35 research outputs found
Baseline characteristics of patients in the reduction of events with darbepoetin alfa in heart failure trial (RED-HF)
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- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- 01/01/2013
- Field of study
<p>Aims: This report describes the baseline characteristics of patients in the Reduction of Events with Darbepoetin alfa in Heart Failure trial (RED-HF) which is testing the hypothesis that anaemia correction with darbepoetin alfa will reduce the composite endpoint of death from any cause or hospital admission for worsening heart failure, and improve other outcomes.</p>
<p>Methods and results: Key demographic, clinical, and laboratory findings, along with baseline treatment, are reported and compared with those of patients in other recent clinical trials in heart failure. Compared with other recent trials, RED-HF enrolled more elderly [mean age 70 (SD 11.4) years], female (41%), and black (9%) patients. RED-HF patients more often had diabetes (46%) and renal impairment (72% had an estimated glomerular filtration rate <60 mL/min/1.73 m2). Patients in RED-HF had heart failure of longer duration [5.3 (5.4) years], worse NYHA class (35% II, 63% III, and 2% IV), and more signs of congestion. Mean EF was 30% (6.8%). RED-HF patients were well treated at randomization, and pharmacological therapy at baseline was broadly similar to that of other recent trials, taking account of study-specific inclusion/exclusion criteria. Median (interquartile range) haemoglobin at baseline was 112 (106–117) g/L.</p>
<p>Conclusion: The anaemic patients enrolled in RED-HF were older, moderately to markedly symptomatic, and had extensive co-morbidity.</p>
Effects of alirocumab on types of myocardial infarction: insights from the ODYSSEY OUTCOMES trial
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- Zuniga JDH
- Zurakowski A
- Zuran I
- Zweiker R
- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- 01/01/2019
- Field of study
Aims The third Universal Definition of Myocardial Infarction (MI) Task Force classified MIs into five types: Type 1, spontaneous; Type 2, related to oxygen supply/demand imbalance; Type 3, fatal without ascertainment of cardiac biomarkers; Type 4, related to percutaneous coronary intervention; and Type 5, related to coronary artery bypass surgery. Low-density lipoprotein cholesterol (LDL-C) reduction with statins and proprotein convertase subtilisin–kexin Type 9 (PCSK9) inhibitors reduces risk of MI, but less is known about effects on types of MI. ODYSSEY OUTCOMES compared the PCSK9 inhibitor alirocumab with placebo in 18 924 patients with recent acute coronary syndrome (ACS) and elevated LDL-C (≥1.8 mmol/L) despite intensive statin therapy. In a pre-specified analysis, we assessed the effects of alirocumab on types of MI. Methods and results Median follow-up was 2.8 years. Myocardial infarction types were prospectively adjudicated and classified. Of 1860 total MIs, 1223 (65.8%) were adjudicated as Type 1, 386 (20.8%) as Type 2, and 244 (13.1%) as Type 4. Few events were Type 3 (n = 2) or Type 5 (n = 5). Alirocumab reduced first MIs [hazard ratio (HR) 0.85, 95% confidence interval (CI) 0.77–0.95; P = 0.003], with reductions in both Type 1 (HR 0.87, 95% CI 0.77–0.99; P = 0.032) and Type 2 (0.77, 0.61–0.97; P = 0.025), but not Type 4 MI. Conclusion After ACS, alirocumab added to intensive statin therapy favourably impacted on Type 1 and 2 MIs. The data indicate for the first time that a lipid-lowering therapy can attenuate the risk of Type 2 MI. Low-density lipoprotein cholesterol reduction below levels achievable with statins is an effective preventive strategy for both MI types.For complete list of authors see http://dx.doi.org/10.1093/eurheartj/ehz299</p
Evacetrapib and Cardiovascular Outcomes in High-Risk Vascular Disease
- Author
- A Gorog D
- Abadier R
- Acheatel R
- Al-Joundi B
- Albert M
- Albirini A
- Albisu Di Gennaro J
- Alcocer Gamba M
- Alexandersen P
- Alexeeva N
- Almassi H
- Altunkeser B
- Amerena J
- Amin J
- Ando K
- Apostolova E
- Appel Kf
- Appel M
- Ardissino D
- Arif I
- Armstrong P
- Asbill B
- Aslam A
- Aviles R
- Azocar J
- Bacon J
- Baden M
- Bakbak A
- Bakhai A
- Ballantyne C
- Bang L
- Barbarash O
- Barr C
- Barter Philip J.
