168 research outputs found
A role for antibiotic biosynthesis monooxygenase domain proteins in fidelity control during aromatic polyketide biosynthesis
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 09/08/2019
- Field of study
We report the formicapyridines which are structurally and biosynthetically related to the pentacyclic fasamycin and formicamycin aromatic polyketides but comprise a rare pyridine moiety. These new compounds are trace level metabolites formed by derailment of the major biosynthetic pathway. Inspired by evolutionary logic we show that rational mutation of a single gene in the biosynthetic gene cluster leads to a significant increase both in total formicapyridine production and their enrichment relative to the fasamycins/formicamycins. Our observations broaden the polyketide biosynthetic landscape and identify a non-catalytic role for ABM superfamily proteins in type II polyketide synthase assemblages for maintaining biosynthetic pathway fidelity
Coffee consumption modifies risk of estrogen-receptor negative breast cancer
- Author
- A Nkondjock
- A Olsen
- A Scalbert
- A Tavani
- BN Ames
- C Magnusson
- CC Orgeas
- D Flesch-Janys
- D Ganmaa
- Dieter Flesch-Janys
- E Lopez-Garcia
- GB Mensink
- Jenny Chang-Claude
- Jianjun Liu
- Jingmei Li
- JP Minton
- K Ishitani
- Kamila Czene
- Keith Humphreys
- KF Allred
- L Arab
- LM Nilsson
- LU Rosenberg
- M Ahlgren
- N Bentzon
- N Bhoo Pathy
- N Tang
- NK Horner
- Per Hall
- Petra Seibold
- PL Horn-Ross
- R Development Core Team
- RC Alves
- RM Alonso-Salces
- SC Larsson
- Sweden Statistics
- TB Young
- WJ Lee
- Y Fukushima
- Publication venue
- BioMed Central
- Publication date
- 01/01/2011
- Field of study
10.1186/bcr2879Breast Cancer Research133R4
Polar extracts from (Tunisian) Acacia salicina Lindl. Study of the antimicrobial and antigenotoxic activities
- Author
- A Cremieux
- A Russo
- A Wadood
- AA Dafallah
- AH Gilani
- BH Shah
- BN Ames
- CC Harris
- D Villaño
- DE Levin
- DM Maron
- G Getachew
- G Hussein
- H Ben-Mansour
- Hedi Ben Mansour
- I Bouhlel
- I Bouhlel
- J Boubaker
- J Vaya
- Jihed Boubaker
- JP Hu
- K Swayamjot
- Kamel Ghedira
- LC Chiang
- Leila Chekir Ghedira
- M Chaib
- M Dhidah
- M Zoran
- MM Bradford
- N Hayder
- NK Ghosh
- P Re
- PE Hartman
- PQ Quillardet
- PS Mead
- R Ben Ammar
- R Edenharder
- R Van der Berg
- RC Horn
- RCP Marques
- RW Teel
- S Amos
- SA Sotohy
- SJ O'Brien
- WH Koch
- Y Oyangagui
- Publication venue
- BioMed Central
- Publication date
- 01/01/2012
- Field of study
<p>Abstract</p> <p>Background</p> <p>Methanolic, aqueous and Total Oligomer Flavonoids (TOF)-enriched extracts obtained from the leaves of <it>Acacia salicina </it>'Lindl.' were investigated for antibacterial, antimutagenic and antioxidant activities.</p> <p>Methods</p> <p>The antimicrobial activity was tested on the Gram positive and Gram negative reference bacterial strains. The Mutagenic and antimutagenic activities against direct acting mutagens, methylmethane sulfonate (MMS) and 4-nitro-o-phenylenediamine (NOPD), and indirect acting mutagens, 2-aminoanthracene (2-AA) and benzo[a]pyrene (B(a)P) were performed with <it>S. typhimurium </it>TA102 and TA98 assay systems. In addition, the enzymatic and nonenzymatic methods were employed to evaluate the anti-oxidative effects of the tested extracts.