS Zinkernagel
- B Albiger
- B Qiao
- DA Hume
- E Karayel
- ET Berends
- Federica Andreoni
- Frank R. DeLeo
- G Plitas
- G Uze
- G Xiao
- H Hemmi
- H Mulcahy
- JA Gold
- JJ Gao
- JN Cole
- JR Carapetis
- JT Buchanan
- K Beiter
- L Johansson
- LW Wannamaker
- LW Wannamaker
- MJ Sweet
- MJ Walker
- MW Cunningham
- N Gratz
- O Goldmann
- OA Chow
- P Hruz
- P Sumby
- PP Cleary
- Reto A. Schuepbach
- RK Aziz
- S Chatellier
- Satoshi Uchiyama
- TL Roberts
- U Bhan
- V Brinkmann
- V Nizet
- Victor Nizet
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2012
- Field of study
Group A Streptococcus (GAS) has developed a broad arsenal of virulence factors that serve to circumvent host defense mechanisms. The virulence factor DNase Sda1 of the hyperinvasive M1T1 GAS clone degrades DNA-based neutrophil extracellular traps allowing GAS to escape extracellular killing. TLR9 is activated by unmethylated CpG-rich bacterial DNA and enhances innate immune resistance. We hypothesized that Sda1 degradation of bacterial DNA could alter TLR9-mediated recognition of GAS by host innate immune cells. We tested this hypothesis using a dual approach: loss and gain of function of DNase in isogenic GAS strains and presence and absence of TLR9 in the host. Either DNA degradation by Sda1 or host deficiency of TLR9 prevented GAS induced IFN-α and TNF-α secretion from murine macrophages and contributed to bacterial survival. Similarly, in a murine necrotizing fasciitis model, IFN-α and TNF-α levels were significantly decreased in wild type mice infected with GAS expressing Sda1, whereas no such Sda1-dependent effect was seen in a TLR9-deficient background. Thus GAS Sda1 suppressed both the TLR9-mediated innate immune response and macrophage bactericidal activity. Our results demonstrate a novel mechanism of bacterial innate immune evasion based on autodegradation of CpG-rich DNA by a bacterial DNase
Skin and soft-tissue infections in suburban primary care: epidemiology of methicillin-resistant Staphylococcus aureus and observations on abscess management
- Author
- A Jeng
- A Robicsek
- AL Cohen
- B Hota
- BW Frazee
- C Liu
- Catherine Ley
- Charles Weiss
- DA Talan
- DM Young
- EM Begier
- EV Barnes
- F Mollema
- G Turabelidze
- GJ Moran
- GJ Moran
- GR Schmitz
- HF Chambers
- JD Szumowski
- JJ Ruhe
- JK Johnson
- John Boggs
- KM Campbell
- LG Miller
- LG Miller
- LG Miller
- M Duong
- MC Lee
- MD King
- MJ Clancy
- P Bernard
- Peggy Kaminsky
- PM Rajendran
- PS Pannaraj
- RJ Gorwitz
- S Iyer
- SK Fridkin
- SL Davis
- TS Naimi
- Publication venue
- BioMed Central
- Publication date
- 01/01/2011
- Field of study
Measurement of the cross-section of high transverse momentum vector bosons reconstructed as single jets and studies of jet substructure in pp collisions at √s = 7 TeV with the ATLAS detector
- Author
- Aad G
- Abajyan T
- Abbott B
- Abdallah J
- Abdinov O
- Aben R
- Abi B
- Abolins M
- AbouZeid OS
- Abramowicz H
- Abreu H
- Abulaitia Y
- Acharya BS
- Adamczyk L
- Adams DL
- Addy TN
- Adelman J
- Adomeit S
- Adye T
- Agatonovic-Jovin T
- Aguilar-Saavedra JA
- Agustoni M
- Ahlen SP
- Ahmadov F
- Aielli G
- Akesson TP
- Akimoto G
- Akimov AV
- Albert J
- Albrand S
- Alconada Verzini MJ
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexander G
- Alexandre G
- Alexopoulos T
- Alhroob M
- Alimonti G
- Alio L
- Alison J
- Allbrooke BMM
- Allison LJ
- Allport PP
- Allwood-Spiers SE
- Almond J
- Aloisio A
- Alon R
- Alonso A
- Alonso F
- Alpigiani C
- Altheimer A
- Alviggi MG
- Amako K
- Amelung C
- Ammosov VV
- Amor Dos Santos SP
- Amorim A
- Amoroso S
- Amram N
- Amundsen G
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders G
- Anderson KJ
- Andreazza A
- Andrei V
- Anduaga XS
- Angelidakis S
- Anger P
- Angerami A
- Anghinolfi F
- Anh TV
- Anisenkov AV
- Anjos N
- Annovi A
- Antonaki A
- Antonelli M
- Antonov A
- Antos J
- Anulli F
- Aoki M
- Apolle R
- Arabidze G
- Aracena I
- Arai Y
- Arce ATH
- Arguin J-F
- Argyropoulos S
- Arik E
- Arik M
- Armbruster AJ
- Arnaez O
- Arnal V
- Arslan O
- Artamonov A
- Artoni G
- Asai S
