9 research outputs found
The Prevalence of Bacterial Infection in Patients Undergoing Elective ACDF for Degenerative Cervical Spine Conditions: A Prospective Cohort Study With Contaminant Control
- Author
- Publication venue
- SAGE Publications
- Publication date
- 01/01/2021
- Field of study
Study Design:
Prospective cohort study.
Objectives:
To determine the prevalence of bacterial infection, with the use of a contaminant control, in patients undergoing anterior cervical discectomy and fusion (ACDF).
Methods:
After institutional review board approval, patients undergoing elective ACDF were prospectively enrolled. Samples of the longus colli muscle and disc tissue were obtained. The tissue was then homogenized, gram stained, and cultured in both aerobic and anaerobic medium. Patients were classified into 4 groups depending on culture results. Demographic, preoperative, and postoperative factors were evaluated.
Results:
Ninety-six patients were enrolled, 41.7% were males with an average age of 54 ± 11 years and a body mass index of 29.7 ± 5.9 kg/m2. Seventeen patients (17.7%) were considered true positives, having a negative control and positive disc culture. Otherwise, no significant differences in culture positivity was found between groups of patients. However, our results show that patients were more likely to have both control and disc negative than being a true positive (odds ratio = 6.2, 95% confidence interval = 2.5-14.6). Propionibacterium acnes was the most commonly identified bacteria. Two patients with disc positive cultures returned to the operating room secondary to pseudarthrosis; however, age, body mass index, prior spine surgery or injection, postoperative infection, and reoperations were not associated with culture results.
Conclusion:
In our cohort, the prevalence of subclinical bacterial infection in patients undergoing ACDF was 17.7%. While our rates exclude patients with positive contaminant control, the possibility of contamination of disc cultures could not be entirely rejected. Overall, culture results did not have any influence on postoperative outcomes
Mapping the human genetic architecture of COVID-19
- Author
- Ahmad H.
- Alarcon C.
- Albertos R.
- Algera A.
- Andrews S.
- Aschard H.
- Aslibekyan S.
- Atanasovska B.
- Auton A.
- Azuure C.
- Baillie J.
- Baillie S.
- Balaconis M.
- Ball C.
- Banagan J.
- Barbour A.
- Bax D.
- Bernasconi A.
- Beudel M.
- Biesecker L.
- Birney E.
- Boer C.
- Bomers M.
- Bonta P.
- Bos L.
- Botta M.
- Boua P.
- Brouwer M.
- Bugiani M.
- Bulle E.
- Butler-Laporte G.
- Buxbaum J.
- Byun J.
- Cadilla C.
- Canakoglu A.
- Carnero-Montoro E.
- Chouchane O.
- Chwialkowska K.
- Cloherty A.
- Coignet M.
- Coker D.
- Colombo F.
- Cordioli M.
- Daly M.
- Darwish D.
- Davis L.
- de Brabander J.
- de Bree G.
- de Bruin S.
- de Jong D.
- de Vries H.
- Deelen P.
- Dongelmans D.
- Dueker N.
- Dunham I.
- Dutta A.
- Elbers P.
- Esmaeeli S.
- Esparza-Gordillo J.
- Faucon A.
- Favé M.
- Fernandez-Cadenas I.
- Ferwerda B.
- Filshtein-Sonmez T.
- Finucane H.
- Fleuren L.
- Folkersen L.
- Francescatto M.
- Francioli L.
- Franke L.
- Friedman P.
- Ganna A.
- Garmendia A.
- Geerlings S.
- Geerts B.
- Geijtenbeek T.
- Ghoussaini M.
- Girbes A.
- Goorhuis B.
- Grobusch M.
- Gómez-Cabrero D.
- Hafkamp F.
- Hagens L.
- Hamann J.
- Hamidi Z.
- Han C.
- Han Y.
- Harris V.
- Harry E.
- Hemke R.
- Hermans S.
- Herrmann S.
- Heunks L.
- Hollmann M.
- Horn J.
- Hovius J.
- Im H.
- Im S.
- Jan Bogaard H.
- Jansen P.
- Julienne H.
- Kaja E.
- Kanai M.
- Kanoni S.
- Karim M.
- Karjalainen J.
- Kaunisto M.
- Kennis-Szilagyi I.
- Kerchberger V.
- Kim H.
- Kim S.
- Knight S.
- Koelling N.
- Koning R.
- Kornilov S.
- Kousathanas A.
- Kromhout A.
- Lee S.
- Lee Y.
- Lemaçon A.
- Lenz T.
- Li R.
