180 research outputs found
Iterative Structure-Based Peptide-Like Inhibitor Design against the Botulinum Neurotoxin Serotype A
- Author
- A Albanese
- AJ McCoy
- Antoni L. Andreu
- AR Hermone
- Axel T. Brunger
- B Willis
- BM Paddle
- BR Singh
- CB Wang
- CM Cheng
- CM Cheng
- D Gui
- D Gui
- D Kumaran
- D Kumaran
- D Kumaran
- DB Lacy
- G Schiavo
- G Schiavo
- G Schiavo
- G Schiavo
- H Li
- J Blasi
- J Blasi
- J Lipozencic
- James C. Burnett
- Jared T. Hammill
- JC Burnett
- JC Burnett
- JC Burnett
- JE Keller
- JE Zuniga
- JG Caya
- JJ Schmidt
- JJ Schmidt
- JJ Schmidt
- JJ Schmidt
- JJ Schmidt
- JJ Schmidt
- Jonathan E. Nuss
- Jorge E. Zuniga
- K Turton
- KA Foster
- KA Foster
- LM Wein
- MA Breidenbach
- MA Breidenbach
- MM Sposito
- MM Sposito
- NR Silvaggi
- NR Silvaggi
- Omri Drory
- P Carter
- P Emsley
- P Wipf
- P Wipf
- P Wipf
- PD Adams
- Peter Wipf
- R Bhidayasiri
- RG Glogau
- RG Glogau
- Rick Gussio
- RL Shapiro
- S Chen
- Sina Bavari
- SM Bachrach
- T Binz
- T Binz
- YH Ye
- Z Otwinowski
- Publication venue
- Public Library of Science
- Publication date
- 01/06/2010
- Field of study
The botulinum neurotoxin serotype A light chain (BoNT/A LC) protease is the catalytic component responsible for the neuroparalysis that is characteristic of the disease state botulism. Three related peptide-like molecules (PLMs) were designed using previous information from co-crystal structures, synthesized, and assayed for in vitro inhibition against BoNT/A LC. Our results indicate these PLMS are competitive inhibitors of the BoNT/A LC protease and their Ki values are in the nM-range. A co-crystal structure for one of these inhibitors was determined and reveals that the PLM, in accord with the goals of our design strategy, simultaneously involves both ionic interactions via its P1 residue and hydrophobic contacts by means of an aromatic group in the P2′ position. The PLM adopts a helical conformation similar to previously determined co-crystal structures of PLMs, although there are also major differences to these other structures such as contacts with specific BoNT/A LC residues. Our structure further demonstrates the remarkable plasticity of the substrate binding cleft of the BoNT/A LC protease and provides a paradigm for iterative structure-based design and development of BoNT/A LC inhibitors
Differential Subcellular Localization of the Splice Variants of the Zinc Transporter ZnT5 Is Dictated by the Different C-Terminal Regions
- Author
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2011
- Field of study
Zinc is emerging as an important intracellular signaling molecule, as well as fulfilling essential structural and catalytic functions through incorporation in a myriad of zinc metalloproteins so it is important to elucidate the molecular mechanisms of zinc homeostasis, including the subcellular localizations of zinc transporters.Two splice variants of the human SLC30A5 Zn transporter gene (ZnT5) have been reported in the literature. These variants differ at their N- and C-terminal regions, corresponding with the use of different 5' and 3' exons. We demonstrate that full length human ZnT5 variant B is a genuine transcript in human intestinal cells and confirm expression of both variant A and variant B in a range of untreated human tissues by splice variant-specific RT-PCR. Using N- or C-terminal GFP or FLAG fusions of both isoforms of ZnT5 we identify that the differential subcellular localization to the Golgi apparatus and ER respectively is a function of their alternative C-terminal sequences. These different C-terminal regions result from the incorporation into the mature transcript of either the whole of exon 14 (variant B) or only the 5' region of exon 14 plus exons 15-17 (variant A).We thus propose that exons 15 to 17 include a signal that results in trafficking of ZnT5 to the Golgi apparatus and that the 3' end of exon 14 includes a signal that leads to retention in the ER