- Asbah N
- Ask S
- Asman B
- Asquith L
- Assamagan K
- Astalos R
- Astbury A
- Atkinson M
- Atlay NB
- Auerbach B
- Auge E
- Augsten K
- Aurousseau M
- Avolio G
- Azuelos G
- Azuma Y
- Baak MA
- Bacci C
- Bach AM
- Bachacou H
- Bachas K
- Backes M
- Backhaus M
- Badescua E
- Bagiacchi P
- Bagnaia P
- Bai Y
- Bailey DC
- Bain T
- Baines JT
- Baker OK
- Baker S
- Balek P
- Balli F
- Banas E
- Banerjee S
- Bangert A
- Bansal V
- Bansil HS
- Barajas CAC
- Barak L
- Baranov SP
- Barber T
- Barberio EL
- Barberis D
- Barbero M
- Barillari T
- Barisonzi M
- Barklow T
- Barlow N
- Barnett BM
- Barnett RM
- Baroncelli A
- Barone G
- Barr AJ
- Barreiro F
- Barrera CO
- Bartoldus R
- Barton AE
- Bartos P
- Bartsch V
- Bassalat A
- Basye A
- Bates RL
- Batkova L
- Batley JR
- Battistin M
- Bauer F
- Bawa HS
- Beau T
- Beauchemin PH
- Beccherle R
- Bechtle P
- Beck HP
- Becker K
- Becker S
- Beckingham M
- Beddall A
- Beddall AJ
- Bedikian S
- Bednyakov VA
- Bee CP
- Beemster LJ
- Beermann TA
- Begel M
- Behr K
- Belanger-Champagne C
- Belenguer MJ
- Bell PJ
- Bell WH
- Bella G
- Bella LA
- Bellagamba L
- Bellerive A
- Bellomo M
- Belloni A
- Belotskiy K
- Beltramello O
- Benary O
- Benchekroun D
- Bendtz K
- Benekos N
- Benhammou Y
- Benjamin DP
- Bensinger JR
- Benslama K
- Bentvelsen S
- Berge D
- Berger N
- Berghaus F
- Berglund E
- Beringer J
- Bernard C
- Bernat P
- Bernius C
- Bernlochner FU
- Berry T
- Berta P
- Bertella C
- Bertolucci F
- Besana MI
- Besjes GJ
- Bessidskaia O
- Besson N
- Betancourt C
- Bethke S
- Bhimji W
- Bianchi RM
- Bianchini L
- Bianco M
- Biebel O
- Bieniek SP
- Bierwagen K
- Biesiada J
- Biglietti M
- Bilokon H
- Bindi M
- Binet S
- Bingul A
- Bini C
- Black CW
- Black JE
- Black KM
- Blackburn D
- Blair RE
- Blanchard J-B
- Blazeka T
- Bloch I
- Blocker C
- Blum W
- Blumenschein U
- Bobbink GJ
- Bobrovnikov VS
- Bocchetta SS
- Bocci A
- Boddy CR
- Boehler M
- Boek J
- Boek TT
- Boeriu OEV
- Bogaerts JA
- Bogdanchikov AG
- Bogouch A
- Bohm C
- Bohm J
- Boisvert V
- Bold T
- Boldea V
- Boldyrev AS
- Bolnet NM
- Bomben M
- Bona M
- Boonekamp M
- Borisov A
- Borissov G
- Borri M
- Borroni S
- Bortfeldt J
- Bortolotto V
- Bos K
- Boscherini D
- Bosman M
- Boterenbrood H
- Boudreau J
- Bouffard J
- Bouhova-Thacker EV
- Boumediene D
- Bourdarios C
- Bousson N
- Boutouil S
- Boveia A
- Boyd J
- Boyko IR
- Bozovic-Jelisavcic I
- Bracinik J
- Branchini P
- Brandt A
- Brandt G
- Brandta O
- Bratzler U
- Brau B
- Brau JE
- Braun HM
- Brazzale SF
- Brelier B
- Brendlinger K
- Brennan AJ
- Brenner R
- Bressler S
- Bristow K
- Bristow TM
- Britton D
- Brochu FM
- Brock I
- Brock R
- Bromberg C
- Bronner J
- Brooijmans G
- Brooks T
- Brooks WK
- Brosamer J
- Brost E
- Brown G
- Brown J
- Bruncko D
- Bruneliere R
- Brunet S
- Bruni A
- Bruni G
- Bruschi M
- Bryngemark L
- Buanes T
- Buat Q
- Bucci F
- Buchholz P
- Buckingham RM
- Buckley AG
- Buda SI
- Budagov IA
- Buehrer F
- Buescher V
- Bugge L
- Bugge MK
- Bulekov O
- Bundock AC
- Burckhart H
- Burdin S
- Burghgrave B
- Burke S
- Burmeister I
- Busato E
- Bussey P
- Bustos ACF
- Buszello CP
- Butler B
- Butler JM
- Butt AI
- Buttar CM
- Butterworth JM
- Buttinger W
- Buzatu A
- Byszewski M
- Cabrera Urban S
- Caforio D
- Cakir IT
- Cakir O
- Calafiura P
- Calderini G
- Calfayan P
- Calkins R
- Caloba LP
- Caloi R
- Calvet D
- Calvet S
- Cameron D
- Camillocci ES
- Caminada LM
- Caminal Armadans R
- Campana S
- Campanelli M
- Campoverde A
- Canale V
- Canelli F
- Canepa A
- Cantero J
- Cantrill R
- Cao T
- Caprini I
- Caprini M
- Capua M
- Caputo R
- Cardarelli R
- Carli T
- Carlino G
- Carminati L
- Caron S
- Carquin E
- Carrillo-Montoya GD
- Carter JR
- Carvalho J
- Casadei D
- Casado MP
- Castaneda-Miranda E
- Castanheira MTD
- Castelli A
- Castillo Gimenez V
- Castillo IS
- Castillo LRF
- Castillo LRF
- Castro NF
- Catastini P
- Catinaccio A
- Catmore JR
- Cattai A
- Cattani G
- Caughron S
- Cavaliere V
- Cavalli D
- Cavalli-Sforza M
- Cavasinni V
- Ceradini F
- Cerio B
- Cerny K
- Cerqueira AS
- Cerri A
- Cerrito L
- Cerutti F
- Cerv M
- Cervelli A
