365 research outputs found
Monocyte Subset Dynamics in Human Atherosclerosis Can Be Profiled with Magnetic Nano-Sensors
- Author
- A Schlitt
- A Stroh
- A Szaflarska
- Aleksey Chudnovskiy
- B Passlick
- C Auffray
- C Murdoch
- C Schinkel
- C Ulrich
- C Weber
- DF Waterhouse
- F Tacke
- Filip K. Swirski
- FK Swirski
- FK Swirski
- G An
- G Draude
- G Fingerle
- GH Simon
- GK Hansson
- H Lee
- H Schmidt
- Hakho Lee
- HW Ziegler-Heitbrock
- JH Lee
- Kevin Croce
- L Josephson
- L Pulliam
- M Nahrendorf
- Martin Etzrodt
- Matthias Nahrendorf
- MG Harisinghani
- Mikael J. Pittet
- Moritz Wildgruber
- P Libby
- P Libby
- P Wunderbaldinger
- Peter Libby
- R Dragu
- R Weissleder
- Ralph Weissleder
- S Gordon
- S Metz
- SW Waldo
- Tae-Jong Yoon
- Terry Means
- Publication venue
- Public Library of Science
- Publication date
- 01/05/2009
- Field of study
Monocytes are circulating macrophage and dendritic cell precursors that populate healthy and diseased tissue. In humans, monocytes consist of at least two subsets whose proportions in the blood fluctuate in response to coronary artery disease, sepsis, and viral infection. Animal studies have shown that specific shifts in the monocyte subset repertoire either exacerbate or attenuate disease, suggesting a role for monocyte subsets as biomarkers and therapeutic targets. Assays are therefore needed that can selectively and rapidly enumerate monocytes and their subsets. This study shows that two major human monocyte subsets express similar levels of the receptor for macrophage colony stimulating factor (MCSFR) but differ in their phagocytic capacity. We exploit these properties and custom-engineer magnetic nanoparticles for ex vivo sensing of monocytes and their subsets. We present a two-dimensional enumerative mathematical model that simultaneously reports number and proportion of monocyte subsets in a small volume of human blood. Using a recently described diagnostic magnetic resonance (DMR) chip with 1 µl sample size and high throughput capabilities, we then show that application of the model accurately quantifies subset fluctuations that occur in patients with atherosclerosis
Commercial Immunoglobulin Products Contain Neutralizing Antibodies Against Severe Acute Respiratory Syndrome Coronavirus 2 Spike Protein
- Author
- Beale Rupert CL
- Burns Siobhan O
- Carr Edward J
- Chauhan Neil
- Dowgier Giulia
- Gandhi Sonia
- Glanville Julie M
- Hanson Steven
- Hart Jennifer CL
- Herman Lou S
- Hobbs Agnieszka
- Lee Simon FK
- Lowe David M
- McCoy Laura
- Moreira Fernando
- Neave Lucy
- O'Sullivan Mary
- Ragno Martina
- Ravichandran Sriram
- Shah Devesh
- Sharma Sonal
- Symes Andrew
- Tadros Susan
- Townsend Katie
- Tynan Aoife
- Upasani Vinit
- Wall Emma C
- Workman Sarita
- Wu Mary Y
- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- 20/06/2023
- Field of study
BACKGROUND: Patients with antibody deficiency respond poorly to COVID-19 vaccination and are at risk of severe or prolonged infection. They are given long-term immunoglobulin replacement therapy (IRT) prepared from healthy donor plasma to confer passive immunity against infection. Following widespread COVID-19 vaccination alongside natural exposure, we hypothesised that immunoglobulin preparations will now contain neutralising SARS-CoV-2 spike antibodies which confer protection against COVID-19 disease and may help to treat chronic infection. METHODS: We evaluated anti-SARS-CoV-2 spike antibody in a cohort of patients before and after immunoglobulin infusion. Neutralising capacity of patient samples and immunoglobulin products was assessed using in vitro pseudo-virus and live-virus neutralisation assays, the latter investigating multiple batches against current circulating omicron variants. We describe the clinical course of nine patients started on IRT during treatment of COVID-19. RESULTS: In 35 individuals with antibody deficiency established on IRT, median anti-spike antibody titre increased from 2123 to 10600 U/ml post-infusion, with corresponding increase in pseudo-virus neutralisation titres to levels comparable to healthy donors. Testing immunoglobulin products directly in the live-virus assay confirmed neutralisation, including of BQ1.1 and XBB variants, but with variation between immunoglobulin products and batches.Initiation of IRT alongside Remdesivir in patients with antibody deficiency and prolonged COVID-19 infection (median 189 days, maximum over 900 days with an ancestral viral strain) resulted in clearance of SARS-CoV-2 virus at a median of 20 days. CONCLUSIONS: Immunoglobulin preparations now contain neutralising anti-SARS-CoV-2 antibodies which are transmitted to patients and help to treat COVID-19 in individuals with failure of humoral immunity