Attentional Processing in C57BL/6J Mice Exposed to Developmental Vitamin D Deficiency
- Author
- A Bari
- A Becker
- AC Stanfield
- CA Winstanley
- CN Vidal
- D Eyles
- DAF de Abreu
- Darryl W. Eyles
- DM Eagle
- DW Eyles
- DW Eyles
- E Hollander
- F Feron
- J Feldon
- J McGrath
- J O'Loan
- J Radua
- Jared W. Young
- JJ McGrath
- JJ McGrath
- John J. McGrath
- JP Kesby
- JP Kesby
- JP Kesby
- JW Young
- Karly M. Turner
- Kenji Hashimoto
- KH Nuechterlein
- KH Nuechterlein
- Lauren R. Harms
- LR Harms
- LR Harms
- LR Harms
- MF Green
- MF Green
- N de Bruin
- NA Fineberg
- PD Harvey
- R Mongeau
- RE Lubow
- RN Hughes
- RS Keefe
- S Scarone
- SG Anagnostaras
- T Humby
- TH Burne
- THJ Burne
- Thomas H. J. Burne
- TW Robbins
- TW Robbins
- Publication venue
- Public Library of Science
- Publication date
- 26/04/2012
- Field of study
Epidemiological evidence suggests that Developmental Vitamin D (DVD) deficiency is associated with an increased risk of schizophrenia. DVD deficiency in mice is associated with altered behaviour, however there has been no detailed investigation of cognitive behaviours in DVD-deficient mice. The aim of this study was to determine the effect of DVD deficiency on a range of cognitive tasks assessing attentional processing in C57BL/6J mice. DVD deficiency was established by feeding female C57BL/6J mice a vitamin D-deficient diet from four weeks of age. After six weeks on the diet, vitamin D-deficient and control females were mated with vitamin D-normal males and upon birth of the pups, all dams were returned to a diet containing vitamin D. The adult offspring were tested on a range of cognitive behavioural tests, including the five-choice serial reaction task (5C-SRT) and five-choice continuous performance test (5C-CPT), as well as latent inhibition using a fear conditioning paradigm. DVD deficiency was not associated with altered attentional performance on the 5C-SRT. In the 5C-CPT DVD-deficient male mice exhibited an impairment in inhibiting repetitive responses by making more perseverative responses, with no changes in premature or false alarm responding. DVD deficiency did not affect the acquisition or retention of cued fear conditioning, nor did it affect the expression of latent inhibition using a fear conditioning paradigm. DVD-deficient mice exhibited no major impairments in any of the cognitive domains tested. However, impairments in perseverative responding in DVD-deficient mice may indicate that these animals have specific alterations in systems governing compulsive or reward-seeking behaviour
Search for high-mass resonances decaying to dilepton final states in pp collisions at s√=7 TeV with the ATLAS detector
- Author
- Aad G
- Abajyan T
- Abbott B
- Abdallah J
- Abdelalim AA
- Abdinov O
- Aben R
- Abi B
- Abolins M
- AbouZeid OS
- Abramowicz H
- Abreu H
- Acharya BS
- Adamczyk L
- Adams DL
- Addy TN
- Adelman J
- Adomeit S
- Adragna P
- Adye T
- Aefsky S
- Aguilar-Saavedra JA
- Agustoni M
- Aharrouche M
- Ahlen SP
- Ahles R
- Ahmad A
- Ahsan M
- Aielli G
- Akesson TPA
- Akimoto G
- Akimov AV
- Alam MA
- Alam MS
- Albert J
- Albrand S
- Aleksa M
- Aleksandrov IN
- Alessandria F
- Alexa C
- Alexander G
- Alexandre G
- Alexopoulos T
- Alhroob M
- Aliev M
- Alimonti G
- Alison J
- Allbrooke BMM
- Allport PP
- Allwood-Spiers SE
- Almenar CC
- Almond J
- Aloisio A
- Alon R
- Alonso A
- Alonso F
- Altheimer A
- Alviggi MG
- Amako K
- Amelung C
- Ammosov VV
- Amor Dos Santos SP
- Amorim A
- Amram N
- Anastopoulos C
- Ancu LS
- Andari N
- Andeen T
- Anders CF
- Anders G
- Anderson KJ
- Andreazza A
- Andrei V
- Andrieux M-L
- Anduaga XS
- Angelidakis S
- Anger P
- Angerami A
- Anghinolfi F
- Anh TV
- Anisenkov A
- Anjos N
- Annovi A
- Antonaki A
- Antonelli M
- Antonov A
- Antos J
- Anulli F
- Aoki M
- Aoun S
- Apolle R
- Arabidze G
- Aracena I
- Arai Y
