79 research outputs found
Malaria Liver Stage Susceptibility Locus Identified on Mouse Chromosome 17 by Congenic Mapping
- Author
- A Fortin
- A Fortin
- A Fortin
- AV Hill
- AV Hill
- C van de Sand
- Carlos Penha-Gonçalves
- CG Huh
- E Lin
- E Nagayasu
- E Ngonseu
- G Min-Oo
- G Min-Oo
- G Min-Oo
- J Lederberg
- KW Broman
- L Flori
- LĂgia Antunes Gonçalves
- M Carrolo
- M Hernandez-Valladares
- M Hernandez-Valladares
- Maria Manuel Mota
- N Sepulveda
- O Silvie
- P Leiriao
- Paulo Almeida
- Pieter Hendrik Reitsma
- RA Burt
- RA Burt
- S Bagot
- S Bennett
- S Campino
- SJ Foote
- T Ohno
- W McGuire
- WR Weiss
- ZM Khan
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2008
- Field of study
Host genetic variants are known to confer resistance to Plasmodium blood stage infection and to control malaria severity both in humans and mice. This work describes the genetic mapping of a locus for resistance to liver stage parasite in the mouse. First, we show that decreased susceptibility to the liver stage of Plasmodium berghei in the BALB/c mouse strain is attributable to intra-hepatic factors and impacts on the initial phase of blood stage infection. We used QTL mapping techniques to identify a locus controlling this susceptibility phenotype (LOD score 4.2) on mouse chromosome 17 (belr1 locus). Furthermore, analysis of congenic mouse strains delimited the belr1 locus boundaries distally to the H2 region. Quantification of parasites in the liver of infected congenic mice strongly suggested that the belr1 locus represents a genetic factor controlling the expansion of P. berghei in the hepatic tissue. The mapping of belr1 locus raises the hypothesis that host gene variation is able to control the progression of Plasmodium liver stage infection and opens the possibility that the human genomic region orthologue to belr1 may contain genes that confer resistance to the human malaria liver stage
Demographic and reproductive associations with nematode infection in a long-lived mammal
- Author
- A Stien
- A Zahavi
- AD Hayward
- AJJ MacIntosh
- AJJ MacIntosh
- AK Simon
- AP Galvani
- BH Craig
- BH Craig
- CA Cizauskas
- CL Lynsdale
- D Bates
- D Ebert
- D Sol
- DH Nussey
- DJ Shaw
- DM Tompkins
- EM Labes
- EM Warburton
- F Pelletier
- H Mpairwe
- HD Crofton
- IA Barger
- IL King
- J Schindelin
- JAH Crawley
- JL Gittleman
- JM Parker
- JM Setchell
- K Norris
- L Baines
- L Cheynel
- M Barthelemy
- M Festa-Bianchet
- M Grzybek
- M LahdenperÀ
- M LahdenperÀ
- M Thurber
- M Zuk
- M Zuk
- MA Beck
- MA Stoffel
- MG Watve
- MJ Denwood
- MJ Smith
- ML East
- ML Roberts
- N Ferrari
- N Ferrari
- O Halvorsen
- P Agnew
- R Clubb
- R Poulin
- R Tinsley
- RJ Wiese
- S Catalano
- SD Albon
- SL Balenger
- SS Godfrey
- WD Hamilton
- YZ Foo
- ZM Oo
- ZM Oo
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 08/06/2020
- Field of study
Infection by macroparasites, such as nematodes, varies within vertebrate host systems; elevated infection is commonly observed in juveniles and males, and, for females, with different reproductive states. However, while such patterns are widely recognized in short-lived model systems, how they apply to long-lived hosts is comparatively understudied. Here, we investigated how infection varies with host age, sex, and female reproduction in a semi-captive population of individually marked Asian elephants Elephas maximus. We carried out 1,977 faecal egg counts (FECs) across five years to estimate nematode loads for 324 hosts. Infection patterns followed an established age-infection curve, whereby calves (5 years) exhibited the highest FECs and adults (45 years) the lowest. However, males and females had similar FECs across their long lifespan, despite distinct differences in life-history strategy and clear sexual dimorphism. Additionally, although mothers invest two years in pregnancy and a further three to five years into lactation, nematode load did not vary with four different measures of female reproduction. Our results provide a much-needed insight into the host-parasite dynamics of a long-lived host; determining host-specific associations with infection in such systems is important for broadening our knowledge of parasite ecology and provides practical applications for wildlife medicine and management
