35 research outputs found
Show Me the Money: Does Shared Capitalism Share the Wealth?
This paper examines the effect of a variety of employee stock ownership programs â including ESOPs and broad based stock options â on employeesâ holdings of their employersâ stock, their earnings and their total wealth. Two major datasets are employed: the NBER Shared Capitalism Research Project employee survey dataset and the 2002 national General Social Survey (GSS). Focusing on permanent, full-time employees with at least one year on the job, we find that 87% of employees in the NBER âshared capitalistâ firms, and 36% of employees in the national survey, own their employersâ stock. The NBER employees (including those who hold no company stock) hold an average of 13,200 for employees nationally. We find no evidence â either between datasets or between employees within datasets â of substitution of pay for stock ownership. Employee-owners earn more on average than non-owners, controlling for confounding factors, and report that it would be somewhat more difficult than GSS employees do to find another job that would replace their current pay and benefits. Finally, we find a rough similarity between the distribution of employer stock among the NBER employees (with the top 10% holding two-thirds) and the distribution of all stock among U.S. households (with the top 10% holding three-quarters). Wealth trickles down a little faster in the shared capitalist firms, perhaps, but itâs still just trickling.
Motivating Employee Owners in ESOP Firms: Human Resource Policies and Company Performance
What enables some employee ownership firms to overcome the free rider problem andmotivate employees to improve performance? This study analyzes the role of humanresource policies in the performance of employee ownership companies, using employeesurvey data from 14 companies and a national sample of employee-owners. Between-firmcomparisons of 11 ESOP firms show that an index of human resource policies, nominallycontrolled by management, is positively related to employee reports of co-workerperformance and other good workplace outcomes (including perceptions of fairness, goodsupervision, and worker input and influence). Within-firm comparisons in three ESOP firms,and exploratory results from a national survey, show that employee-owners who participatein employee involvement committees are more likely to exert peer pressure on shirking coworkers.We conclude that an understanding of how and when employee ownership workssuccessfully requires a three-pronged analysis of: 1) the incentives that ownership gives; 2)the participative mechanisms available to workers to act on those incentives; and 3) thecorporate culture which battles against tendencies to free ride.human resources, industrial relations, employee ownership
Show Me the Money: Does Shared Capitalism Share the Wealth?
This paper examines the effect of a variety of employee ownership programs on employees' holdings of their employers' stock, their earnings and their wealth. Two major datasets are employed: the NBER Shared Capitalism Research Project employee survey dataset and the 2002 and 2006 national General Social Surveys (GSS). The GSS national survey shows that 29% of permanent, full-time employees with at least one year on the job own their employers' stock, compared to the unsurprisingly higher 87% of employees in the NBER "shared capitalist" firms. The employees in the national sample hold an average of 52,800 in the NBER sample. Employee owners in NBER companies with broad-based ownership structures fare better: those in majority-owned ESOPs hold on average 283,000. We find no evidence -- either between datasets or between employee-owners and non-owners within datasets -- of substitution of company stock ownership for pay or benefits. Moreover, our analysis suggests that company stock ownership substantially raises total employee wealth, though it appears to have little effect on the overall distribution of wealth. These results suggest that employee ownership tends to raise both ownership stakes and economic resources of American workers across the economic spectrum.
Deuterated formaldehyde in rho Ophiuchi A
From mapping observations of H2CO, HDCO, and D2CO, we have determined how the
degree of deuterium fractionation changes over the central 3'x3' region of rho
Oph A. The multi-transition data of the various H2CO isotopologues, as well as
from other molecules (e.g., CH3OH and N2D+) present in the observed bands, were
analysed using both the standard type rotation diagram analysis and, in
selected cases, a more elaborate method of solving the radiative transfer for
optically thick emission. In addition to molecular column densities, the
analysis also estimates the kinetic temperature and H2 density. Toward the SM1
core in rho Oph A, the H2CO deuterium fractionation is very high. In fact, the
observed D2CO/HDCO ratio is 1.34+/-0.19, while the HDCO/H2CO ratio is
0.107+/-0.015. This is the first time, to our knowledge, that the D2CO/HDCO
abundance ratio is observed to be greater than 1. The kinetic temperature is in
the range 20-30 K in the cores of rho Oph A, and the H2 density is (6-10)x10^5
cm-3. We estimate that the total H2 column density toward the deuterium peak is
(1-4)x10^23 cm-2. As depleted gas-phase chemistry is not adequate, we suggest
that grain chemistry, possibly due to abstraction and exchange reactions along
the reaction chain H2CO -> HDCO -> D2CO, is at work to produce the very high
deuterium levels observed.Comment: 17 pages, 11 figures, accepted for publication in Astronomy &
Astrophysic
The photodissociation and chemistry of CO isotopologues: applications to interstellar clouds and circumstellar disks
Aims. Photodissociation by UV light is an important destruction mechanism for
CO in many astrophysical environments, ranging from interstellar clouds to
protoplanetary disks. The aim of this work is to gain a better understanding of
the depth dependence and isotope-selective nature of this process.
