86 research outputs found
Efficacy of serology driven “test and treat strategy” for eradication of H. pylori in patients with rheumatic disease in the Netherlands
- Author
- A Kokkola
- A. M. Huisman
- B. A. C. Dijkmans
- BA Wouw van de
- BA Wouw van de
- BC Meijer
- BC Meijer
- E. J. Kuipers
- EJ Kuipers
- EJ Wouden van der
- F Lerang
- F Lerang
- F Megraud
- FK Chan
- FK Chan
- H. H. M. L. Houben
- H. T. J. I. de Leest
- HT LeesT de
- J Labenz
- JC Thijs
- K. S. S. Steen
- L Monteiro
- M. A. F. J. van de Laar
- MF Dixon
- MJ Janssen
- P Malfertheiner
- P. J. Kostense
- Q Gu
- RO Lindsetmo
- S. W. Kadir
- SH Park
- U Kiltz
- W. F. Lems
- WA Boer de
- WD Chey
- WH Wang
- Y Glupczynski
- Y. J. Debets-Ossenkopp
- YJ Debets-Ossenkopp
- Publication venue
- Springer-Verlag
- Publication date
- 01/01/2011
- Field of study
The treatment of choice of H. pylori infections is a 7-day triple-therapy with a proton pump inhibitor (PPI) plus amoxicillin and either clarithromycin or metronidazole, depending on local antibiotic resistance rates. The data on efficacy of eradication therapy in a group of rheumatology patients on long-term NSAID therapy are reported here. This study was part of a nationwide, multicenter RCT that took place in 2000–2002 in the Netherlands. Patients who tested positive for H. pylori IgG antibodies were included and randomly assigned to either eradication PPI-triple therapy or placebo. After completion, follow-up at 3 months was done by endoscopy and biopsies were sent for culture and histology. In the eradication group 13% (20/152, 95% CI 9–20%) and in the placebo group 79% (123/155, 95% CI 72–85%) of the patients were H. pylori positive by histology or culture. H. pylori was successfully eradicated in 91% of the patients who were fully compliant to therapy, compared to 50% of those who were not (difference of 41%; 95% CI 18–63%). Resistance percentages found in isolates of the placebo group were: 4% to clarithromycin, 19% to metronidazole, 1% to amoxicillin and 2% to tetracycline
Less Can Be More: RNA-Adapters May Enhance Coding Capacity of Replicators
- Author
- A Brennicke
- A Fire
- A Kun
- A Philpott
- A Serganov
- A Wochner
- AG Vitreschak
- AR Ferré-D'Amaré
- AS Richter
- BA Shapiro
- CC Mello
- D Herschlag
- DG Silvestre
- DL Black
- E Szathmáry
- E Szathmáry
- EJ Sontheimer
- FJ Isaacs
- FK de Boer
- Folkert K. de Boer
- G Tiana
- GF Joyce
- IL Hofacker
- J Bachellerie
- J Zhang
- JM Maynard-Smith
- L Li
- M Boerlijst
- M Eigen
- M Eigen
- M Eigen
- M Huynen
- M Irimia
- MA Huynen
- MA Huynen
- N Takeuchi
- N Takeuchi
- N Takeuchi
- P Chen
- P Csermely
- P Hogeweg
- P Hogeweg
- P Schuster
- Paulien Hogeweg
- Peter Csermely
- R Knight
- R Sanjuán
- RJ Ellis
- RJ Ellis
- RK Montange
- RM Turk
- T Kiss
- W Fontana
- W Fontana
- W Gilbert
- Publication venue
- Public Library of Science
- Publication date
- 23/01/2012
- Field of study
It is still not clear how prebiotic replicators evolved towards the complexity found in present day organisms. Within the most realistic scenario for prebiotic evolution, known as the RNA world hypothesis, such complexity has arisen from replicators consisting solely of RNA. Within contemporary life, remarkably many RNAs are involved in modifying other RNAs. In hindsight, such RNA-RNA modification might have helped in alleviating the limits of complexity posed by the information threshold for RNA-only replicators. Here we study the possible role of such self-modification in early evolution, by modeling the evolution of protocells as evolving replicators, which have the opportunity to incorporate these mechanisms as a molecular tool. Evolution is studied towards a set of 25 arbitrary ‘functional’ structures, while avoiding all other (misfolded) structures, which are considered to be toxic and increase the death-rate of a protocell. The modeled protocells contain a genotype of different RNA-sequences