498 research outputs found
Regression of target organ damage in children and adolescents with primary hypertension
- Author
- Aldona Wierzbicka
- Andrzej Wisniewski
- Anna Niemirska
- C Hanevold
- C Jourdan
- E Lurbe
- E Urbina
- E Wuhl
- G Simone de
- GJ Schwartz
- HR Black
- J Sorof
- Janusz Feber
- JM Sorof
- Joanna Śladowska-Kozlowska
- JT Flynn
- JW Eriksson
- KL McNiece
- M Gutt
- M Litwin
- M Litwin
- M Litwin
- M Litwin
- MB Lande
- Mieczyslaw Litwin
- ML Bots
- National High Blood Pressure Education Program Working Group on High Blood Pressure in Children and Adolescents
- PA Richey
- PR Khoury
- Roman Janas
- S Julius
- S Laurent
- SC Benson
- SR Daniels
- SR Daniels
- SR Daniels
- T Seeman
- TJ Cole
- TM Brady
- Zbigniew T. Wawer
- Publication venue
- Springer-Verlag
- Publication date
- 01/01/2010
- Field of study
We assessed the effects of 12 months of non-pharmacological and pharmacological therapy on 24-h ambulatory blood pressure, regression of target organ damage (TOD) and metabolic abnormalities in 86 children (14.1 ± 2.4 years) with primary hypertension. Twenty-four hour systolic and diastolic blood pressure (BP) decreased (130 ± 8 vs 126 ± 8, 73 ± 7 vs 70 ± 7, p = 0.0001 and 0.004 respectively). Body mass index (BMI) did not change, but waist-to-hip (0.85 ± 0.07 vs 0.83 ± 0.05, p = 0.01) and waist-to-height ratio (WHtR; 0.49 ± 0.07 vs 0.48 ± 0.05, p = 0.008) decreased. Left ventricular mass index (LVMi; 38.5 ± 10.7 vs 35.2 ± 7.5 g/m2.7, p = 0.0001), prevalence of left ventricular hypertrophy (46.5% vs 31.4%; p = 0.0001), carotid intima-media thickness (cIMT; 0.44 ± 0.05 vs 0.42 ± 0.04 mm, p = 0.0001), wall cross sectional area (WCSA; 7.5 ± 1.3 vs 6.9 ± 1.2 mm2, p = 0.002), hsCRP (1.1 ± 1.0 vs 0.7 ± 0.7 mg/l, p = 0.002), and LDL-cholesterol (115 ± 33 vs 107 ± 26 mg/dl, p = 0.001) decreased. Patients who had lowered BP had a lower cIMT at the second examination (0.41 ± 0.04 vs 0.43 ± 0.04 mm, p = 0.04) and lower initial hsCRP values (0.9 ± 0.7 vs 1.5 ± 1.3 mg/l, p = 0.04) in comparison to non-responders. Regression analysis revealed that the main predictor of LVMi decrease was a decrease in abdominal fat expressed as a decrease in waist circumference (WC) (R2 = 0.280, β = 0.558, p = 0.005), for WCSA-SDS a decrease in WC (R2 = 0.332, β = 0.611, p = 0.009) and for a cIMT-SDS decrease the main predictor was a decrease in hsCRP concentrations (R2 = 0.137, β = 0.412, p = 0.03). Standard antihypertensive treatment lowered BP and led to regression of TOD in hypertensive children. Lean body mass increase and decrease in abdominal obesity correlated with TOD regression
Search for new phenomena in final states with an energetic jet and large missing transverse momentum in pp collisions at √ s = 8 TeV with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abud AA
- Abulaiti Y
- Acharya BS
- Achkar B
- Adachi S
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
- Adersberger M
- Adiguzel A
- Adorni S
- Adye T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
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- Aielli G
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- Akesson TPA
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- Al Khoury K
- Alberghi GL
- Albert J
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- Aleksandrov IN
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- Sen S
- Serfon C
- Serin L
- Serkin L
- Sessa M
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- Sevova S
- Sfiligoj T
- Sforza F
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- Shabalina E
- Shahinian JD
- Shaikh NW
- Shan LY
- Shank JT
- Shapiro M
- Sharma A
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- Sherman AD
