247 research outputs found
The role of clathrin in post-golgi trafficking in toxoplasma gondii
- Author
- A Achbarou
- A Herm-Götz
- A Sadak
- AE Radulescu
- AL Schwartz
- B Lige
- B Striepen
- BF Kafsack
- BJ Nichols
- C Agop-Nersesian
- C Agop-Nersesian
- C Wang
- D Van Damme
- David J. P. Ferguson
- DJ Ferguson
- DJ Ferguson
- DJ Owen
- DS Roos
- E Gaskins
- EM Bennett
- GG van Dooren
- GL Starnes
- H El Hajj
- HT McMahon
- I Coppens
- I Coppens
- J Burgess
- J Hirst
- JB Dacks
- JB Doyon
- JC Schwab
- JJ Hölzenspies
- JM Harper
- K Kremer
- K Lingelbach
- KF Leung
- KL Wan
- L Pelletier
- L Tawk
- LH Bannister
- M Blume
- M Kaksonen
- M Nishi
- MA McNiven
- Manuela S. Pieperhoff
- Markus Meissner
- MH Huynh
- Miriam Schmitt
- ML Allen
- ML Niswonger
- Mohamed Ali Hakimi
- MS Breinich
- N Andenmatten
- NC Elde
- NJ Gerald
- PJ Sloves
- RG Donald
- RG Donald
- S Botero-Kleiven
- S Kitakura
- SH Liu
- SH Liu
- SL Acton
- SL Pfluger
- SM Stagg
- T Mann
- TF Roth
- U Gross
- V Karsten
- VB Carruthers
- WD Nevin
- Z Liu
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2013
- Field of study
Apicomplexan parasites are single eukaryotic cells with a highly polarised secretory system that contains unique secretory organelles (micronemes and rhoptries) that are required for host cell invasion. In contrast, the role of the endosomal system is poorly understood in these parasites. With many typical endocytic factors missing, we speculated that endocytosis depends exclusively on a clathrin-mediated mechanism. Intriguingly, in Toxoplasma gondii we were only able to observe the endogenous clathrin heavy chain 1 (CHC1) at the Golgi, but not at the parasite surface. For the functional characterisation of Toxoplasma gondii CHC1 we generated parasite mutants conditionally expressing the dominant negative clathrin Hub fragment and demonstrate that CHC1 is essential for vesicle formation at the trans-Golgi network. Consequently, the functional ablation of CHC1 results in Golgi aberrations, a block in the biogenesis of the unique secretory microneme and rhoptry organelles, and of the pellicle. However, we found no morphological evidence for clathrin mediating endocytosis in these parasites and speculate that they remodelled their vesicular trafficking system to adapt to an intracellular lifestyle
The chromatin remodelling enzymes SNF2H and SNF2L position nucleosomes adjacent to CTCF and other transcription
- Author
- A Valouev
- A Weiner
- AC Bell
- AN Yadon
- BJ Raney
- BK Lee
- C Trapnell
- D Bohla
- D Ganguli
- D Karolchik
- DE Schones
- DJ Gaffney
- ED Rubio
- F Erdel
- F Pessina
- GC Yuan
- GJ Narlikar
- GS Banting
- H Chen
- H Xiao
- J CÎté
- J Wang
- J Widom
- J Wysocka
- JA Wallace
- JG Kang
- K Ishihara
- K Luger
- K Nasmyth
- K Yen
- KS Wendt
- L Bozhenok
- L Handoko
- M Dluhosova
- M Eckey
- M Li
- M Merkenschlager
- M Renda
- M Smolle
- MA Hakimi
- MH Kagey
- MJ Pazin
- ML Eaton
- MTY Lam
- Nicola Wiechens
- NM Berbenetz
- O Barak
- O Barak
- Patrick Varga-Weisz
- Pieta Schofield
- RD Kornberg
- RM Myers
- S Sofueva
- SA Morris
- Sonia Rocha
- T Ito
- T Tsukiyama
- TH Kim
- TK Quan
- TN Mavrich
- Tom Owen-Hughes
- Triantaffyllos Gkikopoulos
- V Cheung
- V Parelho
- Vijender Singh
- W Lee
- W Stedman
- Y Fu
- YJ Kim
- Z Liu
- Z Qiu
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/03/2016
- Field of study
Within the genomes of metazoans, nucleosomes are highly organised adjacent to the binding sites for a subset of transcription factors. Here we have sought to investigate which chromatin remodelling enzymes are responsible for this. We find that the ATP-dependent chromatin remodelling enzyme SNF2H plays a major role organising arrays of nucleosomes adjacent to the binding sites for the architectural transcription factor CTCF sites and acts to promote CTCF binding. At many other factor binding sites SNF2H and the related enzyme SNF2L contribute to nucleosome organisation. The action of SNF2H at CTCF sites is functionally important as depletion of CTCF or SNF2H affects transcription of a common group of genes. This suggests that chromatin remodelling ATPase's most closely related to the Drosophila ISWI protein contribute to the function of many human gene regulatory elements
