Widespread and accelerating glacier retreat on the Lyngen Peninsula, northern Norway, since their ‘Little Ice Age’ maximum

The recession of mountain glaciers worldwide is increasing global sea level and, in many regions, human activities will have to adapt to changes in surface hydrology. Thus, it is important to provide up-to-date analyses of glacier change and the factors modulating their response to climate warming. Here we report changes in the extent of >120 glaciers on the Lyngen Peninsula, northern Norway, where glacier runoff is utilised for hydropower and where glacial lake outburst floods have occurred. Glaciers covered at least 114 km2 in 1953 and we compare this inventory with those from 1988, 2001 and a new one from 2014, and previously-dated Little Ice Age (LIA) limits. Results show a steady reduction in area (~0.3% a−1) between their LIA maximum (~1915) and 1988, consistent with increasing summer air temperatures, but recession paused between 1988 and 2001, coinciding with increased winter precipitation. Air temperatures increased 0.5°C per decade from the 1990s and the rate of recession accelerated to ~1% a−1 between 2001 and 2014 when glacier area totalled ~95.7 km2. Small glaciers (<0.05 km2) with low maximum elevations (<1400 m) experienced the largest percentage losses and, if warming continues, several glaciers may disappear within the next two decades

Ice thickness measurements and volume estimates for glaciers in Norway

Glacier volume and ice thickness distribution are important variables for water resource management in Norway and the assessment of future glacier changes. We present a detailed assessment of thickness distribution and total glacier volume for mainland Norway based on data and modelling. Glacier outlines from a Landsat-derived inventory from 1999 to 2006 covering an area of 2692 ± 81 km² were used as input. We compiled a rich set of ice thickness observations collected over the past 30 years. Altogether, interpolated ice thickness measurements were available for 870 km² (32%) of the current glacier area of Norway, with a total ice volume of 134 ± 23 km³. Results indicate that mean ice thickness is similar for all larger ice caps, and weakly correlates with their total area. Ice thickness data were used to calibrate a physically based distributed model for estimating the ice thickness of unmeasured glaciers. The results were also used to calibrate volume–area scaling relations. The calibrated total volume estimates for all Norwegian glaciers ranged from 257 to 300 km³

Spatio-temporal variability in geometry and geodetic mass balance of Jostedalsbreen ice cap, Norway

The Jostedalsbreen ice cap is mainland Europe's largest ice cap and accommodates 20% (458 km2 in 2019) of the total glacier area of mainland Norway. Jostedalsbreen and its meltwater contribute to global sea-level rise and to local water management, hydropower and tourism economies and livelihoods. In this study, we construct a digital terrain model (DTM) of the ice cap from 1966 aerial photographs, which by comparing to an airborne LiDAR DTM from 2020, we compute changes in surface elevation and geodetic mass balances. The area mapped in both surveys cover about 3/4 of the ice cap area and 49 of 82 glaciers. The measured glacier area has decreased from 363.4 km2 in 1966 to 332.9 km2 in 2019, i.e. a change of −30 km2 or −8.4% (−0.16% a−1), which is in line with the percentage reduction in area for Jostedalsbreen as a whole. The mean geodetic mass balance over the 49 glaciers was −0.15 ± 0.01 m w.e. a−1, however, large variability is evident between glaciers, e.g. Nigardsbreen (−0.05 m w.e. a−1), Austdalsbreen (−0.28 m w.e. a−1) and Tunsbergdalsbreen (−0.36 m w.e. a−1) confirming differences also found by the glaciological records for Nigardsbreen and Austdalsbreen

Glacier thinning, recession and advance, and the associated evolution of a glacial lake between 1966 and 2021 at Austerdalsbreen, western Norway