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- Shimojima M
- Shipsey IPJ
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- Shlomi J
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- Taylor GN
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- Wagner P
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- Wallangen V
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- Wessels M
- Weston TD
- Whalen K
- Whallon NL
- Wharton AM
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- White AS
- White MJ
- Whiteson D
- Whitmore BW
- Wiedenmann W
- Wiel C
- Wielers M
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- Winkels E
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- Wolters H
- Wong VWS
- Woods NL
- Worm SD
- Wosiek BK
- Wozniak KW
- Wraight K
- Wu SL
- Wu X
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- Wyatt TR
- Wynne BM
- Xella S
- Xi Z
- Xia L
- Xiao X
- Xiotidis I
- Xu D
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- Yabsley B
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- Yallup DP
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- Yamamoto A
- Yamatani M
- Yamazaki T
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- Yao W-M
- Yap YC
- Yasu Y
- Yatsenko E
- Ye H
- Ye J
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- Yeletskikh I
- Yexley MR
- Yigitbasi E
- Yildiz HD
- Yorita K
- Yoshihara K
- Young C
- Young CJS
- Yu J
- Yuan R
- Yue X
- Zaazoua M
- Zabinski B
- Zacharis G
- Zaffaroni E
- Zahreddine J
- Zaitsev AM
- Zakareishvili T
- Zakharchuk N
- Zambito S
- Zanzi D
- Zaripovas DR
- Zeissner S
- Zeitnitz C
- Zemaityte G
- Zeng JC
- Zenin O
- Zenis T
- Zerwas D
- Zgubic M
- Zhang B
- Zhang DF
- Zhang G
- Zhang H
- Zhang J
- Zhang L
- Zhang M
- Zhang R
- Zhang S
- Zhang X
- Zhang Y
- Zhang Z
- Zhao P
- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
- Zhou B
- Zhou C
- Zhou M
- Zhou MS
- Zhou N
- Zhou Y
- Zhu C
- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine N
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zivkovic L
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zu Theenhausen HM
- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
Results of a search for new phenomena in final states with an energetic jet and large missing transverse momentum are reported. The search uses 20.3 fb−1 of √ s = 8 TeV data collected in 2012 with the ATLAS detector at the LHC. Events are required to have at least one jet with pT > 120 GeV and no leptons. Nine signal regions are considered with increasing missing transverse momentum requirements between Emiss T > 150 GeV and Emiss T > 700 GeV. Good agreement is observed between the number of events in data and Standard Model expectations. The results are translated into exclusion limits on models with either large extra spatial dimensions, pair production of weakly interacting dark matter candidates, or production of very light gravitinos in a gauge-mediated supersymmetric model. In addition, limits on the production of an invisibly decaying Higgs-like boson leading to similar topologies in the final state are presente
Change in left ventricular geometry during antihypertensive treatment in children with primary hypertension
- Author
- A Toprak
- AF Lever
- Aldona Wierzbicka
- Anna Niemirska
- B Gutin
- C Hanevold
- CJ Lavie
- D Levy
- DJ Sahn
- DR Matthews
- E Lurbe
- E Urbina
- F Assadi
- G de Simone
- JM Sorof
- JM Sorof
- Joanna Śladowska-Kozłowska
- JS Drukteinis
- JT Flynn
- KL McNiece
- M Litwin
- M Litwin
- M Litwin
- Mieczysław Litwin
- MJ Koren
- ML Muiesan
- National High Blood Pressure Education Program Working Group on High Blood Pressure in Children and Adolescents
- PA Richey
- PR Khoury
- RB Devereux
- Roman Janas