Involvement of global genome repair, transcription coupled repair, and chromatin remodeling in UV DNA damage response changes during development
- Author
- A Gartner
- A Klochendler-Yeivin
- AJ van Gool
- Björn Schumacher
- C Dinant
- C Masutani
- C Masutani
- C Troelstra
- E Citterio
- EC Andersen
- EJ Hubbard
- F Gong
- F Gong
- G Conforti
- G van Haaften
- Gert Jansen
- GG Wang
- H de Waard
- H Sawa
- Hannes Lans
- HI Stewart
- HK Park
- HK Park
- I Clejan
- J Cadet
- J de Boer
- J Pothof
- Jan H. J. Hoeijmakers
- JE Sulston
- JH Hoeijmakers
- JL Youds
- JM Ng
- JN Meyer
- JP Mueller
- JQ Svejstrup
- JT Reardon
- Jurgen A. Marteijn
- JW Astin
- K Sugasawa
- K Sugasawa
- K Ura
- KA Henning
- L Stergiou
- M Araki
- M Cui
- M Fousteri
- M Ljungman
- M Matsumoto
- M Volker
- M Wakasugi
- MA Hakimi
- MH Lee
- MS Luijsterburg
- Nancy Maizels
- NP Dantuma
- PS Hartman
- PS Hartman
- PS Hartman
- PT Yazdi
- R Hara
- R Nishi
- R Verhage
- RC Chan
- S Brenner
- SL Wade
- SN Guzder
- ST Kim
- T Nouspikel
- T Nouspikel
- TL Gumienny
- Wim Vermeulen
- Y Shi
- Publication venue
- 'Public Library of Science (PLoS)'
- Publication date
- 01/01/2010
- Field of study
Nucleotide Excision Repair (NER), which removes a variety of helix-distorting lesions from DNA, is initiated by two distinct DNA damage-sensing mechanisms. Transcription Coupled Repair (TCR) removes damage from the active strand of transcribed genes and depends on the SWI/SNF family protein CSB. Global Genome Repair (GGR) removes damage present elsewhere in the genome and depends on damage recognition by the XPC/RAD23/Centrin2 complex. Currently, it is not well understood to what extent both pathways contribute to genome maintenance and cell survival in a developing organism exposed to UV light. Here, we show that eukaryotic NER, initiated by two distinct subpathways, is well conserved in the nematode Caenorhabditis elegans. In C. elegans, involvement of TCR and GGR in the UV-induced DNA damage response changes during development. In germ cells and early embryos, we find that GGR is the major pathway contributing to normal development and survival after UV irradiation, whereas in later developmental stages TCR is predominantly engaged. Furthermore, we identify four ISWI/Cohesin and four SWI/SNF family chromatin remodeling factors that are implicated in the UV damage response in a developmental stage dependent manner. These in vivo studies strongly suggest that involvement of different repair pathways and chromatin remodeling proteins in UV-induced DNA repair depends on developmental stage of cells
Targeting HER2/neu with a fully human IgE to harness the allergic reaction against cancer cells
- Author
- A Berchuck
- A Garoufali
- A Jemal
- A Rodriguez
- AE Hanbidge
- B Bettler
- Birgit C. Schultes
- BM Weichman
- CA Janeway
- Christopher F. Nicodemus
- CW Adams
- D Dombrowicz
- D Dombrowicz
- D Maurer
- D Zhu
- DH Conrad
- DJ Slamon
- DJ Slamon
- DM King
- E Jensen-Jarolim
- E Nagy
- EA Nigro
- EM Yoo
- FL Heijden van der
- G Helguera
- GD Lewis Phillips
- Gustavo Helguera
- H Meden
- H Meden
- H Satoh
- HJ Gould
- HJ Gould
- J Berek
- J Gong
- J Hakimi
- JB Lyczak
- JM Lafky
- JosĂ© A. RodrĂguez
- JP Kinet
- JR Junutula
- JS Dela Cruz
- K Rafiq
- KM Ferguson
- LM Coussens
- M Hoopmann
- MA Bookman
- Manuel L. Penichet
- MH Kershaw
- ML Disis
- ML Penichet
- N Watanabe
- NE Hynes
- Otoniel MartĂnez-Maza
- P Braly
- P Karagiannis
- PJ Cooper
- R Bheekha Escura
- R Nahta
- R Schier
- R Schier
- RA Manz
- Rafaela Quintero
- Richard K. Leuchter
- S Lennon
- SN Karagiannis
- SN Karagiannis
- SN Karagiannis
- T Yoshimoto
- TH Cheung
- TH Huang
- Tracy R. Daniels
- TW Wiegand
- VC Vries de