ABHD4-dependent developmental anoikis safeguards the embryonic brain
- Author
- A Bulfone
- B Dudok
- B Tasic
- C Cserép
- C Englund
- C Gil-Sanz
- C Kintner
- CA Brady
- CB Forsyth
- CD Simpson
- CJ Wilhelm
- DG Southwell
- E Aaku-Saraste
- E Soliman
- E Taverna
- ES Boyden
- FK Wong
- FK Wong
- G Camargo Ortega
- GM Simon
- H Kitano
- HC Lee
- HHO Schmid
- I Pastushenko
- J Morishita
- J Zhang
- JC Domingo
- JG Camp
- JM Whitacre
- JP McAllister
- K Lefkovics
- KB Leszczynska
- KJ Yoon
- L Barna
- L Galluzzi
- M Baranyi
- M Götz
- M Kielar
- M Maccarrone
- M Mayer
- M Miller
- ML Sutherland
- MT Nieman
- N Tamamaki
- NT Woods
- P Malatesta
- P Paoli
- P Rakic
- R Insolera
- R Miñambres
- R Nyilas
- RD Hodge
- RJ Ferland
- S Frisch
- S Govindan
- S Ishii
- S Kumar
- S Tavano
- SA Fietz
- SC Noctor
- SM Frisch
- T Fujimori
- T Matsuda
- T Namba
- W Humphrey
- Y Itoh
- Y Yokota
- YC Lin
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/08/2020
- Field of study
During embryonic development, neural progenitor cells undergo numerous cell divisions. Here, the authors show that ABHD4-mediated developmental anoikis distinguishes the physiological delamination and the pathological detachment of progenitor cells with relevance to fetal alcohol-induced apoptosis
Chemical–Genetic Profiling of Imidazo[1,2-a]pyridines and -Pyrimidines Reveals Target Pathways Conserved between Yeast and Human Cells
- Author
- A Anaflous
- A Eastman
- A Gueiffier
- A Gueiffier
- A Sacchi
- A Sancar
- AB Parsons
- AB Parsons
- Abdellah Hamal
- Abderrahmane Anaflous
- Andres Lopez
- Angus McQuibban
- AS Basile
- B Stevens
- B Westermann
- Brahim El Bali
- C Tucker
- C Zhang
- CA Weber
- CB Brachmann
- CD Armour
- Charles Boone
- CM Grant
- CM Grant
- Corey Nislow
- DE Lea
- DJ Jamieson
- E Ohler
- EA Winzeler
- Elke Ericson
- EM Gozukara
- ES Goldring
- F Sherman
- F Sherman
- F Thoma
- FK Zimmermann
- G Giaever
- G Giaever
- G Giaever
- GG Danagulyan
- GR Revanker
- Grant W. Brown
- Guri Giaever
- GW Thorpe
- H Kappus
- H Niida
- HI Wu
- HP Hoffmann
- J Fillingham
- J Flattery-O'Brien
- J Heitman
- JA Simon
- JB Allen
- JC Harrison
- JH Houtgraaf
- JM Davies
- JS Armstrong
- K Baetz
- KF Byth
- KP Mahon
- KS Gudmundsson
- Lawrence E. Heisler
- Lisa Yu
- LP Collinson
- LR Kelland
- M Crompton
- M Fousteri
- M Karbowski
- M Monaco
- M Protic-Sabljic
- M Stucki
- Michael Snyder
- Mohammed Bellaoui
- MS Cyert
- MS Cyert
- P Lehoczky
- PY Lum
- R Pabla
- RM Costa
- S Clements-Jewery
- S Izawa
- SC Shuck
- SE Pierce
- T Swainston Harrison
- T Ueda
- V Beljanski
- W Lee
- WP Blackaby
- WW Paudler
- WW Paudler
- Y Rival
- Y Rival
- Y Sanchez
- Z Sun
- Publication venue
- Public Library of Science
- Publication date
- 01/11/2008
- Field of study
Small molecules have been shown to be potent and selective probes to understand cell physiology. Here, we show that imidazo[1,2-a]pyridines and imidazo[1,2-a]pyrimidines compose a class of compounds that target essential, conserved cellular processes. Using validated chemogenomic assays in Saccharomyces cerevisiae, we discovered that two closely related compounds, an imidazo[1,2-a]pyridine and -pyrimidine that differ by a single atom, have distinctly different mechanisms of action in vivo. 2-phenyl-3-nitroso-imidazo[1,2-a]pyridine was toxic to yeast strains with defects in electron transport and mitochondrial functions and caused mitochondrial fragmentation, suggesting that compound 13 acts by disrupting mitochondria. By contrast, 2-phenyl-3-nitroso-imidazo[1,2-a]pyrimidine