Prevalence, awareness, treatment, and control of hypertension in Nigeria in 1995 and 2020: A systematic analysis of current evidence
- Author
- Publication venue
- 'Wiley'
- Publication date
- 18/02/2021
- Field of study
Improved understanding of the current burden of hypertension, including awareness, treatment, and control, is needed to guide relevant preventative measures in Nigeria. A systematic search of studies on the epidemiology of hypertension in Nigeria, published on or after January 1990, was conducted. The authors employed randomeffects meta-analysis on extracted crude hypertension prevalence, and awareness, treatment, and control rates. Using a meta-regression model, overall hypertension cases in Nigeria in 1995 and 2020 were estimated. Fifty-three studies (n = 78 949) met our selection criteria. Estimated crude prevalence of pre-hypertension (120-139/80-89 mmHg) in Nigeria was 30.9% (95% confidence interval [CI]: 22.0%-39.7%), and the crude prevalence of hypertension (â„140/90 mmHg) was 30.6% (95% CI: 27.3%-34.0%). When adjusted for age, study period, and sample, absolute cases of hypertension increased by 540% among individuals aged â„20 years from approximately 4.3 million individuals in 1995 (age-adjusted prevalence 8.6%, 95% CI: 6.5-10.7) to 27.5 million individuals with hypertension in 2020 (age-adjusted prevalence 32.5%, 95% CI: 29.8-35.3). The age-adjusted prevalence was only significantly higher among men in 1995, with the gap between both sexes considerably narrowed in 2020. Only 29.0% of cases (95% CI: 19.7-38.3) were aware of their hypertension, 12.0% (95% CI: 2.7-21.2) were on treatment, and 2.8% (95% CI: 0.1-5.7) had at-goal blood pressure in 2020. Our study suggests that hypertension prevalence has substantially increased in Nigeria over the last two decades. Although more persons are aware of their hypertension status, clinical treatment and control rates, however, remain low. These estimates are relevant for clinical care, population, and policy response in Nigeria and across Africa
NGF Causes TrkA to Specifically Attract Microtubules to Lipid Rafts
- Author
- A Ueda
- AF Palazzo
- AP Hibbert
- AW Harrington
- B Winckler
- BA Tsui-Pierchala
- BJ Nichols
- BP Head
- C Bandtlow
- C Neumann-Giesen
- C Wu
- Catherine Faivre-Sarrailh
- CF Ibåñez
- CL Howe
- CS Bartheld von
- CS Huang
- D Drubin
- D Esposito
- D Hoeller
- D Lingwood
- D Wloga
- DG Drubin
- E Perret
- EC Beattie
- F François
- F Miller
- F-Q Zhou
- F-Q Zhou
- FC Bronfman
- G Dechant
- G Ferrari
- G Mccaffrey
- G Paratcha
- G Valdez
- G Valdez
- Gretchen McCaffrey
- H Chen
- H Higuchi
- H Kamiguchi
- IH Madshus
- J Zheng
- JPS Makkerh
- K Deinhardt
- K Haglund
- K Simons
- K Simons
- L Sadowski
- LA Greene
- LA Schwarz
- Lindsay R. Young
- LJ Huber
- LJ Pike
- LS Zweifel
- M Canossa
- M Lakadamyali
- M Maccormick
- M Nishio
- MA del Pozo
- Mark L. Grimes
- ML Grimes
- ML Grimes
- ML Grimes
- N Fukushima
- N Galjart
- OO Glebov
- P Philippidou
- PA Hantzopoulos
- RC Aguilar
- S Bamji
- S Polo
- S Polo
- S Saxena
- S Sigismund
- S Suzuki
- Shona Pryor
- SJ Dixon
- SJ Rabin
- T Farooqui
- T Geetha
- T Hryciw
- T Mutoh
- T Yamashita
- TR Bilderback
