51 research outputs found
Goat meat products
In general, goat meat is not inferior to other meat types regarding nutritional and biological value-it has a high protein content (up to 29%), and it is a good source of minerals, vitamin B-complex, and essential amino acids. However, the meat of older and culled goats is less juicy, less tender, has a characteristically different odour and taste compared to kids' goat meat (and meat of other animals), and thus tends to be less desirable. Different meat products could be produced using goat meat (including culled goat meat): dry-fermented sausages (e.g. sucuk), dry-cured meats (Violino di capra-goat prosciutto), frankfurters, mortadella, etc. without adverse effects on products' technological properties. The negative impact of goat meat on the properties of meat products is mainly associated with the use of goat fatty tissue. However, this could be overcome by using fatty tissue of other animals (e.g. pork back fat or beef fatty tissue)
The pseudogap state in superconductors: Extended Hartree approach to time-dependent Ginzburg-Landau Theory
It is well known that conventional pairing fluctuation theory at the Hartree
level leads to a normal state pseudogap in the fermionic spectrum. Our goal is
to extend this Hartree approximated scheme to arrive at a generalized mean
field theory of pseudogapped superconductors for all temperatures . While an
equivalent approach to the pseudogap has been derived elsewhere using a more
formal Green's function decoupling scheme, in this paper we re-interpret this
mean field theory and BCS theory as well, and demonstrate how they naturally
relate to ideal Bose gas condensation. Here we recast the Hartree approximated
Ginzburg-Landau self consistent equations in a T-matrix form. This recasting
makes it possible to consider arbitrarily strong attractive coupling, where
bosonic degrees of freedom appear at considerably above . The
implications for transport both above and below are discussed. Below
we find two types of contributions. Those associated with fermionic
excitations have the usual BCS functional form. That they depend on the
magnitude of the excitation gap, nevertheless, leads to rather atypical
transport properties in the strong coupling limit, where this gap (as distinct
from the order parameter) is virtually -independent. In addition, there are
bosonic terms arising from non-condensed pairs whose transport properties are
shown here to be reasonably well described by an effective time-dependent
Ginzburg-Landau theory.Comment: 14 pages, 5 figures, REVTeX4, submitted to PRB; clarification of the
diagrammatic technique added, one figure update
Nernst Effect and Anomalous Transport in Cuprates: A Preformed-Pair Alternative to the Vortex Scenario
We address those puzzling experiments in underdoped high
superconductors which have been associated with normal state "vortices" and
show these data can be understood as deriving from preformed pairs with onset
temperature . For uncorrelated bosons in small magnetic fields, and
arbitrary , we present the exact contribution to \textit{all}
transport coefficients. In the overdoped regime our results reduce to those of
standard fluctuation theories (). Semi-quantitative agreement
with Nernst, ac conductivity and diamagnetic measurements is quite reasonable.Comment: 9 pages, 4 figures; Title, abstract and contents modified, new
references added, figures changed, one more figure added; to be published on
PR
Unboxing mutations: Connecting mutation types with evolutionary consequences
A key step in understanding the genetic basis of different evolutionary outcomes (e.g., adaptation) is to determine the roles played by different mutation types (e.g., SNPs, translocations and inversions). To do this we must simultaneously consider different mutation types in an evolutionary framework. Here, we propose a research framework that directly utilizes the most important characteristics of mutations, their population genetic effects, to determine their relative evolutionary significance in a given scenario. We review known population genetic effects of different mutation types and show how these may be connected to different evolutionary outcomes. We provide examples of how to implement this framework and pinpoint areas where more data, theory and synthesis are needed. Linking experimental and theoretical approaches to examine different mutation types simultaneously is a critical step towards understanding their evolutionary significance
Schimke immunoosseous dysplasia: defining skeletal features
Schimke immunoosseous dysplasia (SIOD) is an autosomal recessive multisystem disorder characterized by prominent spondyloepiphyseal dysplasia, T cell deficiency, and focal segmental glomerulosclerosis. Biallelic mutations in swi/snf-related, matrix-associated, actin-dependent regulator of chromatin, subfamily a-like 1 (SMARCAL1) are the only identified cause of SIOD, but approximately half of patients referred for molecular studies do not have detectable mutations in SMARCAL1. We hypothesized that skeletal features distinguish between those with or without SMARCAL1 mutations. Therefore, we analyzed the skeletal radiographs of 22 patients with and 11 without detectable SMARCAL1 mutations. We found that patients with SMARCAL1 mutations have a spondyloepiphyseal dysplasia (SED) essentially limited to the spine, pelvis, capital femoral epiphyses, and possibly the sella turcica, whereas the hands and other long bones are basically normal. Additionally, we found that several of the adolescent and young adult patients developed osteoporosis and coxarthrosis. Of the 11 patients without detectable SMARCAL1 mutations, seven had a SED indistinguishable from patients with SMARCAL1 mutations. We conclude therefore that SED is a feature of patients with SMARCAL1 mutations and that skeletal features do not distinguish who of those with SED have SMARCAL1 mutations
Microwave Surface Impedance and Complex Conductivity of High-T c Single Crystals: Current State and Unsolved Problems
Increased Amyloid Precursor Protein and Tau Expression Manifests as Key Secondary Cell Death in Chronic Traumatic Brain Injury
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