Alaska Earthquake Center: A 2020 Perspective

The Alaska Earthquake Center is not historically in the habit of producing annual reports. We are in a dynamic time, however. Societally-significant earthquakes and multiple tsunami concerns over the past few years have brought more attention to what we do. At the same time, we are experiencing significant growth in several areas. Our goal in distributing this summary is to communicate the breadth of our activities and the diversity of our stakeholders, helping us become even more effective at meeting the earthquake and tsunami science needs of Alaska and the nation

Continuous Decoupling of Dynamically Expanding Systems

The question of decoupling and freeze-out is reinvestigated and analysed in terms of transparent semi-classical decoupling formulae, which provide a smooth decoupling in time both, for single and two particle inclusive spectra. They generalise frequently employed instantaneous freeze-out procedures and provide simple relations between the damping width and the duration of the decoupling process. The implications on physical phenomena arising from the expansion and decay dynamics of the highly compressed hadronic matter generated in high energy nuclear collisions are discussed.Comment: The paper is significantly revised concentrating on the physics discussion, thereby including material that appeared during the revision stage. The formal derivations are deferred to a forthcoming paper. 20 pages, 2 Figures, Nucl. Phys. A in pres

Alaska Earthquake Center Quarterly Technical Report October-December 2021

This series of technical quarterly reports from the Alaska Earthquake Center (AEC) includes detailed summaries and updates on Alaska seismicity, the AEC seismic network and stations, field work, our social media presence, and lists publications and presentations by AEC staff. Multiple AEC staff members contributed to this report. It is issued in the following month after the completion of each quarter Q1: January-March, Q2: April-June, Q3: July-September, and Q4: October-December.1. Introduction 2. Seismicity 3. Field network 4. Data Quality assurance 5. Real-time earthquake detection system 6. Computer systems 6.1 Computer resources 6.2 Waveform storage 6.3 Metadata 6.4 Software development 7. Fieldwork 8. Social media and outreach 8.1. Website 8.2. Twitter 8.3. Facebook (Page) 8.4. Facebook (Group) 9. Publications and presentations 9.1. Publications 9.2. Public Presentations 9.3. Lunch Seminar Talks 10. References Appendix A: Data availability for broadband stations from the AK network. Appendix B: Gaps for broadband stations from the AK network

Alaska Earthquakes Poster

Alaska is one of the most seismically active places in the world. This poster connects the geographic distribution of earthquakes from the Alaska Earthquake Center catalog with the core concepts that drive Alaska seismicity. Rupture patches, how plate tectonics forms faults throughout Alaska, and how the angle of the sinking Pacific Plate affects earthquake distribution and creates volcanoes are some of the key concepts represented.Ye

Is working memory training in older adults sensitive to music?

Evidence in the literature suggests that listening to music can improve cognitive performance. The aim of the present study was to examine whether the short- and long-term gains of a working memory (WM) training in older adults could be enhanced by music listening\u2014the Mozart\u2019s Sonata K448 and the Albinoni\u2019s Adagio in G minor\u2014which differ in tempo and mode. Seventy-two healthy older adults (age range: 65\u201375 years) participated in the study. They were divided into four groups. At each training session, before starting the WM training activities, one group listened to Mozart (Mozart group, N = 19), one to Albinoni (Albinoni group, N = 19), one to white noise (White noise group, N = 16), while one served as an active control group involved in other activities and was not exposed to any music (active control group, N = 18). Specific training gains on a task like the one used in the training, and transfer effects on visuo-spatial abilities, executive function and reasoning measures were assessed. Irrespective of listening condition (Mozart, Albinoni, White noise), trained groups generally outperformed the control group. The White noise group never differed from the two music groups. However, the Albinoni group showed larger specific training gains in the criterion task at short-term and transfer effects in the reasoning task at both short-and long term compared to the Mozart group. Overall the present findings suggest caution when interpreting the effects of music before a WM training, and are discussed according to aging and music effect literature

The cardiac sympathetic co-transmitter galanin reduces acetylcholine release and vagal bradycardia: Implications for neural control of cardiac excitability

The autonomic phenotype of congestive cardiac failure is characterised by high sympathetic drive and impaired vagal tone, which are independent predictors of mortality. We hypothesize that impaired bradycardia to peripheral vagal stimulation following high-level sympathetic drive is due to sympatho-vagal crosstalk by the adrenergic co-transmitters galanin and neuropeptide-Y (NPY). Moreover we hypothesize that galanin acts similarly to NPY by reducing vagal acetylcholine release via a receptor mediated, protein kinase-dependent pathway. Prolonged right stellate ganglion stimulation (10 Hz, 2 min, in the presence of 10 μM metoprolol) in an isolated guinea pig atrial preparation with dual autonomic innervation leads to a significant (p < 0.05) reduction in the magnitude of vagal bradycardia (5 Hz) maintained over the subsequent 20 min (n = 6). Immunohistochemistry demonstrated the presence of galanin in a small number of tyrosine hydroxylase positive neurons from freshly dissected stellate ganglion tissue sections. Following 3 days of tissue culture however, most stellate neurons expressed galanin. Stellate stimulation caused the release of low levels of galanin and significantly higher levels of NPY into the surrounding perfusate (n = 6, using ELISA). The reduction in vagal bradycardia post sympathetic stimulation was partially reversed by the galanin receptor antagonist M40 after 10 min (1 μM, n = 5), and completely reversed with the NPY Y2 receptor antagonist BIIE 0246 at all time points (1 μM, n = 6). Exogenous galanin (n = 6, 50–500 nM) also reduced the heart rate response to vagal stimulation but had no effect on the response to carbamylcholine that produced similar degrees of bradycardia (n = 6). Galanin (500 nM) also significantly attenuated the release of 3H-acetylcholine from isolated atria during field stimulation (5 Hz, n = 5). The effect of galanin on vagal bradycardia could be abolished by the galanin receptor antagonist M40 (n = 5). Importantly the GalR1 receptor was immunofluorescently co-localised with choline acetyl-transferase containing neurons at the sinoatrial node. The protein kinase C inhibitor calphostin (100 nM, n = 6) abolished the effect of galanin on vagal bradycardia whilst the protein kinase A inhibitor H89 (500 nM, n = 6) had no effect. These results demonstrate that prolonged sympathetic activation releases the slowly diffusing adrenergic co-transmitter galanin in addition to NPY, and that this contributes to the attenuation in vagal bradycardia via a reduction in acetylcholine release. This effect is mediated by GalR1 receptors on vagal neurons coupled to protein kinase C dependent signalling pathways. The role of galanin may become more important following an acute injury response where galanin expression is increased