acted as a DNA poison, causing damage to the nuclear DNA and inducing mutagenesis. We compared compound 15 to known chemotherapeutics and found resistance required intact DNA repair pathways. Thus, subtle changes in the structure of imidazo-pyridines and -pyrimidines dramatically alter both the intracellular targeting of these compounds and their effects in vivo. Of particular interest, these different modes of action were evident in experiments on human cells, suggesting that chemical–genetic profiles obtained in yeast are recapitulated in cultured cells, indicating that our observations in yeast can: (1) be leveraged to determine mechanism of action in mammalian cells and (2) suggest novel structure–activity relationships
Worldwide trends in diabetes since 1980: a pooled analysis of 751 population-based studies with 4.4 million participants
- Author
- Abdeen ZA
- Abu-Rmeileh NM
- Acosta-Cazares B
- Adams R
- Aekplakorn W
- Aguilar-Salinas CA
- Agyemang C
- Ahmadvand A
- Al Nsour M
- Al-Othman AR
- Ali MK
- Alkerwi A
- Amouyel P
- Amuzu A
- Andersen LB
- Anderssen SA
- Anjana RM
- Aounallah-Skhiri H
- Aris T
- Arlappa N
- Arveiler D
- Assah FK
- Avdicova M
- Azizi F
- Balakrishna N
- Bandosz P
- Barbagallo CM
- Barcelo A
- Batieha AM
- Baur LA
- Ben Romdhane H
- Ben Romdhane H
- Benet M
- Bennett JE
- Bentham J
- Bernabe-Ortiz A
- Bharadwaj S
- Bhargava SK
- Bi Y
- Bixby H
- Bjerregaard P
- Bjerregaard P
- Bjertness E
- Bjertness MB
- Bjorkelund C
- Blokstra A
- Bo S
- Boehm BO
- Boissonnet CP
- Bonet Gorbea M
- Bovet P
- Brajkovich I
- Breckenkamp J
- Brenner H
- Brewster LM
- Brian GR
- Bruno G
- Bugge A
- Cabrera de Leon A
- Can G
- Candido APC
- Capuano V
- Carlsson AC
- Carvalho MJ
- Casanueva FF
- Casas J-P
- Caserta CA
- Castetbon K
- Chamukuttan S
- Chaturvedi N
- Chen C-J
- Chen F
- Chen S
- Cheng C-Y
- Chetrit A
- Chiou S-T
- Cho Y
- Chudek J
- Cifkova R
- Claessens F
- Concin H
- Cooper C
- Cooper R
- Costanzo S
- Cottel D
- Cowan MJ
- Cowell C
- Crujeiras AB
- D'Arrigo G
- Dallongeville J
- Danaei G
- Dankner R
- Dauchet L
- de Gaetano G
- De Henauw S
- Deepa M
- Dehghan A
- del Cristo Rodriguez-Perez M
- Deschamps V
- Dhana K
- Di Castelnuovo AF
- Di Cesare M
- Djalalinia S
- dos Santos RN
- Doua K
- Drygas W
- Du Y
- Dzerve V
- Egbagbe EE
- Eggertsen R
- El Ati J
- Elosua R
- Erasmus RT
- Erem C
- Ergor G
- Eriksen L
- Escobedo-de la Pena J
- Eugenio Lozano J
- Ezzati M
- Fall CH
- Farzadfar F
- Felix-Redondo FJ
- Ferguson TS
- Fernanda Lima-Costa M
- Fernandez-Berges D
- Ferrari M
- Ferreccio C
- Feskens EJM
- Finn JD
- Foeger B
- Foo LH
- Forslund A-S
- Fouad HM
- Fouad HM
- Francis DK
- Francisco Miquel J
- Franco MDC
- Franco OH
- Frontera G
- Furusawa T
- Gaciong Z
- Garnett SP
- Gaspoz J-M
- Gasull M
- Gates L
- Geleijnse JM
- Ghasemian A
- Ghimire A
- Giampaoli S
- Gianfagna F
- Giovannelli J
- Giwercman A
- Gonzalez Gross M
- Gonzalez Rivas JP
- Gottrand F
- Grafnetter D
- Grodzicki T
- Grontved A
- Gruden G
- Gu D
- Guan OP
- Guerrero R
- Guessous I
- Guimaraes AL
- Gutierrez L
- Hajifathalian K
- Hambleton IR
- Hardy R
- Hata J
- Heidemann C
- Herrala S
- Hihtaniemi IT
- Ho SC
- Ho SY
- Hofman A
- Hormiga CM
- Horta BL
- Houti L
- Howitt C
- Htay TT
- Htet AS
- Htike MMT
- Htike MMT
- Hu Y
- Hussieni AS
- Huybrechts I
- Hwalla N
- Iacoviello L
- Iannone AG
- Ibrahim MM
- Ikeda N
- Ikram MA
- Irazola VE
- Islam M
- Iwasaki M
- Jacobs JM
- Jafar T
- Jaime Miranda J
- Jamil KM
- Jasienska G
- Jiang CQ
- Jonas JB
- Joshi P
- Kadir KA
- Kafatos A
- Kalter-Leibovici O
- Kasaeian A
- Katz J
- Kaur P
- Kavousi M
- Kelishadi R
- Kengne AP
- Kersting M
- Khader YS
- Khalili D
- Khang Y-H
- Kiechl S
- Kim J
- Kolsteren P
- Kratzer W
- Kromhout D
- Kujala UM
- Kula K
- Kumar RH
- Kyobutungi C
- Laatikainen T
- Lachat C
- Laid Y
- Lam TH
- Landrove O
- Lanska V
- Lappas G
- Laxmaiah A
- Leclercq C
- Lee J
- Lee J
- Lehtimaki T
- Lekhraj R
- Leon-Munoz LM
- Li Y
- Lim W-Y
- Lin H-H
- Lin X
- Lissner L
- Lo W-C
- Lorbeer R
- Lu Y
- Luksiene D
- Lundqvist A
- Lytsy P
- Ma G
- Ma G
- Machado-Coelho GLL
- Machi S
- Maggi S
- Magliano DJ