while their phenotype is the ensemble of secondary structures they can potentially produce from these RNA-sequences. One of the secondary structures explicitly codes for a simple sequence-modification tool. This ‘RNA-adapter’ can block certain positions on other RNA-sequences through antisense base-pairing. The altered sequence can produce an alternative secondary structure, which may or may not be functional. We show that the modifying potential of interacting RNA-sequences enables these protocells to evolve high fitness under high mutation rates. Moreover, our model shows that because of toxicity of misfolded molecules, redundant coding impedes the evolution of self-modification machinery, in effect restraining the evolvability of coding structures. Hence, high mutation rates can actually promote the evolution of complex coding structures by reducing redundant coding. Protocells can successfully use RNA-adapters to modify their genotype-phenotype mapping in order to enhance the coding capacity of their genome and fit more information on smaller sized genomes
Search for the standard model Higgs boson at LEP
- Author
- Abbaneo D
- Abbaneo D
- Abbiendi G
- Abbiendi G
- Abdallah J
- Abreu P
- Achard P
- Adam W
- Adriani O
- Adzic P
- Affholderbach K
- Aguilar-Benitez M
- Ainsley C
- Akesson PF
- Albrecht T
- Alcaraz J
- Alderweireld T
- Alemanni G
- Alemany-Fernandez R
- Alexander G
- Allaby J
- Allison J
- Allmendinger T
- Allport PP
- Aloisio A
- Alviggi MG
- Amaldi U
- Amapane N
- Amaral P
- Amato S
- Anagnostou G
- Anashkin E
- Anderhub H
- Anderson KJ
- Andreazza A
- Andreev VP
- Andringa S
- Anjos N
- Anselmo F
- Antilogus P
- Antonelli A
- Antonelli M
- Apel WD
- Arcelli S
- Arefiev A
- Armstrong SR
- Arnoud Y
- Asai S
- Ask S
- Asman B
- Augustin JE
- Augustinus A
- Awunor O
- Axen D
- Azemoon T
- Aziz T
- Azuelos G
- Azzurri P
- Baarmand M
- Badaud F
- Bagliesi G
- Bagnaia P
- Bailey I
- Baillon P
- Bajo A
- Baksay G
- Baksay L
- Baldew SV
- Ballestrero A
- Bambade P
- Banerjee S
- Banerjee S
- Barate R
- Barate R
- Barberio E
- Barbier R
- Barczyk A
- Bardin D
- Barillere R
- Barker G
- Barklow T
- Barlow RJ
- Baroncelli A
- Bartalini P
- Basile M
- Batalova N
- Batley RJ
- Battaglia M
- Battiston R
- Baubillier M
- Bay A
- Becattini F
- Bechtle P
- Becker U
- Becks KH
- Begalli M
- Behner F
- Behnke T
- Behrmann A
- Bell KW
- Bell PJ
- Bella G
- Bellerive A
- Bellucci L
- Ben-Haim E
- Bencivenni G
- Benekos N
- Benelli G
- Benvenuti A
- Berat C
- Berbeco R
- Berdugo J
- Berges P
- Berggren M
- Berkelman K
- Berntzon L
- Bertrand D
- Bertucci B
- Besancon M
- Besson N
- Betev BL
- Bethke S
- Beuselinck R
- Biasini M
- Biebell O
- Biglietti M
- Biland A
- Binnie DM
- Blair GA
- Blaising JJ
- Bloch D
- Bloch-Devaux B
- Blom M
- Blondel A
- Bloodworth IJ
- Bluj M
- Blumenschein U
- Blyth SC
- Bobbink GJ
- Boccali T
- Bock P
- Boeriun O
- Bohm A
- Bohrer A
- Boix G
- Boldizsar L
- Bologna G
- Bonacorsi D
- Bonesini M
- Bonissent A
- Boonekamp M
- Booth CN
- Booth PSL
- Borean C
- Borgia B
- Borisov G
- Bossi F
- Botner O
- Boucrot J
- Bouhova-Thacker E
- Boumediene D
- Bouquet B
- Bourilkov D
- Bourquin M
- Boutemeur M
- Bowcock TJV
- Bowdery CK
- Boyko I
- Braccini S
- Bracko M
- Braibant S
- Brandt S
- Branson JG
- Bravo S
- Brenner R
- Brient JC
- Brigliadori L
- Brochu F
- Brodet E
- Brown RM
- Bruckman P
- Bruneliere R
- Brunet JM
- Buchmuller O
- Buesser K
- Bugge L
- Buijs A
- Burckhart HJ
- Burger JD
- Burger WJ
- Buschmann P
- Cai XD
- Callot O
- Calvi M
- Cameron W
- Campana R
- Campana S
- Camporesi T
- Canale V
- Capell M
- Capon G
- Carena F
- Carlino G
- Carnegie RK
- Caron B
- Carr J
- Cartacci A
- Carter AA
- Carter JR
- Cartwright S
- Casado MP
- Casaus J
- Castro N
- Cattaneo A
- Cavallari F
- Cavallo F
- Cavallo N
- Cavanaugh R
- Cecchi C
- Cerrada M
- Cerutti F
- Chamizo M
- Chang CY
- Chang YH
- Chapkin M
- Charlton DG