- Arce ATH
- Arfaoui S
- Arguin J-F
- Argyropoulos S
- Arik E
- Arik M
- Armbruster AJ
- Arnaez O
- Arnal V
- Arnault C
- Artamonov A
- Artoni G
- Arutinov D
- Asai S
- Ask S
- Asman B
- Asquith L
- Assamagan K
- Astbury A
- Atkinson M
- Aubert B
- Auge E
- Augsten K
- Aurousseau M
- Avolio G
- Avramidou R
- Axen D
- Azuelos G
- Azuma Y
- Baak MA
- Baccaglioni G
- Bacci C
- Bach AM
- Bachacou H
- Bachas K
- Backes M
- Backhaus M
- Badescu E
- Bagnaia P
- Bahinipati S
- Bai Y
- Bailey DC
- Bain T
- Baines JT
- Baker MD
- Baker OK
- Baker S
- Balek P
- Banas E
- Banerjee P
- Banerjee S
- Banfi D
- Bangert A
- Bansal V
- Bansil HS
- Barajas CAC
- Barak L
- Baranov SP
- Barber T
- Barberio EL
- Barberis D
- Barbero M
- Bardin DY
- Barillari T
- Barisonzi M
- Barklow T
- Barlow N
- Barnett BM
- Barnett RM
- Baroncelli A
- Barone G
- Barr AJ
- Barreiro F
- Barrera CO
- Barrillon P
- Bartoldus R
- Barton AE
- Bartsch V
- Basye A
- Bates RL
- Batkova L
- Batley JR
- Battaglia A
- Battistin M
- Bauer F
- Bawa HS
- Beale S
- Beau T
- Beauchemin PH
- Beccherle R
- Bechtle P
- Beck HP
- Becker AK
- Becker S
- Beckingham M
- Becks KH
- Beddall A
- Beddall AJ
- Bedikian S
- Bednyakov VA
- Bee CP
- Beemster LJ
- Begel M
- Behera PK
- Beimforde M
- Belanger-Champagne C
- Belenguer MJ
- Bell PJ
- Bell WH
- Bella G
- Bella LA
- Bellagamba L
- Bellomo M
- Belloni A
- Beloborodova O
- Belotskiy K
- Beltramello O
- Benary O
- Benchekroun D
- Bendtz K
- Benekos N
- Benhammou Y
- Benjamin DP
- Benoit M
- Bensinger JR
- Benslama K
- Bentvelsen S
- Berge D
- Berger N
- Berghaus F
- Berglund E
- Beringer J
- Bernat R
- Bernhard R
- Bernius C
- Berry T
- Bertella C
- Bertin A
- Bertolucci F
- Besana MI
- Besjes GJ
- Besson N
- Bethke S
- Bhimji W
- Bianchi RM
- Bianchini L
- Bianco M
- Biebel O
- Bieniek SP
- Bierwagen K
- Biesiada J
- Biglietti M
- Bilokon H
- Bindi M
- Binet S
- Bingul A
- Bini C
- Biscarat C
- Bittner B
- Black CW
- Black KM
- Blair RE
- Blanchard J-B
- Blanchot G
- Blazek T
- Bloch I
- Blocker C
- Blocki J
- Blonde A
- Blum W
- Blumenschein U
- Bobbink GJ
- Bobrovnikov VS
- Bocchetta SS
- Bocci A
- Boddy CR
- Boehler M
- Boek J
- Boelaert N
- Boeriu OEV
- Bogaerts JA
- Bogdanchikov A
- Bogouch A
- Bohm C
- Bohm J
- Boisvert V
- Bold T
- Boldea V
- Bolnet NM
- Bomben M
- Bona M
- Boonekamp M
- Bordoni S
- Borer C
- Borisov A
- Borissov G
- Borjanovic I
- Borri M
- Borroni S
- Bortfeldt J
- Bortolotto V
- Bos K
- Boscherini D
- Bosman M
- Boterenbrood H
- Bouchami J
- Boudreau J
- Bouhova-Thacker EV
- Boumediene D
- Bourdarios C
- Bousson N
- Boveia A
- Boyd J
- Boyko IR
- Bozovic-Jelisavcic I
- Bracinik J
- Branchini R
- Brandt A
- Brandt G
- Brandt O
- Bratzler U
- Brau B
- Brau JE
- Braun HM
- Brazzale SF
- Brelier B
- Bremer J
- Brendlinger K
- Brenner R
- Bressler S
- Britton D
- Brochu FM
- Brock I
- Brock R
- Broggi F
- Bromberg C
- Bronner J
- Brooijmans G
- Brooks T
- Brooks WK
- Brown G
- Brown H
- Bruckman de Renstrom PA
- Bruncko D
- Bruneliere R
- Brunet S
- Bruni A
- Bruni G
- Bruschi M
- Buanes T
- Buat Q
- Bucci F
- Buchanan J
- Buchholz P
- Buckingham RM
- Buckley AG
- Buda SI
- Budagov IA
- Budick B
- Buescher V
- Bueso XP
- Bugge L
- Bulekov O
- Bundock AC
- Bunse M
- Buran T
- Burckhart H
- Burdin S
- Burgess T
- Burke S
- Busato E
- Bussey P
- Bustos ACF
- Buszello CP
- Butler B
- Butler JM
- Buttar CM
- Butterworth JM
- Buttinger W
- Byszewski M
- Cabrera Urban S
- Caforio D
- Cakir IT
- Cakir O
- Calafiura P
- Calderini G
- Calfayan P
- Calkins R
- Caloba LP
- Caloi R
- Calvet D
- Calvet S
- Camarri P
- Cameron D
- Camillocci ES
- Caminada LM
- Caminal Armadans R
- Campana S
- Campanelli M
- Canale V
- Canelli F
- Canepa A
- Cantero J
- Cantrill R
- Capasso L
- Caprini I
- Caprini M
- Capriotti D
- Capua M
- Caputo R
- Cardarelli R
- Carli T
- Carlino G
- Carminati L
- Caron B
- Caron S
- Carquin E
- Carrillo-Montoya GD