- Basson M
- Batchelor W
- Beaudry Y
- Benatar J
- Benton R
- Bernhardi D
- Berrouschot J
- Betteridge J
- Bhagwat R
- Bhargava A
- Bhargava R
- Bhatia P
- Binder R
- Bitzur R
- Blain L
- Blombery P
- Boldueva S
- Borghi C
- Bourhaial H
- Brautigam D
- Brewer H. Bryan
- Brodmann M
- Broncel M
- Bronzwaer P
- Brown K
- Bruckert E
- Brunskill J
- Brønnum-Schou J
- Bucci M
- Bugan W
- Butman S
- Caccavo A
- Cahill T
- Canto J
- Carmichael P
- Castano L
- Cecil J
- Chan K
- Chandler G
- Chandrika Parameswaran A
- Chang K
- Chehayeb R
- Chen J
- Cheng S
- Chenier M
- Chiang Ce
- Chizhov P
- Cho B
- Chorin E
- Cinteza M
- Claudio J
- Cohen K
- Cohn J
- Coleman C
- Collis W
- Colombo H
- Colombo T
- Colquhoun D
- Comerota A
- Conde Diego
- Constantinescu M
- Cooke D
- Copaci I
- Coste P
- Cottin Y
- Coulson W
- Crater T
- Cremer P
- Crenshaw B
- Croaning J
- Crowley K
- Dalby Aj
- Davalos J
- de Groot M
- De los Rios Ibarra M
- De Luca G
- De Melker E
- De Wolf L
- Deen C
- Degregorio M
- Dehart D
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- Zrazhevsky K
- Zólyomi S
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2017
- Field of study
BACKGROUND:
The cholesteryl ester transfer protein inhibitor evacetrapib substantially raises the high-density lipoprotein (HDL) cholesterol level, reduces the low-density lipoprotein (LDL) cholesterol level, and enhances cellular cholesterol efflux capacity. We sought to determine the effect of evacetrapib on major adverse cardiovascular outcomes in patients with high-risk vascular disease.
METHODS:
In a multicenter, randomized, double-blind, placebo-controlled phase 3 trial, we enrolled 12,092 patients who had at least one of the following conditions: an acute coronary syndrome within the previous 30 to 365 days, cerebrovascular atherosclerotic disease, peripheral vascular arterial disease, or diabetes mellitus with coronary artery disease. Patients were randomly assigned to receive either evacetrapib at a dose of 130 mg or matching placebo, administered daily, in addition to standard medical therapy. The primary efficacy end point was the first occurrence of any component of the composite of death from cardiovascular causes, myocardial infarction, stroke, coronary revascularization, or hospitalization for unstable angina.
RESULTS:
At 3 months, a 31.1% decrease in the mean LDL cholesterol level was observed with evacetrapib versus a 6.0% increase with placebo, and a 133.2% increase in the mean HDL cholesterol level was seen with evacetrapib versus a 1.6% increase with placebo. After 1363 of the planned 1670 primary end-point events had occurred, the data and safety monitoring board recommended that the trial be terminated early because of a lack of efficacy. After a median of 26 months of evacetrapib or placebo, a primary end-point event occurred in 12.9% of the patients in the evacetrapib group and in 12.8% of those in the placebo group (hazard ratio, 1.01; 95% confidence interval, 0.91 to 1.11; P=0.91).
CONCLUSIONS:
Although the cholesteryl ester transfer protein inhibitor evacetrapib had favorable effects on established lipid biomarkers, treatment with evacetrapib did not result in a lower rate of cardiovascular events than placebo among patients with high-risk vascular disease. (Funded by Eli Lilly; ACCELERATE ClinicalTrials.gov number, NCT01687998 .)
Effect of alirocumab on mortality after acute coronary syndromes. An analysis of the ODYSSEY OUTCOMES randomized clinical trial
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- Welka S
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- Westbrook M
- Westerhausen D
- Whitaker J
- White HD
- White L
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- Wiersma JJ
- Wilkins G
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- Willems FF
- Wilson MD
- Winkelmann B
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- Yao Z
- Yazdani S
- Yerra SK
- Yin W-H
- Yong H
- Yoon JH
- Younis L
- Yu Y
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- Yusoff K
- Zahger D
- Zaidman CJ
- Zainea M
- Zaman A
- Zapata G
- Zdravko B
- Zea Nunez CA
- Zeiher AM
- Zeiher AM
- Zhang R
- Zhang W
- Zhang X
- Zhang Z
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- Zheleznyy V
- Zheng Y
- Zheng Z
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- Zhu H
- Zhu J
- Zirlik A
- Zizka J
- Zobouyan I
- Zong W
- Zrazhevsky K
- Zurakowski A
- Zuran I
- Zweiker R
- Publication venue
- 'Ovid Technologies (Wolters Kluwer Health)'
- Publication date
- 04/01/2019
- Field of study
Background: Previous trials of PCSK9 (proprotein convertase subtilisin-kexin type 9) inhibitors demonstrated reductions in major adverse cardiovascular events, but not death. We assessed the effects of alirocumab on death after index acute coronary syndrome. Methods: ODYSSEY OUTCOMES (Evaluation of Cardiovascular Outcomes After an Acute Coronary Syndrome During Treatment With Alirocumab) was a double-blind, randomized comparison of alirocumab or placebo in 18 924 patients who had an ACS 1 to 12 months previously and elevated atherogenic lipoproteins despite intensive statin therapy. Alirocumab dose was blindly titrated to target achieved low-density lipoprotein cholesterol (LDL-C) between 25 and 50 mg/dL. We examined the effects of treatment on all-cause death and its components, cardiovascular and noncardiovascular death, with log-rank testing. Joint semiparametric models tested associations