- W Tai
- WP Fung-Leung
- WZ Wei
- Y Fernandez
- Y Tang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/07/2012
- Field of study
Breast and ovarian cancer are two of the leading causes of cancer deaths among women in the United States. Overexpression of the HER2/neu oncoprotein has been reported in patients affected with breast and ovarian cancers, and is associated with poor prognosis. To develop a novel targeted therapy for HER2/neu expressing tumors, we have constructed a fully human IgE with the variable regions of the scFv C6MH3-B1 specific for HER2/neu. This antibody was expressed in murine myeloma cells and was properly assembled and secreted. The Fc region of this antibody triggers in vitro degranulation of rat basophilic cells expressing human FcΔRI (RBL SX-38) in the presence of murine mammary carcinoma cells that express human HER2/neu (D2F2/E2), but not the shed (soluble) antigen (ECDHER2) alone. This IgE is also capable of inducing passive cutaneous anaphylaxis in a human FcΔRIα transgenic mouse model, in the presence of a cross-linking antibody, but not in the presence of soluble ECDHER2. Additionally, IgE enhances antigen presentation in human dendritic cells and facilitates cross-priming, suggesting that the antibody is able to stimulate a secondary T-cell anti-tumor response. Furthermore, we show that this IgE significantly prolongs survival of human FcΔRIα transgenic mice bearing D2F2/E2 tumors. We also report that the anti-HER2/neu IgE is well tolerated in a preliminary study conducted in Macaca fascicularis (cynomolgus) monkeys. In summary, our results suggest that this IgE should be further explored as a potential therapeutic against HER2/neu overexpressing tumors, such as breast and ovarian cancers.Fil: Daniels, Tracy R.. University of California at Los Angeles; Estados UnidosFil: Leuchter, Richard K.. University of California at Los Angeles; Estados UnidosFil: Quintero, Rafaela. University of California; Estados UnidosFil: Helguera, Gustavo Fernando. University of California at Los Angeles; Estados Unidos. Universidad de Buenos Aires. Facultad de Farmacia y BioquĂmica; Argentina. Consejo Nacional de Investigaciones CientĂficas y TĂ©cnicas; ArgentinaFil: RodrĂguez, JosĂ© A.. University of California at Los Angeles; Estados UnidosFil: MartĂnez Maza, Otoniel. University of California at Los Angeles; Estados UnidosFil: Schultes, Birgit C.. Advanced Immune Therapeutics, Inc.; Estados Unidos. Momenta Pharmaceuticals, Inc.; Estados UnidosFil: Nicodemus, Christopher F.. Advanced Immune Therapeutics, Inc.; Estados UnidosFil: Penichet, Manuel L.. University of California at Los Angeles; Estados Unido
A novel IgE antibody targeting the prostate-specific antigen as a potential prostate cancer therapy
- Author
- A Katzenwadel
- A Rodriguez
- AA Sinha
- AM Miller
- B Bettler
- Birgit C Schultes
- BO Villoutreix
- C Massard
- C Xuan
- CF Nicodemus
- Christopher F Nicodemus
- CK Gjertson
- CS Hsieh
- D Dombrowicz
- D Dombrowicz
- D Maurer
- D Maurer
- D Maurer
- DH Conrad
- E Jensen-Jarolim
- E Nagy
- EA Nigro
- Elizabeth Ortiz-SĂĄnchez
- EP Diamandis
- FJ Meyers
- FL Van der Heijden
- G Helguera
- G Sonpavde
- Gustavo Helguera
- H Lilja
- HJ Gould
- HJ Gould
- J Hakimi
- J Hoff
- J Singh
- JB Lyczak
- JM Corman
- JosĂ© A RodrĂguez
- JP Kinet
- K Pettersson
- K Rafiq
- KA Berlyn
- L Sottrup-Jensen
- LM Weiner
- M Bilusic
- M Bishr
- MA Cheever
- Maggie Kozman
- Manuel L Penichet
- MH Kershaw
- ML Penichet
- NG Chakraborty
- Otoniel MartĂnez-Maza
- P Karagiannis
- R Bheekha Escura
- R Siegel
- RA Willis
- Rafaela Quintero
- Richard K Leuchter
- RS Chu
- S Gupta
- SN Karagiannis
- SN Karagiannis
- SN Karagiannis
- T Bieber
- TL Stevens
- TR Daniels
- TR Daniels
- TR Daniels
- Tracy R Daniels-Wells
- TW Wiegand
- V Kumar
- V Kumar
- WM Zhang
- WM Zhang
- WP Fung-Leung
- Z Chen
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/04/2013
- Field of study