- W Yu
- Y de Pablo
- Y Shao
- ZM Lasiecka
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2012
- Field of study
Membrane protein sorting is mediated by interactions between proteins and lipids. One mechanism that contributes to sorting involves patches of lipids, termed lipid rafts, which are different from their surroundings in lipid and protein composition. Although the nerve growth factor (NGF) receptors, TrkA and p75NTR collaborate with each other at the plasma membrane to bind NGF, these two receptors are endocytosed separately and activate different cellular responses. We hypothesized that receptor localization in membrane rafts may play a role in endocytic sorting. TrkA and p75NTR both reside in detergent-resistant membranes (DRMs), yet they responded differently to a variety of conditions. The ganglioside, GM1, caused increased association of NGF, TrkA, and microtubules with DRMs, but a decrease in p75NTR. When microtubules were induced to polymerize and attach to DRMs by in vitro reactions, TrkA, but not p75NTR, was bound to microtubules in DRMs and in a detergent-resistant endosomal fraction. NGF enhanced the interaction between TrkA and microtubules in DRMs, yet tyrosine phosphorylated TrkA was entirely absent in DRMs under conditions where activated TrkA was detected in detergent-sensitive membranes and endosomes. These data indicate that TrkA and p75NTR partition into membrane rafts by different mechanisms, and that the fraction of TrkA that associates with DRMs is internalized but does not directly form signaling endosomes. Rather, by attracting microtubules to lipid rafts, TrkA may mediate other processes such as axon guidance
Global, regional, and national incidence, prevalence, and mortality of HIV, 1980â2017, and forecasts to 2030, for 195 countries and territories: a systematic analysis for the Global Burden of Diseases, Injuries, and Risk Factors Study 2017
- Author
- Abbastabar H
- Abu-Raddad LJ
- Abyu DM
- Adabi M
- Adebayo OM
- Adekanmbi V
- Adetokunboh OO
- Ahmadi A
- Ahmadi K
- Ahmadian E
- Ahmadpour E
- Ahmed MB
- Akal CG
- Alahdab F
- Alam N
- Albertson SB
- Alemnew BTT
- Alene KA
- Alipour V
- Alvis-Guzman N
- Amini S
- Anbari Z
- Anber NH
- Anjomshoa M
- Antonio CAT
- Arabloo J
- Aremu O
- Areri HA
- Arora M
- Asfaw ET
- Ashagre AF
- Asmelash D
- Asrat AA
- Avokpaho EFGA
- Awasthi A
- Awoke N
- Ayanore MA
- Azari S
- Badawi A
- Bagherzadeh M
- Banach M
- Barac A
- Basu S
- Bedi N
- Behzadifar M
- Bekele BB
- Belay SA
- Belay YB
- Belayneh YM
- Berhane A
- Bhat AG
- Bhattacharyya K
- Biadgo B
- Biehl MH
- Bijani A
- Bin Sayeed MS
- Bitew H
- Blinov A
- Bogale KA
- Bojia HA
- Burugina Nagaraja SBN
- Butt ZA
- BĂ€rnighausen TW
- Cahuana-Hurtado L
- Campuzano Rincon JC
- Carter A
- Carvalho F
- Chattu VK
- Christopher DJ
- Chu D-T
- Crider R
- Dahiru T
- Dandona L
- Dandona R
- Daryani A
- Das Neves J
- De Neve J-W
- Degenhardt L
- Demeke FM
- Demis AB
- Demissie DB
- Demoz GT
- Deribe K
- Des Jarlais D
- Dhungana GP
- Diaz D
- Djalalinia S
- Do HP
- Doan LP
- Douwes-Schultz D
- Duber H
- Dubey M
- Dubljanin E
- Duken EE
- Duko Adema B
- Dwyer-Lindgren L
- Effiong A
- Eftekhari A
- El Sayed Zaki M
- El-Jaafary SI
- El-Khatib Z
- Elsharkawy A
- Endries AY
- Eskandarieh S
- Eyawo O
- Farzadfar F
- Fatima B
- Fentahun N
- Fernandes E
- Filip I
- Fischer F
- Folayan MO
- Foroutan M
- Frank TD
- Fukumoto T
- Fullman N
- Garcia-Basteiro AL
- Gayesa RT
- Gebremedhin KB
- Gebremeskel GGG
- Gebreyohannes KK
- Gedefaw GA
- Gelaw BK
- Gesesew HA
- Geta B
- Gezae KE
- Ghadiri K
- Ghashghaee A
- Ginindza TTG
- Gugnani HC
- GuimarĂŁes RA
- Haile MT
- Hailu GB
- Haj-Mirzaian A
- Haj-Mirzaian A
- Hamidi S
- Handanagic S
- Handiso DW
- Hanfore LK
- Hasanzadeh A