The Jostedalsbreen is the largest ice cap in Norway and mainland Europe. Rapid retreat of many of its outlet glaciers since the 2000s has led to the formation of several glacial lakes. Processes causing the formation and expansion of glacial lakes and their interaction with a glacier and terminal moraine have not been widely addressed yet. In this study, we investigate the degradation of the front of the southeast-facing outlet glacier Austerdalsbreen. Based on a variety of remotely sensed data (UAV-based and airborne orthophotos and DEMs, satellite images), we analyze the coincident glacial and proglacial changes of Austerdalsbreen and quantify the evolution of this transition zone during the last decades. In particular, we focus on the short-term evolution of the glacial lake since 2010, we examine the context of a glacier advance in the 1990s, and we report long-term changes by utilizing 1960s imagery. We discuss the evolution and conditions of Austerdalsbreen compared to other outlet glaciers of Jostedalsbreen. Overall, the glacier terminus has experienced a recession in the last decades. The 1990s terminus advance was more restricted than at other nearby outlet glaciers due to glacier surface debris cover, which is a critical factor for the glacier and lake evolution. However, in the most recent period, since 2012, a distinct expansion of a glacial lake is quantifiable. Since the rates of glacier surface lowering also considerably increased since approximately 2017 and the glacier retreated since the beginning of the 2000s with a clear maximum length decrease in 2015, we interpret the recently formed glacial lake as a contributory factor of glacial changes

The Intentional Use of Service Recovery Strategies to Influence Consumer Emotion, Cognition and Behaviour

Service recovery strategies have been identified as a critical factor in the success of. service organizations. This study develops a conceptual frame work to investigate how specific service recovery strategies influence the emotional, cognitive and negative behavioural responses of . consumers., as well as how emotion and cognition influence negative behavior. Understanding the impact of specific service recovery strategies will allow service providers' to more deliberately and intentionally engage in strategies that result in positive organizational outcomes. This study was conducted using a 2 x 2 between-subjects quasi-experimental design. The results suggest that service recovery has a significant impact on emotion, cognition and negative behavior. Similarly, satisfaction, negative emotion and positive emotion all influence negative behavior but distributive justice has no effect

School-based prevention for adolescent Internet addiction: prevention is the key. A systematic literature review

Adolescents’ media use represents a normative need for information, communication, recreation and functionality, yet problematic Internet use has increased. Given the arguably alarming prevalence rates worldwide and the increasingly problematic use of gaming and social media, the need for an integration of prevention efforts appears to be timely. The aim of this systematic literature review is (i) to identify school-based prevention programmes or protocols for Internet Addiction targeting adolescents within the school context and to examine the programmes’ effectiveness, and (ii) to highlight strengths, limitations, and best practices to inform the design of new initiatives, by capitalizing on these studies’ recommendations. The findings of the reviewed studies to date presented mixed outcomes and are in need of further empirical evidence. The current review identified the following needs to be addressed in future designs to: (i) define the clinical status of Internet Addiction more precisely, (ii) use more current psychometrically robust assessment tools for the measurement of effectiveness (based on the most recent empirical developments), (iii) reconsider the main outcome of Internet time reduction as it appears to be problematic, (iv) build methodologically sound evidence-based prevention programmes, (v) focus on skill enhancement and the use of protective and harm-reducing factors, and (vi) include IA as one of the risk behaviours in multi-risk behaviour interventions. These appear to be crucial factors in addressing future research designs and the formulation of new prevention initiatives. Validated findings could then inform promising strategies for IA and gaming prevention in public policy and education

Genetic Sharing with Cardiovascular Disease Risk Factors and Diabetes Reveals Novel Bone Mineral Density Loci.