- Charpentier P
- Checchia P
- Chemarin M
- Chen A
- Chen G
- Chen GM
- Chen HF
- Chen HS
- Chiarella V
- Chiefari G
- Chierici R
- Chiliapnikov P
- Chmeissani M
- Chudoba J
- Chung SU
- Cieslik K
- Cifarelli L
- Cindolo F
- Ciulli V
- Clare I
- Clare R
- Clarke DP
- Clerbaux B
- Clifft RW
- Coignet G
- Colaleo A
- Colas P
- Coles J
- Colino N
- Collins P
- Combley F
- Contri R
- Cosme G
- Cossutti F
- Costa MJ
- Costantini S
- Cowan G
- Coyle P
- Cranmer K
- Crawley B
- Creanza D
- Crennell D
- Crespo JM
- Csilling A
- Cucciarelli S
- Cuevas J
- Cuffiani M
- Curtil C
- D'Hondt J
- da Silva T
- Da Silva W
- Dado S
- Dai TS
- Dallavalle GM
- Dallison S
- Dalmau J
- David A
- Davier M
- Davies G
- De Angelis A
- de Asmundis R
- De Boer W
- De Bonis I
- De Clercq C
- De Filippis N
- de Guevara PL
- de Jong R
- de la Cruz B
- De Lotto B
- De Maria N
- De Min A
- de Palma M
- de Paula L
- De Roeck A
- De Salvo A
- De Vivie de Regie JB
- De Wolf EA
- Debreczeni J
- Decamp D
- Deglon P
- Degre A
- Deiters K
- Della Ricca G
- della Volpe D
- Delmeire E
- Denes P
- DeNotaristefani F
- Desch K
- Dessagne S
- Dhamotharan S
- Di Ciaccio L
- Di Simone A
- Diemoz M
- Dienes B
- Dierckxsens M
- Dietl H
- Dionisi C
- Dissertori G
- Dittmar M
- Donkers M
- Doria A
- Dornan PJ
- Doroba K
- Dova MT
- Drees J
- Drevermann H
- Dris M
- Dubbert J
- Duchesneau D
- Duchovni E
- Duckeck G
- Duerdoth IP
- Duflot L
- Duinker P
- Ealet A
- Echenard B
- Edgecock TR
- Eigen G
- Ekelof T
- El Mamouni H
- Elfgren E
- Eline A
- Ellert M
- Ellis G
- Elsing M
- Engler A
- Eppling FJ
- Etzion E
- Ewers A
- Extermann P
- Fabbri F
- Fabbro B
- Falagan MA
- Falciano S
- Falvard A
- Fanourakis G
- Fassouliotis D
- Favara A
- Fay J
- Fayolle D
- Fedin O
- Feindt M
- Felcini M
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- Ferguson DPS
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- Fernandez E
- Fernandez J
- Fernandez-Bosman M
- Ferrari P
- Ferrer A
- Ferro F
- Fesefeldt H
- Fiandrini E
- Fiedler F
- Field JH
- Filthaut F
- Finch AJ
- Fisher PH
- Fisher W
- Fisk I
- Flagmeyer U
- Fleck I
- Foa L
- Focardi E
- Foeth H
- Fokitis E
- Forconi G
- Ford M
- Forty RW
- Foster F
- Fouchez D
- Frank M
- Freudenreich K
- Frey A
- Fulda-Quenzer F
- Furetta C
- Furtjes A
- Fuster J
- Gagnon P
- Galaktionov Y
- Gandelman M
- Ganguli SN
- Ganis G
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- Garcia C
- Garcia-Abia P
- Garcia-Bellido A
- Garrido L
- Gary JW
- Gataullin M
- Gavillet P
- Gay P
- Gaycken G
- Gazis E
- Geich-Gimbel C
- Gentile S
- Geweniger C
- Ghete VM
- Giacomelli G
- Giacomelli P
- Giagu S
- Giammanco A
- Giannini G
- Gianotti F
- Giassi A
- Girone M
- Girtler P
- Giunta M
- Gokieli R
- Goldberg J
- Golob B
- Gomez-Ceballos G
- Goncalves P
- Gong ZF
- Gonzalez S
- Goy C
- Grauges E
- Graziani E
- Green MG
- Greening TC
- Grenier G
- Grimm O
- Grivaz JF
- Grosdidier G
- Gross E
- Gruenewald MW
- Grunhaus J
- Grupen C
- Gruwe M
- Grzelak K
- Guida M
- Gunther PO
- Gupta A
- Gupta VK
- Gurtu A
- Gutay LJ
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- Haas D
- Hajdu C
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- Johansson EK
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- Kellogg RG
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- Kjaer NJ
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- Klimentov A
- Kluge EE
- Kluit P
- Kluth S
- Kneringer E
- Kobayashi T
- Kobel M
- Kokkinias P
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- Konstantinidis N
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- Krumstein Z
- Kucharczyk M
- Kuhn D
- Kunin A
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- Lafferty GD
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- Lamsa J
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- Landi G
- Landsman H
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- Layter JG
- Le Coultre P