- SR Daniels
- SR Daniels
- T McLaughlin
- T Seeman
- TJ Cole
- TM Brady
- Verdecchia P Carini G, Circo A, Dovellini E, Giovannini E, Lombardo M, Solinas P, Gorini M, Maggioni AP
- W Grossman
- WL Border
- Wühl E Witte K, Soergel M, Mehls O, Shaefer F, German Working Group on Pediatric Hypertension
- Zbigniew T. Wawer
- Publication venue
- Springer-Verlag
- Publication date
- 01/01/2011
- Field of study
The pattern of the left ventricle (LV) has important significance in adults with hypertension. The aim of the present study was to analyze changes and determinants of LV geometry after 1 year of antihypertensive treatment in children with primary hypertension (PH) in relation to metabolic abnormalities and anthropometrical parameters. In 86 children (14.1 ± 2.4 years) with newly diagnosed PH, LV geometry and biochemical parameters before and after 12 months of standard antihypertensive therapy were assessed. At baseline, normal LV geometry (NG) was found in 42 (48.9%), concentric remodeling (CR) in 4 (4.6%), concentric hypertrophy (CH) in 8 (9.3%), and eccentric hypertrophy (EH) in 32 (37.2%) patients. The prevalence of NG in patients with severe hypertension was significantly lower than in patients with ambulatory hypertension. There were no differences in dipping status in relation to LV geometry. Patients with CH and EH were more viscerally obese than patients with NG. Patients with CH had higher diastolic blood pressure in comparison with EH patients (p < 0.05). The main predictor of relative wall thickness (RWT) was the triglycerides to high density lipoprotein cholesterol (TG/HDL) ratio (R2 = 0.319, β = 0.246, p = 0.004). Patients received 12 months of antihypertensive treatment, either lifestyle modification only (n = 37) or lifestyle modification plus antihypertensive medications (n = 49) if severe ambulatory hypertension or target organ damage were present. After 12 months of treatment the prevalence of EH (37.2% vs 18.6%, p = 0.003) decreased but prevalence of CH did not change. Patients in whom RWT decreased also decreased waist circumference and TG/HDL; the main predictor of RWT decrease was a decrease of the TG/HDL ratio (β = 0.496, R2 = 0.329, p = 0.002). In adolescents with PH, LV geometry is related to central obesity and insulin resistance. Decrease of abdominal obesity and insulin resistance are the most important predictors of normalization of LV geometry, however CH has lower potential to normalize LV geometry
IL-2 Mediates CD4+ T Cell Help in the Breakdown of Memory-Like CD8+ T Cell Tolerance under Lymphopenic Conditions
- Author
- A Khoruts
- A Le Bon
- A Le Bon
- A Schildknecht
- AW Goldrath
- AW Goldrath
- B Ernst
- B Knoechel
- B Scott
- BK Cho
- C Le Saout
- C Viret
- Chantal Jacquet
- CL Ahonen
- Clémence Cabasse
- CM Paulos
- Cécile Le Saout
- D Kamimura
- D Lo
- Derya Unutmaz
- DJ Morgan
- EB Wilson
- F Castellino
- G Verdeil
- GM Behrens
- J Hernandez
- J Hernandez
- J Waithman
- JA Keene
- Javier Hernandez
- JD Fontenot
- JD Phelan
- JJ Obar
- JP Ridge
- JT Tan
- K Lahl
- K Murali-Krishna
- K Staveley-O'Carroll
- KP Cheung
- KS Schluns
- LM D'Cruz
- M Prlic
- MA Williams
- Marine Villard
- ME Dudley
- ME Pipkin
- MF Bachmann
- ML Janas
- Naomi Taylor
- O Boyman
- PA Gleeson
- PS Ohashi
- Q Tang
- R Setoguchi
- RJ Noelle
- RS McHugh
- S Brode
- S Read
- S Sakaguchi
- SE Hamilton
- SE Hamilton
- SP Schoenberger
- SR Bennett
- T Calzascia
- TM Kundig
- TR Malek
- V Kalia
- W Dummer
- WC Kieper
- WC Kieper
- WL Redmond
- Y Guilloux
- Z Lu
- Z Wu
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2010
- Field of study
