Is the Sunyaev-Zeldovich effect responsible for the observed steepening in the spectrum of the Coma radio halo ?

The spectrum of the radio halo in the Coma cluster is measured over almost two decades in frequency. The current radio data show a steepening of the spectrum at higher frequencies, which has implications for models of the radio halo origin. There is an on-going debate on the possibility that the observed steepening is not intrinsic to the emitted radiation, but is instead caused by the SZ effect. Recently, the Planck satellite measured the SZ signal and its spatial distribution in the Coma cluster allowing to test this hypothesis. Using the Planck results, we calculated the modification of the radio halo spectrum by the SZ effect in three different ways. With the first two methods we measured the SZ-decrement within the aperture radii used for flux measurements of the halo at the different frequencies. First we adopted the global compilation of data from Thierbach et al. and a reference aperture radius consistent with those used by the various authors. Second we used the available brightness profiles of the halo at different frequencies to derive the spectrum within two fixed apertures, and derived the SZ-decrement using these apertures. As a third method we used the quasi-linear correlation between the y and the radio-halo brightness at 330 MHz discovered by Planck to derive the modification of the radio spectrum by the SZ-decrement in a way that is almost independent of the adopted aperture radius. We found that the spectral modification induced by the SZ-decrement is 4-5 times smaller than that necessary to explain the observed steepening. Consequently a break or cut-off in the spectrum of the emitting electrons is necessary to explain current data. We also show that, if a steepening is absent from the emitted spectrum, future deep observations at 5 GHz with single dishes are expected to measure a halo flux in a 40 arcmin radius that would be 7-8 times higher than currently seen.Comment: 8 pages, 6 figures, accepted in Astronomy and Astrophysics (date of acceptance 19/08/2013

Magnetic field evolution in giant radio relics using the example of CIZA J2242.8+5301

Article / Letter to editorSterrewach

Cluster magnetic fields through the study of polarized radio halos in the SKA era

Galaxy clusters are unique laboratories to investigate turbulent fluid motions and large scale magnetic fields. Synchrotron radio halos at the center of merging galaxy clusters provide the most spectacular and direct evidence of the presence of relativistic particles and magnetic fields associated with the intracluster medium. The study of polarized emission from radio halos is extremely important to constrain the properties of intracluster magnetic fields and the physics of the acceleration and transport of the relativistic particles. However, detecting this polarized signal is a very hard task with the current radio facilities.We use cosmological magneto-hydrodynamical simulations to predict the expected polarized surface brightness of radio halos at 1.4 GHz. We compare these expectations with the sensitivity and the resolution reachable with the SKA1. This allows us to evaluate the potential for studying intracluster magnetic fields in the surveys planned for SKA1.Comment: 11 pages, 4 figures; to appear as part of 'Cosmic Magnetism' in Proceedings 'Advancing Astrophysics with the SKA (AASKA14)', PoS(AASKA14)10

The Coma cluster magnetic field from Faraday rotation measures

The aim of the present work is to constrain the Coma cluster magnetic field strength, its radial profile and power spectrum by comparing Faraday Rotation Measure (RM) images with numerical simulations of the magnetic field. We have analyzed polarization data for seven radio sources in the Coma cluster field observed with the Very Large Array at 3.6, 6 and 20 cm, and derived Faraday Rotation Measures with kiloparsec scale resolution. Random three dimensional magnetic field models have been simulated for various values of the central intensity B_0 and radial power-law slope eta, where eta indicates how the field scales with respect to the gas density profile. We derive the central magnetic field strength, and radial profile values that best reproduce the RM observations. We find that the magnetic field power spectrum is well represented by a Kolmogorov power spectrum with minimum scale ~ 2 kpc and maximum scale ~ 34 kpc. The central magnetic field strength and radial slope are constrained to be in the range (B_0=3.9 microG; eta=0.4) and (B_0=5.4 microG; eta=0.7) within 1sigma. The best agreement between observations and simulations is achieved for B_0=4.7 microG; eta=0.5. Values of B_0>7 microG and 1.0 are incompatible with RM data at 99 % confidence level.Comment: 23 pages, 21 figures. Higher resolution available at http://www.ira.inaf.it/~bonafede/paper.pdf. A&A accepte