- Carter AA
- Carter JR
- Carvalho J
- Casadei D
- Casado MP
- Cascella M
- Caso C
- Castaneda Hernandez AM
- Castaneda-Miranda E
- Castanheira MTD
- Castillo IS
- Castillo LRF
- Castro NF
- Cataldi G
- Catastini R
- Catinaccio A
- Catmore JR
- Cattai A
- Cattani G
- Caughron S
- Cavalcanti TP
- Cavaliere V
- Cavalleri P
- Cavalli D
- Cavalli-Sforza M
- Cavasinni V
- Ceradini F
- Cerqueira AS
- Cerri A
- Cerrito L
- Cerutti F
- Cetin SA
- Chafaq A
- Chakraborty D
- Chalupkova I
- Chan K
- Chang P
- Chapleau B
- Chapman JD
- Chapman JW
- Chareyre E
- Charlton DG
- Chavda V
- Cheatham S
- Chekanov S
- Chekulaev SV
- Chelkov GA
- Chelstowska MA
- Chen C
- Chen H
- Chen S
- Chen X
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- Cheng Y
- Cheplakov A
- Chernyatin V
- Cheu E
- Cheung SL
- Chevalier L
- Chiefari G
- Chikovani L
- Childers JT
- Chilingarov A
- Chiodini G
- Chisholm AS
- Chislett RT
- Chitan A
- Chizhov MV
- Choudalakis G
- Chouridou S
- Christidi IA
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- Chromek-Burckhart D
- Chu ML
- Chudoba J
- Ciapetti G
- Ciftci AK
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- Cinca D
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- Ciocca C
- Ciocio A
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- Citron ZH
- Citterio M
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- Clemens JC
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- Codina EP
- Coffey L
- Cogan JG
- Coggeshall J
- Cogneras E
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- Colijn AP
- Collaboration A
- Collins NJ
- Collins-Tooth C
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- Compostella G
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- Coniavitis E
- Conidi MC
- Consonni SM
- Consorti V
- Constantinescu S
- Conta C
- Conti G
- Conventi F
- Cooke M
- Cooper BD
- Cooper-Sarkar AM
- Copic K
- Cornelissen T
- Corradi M
- Correia AMH
- Corriveau F
- Cortes-Gonzalez A
- Cortiana G
- Costa G
- Costa MJ
- Costanzo D
- Cote D
- Courneyea L
- Cowan G
- Cowden C
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- Cranmer K
- Crepe-Renaudin S
- Crescioli F
- Cristinziani M
- Crosetti G
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- Curtis CJ
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- Czirr H
- Czodrowski P
- Czyczula Z
- D'Auria S
- D'Onofrio M
- D'Orazio A
- da Costa JBG
- Da Costa JGPF
- Da Cunha Sargedas De Sousa MJ
- Da Via C
- Dabrowski W
- Dafinca A
- Dai T
- Dallapiccola C
- Dam M
- Damazio DO
- Dameri M
- Damiani DS
- Danielsson HO
- Dao V
- Darbo G
- Darlea GL
- Dassoulas JA
- Davey W
- Davidek T
- Davidson N
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- Davies E
- Davies M
- Davignon O
- Davison AR
- Davygora Y
- Dawe E
- Dawson I
- Daya-Ishmukhametova RK
- de Asmundis R
- De Castro S
- De Cecco S
- de Graat J
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- de Jong P
- De la Taille C
- De la Torre H
- de Lima DEF
- de Lima JGR
- De Lorenzi F
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- De Nooij L
- De Pedis D
- De Regie JBDV
- De Salvo A
- De Sanctis U
- De Santo A
- De Zorzi G
- De K
- Dearnaley WJ
- Debbe R
- Debenedetti C
- Dechenaux B
- Dedovich DV
- Degenhardt J
- Del Peso J
- Del Prete T
- Delemontex T
- Deliyergiyev M
- Dell'Acqua A
- Dell'Asta L
- Della Pietra M
- della Porta GZ
- della Volpe D
- Delmastro M
- Delsart PA
- Deluca C
- Demers S
- Demichev M
- Demirkoz B
- Denis RDS
- Denisov SP
- Derendarz D
- Derkaoui JE
- Derue F
- Dervan P
- Desch K
- Devetak E
- Deviveiros PO
- Dewhurst A
- DeWilde B
- Dhaliwal S
- Dhullipudi R
- Di Ciaccio A
- Di Ciaccio L
- Di Donato C
- Di Girolamo A
- Di Girolamo B
- Di Luise S
- Di Mattia A
- Di Micco B
- Di Nardo R
- Di Simone A
- Di Sipio R
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- Diehl EB
- Dietrich J
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- Dingfelder J