Background: Lymphopenia results in the proliferation and differentiation of naïve T cells into memory-like cells in the apparent absence of antigenic stimulation. This response, at least in part due to a greater availability of cytokines, is thought to promote anti-self responses. Although potentially autoreactive memory-like CD8 + T cells generated in a lymphopenic environment are subject to the mechanisms of peripheral tolerance, they can induce autoimmunity in the presence of antigen-specific memory-like CD4 + T helper cells. Methodology/Principal Findings: Here, we studied the mechanisms underlying CD4 help under lymphopenic conditions in transgenic mice expressing a model antigen in the beta cells of the pancreas. Surprisingly, we found that the self-reactivity mediated by the cooperation of memory-like CD8 + and CD4 + T cells was not abrogated by CD40L blockade. In contrast, treatment with anti-IL-2 antibodies inhibited the onset of autoimmunity. IL-2 neutralization prevented the CD4-mediated differentiation of memory-like CD8 + T cells into pathogenic effectors in response to self-antigen cross-presentation. Furthermore, in the absence of helper cells, induction of IL-2 signaling by an IL-2 immune complex was sufficient to promote memory-like CD8 + T cell self-reactivity. Conclusions/Significance: IL-2 mediates the cooperation of memory-like CD4 + and CD8 + T cells in the breakdown of crosstolerance, resulting in effector cytotoxic T lymphocyte differentiation and the induction of autoimmune disease
PI3Kδ and primary immunodeficiencies.
- Author
- A Alkhatib
- A Suzuki
- A Suzuki
- AK Gopal
- Alison M. Condliffe
- AM Condliffe
- Anita Chandra
- B Boisson
- B Vanhaesebroeck
- BF Nashed
- BS Jaiswal
- C Schroeder
- C Thauvin-Robinet
- CA Durand
- Carrie L. Lucas
- CL Lucas
- CL Lucas
- D Dominguez-Sola
- D Liu
- DA Dyment
- DA Fruman
- DA Fruman
- DL Barber
- DR Soond
- DR Soond
- DT Gracias
- DT Patton
- E Aksoy
- E Ciraolo
- E Clayton
- E Elkaim
- E Sapey
- EM Putz
- F Ramadani
- GD Victora
- GM Delgoffe
- H Suzuki
- HN Hartman
- HS Dengler
- I Angulo
- J Rolf
- JE Burke
- JL Hukelmann
- JM Brenchley
- K Ali
- K Araki
- K Kinoshita
- K Okkenhaug
- K Okkenhaug
- K Okkenhaug
- KJ Zhang
- KK Chudasama
- Klaus Okkenhaug
- KS Hathcock
- LM Webb
- LR Borlado
- LV Sinclair
- M Elgizouli
- M Gigoux
- M Kuhlen
- M Llorian
- MC Crank
- MC Deau
- ME Conley
- ME Urick
- MJ Browning
- ML Janas
- MV Kim
- PT Hawkins
- R Di Fonte
- R Fritsch
- RR Furman
- S Haylock-Jacobs
- S Kracker
- S Kulkarni
- S Petrovski
- S Sander
- S Shojaee
- SA Omori
- SE Coutre
- Sergey Nejentsev
- SM Kaech
- SM O'Brien
- ST Jou
- ST Jou
- TT Zhang
- V Lougaris
- VQ Pearce
- W Ouyang
- Y Kurebayashi
- Publication venue
- Nat Rev Immunol
- Publication date
- 12/09/2016
- Field of study
Primary immunodeficiencies are inherited disorders of the immune system, often caused by the mutation of genes required for lymphocyte development and activation. Recently, several studies have identified gain-of-function mutations in the phosphoinositide 3-kinase (PI3K) genes PIK3CD (which encodes p110δ) and PIK3R1 (which encodes p85α) that cause a combined immunodeficiency syndrome, referred to as activated PI3Kδ syndrome (APDS; also known as p110δ-activating mutation causing senescent T cells, lymphadenopathy and immunodeficiency (PASLI)). Paradoxically, both loss-of-function and gain-of-function mutations that affect these genes lead to immunosuppression, albeit via different mechanisms. Here, we review the roles of PI3Kδ in adaptive immunity, describe the clinical manifestations and mechanisms of disease in APDS and highlight new insights into PI3Kδ gleaned from these patients, as well as implications of these findings for clinical therapy
PI3K Signaling in Normal B Cells and Chronic Lymphocytic Leukemia (CLL).
