Search for the Rare Decay KL --> pi0 ee

The KTeV/E799 experiment at Fermilab has searched for the rare kaon decay KL--> pi0ee. This mode is expected to have a significant CP violating component. The measurement of its branching ratio could support the Standard Model or could indicate the existence of new physics. This letter reports new results from the 1999-2000 data set. One event is observed with an expected background at 0.99 +/- 0.35 events. We set a limit on the branching ratio of 3.5 x 10^(-10) at the 90% confidence level. Combining the results with the dataset taken in 1997 yields the final KTeV result: BR(KL --> pi0 ee) < 2.8 x 10^(-10) at 90% CL.Comment: 4 pages, three figure

Bounded and compact multipliers between Bergman and Hardy spaces

This paper studies the boundedness and compactness of the coefficient multiplier operators between various Bergman spaces A p and Hardy spaces H q . Some new characterizations of the multipliers between the spaces with exponents 1 or 2 are derived which, in particular, imply a Bergman space analogue of the Paley-Rudin Theorem on sparse sequences. Hardy and Bergman spaces are shown to be linked using mixed-norm spaces, and this linkage is used to improve a known result on ( A p , A 2 ), 1< p <2.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/42949/1/20_2005_Article_BF01225524.pd

Management of KPC-Producing Klebsiella pneumoniae Infections

Klebsiella pneumoniae carbapenemase (KPC)-producing K. pneumoniae (KPC-KP) has become one of the most important contemporary pathogens, especially in endemic areas

Measurements Of The Decay Kl → E+e-μ+μ-

Several 132 KL → e+e- μ+ μ- events were observed from the 1997 and 1999 runs of the KTeV experiments, with an estimated background of 0.8 events. In the first measurement of the parameter α using this decay mode, it was found that α=-1.59±0.37. No evidence was found for CP-violating contributions to the KLγ*γ* interaction.9014141801/1141801/5Wolfenstein, L., (1983) Phys. Rev. Lett., 51, p. 1945Belanger, G., Geng, C.Q., (1991) Phys. Rev. D, 43, p. 140Buras, A.J., Fleischer, R., (1998) Advanced Ser. Direct. High Energy Phys., 15, p. 65Uy, Z.E.S., (1991) Phys. Rev. D, 43, p. 802D'Ambrosio, G., Isidori, G., Portolès, J., (1998) Phys. Lett. B, 423, p. 385Alavi-Harati, A., (2001) Phys. Rev. Lett., 87, p. 71801. , KTeV CollaborationAlavi-Harati, A., (2001) Phys. Rev. Lett., 86, p. 5425. , KTeV CollaborationUy, Z.E.S., (2002) Eur. Phys. J. C, 23, p. 113Alavi-Harati, A., (2001) Phys. Rev. Lett., 87, p. 111802. , KTeV CollaborationHamm, J.C., (2002), Ph.D. thesis, The University of Arizona(Fermilab Report No. fERMILAB-THESIS-2002-09)Alavi-Harati, A., (1999) Phys. Rev. Lett., 83, p. 922. , KTeV CollaborationAlavi-Harati, A., (2000) Phys. Rev. D, 61, p. 072006. , KTeV CollaborationBrown, C., (1996) Nucl. Instrum. Methods Phys. Res., Sect. A, 369, p. 248Quinn, G.B., (2000), Ph.D. thesis, The University of ChicagoBarker, A.R., Huang, H., Toale, P.A., Engle, J., hep-ph/0210174Bergström, L., Massó, E., Singer, P., (1983) Phys. Lett., 131 B, p. 229Fanti, V., (1999) Phys. Lett. B, 458, p. 553. , NA48 Collaboratio

Meta-analysis of type 2 Diabetes in African Americans Consortium

Type 2 diabetes (T2D) is more prevalent in African Americans than in Europeans. However, little is known about the genetic risk in African Americans despite the recent identification of more than 70 T2D loci primarily by genome-wide association studies (GWAS) in individuals of European ancestry. In order to investigate the genetic architecture of T2D in African Americans, the MEta-analysis of type 2 DIabetes in African Americans (MEDIA) Consortium examined 17 GWAS on T2D comprising 8,284 cases and 15,543 controls in African Americans in stage 1 analysis. Single nucleotide polymorphisms (SNPs) association analysis was conducted in each study under the additive model after adjustment for age, sex, study site, and principal components. Meta-analysis of approximately 2.6 million genotyped and imputed SNPs in all studies was conducted using an inverse variance-weighted fixed effect model. Replications were performed to follow up 21 loci in up to 6,061 cases and 5,483 controls in African Americans, and 8,130 cases and 38,987 controls of European ancestry. We identified three known loci (TCF7L2, HMGA2 and KCNQ1) and two novel loci (HLA-B and INS-IGF2) at genome-wide significance (4.15 × 10(-94)<P<5 × 10(-8), odds ratio (OR)  = 1.09 to 1.36). Fine-mapping revealed that 88 of 158 previously identified T2D or glucose homeostasis loci demonstrated nominal to highly significant association (2.2 × 10(-23) < locus-wide P<0.05). These novel and previously identified loci yielded a sibling relative risk of 1.19, explaining 17.5% of the phenotypic variance of T2D on the liability scale in African Americans. Overall, this study identified two novel susceptibility loci for T2D in African Americans. A substantial number of previously reported loci are transferable to African Americans after accounting for linkage disequilibrium, enabling fine mapping of causal variants in trans-ethnic meta-analysis studies.Peer reviewe