12 research outputs found
The catalytically active secretory phospholipase A2 type IIA is involved in restenosis development after PTCA in human coronary arteries and generation of atherogenic LDL
No full textInhibition of Secretory Phospholipase A2 in Patients with Acute Coronary Syndromes: Rationale and Design of the Vascular Inflammation Suppression to Treat Acute Coronary Syndrome for 16 Weeks (VISTA-16) Trial
No full textEarly regulation of primary metabolism, antioxidant, methyl cycle and phenylpropanoid pathways during the Mycosphaerella fijiensis-Musa spp. interaction
No full textProfiling population-level diversity and dynamics of Accumulibacter via high throughput sequencing of ppk1
No full textComammox Nitrospira within the Yangtze River continuum: community, biogeography, and ecological drivers
No full textLow yield and abiotic origin of N2O formed by the complete nitrifier Nitrospira inopinata
No full textA secreted metal-binding protein protects necrotrophic phytopathogens from reactive oxygen species
No full textCell biology and immunology of Listeria monocytogenes infections: novel insights
No full textInternational audienceListeria monocytogenes is an intracellular bacterium responsible for a disease characterized by several clinical features, such as septicemia, brain infection, abortion, and perinatal infection. These clinical features are attributed to its amazing capacity to cross several barriers in susceptible hosts. Intracellular infection is a consequence of the bacterium's capacity to enter a wide variety of mammalian cells, to not only survive but also replicate therein, and to its faculty to spread from one cell to the next, thereby escaping the humoral immune response. Here, we review both the well-established and the newly discovered strategies used by this bacterium to achieve this intracellular lifestyle while escaping from the host innate immune response. More than ever, Listeria appears as a model system and a reference in infection biology