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- Dionisi C
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- Djama F
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- do Vale MAB
- Doan TKO
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- Dodd J
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- Doherty T
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- Dolezal Z
- Dolgoshein BA
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- Donini J
- Donszelmann TC
- Dopke J
- Doria A
- Dos Anjos A
- Dos Santos DR
- Dotti A
- Dova MT
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- Dris M
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- Dudarev A
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- Duerdoth IP
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- Edwards CA
- Edwards NC
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- Eigen G
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- Eisenhandler E
- Ekelof T
- El Kacimi M
- El Moursli RC
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- Ereditato A
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- Espinal Curull X
- Esposito B
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- Etienvre AI
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- Fabre C
- Fajardo LSG
- Fakhrutdinov RM
- Falciano S
- Fang Y
- Fanti M
- Farbin A
- Farilla A
- Farley J
- Farooque T
- Farrell S
- Farrington SM
- Farthouat P
- Fassi F
- Fassnacht P
- Fassouliotis D
- Fatholahzadeh B
- Favareto A
- Fayard L
- Fazio S
- Febbraro R
- Federic P
- Fedin OL
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- Fehling-Kaschek M
- Feligioni L
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- Fenyuk AB
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- Ferrando BMS
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- Ferrari P
- Ferrari R
- Ferrer A
- Ferrere D
- Ferretti C
- Fiascaris M
- Fiedler F
- Filipcic A
- Filthaut F
- Fincke-Keeler M
- Fiolhais MCN
- Fiorini L
- Firan A
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- Fisher MJ
- Flechl M
- Fleck I
- Fleckner J
- Fleischmann P
- Fleischmann S
- Flick T
- Floderus A
- Flowerdew MJ
- Formica A
- Forti A
- Fortin D
- Fournier D
- Fowler AJ
- Fox H
- Francavilla P
- Franchini M
- Franchino S
- Francis D
- Frank T
- Franklin M
- Franz S
- Fraternali M
- Fratina S
- French ST
- Friedrich C
- Friedrich F
- Froeschl R
- Froidevaux D
- Frost JA
- Fukunaga C
- Fulsom BG
- Fuster J
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- Gabizon T
- Gadfort T
- Gadomski S
- Gagliardi G
- Gagnon P
- Galea C
- Galhardo B
- Gallas EJ
- Gallo V
- Gallop BJ
- Gallus P
- Galtieri AB
- Gan KK
- Gao YS
- Gaponenko A
- Garberson F
- Garcia Navarro JE
- Garcia BRM
- Garcia C
- Garcia JAB
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- Gardner RW
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- Garitaonandia H
- Garonne V
- Garrido MDMC
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- Gaudio G
- Gaur B
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- Gavrilenko IL
- Gay C
- Gaycken G
- Gazis EN
- Ge P
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- Gee CNP
- Geerts DAA
- Geich-Gimbel C
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- Gemme C
- Gemmell A
- Genest MH
- Gentile S
- George M
- George S
- Gerlach P
- Gershon A
- Geweniger C
- Ghazlane H
- Ghodbane N
- Giacobbe B
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- Gimenez VC
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- Gladilin LK
- Glasman C
- Glatzer J
- Glazov A
- Glitza KW
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- Goepfert T
- Goeringer C
- Goessling C
- Goldfarb S
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- Zhemchugov A
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- Zibell A
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- Zinonos Z