- Liao R.
- Lim J.
- Limou S.
- Lio P.
- Mari F.
- Marouli E.
- Martin A.
- Marttila M.
- Mazurek S.
- Mbarek H.
- McCurdy S.
- Medina-Gómez C.
- Mehtonen J.
- Meltzer D.
- Migeotte I.
- Minica C.
- Minnaar R.
- Moltke I.
- Moreno D.
- Moreno-Estrada A.
- Moya L.
- Nakanishi T.
- Nasir J.
- Neale B.
- Nellen J.
- Nguyen H.
- Niemi M.
- Nossent E.
- Nutescu E.
- Okada Y.
- O’Brien T.
- O’Connell J.
- O’Donnell P.
- O’Leary K.
- Park D.
- Partha R.
- Pasaniuc B.
- Pasko D.
- Patel S.
- Pathak G.
- Paulus F.
- Pearson N.
- Perera M.
- Perumal S.
- Peters E.
- Polimanti R.
- Posthuma D.
- Preckel B.
- Priest J.
- Prijatelj V.
- Prins J.
- Prokić I.
- Pérez-Tur J.
- Raasveld J.
- Raffat N.
- Rahmouni S.
- Reijnders T.
- Renieri A.
- Rhead B.
- Richards J.
- Roberts G.
- Sankaran V.
- Savage J.
- Schinkel M.
- Schulte E.
- Schultz M.
- Schurmann C.
- Schuurman A.
- Schwartzentruber J.
- Sedaghati-Khayat B.
- Shastri A.
- Shelton J.
- Shi H.
- Sigaloff K.
- Sipeky C.
- Sivanadhan I.
- Sloofman L.
- Smit M.
- Smith G.
- Solomonson M.
- Song H.
- Stijnis C.
- Stilma W.
- Striano P.
- Symons A.
- Szentpeteri J.
- Tanigawa Y.
- Teles A.
- Teumer A.
- Teunissen C.
- Thoral P.
- Tissink E.
- Trankiem A.
- Tsonas A.
- Tuck M.
- Uddin M.
- Uddin M.
- Uffelmann E.
- Utrilla A.
- Vadgama N.
- Vallerga C.
- van Agtmael M.
- van Baarle F.
- van de Beek D.
- van der Poll T.
- van der Valk M.
- van Heel D.
- van Mourik N.
- van Uffelen J.
- van Vugt M.
- Varnai R.
- Veelo D.
- Veerapen K.
- Verdugo R.
- Vlaar A.
- von Hohenstaufen K.
- Wang Q.
- Weldon C.
- Wendt F.
- Wiersinga W.
- Willer C.
- Wolford B.
- Wolterman R.
- Wouters D.
- Yang G.
- Ye C.
- Yengo L.
- Zhou W.
- Zwinderman K.
- Zárate R.
- Özer O.
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2021
- Field of study
The genetic make-up of an individual contributes to the susceptibility and response to viral infection. Although environmental, clinical and social factors have a role in the chance of exposure to SARS-CoV-2 and the severity of COVID-19(1,2), host genetics may also be important. Identifying host-specific genetic factors may reveal biological mechanisms of therapeutic relevance and clarify causal relationships of modifiable environmental risk factors for SARS-CoV-2 infection and outcomes. We formed a global network of researchers to investigate the role of human genetics in SARS-CoV-2 infection and COVID-19 severity. Here we describe the results of three genome-wide association meta-analyses that consist of up to 49,562 patients with COVID-19 from 46 studies across19 countries. We report 13 genome-wide significant loci that are associated with SARS-CoV-2 infection or severe manifestations of COVID-19. Several of these loci correspond to previously documented associations to lung or autoimmune and inflammatory diseases(3-7). They also represent potentially actionable mechanisms in response to infection. Mendelian randomization analyses support a causal role for smoking and body-mass index for severe COVID-19 although not for type II diabetes. The identification of novel host genetic factors associated with COVID-19 was made possible by the community of human genetics researchers coming together to prioritize the sharing of data, results, resources and analytical frameworks. This working model of international collaboration underscores what is possible for future genetic discoveries in emerging pandemics, or indeed for any complex human disease.Radiolog
RECENT RESEARCH ON RED SQUILL AS A RODENTICIDE
- Publication venue
- DigitalCommons@University of Nebraska - Lincoln
- Publication date
- 01/01/1986
- Field of study
Red squill has been used in rodent control for several hundred years but in the United States its use has decreased since the 1950s. However, there is now a recognized need for rodenticides with different kinds of toxic activity. Red squill is being investigated as an acute rodenticide and an economic crop for the southwestern states. Clones from a prior USDA collection have been assayed by high-performance liquid chromatography and selections are being propagated in California and Arizona. The major toxicant, scilliroside, is relatively fast acting, causing convulsions and death to rats and mice. This glycoside is also strongly emetic to humans, cats and dogs, affording a safety factor uncommon to high-toxicity rodenticides. Our chemical, processing, agronomic, and toxicological studies are a technical basis for further developing this potentially superior rodenticide
Safety and the Anatomy of the Retroperitoneal Lateral Corridor with Respect to the Minimally Invasive Lateral Lumbar Intervertebral Fusion Approach
- Author
- Publication venue
- 'Elsevier BV'
- Publication date
- Field of study
Mapping the human genetic architecture of COVID-19
- Author
- Abdelrazik M.
