71 research outputs found

    Pension helpdesk calls:A repair mechanism in the client communication of financial institutions

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    This paper analyzes the role of helpdesk calls in the client communication package of pension funds.Our audio-corpus of 77 helpdesk calls contained 104 client questions. These show that clients seem tocall the helpdesk in order to repair a comprehension problem, to find specific information they missed,to repair incorrect information or an administrative failure. In terms of Media Synchronicity Theory,helpdesk calls are most often used to repair unsuccessful conveyance processes by providing extra infor-mation, rectifying information or by addressing misunderstandings. Overall, the helpdesk is only usedfor straightforward inquiries and problems, not for financial advice

    How the World Changed Social Media

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    How the World Changed Social Media is the first book in Why We Post, a book series that investigates the findings of nine anthropologists who each spent 15 months living in communities across the world. This book offers a comparative analysis summarising the results of the research and exploring the impact of social media on politics and gender, education and commerce. What is the result of the increased emphasis on visual communication? Are we becoming more individual or more social? Why is public social media so conservative? Why does equality online fail to shift inequality offline? How did memes become the moral police of the internet? Supported by an introduction to the project’s academic framework and theoretical terms that help to account for the findings, the book argues that the only way to appreciate and understand something as intimate and ubiquitous as social media is to be immersed in the lives of the people who post. Only then can we discover how people all around the world have already transformed social media in such unexpected ways and assess the consequences.published_or_final_versio

    Beyond Electronics

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    Amundson traces the histories of electronic technologies, video, and electronic art, examining the shift from art about the technology itself to work that uses it as an expressive tool or addresses its social dimension. Lists exhibitions related to art and technology. Statements by 11 artists. Biographical notes. 14 bibl. ref

    दुनिया ने जैसे सामाजिक मीडिया को बदल दिया

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    दुनिया ने जैसे सामाजिक मीडिया को बदल दिया, हम क्यों पोस्ट करते हैं ग्रन्थ श्रृंखला का पहला ग्रन्थ है जो उन नौ मानवविज्ञानियों के निष्कर्षों पर जाँच करता है जिन्होंने दुनिया भर के समूहों में १५ महीने तक बिताया जिसमे शामिल है ब्राज़ील, चिली, चीन, इंग्लैंड, भारत, इटली, ट्रिनिडाड और टर्की. यह ग्रन्थ एक तुलनात्मक विश्लेषण को प्रदान करता है जो अनुसंधान के परिणाम को संक्षेप में प्रस्तुत करता है और राजनीति और लिंग, शिक्षा और व्यापार पर सामाजिक मीडिया के प्रभाव का पता लगाता है. दृश्य संचार पर बढ़ते हुए ज़ोर का परिणाम क्या है? क्या हम अधिक व्यक्तिगत या सामाजिक बनते हैं? क्यों सार्वजनिक सामाजिक मीडिया अधिक रूढ़िवादी होता है? क्यों ऑनलाइन समानता ऑफलाइन असमानता को बदलने में असफल होता है? कैसे मिमी इंटरनेट के नैतिक पुलिस बन गए? परियोजना के शैक्षिक ढाँचा और सैद्धांतिक शर्तों, जो निष्कर्षों के उत्तरदायी होने में मदद करते हैं, के परिचय से समर्थित होकर यह ग्रन्थ तर्क करता है कि सामाजिक मीडिया जैसे अन्तरंग और सर्वव्यापक वास्तु को समझने और मूल्यांकन करने का एक ही रास्ता पोस्ट करनेवाले लोगों के जीवन में तल्लीन होकर रहना है. तभी हम पता लगा सकते हैं कि दुनिया भर के लोगों ने जैसे सामाजिक मीडिया को अभी तक अप्रत्याशित तरीकों से बदल दिया हैं और उनके परिणाम पर आकलन कर सकते हैं

    உலகம் சமூக ஊடகங்களை எப்படி மாற்றியிருக்கிறது How the world changed social media (Tamil)