between nonfatal cardiovascular events and cardiovascular or noncardiovascular death. Results: Median follow-up was 2.8 years. Death occurred in 334 (3.5%) and 392 (4.1%) patients, respectively, in the alirocumab and placebo groups (hazard ratio [HR], 0.85; 95% CI, 0.73 to 0.98; P=0.03, nominal P value). This resulted from nonsignificantly fewer cardiovascular (240 [2.5%] vs 271 [2.9%]; HR, 0.88; 95% CI, 0.74 to 1.05; P=0.15) and noncardiovascular (94 [1.0%] vs 121 [1.3%]; HR, 0.77; 95% CI, 0.59 to 1.01; P=0.06) deaths with alirocumab. In a prespecified analysis of 8242 patients eligible for ≥3 years follow-up, alirocumab reduced death (HR, 0.78; 95% CI, 0.65 to 0.94; P=0.01). Patients with nonfatal cardiovascular events were at increased risk for cardiovascular and noncardiovascular deaths (P<0.0001 for the associations). Alirocumab reduced total nonfatal cardiovascular events (P<0.001) and thereby may have attenuated the number of cardiovascular and noncardiovascular deaths. A post hoc analysis found that, compared to patients with lower LDL-C, patients with baseline LDL-C ≥100 mg/dL (2.59 mmol/L) had a greater absolute risk of death and a larger mortality benefit from alirocumab (HR, 0.71; 95% CI, 0.56 to 0.90; Pinteraction=0.007). In the alirocumab group, all-cause death declined wit h achieved LDL-C at 4 months of treatment, to a level of approximately 30 mg/dL (adjusted P=0.017 for linear trend). Conclusions: Alirocumab added to intensive statin therapy has the potential to reduce death after acute coronary syndrome, particularly if treatment is maintained for ≥3 years, if baseline LDL-C is ≥100 mg/dL, or if achieved LDL-C is low. Clinical Trial Registration: URL: https://www.clinicaltrials.gov. Unique identifier: NCT01663402
Pharmacological cardioversion of atrial fibrillation--a double-blind, randomized, placebo-controlled, multicentre, dose-escalation study of AZD1305 given intravenously
- Author
- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- Field of study
Early eplerenone treatment in patients with acute ST-elevation myocardial infarction without heart failure: The Randomized Double-Blind Reminder Study
- Author
- A. Chauhan
- A. F. Ortiz
- A. Grieve
- B. O'Neill
- B. Pitt
- B. Sussex
- Beygui
- Beygui
- C. Dezsi
- C. G. Garcia
- C. P. Munoz
- C. Tschope
- C. W. Hamm
- D. Adamson
- D. Alexopoulos
- D. Babalis
- D. Ducos
- D. Fitchett
- D. Newby
- D. Smith
- de Lemos
- E. Lopez de Sa
- E. Teiger
- E. Turgonyi
- F. Alamgir
- F. Gerasimos
- F. Kovar
- F. R. Soriano
- F. Verheugt
- F. Zannad
- G. Hauf
- G. Lip
- G. Lupkovics
- G. McCann
- G. Montalescot
- G. Noll
- G. Tremblay
- G. Wong
- G. Zemour
- Goyal
- H. Heuer
- H. Mudra
- H. Shi
- J. Bauersachs
- J. Gorny
- J. Herrman
- J. Kettner
- J. Kubica
- J. Lewczuk
- J. M. ten Berg
- J. R. Rey Blas
- J. Spinar
- J. Sumbal
- J. Tomcsanyi
- J. Vincent
- J. Vojacek
- J.-L. Rouleau
- J.-P. Collet
- K. Micko
- K. Sydow
- Lopez-de-Sa
- M. Barboteu
- M. Bohm
- M. F. Sala
- M. Flather
- M. Galinier
- M. Hranai
- M. Hutyra
- M. Madan
- M. Orri
- Montalescot
- N. Werner
- P. Coste
- P. Ostadal
- P. Polgar
- R. Hambrecht
- R. Margoczy
- R. Strasser
- R. Wachter
- R. Weir
- R. Welsh
- S. Lepage
- S. Steiner
- Sabate
- Struthers
- T. Martin
- T. Munzel
- V. Pyrgakis
- W. S. Hillis
- W. Zmuda
- Wei
- X. B. Genover
- Y. Cottin
- Zannad
- Publication venue
- 'Oxford University Press (OUP)'
- Publication date
- 30/04/2014
- Field of study
Aims: We aimed to assess the impact of eplerenone on cardiovascular (CV) outcomes in STEMI without known heart failure, when initiated within 24 h of symptom onset. Methods and results: In this randomized, placebo-controlled, double-blind trial, we assigned 1012 patients with acute STEMI and without a history of heart failure to receive either eplerenone (25–50 mg once daily) or placebo in addition to standard therapy. The primary endpoint was the composite of CV mortality, re-hospitalization, or, extended initial hospital stay, due to diagnosis of HF, sustained ventricular tachycardia or fibrillation, ejection fraction ≤40%, or elevated BNP/NT-proBNP at 1 month or more after randomization. BNP elevation was defined as BNP levels or values above 200 pg/mL or NT-proBNP values above 450 pg/mL (in patients aged below 50); above 900 pg/mL (age 50–75 years) or above 1800 pg/mL (patients older than 75). After a mean follow-up of 10.5 months, the primary endpoint occurred in 92 patients (18.2%) in the eplerenone group and in 149 patients (29.4%) in the placebo group [adjusted hazard ratio (HR), 0.58; 95% confidence interval (CI), 0.45–0.76; P < 0.0001]. The primary endpoint was driven by a high BNP/NT-proBNP level (adjusted HR, 0.60; 95% CI, 0.45–0.79; P < 0.0003). Adverse event rates were similar in both groups. Serum potassium levels exceeded 5.5 mmol/L in 5.6 vs. 3.2% (P = 0.09) and were below 3.5 mmol/L in 1.4 vs. 5.6% of patients (P = 0.0002), in the eplerenone and placebo groups, respectively. Conclusion: The addition of eplerenone during the acute phase of STEMI was safe and well tolerated. It reduced the primary endpoint over a mean 13 months follow-up mostly because of significantly lower BNP/NT-proBNP levels. Additional studies are needed to clarify the role of early use of MRAs in STEMI patients without heart failure
Rivaroxaban with or without aspirin in stable cardiovascular disease
- Author
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- Abatello M.