- Abdullah T.
- Abe R.
- Abe S.
- Abecasis G. R.
- Abel L.
- Abernathy C.
- Abraheem A.
- Abul-Husn N. S.
- Acosta-Herrera M.
- Acquilini D.
- Adachi T.
- Adachi Y.
- Adams C.
- Adams K.
- Adanini O.
- Adeleye O.
- Adeniji K.
- Adra D.
- Afifi N.
- Afilalo J.
- Afilalo M.
- Afolabi D.
- Afrasiabi Z.
- Afset J. E.
- Agasou A.
- Aghemo A.
- Agranoff D.
- Agrawal S.
- Aguirre L. A.
- Agwuh K.
- Ahmad H. F.
- Ahmad N.
- Ahmed A.
- Ahmed C.
- Ahmed K. A.
- Ai M.
- Ail D.
- Akeroyd L.
- Akhtar M. N.
- Akhtar S.
- Akinkugbe O.
- Aksentijevich A.
- Al Thani A.
- Al-Muftah W.
- Al-Sarraj Y.
- Alarcon C.
- Alarcon-Riquelme M. E.
- Alasdair F.
- Alaverdian D.
- Alavere H.
- Albertos R.
- Albillos A.
- Albrecht W.
- Albrich W.
- Albrich W. C.
- Aldera E. L.
- Aldridge J.
- Alegria A.
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- Alfonso J.
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- Ali A.
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- Aliberti S.
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- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2021
- Field of study
The genetic make-up of an individual contributes to the susceptibility and response to viral infection. Although environmental, clinical and social factors have a role in the chance of exposure to SARS-CoV-2 and the severity of COVID-191,2, host genetics may also be important. Identifying host-specific genetic factors may reveal biological mechanisms of therapeutic relevance and clarify causal relationships of modifiable environmental risk factors for SARS-CoV-2 infection and outcomes. We formed a global network of researchers to investigate the role of human genetics in SARS-CoV-2 infection and COVID-19 severity. Here we describe the results of three genome-wide association meta-analyses that consist of up to 49,562 patients with COVID-19 from 46 studies across 19 countries. We report 13 genome-wide significant loci that are associated with SARS-CoV-2 infection or severe manifestations of COVID-19. Several of these loci correspond to previously documented associations to lung or autoimmune and inflammatory diseases3–7. They also represent potentially actionable mechanisms in response to infection. Mendelian randomization analyses support a causal role for smoking and body-mass index for severe COVID-19 although not for type II diabetes. The identification of novel host genetic factors associated with COVID-19 was made possible by the community of human genetics researchers coming together to prioritize the sharing of data, results, resources and analytical frameworks. This working model of international collaboration underscores what is possible for future genetic discoveries in emerging pandemics, or indeed for any complex human disease
Biogeochemical processes and geotechnical applications: progress, opportunities and challenges
- Author
- Banagan B. L.
- Bang S.
- Barnes R. S. K.
- Bonala M. V. S.
- Burbank M.
- Cabalar A. F.
- Chaney R.
- Chou C.-W.
- Cullimore D. R.
- Cunningham A.
- Darcy H.
- Day J. L.
- DeJong J. T.
- DeJong J. T.
- Ehlers C. J.
- Fauriel S.
- Gray D. H.
- Hamdan N.
- Ishihara K.
- Ivanov V.
- Karatas I.
- Kavazanjian E.
- Kavazanjian E.
- Kohnhauser K.
- Kuo M. Y. H.
- Li M.
- Mitchell J. K.
- NRC
- Ramachandran S. K.
- Schofield A. N.
- Sherif M. A.
- Sivavec T.
- Slichter C. S.
- Taylor D.
- Terzaghi K.