- Hassankhani H
- Hassen HY
- Hay SI
- Henok A
- Hoang CL
- Hosgood HD
- Hosseinzadeh M
- Hsairi M
- Ibitoye SE
- Idrisov B
- Ikuta KS
- Ilesanmi OS
- Irvani SSN
- Iwu CJ
- Jacobsen KH
- Jahagirdar D
- James SL
- Jenabi E
- Jha RP
- Jonas JB
- Jorjoran Shushtari Z
- Kabir A
- Kabir Z
- Kadel R
- Kasaeian A
- Kassa B
- Kassa GM
- Kassa TD
- Kayode GA
- Kebede MM
- Kefale AT
- Kengne AP
- Khader YS
- Khafaie MA
- Khalid N
- Khan EA
- Khan G
- Khan J
- Khang Y-H
- Khatab K
- Khazaei S
- Khoja AT
- Kiadaliri AA
- Kim YJ
- Kisa A
- Kisa S
- Kochhar S
- Komaki H
- Koul PA
- Koyanagi A
- Kuate Defo B
- Kumar GA
- Kumar M
- Kuupiel D
- Kyu HH
- Lal DK
- Larson SL
- Lee JJ-H
- Lenjebo TL
- Leshargie CT
- Lim SS
- Macarayan ERK
- Maddison ER
- Magdy Abd El Razek H
- Magis-Rodriguez C
- Mahasha PW
- Majdan M
- Majeed A
- Malekzadeh R
- Manafi N
- Mapoma CC
- Martins-Melo FR
- Masaka A
- Mayenga ENL
- Mehta V
- Meles GG
- Meles HG
- Melese A
- Melku M
- Memiah PTN
- Memish ZA
- Mena AT
- Mendoza W
- Mengistu DT
- Mengistu G
- Meretoja TJ
- Mestrovic T
- Miller TR
- Moazen B
- Mohajer B
- Mohamadi-Bolbanabad A
- Mohammad Darwesh A
- Mohammad Gholi Mezerji N
- Mohammad KA
- Mohammad Y
- Mohammadi M
- Mohammadibakhsh R
- Mohammadoo-Khorasani M
- Mohammed JA
- Mohammed S
- Mohebi F
- Mokdad AH
- Moodley Y
- Moossavi M
- Moradi G
- Moradi-Lakeh M
- Moschos MM
- Mossie TB
- Mousavi SM
- Muchie KF
- Muluneh AG
- Muriithi MK
- Murray CJL
- Mustafa G
- Muthupandian S
- Nagarajan AJ
- Naghavi M
- Naik G
- Najafi F
- Nazari J
- Ndwandwe DE
- Nguyen CT
- Nguyen HLT
- Nguyen SH
- Nguyen TH
- Ningrum DNA
- Nixon MR
- Nnaji CA
- Noroozi M
- Noubiap JJ
- Nourollahpour Shiadeh M
- Obsa MS
- Odame EA
- Ofori-Asenso R
- Ogbo FA
- Okoro A
- Oladimeji O
- Olagunju AT
- Olagunju TO
- Olum S
- Oppong Asante KOA
- Oren E
- Otstavnov SS
- PA M
- Padubidri JR
- Pakhale S
- Pakpour AH
- Patel SK
- Paulos K
- Pepito VCF
- Peprah EK
- Piroozi B
- Pourshams A
- Qorbani M
- Rabiee M
- Rabiee N
- Radfar A
- Rafay A
- Rafiei A
- Rahim F
- Rahimi-Movaghar A
- Rahimi-Movaghar V
- Rahman SU
- Ranabhat CL
- Rawaf S
- Reis C
- Renjith V
- Reta MA
- Rezai MS
- Rios GonzĂĄlez CM
- Roro EM
- Rostami A
- Rubino S
- Saeedi Moghaddam S
- Safari S
- Sagar R
- Sahraian MA
- Salem MRR
- Salimi Y
- Salomon JA
- Sambala EZ
- Samy AM
- Sartorius B
- Satpathy M
- Sawhney M
- Sayyah M
- Schutte AE
- Sepanlou SG
- Seyedmousavi S
- Shabaninejad H
- Shaheen AA
- Shaikh MA
- Shallo SA
- Shamsizadeh M
- Sharifi H
- Shibuya K
- Shin JI
- Shirkoohi R
- Silva DAS
- Silveira DGA
- Singh JA
- Sisay M
- Sisay MMM
- Sisay S
- Smith AE
- Sokhan A
- Somayaji R
- Soshnikov S
- Stein DJ
- Steuben KM
- Sufiyan MB
- Sunguya BF
- Sykes BL
- Tadesse BT
- Tadesse DB
- Tamirat KS
- Taveira N
- Tekelemedhin SW
- Temesgen HD
- Tesfay FH
- Teshale MY
- Thapa S
- Tlaye KG
- Topp SM
- Tovani-Palone MR
- Tran BX
- Tran KB
- Ullah I
- Unnikrishnan B
- Uthman OA
- Veisani Y
- Vladimirov SK
- Vollset SE
- Wada FW
- Waheed Y
- Wang H
- Weldegwergs KG
- Weldesamuel GTT
- Westerman R
- Wijeratne T
- Wolde HF
- Wondafrash DZ
- Wonde TE
- Wondmagegn BY
- Yeshanew AG
- Yilma MT
- Yimer EM
- Yonemoto N
- Yotebieng M
- Youm Y
- Yu C
- Zaidi Z
- Zarghi A
- Zenebe ZM
- Zewale TA
- Ziapour A
- Zodpey S
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2019
- Field of study
Background
Understanding the patterns of HIV/AIDS epidemics is crucial to tracking and monitoring the progress of prevention and control efforts in countries. We provide a comprehensive assessment of the levels and trends of HIV/AIDS incidence, prevalence, mortality, and coverage of antiretroviral therapy (ART) for 1980â2017 and forecast these estimates to 2030 for 195 countries and territories.