- Makdisse M
- Manios Y
- Manzato E
- Margozzini P
- Maria Navarrete-Munoz E
- Marques-Vidal P
- Martorell R
- Masoodi SR
- Mathiesen EB
- Matsha TE
- Mbanya JCN
- Mbanya JCN
- McFarlane SR
- McGarvey ST
- McGarvey ST
- McLachlan S
- McNulty BA
- Mediene-Benchekor S
- Meirhaeghe A
- Menezes AMB
- Merat S
- Meshram II
- Mi J
- Miranda JJ
- Mohamed MK
- Mohammad K
- Mohammadifard N
- Mohamud WNW
- Mohan V
- Mohan V
- Moller NC
- Molnar D
- Mondo CK
- Morejon A
- Moreno LA
- Morgan K
- Moschonis G
- Mossakowska M
- Mostafa A
- Mota J
- Motta J
- Mu TT
- Mueller-Nurasyid M
- Muiesan ML
- Mursu J
- Nagel G
- Namesna J
- Nang EEK
- Nangia VB
- NCD Risk Factor Collaboration (NCD-RisC) .
- Ndiaye NC
- Nenko I
- Nervi F
- Nguyen ND
- Nguyen QN
- Nieto-Martinez RSE
- Ning G
- Ninomiya T
- Noale M
- Noto D
- Ochoa-Aviles AM
- Oh K
- Onat A
- Onat A
- Ordunez P
- Osmond C
- Otero JA
- Owusu-Dabo E
- Paciorek CJ
- Pahomova E
- Palmieri L
- Panda-Jonas S
- Panza F
- Parsaeian M
- Peixoto SV
- Peltonen M
- Peters A
- Peykari N
- Pham ST
- Pilav A
- Pitakaka F
- Piwonska A
- Piwonski J
- Plans-Rubio P
- Porta M
- Portegies MLP
- Poustchi H
- Pradeepa R
- Price JF
- Punab M
- Qasrawi RF
- Qorbani M
- Radisauskas R
- Rahman M
- Raitakari O
- Ramachandran A
- Ramachandran A
- Ramke J
- Ramos R
- Rampal S
- Rao KM
- Rao SR
- Rathmann W
- Redon J
- Reganit PFM
- Reis-Santos B
- Rigo F
- Riley LM
- Rivera JA
- Robinson SM
- Robitaille C
- Rodriguez-Artalejo F
- Rodriguez-Villamizar LA
- Rojas-Martinez R
- Ronkainen K
- Rosengren A
- Rubinstein A
- Rui O
- Russo Horimoto ARV
- Rutkowski M
- Sabanayagam C
- Sachdev HS
- Saidi O
- Sakarya S
- Salanave B
- Salonen JT
- Salvetti M
- Sanchez-Abanto J
- Sandra Ruiz-Betancourt B
- Santos D
- Santos R
- Saramies JL
- Sardinha LB
- Sarrafzadegan N
- Saum K-U
- Scazufca M
- Schargrodsky H
- Scheidt-Nave C
- Sein AA
- Sharma SK
- Shaw JE
- Shibuya K
- Shin Y
- Shiri R
- Siantar R
- Sibai AM
- Simon M
- Simons J
- Simons LA
- Sjostrom M
- Slowikowska-Hilczer J
- Slusarczyk P
- Smeeth L
- Snijder MB
- So H-K
- Sobngwi E
- Soderberg S
- Solfrizzi V
- Sonestedt E
- Soumare A
- Staessen JA
- Stathopoulou MG
- Steene-Johannessen J
- Stehle P
- Stein AD
- Stessman J
- Stevens GA
- Stoeckl D
- Stokwiszewski J
- Stronks K
- Strufaldi MW
- Sun C-A
- Sundstrom J
- Sung Y-T
- Suriyawongpaisal P
- Sy RG
- Taddei C
- Tai ES
- Tamosiunas A
- Tang L
- Tarawneh M
- Tarqui-Mamani CB
- Taylor A
- Theobald H
- Thijs L
- Thuesen BH
- Tolonen HK
- Tolstrup JS
- Topbas M
- Torrent M
- Traissac P
- Trinh OTH
- Tulloch-Reid MK
- Tuomainen T-P
- Turley ML
- Tzourio C
- Ueda P
- Ukoli FAM
- Ulmer H
- Uusitalo HMT
- Valdivia G
- Valvi D
- van Rossem L
- van Valkengoed IGM
- Vanderschueren D
- Vanuzzo D
- Vega T
- Velasquez-Melendez G
- Veronesi G
- Verschuren WMM
- Verstraeten R
- Viet L
- Vioque J
- Virtanen JK
- Viswanathan B
- Vollenweider P
- Voutilainen S
- Vrijheid M
- Wade AN
- Wagner A
- Walton J
- Wang F
- Wang M-D
- Wang Q
- Wang YX
- Wannamethee SG
- Weerasekera D
- Whincup PH
- Widhalm K
- Wijga AH
- Wilks RJ
- Willeit J
- Wilsgaard T
- Wojtyniak B
- Wong TY
- Woo J
- Woodward M
- Wu FC
- Wu SL
- Xu H
- Yan W
- Yang X
- Ye X
- Yoshihara A
- Younger-Coleman NO
- Yusoff MFM
- Zambon S
- Zargar AH
- Zdrojewski T
- Zhao W
- Zheng Y
- Zhou B
- Zuniga Cisneros J
- Publication venue
- Publication date
- 09/04/2016
- Field of study
BACKGROUND: One of the global targets for non-communicable diseases is to halt, by 2025, the rise in the age-standardised adult prevalence of diabetes at its 2010 levels. We aimed to estimate worldwide trends in diabetes, how likely it is for countries to achieve the global target, and how changes in prevalence, together with population growth and ageing, are affecting the number of adults with diabetes. METHODS: We pooled data from population-based studies that had collected data on diabetes through measurement of its biomarkers. We used a Bayesian hierarchical model to estimate trends in diabetes prevalence—defined as fasting plasma glucose of 7·0 mmol/L or higher, or history of diagnosis with diabetes, or use of insulin or oral hypoglycaemic