- Cetin SA
- Chafaq A
- Chakraborty D
- Chalupkova I
- Chan K
- Chang P
- Chapleau B
- Chapman JD
- Charfeddine D
- Charlton DG
- Cheatham S
- Chekanov S
- Chekulaev SV
- Chelkov GA
- Chelstowska MA
- Chen C
- Chen H
- Chen K
- Chen L
- Chen S
- Chen X
- Chen Y
- Cheng HC
- Cheng Y
- Cheplakov A
- Chernyatin V
- Cheu E
- Chevalier L
- Chiarella V
- Chiefari G
- Childers JT
- Chilingarov A
- Chiodini G
- Chisholm AS
- Chislett RT
- Chitan A
- Chizhov MV
- Chouridou S
- Chow BKB
- Christidi IA
- Chromek-Burckhart D
- Chu ML
- Chudoba J
- Ciapetti G
- Ciftci AK
- Ciftci R
- Cinca D
- Cindro V
- Ciocio A
- Cirkovic P
- Citron ZH
- Citterio M
- Ciubancan M
- Clark A
- Clark PJ
- Clarke RN
- Cleland W
- Clemens JC
- Clement B
- Clement C
- Coadou Y
- Cobal M
- Coccaro A
- Cochran J
- Codina EP
- Coffey L
- Cogan JG
- Coggeshall J
- Cole B
- Cole S
- Colijn AP
- Collaboration ATLAS
- Collins-Tooth C
- Collot J
- Colombo T
- Colon G
- Compostella G
- Conde Muino P
- Coniavitis E
- Conidi MC
- Connelly IA
- Consonni SM
- Consorti V
- Constantinescu S
- Conta C
- Conti G
- Conventi F
- Cooke M
- Cooper BD
- Cooper-Sarkar AM
- Cooper-Smith NJ
- Copic K
- Cornelissen T
- Corradi M
- Correia AMH
- Corriveau F
- Corso-Radu A
- Cortes-Gonzalez A
- Cortiana G
- Costa G
- Costa MJ
- Costanzo D
- Cote D
- Cottin G
- Coutinho YA
- Cowan G
- Cox BE
- Cranmer K
- Cree G
- Crepe-Renaudin S
- Crescioli F
- Cristinziani M
- Crosetti G
- Cuciuc C-M
- Cummings J
- Curatolo M
- Cuthbert C
- Czirr H
- Czodrowski P
- Czyczula Z
- D'Auria S
- D'Onofrio M
- Da Costa JBG
- Da Costa JGPF
- Da Costa JGPF
- Da Cunha Sargedas De Sousa MJ
- Da Via C
- Dabrowski W
- Dafinca A
- Dai T
- Dale O
- Dallaire F
- Dallapiccola C
- Dam M
- Damazio DO
- Daniells AC
- Dao V
- Darbo G
- Darlea GL
- Darmora S
- Dassoulas JA
- Davey W
- David C
- Davidek T
- Davies E
- Davies M
- Davignon O
- Davison AR
- Davison P
- Davygora Y
- Dawe E
- Dawson I
- Daya-Ishmukhametova RK
- de Asmundis R
- De Bruin PHS
- De Castro S
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- De la Taille C
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- De Pedis D
- De Regie JBDV
- de Renstrom PAB
- De Salvo A
- De Sanctis U
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- della Porta GZ
- della Volpe D
- Delmastro M
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- Deluca C
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- Demilly A
- Demirkoz B
- Denisov SP
- Derendarz D
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- Derue F
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- Deviveiros PO
- Dewhurst A
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- Di Ciaccio L
- Di Domenico A
- Di Donato C
- Di Girolamo A
- Di Girolamo B
- Di Mattia A
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- Diglio S
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- Doglioni C
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- Ekelof T
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- Enari Y
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- Endner OC
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- Endo M
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- Engelmann R
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- Erdmann J
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- Errede D
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- Errede S
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- Ertel E
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- Escalier M
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- Esch H
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- Escobar C
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- Espinal Curull X
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- Esposito B
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- Etienne F
- Etienne F
- Etienvre AI
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- Etzion E
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- Evans H
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- Fabbri L