Methods
We determined a modelling strategy for each country on the basis of the availability and quality of data. For countries and territories with data from population-based seroprevalence surveys or antenatal care clinics, we estimated prevalence and incidence using an open-source version of the Estimation and Projection Packageâa natural history model originally developed by the UNAIDS Reference Group on Estimates, Modelling, and Projections. For countries with cause-specific vital registration data, we corrected data for garbage coding (ie, deaths coded to an intermediate, immediate, or poorly defined cause) and HIV misclassification. We developed a process of cohort incidence bias adjustment to use information on survival and deaths recorded in vital registration to back-calculate HIV incidence. For countries without any representative data on HIV, we produced incidence estimates by pulling information from observed bias in the geographical region. We used a re-coded version of the Spectrum model (a cohort component model that uses rates of disease progression and HIV mortality on and off ART) to produce age-sex-specific incidence, prevalence, and mortality, and treatment coverage results for all countries, and forecast these measures to 2030 using Spectrum with inputs that were extended on the basis of past trends in treatment scale-up and new infections.
Findings
Global HIV mortality peaked in 2006 with 1·95 million deaths (95% uncertainty interval 1·87â2·04) and has since decreased to 0·95 million deaths (0·91â1·01) in 2017. New cases of HIV globally peaked in 1999 (3·16 million, 2·79â3·67) and since then have gradually decreased to 1·94 million (1·63â2·29) in 2017. These trends, along with ART scale-up, have globally resulted in increased prevalence, with 36·8 million (34·8â39·2) people living with HIV in 2017. Prevalence of HIV was highest in southern sub-Saharan Africa in 2017, and countries in the region had ART coverage ranging from 65·7% in Lesotho to 85·7% in eSwatini. Our forecasts showed that 54 countries will meet the UNAIDS target of 81% ART coverage by 2020 and 12 countries are on track to meet 90% ART coverage by 2030. Forecasted results estimate that few countries will meet the UNAIDS 2020 and 2030 mortality and incidence targets.
Interpretation
Despite progress in reducing HIV-related mortality over the past decade, slow decreases in incidence, combined with the current context of stagnated funding for related interventions, mean that many countries are not on track to reach the 2020 and 2030 global targets for reduction in incidence and mortality. With a growing population of people living with HIV, it will continue to be a major threat to public health for years to come. The pace of progress needs to be hastened by continuing to expand access to ART and increasing investments in proven HIV prevention initiatives that can be scaled up to have population-level impact
Laparoscopy in management of appendicitis in high-, middle-, and low-income countries: a multicenter, prospective, cohort study.
- Author
- Abantanga F
- Abbas M
- Abbo O
- Abd El Galeel A
- Abd El Hameed OS
- Abd El Hamid N
- Abd El Salam Y
- Abd El Slam M
- Abd El-Salam F
- Abd Elrasoul Y
- Abd-Elrasoul Y
- Abdallah E
- Abdel-Wahab NYE
- AbdelFattah I
- Abdelgelil A
- Abdelghany S
- Abdelkader M
- Abdelkareem A
- Abdelraheim M
- Abdelshafy M
- Abdulgawad M
- Abdullah AAN
- Abdullah NAN
- Abebe NS
- Abebe NS
- Aber A
- Abington E
- Aboarab A
- Aboraya M
- Aboraya M
- Abouelnasr A
- Abouzahra A
- Abozaid M
- Abozeid K
- Abozyed MAF
- Abudeeb H
- Abuseif M
- Acharya P
- Adams R
- Adebanjo A
- Adekoya A
- Adel A
- Adel A
- Ademuyiwa A
- Ademuyiwa AO
- Adeniran J
- Adeniyi A
- Adesanya O
- Adesina A
- Adesuyi A
- Adeyeye A
- Adham W
- Afandy A
- Afifi AM
- Afzal M
- Agalianos C
- Agarwal A
- Agius M
- Aguilar-Jimenez J
- Aguilera M
- Aguilera O
- Ahlqvist S
- Ahmed A
- Ahmed EA
- Ahmed H
- Ahmed SM
- Aigrain Y
- Ajao AE
- Akeel N
- Akhtar M
- Al Awwad S
- Al-Aarag AA-R
- Al-Azraqi IAA
- Al-Faifi JJ
- Al-Hameedi HII
- Al-Hasani RKMJ
- Al-Mallah A
- Al-Mousa M
- Al-Nahrawi S
- Al-Obaedi O
- Al-shammari A
- Alahmady O
- Alakaloko F
- Alamoudi U
- Albeladi B
- Aldalaq A
- Aldhafeeri S
- Aledrisy M
- Alfarsi MA
- Alghamdi B
- Alghamdi M
- Algohary O
- Alhabli I
- Alhendy M
- Ali A
- Ali A
- Ali H
- Ali SS
- Alimi Q
- Aljiffry M
- Aljohani E
- Aljohani S
- Alkammash A
- Alkhabbaz D
- Alkhouri M
- Allam M
- Allegri A
- Allen JLY
- Allott R
- Almallah A
- Almoflihi A
- Almuallem S
- Almy J
- Alnawam E
- Alnuqaydan SA
- Alomar IN
- Alqahtani E
- Alrahawy M
- Alrajraji M
- Alrifaie A
- AlRowais M
- Alsaggaf M
- Alsaggaf MU
- Altaf A
- Altamimi A
- Althwainy A
- Altoe F
- Altwigry AM
- Alvarado Jaramillo R
- Alvarez Barreda LM
- Alwafai MG
- Aly AK
- Alyami R
- AlYoussef I
- Alzahrani M
- Alzayat T
- Amandito R
- Ambrozeviciute V
- Ameen M