drugs—in 200 countries and territories in 21 regions, by sex and from 1980 to 2014. We also calculated the posterior probability of meeting the global diabetes target if post-2000 trends continue. FINDINGS: We used data from 751 studies including 4 372 000 adults from 146 of the 200 countries we make estimates for. Global age-standardised diabetes prevalence increased from 4·3% (95% credible interval 2·4–7·0) in 1980 to 9·0% (7·2–11·1) in 2014 in men, and from 5·0% (2·9–7·9) to 7·9% (6·4–9·7) in women. The number of adults with diabetes in the world increased from 108 million in 1980 to 422 million in 2014 (28·5% due to the rise in prevalence, 39·7% due to population growth and ageing, and 31·8% due to interaction of these two factors). Age-standardised adult diabetes prevalence in 2014 was lowest in northwestern Europe, and highest in Polynesia and Micronesia, at nearly 25%, followed by Melanesia and the Middle East and north Africa. Between 1980 and 2014 there was little change in age-standardised diabetes prevalence in adult women in continental western Europe, although crude prevalence rose because of ageing of the population. By contrast, age-standardised adult prevalence rose by 15 percentage points in men and women in Polynesia and Micronesia. In 2014, American Samoa had the highest national prevalence of diabetes (>30% in both sexes), with age-standardised adult prevalence also higher than 25% in some other islands in Polynesia and Micronesia. If post-2000 trends continue, the probability of meeting the global target of halting the rise in the prevalence of diabetes by 2025 at the 2010 level worldwide is lower than 1% for men and is 1% for women. Only nine countries for men and 29 countries for women, mostly in western Europe, have a 50% or higher probability of meeting the global target. INTERPRETATION: Since 1980, age-standardised diabetes prevalence in adults has increased, or at best remained unchanged, in every country. Together with population growth and ageing, this rise has led to a near quadrupling of the number of adults with diabetes worldwide. The burden of diabetes, both in terms of prevalence and number of adults affected, has increased faster in low-income and middle-income countries than in high-income countries. FUNDING: Wellcome Trust
A922 Sequential measurement of 1 hour creatinine clearance (1-CRCL) in critically ill patients at risk of acute kidney injury (AKI)
- Author
- Abbott TE
- Abdallah RI
- Abdel-Aal IR
- Abdelmonem SA
- Abdo WF
- Abellan AN
- Abellán AN
- Abellán AN
- Abellán AN
- Abrams ST
- Ackerley C
- Acuña M
- Adda M
- Adhikari NK
- Adriano G
- Adrie C
- Affatato R
- Affatato R
- Afonso-Rivero D
- Agarossi A
- Agarwal LK
- Ageeva T
- Agrawal R
- Aguayo-DeHoyos E
- Aguilar AL
- Aguilar-Alonso E
- Aguilar-Alonso E
- Aguilar-Alonso E
- Aguirregabyria M
- Ahmadnia E
- Ahn C
- Ahn JY
- Ahn JY
- Ahn MY
- Ahn MY
- Aitken LM
- Akalaev R
- Akbarzadeh A
- Akcan-Arikan A
- Akhlagh SH
- Akhtar N
- AKI Research Group
- Akiduki N
- Akin S
- Akizuki N
- Akkoc T
- Ala-Kokko T
- Alanio A
- Albaiceta GM
- Albaladejo P
- Alberto F
- Albuisson E
- Alcala MA
- Alcázar L
- Aldabo-Pallas T
- Aldana NN
- Aldecoa C
- Alegría L
- Alejandro R
- Alejo GC
- Alejos RM
- Alexandrova E
- Algarte R
- Algarte R
- Algieri I
- Algora-Weber A
- Alhamdi Y
- Aliaga FA
- Aliberti S
- Alkan A
- Almudena PM
- Almudevar PM
- Almudevar PM
- Almudévar PM
- Altıntas ND
- Alvarez J
- Alves SC
- Alzola LM
- Amargianitakis V
- Amaro R
- Amato MB
- Ambler M
- Amerongen MP
- Amininejad T
- Amor LL
- Amorim V
- Anand RK
- Andersen LW
- Andersson S
- Andoh K
- Andrade AH
- Andrade AH
- Andrade AH
- Andrade G
- Andreis DT
- Andreis DT
- Annane D
- Antonelli M
- Antoniadou A
- Antonio C
- Antonucci E
- Antón DG
- Anzueto A
- Appiani F
- Aquevedo A
- Aragon C
- Araujo NJ
- Araujo VF
- ARIAM registry of adult cardiac surgery
- ARIAM-ANDALUCIA
- Arias CC
- Arias N
- Arias-Verdu MD
- Arias-Verdu MD
- Arias-Verdu MD
- Armaganidis A
- Arora MK
- Arora N
- Arrigo M
- Arslantas MK
- Artiguenave M
- Aslanian P
- Aspide R
- Asyralyyeva G
- Ataman S
- Ataman S
- Atchade E
- Atchade E