Bone Mineral Density (BMD) is a highly heritable trait, but genome-wide association studies have identified few genetic risk factors. Epidemiological studies suggest associations between BMD and several traits and diseases, but the nature of the suggestive comorbidity is still unknown. We used a novel genetic pleiotropy-informed conditional False Discovery Rate (FDR) method to identify single nucleotide polymorphisms (SNPs) associated with BMD by leveraging cardiovascular disease (CVD) associated disorders and metabolic traits. By conditioning on SNPs associated with the CVD-related phenotypes, type 1 diabetes, type 2 diabetes, systolic blood pressure, diastolic blood pressure, high density lipoprotein, low density lipoprotein, triglycerides and waist hip ratio, we identified 65 novel independent BMD loci (26 with femoral neck BMD and 47 with lumbar spine BMD) at conditional FDR < 0.01. Many of the loci were confirmed in genetic expression studies. Genes validated at the mRNA levels were characteristic for the osteoblast/osteocyte lineage, Wnt signaling pathway and bone metabolism. The results provide new insight into genetic mechanisms of variability in BMD, and a better understanding of the genetic underpinnings of clinical comorbidity

State of the climate in 2013

In 2013, the vast majority of the monitored climate variables reported here maintained trends established in recent decades. ENSO was in a neutral state during the entire year, remaining mostly on the cool side of neutral with modest impacts on regional weather patterns around the world. This follows several years dominated by the effects of either La Niña or El Niño events. According to several independent analyses, 2013 was again among the 10 warmest years on record at the global scale, both at the Earths surface and through the troposphere. Some regions in the Southern Hemisphere had record or near-record high temperatures for the year. Australia observed its hottest year on record, while Argentina and New Zealand reported their second and third hottest years, respectively. In Antarctica, Amundsen-Scott South Pole Station reported its highest annual temperature since records began in 1957. At the opposite pole, the Arctic observed its seventh warmest year since records began in the early 20th century. At 20-m depth, record high temperatures were measured at some permafrost stations on the North Slope of Alaska and in the Brooks Range. In the Northern Hemisphere extratropics, anomalous meridional atmospheric circulation occurred throughout much of the year, leading to marked regional extremes of both temperature and precipitation. Cold temperature anomalies during winter across Eurasia were followed by warm spring temperature anomalies, which were linked to a new record low Eurasian snow cover extent in May. Minimum sea ice extent in the Arctic was the sixth lowest since satellite observations began in 1979. Including 2013, all seven lowest extents on record have occurred in the past seven years. Antarctica, on the other hand, had above-average sea ice extent throughout 2013, with 116 days of new daily high extent records, including a new daily maximum sea ice area of 19.57 million km2 reached on 1 October. ENSO-neutral conditions in the eastern central Pacific Ocean and a negative Pacific decadal oscillation pattern in the North Pacific had the largest impacts on the global sea surface temperature in 2013. The North Pacific reached a historic high temperature in 2013 and on balance the globally-averaged sea surface temperature was among the 10 highest on record. Overall, the salt content in nearsurface ocean waters increased while in intermediate waters it decreased. Global mean sea level continued to rise during 2013, on pace with a trend of 3.2 mm yr-1 over the past two decades. A portion of this trend (0.5 mm yr-1) has been attributed to natural variability associated with the Pacific decadal oscillation as well as to ongoing contributions from the melting of glaciers and ice sheets and ocean warming. Global tropical cyclone frequency during 2013 was slightly above average with a total of 94 storms, although the North Atlantic Basin had its quietest hurricane season since 1994. In the Western North Pacific Basin, Super Typhoon Haiyan, the deadliest tropical cyclone of 2013, had 1-minute sustained winds estimated to be 170 kt (87.5 m s-1) on 7 November, the highest wind speed ever assigned to a tropical cyclone. High storm surge was also associated with Haiyan as it made landfall over the central Philippines, an area where sea level is currently at historic highs, increasing by 200 mm since 1970. In the atmosphere, carbon dioxide, methane, and nitrous oxide all continued to increase in 2013. As in previous years, each of these major greenhouse gases once again reached historic high concentrations. In the Arctic, carbon dioxide and methane increased at the same rate as the global increase. These increases are likely due to export from lower latitudes rather than a consequence of increases in Arctic sources, such as thawing permafrost. At Mauna Loa, Hawaii, for the first time since measurements began in 1958, the daily average mixing ratio of carbon dioxide exceeded 400 ppm on 9 May. The state of these variables, along with dozens of others, and the 2013 climate conditions of regions around the world are discussed in further detail in this 24th edition of the State of the Climate series. © 2014, American Meteorological Society. All rights reserved