Prostate cancer (PCa) is the second leading cause of cancer deaths in men in the United States. The
prostate-specific antigen (PSA), often found at high levels in the serum of PCa patients, has been used as a marker for PCa detection and as a target of immunotherapy. The murine IgG1 monoclonal antibody AR47.47, specific for human PSA, has been shown to enhance antigen presentation by human dendritic cells and induce both CD4 andCD8 T-cell activation when complexed with PSA. In this study, we explored the properties of a novel mouse/human chimeric anti-PSA IgE containing the variable regions of AR47.47 as a potential therapy for PCa. Our goal was to take advantage of the unique properties of IgE in order to trigger immune activation against PCa.Fil: Daniels-Wells, Tracy R. University of California. David Geffen School of Medicine. Department of Surgery. Division of Surgical Oncology; Estados Unidos de AmĂ©rica;Fil: Helguera, Gustavo Fernando. Universidad de Buenos Aires. Facultad de Farmacia y Bioquimica. Departamento de Tecnologia Farmaceutica; Argentina; University of California. David Geffen School of Medicine. Department of Surgery. Division of Surgical Oncology; Estados Unidos de AmĂ©rica;Fil: Leuchter, Richard K. University of California. David Geffen School of Medicine. Department of Surgery. Division of Surgical Oncology; Estados Unidos de AmĂ©rica;Fil: Quintero, Rafael. University of California. David Geffen School of Medicine. Department of Surgery. Division of Surgical Oncology; Estados Unidos de AmĂ©rica;Fil: Kozman, Maggie. University of California. David Geffen School of Medicine. Department of Surgery. Division of Surgical Oncology; Estados Unidos de AmĂ©rica;Fil: RodrĂguez, JosĂ© A.. University of California. David Geffen School of Medicine. Department of Surgery. Division of Surgical Oncology; Estados Unidos de AmĂ©rica; University of California. The Molecular Biology Institute; Estados Unidos de AmĂ©rica;Fil: Ortiz-SĂĄnchez, E. University of California. David Geffen School of Medicine. Department of Surgery. Division of Surgical Oncology; Estados Unidos de AmĂ©rica; Biomedical Research in Cancer. Basic Research Division. National Institute of Cancerology; Mexico.;Fil: MartĂnez-Maza, Otonel. University of California. David Geffen School of Medicine. Department of Surgery. Division of Surgical Oncology; Estados Unidos de AmĂ©rica;Fil: Schultes, Brigit C.. Advanced Immune Therapeutics; Estados Unidos de AmĂ©rica;Fil: Nicodemus Christopher. Advanced Immune Therapeutics; Estados Unidos de AmĂ©rica;Fil: Penichet, Manuel. University of California. David Geffen School of Medicine. Department of Surgery. Division of Surgical Oncology; Estados Unidos de AmĂ©rica; University of California. The Molecular Biology Institute; Estados Unidos de AmĂ©rica
Search for strongly interacting massive particles generating trackless jets in proton-proton collisions at s = 13 TeV
- Author
- Aarrestad TK
- Aarup Petersen H
- Abbaneo D
- Abbiendi G
- Abbrescia M
- Abdullin S
- Abercrombie D
- Acharya H
- Acosta D
- Acosta JG
- Adam W
- Adams E
- Adams MR
- Adams T
- Adloff C
- Adzic P
- Agapitos A
- Agarwal G
- Aggleton R
- Agram J-L
- Aguilar-Benitez M
- Ahmad A
- Ahmad M
- Ahmed A
- Ahuja S
- Aime C
- Akchurin N
- Akgun B
- Akpinar A
- Albajar C
- Albergo S
- Albert A
- Albrow M
- Alcaraz Maestre J
- Aldaya Martin M
- AldĂĄ JĂșnior WL
- Aleksandrov A
- Alexander J
- Alhusseini M
- Alimena J
- Alison J
- Allen B
- Almond J
- Alvarez Gonzalez B
- Alverson G
- Alves GA
- Aly R
- Alyari M
- Amapane N
- Amendola C
- Amin N
- Amram O
- Amsler C
- Anagnostou G
- Andrea J
- Andreev V
- Andreev Y
- Andrews MB
- Androsov K
- Antchev G
- Antunovic Z
- Apanasevich L
- Apollinari G
- Appelt E
- Apresyan A
- Apyan A
- Araujo M
- Arcaro D
- Arcidiacono R
- Arenton MW
- Argiro S
- Arneodo M
- Aruta C
- Asavapibhop B
- Asawatangtrakuldee C
- Asenov P
- Asghar MI
- Asilar E
- Askew A
- Asmuss P
- Aspell P
- Atakisi IO
- Atanassov I
- Ather MW
- Attikis A
- Auffray E