- Abdul Hafidz M. I.
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- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2017
- Field of study
BACKGROUND: We evaluated whether rivaroxaban alone or in combination with aspirin would be more effective than aspirin alone for secondary cardiovascular prevention. METHODS: In this double-blind trial, we randomly assigned 27,395 participants with stable atherosclerotic vascular disease to receive rivaroxaban (2.5 mg twice daily) plus aspirin (100 mg once daily), rivaroxaban (5 mg twice daily), or aspirin (100 mg once daily). The primary outcome was a composite of cardiovascular death, stroke, or myocardial infarction. The study was stopped for superiority of the rivaroxaban-plus-aspirin group after a mean follow-up of 23 months. RESULTS: The primary outcome occurred in fewer patients in the rivaroxaban-plus-aspirin group than in the aspirin-alone group (379 patients [4.1%] vs. 496 patients [5.4%]; hazard ratio, 0.76; 95% confidence interval [CI], 0.66 to 0.86; P<0.001; z=−4.126), but major bleeding events occurred in more patients in the rivaroxaban-plus-aspirin group (288 patients [3.1%] vs. 170 patients [1.9%]; hazard ratio, 1.70; 95% CI, 1.40 to 2.05; P<0.001). There was no significant difference in intracranial or fatal bleeding between these two groups. There were 313 deaths (3.4%) in the rivaroxaban-plus-aspirin group as compared with 378 (4.1%) in the aspirin-alone group (hazard ratio, 0.82; 95% CI, 0.71 to 0.96; P=0.01; threshold P value for significance, 0.0025). The primary outcome did not occur in significantly fewer patients in the rivaroxaban-alone group than in the aspirin-alone group, but major bleeding events occurred in more patients in the rivaroxaban-alone group. CONCLUSIONS: Among patients with stable atherosclerotic vascular disease, those assigned to rivaroxaban (2.5 mg twice daily) plus aspirin had better cardiovascular outcomes and more major bleeding events than those assigned to aspirin alone. Rivaroxaban (5 mg twice daily) alone did not result in better cardiovascular outcomes than aspirin alone and resulted in more major bleeding events
Cardiovascular Efficacy and Safety of Bococizumab in High-Risk Patients
- Author
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- Abdullah S.
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- Willits V. L.
- Winkelmann B.
- Witt N.
- Wittmann I.
- Woehrle J.
- Worner Diz F.
- Wozniak I.
- Wright R. S.
- Wronska D.
- Wuerzner G.
- Wychota K.
- Xu P.
- Xue L.
- Yakusevich V. V.
- Yang P.
- Yang X.
- Yao L. C.
- Yared Z.
- Yarows S. A.
- Yigit Z.
- Yoon J.
- Yunis C.
- Zadra R.
- Zaidman C. J.
- Zamorano Gomez J. L.
- Zannad Faiez
- Zarandon R. B. S.
- Zarich S. W.
- Zateyshchikov D.
- Zavaro S. H.
- Zebrack J. S.
- Zeig S.
- Zemek S.
- Zemour G.
- Zeng X.
- Zeno M. L.
- Zhang L.
- Zhang X.
- Zhang Y.
- Zhao Y.
- Zhou C.
- Zhou Y.
- Zhu Y.
- Zidkova E.
- Zieba B.
- Zilahi Z.
- Zoet-Nugteren S. K.
- Zrazhevskiy K.
- Zubek V.