- Atchasiri S
- Athapattu P
- Atkins G
- Aublanc M
- Aublanc M
- Augustin P
- Augustin P
- Auquier P
- Avilés-Jurado FX
- Avramidis V
- Ayala LY
- Aydin M
- Azevedo JC
- Azevedo JR
- Aznar GP
- Babalis D
- Babini G
- Bachli EB
- Bader A
- Bader A
- Baena J
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- Chhor V
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- Curiel-Balsera E
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- Dalhuisen A
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- De Brito-Ashurst I
- De Cassai A
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- Engelberts D
- ENVIN-HELICS Study Group
- Erb J
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- Escalada SH
- Escoval A
- Escribá A
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- FINNRESUSCI Study Group
- Fischer G
- Fleischmann E
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- FROG ICU Investigators
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- GETGAG Working Group
- GETGAG/SEMICYUC
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- Hung WT
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- Ignacio ML
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- Imanaka H
- Inaloo S
- Ince C
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- IPREA Study Group
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- Japan Septic Disseminated Intravascular Coagulation (JSEPTIC DIC) study group
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- Jayasinghe S
- Jean-Baptiste S
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- Jensen JU
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- JSEPTIC (Japanese Society of Education for Physicians and Trainees in Intensive Care) Clinical Trial Group
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- Juanas CA
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- Juliarena A
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- Koco E
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- Kodate A
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- Kolnikova I
- Komuro T
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- Kosuk ME
- Kotsopoulos A
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- Krishna B
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- Kulkarni AP
- Kumari BK
- Kumbamu A
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- Kwon WY
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- Kyle UG
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- Limuti R
- Lin KC
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- Lin SC
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- Lischinsky A
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- Lobo-Civico A
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- Lopez-Gude MJ
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- Lortat-Jacob B
- Lortat-Jacob B
- Lotay R
- Loudet CI
- Louf-Durier A
- Louf-Durier A
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- Lozano JA
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- Lukaszewicz AC
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- MacKay A
- MacKay A
- Mackey G
- Mackey G
- MacPhee I
- Macrì M
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- Maekawa K
- Maekawa K
- Maekawa K
- Maekawa K
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- Maghsoudi B
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- Maistry N
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- Marcos-Zambrano LJ
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- Marin-Mateos H
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- Martín-Loeches I
- Martínez F
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- Martínez MV
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- Mejías CR
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- Melo N
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- Mercat A
- Mercat A
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- Merino-Vega D
- Messenger D
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- Midwinter MJ
- Mignot T
- Miguelena PR
- Mihara Y