- Ziolkowski M
- Zitoun R
- Zivkovic L
- Zmouchko VV
- Zobernig G
- Zoccoli A
- zur Nedden M
- Zutshi V
- Zwalinski L
- Publication venue
- Springer
- Publication date
- 23/11/2012
- Field of study
The ATLAS detector at the Large Hadron Collider is used to search for high-mass resonances decaying to an electron-positron pair or a muon-antimuon pair. The search is sensitive to heavy neutral Z′ gauge bosons, Randall-Sundrum gravitons, Z * bosons, techni-mesons, Kaluza-Klein Z/γ bosons, and bosons predicted by Torsion models. Results are presented based on an analysis of pp collisions at a center-of-mass energy of 7 TeV corresponding to an integrated luminosity of 4.9 fb−1 in the e + e − channel and 5.0 fb−1 in the μ + μ −channel. A Z ′ boson with Standard Model-like couplings is excluded at 95 % confidence level for masses below 2.22 TeV. A Randall-Sundrum graviton with coupling k/MPl=0.1 is excluded at 95 % confidence level for masses below 2.16 TeV. Limits on the other models are also presented, including Technicolor and Minimal Z′ Models
Observation of associated near-side and away-side long-range correlations in √sNN=5.02 TeV proton-lead collisions with the ATLAS detector
- Author
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- Yan Z
- Yang H
- Yang H
- Yang UK
- Yang Y
- Yang Z
- Yanush S
- Yao L
- Yasu Y
- Yatsenko E
- Ye J
- Ye S
- Yen AL
- Yilmaz M
- Yoosoofmiya R
- Yorita K
- Yoshida R
- Yoshihara K
- Young C
- Young CJ
- Youssef S
- Yu D
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- Yu J
- Yuan L
- Yurkewicz A
- Zabinski B
- Zaidan R
- Zaitsev AM
- Zambito S
- Zanello L
- Zanzi D
- Zaytsev A
- Zeitnitz C
- Zeman M
- Zemla A
- Zenin O
- Zeniš T
- Zerwas D
- Zevi Della Porta G
- Zhang D
- Zhang H
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- Zhao L
- Zhao Z
- Zhemchugov A
- Zhong J
- Zhou B
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- Zhu CG
- Zhu H
- Zhu J
- Zhu Y
- Zhuang X
- Zhuravlov V
- Zibell A
- Zieminska D
- Zimin NI
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zitoun R
- Zivković L
- Zmouchko VV
- Zobernig G
- Zoccoli A
- Zur Nedden M
- Zutshi V
- Zwalinski L
- Publication venue
- American Physical Society
- Publication date
- 01/01/2012
- Field of study
Two-particle correlations in relative azimuthal angle (Δϕ) and pseudorapidity (Δη) are measured in √sNN=5.02 TeV p+Pb collisions using the ATLAS detector at the LHC. The measurements are performed using approximately 1 μb-1 of data as a function of transverse momentum (pT) and the transverse energy (ΣETPb) summed over 3.1<η<4.9 in the direction of the Pb beam. The correlation function, constructed from charged particles, exhibits a long-range (2<|Δη|<5) “near-side” (Δϕ∼0) correlation that grows rapidly with increasing ΣETPb. A long-range “away-side” (Δϕ∼π) correlation, obtained by subtracting the expected contributions from recoiling dijets and other sources estimated using events with small ΣETPb, is found to match the near-side correlation in magnitude, shape (in Δη and Δϕ) and ΣETPb dependence. The resultant Δϕ correlation is approximately symmetric about π/2, and is consistent with a dominant cos2Δϕ modulation for all ΣETPb ranges and particle pT
Search for the neutral Higgs bosons of the minimal supersymmetric standard model in pp collisions at root s=7 TeV with the ATLAS detector
- Author
- Aad G
- Abajyan T
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- Abdallah J
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- Publication venue
- Publication date
- 01/02/2013
- Field of study