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2017
- Field of study
BACKGROUN
Edoxaban versus warfarin in patients with atrial fibrillation
- Author
- Abdullakutty J
- Abi-Mansour P
- Abril SB
- Accini J
- Accini M
- Acikel M
- Acikel M
- Adachi C
- Adams K
- Adams K
- Adamus J
- Adler J
- Adler P
- Agarwal D
- Aggarwal R
- Aggarwal R
- Agirov M
- Agraou B
- Ahmad A
- Ahmadpour H
- Ahuad Guerrero R
- Ajioka M
- Akhter F
- Akihisa U
- Akilli H
- Al-Maliky T
- Al-Zoebi A
- Albert L. Waldo
- Albisu J
- Albulescu P
- Alexander J
- Alexandrova L
- Alexopoulos D
- Alexopoulos D
- Alhakim M
- Aliyar P
- Allall O
- Allison J
- Allman J
- Almaraz SP
- Almeida A
- Almeida M
- Almquist A
- Aloni I
- Alsheikh T
- Altonen D
- Alvarez C
- Alvarez M
- Alvarez RF
- Amarenco P
- Amato Vincenzo de Paola A
- Ambrovic I
- Ambrovicova V
- Ameriso P
- Ameriso S
- Amin K
- Amin M
- Amuchastegui M
- Anciu M
- Anderson C
- Anderson J
- Anderson J
- Andersson R
- Andor M
- Andrade Lotufo P
- Andresen T
- Andrews A
- Angel J
- Anscombe R
- Anthony A
- Antle S
- Antman EM
- Antman EM
- Antonino M
- Anzai T
- Apetrei E
- Apostolovic S
- Appelros P
- Aquino M
- Arakawa S
- Araújo E
- Archibald J
- Arcocha Torres M
- Ardelean A
- Arfaoui M
- Arias J
- Armas BH
- Arneja J
- Arnold T
- Arora T
- Arora V
- Arouni A
- Arrieta M
- Arroyave C
- Arstall M
- Arutyunov G
- Asakura H
- Asensio A
- Astier L
- Ata N
- Ata N
- Atar D
- Atar D
- Atar S
- Atie J
- Atkinson C
- Attanti S
- Aude Y
- Augusto Alves da Costa F
- Aull L
- Ault S
- Ausperger KM
- Avellar K
- Averett P
- Avery D
- Avila H
- Avvaru G
- Awasty V
- Awtry E
- Ayasse D
- Ayau Milla O
- Ayesu K
- Aylward P
- Azhdari M
- Azua MG
- Babbar A
- Babbolin M
- Babilonia N
- Babilonia N
- Babu P
- Babu R
- Babuty D
- Badenhorst J
- Badila E
- Bae HJ
- Bagatin J
- Bagby J
- Bai F
- Baine S
- Bakliza Z
- Balboa W
- Baldwin E
- Ball E
- Ballyuzek M
- Baman R
- Bamhorst M
- Bamrungpong P
- Banaeian F
- Banikova A
- Banikova A
- Banker D
- Bansal N
- Banville P
- Bar M
- Barai A
- Baranyai M
- Barash B
- Barbarash O
- Barber M
- Barbera S
- Barcinas R
- Barker T
- Barnhorst M
- Barquero R
- Barrington P
- Barros R
- Barroso D
- Bart B
- Bartholomaus D
- Bartkowiak A
- Bartos D
- Bashkireva A
- Basson M
- Baszak J
- Baszak J
- Batchell K
- Batista M
- Batta C
- Battistelli E
- Batushkin V
- Bauch F
- Bauch F
- Bauer K
- Bautista C
- Bayata S
- Bayata S
- Baylacher M
- Bayram Llamas E
- Bazylevych A
- Beasley R
- Beck P
- Bedenko E
- Bedoya R
- Bedregal SA
- Bedwell N
- Beerts C
- Beeton I
- Behm K
- Beinart S
- Bekal S
- Belatsky V
- Belcuore M
- Belenky D
- Belhassane A
- Beloscar J
- Belousova L
- Ben Ari M
- Benatar J
- Bengtsson AS
- Bengus C
- Benicka E
- Benjarge P
- Bensimhon D
- Bentivenga L
- Benton J
- Berbari H
- Berbari H
- Bereznyakov I
- Berger CJ
- Berger N
- Bergovec M
- Bergovec M
- Berk M
- Berka L
- Berli M
- Berli M
- Bernard J
- Bernhardt P
- Berni A
- Berns S
- Berrouschot J
- Bertolim Precoma D
- Betcher J
- Beyer-Westendorf J
- Bharani A
- Bhargava R
- Bhargava V
- Bhaskarwar P
- Bhuti M
- Bianchini M
- Bilal B
- Bilazarian S
- Bingham S
- Biniek D
- Biniwale A
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- Biscardi R
- Bjelobrk M
- Blagden M
- Blanc J
- Blanc JJ
- Blanco Coronado J
- Blihar O
- Block T
- Blom KB
- Blomström P
- Blonder R
- Bloomberg K
- Bloomberg R
- Blue B
- Bobade M
- Bobbie C
- Bobescu E
- Bocanera M
- Bognar A
- Bojanowska E
- Bokarev I
- Bollova D
- Bolohan F
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- Bolton R
- Bonds C
- Bonin K
- Bonora M
- Boriani G
- Borowski W
- Borsheim M
- Borthwick L
- Bosco B
- Bose S
- Bossaers J
- Botelho R
- Botero R
- Botha L
- Bouchard A
- Bourlios P
- Boursin P
- Bouzo M
- Bowden W
- Bowen L
- Bowman B
- Boyer M
- Bozek B
- Bradley A
- Braemer U
- Braun D
- Braun R
- Braunwald E
- Braunwald E
- Breschi M
- Bridge C
- Brilakis E
- Briscoe C
- Broberg M
- Brodova P
- Brondani R
- Bronisz M
- Brown F
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- Browning J
- Brtko M
- Brtko M
- Brubaker S
- Bruguera Cortada J
- Bruning T
- Bruton T
- Buchholz M
- Budassi N
- Buelow-Johansen B
- Bugueño C
- Buhler A
- Buhr M