</p> <p>Results</p> <p>A significant effect against the Gram positive and Gram negative reference bacterial strains was observed with all the extracts. The mutagenic and antimutagenic studies revealed that all the extracts decreased the mutagenicity induced by B(a)P (7.5 μg/plate), 2-AA (5 μg/plate), MMS (1.3 mg/plate) and NOPD (10 μg/plate). Likewise, all the extracts showed an important free radical scavenging activity towards the superoxide anion generated by the xanthine/xanthine oxidase assay system, as well as high Trolox Equivalent Antioxidant Capacity (TEAC), against the 2,2'-azino-bis(3-ethylbenzothiazoline-6-sulfonic acid) diammonium salt (ABTS)<sup>+</sup>• radical. TOF-enriched extract exhibited the highest protective effect against free radicals, direct acting-mutagen and metabolically activated S9-dependent mutagens.</p> <p>Conclusions</p> <p>The present study indicates that the extracts from <it>A. salicina </it>leaves are a significant source of compounds with the antimutagenic and antioxidant activities, and this may be useful for developing potential chemopreventive substances.</p
Population genomics of marine zooplankton
- Author
- A Biscontin
- A Bucklin
- A Bucklin
- A Bucklin
- A Bucklin
- A Bucklin
- A Bucklin
- A Bucklin
- A Bucklin
- A Cornils
- A Levasseur
- A Weydmann
- A Whitehead
- AA Ramos
- AA Thabet
- AB Kohn
- AE Maas
- AM Reitzel
- AM Tarrant
- AR Longhurst
- B Meyer
- B Miller
- B Nilsson
- B Planque
- B Young de
- BE Deagle
- BK Peterson
- BR Foley
- BY Lee
- C Castellani
- C Hahn
- C Lauritano
- C Papetti
- C Papot
- C Pittà De
- C Riginos
- C Schlötterer
- C Schunter
- C Sun
- C Sun
- C Vargas de
- CC Caudill
- CD Prokopowich
- CDH Sherman
- CE Lee
- CE Lee
- CL Ames
- CR Voolstra
- D Abad
- D Papillon
- D-S Hwang
- DE Schindel
- DL Crawford
- DN Kulagin
- DO Hessen
- DPL Toews
- E Bortolotto
- E Faure
- E Goetze
- E Goetze
- E Goetze
- E Hodges
- E Kayal
- E Petkeviciute
- E Unal
- EE Nielsen
- EJ Yang
- EJH Head
- EL Norton
- EM Rasch
- F Alberto
- F Carlotti
- F Denoeud
- F Dufresne
- F Li
- F Marlétaz
- FS Barreto
- G Beaugrand
- G Chen
- G Chen
- G Chust
- G Drillet
- G Luikart
- G Sales
- G Tsagkogeorga
- GA Wyngaard
- GA Wyngaard
- GA Wyngaard
- Genome 10K Community of Scientists
- GH Pogson
- GIGA Community of Scientists
- H Ellegren
- H Miyamoto
- H Miyamoto
- HM Bik
- HM Galindo
- HP Leinaas
- HR Skjoldal
- HS Kim
- HS Kim
- HY Koh
- I Smolina
- I Smolina
- I Smolina
- i5K Consortium
- IA McLaren
- IA McLaren
- IG Romero
- J Bailey
- J Hemmer-Hansen
- J Hirai
- J Hirai
- J Provan
- J Rahlff
- J Stapley
- JC Avise
- JC Avise
- JC Havird
- JE Bron
- Jennifer Marie Questel
- JF O’Grady
- JF Ryan
- JK Pearman
- JL Davey
- JS Ki
- JS Ki
- JT Kool
- JY Toullec
- K Alfsnes
- K Ogoh
- K Stopar
- K Wang
- K Weersing
- KG Helfenbein
- KM Johnson
- KR Andrews
- KTCA Peijnenburg
- KTCA Peijnenburg
- KTCA Peijnenburg
- KTCA Peijnenburg
- L Blanco-Bercial
- L Blanco-Bercial
- L Excoffier
- L Excoffier
- L Zane
- L Zane
- L Zane
- Leocadio Blanco-Bercial
- LL Knowles
- LL Moroz
- LN Papadopoulos
- M Baratti
- M Choquet
- M Foll
- M Iacchei
- M Kuriyama
- M Lechner
- M Leray
- M Omori
- M-A Madoui
- ME Cristescu
- ME Hellberg
- MM Hansen
- MN Dawson
- MR Jones
- MS Clark
- N Bierne
- N Bierne
- NA Baird
- NK Jue
- NW Jeffery
- NW Jeffery
- NW Jeffery
- O Savolainen
- ONS Aarbakke
- ONS Aarbakke
- P Beerli
- P Gayral
- P Li
- P Wit De
- P Wit De
- PA Gagnaire
- PA Gagnaire
- PD Rawson