A search for neutral Higgs bosons of the Minimal Supersymmetric Standard Model (MSSM) is reported. The analysis is based on a sample of proton-proton collisions at a centre-of-mass energy of 7TeV recorded with the ATLAS detector at the Large Hadron Collider. The data were recorded in 2011 and correspond to an integrated luminosity of 4.7 fb-1 to 4.8 fb-1. Higgs boson decays into oppositely-charged muon or τ lepton pairs are considered for final states requiring either the presence or absence of b-jets. No statistically significant excess over the expected background is observed and exclusion limits at the 95% confidence level are derived. The exclusion limits are for the production cross-section of a generic neutral Higgs boson, φ, as a function of the Higgs boson mass and for h/A/H production in the MSSM as a function of the parameters mA and tan β in the mhmax scenario for mA in the range of 90GeV to 500 GeV. Copyright CERN
Search for R-parity-violating supersymmetry in events with four or more leptons in sqrt(s) =7 TeV pp collisions with the ATLAS detector
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- Winkelmann S
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- Yabsley B
- Yacoob S
- Yamada M
- Yamaguchi H
- Yamamoto A
- Yamamoto K
- Yamamoto S
- Yamamura T
- Yamanaka T
- Yamazaki T
- Yamazaki Y
- Yan Z
- Yang H
- Yang UK
- Yang Y
- Yang Z
- Yanush S
- Yao L
- Yao Y
- Yasu Y
- Smit GVY
- Ye J
- Ye S
- Yilmaz M
- Yoosoofmiya R
- Yorita K
- Yoshida R
- Yoshihara K
- Young C
- Young CJ
- Youssef S
- Yu D
- Yu DR
- Yu J
- Yu J
- Yuan L
- Yurkewicz A
- Zabinski B
- Zaidan R
- Zaitsev AM
- Zajacova Z
- Zanello L
- Zanzi D
- Zaytsev A
- Zeitnitz C
- Zeman M
- Zemla A
- Zendler C
- Zenin O
- Zenis T
- Zinonos Z
- Zerwas D
- della Porta GZ
- Zhang D
- Zhang H
- Zhang J
- Zhang X
- Zhang Z
- Zhao L
- Zhao Z
- Zhemchugov A
- Zhong J
- Zhou B
- Zhou N
- Zhou Y
- Zhu CG
- Zhu H
- Zhu J
- Zhu Y
- Zhuang X
- Zhuravlov V
- Zibell A
- Zieminska D
- Zimin NI
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Ziolkowski M
- Zitoun R
- Zivkovic L
- Zmouchko VV
- Zobernig G
- Zoccoli A
- zur Nedden M
- Zutshi V
- Zwalinski L
- Collaboration ATLAS
- Publication venue
- Springer Verlag
- Publication date
- 01/01/2008
- Field of study
A search for new phenomena in final states with four or more leptons (electrons or muons) is presented. The analysis is based on 4.7 fb−1 of s=7TeV proton-proton collisions delivered by the Large Hadron Collider and recorded with the ATLAS detector. Observations are consistent with Standard Model expectations in two signal regions: one that requires moderate values of missing transverse momentum and another that requires large effective mass. The results are interpreted in a simplified model of R-parity-violating supersymmetry in which a 95% CL exclusion region is set for charged wino masses up to 540 GeV. In an R-parity-violating MSUGRA/CMSSM model, values of m 1/2 up to 820 GeV are excluded for 10 < tan β < 40
Dual selection enhances the signaling specificity of a variant of the quorum-sensing transcriptional activator LuxR
- Author
- A Aharoni
- AL Schaefer
- AL Schaefer
- AL Schaefer
- BP Cormack
- C Fuqua
- CH Collins
- Cynthia H Collins
- D Karig
- Frances H Arnold
- GS Shadel
- H Kobayashi
- I Matsumura
- Jared R Leadbetter
- JD Bloom
- JJ Havranek
- KA Egland
- KM Gray
- L You
- ME Taga
- ML Urbanowski
- MR Parikh
- NA Whitehead
- NC Shaner
- PC Cirino
- R Weiss
- R Weiss
- RM Horton
- S Basu
- S Basu
- SM Gould
- Y Voziyanov
- Y Yokobayashi
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Noncardiac genetic predisposition in sudden infant death syndrome.
- Author
- AJ Yun
- Alban-Elouen Baruteau
- Amie Jaye
- Belinda Gray
- CA Brownstein
- D Fard
- David J. Tester
- DE Weese-Mayer
- DE Weese-Mayer
- DE Weese-Mayer
- DW Norstrand Van
- E Cutz
- Elijah R. Behr
- HC Kinney
- HF Krous
- Iona Jeffrey
- Jacob Tfelt-Hansen
- Jared M. Evans
- JJ Filiano
- JR Lemke
- K Laer
- L Ferrante
- L Filonzi
- Leonie CH Wong
- M Frigeni
- M Kircher
- Margaret Evans
- Marta Cohen
- Michael A. Simpson
- Michael J. Ackerman
- N Lahrouchi
- N Salomonis
- Peter Fleming
- PJ Schwartz
- Pritha Chanana
- Q Li
- RM Hofstra
- RWL Mannikko.