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- Facini G
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- Fakhrutdinov RM
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- Falciano S
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- Faltova J
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- Fang Y
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- Fanti M
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- Farbin A
- Farbin A
- Farilla A
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- Farooque T
- Farooque T
- Farrell S
- Farrell S
- Farrington SM
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- Farthouat P
- Farthouat P
- Fassi F
- Fassi F
- Fassnacht P
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- Fassouliotis D
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- Favareto A
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- Fayard L
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- Federic P
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- Fedin OL
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- Fedorko W
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- Fehling-Kaschek M
- Fehling-Kaschek M
- Feigl S
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- Feligioni L
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- Feng C
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- Feng EJ
- Feng EJ
- Feng H
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- Fenyuk AB
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- Fernando W
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- Ferrag S
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- Ferrando BMS
- Ferrando J
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- Ferrara V
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- Ferrari A
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- Ferrari P
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- Ferrari R
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- Ferretti C
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- Fiascaris M
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- Fiedler F
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- Filipcic A
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- Filipuzzi M
- Filipuzzi M
- Filthaut F
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- Fincke-Keeler M
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- Fiorini L
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- Firan A
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- Fisher MJ
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- Fitzgerald EA
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- Fleck I
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- Fleischmann P
- Fleischmann P
- Fleischmann S
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- Fletcher G
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- Floderus A
- Floderus A
- Florez Bustos AC
- Flowerdew MJ
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- Formica A
- Formica A
- Forti A
- Forti A
- Fortin D
- Fortin D
- Fournier D
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- Francavilla P
- Francavilla P
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- Franchini M
- Franchino S
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- Francis D
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- Franklin M
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- Franz S
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- Fraternali M
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- French ST
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- Gadomski S
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- Gagnon P
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- Galea C
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- Gallas EJ
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- Gallo V
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- Gallop BJ
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- Gao YS