- Amer MA
- Amin J
- Ammar M
- Ammar MS
- Amreia M
- Anandkumar D
- Anderson D
- Anderson R
- Anderson-Knight H
- Andersson L
- Andres Garcia-Marin J
- Ansaloni L
- Antar S
- Anthoulakis C
- Anwar AW
- Anwer W
- Anyanwu L-J
- Aql S
- Arachchi PP
- Arachchige LAJJ
- Aranzabal Durand SY
- Araujo R
- Ardley R
- Arenas A
- Arevalo Azmitia JR
- Arias Pacheco RD
- Arman S
- Arnaud AP
- Arnaud AP
- Arroyo Basto CA
- Arulampalam T
- Asal M
- Asele FA
- Ashraf SF
- Ashton A
- Assouto PA
- Ata AAA
- Atkinson K
- Atobatele K
- Attallah NA
- Attard M
- Attia AM
- Aung KP
- Austraite A
- Awuku-Asabre J
- Ayad S
- Ayandipo OO
- Ayyar S
- Azmitia Mendizabal CA
- Azodo IA
- Azzie G
- Badr A
- Badwi NM
- Badwy A
- Baheeg M
- Bailey K
- Baillie K
- Baird A
- Bakhaidar M
- Baldacchino M
- Balila RMH
- Balkan AZA
- Balmaceda R
- Bamicha A
- Barakat SAE
- Barker T
- Barnacle J
- Barneo L
- Barrionuevo Ojeda RR
- Barrios E
- Barrios E
- Barros De Castro Segundo LC
- Barroso de Lima CV
- Barry J
- Basson S
- Batista Lemaire S
- Batt J
- Beasley W
- Beaton C
- Behar N
- Beisa V
- Benevento A
- Benger M
- Benons N
- Beuran M
- Beyene R
- Bezerra Morais PA
- Bezzina M
- Bhangu A
- Bhargava A
- Bharj IS
- Bhat S
- Bhattacharya S
- Bhopal KF
- Bin Mahamood A
- Birring A
- Bjorklund I
- Blencowe N
- Blower E
- Blundell R
- Boakye-Yiadom K
- Boal M
- Boddy A
- Bode C
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- Iqbal FM
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- Macfarlane A
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- Mclean AL
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- Mcnish D
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- Mendoza I
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- Menshawy A
- Mentula P
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- Messa Aguilar WL
- Metwally MM
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- Mijuskovic B
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- Mustafa M-A
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- Nair H
- Napolitano L
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- Naranjo Aristizabal FA
- Narvestad JK
- Nashaat A
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- Neophytou C
- Nepogodiev D
- Nesaratnam A
- Newton T
- Ng S
- Ngayu N
- Ngwa TE
- Nighat B
- Nigo S
- Nikoloudis N
- Niragire A
- Niyirera E
- Nnajiuba H
- Nocito A
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- Normile C
- North AS
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- Obute RD
- Odeyemi B
- Oefner D
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- Palkhi EYA
- Palliyil MM
- Palma AF
- Palomino Castillo VD
- Paludi MA
- Panait N
- Panieri E
- Parakh J
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- Parent V
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- Pereira B
- Perez Velasquez CI
- Periasammy D
- Peycelon M
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- Pimenta ML
- Pino W
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- Pitt SK
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- Popoola A
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- Regalado F
- Reinisch KB
- Restrepo J
- Reza L
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- Skardziukaite A
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- Soeselo DA
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- Ubhi HK
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- Urbanavicius L
- Urbina Rojas NM
- Urbonas T
- Urniezius Z
- Usaityte A
- Utter M
- Valente Laufer LA
- Van Duren B
- Varcada M
- Varkalys K
- Vartic M
- Vella-Baldacchino M
- Venskutonis D
- Venugopal C
- Vergara Celis JM
- Veronese E
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- Vimalachandran D
- Vincent J
- Vivekanantham S
- Vlachogiorgos A
- Wagener M
- Waleed H
- Walsh U
- Ward AE
- Ward S
- Warda M
- Warren O
- Warwick AM
- Watson NFS
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- Williams G
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- Wilson A
- Wilson MSJ
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- Witherspoon P
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- Wold TB
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- Yallop L
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- Yaseen TM
- Yavuz MA
- Yee J
- Yeo LS
- Yeung ENW
- Yong CO
- Youssef D
- Youssef H
- Youssif JH
- Zachariades D-C
- Zakir M
- Zalabia MF
- Zamarin K
- Zani A
- Zanini N
- Zapata Ponze De Leon M
- Zapata Ponze De Leon M
- Zardab A
- Zegarra S
- Zeidan S
- Zelada Alvarez II
- Zerihun AB
- Zhang Z
- Zheng F
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- Zilinskas J
- Zilinskiene R
- Zimmermann EF
- Zuber M
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2018
- Field of study