- Buiks C
- Buiks C
- Builes A
- Buksa B
- Bulgari M
- Bulygin S
- Burianova H
- Burianova H
- Bush D
- Bustamante G
- Bustamante Labarta G
- Busz-Papiez B
- Butcher S
- Buza V
- Buzzetti C
- Bybee K
- Cabero P
- Cabrera J
- Caccavo A
- Caceres M
- Cader C
- Cajulis C
- Cakmak N
- Calambur N
- Calderon M
- Calik A
- Calvo C
- Calvo G
- Calvo M
- Calvo Vargas C
- Camm J
- Camm J
- Cammell R
- Campbell G
- Campisto Y
- Canaday D
- Cannoni S
- Cano N
- Canosa R
- Capasso-Gulve E
- Capiau L
- Caplan J
- Car S
- Cardona Muñoz E
- Cardoza T
- Carevic V
- Carey G
- Carey J
- Carlsson T
- Carne M
- Carr K
- Carraro A
- Carrasco GT
- Carrillo J
- Carrion A
- Carroll L
- Cartasegna L
- Caruso O
- Casares E
- Casas R
- Castro C
- Caterini T
- Cavanna L
- Cayli M
- Cayli M
- Ceirano C
- Celic V
- Cemerlic Adjic N
- Cendali G
- Cermak O
- Cermak O
- Cervellione K
- Cervi E
- Ceyhan C
- Ceyhan C
- Cha J
- Cha JK
- Cha MJ
- Chacon L
- Champagne M
- Chang M
- Chapman O
- Charlat M
- Chaturvedi S
- Chaves Junior H
- Chavez C
- Chehayeb R
- Chen JZ
- Chen SA
- Chen XP
- Chen YF
- Chen YP
- Chendey T
- Cheng CC
- Cheng CC
- Chernyavskaya T
- Cherrico M
- Chiang CE
- Chiang FT
- Chilvers M
- Chiou CW
- Chiu CZ
- Chmielowski A
- Cho BR
- Choi HH
- Choi KJ
- Choi Y
- Chompalova B
- Chopda M
- Choy A
- Christensen H
- Christensen L
- Christian T. Ruff
- Chumakova G
- Chun MY
- Chung A
- Chung N
- Chung WS
- Ciaramita J
- Cieslak B
- Ciezak P
- Ciglenecki N
- Ciobotaru G
- Ciortea M
- Clapp T
- Claussen H
- Clay A
- Cleveland D
- Clifton R
- Clinckemaille N
- Cobos D
- Cobos J
- Coching R
- Codutti O
- Cohen A
- Cohen A
- Cohen R
- Coisne D
- Colan D
- Cole J
- Colin MA
- Colombo H
- Colquhoun D
- Coman I
- Comi G
- Concha M
- Connolly S
- Constance C
- Constantinescu M
- Constantinescu M
- Contzen C
- Cook S
- Coombs V
- Copley M
- Corbalan R
- Corbalán R
- Corbett B
- Corbut S
- Corchado D
- Correia J
- Correia J
- Cossu S
- Costache L
- Costantini C
- Costi P
- Cottiero R
- Coutinho E
- Coutu B
- Cowan L
- Cox C
- Coyle GS
- Cozma M
- Craft D
- Craig M
- Crerand W
- Creteanu M
- Crimmins D
- Cristaldi J
- Cristea M
- Crisu D
- Croce K
- Cuccia C
- Cuello J
- Cullen T
- Cuneo C
- Cunha S
- Curt V
- Curtis B
- Curtis S
- Cury C
- Cush S
- Cymerman K
- Cymerman K
- Cásares Ramirez M
- D'Amico A
- D'Costa D
- D'Souza A
- Dabrowska M
- Dagan T
- Daitz C
- Dalby A
- Dalby A
- Dandekar U
- Dang N
- Daniels R
- Daniels S
- Dart A
- Daskalaki A
- Dastoli K
- Datta S
- Daut W
- Dave B
- Dave K
- Davicevic Z
- David D
- Davids M
- Davies D
- Davis A
- Davis C
- Davis C
- Davis S
- Davolos M
- Davuluri A
- Davé J
- de Arce Borda A
- De Bleecker J
- De Bruyne L
- De Caterina R
- de Groot J
- de Koning K
- de la Mora S
- De Lemos J
- de Melo RF
- De Pace D
- De Patta M
- De Servi S
- De Tollenaere M
- de Vos R
- de Vos R
- Dean J
- Debes C
- Decoulx E
- Deenadayalu N
- DeGarmo R
- Degtyareva E
- Delgadillo T
- Delgado A
- Delgado MD
- Delgado P
- Delgado Restrepo J
- Delgado S
- Demir H
- Demir H
- Demko A
- Denchev S
- Denie D
- Denis I
- Dennison K
- Deokar M
- Derlaga B
- Deruche E
- Derycker K
- Desai A
- Desai A
- DeSalle D
- Desfontaines P
- Desire A
- Despotovic N
- Dettmann S
- Devenport S
- Dewar C
- Dhar S
- Dharmarao A
- Di Lenarda A
- Di Niro M
- Di Pasquale G
- Dias P
- Diaz IA
- Diaz PA
- Diaz-Pintado M
- Dichristin U
- Dicker D
- Dickinson D
- Dickinson D
- DiCorcia L
- Dieckheuer U
- Diez Tejedor E
- DiGiovanna M
- Dimitra K
- Dinanian S
- Dioszeghy P
- Djokic I
- Djokovic A
- Djordjevic-Radojkovic D
- do Val R
- Dobre I
- Dobreanu D
- Doctor A
- Dodt K
- Doka B
- Domanska E
- Domingo D
- Donahue S
- Dong YG
- Dongas B
- Donova T
- Donovan D
- Dorn S
- Dorobantu M
- Dotani I
- Dotsenko S
- Dougal K
- Doughty R
- Douglas H
- Douglas S
- Dovgalevsky P
- Dovgan N
- Dowling C
- Dragomir D
- Dran R
- Drapchak I
- Drapkina O
- Drescher T
- Drew B
- Dreykluft K
- Dronov D
- Drozdiak R
- du Preez M
- Duda N
- Duff J
- Dufresne M
- Dugal J
- Duggal A
- Dukat A
- Duki Y
- Dulka T
- Dulkova K
- Duncan D
- Dunn K
- Dunn S
- Duprat C
- Duris T
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- Dutka C
- Dutra O
- Dutter S
- Dvoieglazova M
- Dzublik Y