- Terzaghi K.
- Van Meurs G.
- Van Paassen L. A.
- Weil M. H.
- Publication venue
- 'Thomas Telford Ltd.'
- Publication date
- Field of study
Biogeochemical processes and geotechnical applications: progress, opportunities and challenges
- Author
- A. AL QABANY
- A. AYDILEK
- A. ESNAULT-FILET
- A. PALOMINO
- B. MARTINEZ
- B. TANYU
- B.M. MONTOYA
- Banagan B. L.
- Bang S.
- Barnes R. S. K.
- Bonala M. V. S.
- Burbank M.
- C.Y. CHEN
- Cabalar A. F.
- Chaney R.
- Chou C.-W.
- Chu J.
- Cullimore D. R.
- Cunningham A.
- D.A.C. MANNING
- D.C. NELSON
- Darcy H.
- Day J. L.
- DeJong J. T.
- DeJong J. T.
- E. KAVAZANJIAN
- E.A. SEAGREN
- Ehlers C. J.
- Fauriel S.
- Gray D. H.
- Hamdan N.
- Ishihara K.
- Ivanov V.
- J. CHU
- J. LARRAHONDO
- J.C. SANTAMARINA
- J.T. DEJONG
- K. SOGA
- Karatas I.
- Kavazanjian E.
- Kavazanjian E.
- Kohnhauser K.
- Kuo M. Y. H.
- L. LALOUI
- L.A. VAN PAASSEN
- L.F. CASLAKE
- Legge K. R.
- Li M.
- M. BURBANK
- M. KUO
- M. TSESARSKY
- Martinez B. C.
- Martinez B. C.
- Martinez B. C.
- Mitchell J. K.
- Montoya B. M.
- N. HAMDAN
- NRC
- P. RENFORTH
- Ramachandran S. K.
- S. BURNS
- S. CIURLI
- S. FAURIEL
- S. JEFFERIS
- S.S. BANG
- Sanderson K.
- Schofield A. N.
- Sherif M. A.
- Sivavec T.
- Slichter C. S.
- T. HATA
- T. WEAVER
- Taylor D.
- Terzaghi K.
- Terzaghi K.
- Van Meurs G.
- Van Paassen L. A.
- Weil M. H.
- X. CHENG
- Y. INAGAKI
- Publication venue
- 'Thomas Telford Ltd.'
- Publication date
- Field of study
Recommended from our members
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- Author
- Abbasifard Salman
- Abd-El-Barr Muhammad M
- Ahmadian Amir A
- Ahn Junyoung
- Alikhani Puya
- Asghar Jahangir
- Baaj Ali A
- Bamford Colin R
- Banagan Kelley
- Basho Rahul
- Bono Christopher M
- Burkett Clinton J
- Buser Zorica
- Bydon Ali
- Bydon Mohamad
- Chang Steven W
- Chapman Jr. Todd M
- Chapman Jens R
- Chin Matthew
- Chou Dean
- Corredor José A
- Dasenbrock Hormuzdiyar H
- Dhall Sanjay S
- Eastman Jonathan G
- Elder Benjamin D
- Ferrone Marco
- Filis Andreas K
- Foley Kevin T
- Fridley Jared
- Garfin Steven R
- Garza-Ramos Rafael De la
- Gerszten Peter Carlos
- Ghobrial George M
- Gokaslan Ziya L
- Goodwin C. Rory
- Greenberg Mark S
- Groff Michael W
- Groves Mari L
- Harris Colin B
- Harrop James S
- Hobbs Jonathan
- Horak Holli A
- Horne Phillip
- Hsieh Patrick C
- Hwang Steven W
- Härtl Roger
- Jackson Keith
- Jallo George I
- James Whitney S
- Jea Andrew
- Joaquim Andrei Fernandes
- Kakarla U. Kumar
- Kalra Ricky
- Kanter Adam S
- Khalsa Amrit
- Kiely Paul D
- Klineberg Eric O
- Kosztowski Thomas
- Le Tien V
- Lee Joon Y
- Lenke Lawrence G
- Levi Allan D
- Long III William D
- Lu Daniel C
- Ludwig Steven C
- Manzano Glen R
- Marca Frank La
- Mashaly Hazem A
- Massel Dustin H
- Mayo Benjamin C
- Miller Justin W
- Minges David
- Mitha Alim P
- Modi Krishna D
- Molina Camilo
- Momin Eric
- Monaco III Edward A
- Mummaneni Praveen V
- Murray Gisela
- Oskouian Jr. Rod J
- Palejwala Sheri
- Papanastassiou Ioannis
- Park Paul
- Patel Amit R
- Patel Kamakshi
- Phillips Frank M
- Poelstra Kornelis
- Ponnappan Ravi K
- Presciutti Steven M
- Quirno Martin
- Ramachandran Ravi
- Ramey Wyatt L
- Ramos Edwin
- Readdy William J
- Rhee Jay
- Roguski Marie
- Rowan F. Andrew
- Saigal Rajiv
- Sama Andrew A
- Samdani Amer F
- Sasso Rick C
- Schmidt Meic H
- Schroeder Gregory D
- Sciubba Daniel M
- Shridharani Shyam M
- Shufflebarger Harry L
- Siemionow Krzysztof
- Singh Harminder
- Singh Kern
- Skoch Jesse
- Sosio Jessica
- Sribnick Eric