An improved SPH scheme for cosmological simulations

We present an implementation of smoothed particle hydrodynamics (SPH) with improved accuracy for simulations of galaxies and the large-scale structure. In particular, we combine, implement, modify and test a vast majority of SPH improvement techniques in the latest instalment of the GADGET code. We use the Wendland kernel functions, a particle wake-up time-step limiting mechanism and a time-dependent scheme for artificial viscosity, which includes a high-order gradient computation and shear flow limiter. Additionally, we include a novel prescription for time-dependent artificial conduction, which corrects for gravitationally induced pressure gradients and largely improves the SPH performance in capturing the development of gas-dynamical instabilities. We extensively test our new implementation in a wide range of hydrodynamical standard tests including weak and strong shocks as well as shear flows, turbulent spectra, gas mixing, hydrostatic equilibria and self-gravitating gas clouds. We jointly employ all modifications; however, when necessary we study the performance of individual code modules. We approximate hydrodynamical states more accurately and with significantly less noise than standard SPH. Furthermore, the new implementation promotes the mixing of entropy between different fluid phases, also within cosmological simulations. Finally, we study the performance of the hydrodynamical solver in the context of radiative galaxy formation and non-radiative galaxy cluster formation. We find galactic disks to be colder, thinner and more extended and our results on galaxy clusters show entropy cores instead of steadily declining entropy profiles. In summary, we demonstrate that our improved SPH implementation overcomes most of the undesirable limitations of standard SPH, thus becoming the core of an efficient code for large cosmological simulations.Comment: 21 figures, 2 tables, accepted to MNRA

Radio Halos From Simulations And Hadronic Models II: The Scaling Relations of Radio Halos

We use results from a constrained, cosmological MHD simulation of the Local Universe to predict radio halos and their evolution for a volume limited set of galaxy clusters and compare to current observations. The simulated magnetic field inside the clusters is a result of turbulent amplification within them, with the magnetic seed originating from star-burst driven, galactic outflows. We evaluate three models, where we choose different normalizations for the Cosmic Ray proton population within clusters. Similar to our previous analysis of the Coma cluster (Donnert et al. 2010), the radial profile and the morphological properties of observed radio halos can not be reproduced, even with a radially increasing energy fraction within the cosmic ray proton population. Scaling relations between X-ray luminosity and radio power can be reproduced by all models, however all models fail in the prediction of clusters with no radio emission. Also the evolutionary tracks of our largest clusters in all models fail to reproduce the observed bi-modality in radio luminosity. This provides additional evidence that the framework of hadronic, secondary models is disfavored to reproduce the large scale diffuse radio emission of galaxy clusters. We also provide predictions for the unavoidable emission of $\gamma$-rays from the hadronic models for the full cluster set. None of such secondary models is yet excluded by the observed limits in $\gamma$-ray emission, emphasizing that large scale diffuse radio emission is a powerful tool to constrain the amount of cosmic ray protons in galaxy clusters

Immunolocalization of fibroblast growth factor-1 (FGF-1), its receptor (FGFR-1), and fibroblast-specific protein-1 (FSP-1) in inflammatory renal disease