- Dzupina A
- Dzupinova M
- Dzyak G
- Eade J
- Easton JD
- Ebano P
- Ebano P
- Ebert J
- Ebina E
- Ebreo N
- Edelstein J
- Eden M
- Edes I
- Edlund M
- Egorova L
- Egorova L
- Eikelboom J
- El Jarroudi F
- El Jarroudi F
- El Jarroudi M
- Elbl L
- Elbl L
- Eldadah Z
- Eldred R
- Elias M
- Elliott M. Antman
- Ellis G
- Ellis J
- Enad C
- Endo H
- Engdahl J
- Engelbrecht J
- Enger S
- Ensminger E
- Erasmus L
- Erceg P
- Erdogan D
- Erdogan D
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- Erenrich N
- Eriksson G
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- Esip V
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- Eugene Braunwald
- Everhart B
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- Facello A
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- Faghih M
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- Faircloth C
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- Faustino Saporito W
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- Feil JF
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- Jambula H
- James J. Hanyok
- Jameson L
- Janczewska D
- Jandik J
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- Januszewicz A
- Jaramillo A
- Jaramillo Muñoz C
- Jardula M
- Jarkovsky P
- Jarkovsky P
- Jasani B
- Jauregui V
- Jazwinska-Tarnawska E
- Jeffrey I. Weitz
- Jelic V
- Jelinek Z
- Jelinek Z
- Jemna D
- Jeon HK
- Jerabek O
- Jerabek O
- Jeric M
- Jernhed H
- Jiang XJ
- Jimenez T
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- Zateyshchikova A
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- Zharinov O
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- Åkerberg A
- Östberg S
- Ŝpinar J
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2013
- Field of study
Contains fulltext :
125374.pdf (publisher's version ) (Open Access)BACKGROUND: Edoxaban is a direct oral factor Xa inhibitor with proven antithrombotic effects. The long-term efficacy and safety of edoxaban as compared with warfarin in patients with atrial fibrillation is not known. METHODS: We conducted a randomized, double-blind, double-dummy trial comparing two once-daily regimens of edoxaban with warfarin in 21,105 patients with moderate-to-high-risk atrial fibrillation (median follow-up, 2.8 years). The primary efficacy end point was stroke or systemic embolism. Each edoxaban regimen was tested for noninferiority to warfarin during the treatment period. The principal safety end point was major bleeding. RESULTS: The annualized rate of the primary end point during treatment was 1.50% with warfarin (median time in the therapeutic range, 68.4%), as compared with 1.18% with high-dose edoxaban (hazard ratio, 0.79; 97.5% confidence interval [CI], 0.63 to 0.99; P<0.001 for noninferiority) and 1.61% with low-dose edoxaban (hazard ratio, 1.07; 97.5% CI, 0.87 to 1.31; P=0.005 for noninferiority). In the intention-to-treat analysis, there was a trend favoring high-dose edoxaban versus warfarin (hazard ratio, 0.87; 97.5% CI, 0.73 to 1.04; P=0.08) and an unfavorable trend with low-dose edoxaban versus warfarin (hazard ratio, 1.13; 97.5% CI, 0.96 to 1.34; P=0.10). The annualized rate of major bleeding was 3.43% with warfarin versus 2.75% with high-dose edoxaban (hazard ratio, 0.80; 95% CI, 0.71 to 0.91; P<0.001) and 1.61% with low-dose edoxaban (hazard ratio, 0.47; 95% CI, 0.41 to 0.55; P<0.001). The corresponding annualized rates of death from cardiovascular causes were 3.17% versus 2.74% (hazard ratio, 0.86; 95% CI, 0.77 to 0.97; P=0.01), and 2.71% (hazard ratio, 0.85; 95% CI, 0.76 to 0.96; P=0.008), and the corresponding rates of the key secondary end point (a composite of stroke, systemic embolism, or death from cardiovascular causes) were 4.43% versus 3.85% (hazard ratio, 0.87; 95% CI, 0.78 to 0.96; P=0.005), and 4.23% (hazard ratio, 0.95; 95% CI, 0.86 to 1.05; P=0.32). CONCLUSIONS: Both once-daily regimens of edoxaban were noninferior to warfarin with respect to the prevention of stroke or systemic embolism and were associated with significantly lower rates of bleeding and death from cardiovascular causes. (Funded by Daiichi Sankyo Pharma Development; ENGAGE AF-TIMI 48 ClinicalTrials.gov number, NCT00781391.)