- Aushev T
- Auzinger G
- Avati V
- Avery P
- Avila C
- Awais A
- Awan MIM
- Ayala E
- Ayala G
- Aydogmus Sen F
- Azarkin M
- Azhgirey I
- Aziz T
- Azzi P
- Azzurri P
- Baarmand MM
- Babaev A
- Babounikau I
- Bacchetta N
- Bachiller I
- Bachtis M
- Backhaus M
- Baden A
- Baechler J
- Baginyan A
- Bagliesi G
- Bahinipati S
- Baillon P
- Bainbridge R
- Bakas G
- Bakhshiansohi H
- Bakirci MN
- Baldenegro Barrera C
- Ball AH
- Ban Y
- Band R
- Bandyopadhyay H
- Banerjee S
- Banerjee S
- Bansal S
- Barbagli G
- Barberis E
- Bargassa P
- Baringer P
- Barnes VE
- Barney D
- Baron O
- Barrio Luna M
- Bartek R
- Bartosik N
- BartĂłk M
- Bastos D
- Battilana C
- Baty A
- Bauer G
- Bauerdick LAT
- Baxter S
- Bayshev I
- Bean A
- Beauceron S
- Beaudette F
- Becerril Gonzalez H
- Bechtel J
- Bedoya CF
- Beghin D
- Behera PK
- Behera SC
- Behnke O
- Bein S
- Belchior Batista Das Chagas E
- Belforte S
- Bell KW
- Bellan R
- Belloni A
- Bellora A
- Belyaev A
- Belyaev A
- Benaglia A
- Benato L
- Bencze G
- Bendavid J
- Benecke A
- Benelli G
- Beni N
- Benitez JF
- Benussi L
- Berardi V
- Berenguer Antequera J
- Beretvas A
- Bergauer T
- Berger P
- Berger T
- Beri SB
- BermĂșdez MartĂnez A
- Bernardes CA
- Bernet C
- Berretti M
- Berry D
- Berryhill J
- Bertacchi V
- Besancon M
- Beschi A
- Bhal E
- Bhardwaj A
- Bharti M
- Bhat MA
- Bhat PC
- Bhatnagar V
- Bhattacharya R
- Bhattacharya S
- Bhattacharya S
- Bhattacharya S
- Bheesette S
- Bhowmik D
- Bhowmik S
- Bhyun JH
- Bi R
- Bialkowska H
- Bianchini L
- Bianco M
- Bianco S
- Biasini M
- Biino C
- Bilei GM
- Bilin B
- Bin Anuar AA
- Bisello D
- Black K
- Blekman F
- Blinov V
- Bloch D
- Bloch P
- Bloom K
- Bluj M
- Blumenfeld B
- Boccali T
- Bocci A
- Bodek A
- Boimska B
- Boletti A
- Bologna S
- Bols ES
- Bonacorsi D
- Bonanomi M
- Bonham B
- Bonomally S
- Boos E
- Boran F
- Borchsh V
- Borg J
- Borgonovi L
- Bornheim A
- Borras K
- Bortignon P
- Bose T
- Bossini E
- Botta C
- Botta V
- Bottigli U
- Boudoul G
- Bouhali O
- Bourgatte G
- Bourilkov D
- Bozzo M
- Bragagnolo A
- Braghieri A
- Braibant-Giacomelli S
- Brainerd C
- Brandao Malbouisson H
- Brandt S
- Branson JG
- Breedon R
- Breeze S
- Brew C
- Brigljevic V
- Brinkerhoff A
- Brivio F
- Brochero Cifuentes JA
- Brom J-M
- Brondolin E
- Brooke JJ
- Brown RM
- Brunner D
- Bruno G
- Brzhechko D
- BrĂŒcken E
- Bucci R
- Buccilli A
- Buchanan J
- Buchmuller O
- Buchot Perraguin A
- Bueghly J
- Bundock A
- Bunkowski K
- Buontempo S
- Burkett K
- Burkhardt H
- Burkle B
- Burt K
- Bury F
- Busson P
- Busza W
- Butalla S
- Butler JN
- Butler PH
- Butz E
- Bylinkin A
- Bylsma B
- Cabrera A
- Cabrillo IJ
- Cadamuro L
- Cafagna FS
- Caillol C
- Cakir A
- Calandri A
- Calderon De La Barca Sanchez M
- Calderon A
- Cali IA
- Call K
- Calligaris L
- Calzaferri S
- Camen C
- Caminada L
- Campagnari C
- Campanini R
- Campbell A
- Camporesi T
- Candelise V
- Canelli MF
- Canepa A
- Cankocak K
- Capeans Garrido M
- Capiluppi P
- Cappati A
- Caputo C
- Caraway B
- Cardini A
- Cardwell B
- Carle A
- Carlin R
- Carrera Jarrin E
- Carrillo Montoya CA
- Carrillo Moreno S
- Cartiglia N
- Carvalho Antunes De Oliveira A
- Carvalho W
- Casarsa M
- Caspart R
- Cassese A
- Castaldi R
- Castaneda Hernandez A
- Castilla-Valdez H
- Castro A
- Catanesi MG
- Cavallari F
- Cavallo FR
- Cavallo N
- Cavanaugh R
- Ceard L
- Ceccarelli R
- Cepaitis V
- Cepeda M
- Cerati GB
- Cerci S
- Cerminara G
- Cerrada M
- Cerri O
- Cetorelli F
- Chabert EC
- Chahal GS
- Chang P
- Chang P
- Chanon N
- Chao Y
- Chapon E
- Charaf O
- Charlot C
- Chatterjee RM
- Chatterjee S
- Chaudhary G
- Chauhan S
- Chauhan S
- Chawla R
- Chazin Quero B
- Checchia P
- Chekhovsky V
- Chen C
- Chen GM
- Chen HS
- Chen KF
- Chen M
- Chen PH
- Chen X
- Chen Y
- Chen Y
- Chen Z
- Chenarani S
- Cheng T
- Cheng Y
- Cherepanov V
- Chernyavskaya N
- Chertok M
- Cheung HWK
- Chhibra SS
- Chiarito B
- Chinellato J
- Chistov R
- Chlebana F
- Cho S
- Choi J
- Choi S
- Choi Y
- Chou JP
- Choudhary BC