- Author
- A Alkhatib
- A Bilancio
- A Burkle
- A Castello
- A Patke
- A Philippen
- A Rosenwald
- A Suzuki
- A. Lanemo Myhrinder
- AL Shaffer
- AM Buhl
- AM Scharenberg
- AV Miletic
- B Moser
- BM Broker
- BT Messmer
- BY Chang
- C Calissano
- CA Durand
- CC Chu
- DA Fruman
- DA Tuveson
- DR Soond
- DT Patton
- E Arana
- E Clayton
- E Schweighoffer
- EC Steinbach
- F Bennett
- F Castellino
- F Fais
- F Mackay
- FK Stevenson
- HA Dbouk
- Harumi Suzuki
- HS Dengler
- Huimin Geng
- I Sagiv-Barfi
- J Eberlein
- J Hoellenriegel
- J Hoellenriegel
- J Jellusova
- J Rolf
- J. C. Byrd
- JA Burger
- JA Burger
- JA Burger
- JA Burger
- JA Burger
- JA Burger
- JA Dubovsky
- JA Woyach
- JC Byrd
- JC Byrd
- Jennifer K. Uno
- JJ Campbell
- JJ Limon
- JR Brown
- JW Friedberg
- K Ali
- K Heinig
- K Okkenhaug
- K Okkenhaug
- K Okkenhaug
- K Okkenhaug
- K Okkenhaug
- K Reif
- K Stamatopoulos
- KL Otipoby
- L Borche
- L Srinivasan
- LR Pearce
- M Binder
- M Binder
- M Herve
- M Sivina
- M Spaargaren
- M. P. Quiroga
- Marcus Dühren-von Minden
- MF Rooij de
- ML Janas
- MM Al-Alwan
- N Chiorazzi
- N Chiorazzi
- N Chiorazzi
- NE Harwood
- P Baliakas
- P Ghia
- PE Patten
- R Catera
- R Hoogeboom
- R Krzysiek
- RC Rickert
- RH Amin
- RN Damle
- RR Furman
- RS Bystry
- S Burgler
- S Fiorcari
- S Iyengar
- S Matsuda
- S Ponader
- S Ponader
- S. Iacovelli
- SE Herman
- Sidne A. Omori
- SS Chen
- T Henley
- T Kurosaki
- T Yamazaki
- TA Springer
- TJ Hamblin
- TJ Schall
- TT Zhang
- TT Zhang
- Y Aiba
- Y Herishanu
- Y Wang
- ZM Sthoeger
- Publication venue
- Curr Top Microbiol Immunol
- Publication date
- 09/09/2015
- Field of study
B cells provide immunity to extracellular pathogens by secreting a diverse repertoire of antibodies with high affinity and specificity for exposed antigens. The B cell receptor (BCR) is a transmembrane antibody, which facilitates the clonal selection of B cells producing secreted antibodies of the same specificity. The diverse antibody repertoire is generated by V(D)J recombination of heavy and light chain genes, whereas affinity maturation is mediated by activation-induced cytidine deaminase (AID)-mediated mutagenesis. These processes, which are essential for the generation of adaptive humoral immunity, also render B cells susceptible to chromosomal rearrangements and point mutations that in some cases lead to cancer. In this chapter, we will review the central role of PI3K s in mediating signals from the B cell receptor that not only facilitate the development of functional B cell repertoire, but also support the growth and survival of neoplastic B cells, focusing on chronic lymphocytic leukemia (CLL) B cells. Perhaps because of the central role played by PI3K in BCR signaling, B cell leukemia and lymphomas are the first diseases for which a PI3K inhibitor has been approved for clinical use
RNA delivery by extracellular vesicles in mammalian cells and its applications.
