4 research outputs found
Effects of inertial overload resistance training on muscle function.
El entrenamiento de fuerza debe incluirse en todos los programas de
ejercicio encaminados a mejorar la salud y la calidad de vida. Los programas de
entrenamiento de fuerza han priorizado los regĂmenes de contracciĂłn
concéntricos a los excéntricos, sin embargo, actualmente se estå realizando un
tipo de entrenamiento de fuerza basado en contracciones excéntricas mediante
sobrecargas de tipo inercial. Por tanto, el objetivo del presente trabajo de revisiĂłn
bibliogrĂĄfica ha sido comprobar los efectos de este tipo de entrenamiento basado
en contracciones excéntricas mediante sobrecargas de tipo inercial sobre la
funciĂłn muscular. Para ello se realizĂł una bĂșsqueda bibliogrĂĄfica en las bases
de datos Web of Science, Pubmed, Medline, Dialnet y Scielo. Los resultados de
nuestra revisiĂłn sugieren que este tipo de entrenamiento da lugar a una mayor
actividad electromiogrĂĄfica e hipertrofia muscular con respecto a programas de
entrenamiento convencionales, al tiempo que podrĂan ser efectivos en la
recuperaciĂłn de lesiones mĂșsculo-tendinosasResistance training should be included in all exercise programmes which
improve health and quality of live. These programmes have been focusing on
both concentric-eccentric contractions, however, a new type of resistance
training based on eccentric contractions provided by inertial overload is being
carried out. Therefore, the aim of the present study is to prove the effects of this
kind of training based on eccentric contractions by inertial overload. Databases
utilized to carry out information research were Web of Science, Pubmed,
Medline, Dialnet and Scielo. Results would suggest that inertial training based
on inertial overload produces maximal EMG and an earlier muscular
hypertrophy compared to conventional resistance training, besides the fact it
could have successful on muscle-tendon injurie
Paracrine cellular senescence exacerbates biliary injury and impairs regeneration
Senescence has been suggested as causing biliary cholangiopathies but how this is regulated is unclear. Here, the authors generate a mouse model of biliary senescence by deleting Mdm2 in bile ducts and show that inhibiting TGFÎČ limits senescence-dependent aggravation of cholangiopathies
Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018.
Over the past decade, the Nomenclature Committee on Cell Death (NCCD) has formulated guidelines for the definition and interpretation of cell death from morphological, biochemical, and functional perspectives. Since the field continues to expand and novel mechanisms that orchestrate multiple cell death pathways are unveiled, we propose an updated classification of cell death subroutines focusing on mechanistic and essential (as opposed to correlative and dispensable) aspects of the process. As we provide molecularly oriented definitions of terms including intrinsic apoptosis, extrinsic apoptosis, mitochondrial permeability transition (MPT)-driven necrosis, necroptosis, ferroptosis, pyroptosis, parthanatos, entotic cell death, NETotic cell death, lysosome-dependent cell death, autophagy-dependent cell death, immunogenic cell death, cellular senescence, and mitotic catastrophe, we discuss the utility of neologisms that refer to highly specialized instances of these processes. The mission of the NCCD is to provide a widely accepted nomenclature on cell death in support of the continued development of the field