- Choudhury S
- Chu J
- Chudasama R
- Chwalek T
- Ciangottini D
- Ciocca C
- Ciocci MA
- Cipriani M
- Ciriolo V
- Cirkovic P
- Citron M
- Cittolin S
- Ciulli V
- Civinini C
- Claes DR
- Clare R
- Clement E
- Clerbaux B
- CMS Collaboration<sup>â </sup>
- Cockerill DJA
- Coelho E
- Colaleo A
- Cole JE
- Colino N
- Collard C
- Colling D
- Cometti S
- Connor P
- Consuegra RodrĂguez S
- Contardo D
- Conway J
- Conway R
- Cooper SI
- Cooperstein S
- Corcodilos L
- Cornelis T
- Cosby C
- Cossutti F
- Costa M
- Costa S
- Coubez X
- Couderc F
- Cousins R
- Covarelli R
- Cox B
- Cox PT
- Cranshaw DJ
- Creanza D
- Cremonesi M
- Cristella L
- CsanĂĄd M
- CsörgĆ T
- Cuevas J
- Cuffiani M
- Cumalat JP
- Cummings G
- Cussans D
- Cutts D
- Czellar S
- D'Alessandro R
- D'Alfonso M
- D'Enterria D
- D'Hondt J
- Da Costa EM
- Da Rold A
- Da Silveira GG
- Dabrowski A
- Daci N
- Dalchenko M
- Dallavalle GM
- Damarseckin S
- Damgov J
- Danilov M
- Danilov V
- Daponte V
- Darwish MR
- Das P
- Das S
- Dasgupta A
- Dash D
- Daskalakis G
- Dasu S
- Datta A
- Dauncey P
- David A
- David P
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- Ăzçelik Ă
- Ć irokĂœ J
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2022
- Field of study
A search for dark matter in the form of strongly interacting massive particles (SIMPs) using the CMS detector at the LHC is presented. The SIMPs would be produced in pairs that manifest themselves as pairs of jets without tracks. The energy fraction of jets carried by charged particles is used as a key discriminator to suppress efficiently the large multijet background, and the remaining background is estimated directly from data. The search is performed using proton-proton collision data corresponding to an integrated luminosity of 16.1 fb - 1 , collected with the CMS detector in 2016. No significant excess of events is observed above the expected background. For the simplified dark matter model under consideration, SIMPs with masses up to 100 GeV are excluded and further sensitivity is explored towards higher masses
Observation of a New Excited Beauty Strange Baryon Decaying to Îb- Ï+Ï-
- Author
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- Zahid S
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- Zorbilmez C
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- Ăzçelik Ă
- Publication venue
- American Physical Society
- Publication date
- 01/01/2021
- Field of study
The Îb-Ï+Ï- invariant mass spectrum is investigated with an event sample of proton-proton collisions at s=13 TeV, collected by the CMS experiment at the LHC in 2016-2018 and corresponding to an integrated luminosity of 140 fb-1. The ground state Îb- is reconstructed via its decays to J/ÏÎ- and J/ÏÎK-. A narrow resonance, labeled Îb(6100)-, is observed at a Îb-Ï+Ï- invariant mass of 6100.3±0.2(stat)±0.1(syst)±0.6(Îb-) MeV, where the last uncertainty reflects the precision of the Îb- baryon mass. The upper limit on the Îb(6100)- natural width is determined to be 1.9 MeV at 95% confidence level. The low Îb(6100)- signal yield observed in data does not allow a measurement of the quantum numbers of the new state. However, following analogies with the established excited Îc baryon states, the new Îb(6100)- resonance and its decay sequence are consistent with the orbitally excited Îb- baryon, with spin and parity quantum numbers JP=3/2-
Measurement of the inclusive and differential Higgs boson production cross sections in the decay mode to a pair of Ï Leptons in pp collisions at sqrt[s]=13ââTeV
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- Aarup Petersen H
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- Yumiceva F
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- Zacharopoulou A
- Zahid S
- Zaleski S
- Zalewski P
- Zanetti M
- Zarubin A
- Zarucki M
- Zecchinelli AG
- Zeinali M
- Zeuner WD
- Zghiche A
- Zhang F
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- Zhizhin I
- Zhokin A
- Zhu DH