- Author
- A Ahadi
- A Buonfiglioli
- A Datta
- A Gori
- A Herrmann
- A Hoshino
- A Iavello
- A Khvorova
- A Longatti
- A Mondal
- A Montecalvo
- A Morales-Kastresana
- A Rustom
- A Savina
- A Varcianna
- A Wittrup
- A Yeri
- A Zomer
- AD Flynn
- AD Grabarek
- AF Saleh
- AH Van Craenenbroeck
- AJ McKenzie
- AK Rupp
- AL Blasius
- AM Leidal
- AM Marleau
- AR Soares
- AT Da Poian
- B Escudier
- B György
- B György
- B Mannerström
- B Mateescu
- B Meehan
- B Shen
- BH Sung
- BM Lehrich
- BN Hannafon
- BWM Van Balkom
- BY Nabet
- C Eliscovich
- C Escrevente
- C Geoffroy
- C Gong
- C Grange
- C Lässer
- C Ma
- C Meyer
- C Théry
- C Villarroya-Beltri
- C Williams
- C Williams
- CA Thompson
- CC Chen
- CG Liu
- CM Di Liegro
- CP Lai
- CP Lai
- D Choi
- D De Gonzalo-Calvo
- D Fanale
- D Granchi
- D Hoshino
- D Planchon
- D Shukla
- DD Yanshina
- DE Murphy
- DJ Gibbings
- DK Jeppesen
- DK Struck
- DM Jones
- DX Zhang
- E Reátegui
- E Rohde
- E Tutucci
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- ED Pastuzyn
- EJ Im
- EM Guerreiro
- EN Hoen
- ENM Nolte’T Hoen
- ER Abels
- F Chen
- F De La Cuesta
- F Ghamloush
- F Gámbaro
- F Hobor
- F Kenneweg
- F Pi
- F Royo
- FJ Giles
- FM Khan
- G Haimovich
- G Van Niel
- H Goodarzi
- H Persson
- H Pollet
- H Ragelle
- H Shao
- H Sork
- H Valadi
- H Yu
- H Yuhua
- H Zhang
- H Zhang
- H Zhang
- HC Christianson
- HM van Dongen
- HW King
- I Nakase
- I Nakase
- I Nakase
- I Parolini
- I Prada
- IKH Poon
- J Ashley
- J Grove
- J Guduric-Fuchs
- J He
- J Jiang
- J Karttunen
- J Kowal
- J Liu
- J Matsuzaki
- J McKiernan
- J Schorey
- J Skog
- J Van Deun
- J Van Deun
- J Wang
- J Wang
- J Zou
- JA Tsatsaronis
- JC Akers
- JC Akers
- JD Arroyo
- JD Martin-Rufino
- JE Freedman
- JF Quinn
- JM Rimer
- JM Wahlberg
- JPK Armstrong
- JR Chevillet
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- JR Schnell
- JW Clancy
- K Burgos
- K Ekström
- K Glebov
- K Hagiwara
- K Koga
- K Lee
- K Miyauchi
- K Mukherjee
- K Otake
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- K Yin
- KC Vickers
- KM Danielson
- KW Witwer
- L Alvarez-Erviti
- L Antoury
- L Morel
- L Pigati
- L Santangelo
- L Statello
- L Wang
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- LA Beninson
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- M Alexander
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- M Gimona
- M Li
- M Mathieu
- M Maugeri
- M Mendt
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- M Mittelbrunn
- M Miyanishi
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- M Shen
- M Tkach
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- MJ Shurtleff
- MJ Shurtleff
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- MP Hardy
- MP Zaborowski
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- N Heath
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- OPB Wiklander
- P Gangadaran
- P Gaurivaud
- P Lönn
- P Song
- PE Mangeot
- PJ Quesenberry
- PM Godoy
- PM Godoy
- Q Liu
- Q Wang
- Q Wang
- Q Zhang
- R Bayraktar
- R Crescitelli
- R Horos
- R Kojima
- R Lucero
- R Munagala
- R Shah
- R Xu
- RA Haraszti
- RA Haraszti
- RF D’souza
- RK Tweten
- RL Maute
- S Atienzar-Aroca
- S Bala
- S Caruso
- S Chen
- S Das
- S Gerstberger
- S Kamerkar
- S Sinha
- S Srinivasan
- SA Melo
- SAA Kooijmans
- SAA Kooijmans
- SAA Kooijmans
- SAA Kooijmans
- SC Jang
- SG Antimisiaris
- SI Ohno
- SK Chakrabortty
- SM Kim
- SM Lehmann
- SN Hurwitz
- SR Baglio
- SR Baglio
- T Janas
- T Thomou
- T Tian
- TAP Driedonks
- TG Iversen
- TJ Antes
- TN Bukong
- TN Lamichhane
- V Bhaskaran
- V Ramakrishnaiah
- V Toribio
- VD Atayde
- VR Minciacchi
- W Aoi
- W Heusermann
- W Ying
- WD Gray
- WM Usman
- WP Kuo
- X Gao
- X Huang
- X Liu
- X Zhu
- X Zou
- Y Lin
- Y Long
- Y Ma
- Y Tao