- Le Goff JM
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- Loebinger FK
- Lohmann W
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- Piotto E
- Piroue PA
- Pistolesi E
- Plane DE
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- Prokofiev D
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- Trigger I
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- Waller D
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- Zichichi A
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- Zilizi G
- Zimin NI
- Zimmermann B
- Zintchenko A
- Zito G
- Zivkovic L
- Zobernig G
- Zoller M
- Zupan M
- Publication venue
- ELSEVIER SCIENCE BV
- Publication date
- 17/07/2003
- Field of study
Reconstruction of ancestral RNA sequences under multiple structural constraints
- Author
- A Srivastava
- AR Gruber
- B Knudsen
- B Paten
- C Bustamante
- CA Nasrallah
- D Sankoff
- DA Sorescu
- E Rogers
- EP Nawrocki
- F Vogel
- FK de Boer
- J Stombaugh
- JH Urban
- JJ Gillespie
- JL Knies
- Jérôme Waldispühl
- K Robert
- KM Kutchko
- L Pauling
- LJ Jerome
- M Blanchette
- MP Hoeppner
- O Tremblay-Savard
- Olivier Tremblay-Savard
- P Schuster
- PG Higgs
- PP Gardner
- PS Klosterman
- R Lorenz
- RB Lyngsø
- RK Bradley
- RR Gutell
- SR Eddy
- T Biesen
- V Reinharz
- Vladimir Reinharz
- WM Fitch
- Y Chen
- Z Yang
- Z Yao
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
The Roots of Bioinformatics in Theoretical Biology
- Author
- A Anderson
- A Boiteux
- A Crombach
- A Crombach
- A Lindenmayer
- A Lindenmayer
- A Marée
- A Marée
- A Neyfakh
- A Turing
- A Varma
- A Wagner
- A Wagner
- B Goodwin
- B Hesper
- B Turner
- C Hewitt
- C Honk
- C Pál
- CH Waddington
- D Gillespie
- D Konings
- D Konings
- David B. Searls
- E Koonin
- E van Nimwegen
- EP Odum
- F Crick
- F Graner
- F Rosenblatt
- FK de Boer
- G Lance
- G Odell
- H Abelson
- H Kacser
- J Draghi
- J Draghi
- J Griffith
- J Hagen
- J Holland
- L Hurst
- L Segel
- L Von Bertalanffy
- L Von Bertalanffy
- L Wolpert
- M Boerlijst
- M Covert
- M Dayhoff
- M Dayhoff
- M Huynen
- M Huynen
- M Huynen
- M Kertesz
- M Kozak
- M Minsky
- M Szekely
- M Thomson
- M Van Hoek
- N Batada
- N Savill
- N Stoletzki
- O Mastenbroek
- O Soyer
- P Hogeweg
- P Hogeweg
- P Hogeweg
- P Hogeweg
- P Hogeweg
- P Hogeweg
- P Hogeweg
- P Hogeweg
- P Hogeweg
- P Hogeweg
- P Hogeweg
- P Macnaughton-Smith
- P Nurse
- P Schuster
- P Sneath
- Paulien Hogeweg
- R Goldstein
- R May
- R Rosen
- S Freilich
- S Huang
- S Huang
- S Kauffman
- S Papert
- S Rafelski
- W Gu
- Publication venue
- Public Library of Science
- Publication date
- 01/03/2011
- Field of study
From the late 1980s onward, the term “bioinformatics” mostly has been used to refer to computational methods for comparative analysis of genome data. However, the term was originally more widely defined as the study of informatic processes in biotic systems. In this essay, I will trace this early history (from a personal point of view) and I will argue that the original meaning of the term is re-emerging
Factors associated with work disability in employed cancer survivors at 24-month sick leave
- Author
- A De Boer
- A Johnsson
- A Mehnert
- AE Buckwalter
- AGEM De Boer
- AJ Schellart
- Allard J van der Beek
- B Bergman
- CA Roelen
- DR Fenlon
- ER Spelten
- ER Spelten
- F Kennedy
- FK Tangka
- GP Guy Jr
- HP Greenwald
- J Ilmarinen
- JG Vartanian
- JH Park
- JH Park
- Johannes R Anema
- JP Broersen
- JR Peteet
- K Carlsen
- Karin Bonefaas-Groenewoud
- Kenniscentrum UWV Directie SBK
- KR Yabroff
- KS Choi
- L Calvio
- L Calvio
- LS Radloff
- M Bergner
- M Feuerstein
- NK Aaronson
- P Poirier
- P van Muijen
- P van Muijen
- P Wynn
- Peter van Muijen
- PF Short
- PF Short
- R Grewal
- RR Bouknight
- S Lauzier
- Saskia FA Duijts
- SB Gudbergsson
- SB Yellen
- T Taskila
- V Peuckmann
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