- Zhu RY
- Ziemons T
- Zoi I
- Zolkapli Z
- Zorbakir IS
- Zorbilmez C
- Zotto P
- Zotz A
- Zou D
- Zou R
- Zucchetta A
- Zumerle G
- Zuo X
- Zuolo D
- Zygala L
- Ălvarez FernĂĄndez A
- Ăzçelik Ă
- Publication venue
- 'American Physical Society (APS)'
- Publication date
- 01/01/2022
- Field of study
Measurements of the inclusive and differential fiducial cross sections of the Higgs boson are presented, using the Ï lepton decay channel. The differential cross sections are measured as functions of the Higgs boson transverse momentum, jet multiplicity, and transverse momentum of the leading jet in the event, if any. The analysis is performed using proton-proton collision data collected with the CMS detector at the LHC at a center-of-mass energy of 13ââTeV and corresponding to an integrated luminosity of 138ââfb^{-1}. These are the first differential measurements of the Higgs boson cross section in the final state of two Ï leptons. In final states with a large jet multiplicity or with a Lorentz-boosted Higgs boson, these measurements constitute a significant improvement over measurements performed in other final states
Search for dark matter produced in association with a Higgs boson decaying to a pair of bottom quarks in proton-proton collisions at root s=13TeV
- Author
- Aarrestad TK
- Abbaneo D
- Abbiendi G
- Abbrescia M
- Abdullah WATW
- Abdullin S
- Abercrombie D
- Acharya H
- Acosta D
- Acosta JG
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- Adloff C
- Adzic P
- AdĂĄn DP
- Afanasiev S
- Agapitos A
- Agarwal G
- Aggleton R
- Agram J-L
- Aguilar-Benitez M
- Ahmad A
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- Ahmad WH
- Ahmed A
- Ahuja S
- Aime' C
- Akchurin N
- Akgun B
- Akpinar A
- Albajar C
- Albergo S
- Albert A
- Albrow M
- Aleksandrov A
- Alexander J
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- Alimena J
- Alison J
- Allen B
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- Alonso AG
- Alvarez JDR
- Alverson G
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- Aly R
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- Amaral SMSD
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- Amram O
- Amsler C
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- Angioni GLP
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- Anuar AAB
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- Apollinari G
- Appelt E
- Apresyan A
- Apyan A
- Araujo M
- Arbelaez NV
- Arbol PMRD
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- Arenton MW
- Argiro S
- Arneodo M
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- Asawatangtrakuldee C
- Asenov P
- Asghar MI
- Asilar E
- Askew A
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- Atakisi IO
- Atanasov I
- Ather MW
- Attikis A
- Auffray E
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- Bindhu VKMN
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- Brigljevic V
- Brinkerhoff A
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- Brom J-M
- Brondolin E
- Brooke JJ
- Brown RM
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- Bruno G
- Brzhechko D
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- Buontempo S
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- Butler JN
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- Caballero IG
- Cabrera A
- Cabrillo IJ
- Cadamuro L
- Caillol C
- Cakir A
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- Calderon A
- Cali IA
- Call K
- Calligaris L
- Calzaferri S
- Camen C
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- Campagnari C
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- Campbell A
- Campderros JD
- Camporesi T
- Candelise V
- Canelli MF
- Canepa A
- Cankocak K
- Capiluppi P
- Cappati A
- Caputo C
- Caraway B
- Cardini A
- Cardwell B
- Carle A
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- Casarsa M
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- Castro A
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- Ceballos GG