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- Olaechea P
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- Radu V
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- Rodríguez FG
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- Tabei SH
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- Tapponnier R
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- Temprano-Gómez I
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- WIND study group
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- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
Meeting abstrac
A classification approach for genotyping viral sequences based on multidimensional scaling and linear discriminant analysis
- Author
- A Rambaut
- AJ Brown
- AK Schultz
- AK Schultz
- AM Land
- B Wang
- C Kuiken
- C Pace
- CE Monken
- DG Higgins
- DL Robertson
- FK Yoshimura
- GH Kijak
- J Timm
- JE Fitzgibbon
- JF Hair
- JH Simon
- Jiwoong Kim
- JP Vartanian
- JP Vartanian
- K Wilbe
- Kichan Lee
- M Goodenow
- M Janini
- M Rozanov
- M Wei
- M Zhang
- MO Salminen
- N Vidal
- P Hraber
- P Simmonds
- R Gifford
- RE Myers
- Sangsoo Kim
- SK Gandhi
- Sung Hee Park
- T de Oliveira
- T Leitner
- TF Cox
- VA Johnson
- WN Venables
- X Wu
- Yongju Ahn
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Measurement of the W±Z boson pair-production cross section in pp collisions at √s=13TeV with the ATLAS detector
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- Zivkovic L
- Zobernig G
- Zoccoli A
- Zu Theenhausen HM
- zur Nedden M
- Zwalinski L
- Publication venue
- Netherlands
- Publication date
- 01/01/2016
- Field of study
published_or_final_versio
Anatomy of the sign-problem in heavy-dense QCD
- Author
- Adam E Romero
- Aranda C Padilla
- Arjanovic MM
- Armadans R Caminal
- Astigarraga ME Pozo
- Barajas CA Chavez
- Bechtle P
- Beck HP
- Becker K
- Becker M
- Beckingham M
- Becot C
- Beddall A
- Beddall AJ
- Bednyakov VA
- Bedognetti M
- Bee CP
- Beemster LJ
- Beermann TA
- Begel M
- Behr JK
- Belanger-Champagne C
- Bell AS
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- Bellerive A
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- Belotskiy K
- Beltramello O
- Belyaev NL
- Benary O
- Benchekroun D
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- Bendtz K
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- Benhammou Y
- Benitez J
- Benjamin DP
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Berge D
- Berger N
- Berghaus F
- Beringer J
- Berlendis S
- Berlingen J Montejo
- Bernard NR
- Bernius C
- Bernlochner FU
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- Bertolucci F
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- Bertsche D
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- Bessner M
- Besson N
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- Bethke S
- Bevan AJ
- Bhimji W
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- Biesuz NV
- Biglietti M
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- Bindi M
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- Biondi S
- Bjergaard DM
- Black CW
- Black JE
- Black KM
- Blackburn D
- Blair RE
- Blanchard J-B
- Blanco JE
- Blazek T
- Bloch I
- Blocker C
- Blum W
- Blumenschein U
- Blunier S
- Bobbink GJ
- Bobrovnikov VS
- Bocchetta SS
- Bocci A
- Bock C
- Boehler M
- Boeriu OE Vickey
- Boerner D
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- Borisov A
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- Boscherini D
- Bosman M
- Bossio Sola JD
- Boudreau J
- Bouffard J
- Bouhova-Thacker EV
- Boumediene D
- Bourdarios C
- Bousson N
- Boutle SK
- Boveia A
- Boyd J
- Boyko IR
- Bracinik J
- Brandt A
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- Zimine NI
- Zimmermann C
- Zimmermann S
- Zinonos Z
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- Zobernig G
- Zoccoli A
- zur Nedden M
- Zurzolo G
- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
QCD at finite densities of heavy quarks is investigated
using the density-of-states method. The phase factor
expectation value of the quark determinant is calculated to
unprecedented precision as a function of the chemical potential.