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    How the World Changed Social Media is the first book in Why We Post, a book series that investigates the findings of anthropologists who each spent 15 months living in communities across the world. This book offers a comparative analysis summarising the results of the research and explores the impact of social media on politics and gender, education and commerce. What is the result of the increased emphasis on visual communication? Are we becoming more individual or more social? Why is public social media so conservative? Why does equality online fail to shift inequality offline? How did memes become the moral police of the internet? Supported by an introduction to the project’s academic framework and theoretical terms that help to account for the findings, the book argues that the only way to appreciate and understand something as intimate and ubiquitous as social media is to be immersed in the lives of the people who post. Only then can we discover how people all around the world have already transformed social media in such unexpected ways and assess the consequences

    Evolution of long-term vaccine-induced and hybrid immunity in healthcare workers after different COVID-19 vaccine regimens

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    BACKGROUND: Both infection and vaccination, alone or in combination, generate antibody and T cell responses against severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). However, the maintenance of such responses-and hence protection from disease-requires careful characterization. In a large prospective study of UK healthcare workers (HCWs) (Protective Immunity from T Cells in Healthcare Workers [PITCH], within the larger SARS-CoV-2 Immunity and Reinfection Evaluation [SIREN] study), we previously observed that prior infection strongly affected subsequent cellular and humoral immunity induced after long and short dosing intervals of BNT162b2 (Pfizer/BioNTech) vaccination. METHODS: Here, we report longer follow-up of 684 HCWs in this cohort over 6-9 months following two doses of BNT162b2 or AZD1222 (Oxford/AstraZeneca) vaccination and up to 6 months following a subsequent mRNA booster vaccination. FINDINGS: We make three observations: first, the dynamics of humoral and cellular responses differ; binding and neutralizing antibodies declined, whereas T and memory B cell responses were maintained after the second vaccine dose. Second, vaccine boosting restored immunoglobulin (Ig) G levels; broadened neutralizing activity against variants of concern, including Omicron BA.1, BA.2, and BA.5; and boosted T cell responses above the 6-month level after dose 2. Third, prior infection maintained its impact driving larger and broader T cell responses compared with never-infected people, a feature maintained until 6 months after the third dose. CONCLUSIONS: Broadly cross-reactive T cell responses are well maintained over time-especially in those with combined vaccine and infection-induced immunity ("hybrid" immunity)-and may contribute to continued protection against severe disease

    EULAR recommendations for the management of rheumatoid arthritis with synthetic and biological disease-modifying antirheumatic drugs: 2016 update

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    Recent insights in rheumatoid arthritis (RA) necessitated updating the European League Against Rheumatism (EULAR) RA management recommendations. A large international Task Force based decisions on evidence from 3 systematic literature reviews, developing 4 overarching principles and 12 recommendations (vs 3 and 14, respectively, in 2013). The recommendations address conventional synthetic (cs) disease-modifying antirheumatic drugs (DMARDs) (methotrexate (MTX), leflunomide, sulfasalazine); glucocorticoids (GC); biological (b) DMARDs (tumour necrosis factor (TNF)-inhibitors (adalimumab, certolizumab pegol, etanercept, golimumab, infliximab), abatacept, rituximab, tocilizumab, clazakizumab, sarilumab and sirukumab and biosimilar (bs) DMARDs) and targeted synthetic (ts) DMARDs (Janus kinase (Jak) inhibitors tofacitinib, baricitinib). Monotherapy, combination therapy, treatment strategies (treat-to-target) and the targets of sustained clinical remission (as defined by the American College of Rheumatology-(ACR)-EULAR Boolean or index criteria) or low disease activity are discussed. Cost aspects were taken into consideration. As first strategy, the Task Force recommends MTX (rapid escalation to 25 mg/week) plus short-term GC, aiming at >50% improvement within 3 and target attainment within 6 months. If this fails stratification is recommended. Without unfavourable prognostic markers, switching to—or adding—another csDMARDs (plus short-term GC) is suggested. In the presence of unfavourable prognostic markers (autoantibodies, high disease activity, early erosions, failure of 2 csDMARDs), any bDMARD (current practice) or Jak-inhibitor should be added to the csDMARD. If this fails, any other bDMARD or tsDMARD is recommended. If a patient is in sustained remission, bDMARDs can be tapered. For each recommendation, levels of evidence and Task Force agreement are provided, both mostly very high. These recommendations intend informing rheumatologists, patients, national rheumatology societies, hospital officials, social security agencies and regulators about EULAR's most recent consensus on the management of RA, aimed at attaining best outcomes with current therapies
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