BACKGROUND: Appendicitis is the most common abdominal surgical emergency worldwide. Differences between high- and low-income settings in the availability of laparoscopic appendectomy, alternative management choices, and outcomes are poorly described. The aim was to identify variation in surgical management and outcomes of appendicitis within low-, middle-, and high-Human Development Index (HDI) countries worldwide. METHODS: This is a multicenter, international prospective cohort study. Consecutive sampling of patients undergoing emergency appendectomy over 6Â months was conducted. Follow-up lasted 30Â days. RESULTS: 4546 patients from 52 countries underwent appendectomy (2499 high-, 1540 middle-, and 507 low-HDI groups). Surgical site infection (SSI) rates were higher in low-HDI (OR 2.57, 95% CI 1.33-4.99, pâ=â0.005) but not middle-HDI countries (OR 1.38, 95% CI 0.76-2.52, pâ=â0.291), compared with high-HDI countries after adjustment. A laparoscopic approach was common in high-HDI countries (1693/2499, 67.7%), but infrequent in low-HDI (41/507, 8.1%) and middle-HDI (132/1540, 8.6%) groups. After accounting for case-mix, laparoscopy was still associated with fewer overall complications (OR 0.55, 95% CI 0.42-0.71, pâ<â0.001) and SSIs (OR 0.22, 95% CI 0.14-0.33, pâ<â0.001). In propensity-score matched groups within low-/middle-HDI countries, laparoscopy was still associated with fewer overall complications (OR 0.23 95% CI 0.11-0.44) and SSI (OR 0.21 95% CI 0.09-0.45). CONCLUSION: A laparoscopic approach is associated with better outcomes and availability appears to differ by country HDI. Despite the profound clinical, operational, and financial barriers to its widespread introduction, laparoscopy could significantly improve outcomes for patients in low-resource environments. TRIAL REGISTRATION: NCT02179112
Muon reconstruction and identification efficiency in ATLAS using the full Run 2 pp collision data set at \sqrt{s}=13 TeV
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- Zhemchugov A
- Zheng Z
- Zhong D
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- Zorbas TG
- Zou R
- Zwalinski L
- Publication venue
- 'Springer Fachmedien Wiesbaden GmbH'
- Publication date
- 01/01/2021
- Field of study
This article documents the muon reconstruction and identification efficiency obtained by the ATLAS experiment for 139 \hbox {fb}^{-1} of pp collision data at \sqrt{s}=13 TeV collected between 2015 and 2018 during Run 2 of the LHC. The increased instantaneous luminosity delivered by the LHC over this period required a reoptimisation of the criteria for the identification of prompt muons. Improved and newly developed algorithms were deployed to preserve high muon identification efficiency with a low misidentification rate and good momentum resolution. The availability of large samples of Z\rightarrow \mu \mu and J/\psi \rightarrow \mu \mu decays, and the minimisation of systematic uncertainties, allows the efficiencies of criteria for muon identification, primary vertex association, and isolation to be measured with an accuracy at the per-mille level in the bulk of the phase space, and up to the percent level in complex kinematic configurations. Excellent performance is achieved over a range of transverse momenta from 3 GeV to several hundred GeV, and across the full muon detector acceptance of |\eta |<2.7
Configuration and performance of the ATLAS b-jet triggers in Run 2
- Author
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- Zeitnitz C
- Zemaityte G
- Zeng JC
- Zenin O
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- Zerradi S
- Zerwas D
- Zgubic M
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- Zhang DF
- Zhang G
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- Zhang R
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- Zhang X
- Zhang X
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- Zhang Z
- Zhao P
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zheng Z
- Zhong D
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- Zhou M
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- Zhu CG
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- Zhuang X
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- Zhulanov V
- Zieminska D
- Zimine NI
- Zimmermann S
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- Zivkovic L
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- Zorbas TG
- Zou R
- Zou W
- Zwalinski L
- Publication venue
- 'Springer Fachmedien Wiesbaden GmbH'
- Publication date
- 01/12/2021
- Field of study