A922 Sequential measurement of 1 hour creatinine clearance (1-CRCL) in critically ill patients at risk of acute kidney injury (AKI)
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- Abbott TE
- Abdallah RI
- Abdel-Aal IR
- Abdelmonem SA
- Abdo WF
- Abellan AN
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- Aguirregabyria M
- Ahmadnia E
- Ahn C
- Ahn JY
- Ahn JY
- Ahn MY
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- Aitken LM
- Akalaev R
- Akbarzadeh A
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- Akhlagh SH
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- AKI Research Group
- Akiduki N
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- CAPCRI Study
- Capellier G
- Carbonara M
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- Cariou A
- Cariou A
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- Carratalá A
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- Carreño ER
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- Carvalho JP
- Carvalho KV
- Casals XN
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- Castañeda DP
- Castellana D
- Castellanos A
- Castellanos A
- Castillo-Lorente E
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- Catalan M
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- Catena E
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- Cauwenberghs H
- Cavalcanti D
- Cavalheiro AM
- Cavalier E
- Cavicchi FZ
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- Cengiz M
- Ceriani R
- Cermelli S
- Cerović O
- Cha YS
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- Champigneulle B
- Champigneulle B
- Chan KC
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- Chang WY
- Chao A
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- Charitidou E
- Charpentier C
- Charpentier J
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- Chaudhari HK
- Chaussard M
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- Chelly J
- Chemam S
- Chen CM
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- Chrétien JM
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- Fang M
- Fanzaghi P
- Farias P
- Faulds MC
- Fave G
- Feehan A
- Feijoo AM
- Feliot E
- Ferez MA
- Fernandes MJ
- Fernandes N
- Fernandez NG
- Fernandez R
- Fernandez-Zamora MD
- Fernández IF
- Fernández L
- Fernández MV
- Fernández PA
- Fernández-Barat L
- Fernández-Mondéjar E
- Fernández-Ortega JF
- Ferraz AB
- Ferrero-Calleja J
- Ferri F
- Ferry A
- Ferré A
- Ferrón FR
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- Fiks T
- Fiks T
- Fileković S
- Filho CA
- Filho CA
- Filho CA
- Filipe E
- FINNRESUSCI Study Group
- Fischer G
- Fleischmann E
- Fless K
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- Follin A
- Follin A
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- Fontaiña LP
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- Fontenot T
- Forel JM
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- Frat JP
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- Freile MT
- Frenzel T
- Frigeni B
- Frithiof R
- Fritz C
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- Fromentin M
- Frontera PR
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- Fumagalli F
- Fumagalli F
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- Fushimi K
- Gabán-Díez Á
- Gadgil S
- Gaide-Chevronnay L
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- Gaite FB
- Gaizauskas E
- Gajic O
- Galache JA
- Gallego-Corpa AI
- Gameiro J
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- Gando S
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- Gandola A
- Gandolfi S
- Ganuza MS
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- Garcia M
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- Garcia-Alvarez JM
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- Garcia-Delgado M
- Garcia-Gigorro R
- García MF
- García-Martínez MV
- García-Olivares P
- Garnacho-Montero J
- Garrido BB