- Tempel Zachary J
- Than Khoi D
- Toy Jason O
- Tuchman Alexander
- Uribe Juan S
- Uschold Timothy D
- Vaccaro Alexander R
- Valdivia Juan M
- Vale Fernando L
- Varthi Arya Giri
- Vasudeva Viren S
- Volkov Andrey Alex
- Vrionis Frank D
- Wang Jeffrey C
- Wang Michael Y
- Whang Peter G
- Witham Timothy F
- Wolinsky Jean-Paul
- Wu Jau-Ching
- Yeung Anthony T
- Yeung Christopher
- Zaidi Hasan A
- Zlomislic Vinko
- Zussman Benjamin M
- Publication venue
- Stuttgart
- Publication date
- 01/01/2016
- Field of study
Mapping the human genetic architecture of COVID-19
- Author
- Abdelrazik M
- Abdullah T
- Abe R
- Abe S
- Abecasis GR
- Abel L
- Abernathy C
- Abraheem A
- Abul-Husn NS
- Acosta-Herrera M
- Acquilini D
- Acquilini D
- Adachi T
- Adachi Y
- Adams C
- Adams K
- Adanini O
- Adeleye O
- Adeniji K
- Adra D
- Afifi N
- Afilalo J
- Afilalo M
- Afolabi D
- Afrasiabi Z
- Afset JE
- Agasou A
- Aghemo A
- Agranoff D
- Agrawal S
- Aguirre LA
- Agwuh K
- Ahmad HF
- Ahmad N
- Ahmed A
- Ahmed C
- Ahmed KA
- Ai M
- Ail D
- Akeroyd L
- Akhtar MN
- Akhtar S
- Akinkugbe O
- Aksentijevich A
- Al Thani A
- Al-Muftah W
- Al-Sarraj Y
- Alarcon C
- Alarcon-Riquelme ME
- Alasdair F
- Alaverdian D
- Alavere H
- Albertos R
- Albillos A
- Albrecht W
- Albrich W
- Albrich WC
- Aldera EL
- Aldridge J
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- Cossarizza A
- Costantino G
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- Coton Z
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- de Gordoa LO-R
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- De la Horra C
- De Neef M
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- de Rojas I
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- zu Bentrup FM
- Zucchi P
- Zwinderman AHK
- Zyndorf M
- Publication venue
- Publication date
- 01/01/2021
- Field of study
The genetic make-up of an individual contributes to the susceptibility and response to viral infection. Although environmental, clinical and social factors have a role in the chance of exposure to SARS-CoV-2 and the severity of COVID-191,2, host genetics may also be important. Identifying host-specific genetic factors may reveal biological mechanisms of therapeutic relevance and clarify causal relationships of modifiable environmental risk factors for SARS-CoV-2 infection and outcomes. We formed a global network of researchers to investigate the role of human genetics in SARS-CoV-2 infection and COVID-19 severity. Here we describe the results of three genome-wide association meta-analyses that consist of up to 49,562 patients with COVID-19 from 46 studies across 19 countries. We report 13 genome-wide significant loci that are associated with SARS-CoV-2 infection or severe manifestations of COVID-19. Several of these loci correspond to previously documented associations to lung or autoimmune and inflammatory diseases3,4,5,6,7. They also represent potentially actionable mechanisms in response to infection. Mendelian randomization analyses support a causal role for smoking and body-mass index for severe COVID-19 although not for type II diabetes. The identification of novel host genetic factors associated with COVID-19 was made possible by the community of human genetics researchers coming together to prioritize the sharing of data, results, resources and analytical frameworks. This working model of international collaboration underscores what is possible for future genetic discoveries in emerging pandemics, or indeed for any complex human disease