- PG Batta-Lona
- PG Batta-Lona
- PH Lenz
- PH Wiebe
- PH Wiebe
- PH Wiebe
- PJ Milligan
- PK Lindeque
- PM Jepsen
- PW Hedrick
- R Ekblom
- R Escribano
- R Saborowski
- RJ Elshire
- RJ Machida
- RJ Pereira
- RJ Pereira
- RK Cowen
- RS Burton
- RS Burton
- RS Waples
- RS Waples
- S Bolte
- S Edmands
- S Eyun
- S Falk-Petersen
- S Kang
- S Kim
- S Kim
- S Kollias
- S Laakmann
- S Raisuddin
- S Renaut
- S Wang
- S Wei
- SA Levin
- SD Schoville
- SJ Helyar
- SM Bybee
- SM Francisco
- SN Jarman
- SO Jung
- SR Narum
- T Maricic
- TA Elliott
- TG Lima
- TM McGovern
- Tomaso Patarnello
- TR Gregory
- TR Gregory
- V Brekhman
- V Siegel
- W Minxiao
- W Pett
- WC Black
- X Shen
- X Shen
- Y Li
- Z Shao
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 22/08/2017
- Field of study
Author Posting. © The Author(s), 2017. This is the author's version of the work. It is posted here for personal use, not for redistribution. The definitive version was published in Bucklin, Ann et al. "Population Genomics of Marine Zooplankton." Population Genomics: Marine Organisms. Ed. Om P. Rajora and Marjorie Oleksiak. Springer, 2018. doi:10.1007/13836_2017_9.The exceptionally large population size and cosmopolitan biogeographic distribution that
distinguish many – but not all – marine zooplankton species generate similarly exceptional patterns of
population genetic and genomic diversity and structure. The phylogenetic diversity of zooplankton has
slowed the application of population genomic approaches, due to lack of genomic resources for closelyrelated
species and diversity of genomic architecture, including highly-replicated genomes of many
crustaceans. Use of numerous genomic markers, especially single nucleotide polymorphisms (SNPs), is
transforming our ability to analyze population genetics and connectivity of marine zooplankton, and
providing new understanding and different answers than earlier analyses, which typically used
mitochondrial DNA and microsatellite markers. Population genomic approaches have confirmed that,
despite high dispersal potential, many zooplankton species exhibit genetic structuring among geographic
populations, especially at large ocean-basin scales, and have revealed patterns and pathways of population
connectivity that do not always track ocean circulation. Genomic and transcriptomic resources are
critically needed to allow further examination of micro-evolution and local adaptation, including
identification of genes that show evidence of selection. These new tools will also enable further
examination of the significance of small-scale genetic heterogeneity of marine zooplankton, to
discriminate genetic “noise” in large and patchy populations from local adaptation to environmental
conditions and change.Support was provided by the
US National Science Foundation to AB and RJO (PLR-1044982) and to RJO (MCB-1613856); support to
IS and MC was provided by Nord University (Norway)
Study of 300,486 individuals identifies 148 independent genetic loci influencing general cognitive function
- Author
- Adams HHH
- Ahola-Olli AV
- Ames D
- Amin N
- Amouyel P
- Andreassen OA
- Arking DE
- Armstrong NJ
- Assareh AA
- Attia JR
- Attix D
- Avramopoulos D
- Barnes CLK
- Bennett DA
- Bertram L
- Bilder RM
- Bis JC
- Bitsios P
- Boerwinkle E
- Boyle PA
- Bressler J
- Brodaty H
- Burdick KE
- Böhmer AC
- Campbell H
- Cannon TD
- Chiba-Falek O
- Christoforou A