- Y Teng
- Y Wang
- YY Jhan
- Z Feng
- Z Wei
- Z Wei
- ZW Luo
- Publication venue
- eScholarship, University of California
- Publication date
- 01/10/2020
- Field of study
The term 'extracellular vesicles' refers to a heterogeneous population of vesicular bodies of cellular origin that derive either from the endosomal compartment (exosomes) or as a result of shedding from the plasma membrane (microvesicles, oncosomes and apoptotic bodies). Extracellular vesicles carry a variety of cargo, including RNAs, proteins, lipids and DNA, which can be taken up by other cells, both in the direct vicinity of the source cell and at distant sites in the body via biofluids, and elicit a variety of phenotypic responses. Owing to their unique biology and roles in cell-cell communication, extracellular vesicles have attracted strong interest, which is further enhanced by their potential clinical utility. Because extracellular vesicles derive their cargo from the contents of the cells that produce them, they are attractive sources of biomarkers for a variety of diseases. Furthermore, studies demonstrating phenotypic effects of specific extracellular vesicle-associated cargo on target cells have stoked interest in extracellular vesicles as therapeutic vehicles. There is particularly strong evidence that the RNA cargo of extracellular vesicles can alter recipient cell gene expression and function. During the past decade, extracellular vesicles and their RNA cargo have become better defined, but many aspects of extracellular vesicle biology remain to be elucidated. These include selective cargo loading resulting in substantial differences between the composition of extracellular vesicles and source cells; heterogeneity in extracellular vesicle size and composition; and undefined mechanisms for the uptake of extracellular vesicles into recipient cells and the fates of their cargo. Further progress in unravelling the basic mechanisms of extracellular vesicle biogenesis, transport, and cargo delivery and function is needed for successful clinical implementation. This Review focuses on the current state of knowledge pertaining to packaging, transport and function of RNAs in extracellular vesicles and outlines the progress made thus far towards their clinical applications
Measurement of the tt¯tt¯ production cross section in pp collisions at √s=13 TeV with the ATLAS detector
- Author
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- Zhemchugov A
- Zheng Z
- Zhong D
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- Zhu Y
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- Zhukov K
- Zhulanov V
- Zieminska D
- Zimine NI
- Zimmermann S
- Ziolkowski M
- Zoccoli A
- Zoch K
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- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 16/11/2021
- Field of study
A measurement of four-top-quark production using proton-proton collision data at a centre-of-mass energy of 13 TeV collected by the ATLAS detector at the Large Hadron Collider corresponding to an integrated luminosity of 139 fb−1 is presented. Events are selected if they contain a single lepton (electron or muon) or an opposite-sign lepton pair, in association with multiple jets. The events are categorised according to the number of jets and how likely these are to contain b-hadrons. A multivariate technique is then used to discriminate between signal and background events. The measured four-top-quark production cross section is found to be 26+17−15 fb, with a corresponding observed (expected) significance of 1.9 (1.0) standard deviations over the background-only hypothesis. The result is combined with the previous measurement performed by the ATLAS Collaboration in the multilepton final state. The combined four-top-quark production cross section is measured to be 24+7−6 fb, with a corresponding observed (expected) signal significance of 4.7 (2.6) standard deviations over the background-only predictions. It is consistent within 2.0 standard deviations with the Standard Model expectation of 12.0 ± 2.4 fb