Methods. We present a photodissociation model based on recent spectroscopic
data from the literature, which allows us to compute depth-dependent and
isotope-selective photodissociation rates at higher accuracy than in previous
work. The model includes self-shielding, mutual shielding and shielding by
atomic and molecular hydrogen, and it is the first such model to include the
rare isotopologues C17O and 13C17O. We couple it to a simple chemical network
to analyse CO abundances in diffuse and translucent clouds, photon-dominated
regions, and circumstellar disks.
Results. The photodissociation rate in the unattenuated interstellar
radiation field is 2.6e-10 s^-1, 30% higher than currently adopted values.
Increasing the excitation temperature or the Doppler width can reduce the
photodissociation rates and the isotopic selectivity by as much as a factor of
three for temperatures above 100 K. The model reproduces column densities
observed towards diffuse clouds and PDRs, and it offers an explanation for both
the enhanced and the reduced N(12CO)/N(13CO) ratios seen in diffuse clouds. The
photodissociation of C17O and 13C17O shows almost exactly the same depth
dependence as that of C18O and 13C18O, respectively, so 17O and 18O are equally
fractionated with respect to 16O. This supports the recent hypothesis that CO
photodissociation in the solar nebula is responsible for the anomalous 17O and
18O abundances in meteorites.Comment: Accepted by A&
Molecular line mapping of the giant molecular cloud associated with RCW 106 - III. Multi-molecular line mapping
We present multi-molecular line maps obtained with the Mopra Telescope
towards the southern giant molecular cloud (GMC) complex G333, associated with
the HII region RCW 106. We have characterised the GMC by decomposing the 3D
data cubes with GAUSSCLUMPS, and investigated spatial correlations among
different molecules with principal component analysis (PCA). We find no
correlation between clump size and line width, but a strong correlation between
emission luminosity and line width. PCA classifies molecules into high and low
density tracers, and reveals that HCO+ and N2H+ are anti-correlated.Comment: 24 pages, 21 figures accepted by MNRA
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Recognition of effectors by the bacterial type III secretion system
Pathogenic bacteria possess virulence proteins which are exquisitely tuned to modulate an array of targets. The type III secretion (TTS) system is broadly utilized by Gram-negative bacteria to deliver these effectors into host cells. Yersinia pseudotuberculosis delivers six virulence proteins, called "Yops," into host cells. Yops modulate the inflammatory response, cell survival, and phagocytosis - effective tactics which enable the bacterium to colonize phagocyte-rich lymph nodes. YopE is particularly well-studied, and disables members of the Rho protein family, which are critical to phagocytosis through their modulation of the actin cytoskeleton. Like many TTS effectors, translocation of YopE into host cells requires its prior association with a dedicated chaperone protein within the bacterium. The mechanism by which this association promotes YopE translocation is the topic of this dissertation. I present evidence that SycE promotes a disorder-to-order transition within the chaperone-binding region of YopE. Among the secondary structure generated upon association between SycE and YopE is a [beta]-hairpin in YopE, and I hypothesize that the ordering of specific residues within this domain serves to target the effector to a component of the TTS system required for translocation. Structures of diverse TTS effectors from several species reveal that this motif is conserved, indicating that this motif might function as a general targeting motif. I mutated a set of three or five residues within the YopE [beta]-hairpin and find the translocation of the altered protein to be dramatically reduced. We demonstrate that these mutations do not destabilize the YopE-SycE interaction, and we propose that the ordering of these YopE residues by the chaperone SycE identifies the effector as a substrate for the TTS system. We seek to identify the receptor for this targeting signal, and we present preliminary results on purification of several proteins that might function as receptors for translocation targeting signals. Finally, recommendations are made for future investigation
Recognition of effectors by the bacterial type III secretion system
Pathogenic bacteria possess virulence proteins which are exquisitely tuned to modulate an array of targets. The type III secretion (TTS) system is broadly utilized by Gram-negative bacteria to deliver these effectors into host cells. Yersinia pseudotuberculosis delivers six virulence proteins, called "Yops," into host cells. Yops modulate the inflammatory response, cell survival, and phagocytosis - effective tactics which enable the bacterium to colonize phagocyte-rich lymph nodes. YopE is particularly well-studied, and disables members of the Rho protein family, which are critical to phagocytosis through their modulation of the actin cytoskeleton. Like many TTS effectors, translocation of YopE into host cells requires its prior association with a dedicated chaperone protein within the bacterium. The mechanism by which this association promotes YopE translocation is the topic of this dissertation. I present evidence that SycE promotes a disorder-to-order transition within the chaperone-binding region of YopE. Among the secondary structure generated upon association between SycE and YopE is a [beta]-hairpin in YopE, and I hypothesize that the ordering of specific residues within this domain serves to target the effector to a component of the TTS system required for translocation. Structures of diverse TTS effectors from several species reveal that this motif is conserved, indicating that this motif might function as a general targeting motif. I mutated a set of three or five residues within the YopE [beta]-hairpin and find the translocation of the altered protein to be dramatically reduced. We demonstrate that these mutations do not destabilize the YopE-SycE interaction, and we propose that the ordering of these YopE residues by the chaperone SycE identifies the effector as a substrate for the TTS system. We seek to identify the receptor for this targeting signal, and we present preliminary results on purification of several proteins that might function as receptors for translocation targeting signals. Finally, recommendations are made for future investigation