- Cirulli ET
- Congdon E
- Conley ED
- Corley J
- Corvin A
- Cox SR
- Dale AM
- Dan Rujescu
- Davies G
- De Jager PL
- Deary IJ
- Debette S
- Dehghan A
- DeRosse P
- Dick D
- Dickinson D
- Djurovic S
- Donohoe G
- Elliott P
- Eriksson JG
- Espeseth T
- Evangelou E
- Faul JD
- Fawns-Ritchie C
- Fitzpatrick AL
- Ford I
- Freimer NA
- Gale CR
- Gao H
- Giakoumaki S
- Giddaluru S
- Giegling I
- Gill M
- Gillespie NA
- Glahn DC
- Gordon SD
- Gottesman RF
- Griswold ME
- Gudnason V
- Gustavson DE
- Hagenaars SP
- Hansell NK
- Hariri AR
- Harris SE
- Harris TB
- Hartmann AM
- Hatzimanolis A
- Hayward C
- Heiss G
- Hill WD
- Hofer E
- Holliday EG
- Hägg S
- Ikram MA
- Ikram MK
- Joshi PK
- Jukema JW
- Kaprio J
- Kardia SLR
- Karlsson I
- Karlsson R
- Keller MC
- Kleineidam L
- Knopman DS
- Knowles E
- Kochan NA
- Koini M
- Konte B
- Kremen WS
- Kwok JB
- Kähönen M
- Lahti J
- Lam M
- Launer L
- Le Hellard S
- Leber M
- Lee T
- Lehtimäki T
- Lencz T
- Li S
- Li S-C
- Liewald DCM
- Lill CM
- Lindenberger U
- Liu T
- London E
- Longstreth WT
- Lopez OL
- Loukola A
- Luciano M
- Luck T
- Lundervold AJ
- Lundquist A
- Lyytikäinen L-P
- Malhotra A
- Marioni RE
- Martin NG
- Mather KA
- McIntosh AM
- Melle I
- Montgomery GW
- Morris D
- Mosley TH
- Murray AD
- Need AC
- Noordam R
- Nyberg L
- Okely JA
- Oldmeadow C
- Ollier W
- Palotie A
- Palviainen T
- Papenberg G
- Pattie A
- Payton A
- Pazoki R
- Pedersen NL
- Pendleton N
- Petrovic K
- Polasek O
- Poldrack RA
- Porteous DJ
- Psaty BM
- Raitakari OT
- Ramirez A
- Reinvang I
- Reppermund S
- Reynolds CA
- Riedel-Heller SG
- Ritchie SJ
- Rose RJ
- Rotter JI
- Roussos P
- Rovio SP
- Rudan I
- Räikkönen K
- Saba Y
- Sabb FW
- Sachdev PS
- Sargurupremraj M
- Satizabal CL
- Schmid M
- Schmidt H
- Schmidt R
- Schofield PR
- Schofield PW
- Scholz M
- Scott RJ
- Scult MA
- Seshadri S
- Simino J
- Slagboom PE
- Smith AV
- Smith JA
- Smyrnis N
- Soumaré A
- Starr JM
- Steen VM
- Stefanis NC
- Stott DJ
- Straub RE
- Sundet K
- Taylor AM
- Taylor KD
- Terzikhan N
- Thalamuthu A
- Trampush JW
- Trollor JN
- Trompet S
- Turner ST
- Tzoulaki I
- Tzourio C
- Uitterlinden A
- van der Lee SJ
- Van Duijn CM
- Villringer A
- Vitart V
- Voineskos AN
- Vuoksimaa E
- Wagner H
- Wagner M
- Ware EB
- Weinberger DR
- Weinhold L
- Weir DR
- Wen KH
- Widen E
- Wilson JF
- Windham BG
- Wright MJ
- Yang J
- Yang Q
- Yu J
- Zhao W
- Publication venue
- Publication date
- 01/01/2018
- Field of study
Correction Volume: 10, Article Number: 2068 DOI: 10.1038/s41467-019-10160-w WOS:000466339700001General cognitive function is a prominent and relatively stable human trait that is associated with many important life outcomes. We combine cognitive and genetic data from the CHARGE and COGENT consortia, and UK Biobank (total N = 300,486; age 16-102) and find 148 genome-wide significant independent loci (P <5 x 10(-8)) associated with general cognitive function. Within the novel genetic loci are variants associated with neurodegenerative and neurodevelopmental disorders, physical and psychiatric illnesses, and brain structure. Gene-based analyses find 709 genes associated with general cognitive function. Expression levels across the cortex are associated with general cognitive function. Using polygenic scores, up to 4.3% of variance in general cognitive function is predicted in independent samples. We detect significant genetic overlap between general cognitive function, reaction time, and many health variables including eyesight, hypertension, and longevity. In conclusion we identify novel genetic loci and pathways contributing to the heritability of general cognitive function.Peer reviewe
