On-Shell Unitarity Bootstrap for QCD Amplitudes

We describe the recently developed on-shell bootstrap for computing one-loop amplitudes in non-supersymmetric theories such as QCD. The method combines the unitarity method with loop-level on-shell recursion. The unitarity method is used to compute cut-containing parts of amplitudes, and on-shell recursion is used for the remaining rational terms.Comment: 10 pages, 9 figures, write-up of talks given by Z. Bern and D. A. Kosower at Loops & Legs 2006, Eisenach, Germany; v2: added referenc

Complete Genome Sequence of Serotype III Streptococcus agalactiae Sequence Type 17 Strain 874391.

Here we report the complete genome sequence of Streptococcus agalactiae strain 874391. This serotype III isolate is a member of the hypervirulent sequence type 17 (ST-17) lineage that causes a disproportionate number of cases of invasive disease in humans and mammals. A brief historical context of the strain is discussed

Anesthetic action on the transmission delay between cortex and thalamus explains the beta-buzz observed under propofol anesthesia

In recent years, more and more surgeries under general anesthesia have been performed with the assistance of electroencephalogram (EEG) monitors. An increase in anesthetic concentration leads to characteristic changes in the power spectra of the EEG. Although tracking the anesthetic-induced changes in EEG rhythms can be employed to estimate the depth of anesthesia, their precise underlying mechanisms are still unknown. A prominent feature in the EEG of some patients is the emergence of a strong power peak in the β–frequency band, which moves to the α–frequency band while increasing the anesthetic concentration. This feature is called the beta-buzz. In the present study, we use a thalamo-cortical neural population feedback model to reproduce observed characteristic features in frontal EEG power obtained experimentally during propofol general anesthesia, such as this beta-buzz. First, we find that the spectral power peak in the α– and δ–frequency ranges depend on the decay rate constant of excitatory and inhibitory synapses, but the anesthetic action on synapses does not explain the beta-buzz. Moreover, considering the action of propofol on the transmission delay between cortex and thalamus, the model reveals that the beta-buzz may result from a prolongation of the transmission delay by increasing propofol concentration. A corresponding relationship between transmission delay and anesthetic blood concentration is derived. Finally, an analytical stability study demonstrates that increasing propofol concentration moves the systems resting state towards its stability threshold

Shaping 3D root system architecture

Plants are sessile organisms rooted in one place. The soil resources that plants require are often distributed in a highly heterogeneous pattern. To aid foraging, plants have evolved roots whose growth and development are highly responsive to soil signals. As a result, 3D root architecture is shaped by myriad environmental signals to ensure resource capture is optimised and unfavourable environments are avoided. The first signals sensed by newly germinating seeds — gravity and light — direct root growth into the soil to aid seedling establishment. Heterogeneous soil resources, such as water, nitrogen and phosphate, also act as signals that shape 3D root growth to optimise uptake. Root architecture is also modified through biotic interactions that include soil fungi and neighbouring plants. This developmental plasticity results in a ‘custom-made’ 3D root system that is best adapted to forage for resources in each soil environment that a plant colonises

Nitrate uptake and its regulation in relation to improving nitrogen use efficiency in cereals

On average less than half of the applied N is captured by crops, thus there is scope and need to improveN uptake in cereals. With nitrate (NO3−) being the main form of N available to cereal crops there hasbeen a significant global research effort to understand plant NO3−uptake. Despite this, our knowledgeof the NO3−uptake system is not sufficient to easily target ways to improve NO3−uptake. Based on thisthere is an identified need to better understand the NO3−uptake system and the signalling moleculesthat modulate it. With strong transcriptional control governing the NO3−uptake system, we also neednew leads for modulating transcription of NO3−transporter genesDarren C. Plett, Luke R. Holtham, Mamoru Okamoto, Trevor P. Garnet