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- Gardner RW
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- Garelli N
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- Garonne V
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- Garrido MDMC
- Gatti C
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- Gaudio G
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- Gaur B
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- Gauzzi P
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- Gavrilenko IL
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- Gaycken G
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- Gazis EN
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- Ge P
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- Gecse Z
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- Gee CNP
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- Geerts DAA
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- Geich-Gimbel C
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- Gellerstedt K
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- Gemme C
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- Gemmell A
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- Genest MH
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- Gentile S
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- George M
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- George S
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- Gerbaudo D
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- Gershon A
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- Ghazlane H
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- Ghodbane N
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- Giangiobbe V
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- Giannetti P
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- Gillam TPS
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- Gillberg D
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- Gillman AR
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- Gingrich DM
- Gingrich M
- Giokaris N
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- Giordani MP
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- Giordano R
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- Giugni D
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- Gornicki E
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- Goshaw AT
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- Gostkin MI
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- Zabinski B
- Zaidan R
- Zaitsev AM
- Zaman A
- Zambito S
- Zanello L
- Zanzi D
- Zaytsev A
- Zeitnitz C
- Zeman M
- Zemla A
- Zengel K
- Zenin O
- Zenis T
- Zerwas D
- Zhang D
- Zhang F
- Zhang H
- Zhang J
- Zhang L
- Zhang X
- Zhang Z
- Zhao Z
- Zhemchugov A
- Zhong J
- Zhou B
- Zhou L
- Zhou N
- Zhu CG
- Zhu H
- Zhu J
- Zhu Y
- Zhuang X
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann C
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zitoun R
- Zobernig G
- Zoccoli A
- Zurzolo G
- Zutshi V
- Zwalinski L
- Publication venue
- 'IOP Publishing'
- Publication date
- 01/01/2014
- Field of study
This paper presents a measurement of the cross-section for high transverse momentum W and Z bosons produced in pp collisions and decaying to all-hadronic final states. The data used in the analysis were recorded by the ATLAS detector at the CERN Large Hadron Collider at a centre-of-mass energy of √s = 7 TeV;{\rm Te}{\rm V}andcorrespondtoanintegratedluminosityof4.6\;{\rm f}{{{\rm b}}^{-1}}.ThemeasurementisperformedbyreconstructingtheboostedWorZbosonsinsinglejets.ThereconstructedjetmassisusedtoidentifytheWandZbosons,andajetsubstructuremethodbasedonenergyclusterinformationinthejetcentre−of−massframeisusedtosuppressthelargemulti−jetbackground.Thecross−sectionforeventswithahadronicallydecayingWorZboson,withtransversemomentum{{p}_{{\rm T}}}\gt 320\;{\rm Ge}{\rm V}andpseudorapidity|\eta |\lt 1.9,ismeasuredtobe{{\sigma }_{W+Z}}=8.5\pm 1.7$ pb and is compared to next-to-leading-order calculations. The selected events are further used to study jet grooming techniques