Exploration of Shared Genetic Architecture Between Subcortical Brain Volumes and Anorexia Nervosa
- Author
- Abramovic L
- Adams HHH
- Adan R
- Agartz I
- Alfredsson L
- Alhusaini S
- Almasy L
- Ames D
- Andersson M
- Ando T
- Andreassen O
- Andreassen OA
- Anttila V
- Arepalli S
- Arias-Vasquez A
- Aribisala BS
- Armstrong NJ
- Aschauer H
- Ashbrook DG
- Assareh AA
- Athanasiu L
- Baker J
- Barrett J
- Bastin ME
- Batury V-L
- Becker JT
- Bencko V
- Bergen A
- Bernard M
- Berrettini W
- Birgegard A
- Bis JC
- Blangero J
- Bohlken MM
- Boks MP
- Boni C
- Boomsma DI
- Bralten J
- Brandt H
- Breen G
- Brodaty H
- Brouwer RM
- Brown AA
- Brunner HG
- Buckner RL
- Buitelaar JK
- Bulayeva KB
- Bulik CM
- Bulik CM
- Bulik-Sullivan B
- Burghardt R
- Cahn W
- Calhoun VD
- Cannon DM
- Carlberg L
- Carless MA
- Cassina M
- Cavalleri GL
- Chakravarty MM
- Chauhan G
- Chen Q
- Cheung J
- Ching CRK
- Cichon CCS
- Cichon S
- Clementi M
- Cohen-Woods S
- Coleman J
- Cone R
- Cookson MR
- Corvin A
- Courtet P
- Crawford S
- Crespo-Facorro B
- Crow S
- Crowley J
- Cuellar-Partida G
- Curran JE
- Czisch M
- Dale AM
- Danner U
- Davies GE
- Davis O
- de Geus EJC
- de Zubicaray GI
- de Zwaan M
- Deary IJ
- Dedoussis G
- Degortes D
- Delanty N
- Den Braber A
- Depondt C
- DeSocio J
- Desrivieres S
- Dick D
- Dikeos D
- Dillman A
- Dina C
- Ding B
- Djurovic S
- Dmitrzak-Weglarz M
- Docampo E
- Donohoe G
- Drevets WC
- Duggirala R
- Duncan L
- Dyer TD
- Ebling M
- Egberts K
- Ehrlich S
- Ehrlich S
- Ehrlich S
- Erk S
- Escaramis G
- Esko T
- Espeseth T
- Espeseth T
- Estivill X
- Favaro A
- Fedko IO
- Fernandez G
- Fernandez-Aranda F
- Ferrucci L
- Fichter M
- Finan C
- Fischer K
- Fischl B
- Fisher SE
- Floyd J
- Focker M
- Foretova L
- Foroud TM
- Forzan M
- Fox C
- Fox PT
- Francks C
- Franke B
- Franklin C
- Fukunaga M
- Gallinger VGS
- Gambaro G
- Gaspar H
- Gibbs JR
- Giddaluru S
- Giegling I
- Glahn DC
- Goldman AL
- Goldstein J
- Gollub RL
- Gonidakis F
- Goring HHH
- Gorwood P
- Grabe HJ
- Gratacos M
- Green RC
- Greve D
- Grimm O
- Gruber O
- Guadalupe T
- Gudnason V
- Guelfi S
- Guillaume S
- Guo Y
- Hager R
- Hakobjan MMH
- Hakonarson H
- Halmi K
- Hansell NK
- Hardy J
- Harrison R
- Hartberg CB
- Hartman CA
- Hashimoto R
- Hass J
- Hatzikotoulas K
- Haukvik UK
- Hauser J
- Hebebrand J
- Hegenscheid K
- Heinz A
- Heister AJGAM
- Helder S
- Hendriks J
- Herms S
- Hernandez DG
- Hernandez MCV
- Herpertz-Dahlmann B
- Herzog W
- Heslenfeld DJ
- Hibar D
- Hibar DP
- Hilliard C
- Hinney A
- Ho B-C
- Hoekstra PJ
- Hoffmann W
- Hohn D
- Holmes AJ
- Holsboer F
- Homuth G
- Hoogman M
- Hosten N
- Hottenga J-J
- Huckins L
- Hudson J
- Huemer J
- Ikeda M
- Ikram MA
- Imgart H
- Inoko H
- Jack CR
- Jahanshad N
- Jahanshad N
- Jamain SJS
- Janout V
- Janowitz D
- Jenkinson M
- Jia T
- Jimenez-Murcia S
- Johnson C
- Johnson R
- Jonsson EG
- Jordan J
- Julia A
- Jureus A
- Kahn RS
- Kalsi G
- Kaminska D
- Kanai R
- Kaplan A
- Kaprio J
- Karhunen L
- Karwautz A
- Kas M
- Kasperaviciute D
- Kaye W
- Keil M
- Kennedy J
- Kennedy M
- Kent JW
- Keski-Rahkonen A
- Kiezebrink K
- Kim S
- Kim Y-R
- Klareskog L
- Klein M
- Kloszewska I