The genetic architecture of the human cerebral cortex

INTRODUCTION The cerebral cortex underlies our complex cognitive capabilities. Variations in human cortical surface area and thickness are associated with neurological, psychological, and behavioral traits and can be measured in vivo by magnetic resonance imaging (MRI). Studies in model organisms have identified genes that influence cortical structure, but little is known about common genetic variants that affect human cortical structure. RATIONALE To identify genetic variants associated with human cortical structure at both global and regional levels, we conducted a genome-wide association meta-analysis of brain MRI data from 51,665 individuals across 60 cohorts. We analyzed the surface area and average thickness of the whole cortex and 34 cortical regions with known functional specializations. RESULTS We identified 306 nominally genome-wide significant loci (P < 5 × 10−8) associated with cortical structure in a discovery sample of 33,992 participants of European ancestry. Of the 299 loci for which replication data were available, 241 loci influencing surface area and 14 influencing thickness remained significant after replication, with 199 loci passing multiple testing correction (P < 8.3 × 10−10; 187 influencing surface area and 12 influencing thickness). Common genetic variants explained 34% (SE = 3%) of the variation in total surface area and 26% (SE = 2%) in average thickness; surface area and thickness showed a negative genetic correlation (rG = −0.32, SE = 0.05, P = 6.5 × 10−12), which suggests that genetic influences have opposing effects on surface area and thickness. Bioinformatic analyses showed that total surface area is influenced by genetic variants that alter gene regulatory activity in neural progenitor cells during fetal development. By contrast, average thickness is influenced by active regulatory elements in adult brain samples, which may reflect processes that occur after mid-fetal development, such as myelination, branching, or pruning. When considered together, these results support the radial unit hypothesis that different developmental mechanisms promote surface area expansion and increases in thickness. To identify specific genetic influences on individual cortical regions, we controlled for global measures (total surface area or average thickness) in the regional analyses. After multiple testing correction, we identified 175 loci that influence regional surface area and 10 that influence regional thickness. Loci that affect regional surface area cluster near genes involved in the Wnt signaling pathway, which is known to influence areal identity. We observed significant positive genetic correlations and evidence of bidirectional causation of total surface area with both general cognitive functioning and educational attainment. We found additional positive genetic correlations between total surface area and Parkinson’s disease but did not find evidence of causation. Negative genetic correlations were evident between total surface area and insomnia, attention deficit hyperactivity disorder, depressive symptoms, major depressive disorder, and neuroticism. CONCLUSION This large-scale collaborative work enhances our understanding of the genetic architecture of the human cerebral cortex and its regional patterning. The highly polygenic architecture of the cortex suggests that distinct genes are involved in the development of specific cortical areas. Moreover, we find evidence that brain structure is a key phenotype along the causal pathway that leads from genetic variation to differences in general cognitive function

Novel genetic loci associated with hippocampal volume

The hippocampal formation is a brain structure integrally involved in episodic memory, spatial navigation, cognition and stress responsiveness. Structural abnormalities in hippocampal volume and shape are found in several common neuropsychiatric disorders. To identify the genetic underpinnings of hippocampal structure here we perform a genome-wide association study (GWAS) of 33,536 individuals and discover six independent loci significantly associated with hippocampal volume, four of them novel. Of the novel loci, three lie within genes (ASTN2, DPP4 and MAST4) and one is found 200 kb upstream of SHH. A hippocampal subfield analysis shows that a locus within the MSRB3 gene shows evidence of a localized effect along the dentate gyrus, subiculum, CA1 and fissure. Further, we show that genetic variants associated with decreased hippocampal volume are also associated with increased risk for Alzheimer's disease (rg =-0.155). Our findings suggest novel biological pathways through which human genetic variation influences hippocampal volume and risk for neuropsychiatric illness