Observation of associated near-side and away-side long-range correlations in √sNN=5.02 TeV proton-lead collisions with the ATLAS detector
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- Zhemchugov A
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- Zobernig G
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- Zutshi V
- Zwalinski L
- Publication venue
- American Physical Society
- Publication date
- 01/01/2012
- Field of study
Two-particle correlations in relative azimuthal angle (Δϕ) and pseudorapidity (Δη) are measured in √sNN=5.02 TeV p+Pb collisions using the ATLAS detector at the LHC. The measurements are performed using approximately 1 μb-1 of data as a function of transverse momentum (pT) and the transverse energy (ΣETPb) summed over 3.1<η<4.9 in the direction of the Pb beam. The correlation function, constructed from charged particles, exhibits a long-range (2<|Δη|<5) “near-side” (Δϕ∼0) correlation that grows rapidly with increasing ΣETPb. A long-range “away-side” (Δϕ∼π) correlation, obtained by subtracting the expected contributions from recoiling dijets and other sources estimated using events with small ΣETPb, is found to match the near-side correlation in magnitude, shape (in Δη and Δϕ) and ΣETPb dependence. The resultant Δϕ correlation is approximately symmetric about π/2, and is consistent with a dominant cos2Δϕ modulation for all ΣETPb ranges and particle pT
Search for the neutral Higgs bosons of the minimal supersymmetric standard model in pp collisions at root s=7 TeV with the ATLAS detector
- Author
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- Publication venue
- Publication date
- 01/02/2013
- Field of study
A search for neutral Higgs bosons of the Minimal Supersymmetric Standard Model (MSSM) is reported. The analysis is based on a sample of proton-proton collisions at a centre-of-mass energy of 7TeV recorded with the ATLAS detector at the Large Hadron Collider. The data were recorded in 2011 and correspond to an integrated luminosity of 4.7 fb-1 to 4.8 fb-1. Higgs boson decays into oppositely-charged muon or τ lepton pairs are considered for final states requiring either the presence or absence of b-jets. No statistically significant excess over the expected background is observed and exclusion limits at the 95% confidence level are derived. The exclusion limits are for the production cross-section of a generic neutral Higgs boson, φ, as a function of the Higgs boson mass and for h/A/H production in the MSSM as a function of the parameters mA and tan β in the mhmax scenario for mA in the range of 90GeV to 500 GeV. Copyright CERN
Search for direct pair production of the top squark in all-hadronic final states in proton-proton collisions at s√=8 TeV with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abdallah J
- Abdinov O
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2014
- Field of study
The results of a search for direct pair production of the scalar partner to the top quark using an integrated luminosity of 20.1fb−1 of proton–proton collision data at √s = 8 TeV recorded with the ATLAS detector at the LHC are reported. The top squark is assumed to decay via t˜→tχ˜01 or t˜→ bχ˜±1 →bW(∗)χ˜01 , where χ˜01 (χ˜±1 ) denotes the lightest neutralino (chargino) in supersymmetric models. The search targets a fully-hadronic final state in events with four or more jets and large missing transverse momentum. No significant excess over the Standard Model background prediction is observed, and exclusion limits are reported in terms of the top squark and neutralino masses and as a function of the branching fraction of t˜ → tχ˜01 . For a branching fraction of 100%, top squark masses in the range 270–645 GeV are excluded for χ˜01 masses below 30 GeV. For a branching fraction of 50% to either t˜ → tχ˜01 or t˜ → bχ˜±1 , and assuming the χ˜±1 mass to be twice the χ˜01 mass, top squark masses in the range 250–550 GeV are excluded for χ˜01 masses below 60 GeV
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