- Klump K
- Knudsen GP
- Kochunov P
- Koeleman B
- Koubek D
- Kraemer B
- Kwok JB
- La Via M
- Landen M
- Launer LJ
- Lawrie SM
- Le Hellard S
- Le Hellard S
- Leboyer M
- Lee PH
- Levitan R
- Li D
- Lichtenstein P
- Liewald DCM
- Lilenfeld L
- Lissowska J
- Liu X
- Longo DL
- Longstreth WT
- Loohuis LMO
- Lopez LM
- Lovestone S
- Lu L
- Luciano M
- Lundervold A
- Macare C
- Magistretti P
- Maj M
- Makkinje RRR
- Mannik K
- Marsal S
- Martin N
- Martin NG
- Matarin M
- Mather KA
- Mattay VS
- Mattheisen M
- Mattingsdal M
- Mazoyer B
- McDevitt S
- McDonald C
- McGuffin P
- McIntosh AM
- Mckay DR
- McMahon FJ
- McMahon KL
- Mecocci P
- Medland SE
- Meisenzahl E
- Melle I
- Merl E
- Metspalu A
- Meulenbelt I
- Meyer-Lindenberg A
- Micali N
- Milaneschi Y
- Mitchell J
- Mitchell K
- Mohnke S
- Monteleone AM
- Monteleone P
- Montgomery G
- Montgomery GW
- Morris DW
- Mortensen P
- Mostert JC
- Mueller T
- Mueller-Myhsok B
- Muhleisen TW
- Munn-Chernoff M
- Naber MAM
- Nacmias B
- Nalls MA
- Nauck M
- Navratilova M
- Needham M
- Nho K
- Nichols TE
- Nilsson I
- Nilsson LG
- Norring C
- Nothen MM
- Ntalla I
- Nugent AC
- Nyberg L
- Nyquist P
- O'Toole J
- Ohi K
- Olvera RL
- Ophoff R
- Ophoff RA
- Palotie A
- Pandolfo M
- Pantel J
- Papezova H
- Papmeyer M
- Paus T
- Pausova Z
- Penninx BWJH
- Perez-Iglesias R
- Perica VB
- Pike GB
- Pinto RPD
- Pol HEH
- Potkin SG
- Putz B
- Rabionet R
- Raevuori A
- Rajewski A
- Ramasamy A
- Ramoz N
- Rayner NW
- Reichborn-Kjennerud T
- Reinvang I
- Renteria ME
- Reppermund S
- Ricca V
- Rietschel M
- Ripatti S
- Ripke S
- Risacher SL
- Ritschel F
- Roberts M
- Roffman JL
- Roiz-Santianez R
- Romanczuk-Seiferth N
- Rose EJ
- Rosen GD
- Rotondo A
- Rujescu D
- Rujescu D
- Rybakowski F
- Ryten M
- Sachdev PS
- Salami A
- Samann PG
- Santonastaso P
- Satizabal CL
- Saykin AJ
- Scherag A
- Scherer S
- Schmaal L
- Schmidt H
- Schmidt R
- Schmidt U
- Schnell K
- Schofield PR
- Schork AJ
- Schork N
- Schosser A
- Schumann G
- Scott L
- Seiler S
- Seitz J
- Seitz J
- Seshadri S
- Shen L
- Shin J
- Simmons A
- Singleton A
- Sisodiya SM
- Slachtova L
- Sladek R
- Slagboom PE
- Slof-Op't Landt M
- Slopien A
- Smith C
- Smith T
- Smoller JW
- Soininen H
- Soranzo N
- Sorbi S
- Southam L
- Sprooten E
- Steen V
- Steen VM
- Stein JL
- Strengman E
- Strike LT
- Strober M
- Sussmann JE
- Szatkiewicz J
- Szeszenia-Dabrowska N
- Tachmazidou I
- Tenconi E
- Teumer A
- Thalamuthu A
- Thompson PM
- Thompson PM
- Thornton L
- Toga AW
- Toro R
- Tortorella A
- Tozzi F
- Trabzuni D
- Traynor BJ
- Treasure J
- Troncoso J
- Tschop M
- Tsitsika A
- Turner JA
- Tziouvas K
- van Bokhoven H
- van der Brug M
- van der Lee SJ
- van der Marel SSL
- van der Wee NJA
- van Donkelaar MMJ
- van Duijn CM
- van Eijk KR
- van Elburg A
- van Furth E
- van Haren NEM
- van Hulzen KJE
- van Tol M-J
- van't Ent D
- Veltman DJ
- Vinke LN
- Volzke H
- Wade T
- Wagner G
- Walter H
- Walters R
- Walters RK
- Walton E
- Walton E
- Walton E
- Wardlaw JM
- Wassink TH
- Watson H
- Weale ME
- Weinberger DR
- Weiner MW
- Wen W
- Westlye LT
- Westman E
- Whelan CD
- White T
- Wichmann H-E
- Widen E
- Williams RW
- Winkler AM
- Wittfeld K
- Woldehawariat G
- Wolf C
- Woodside DB
- Wright MJ
- Xue L
- Yanek LR
- Yanovski J
- Yao S
- Yilmaz Z
- Yilmaz Z
- Zerwas S
- Zielke RH
- Zipfel S
- Zonderman AB
- Zwiers MP
- Publication venue
- 'Springer Fachmedien Wiesbaden GmbH'