- RY Moon
- S Richards
- SE Olpin
- T Yamamoto
- TJ Matthews
- WG Guntheroth
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 24/08/2018
- Field of study
PURPOSE: Sudden infant death syndrome (SIDS) is the commonest cause of sudden death of an infant; however, the genetic basis remains poorly understood. We aimed to identify noncardiac genes underpinning SIDS and determine their prevalence compared with ethnically matched controls. METHODS: Using exome sequencing we assessed the yield of ultrarare nonsynonymous variants (minor allele frequency [MAF] ≤0.00005, dominant model; MAF ≤0.01, recessive model) in 278 European SIDS cases (62% male; average age =2.7 ± 2 months) versus 973 European controls across 61 noncardiac SIDS-susceptibility genes. The variants were classified according to American College of Medical Genetics and Genomics criteria. Case-control, gene-collapsing analysis was performed in eight candidate biological pathways previously implicated in SIDS pathogenesis. RESULTS: Overall 43/278 SIDS cases harbored an ultrarare single-nucleotide variant compared with 114/973 controls (15.5 vs. 11.7%, p=0.10). Only 2/61 noncardiac genes were significantly overrepresented in cases compared with controls (ECE1, 3/278 [1%] vs. 1/973 [0.1%] p=0.036; SLC6A4, 2/278 [0.7%] vs. 1/973 [0.1%] p=0.049). There was no difference in yield of pathogenic or likely pathogenic variants between cases and controls (1/278 [0.36%] vs. 4/973 [0.41%]; p=1.0). Gene-collapsing analysis did not identify any specific biological pathways to be significantly associated with SIDS. CONCLUSIONS: A monogenic basis for SIDS amongst the previously implicated noncardiac genes and their encoded biological pathways is negligible
Variants of the EAAT2 Glutamate Transporter Gene Promoter Are Associated with Cerebral Palsy in Preterm Infants
- Author
- A Mitani
- AH MacLennan
- Alexandra C. Spittle
- Anikó Váradi
- Avon Premature Infant Project
- Charlene Crosby
- CM Elitt
- D Sun
- D Wilson-Costello
- David Harding
- David Odd
- DE Odd
- DJ Field
- Elek Molnár
- FE Jensen
- H Spiers
- HE Shin
- J Feng
- J Mallolas
- J Zschocke
- Jared Ching
- JJ Volpe
- JJ Volpe
- JP Boardman
- K Luyt
- K Luyt
- K Tanaka
- Karen Luyt
- KD McCarthy
- KL Costeloe
- L Vivo de
- LJ Mangham
- Louis Dwomoh
- LS Vries de
- M Endres
- M Spangaro
- Maggie Williams
- Marie Owen
- Matthew T. Robinson
- Miles Wagstaff
- N Marlow
- NC Danbolt
- NJ Maragakis
- NS Wood
- O Pampliega
- PJ Mitchell
- R Fern
- R Galinsky
- R Lister
- S Rajatileka
- S Rajatileka
- S Sparrow
- SA Back
- SE Andiman
- Shavanthi Rajatileka
- SP Miller
- TM DeSilva
- TM DeSilva
- TM DeSilva
- X-D Chao
- Y Yang
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2017
- Field of study
© 2017, The Author(s). Preterm delivery is associated with neurodevelopmental impairment caused by environmental and genetic factors. Dysfunction of the excitatory amino acid transporter 2 (EAAT2) and the resultant impaired glutamate uptake can lead to neurological disorders. In this study, we investigated the role of single nucleotide polymorphisms (SNPs; g.-200CCloseSPigtSPiA and g.-181ACloseSPigtSPiC) in the EAAT2 promoter in susceptibility to brain injury and neurodisability in very preterm infants born at or before 32-week gestation. DNA isolated from newborns’ dried blood spots were used for pyrosequencing to detect both SNPs. Association between EAAT2 genotypes and cerebral palsy, cystic periventricular leukomalacia and a low developmental score was then assessed. The two SNPs were concordant in 89.4% of infants resulting in three common genotypes all carrying two C and two A alleles in different combinations. However, in 10.6% of cases, non-concordance was found, generating six additional rare genotypes. The A alleles at both loci appeared to be detrimental and consequently, the risk of developing cerebral palsy increased four- and sixfold for each additional detrimental allele at -200 and -181bp, respectively. The two SNPs altered the regulation of the EAAT2 promoter activity and glutamate homeostasis. This study highlights the significance of glutamate in the pathogenesis of preterm brain injury and subsequent development of cerebral palsy and neurodevelopmental disabilities. Furthermore, the described EAAT2 SNPs may be an early biomarker of vulnerability to neurodisability and may aid the development of targeted treatment strategies
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