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- Garrido EM
- Garrouste M
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- Gasparovic V
- Gattinoni L
- Gaudard P
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- Gavrychenko D
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- Geantot MA
- Gemmell L
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- Genc D
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- Geri G
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- GETGAG/SEMICYUC
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- Gherli T
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- Giannantonio M
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- Gimenez J
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- Gordo F
- Gordon M
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- Gorjup V
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- Gottlieb J
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- Grando M
- Granger CL
- Grangé S
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- Grassi L
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- Hashimoto Y
- Hassanin H
- Hatakeyama J
- Hatib F
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- Hayashi K
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- Herruzo-Aviles A
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- Hewitt H
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- Hickey S
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- Hirayama T
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- Hodgson LE
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- Hofmeijer J
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- Hom H
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- Hou DS
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- Hozhabri K
- Hrachovina M
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- Jardim JJ
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- Jovaisa T
- JSEPTIC (Japanese Society of Education for Physicians and Trainees in Intensive Care) Clinical Trial Group
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- Kaese S
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- Kalenka A
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- Kaneko M
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- Karanjia N
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- Kasdan H
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- Katira BH
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- Kayambu G
- Kazutoshi F
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- Kirca H
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- Kleinpell R
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- Kokkini S
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- Kovacikova L
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- Kurtz P
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- Kwon HM
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- Lafaurie M
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- Lagonidis D
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- Leone M
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- Lortat-Jacob B
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- Macfarlane B
- MacKay A
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- Mackey G
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- Maekawa K
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- Maertens B
- Maggiorini M
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- Magnanimi E
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- Mainardi JL
- Maistry N
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- Martínez F
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- Mion MM
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- Mistry M
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- Mitchell S
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- Mlcek M