Immunolocalization of fibroblast growth factor-1 (FGF-1), its receptor (FGFR-1), and fibroblast-specific protein-1 (FSP-1) in inflammatory renal disease.BackgroundThe fibroblast growth factor (FGF) family has functions in development, cell proliferation, migration, and differentiation. While FGF-2 induces fibrosis, the role of FGF-1 in inflammation and fibrosis is less defined. We examined the expression of FGF-1 and FGF receptor (FGFR-1) to determine if renal diseases with varying etiologies of inflammation, including lupus nephritis (LN), acute interstitial nephritis (AIN) and acute rejection superimposed on chronic allograft nephropathy (CAN), showed varying patterns of expression. We also examined the expression of fibroblast-specific protein-1 (FSP-1), which has been linked to epithelial-mesenchymal transition (EMT) and fibrosis, to determine whether it was linked to potential profibrotic and inflammatory FGF-1 mechanisms.MethodsProliferative LN (PLN) (N = 12), nonproliferative lupus nephritis (NPLN) (N = 5), AIN (N = 6), CAN (N = 4), and normal kidneys (N = 3) were studied. FGF, FGFR-1, and FSP-1 were localized by immunohistochemistry, and intensity scored on a 0 to 3+ scale. Double staining with CD68 and separate immunohistochemical staining for CD4 and CD8 with serial sections analysis were done to identify if T lymphocytes or macrophages showed staining for FGF-1 and FGFR-1 or FSP-1.ResultsIn normal kidneys, FGF-1 was expressed in mesangial cells (0.67 ± 0.58), glomerular endothelial (0.67 ± 0.58), visceral, and parietal epithelial cells (1.67 ± 0.58). FGFR-1 showed a similar pattern of staining but also was expressed in tubular epithelium, and arterial endothelium and smooth muscle. Expression of FGF-1 was increased over normal in glomerular parenchymal cells only in CAN in podocytes (2.30 ± 0.58 vs. 3.00 ± 0.00) (P < 0.05) and parietal epithelial cells (1.67 ± 0.58 vs. 2.25 ± 0.50) (P < 0.05). Infiltrating glomerular and interstitial inflammatory cells in diseased glomeruli also expressed FGF-1 and FGFR-1. Tubular cells expressed slightly increased FGFR-1 in renal diseases vs. normal, whereas tubules remained negative for FGF-1 in diseased kidneys. FSP-1 expression was prominent in the interstitium in all kidneys with interstitial inflammation, and most prominent in CAN. Interstitial FSP-1+ cells were consistent with a myofibroblast-type morphology, and did not stain with CD-68. FSP-1 expression was closely associated with inflammatory cells expressing FGF-1 and FGFR-1. FSP-1 also showed positivity within crescents and occasional podocytes in PLN.ConclusionThe expression of FGF-1 and FGFR-1 in infiltrating lymphocytes and macrophages, and of FGFR-1 in tubules, is supportive, but does not prove causality, of the possibility that FGF-1 might have both autocrine and paracrine functions in renal inflammation. However, the initial stimulus for renal inflammation, whether immune complex, hypersensitivity or rejection, did not alter expression patterns of FGF-1 or its receptor. The colocalization of inflammatory infiltrates with interstitial fibrosis supports the possibility of a contribution of FGF-1 for chemotaxis and associated fibrosis, further supported by interstitial FSP-1 expression closely associated with these inflammatory cells expressing FGF-1 and FGFR-1

Macromolecular-scale resolution in biological fluorescence microscopy

We demonstrate far-field fluorescence microscopy with a focal-plane resolution of 15–20 nm in biological samples. The 10- to 12-fold multilateral increase in resolution below the diffraction barrier has been enabled by the elimination of molecular triplet state excitation as a major source of photobleaching of a number of dyes in stimulated emission depletion microscopy. Allowing for relaxation of the triplet state between subsequent excitation–depletion cycles yields an up to 30-fold increase in total fluorescence signal as compared with reported stimulated emission depletion illumination schemes. Moreover, it enables the reduction of the effective focal spot area by up to ≈140-fold below that given by diffraction. Triplet-state relaxation can be realized either by reducing the repetition rate of pulsed lasers or by increasing the scanning speed such that the build-up of the triplet state is effectively prevented. This resolution in immunofluorescence imaging is evidenced by revealing nanoscale protein patterns on endosomes, the punctuated structures of intermediate filaments in neurons, and nuclear protein speckles in mammalian cells with conventional optics. The reported performance of diffraction-unlimited fluorescence microscopy opens up a pathway for addressing fundamental problems in the life sciences