Measurement and interpretation of same-sign W boson pair production in association with two jets in pp collisions at s = 13 TeV with the ATLAS detector
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- Abbott B
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- Abicht NJ
- Abidi SH
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- Abusleme Hoffman AC
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- Zhang Z
- Zhao H
- Zhao P
- Zhao T
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zheng J
- Zheng K
- Zheng X
- Zheng Z
- Zhong D
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- Zhou Y
- Zhu CG
- Zhu J
- Zhu Y
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 01/04/2024
- Field of study
This paper presents the measurement of fducial and diferential cross sections for both the inclusive and electroweak production of a same-sign W-boson pair in association with two jets (W±W±jj) using 139 fb−1 of proton-proton collision data recorded at a centre-of-mass energy of √s = 13 TeV by the ATLAS detector at the Large Hadron Collider. The analysis is performed by selecting two same-charge leptons, electron or muon, and at least two jets with large invariant mass and a large rapidity diference. The measured fducial cross sections for electroweak and inclusive W±W±jj production are 2.92 ± 0.22 (stat.) ± 0.19 (syst.)fb and 3.38±0.22 (stat.)±0.19 (syst.)fb, respectively, in agreement with Standard Model predictions. The measurements are used to constrain anomalous quartic gauge couplings by extracting 95% confdence level intervals on dimension-8 operators. A search for doubly charged Higgs bosons H±± that are produced in vector-boson fusion processes and decay into a same-sign W boson pair is performed. The largest deviation from the Standard Model occurs for an H±± mass near 450 GeV, with a global signifcance of 2.5 standard deviations
Measurements of W+W−+ ≥ 1 jet production cross-sections in pp collisions at s = 13 TeV with the ATLAS detector
- Author
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- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/06/2021
- Field of study
Fiducial and differential cross-section measurements of W+W− production in
association with at least one hadronic jet are presented. These measurements are sensitive
to the properties of electroweak-boson self-interactions and provide a test of perturbative
quantum chromodynamics and the electroweak theory. The analysis is performed using
proton-proton collision data collected at p
s = 13TeV with the ATLAS experiment, corresponding
to an integrated luminosity of 139 fb−1. Events are selected with exactly one oppositely
charged electron-muon pair and at least one hadronic jet with a transverse momentum
of pT > 30 GeV and a pseudorapidity of |�| < 4.5. After subtracting the background
contributions and correcting for detector effects, the jet-inclusive W+W−+ � 1 jet fiducial
cross-section and W+W−+ jets differential cross-sections with respect to several kinematic
variables are measured. These measurements include leptonic quantities, such as the lepton
transverse momenta and the transverse mass of the W+W− system, as well as jet-related
observables such as the leading jet transverse momentum and the jet multiplicity. Limits on
anomalous triple-gauge-boson couplings are obtained in a phase space where interference
between the Standard Model amplitude and the anomalous amplitude is enhanced
- …