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- Moffatt S
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- Pifferi S
- Pinsky M
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- Piquilloud L
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- Pons S
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- Post E
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- Pottecher J
- Poukkanen M
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- Pozzebon S
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- Pravettoni D
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- Puybasset L
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- Álvarez JT
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
Meeting abstrac
Precise mapping of the magnetic field in the CMS barrel yoke using cosmic rays
- Author
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- Publication date
- 01/01/1
- Field of study
This is the Pre-print version of the Article. The official published version can be accessed from the link below - Copyright @ 2010 IOPThe CMS detector is designed around a large 4 T superconducting solenoid, enclosed in a 12 000-tonne steel return yoke. A detailed map of the magnetic field is required for the accurate simulation and reconstruction of physics events in the CMS detector, not only in the inner tracking region inside the solenoid but also in the large and complex structure of the steel yoke, which is instrumented with muon chambers. Using a large sample of cosmic muon events collected by CMS in 2008, the field in the steel of the barrel yoke has been determined with a precision of 3 to 8% depending on the location.This work is supported by FMSR (Austria); FNRS and FWO (Belgium); CNPq, CAPES, FAPERJ,
and FAPESP (Brazil); MES (Bulgaria); CERN; CAS, MoST, and NSFC (China); COLCIENCIAS
(Colombia); MSES (Croatia); RPF (Cyprus); Academy of Sciences and NICPB (Estonia);
Academy of Finland, ME, and HIP (Finland); CEA and CNRS/IN2P3 (France); BMBF, DFG, and HGF (Germany); GSRT (Greece); OTKA and NKTH (Hungary); DAE and DST (India); IPM (Iran); SFI (Ireland); INFN (Italy); NRF (Korea); LAS (Lithuania); CINVESTAV, CONACYT, SEP, and UASLP-FAI (Mexico); PAEC (Pakistan); SCSR (Poland); FCT (Portugal); JINR
(Armenia, Belarus, Georgia, Ukraine, Uzbekistan); MST and MAE (Russia); MSTDS (Serbia); MICINN and CPAN (Spain); Swiss Funding Agencies (Switzerland); NSC (Taipei); TUBITAK and TAEK (Turkey); STFC (United Kingdom); DOE and NSF (USA)
Iron Behaving Badly: Inappropriate Iron Chelation as a Major Contributor to the Aetiology of Vascular and Other Progressive Inflammatory and Degenerative Diseases
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- The Long-Term Intervention with Pravastatin in Ischaemic Disease (LIPID) Study Group
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- Publication venue
- Publication date
- 09/08/2008
- Field of study
The production of peroxide and superoxide is an inevitable consequence of
aerobic metabolism, and while these particular "reactive oxygen species" (ROSs)
can exhibit a number of biological effects, they are not of themselves
excessively reactive and thus they are not especially damaging at physiological
concentrations. However, their reactions with poorly liganded iron species can
lead to the catalytic production of the very reactive and dangerous hydroxyl
radical, which is exceptionally damaging, and a major cause of chronic
inflammation. We review the considerable and wide-ranging evidence for the
involvement of this combination of (su)peroxide and poorly liganded iron in a
large number of physiological and indeed pathological processes and
inflammatory disorders, especially those involving the progressive degradation
of cellular and organismal performance. These diseases share a great many
similarities and thus might be considered to have a common cause (i.e.