- Ceccarelli R
- Cepaitis V
- Cepeda M
- Cerati GB
- Cerci DS
- Cerci S
- Cerminara G
- Cerrada M
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- Cetorelli F
- Chabert EC
- Chagas EBBD
- Chahal GS
- Chang P
- Chang P
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- Chao Y
- Chapon E
- Charaf O
- Charlot C
- Chatterjee RM
- Chatterjee S
- Chauhan S
- Chauhan S
- Chawla R
- Checchia P
- Chekhovsky V
- Chen C
- Chen GM
- Chen HS
- Chen KF
- Chen M
- Chen PH
- Chen X
- Chen Y
- Chen Y
- Chen Z
- Chenarani S
- Cheng T
- Cheng Y
- Cherepanov V
- Chernyavskaya N
- Chertok M
- Cheung HWK
- Chhibra SS
- Chiarito B
- Chinellato J
- Chistov R
- Chlebana F
- Cho S
- Choi J
- Choi S
- Choi Y
- Chou JP
- Choudhary BC
- Choudhury S
- Chowdhury SR
- Chu J
- Chudasama R
- Chwalek T
- Ciangottini D
- Cifuentes JAB
- Ciocci MA
- Cipriani M
- Ciriolo V
- Cirkovic P
- Citron M
- Cittolin S
- Ciulli V
- Civinini C
- Claes DR
- Clare R
- Clement E
- Clerbaux B
- CMS Collaboration
- Cockerill DJA
- Coelho E
- Colaleo A
- Cole JE
- Colino N
- Collard C
- Colling D
- Cometti S
- Connor P
- Contardo D
- Conway J
- Conway R
- Cooper SI
- Cooperstein S
- Corcodilos L
- Cornelis T
- Cortezon EP
- Cosby C
- Cossutti F
- Costa M
- Costa S
- Coubez X
- Couderc F
- Cousins R
- Covarelli R
- Cox B
- Cox PT
- Cranshaw DJ
- Creanza D
- Cremonesi M
- Cristella L
- Croce DD
- Cruz SS
- Csanad M
- Csorgo T
- Cuevas J
- Cuffiani M
- Cumalat JP
- Cummings G
- Cussans D
- Cutts D
- Czellar S
- D'Alessandro R
- D'Alfonso M
- D'Enterria D
- D'Hondt J
- Da Costa EM
- Da Rold A
- Da Silva De Araujo FT
- Da Silveira GG
- Dabrowski A
- Daci N
- Dalchenko M
- Dallavalle GM
- Damarseckin S
- Damgov J
- Damiani DD
- Danilov M
- Danilov V
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- Ăzçelik Ă
- Publication venue
- Publication date
- 01/01/2018
- Field of study
A search for dark matter produced in association with a Higgs boson decaying to a pair of bottom quarks is performed in proton-proton collisions at a center-of-mass energy of 13 TeV collected with the CMS detector at the LHC. The analyzed data sample corresponds to an integrated luminosity of 35.9 fb(-1). The signal is characterized by a large missing transverse momentum recoiling against a bottom quark-antiquark system that has a large Lorentz boost. The number of events observed in the data is consistent with the standard model background prediction. Results are interpreted in terms of limits both on parameters of the type-2 two-Higgs doublet model extended by an additional light pseudoscalar boson a (2HDM+a) and on parameters of a baryonic Z simplified model. The 2HDM+a model is tested experimentally for the first time. For the baryonic Z model, the presented results constitute the most stringent constraints to date.Peer reviewe
Observation of the B-s(0) -> X(3872)phi Decay
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- Ăzçelik Ă
- Publication venue
- Publication date
- 01/01/2020
- Field of study
Using a data sample of proton-proton collisions at root s = 13 TeV, corresponding to an integrated luminosity of 140 fb(-1) collected by the CMS experiment in 2016-2018, the B-s(0) -> X(3872)phi decay is observed. Decays into J/psi pi(+)pi(-) and K+K- are used to reconstruct, respectively, the X(3872) and phi. The ratio of the product of branching fractions B[B-s(0) -> X(3872)phi]B[X(3872) -> J/psi pi(+)pi(-)] to the product B[B-s(0) ->psi(2S)phi]B[psi(2S) -> J/psi pi(+)pi(-)] is measured to be [2.21 +/- 0.29(stat) +/- 0.17(syst)]%. The ratio B[B-s(0) -> X(3872)phi]/B[B-0 -> X(3872)K-0] is found to be consistent with one, while the ratio B[B-s(0) -> X(3872)phi]/B[B+-> X(3872)K+] is two times smaller. This suggests a difference in the production dynamics of the X(3872) in B-0 and B(0)s meson decays compared to B+. The reported observation may shed new light on the nature of the X(3872) particle.Peer reviewe
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