- Publication date
- 01/07/2019
- Field of study
In MRI scans of patients with anorexia nervosa (AN), reductions in brain volume are often apparent. However, it is unknown whether such brain abnormalities are influenced by genetic determinants that partially overlap with those underlying AN. Here, we used a battery of methods (LD score regression, genetic risk scores, sign test, SNP effect concordance analysis, and Mendelian randomization) to investigate the genetic covariation between subcortical brain volumes and risk for AN based on summary measures retrieved from genome-wide association studies of regional brain volumes (ENIGMA consortium, n = 13,170) and genetic risk for AN (PGC-ED consortium, n = 14,477). Genetic correlations ranged from − 0.10 to 0.23 (all p > 0.05). There were some signs of an inverse concordance between greater thalamus volume and risk for AN (permuted p = 0.009, 95% CI: [0.005, 0.017]). A genetic variant in the vicinity of ZW10, a gene involved in cell division, and neurotransmitter and immune system relevant genes, in particular DRD2, was significantly associated with AN only after conditioning on its association with caudate volume (pFDR = 0.025). Another genetic variant linked to LRRC4C, important in axonal and synaptic development, reached significance after conditioning on hippocampal volume (pFDR = 0.021). In this comprehensive set of analyses and based on the largest available sample sizes to date, there was weak evidence for associations between risk for AN and risk for abnormal subcortical brain volumes at a global level (that is, common variant genetic architecture), but suggestive evidence for effects of single genetic markers. Highly powered multimodal brain- and disorder-related genome-wide studies are needed to further dissect the shared genetic influences on brain structure and risk for AN
Combination of searches for heavy spin-1 resonances using 139 fb−1 of proton-proton collision data at s = 13 TeV with the ATLAS detector
- Author
- Aad G
- Aakvaag E
- Abbott B
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- Abicht NJ
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- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 19/04/2024
- Field of study
A combination of searches for new heavy spin-1 resonances decaying into different pairings of W, Z, or Higgs bosons, as well as directly into leptons or quarks, is presented. The data sample used corresponds to 139 fb−1 of proton-proton collisions at
= 13 TeV collected during 2015–2018 with the ATLAS detector at the CERN Large Hadron Collider. Analyses selecting quark pairs (qq, bb,
, and tb) or third-generation leptons (τν and ττ) are included in this kind of combination for the first time. A simplified model predicting a spin-1 heavy vector-boson triplet is used. Cross-section limits are set at the 95% confidence level and are compared with predictions for the benchmark model. These limits are also expressed in terms of constraints on couplings of the heavy vector-boson triplet to quarks, leptons, and the Higgs boson. The complementarity of the various analyses increases the sensitivity to new physics, and the resulting constraints are stronger than those from any individual analysis considered. The data exclude a heavy vector-boson triplet with mass below 5.8 TeV in a weakly coupled scenario, below 4.4 TeV in a strongly coupled scenario, and up to 1.5 TeV in the case of production via vector-boson fusion