iron-catalysed free radical and especially hydroxyl radical generation). The
studies reviewed include those focused on a series of cardiovascular, metabolic
and neurological diseases, where iron can be found at the sites of plaques and
lesions, as well as studies showing the significance of iron to aging and
longevity. The effective chelation of iron by natural or synthetic ligands is
thus of major physiological (and potentially therapeutic) importance. As
systems properties, we need to recognise that physiological observables have
multiple molecular causes, and studying them in isolation leads to inconsistent
patterns of apparent causality when it is the simultaneous combination of
multiple factors that is responsible. This explains, for instance, the
decidedly mixed effects of antioxidants that have been observed, etc...Comment: 159 pages, including 9 Figs and 2184 reference
Worldwide trends in underweight and obesity from 1990 to 2022: a pooled analysis of 3663 population-representative studies with 222 million children, adolescents, and adults
- Author
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- Publication venue
- Elsevier
- Publication date
- Field of study
Background Underweight and obesity are associated with adverse health outcomes throughout the life course. We estimated the individual and combined prevalence of underweight or thinness and obesity, and their changes, from 1990 to 2022 for adults and school-aged children and adolescents in 200 countries and territories. Methods We used data from 3663 population-based studies with 222 million participants that measured height and weight in representative samples of the general population. We used a Bayesian hierarchical model to estimate trends in the prevalence of different BMI categories, separately for adults (age ≥20 years) and school-aged children and adolescents (age 5–19 years), from 1990 to 2022 for 200 countries and territories. For adults, we report the individual and combined prevalence of underweight (BMI 2 SD above the median). Findings From 1990 to 2022, the combined prevalence of underweight and obesity in adults decreased in 11 countries (6%) for women and 17 (9%) for men with a posterior probability of at least 0·80 that the observed changes were true decreases. The combined prevalence increased in 162 countries (81%) for women and 140 countries (70%) for men with a posterior probability of at least 0·80. In 2022, the combined prevalence of underweight and obesity was highest in island nations in the Caribbean and Polynesia and Micronesia, and countries in the Middle East and north Africa. Obesity prevalence was higher than underweight with posterior probability of at least 0·80 in 177 countries (89%) for women and 145 (73%) for men in 2022, whereas the converse was true in 16 countries (8%) for women, and 39 (20%) for men. From 1990 to 2022, the combined prevalence of thinness and obesity decreased among girls in five countries (3%) and among boys in 15 countries (8%) with a posterior probability of at least 0·80, and increased among girls in 140 countries (70%) and boys in 137 countries (69%) with a posterior probability of at least 0·80. The countries with highest combined prevalence of thinness and obesity in school-aged children and adolescents in 2022 were in Polynesia and Micronesia and the Caribbean for both sexes, and Chile and Qatar for boys. Combined prevalence was also high in some countries in south Asia, such as India and Pakistan, where thinness remained prevalent despite having declined. In 2022, obesity in school-aged children and adolescents was more prevalent than thinness with a posterior probability of at least 0·80 among girls in 133 countries (67%) and boys in 125 countries (63%), whereas the converse was true in 35 countries (18%) and 42 countries (21%), respectively. In almost all countries for both adults and school-aged children and adolescents, the increases in double burden were driven by increases in obesity, and decreases in double burden by declining https://researchonline.ljmu.ac.uk/images/research_banner_face_lab_290.jpgunderweight or thinness. Interpretation The combined burden of underweight and obesity has increased in most countries, driven by an increase in obesity, while underweight and thinness remain prevalent in south Asia and parts of Africa. A healthy nutrition transition that enhances access to nutritious foods is needed to address the remaining burden of underweight while curbing and reversing the increase in obesity
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