Treatment of Anemia with Darbepoetin Alfa in Systolic Heart Failure
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- Rodríguez Lambert J
- Roig Minguell E
- Rokkedal J
- Romero J
- Ronaszeki A
- Ronner E
- Ross A
- Rossert J
- Roth S
- Roul G
- Rudys A
- Russo T
- Rydberg E
- Ryden L
- Rynkiewicz A
- Sabatier R
- Sakalyte G
- Sakalyte G
- Saleh N
- Saltzberg M
- Sanchez A
- Sandri M
- Santos D
- Saraiva J
- Sarkar S
- Sastry B
- Sathe S
- Scarlata D
- Scelsi L
- Schmedtje J
- Schneider R
- Schuyler G
- Schygiel P
- Schäufele T
- Scott D. Solomon
- Seabra Gomes R
- Semernin E
- Senior R
- Senni M
- Sestier F
- Shanes J
- Sharma A
- Shogenov Z
- Shotan A
- Sidorenko B
- Siegel C
- Siegel R
- Silber D
- Simões M
- Sindone A
- Singh J
- Singh N
- Singh V
- Sinha N
- Skagen K
- Sklar J
- Skvortsov A
- Sliaziene S
- Sluzar V
- Small R
- Smirnakis K
- Smith A
- Smith E
- Smith L
- Smull D
- Snyder W
- Sojka G
- Solomon S
- Somsen A
- Sotolongo R
- Soucek M
- Souza Neto J
- Spac J
- Spacek R
- Spisak V
- Squire I
- Srikanthan V
- Stamate S
- Staniloae C
- Stapleton D
- Steele P
- Stehlik J
- Stein M
- Stockins B
- Storzhakov G
- Subburamakrishnan P
- Sulimov V
- Sun Y
- Sunfa Cheng
- Sussex B
- Swedberg K
- Szelemej R
- Talibov O
- Tan L
- Tang W
- Tavazzi L
- Teixeira M
- Tereshenko S
- Thacker H
- Thadani U
- Thalamus J
- Thoeng J
- Thompson P
- Timberg I
- Toblli J
- Tomcsanyi J
- Tomescu M
- Torp-Pedersen C
- Torre-Amoine G
- Trichon B
- Trochu J
- Trotman ML
- Trusz-Gluza M
- Tsai C
- Tse H
- Tsyrline V
- Tummala R
- Tuxen C
- Tzekova M
- Ujda M
- Vahala P
- Vahula V
- Valdovinos P
- Van Bakel A
- van Dantzig J
- van der Burgh P
- van Kempen L
- van Vlies B
- Van Zyl L
- Vanderheyden M
- Vanhaecke J
- Vanhaecke J
- Veze I
- Vicari R
- Videbak L
- Vijay N
- Vijayaraghavan K
- Vinanska D
- Vita I
- Vittorio T
- Vizel S
- Vodnansky P
- Vogel D
- Volans E
- von Hodenberg E
- Voors A
- Vossler M
- Vukasovic J
- Wagoner L
- Waites J
- Wallis D
- Wander G
- Ward N
- Wardeh A
- Wasserman S
- Watkins A
- Weich H
- Weinrauch LA
- Weinstein J
- Westheim A
- Weyland K
- Weytjens C
- Widmer M
- Wight J
- Wikstrom G
- Wilkins C
- Willems F
- Willenheimer R
- Williams C
- Williams G
- Winchester M
- Winkel E
- Winkelmann B
- Witte K
- Wittmer B
- Wojciechowski D
- Wollaert B
- Wong M
- Wood D
- Wormer D
- Wright R
- Wysokinski A
- Xu Z
- Yan Sun
- Yan B
- Yasin M
- Young J
- Zadionchenko V
- Zamolyi K
- Zateyshchikov D
- Zhang Y
- Zimlichman R
- Zolty R.
- Publication venue
- Publication date
- 01/01/2013
- Field of study
<p>BACKGROUND</p><p>Patients with systolic heart failure and anemia have worse symptoms, functional capacity, and outcomes than those without anemia. We evaluated the effects of darbepoetin alfa on clinical outcomes in patients with systolic heart failure and anemia.</p><p>METHODS</p><p>In this randomized, double-blind trial, we assigned 2278 patients with systolic heart failure and mild-to-moderate anemia (hemoglobin level, 9.0 to 12.0 g per deciliter) to receive either darbepoetin alfa (to achieve a hemoglobin target of 13 g per deciliter) or placebo. The primary outcome was a composite of death from any cause or hospitalization for worsening heart failure.</p><p>RESULTS</p><p>The primary outcome occurred in 576 of 1136 patients (50.7%) in the darbepoetin alfa group and 565 of 1142 patients (49.5%) in the placebo group (hazard ratio in the darbepoetin alfa group, 1.01; 95% confidence interval, 0.90 to 1.13; P = 0.87). There was no significant between-group difference in any of the secondary outcomes. The neutral effect of darbepoetin alfa was consistent across all prespecified subgroups. Fatal or nonfatal stroke occurred in 42 patients (3.7%) in the darbepoetin alfa group and 31 patients (2.7%) in the placebo group (P = 0.23). Thromboembolic adverse events were reported in 153 patients (13.5%) in the darbepoetin alfa group and 114 patients (10.0%) in the placebo group (P = 0.01). Cancer-related adverse events were similar in the two study groups.</p><p>CONCLUSIONS</p><p>Treatment with darbepoetin alfa did not improve clinical outcomes in patients with systolic heart failure and mild-to-moderate anemia. Our findings do not support the use of darbepoetin alfa in these patients. (Funded by Amgen; RED-HF ClinicalTrials.gov number, NCT00358215.)</p>