Iron Behaving Badly: Inappropriate Iron Chelation as a Major Contributor to the Aetiology of Vascular and Other Progressive Inflammatory and Degenerative Diseases
- Author
- A Abbott
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- Publication venue
- Publication date
- 09/08/2008
- Field of study
The production of peroxide and superoxide is an inevitable consequence of
aerobic metabolism, and while these particular "reactive oxygen species" (ROSs)
can exhibit a number of biological effects, they are not of themselves
excessively reactive and thus they are not especially damaging at physiological
concentrations. However, their reactions with poorly liganded iron species can
lead to the catalytic production of the very reactive and dangerous hydroxyl
radical, which is exceptionally damaging, and a major cause of chronic
inflammation. We review the considerable and wide-ranging evidence for the
involvement of this combination of (su)peroxide and poorly liganded iron in a
large number of physiological and indeed pathological processes and
inflammatory disorders, especially those involving the progressive degradation
of cellular and organismal performance. These diseases share a great many
similarities and thus might be considered to have a common cause (i.e.
iron-catalysed free radical and especially hydroxyl radical generation). The
studies reviewed include those focused on a series of cardiovascular, metabolic
and neurological diseases, where iron can be found at the sites of plaques and
lesions, as well as studies showing the significance of iron to aging and
longevity. The effective chelation of iron by natural or synthetic ligands is
thus of major physiological (and potentially therapeutic) importance. As
systems properties, we need to recognise that physiological observables have
multiple molecular causes, and studying them in isolation leads to inconsistent
patterns of apparent causality when it is the simultaneous combination of
multiple factors that is responsible. This explains, for instance, the
decidedly mixed effects of antioxidants that have been observed, etc...Comment: 159 pages, including 9 Figs and 2184 reference
Combined measurement of the Higgs boson mass from the H → γγ and H → ZZ∗ → 4ℓ decay channels with the ATLAS detector using √s = 7, 8, and 13 TeV pp collision data
- Author
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- Zhong D
- Zhou B
- Zhou H
- Zhou N
- Zhou Y
- Zhu CG
- Zhu J
- Zhu Y
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- American Physical Society
- Publication date
- 01/01/2023
- Field of study
A measurement of the mass of the Higgs boson combining the
H
→
Z
Z
∗
→
4
ℓ
and
H
→
γ
γ
decay channels is presented. The result is based on
140
fb
−
1
of proton-proton collision data collected by the ATLAS detector during LHC run 2 at a center-of-mass energy of 13 TeV combined with the run 1 ATLAS mass measurement, performed at center-of-mass energies of 7 and 8 TeV, yielding a Higgs boson mass of
125.11
±
0.09
(
stat
)
±
0.06
(
syst
)
=
125.11
±
0.11
GeV
. This corresponds to a 0.09% precision achieved on this fundamental parameter of the Standard Model of particle physics
The Numerical Solution of Non-Linear Parabolic Equations on MIMD Parallel Computers
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/1986
- Field of study
- …