Several improvements to the ATLAS triggers used to identify jets containing b-hadrons (b-jets) were implemented for data-taking during Run 2 of the Large Hadron Collider from 2016 to 2018. These changes include reconfiguring the b-jet trigger software to improve primary-vertex finding and allow more stable running in conditions with high pile-up, and the implementation of the functionality needed to run sophisticated taggers used by the offline reconstruction in an online environment. These improvements yielded an order of magnitude better light-flavour jet rejection for the same b-jet identification efficiency compared to the performance in Run 1 (2011â2012). The efficiency to identify b-jets in the trigger, and the conditional efficiency for b-jets that satisfy offline b-tagging requirements to pass the trigger are also measured. Correction factors are derived to calibrate the b-tagging efficiency in simulation to match that observed in data. The associated systematic uncertainties are substantially smaller than in previous measurements. In addition, b-jet triggers were operated for the first time during heavy-ion data-taking, using dedicated triggers that were developed to identify semileptonic b-hadron decays by selecting events with geometrically overlapping muons and jets
The ATLAS inner detector trigger performance in pp collisions at 13 TeV during LHC Run 2
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abud AA
- Abulaiti Y
- Acharya BS
- Achkar B
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
- Adiguzel A
- Adorni S
- Adye T
- Aenis T
- Affolder AA
- Afik Y
- Agapopoulou C
- Agaras MN
- Aggarwal A
- Agheorghiesei C
- Aguilar-Saavedra JA
- Agullo PM
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahnen JV
- Ai X
- Aielli G
- Aivkovic L
- Akatsuka S
- Akbiyik M
- Akesson TPA
- Akilli E
- Akimov AV
- Alberghi GL
- Albert J
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Alhroob M
- Ali B
- Ali S
- Aliev M
- Alimonti G
- Allaire C
- Allbrooke BMM
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alves FLL
- Alviggi MG
- Ambler A
- Ambroz L
- Amelung C
- Amidei D
- Amoroso S
- Amrouche CS
- Anastopoulos C
- Andari N
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
- Andrei V
- Anelli CR
- Angelidakis S
- Angerami A
- Anisenkov AV
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Aparo MA
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- Araya ST
- Arcangeletti C
- Arce ATH
- Arguin JF
- Argyropoulos S
- Arling JH
- Armbruster AJ
- Armstrong A
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- Chevalerias TJA
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- Publication venue
- Publication date
- 01/01/2022
- Field of study
The design and performance of the inner detector trigger for the high level trigger of the ATLAS experiment at the Large Hadron Collider during the 2016-2018 data taking period is discussed. In 2016, 2017, and 2018 the ATLAS detector recorded 35.6 fb(-1), 46.9 fb(-1), and 60.6 fb(-1) respectively of proton-proton collision data at a centre-of-mass energy of 13TeV. In order to deal with the very high interaction multiplicities per bunch crossing expected with the 13TeV collisions the inner detector trigger was redesigned during the long shutdown of the Large Hadron Collider from 2013 until 2015. An overview of these developments is provided and the performance of the tracking in the trigger for the muon, electron, tau and b-jet signatures is discussed. The high performance of the inner detector trigger with these extreme interaction multiplicities demonstrates how the inner detector tracking continues to lie at the heart of the trigger performance and is essential in enabling the ATLAS physics programme
Two-particle azimuthal correlations in photonuclear ultraperipheral Pb plus Pb collisions at 5.02 TeV with ATLAS
- Author
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- Publication venue
- AMER PHYSICAL SOC
- Publication date
- 01/07/2021
- Field of study
Two-particle long-range azimuthal correlations are measured in photonuclear collisions using
1.7
nb
â
1
of 5.02 TeV
Pb
+
Pb
collision data collected by the ATLAS experiment at the CERN Large Hadron Collider. Candidate events are selected using a dedicated high-multiplicity photonuclear event trigger, a combination of information from the zero-degree calorimeters and forward calorimeters, and from pseudorapidity gaps constructed using calorimeter energy clusters and charged-particle tracks. Distributions of event properties are compared between data and Monte Carlo simulations of photonuclear processes. Two-particle correlation functions are formed using charged-particle tracks in the selected events, and a template-fitting method is employed to subtract the nonflow contribution to the correlation. Significant nonzero values of the second- and third-order flow coefficients are observed and presented as a function of charged-particle multiplicity and transverse momentum. The results are compared with flow coefficients obtained in proton-proton and proton-lead collisions in similar multiplicity ranges, and with theoretical expectations. The unique initial conditions present in this measurement provide a new way to probe the origin of the collective signatures previously observed only in hadronic collision
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