Results are validated using those from a reweighting
approach where the latter can produce a significant signalto-noise
ratio. We confirm the particle–hole symmetry at low
temperatures, find a strong sign problem at intermediate values
of the chemical potential, and an inverse Silver Blaze
feature for chemical potentials close to the onset value: here,
the phase-quenched theory underestimates the density of the
full theory
Combined measurement of differential and total cross sections in the H → γγ and the H → ZZ* → 4ℓ decay channels at s=13 TeV with the ATLAS detector
- Author
- Aaboud M
- Aad G
- Abbott B
- Abdinov O
- Abeloos B
- Abhayasinghe DK
- Abidi SH
- Abouzeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Adachi S
- Adamczyk L
- Adelman J
- Adersberger M
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agheorghiesei C
- Aguilar-Saavedra JA
- Ahmadov F
- Aielli G
- Akatsuka S
- Akilli E
- Akimov AV
- Alberghi GL
- Albert J
- Albicocco P
- Alconada Verzini MJ
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alhroob M
- Ali B
- Alimonti G
- Alison J
- Alkire SP
- Allaire C
- Allbrooke BMM
- Allen BW
- Allport PP
- Aloisio A
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- Alshehri AA
- Alstaty MI
- Alvarez Gonzalez B
- Alviggi MG
- Amadio BT
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- Argyropoulos S
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- Ashkenazi A
- Asimakopoulou EM
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- Atlas Collaboration
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- White R
- Whiteson D
- Whitmore BW
- Wickens FJ
- Wiedenmann W
- Wielers M
- Wiglesworth C
- Wiik-Fuchs LAM
- Wildauer A
- Wilk F
- Wilkens HG
- Wilkins LJ
- Williams HH
- Williams S
- Willis C
- Willocq S
- Wilson JA
- Wingerter-Seez I
- Winkels E
- Winklmeier F
- Winston OJ
- Winter BT
- Wittgen M
- Wobisch M
- Wolf A
- Wolf TMH
- Wolff R
- Wolter MW
- Wolters H
- Wong VWS
- Woods NL
- Worm SD
- Wosiek BK
- Woźniak KW
- Wraight K
- Wu M
- Wu SL
- Wu X
- Wu Y
- Wyatt TR
- Wynne BM
- Xella S
- Xi Z
- Xia L
- Xu D
- Xu H
- Xu L
- Xu T
- Xu W
- Yabsley B
- Yacoob S
- Yajima K
- Yallup DP
- Yamaguchi D
- Yamaguchi Y
- Yamamoto A
- Yamanaka T
- Yamane F
- Yamatani M
- Yamazaki T
- Yamazaki Y
- Yan Z
- Yang HJ
- Yang HT
- Yang S
- Yang Y
- Yang Z
- Yao W-M
- Yap YC
- Yasu Y
- Yatsenko E
- Ye J
- Ye S
- Yeletskikh I
- Yigitbasi E
- Yildirim E
- Yorita K
- Yoshihara K
- Young C
- Young CJS
- Yu J
- Yu J
- Yue X
- Yuen SPY
- Yusuff I
- Zabinski B
- Zacharis G
- Zaffaroni E
- Zaidan R
- Zaitsev AM
- Zakharchuk N
- Zalieckas J
- Zambito S
- Zanzi D
- Zaripovas DR
- Zeitnitz C
- Zeißner SV
- Zemaityte G
- Zeng JC
- Zeng Q
- Zenin O
- Zerwas D
- Zgubič M
- Zhang D
- Zhang DF
- Zhang F
- Zhang G
- Zhang H
- Zhang J
- Zhang L
- Zhang L
- Zhang M
- Zhang P
- Zhang R
- Zhang R
- Zhang X
- Zhang Y
- Zhang Z
- Zhao P
- Zhao X
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zhou B
- Zhou C
- Zhou L
- Zhou M
- Zhou MS
- Zhou N
- Zhou Y
- Zhu CG
- Zhu H
- Zhu HL
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann S
- Zinonos Z
- Zinser M
- Ziolkowski M
- Zobernig G
- Zoccoli A
- Zoch K
- Zorbas TG
- Zou R
- Zur Nedden M
- Zwalinski L
- Álvarez Piqueras D
- Åkesson TPA
- Šfiligoj T
- Ženiš T
- Živković L
- Publication venue
- Elsevier
- Publication date
- 01/01/2018
- Field of study
A combined measurement of differential and inclusive total cross sections of Higgs boson production is performed using 36.1 fb−1 of 13 TeV proton–proton collision data produced by the LHC and recorded by the ATLAS detector in 2015 and 2016. Cross sections are obtained from measured H→γγ and H→ZZ*(→4ℓ event yields, which are combined taking into account detector efficiencies, resolution, acceptances and branching fractions. The total Higgs boson production cross section is measured to be 57.0−5.9 +6.0 (stat.) −3.3 +4.0 (syst.) pb, in agreement with the Standard Model prediction. Differential cross-section measurements are presented for the Higgs boson transverse momentum distribution, Higgs boson rapidity, number of jets produced together with the Higgs boson, and the transverse momentum of the leading jet. The results from the two decay channels are found to be compatible, and their combination agrees with the Standard Model predictions
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