48 research outputs found

    Terahertz electron-hole recollisions in GaAs/AlGaAs quantum wells: robustness to scattering by optical phonons and thermal fluctuations

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    Electron-hole recollisions are induced by resonantly injecting excitons with a near-IR laser at frequency fNIRf_{\text{NIR}} into quantum wells driven by a ~10 kV/cm field oscillating at fTHz=0.57f_{\text{THz}} = 0.57 THz. At T=12T=12 K, up to 18 sidebands are observed at frequencies fsideband=fNIR+2nfTHzf_{\text{sideband}}=f_{\text{NIR}}+2n f_{\text{THz}}, with 82n28-8 \le 2n \le 28. Electrons and holes recollide with total kinetic energies up to 57 meV, well above the ELO=36E_{\text{LO}} = 36 meV threshold for longitudinal optical (LO) phonon emission. Sidebands with order up to 2n=222n=22 persist up to room temperature. A simple model shows that LO phonon scattering suppresses but does not eliminate sidebands associated with kinetic energies above ELOE_{\text{LO}}.Comment: 5 pages, 4 figure

    Renal amyloidosis in children

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    Renal amyloidosis is a detrimental disease caused by the deposition of amyloid fibrils. A child with renal amyloidosis may present with proteinuria or nephrotic syndrome. Chronic renal failure may follow. Amyloid fibrils may deposit in other organs as well. The diagnosis is through the typical appearance on histopathology. Although chronic infections and chronic inflammatory diseases used to be the causes of secondary amyloidosis in children, the most frequent cause is now autoinflammatory diseases. Among this group of diseases, the most frequent one throughout the world is familial Mediterranean fever (FMF). FMF is typically characterized by attacks of clinical inflammation in the form of fever and serositis and high acute-phase reactants. Persisting inflammation in inadequately treated disease is associated with the development of secondary amyloidosis. The main treatment is colchicine. A number of other monogenic autoinflammatory diseases have also been identified. Among them cryopyrin-associated periodic syndrome (CAPS) is outstanding with its clinical features and the predilection to develop secondary amyloidosis in untreated cases. The treatment of secondary amyloidosis mainly depends on the treatment of the disease. However, a number of new treatments for amyloid per se are in the pipeline

    The Kuramoto model in complex networks

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    181 pages, 48 figures. In Press, Accepted Manuscript, Physics Reports 2015 Acknowledgments We are indebted with B. Sonnenschein, E. R. dos Santos, P. Schultz, C. Grabow, M. Ha and C. Choi for insightful and helpful discussions. T.P. acknowledges FAPESP (No. 2012/22160-7 and No. 2015/02486-3) and IRTG 1740. P.J. thanks founding from the China Scholarship Council (CSC). F.A.R. acknowledges CNPq (Grant No. 305940/2010-4) and FAPESP (Grants No. 2011/50761-2 and No. 2013/26416-9) for financial support. J.K. would like to acknowledge IRTG 1740 (DFG and FAPESP).Peer reviewedPreprin
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